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IV. the PATIENTS ILLNESS (Nephrolithiasis) Pathophysiology (Book-Based)

IV. the PATIENTS ILLNESS (Nephrolithiasis) Pathophysiology (Book-Based)

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Published by wapakalypse
pathophysiology
pathophysiology

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Published by: wapakalypse on Feb 28, 2010
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11/21/2012

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IV. THE PATIENTS ILLNESS (Nephrolithiasis)Pathophysiology (Book-based)
 
Non-modifiable Factors
Age (over 20 to 40 yearsold)
Sex (Male>Female 4:1 )
Family history,Hereditary
Race (Mostly Caucasian),influence with lifestyle,diet
Seasonal factors
Urinary tractmalformation
Hyperparathyroidism
Modifiable Factors 
Faulty Diet
Metabolic disorders, obesity
Excessive medications (ceftriaxone)
Low fluid intake and excessive intake of protein, saltand oxalate
 
Nature of work (occupation)
Stimulation of Calcium precursor
Weight Loss
Hypertension
 
RENAL CALCULI FORMATION
Super saturation of one or more salts inthe urine
Precipitation of thesalts from a liquid toa solid stateGrowth throughcrystallization or agglomeration(aggregation)Super saturation of salt in the urineMultiple salts may precipitate intocrystalsFormation of Nidus(nucleus) of aurinary calculus.
 
Intermittentsupersaturation due toingestion of a meal or during dehydrationmay also contributePHAlkaline urineincreases risk of forming calciumstones due toAcidic urine increasesrisk of forming uricacid stones due toincrease in acidic foodXanthine readily precipitates withacidic urine but Phdoes not directlyaffect the formation of this type of stoneTemperatureSalt forms crystalswhich can grow intostones in the presenceof super saturated urine

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