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Vol. 22, No. 5May 2000
Refereed Peer Review
FOCAL POINTKEY FACTS
#
Several traditional antifungalcompounds are still widely usedand recommended for manycutaneous fungal disorders.
Traditional AntifungalDermatologic Agents
Centre Vétérinaire DMV, Ville St-Laurent, Quebec
Caroline de Jaham, DMV, MSc
Université de Montréal
Manon Paradis, DMV, MVSc
North Carolina State University 
Mark G. Papich, DMV, MS
ABSTRACT:
In the past two decades, remarkable advances have occurred in the treatment ofcutaneous fungal diseases. New generations of antifungal drugs have altered the therapeuticapproaches to both systemic and superficial mycoses of companion animals. To understandthese advances and their impact in veterinary dermatology, traditional therapies and their clini-cal applications are reviewed. The pharmacokinetics, methods of action, principal adverse ef-fects, and dermatologic uses of polyenes, iodides, and griseofulvin are summarized.
 A 
ntifungal agents target the cytoplasmic membrane and nucleus of cells(Figure 1). Older antifungal agents, including ampheroticin B, remainthe mainstay of treatment of certain fungal infections despite the adventof newer agents (Table I). The antifungal agents discussed in this article includethe polyenes, flucytosine, iodides, and griseofulvin.
POLYENES
The two clinical polyene macrolide antibiotics used in veterinary dermatology are nystatin and amphotericin B. Approximately 87 polyene antibiotics havebeen developed since the discovery of nystatin in 1950.
1
Nystatin
Because systemic administration of nystatin is associated with a high risk fortoxicity, only the topical preparation is used in veterinary medicine. Nystatin, acommon ingredient in over-the-counter products, is active against the yeast
Malassezia pachydermatis 
, a frequent complicating organism of otitis externa.Topical ear preparation of nystatin has been incriminated as an occasional causeof allergic contact dermatitis in the topical treatment of canine otitis externa.
2
Amphotericin B
Discovered in 1956,
3
amphotericin B was the first effective systemic antifungalagent and rapidly became the treatment of choice for systemic mycoses. Thedrug is still recommended for invasive fungal infections despite its renal toxicity 
CE
V
I
Amphotericin B continues to be arecommended treatment option forseveral invasive fungal infectionsdespite the need for intravenousadministration and the possibilityof nephrotoxicity.
I
Interest in the antifungal drugamphotericin B has beenrenewed because encapsulatedformulations, have a highermargin of safety than does thetraditional formulation.
I
Potassium iodide remains aneconomical, efficient initialtreatment of choice for caninesporotrichosis.
I
Although ineffective againstyeast, griseofulvin is highlyeffective against dermatophytesand remains the treatment ofchoice.
I
Cats are apparently susceptible tothe adverse effects of griseofulvintherapy.
 
and the need for intravenous(IV) administration. Interestin this drug has been re-newed because encapsulatedformulations, such as am-photericin B lipid complex,have a higher margin of safety than does the tradi-tional formulation (ampho-tericin B deoxycholate).
Mechanism of Action 
 Amphotericin B binds ir-reversibly to ergosterol, theprincipal sterol in the cellmembrane of fungi. Thisbinding results in cell deathcaused by altered cellularpermeability and leakage of intracellular constituents. Amphotericin B binds to a lesser extent to other sterols(e.g., cholesterol) in mammalian cells. This interactionaccounts for most of the toxicity and side effects inmammals. Amphotericin B is fungicidal in sufficiently high concentrations but is considered fungistatic atlower concentrations.
1
Pharmacokinetics 
 Amphotericin B is poorly absorbed from the gas-trointestinal tract, thereby necessitating IV infusion.The drug binds primarily to protein, resulting in poorpenetration of body fluids (e.g., cerebrospinal fluid),but intrathecal administration of amphotericin B hasbeen described.
