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The History of Herpes Cold Sores - Part 1

The History of Herpes Cold Sores - Part 1

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Published by Dan Adams
The Herpes Cold Sore History - Part 1 is a journey through time as the cold sore virus evolves and adapts to the human body as its living host. Learn why a Herpes cure hasn't been possible and discover how this tricky virus manages to spread so easily from person to person.
The Herpes Cold Sore History - Part 1 is a journey through time as the cold sore virus evolves and adapts to the human body as its living host. Learn why a Herpes cure hasn't been possible and discover how this tricky virus manages to spread so easily from person to person.

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Published by: Dan Adams on Oct 27, 2009
Copyright:Attribution Non-commercial


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The fight probably began millions of years ago when Oxygen began to replace Sulfur in theEarths lower atmosphere. That was when micro-life came out of the planetary waters seekingthe best host they could locate on dry land.All of these microscopic life forms were in need of varying requirements for survival. Andprogress in their adaption over millions of years helped them to determine where they couldbest survive. They were constantly evolving to adapt and survive in an ever changing world.Among those simple life forms included the especially adaptive micro-life that we now classifyas the many varieties of virus.Throughout Earth's existence these Virus have survived in twounique ways.The first way is by openly invading the host in an attempt toconsume all that is needed from the host to survive. Suchinvasion is readily recognized by the host immune system anda life and death battle eventually determines the outcome.Either the virus is conquered or the host dies. A primeexample of this particular type would be a flu virus.But the second method is a much more highly evolved virusthat has learned to live within the host without doing anything that would alert the immunesystem.The Herpes Virus falls in this category as a compatible virus in human hosts. Over millions of years the Herpes Virus has adapted not only to the human host but has found the best ways touse us to reproduce its own species. It wisely chose areas of human intimate contact to provideeasy transfer and replication using the human oral and sexual areas of body,Once the Herpes Virus enters a host it follows down a nerve to hibernate which helps it avoid afull force attack from the human host immune system. Whenever the Herpes Virus senses thatthe host immune system is preoccupied or weakened it heads to the skins surface to seek anew host to infect. After infecting the surface skin cells it retreats back into hibernation before
the immune system can attack and destroy it. (This ability to run and hide is why a cure has notbeen found for the Herpes Virus)Of course, that still leaves the symptoms of the Herpes Virus to contend with. As previouslystated the Herpes Virus is a real opportunist. It only attacks the surface of the skin when thehost immune system is in a weakened condition. When the Virus senses an opportunity itascends to the skins surface and infects the local skin cells.The body recognizes this as an invasion from the outside of the host body rather than from theinside. This causes the immune system to send help to the skin damaged area caused by theVirus rather than trace down the actual virus that is hiding within the body.Because of the body misdirecting the white cells to the skin rather than seeking out the hidingHerpes Virus the resident Virus escapes unscathed while the skin erupts in a battle of whitecells versus the alien skin invasion.The resulting battle creates a developing tender redness as the skin becomes inflamed but isquickly acted on by the body when the Virus causes the skin to open (split). At this point thebody is not only fighting the Virus but is now engaged in a totally different struggle againstinvading bacteria that has entered Simply because the skin is considered by the host body to bethe FRONT LINE of protection for the entire body.The huge battle between the white cells surrounding the Virus in battle causes dead Virus cellsand dead bacteria to become a swelling pus pocket that becomes covered with drying cells tocreate a scab. As the battle rages on the huge army of sacrificing white cells eventually beatback the virus and invading bacteria. When the battle is over the red cells come in to repair thedamaged skin.One thing that can complicate this entire process is the removal of the crusted scab at thewrong time. If the scab is removed while the battle is still in progress the removal of the scabwill allow a renewed invasion of bacteria. This stalls the battle of the white cells which allowsthe Virus to regain strength and can even give it an extra opportunity to spread to other areas.So, premature removal of the scab is probably not your best move.But that is not the only way the Herpes Virus can spread. Days before a sore becomes visible ithas already invaded skin cells and several days after the sore seems to have gone away thereare still infected cells that are slothing off. So a simple touch of the contaminated area can helptransfer the Virus to other hosts and even to other areas on the original host.

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