• Embed Doc
  • Readcast
  • Collections
  • CommentGo Back
Download
 
Pediatric Asthma
OS 213: Pulmunology
Maria Liza B. Zabala, M.D.Exam 1
Dec 11, 2008 | Thursday
Page 1 of 8SexyBacks
 
Lecture Outline
I.
Epidmemiology
II.
Definition
III.
Review of Anatomy
IV.
Pathophysiology
V.
Risk Factors
VI.
Clinical Features/Diagnosis
VII.
Management
EPIDEMIOLOGY
Asthma ranked number 1 among the non-infections admissions in 57 of accreditedhospitalsPPS Registry of Diseases, 1994
Prevalence of wheezing among 6-19 years inMetro Manila schools was 27.45%Del Mundo, textbook of Pediatrics 2002
A large international survey study of childhoodasthma prevalence in 56 countries found a widerange in asthma prevalence, from 1.6 to 36.8%ISAAC Study
Source
: Masoli M et al. Allergy 2004
DEFINITION
a chronic inflammatory disorder of the airways inwhich many cells play a role, including mast cellsand eosinophils
this
inflammation
causes symptoms that areusually associated with widespread but variableairflow
obstruction
that is often reversible either spontaneously or with treatment, and causesassociated increase in
airway hyperresponsiveness
to a variety of stimuli.
a disorder defined by its clinical, physiologicaland pathological characteristics
Clinically, asthma is characterized by airwayhyperresponsiveness presenting as widespreadnarrowing of the airway which results from avariety of stimuli like allergens, exercise, physicalfactors and irritant gases
Primary physiologic manifestation isspontaneously variable airway obstruction whichcan be modulated by:
Increased obstruction caused by manystimuli
Alleviation of obstruction bybronchodilators and/or anti-inflammatoryagents
REVIEW OF ANATOMYPATHOPHYSIOLOGY
is complex and involves the following components:1)Airway inflammation2)Intermittent airflow obstruction3)Bronchial hyperresponsiveness
 
Pediatric Asthma
OS 213: Pulmunology
Maria Liza B. Zabala, M.D.Exam 1
Dec 11, 2008 | Thursday
Page 2 of 8SexyBacks
Asthma Inflammation: Cells and MediatorsKey Mediators of Asthma:
Chemokines
recruitment of inflammatory cells into theairways and are mainly expressed inairway epithelial cells
Cysteinyl leukotrienes
potent bronchoconstrictors andproinflammatory mediators mainlyderived from mast cells and eosinophils
only mediator whose inhibition has beenassociated with an improvement in lungfunction and asthma symptoms
Cytokines
orchestrate the inflammatory responsein asthma and determine its severity
Histamine
contributes to bronchoconstriction andto the inflammatory response
Nitric Oxide
a potent vasodilator, producedpredominantly from the action of inducible nitric oxide synthase in airwayepithelial cellAsthma Inflammation: Cells and MediatorsMechanisms Of Airway Narrowing in Asthma
Contraction of Airway smooth muscle (ASM) isthe predominant mechanism largely reversed bybronchodilators
Airway wall thickening
Accumulation of airway secretions, mucus casts,and cellular debris may partially occlude thelumenRegulation of Airway Caliber 
Cholinergic (parasympathetic) motoneuronsinnervate the airways via the vagus nerve
Nonadrenergic Noncholinergic (NANC) Nervoussystem
NANC system neurons in the vagus nerverelease the peptides, SUBSTANCE P andVASOACTIVE INTESTINAL PEPTIDE
Appears to be the most potent relaxantcomponent of the nervous system involved inregulation of airway diameter Factors that Influence Asthma Development andExpressionHost Factors
Genetic
Atopy
Airway hyperresponsiveness
Gender 
ObesityEnvironmental Factors
Indoor allergens
Outdoor allergens
Occupational sensitizers
Tobacco smoke
Air Pollution
Respiratory Infections
Diet
RISK FACTORS FOR ASTHMA
Host factors: predispose individuals to, or protectthem from, developing asthma
Environmental factors: influence susceptibility todevelopment of asthma in predisposedindividuals, precipitate asthma exacerbations,and/or cause symptoms to persistWho gets asthma? Anyone!!!
Most children develop asthma before age 8 yearsand over half before 3 years
 
Pediatric Asthma
OS 213: Pulmunology
Maria Liza B. Zabala, M.D.Exam 1
Dec 11, 2008 | Thursday
Page 3 of 8SexyBacks
30% < 1 year 
80-90% before 4-5 years old
Before puberty: asthma occurs 11/2-3x male >female
Adolescence male=femalePredisposing Factors involved in the Development of Asthma
Atopy
defined as the preponderance toproduce abnormal amounts of IgE inresponse to environmental allergens
Familial association among asthma,allergic rhinitis and atopic dermatitissuggests a common genetic basis-chromosomes 5, 11
90% of asthmatic children have anallergic component
64-84% (+) family history of asthmaamong 1st degree relatives
30% & 3.5% of asthmatic patientsreported asthma in one parent and innone respectively
Gender 
Male preponderanceTriggers
Risk factors that cause asthma exacerbation byinducing inflammation or provoking acutebronchoconstriction or bothTrigger Factors of Asthma in Various Age GroupsAnatomic and physiologic peculiarities that predispose toobstructive airway disease1.Decreased amount of smooth muscle inperipheral airways2.Mucosal gland hyperplasia in the major bronchicompared to adults favors increased intraluminalmucus production3.Disproportionately narrow peripheral airways upto 5 years of age4.Decreased static elastic recoil of the young lungpredisposes to early airway closure during tidalbreathing5.Highly compliant rib cage and mechanicallydisadvantageous angle of insertion of diaphragmto ribcage increases diaphragmatic work of breathing6.Decreased number of fatigue-resistant skeletalmuscles in the diaphragm7.Deficient collateral ventilation with the pores of Kohn and the Lambert canals deficient in number and sizeMasqueraders of asthma in children
Upper airway noise/congestion
Cystic fibrosis (CF)
Gastroesophageal reflux disease (GERD)
Bronchopulmonary dysplasia (BPD)
Foreign body aspiration
Immunodeficiency (ID)
Vocal cord dysfunction
CLINICAL FEATURES
Frequent episodes of wheeze (more than once amonth)
Activity induced cough or wheeze
Nocturnal coughs in periods without viralinfections
Absence of seasonal variations in wheeze
Symptoms that persist after the age of 3
Wheeze before the age of 3 and one major riskfactor 
parental history of asthma or eczema or two or three risk factors (eosinophilia,wheezing without colds, and allergicrhinitis) has been shown to predict thepresence of asthma in later childhood
DIAGNOSIS
Signs and symptoms to look for include:
Frequent coughing spells, which may occur during play, at night, or while laughing. It isimportant to know that cough may be the onlysymptom present.
Less energy during play
Rapid breathing
Complaint of chest tightness or chest "hurting”
Whistling sound (wheezing) when breathing inor out
See-saw motions (retractions) in the chestfrom labored breathing
Shortness of breath, loss of breath
Tightened neck and chest muscles
Feelings of weakness or tirednessSpirometry
Recommended in the initial assessment of patients suspected to have asthma
of 00

Leave a Comment

You must be to leave a comment.
Submit
Characters: ...
You must be to leave a comment.
Submit
Characters: ...