4
The metabolism of ampho-tericin B is not clearly under-stood. It is incorporated intocholesterol cell membranesat various times, resulting incomplicated elimination anddistribution of the drug dur-ing cellular metabolism.
5
Clinical Use 
 Amphotericin B is activeagainst
Blastomyces dermati- tidis, Histoplasma capsulatum,Cryptococcus neoformans,Coccidioides immitis,
Muco-rales
, Candida 
species,
 As-  pergillus 
species,
Sporothrix schenckii 
, and
Fusarium 
species. The minimum inhibitory concentration of am-photericin B varies for each group of fungi. The drug ismost commonly used to treat blastomycosis, histoplas-mosis, and coccidioidomycosis, all of which can havecutaneous manifestations as markers of systemic infec-tion. Despite the availability of newer agents, ampho-tericin B combined with flucytosine has remained thetreatment of choice for humans with cryptococcosis.
6
Fungal resistance rarely develops during treatment with amphotericin B; however, relapse may occur whentherapy is discontinued.
4
Concurrent use of other drugs(e.g., flucytosine, the azole derivatives) improves theoverall efficacy of therapy and has been shown to re-duce the number of relapses.
7,8
 Amphotericin B remainsa mainstay of initial therapy; but because of the need
Small Animal/Exotics
Compendium 
May 2000
ERGOSTEROL
I
CELLULAR METABOLISM
I
FLUCYTOSINE
I
FUNGAL RESISTANCE
TABLE IClass and Site of Action of Antifungal Agents
ClassAgentsSite of Actio
PolyenesAmphotericin B,Bind tonystatin, natamycinergosterol anddisrupt cellmembranesMiscellaneousFlucytosineInhibits RNsynthesisGriseofulvinInterferes withspindlemicrotubulesIodidesUnknownTolnaftateInhibitsqualeneepoxidase
Figure 1—
Schematic representation of a fungal cell and target sites of action of antifungal agents.
Cytoplasmic Membrane
Contains ergosterol asthe major sterol• Site of amphotericin B activity:binds to ergosterol and disruptsthe cell membrane• Site of azole activity: inhibits theenzyme lanosterol 14-demethylase,thereby inhibiting ergosterolsynthesis• Site of allylamine activity: inhibitssqualene epoxidase enzyme,thereby inhibiting ergosterolsynthesis
Nucleus
Contains nucleic acid• Site of griseofulvin activity:interferes with spindlemicrotubules, thereby inhibitingnucleic acid synthesis• Site of flucytosine activity:converts to 5-fluorouracil incytoplasm of fungi, therebyinhibiting RNA synthesis
Cell Wall
Contains chitin
 
for hospitalization, intensive patient monitoring, labo-ratory testing, and fluid administration during IV ther-apy, the cost of amphotericin B treatment is similar tothat of the newer, more expensive azole derivatives.Dose regimens of amphotericin B to treat specific dis-eases in companion animals have been reviewed, andthis information should be consulted before veterinari-ans attempt treatment using amphotericin B.
4,9
Administration and Dose 
 Amphotericin B is usually administered IV, and rapidand slow IV infusion techniques for amphotericin Bhave been described.
9,10
The rapid IV infusion tech-nique involves administration of boluses of 0.25 to 0.5mg/kg amphotericin B diluted in 30 to 120 ml of 5%dextrose over 10 to 15 minutes. Although these tech-niques have been widely used, the risk for renal toxicity is higher with this method than with slower infusiontechniques.
10
The slower technique requires an in-dwelling catheter and 0.25 to 0.5 mg/kg amphotericinB diluted in 250 to 1000 ml of 5% dextrose adminis-tered slowly for 4 to 6 hours. Before beginning eachtreatment, the packed cell volume, total protein, creati-nine, blood urea nitrogen, and potassium should bemeasured and urinalysis performed. Regardless of theIV infusion technique used, amphotericin B shouldonly be administered every other day to minimize ad-verse effects. Amphotericin B has also been used orally for local-ized treatment of gastrointestinal candidiasis.
