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PATHOPHYSIOLOGY of Neurocysticercosis

PATHOPHYSIOLOGY of Neurocysticercosis

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Published by teddydeclines14
THIS IS NOT AN ACTUAL PATIENT BASED CASE PRESENTATION.
MADE BY ME... HEHEHEHE... FROM PPTS CDO...BATCH 2
THIS IS NOT AN ACTUAL PATIENT BASED CASE PRESENTATION.
MADE BY ME... HEHEHEHE... FROM PPTS CDO...BATCH 2

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Categories:Types, Research, Science
Published by: teddydeclines14 on Feb 26, 2010
Copyright:Attribution Non-commercial

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01/19/2013

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II. PATHOPHYSIOLOGY OF NEUROCYSTICERCOSISDefinition
: It is a parasitic infection in the central nervous system that is caused by a tapeworm called Taenia solium (T. solium).
Schematic Diagram:Predisposing Factors:
>Race: Hispanics ethnicity>Age: Children older than 7years old>Geographical area: LatinAmerica, Asia, Africa, Spain, andEastern Europe. California and Texas and in the city of Chicago>Immigrant status from Mexico,Central and South America>Family history of taeniasis
Precipitating Factors:
>Exposure to areas of endemicity>Poor sanitation>Poor hygiene>Use of sewage for fertilizer>Lack of controlled pens forpigs>Ingestion of undercook porkmeats>Sharing of food from thesame plateIngestion of Tapeworm larvaewith undercooked porkWorm attaches to the intestinal wall by meansof suckers and hooks and it develops in thesmall intestine by forming segments(hermaphrodite proglottids containing morethan 50,000 eggs) that arise from the caudalend of the scolex. The tapeworm matures over-
 
 The eggs are intermittentlypushed out from the proglottidinto the intestine andproglottids may be shed in theIngestion of food containing eggs that aretransferred from anus to mouth by unclean handsof an infected personDiagnostic:Fecalysis:Parasites:Opaque, off-whitein color, 1-2 cmlong, 1 cm wide-When these eggs are ingested and exposed to gastric acid in thehuman stomach, they lose their protective capsule and release a cystform called oncosphere (the larva of the tapeworm armed with 4hooks) and becomes active in the intestinal wall. The larval cysts penetrate and erodes the intestinal mucosa andmigrate throughout the body via the vascular system to the brain,muscle, eyes and other structures and lives in tissues as fluid-filled
Human Cysticercosis
 The larval cysts lodge to the different parts of the brain with varying macroscopic
 
Ventricular area:
Viablecysts are usually singlelesions, that lodge to thechoroid plexus or float freelywithin the ventricular cavities
Brain parenchyma
: Viablecysticerci measureapproximately 10 mm tendsto lodge in the cerebralcortex or the basal gangliabecause of the high vascular
Subarachnoid space:
Viable cysticerci tends tolodge within cortical sulci orin the CSF (CerebrospinalFluid) cisterns at the base of the brain and grow to reach50mm or more because theirgrowth is not limited by the The parasite remains alive and undergo different stages such as vesicularstage, colloidal stage, granular nodular stage, and nodular calcifiedAfter entering the CNS, cysticerci are viable and induce slightinflammatory changes in the surrounding tissues. Minimum inflammatoryreaction around the cyst because the parasite carry out prostaglandinsand low molecular wieght molecules which decrease perilesionalinflammation and secrete proteases that can degrade interleukin 2 and
Vesicular stage:
parasites have a transparent membrane, clear vesicularfluid, and larva or scolex and may remain for decades (asymptomatic).As the result of a complex immunological attack from the host byreleasing lymphocyte proliferation and macrophages, cytokines, T cellsand antibodies, the cysticerci enters into a process of degeneration that
Neurocysticerco
SuddenHeadacheManagement:CorticosteroidsMRI scan,CT scan

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