statistically higher in healthy turkeys fed a diet free of monensin than in the livers of birds from the 3 groups exposed to monensin. This suggests that the concentrationof monensin in serum positively correlates to the severity of clinical signs and pathologyand to the amount of monensin in the feed. The current study also suggeststhat monensinin the feed could induce lower concentrations of vitamin E in the liver of turkeys and can predispose the turkeys to knockdown syndrome.
Diffuse intestinalT-cell lymphosarcomain a yellow-naped Amazon parrot(Amazona ochrocephala auropalliata). J Vet Diagn Invest 20:656–660 (2008).
Lymphosarcoma (LSA) is themost commonlyreported lymphoid neoplasm in parrots.
Pathology of Natural Infections byH5N1 Highly Pathogenic Avian Influenza Virusin Mute(Cygnus olor) and Whooper (Cygnus cygnus) Swans.
Vet Pathol 44:137–143(2007). multifocalhemorrhagic necrosis in the pancreas, pulmonary congestion andedema, and subepicardial hemorrhages. Major histologic lesions were acute pancreaticnecrosis, multifocal necrotizing hepatitis, andlymphoplasmacytic encephalitis withneuronal necrosis. Adrenals displayed consistently scattered cortical and medullarynecrosis. In spleen and Peyer’s patches, mild lymphocyte necrosis was present. In the brain, a large number of neurons and glial cells, especially Purkinje cells, showedimmunostaining.
Pathology and Virus Tissue Distribution of Turkey Origin ReovirusesinExperimentally Infected Turkey Poults.
Vet Pathol 44:185–195 (2007). bursalatrophy characterized by lymphoid depletion and increased fibroplasiabetween the bursalfollicles. Both IHC and ISH revealed viral antigen andRNA in the surface epithelialcellsof the bursa, in macrophages in the interstitium of the bursa and, to lesser degree, insplenic red pulp macrophages and intestinal epithelial cells. The lymphoid depletionobserved in the bursa appears to be the effect of anindirectly induced apoptosis andwould most likely result in immune dysfunction in poults infected with TRV.
Pathologic Findings of Highly Pathogenic Avian Influenza VirusA/Duck/Vietnam/12/05 (H5N1) in Experimentally Infected Pekin Ducks, Based onImmunohistochemistry and In Situ Hybridization
. Vet Pathol 44:635–642 (2007).Clinical signs -conjunctivitis and slight depression, severe neurologic signs consisting of torticollis, incoordination, tremors, and seizures. Gross lesions- hemorrhages in theduodenum, ceca, proventriculus, ventriculus, trachea,
pancreas, and brain
. Histologiclesions, as well as immunohistochemistry positivity, were recorded in the
Fatal Coxiellosisin Swainson's Blue Mountain Rainbow Lorikeets Vet Path 2008 Mar:
signs= head pressing, hemiparesis,
seizures, obtunded mentation. Grossly= hepatomegaly, splenomegaly; Histo=disseminated microgranulomas
in the liver, spleen, and brain, andlymphohistocytic perivascular encephalitis and cephalic vasculitis. Bacteria were Gimenez and PAS positive.
Feather-picking Psittacines: Histopathology and Species Trends Vet Path 2008 May
: Inflammatoryskin disease was diagnosed in 210 birds, and traumatic skin disease was diagnosed in 198 birds. Theinflammatory cells associated with the lesions were typically lymphocytes and occasionally plasma