11,12
Morerecently, subcutaneous infusion of 0.5 to 0.8 mg/kgamphotericin B diluted in 400 or 500 ml of 0.45%saline containing 2.5% dextrose (for dogs and cats, re-spectively) has been successful in treating cryptococco-sis. Two dogs and three cats received infusions two tothree times a week for several months, and local irrita-tion was only observed when concentrations of ampho-tericin B exceeded 20 mg/L.
13
Adverse Effects 
The most common and serious side effect of ampho-tericin B administration is both acute and chronic nephro-toxicosis. Although clinical signs of early and acute re-versible nephrotoxicosis can occur with each daily dose,permanent renal dysfunction is related to the total cu-mulative dose of amphotericin B. A maximum total cu-mulative dose of 4 to 8 mg/kg is commonly described;but the total cumulative dose, and ultimately the treat-ment duration, is always limited by nephrotoxicosis.
4
Cats are more sensitive to this disorder than are dogs,and the maximum cumulative dose recommended incats is 4 mg/kg.
14
Several treatments have been used toprevent or delay nephrotoxicosis associated with am-photericin B administration. Concomitant administra-tion of mannitol, sodium bicarbonate, furosemide,dopamine, and aminophylline has been tried empirical-ly or experimentally; some of these agents have shownempiric beneficial effects.
4
Pretreatment diuresis withsaline-containing fluids helps decrease nephrotoxici-ty.
4,14
The availability of new formulations of ampho-tericin B in a lipid or cholesterol complex allows ad-ministration of amphotericin B at higher doses withgreater safety.
15
Three formulations are now available:an amphotericin B–cholesteryl sulfate complex; a lipo-somal complex of amphotericin B; and an ampho-tericin B lipid complex, which is a suspension of am-photericin B combined with two phospholipids. Theamphotericin B lipid complex was shown to be safe andeffective for treating blastomycosis in dogs at a dose of 8 to 12 mg/kg.
16
The liposomal complex of ampho-tericin B was used in another study of 13 dogs for treat-ment of 
Leishmania infantum 
at a dose of 3 to 3.3mg/kg.
17
 Although clinical improvement was rapid, thedogs remained positive for
Leishmania 
.Lipid and cholesterol formulations (3 mg/kg ormore) can be administered at higher doses than can theconventional formulation (0.25 to 0.5 mg/kg) and thusproduce greater efficacy with less toxicity.
18,19
Decreasedtoxicity is attributed to selective transfer of the ampho-tericin B lipid complex, which releases the drug directly to the fungal cell membrane and spares mammalian cellmembranes. Reduced drug concentrations in the kid-neys as well as release of fewer inflammatory cytokinesfrom the amphotericin B lipid complex than with theconventional formulation may also prevent adverse re-actions. Administration of amphotericin B as a subcu-taneous infusion also allows higher cumulative doses tobe administered without producing marked azotemia.
13
Other adverse effects caused by IV administration of amphotericin B are hypotension, vomiting, tremors,pyrexia, hypokalemia, anemia, and anorexia. Becausephlebitis should be expected with IV administration,the catheter site should be alternated.
4
MISCELLANEOUS SYSTEMIC AGENTS
The miscellaneous class of antifungal agents includethe systemic compounds flucytosine, potassium andsodium iodide, and griseofulvin. Also included in thisclass are numerous topical agents, such as tolnaftate, which is commonly used in human dermatology.
Flucytosine
Flucytosine is a fluorinated pyrimidine that is convert-ed into 5-fluorouracil in the cytoplasm of fungi contain-ing the enzyme cytosine permease.
4
This inhibits RNA synthesis and leads to cell death. Flucytosine is well ab-
Compendium 
May 2000Small Animal/Exotics
AZOLE DERIVATIVES
I
RENAL TOXICITY
I
LEISHMANIA INFANTUM 
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