Similar to the patients of yester-year, stoke patients today still continue to struggle to regain sensory-motor control and constantly struggle to get rid of spasticity. Therapeutics in the modern times withmost modern machines remains to be palliative and focus on negative and positive symptoms. Passivetissue contracture, synergic grouping, spasticity, associated reactions, depression, spatial neglect,Paraesthesia and central pain are assumed to be part and parcel of the condition. Spasticity is welldefined by physiologist in the last century yet, there is no effective treatment of spasticity till date. Ithink time is ripe in this 21
century for clinicians to relook into stroke symptoms and revisit spasticity.Burning desire in me to help stroke subjects made me clinically experiment in unchartered waters of stroke rehabilitation to find root cause of all complex problems of stroke patients. Theoreticalunderpinning of Vasa Concept evolved with new clinically applied definition of spasticity. This led me toevolve a new solution to complex problems of sensory motor control and getting rid of spasticity by re-reorganizing the stroke brain with Vasa Concept. New clinically applied definition of spasticity andtheoretical underpinning of Vasa Concept is described with “what to exploit”, “what to do” and “whatnot to do” to a self organized brain and how to re-reorganize it.
Clinically Drawn Conclusion:
Increased degrees of freedom of paretic flail MSS (Musculoskeletal system) of one side of thebody from a small lesion in CNS make self organizing dynamic system unstable from within.2.
Action plans of self organizing stroke CNS and MSS to re-stabilize the system and to combatexternal forces like gravity to control and defend COM (Centre of Mass), a priority of all livingorganisms, becomes the added constraints to restoration of lost control besides the presence of lesion acting as a catalyst.3.
Self organizing stroke CNS exploits anticipatory postural activity to induce muscle contraction inchain of paretic muscles during functional acts with slightest movement of COM whereasmuscle’s velocity dependant spastic behavior in laboratory set up in unloaded condition whenthe limb segment is moved passively by examiner is a reflex action.
Synergic extended anticipatory activity in chain of paretic muscles known as spasticiy,and passive tissue contracture help restrict and reduce increased degrees of freedomfrom flaccidity and help defend COM.
Spasticity, synergic grouping and contracture act optimally as a “BRAKE” on the fluidmovement of COM for safety a priority.
Self organizing stroke CNS promotes automatic central postural control of global COMwith synergic grouping of chain of muscles in priority over the development of selectivecontrol on segmental COM.
Automatic muscle activities allow segmental COM to move only in the direction towards