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INTRODUCTION
Cholecystitis is an inflammation of the gallbladder wall and nearby
abdominal lining. Cholecystitis is usually caused by a gallstone in the cystic
duct, the duct that connects the gallbladder to the hepatic duct. The
presence of gallstones in the gallbladder is called cholelithiasis. Cholelithiasis
is the pathologic state of stones or calculi within the gallbladder lumen. A
common digestive disorder worldwide, the annual overall cost of
cholelithiasis is approximately $5 billion in the United States, where 75-80%
of gallstones are of the cholesterol type, and approximately 10-25% of
gallstones are bilirubinate of either black or brown pigment. In Asia,
pigmented stones predominate, although recent studies have shown an
increase in cholesterol stones in the Far East.
Gallstones are crystalline structures formed by concretion (hardening) or
accretion (adherence of particles, accumulation) of normal or abnormal bile
constituents. According to various theories, there are four possible
explanations for stone formation. First, bile may undergo a change in
composition. Second, gallbladder stasis may lead to bile stasis. Third,
infection may predispose a person to stone formation. Fourth, genetics and
demography can affect stone formation.
Risk factors associated with development of gallstones include heredity,
Obesity, rapid weight loss, through diet or surgery, age over 60, Native
American or Mexican American racial makeup, female gender-gallbladder
disease is more common in women than in men. Women with high estrogen
levels, as a result of pregnancy, hormone replacement therapy, or the use of
birth control pills, are at particularly high risk for gallstone formation, Diet-
Very low calorie diets, prolonged fasting, and low-fiber/high-cholesterol/high-
starch diets all may contribute to gallstone formation.
A. Personal History
This is the second time Mr. Cholengty been admitted into this hospital.
On his first admission into this hospital he had undergone fistulectomy
operation, which is almost 29 years ago. He had not experience any accident
and injuries, even though his job is prone to accident.
He was admitted into this hospital because of cholelithiases, he was admitted
last February 09, 2009. He was been diagnosed with cholelithiases with
multiple polyps two months prior to admission due to right upper quadrant
pain. He was diagnosed and surgically operated by Dr. Sales last February 10,
2009.According to Mr. Cholengty, upon admission he had undergone some
laboratory examination such as Chest X-ray, CBC,Creatinine, Sodium,
Potassium and ALT.
generally about 7.5 cm (about 3 in) long and 2.5 cm (1 in) in diameter at its
thickest part; it has a capacity varying from 1 to 1.5 fluid ounces. The body
(corpus) and neck (collum) of the gallbladder extend backward, upward, and
to the left. The wide end (fundus) points downward and forward, sometimes
extending slightly beyond the edge of the liver. Structurally, the gallbladder
consists of an outer peritoneal coat (tunica serosa); a middle coat of fibrous
tissue and unstriped muscle (tunica muscularis); and an inner mucous
membrane coat (tunica mucosa).
The function of the gallbladder is to store bile, secreted by the liver and
transmitted from that organ via the cystic and hepatic ducts, until it is
needed in the digestive process. The gallbladder, when functioning normally,
empties through the biliary ducts into the duodenum to aid digestion by
promoting peristalsis and absorption, preventing putrefaction, and
emulsifying fat. Digestion of fat occurs mainly in the small intestine, by
pancreatic enzymes called lipases. The purpose of bile is to; help the Lipases
to Work, by emulsifying fat into smaller droplets to increase access for the
enzymes, Enable intake of fat, including fat-soluble vitamins: Vitamin A, D, E,
and K, rid the body of surpluses and metabolic wastes Cholesterol and
Bilirubin.
Risk factor
V. PATHOPHYSIOLOGY
Heredity
Obesity
Age Over 60
Female Gender
Gallstones
Localized cellular
Venous and Areas of
Proliferation of irritation or
lymphatic drainage ischemia may
bacteria infiltration or both
is impaired occur
take place
CHOLECYSTITIS
V. Medications
NAME OF CLASSIFICATION INDICATION
DRUGS/DOSAGE
ZEPTRIGEN 1gm IV q80 Antibiotic Treatment for urinary
(8AM-4PM-12MN)
tract infection.
MORPHIN SO4 0.03% sol. Analgesic and Used fro the control of
10cc per EC q 120 x 3 Antipyretic moderate to severe
doses pain and as an adjunct
(4PM-4AM) to anesthesia.
OMEPRON 40mg IV OD Antacid and Antiulcerant Active duodenal ulcer
(6AM) and GERD.
PONSTAN SF 500MG 1 Antirheumatic, Anti-
CAP TID inflammatory Analgesic For post-operative pain.
(8AM-1PM-8PM)
PARACETAMOL 500MG Analgesic and For relief of fever, minor
Q40 Antipyretic aches and pains.
Results:
WBC - 9.79K/uL
NEU -5.58%
LYM -3.42%
MONO-.367%
EOS-.371%
BASO-.046%
Hemoglobin - 14.5g/dl
Hematocrit - 44.1%
Conclusion:
Complete Blood Count is within the normal range.
2. Creatinine
This is the indicator of the renal function
Results:
84umol/L
Conclusions:
The result is within normal range based on the normal value of
58-110.
3. Sodium
Date Ordered: February 09, 2009
Date Result In: February 09, 2009
Results:
144mmol/L
Conclusion: The result is within normal range based on the normal
value of 137-145.
4. Potassium
Date Ordered: February 09, 2009
Date Result In: February 09, 2009
Result:
4.0mmol/L
Conclusion: The result is within normal range based on the normal
value of 3.6-5.0.
5. ALT
Date Ordered: February 09, 2009
Date Result In: February 09, 2009
Result:
46 U/L
Conclusion: The result is within normal range based on the normal
value of 21-72.
6. Ultrasound
** Normal liver parenchyma and intra-hepatic ducts.
** Well distended gallbladder with multiple polyps and lithiases.
**No free fluid noted.
7. Chest X-Ray
Date Ordered: February 09, 2009
Date Result In: February 09, 2009
No active infiltrate noted nor pneumothorax seen.
The heart is not enlarged.
The right hemidiaphragm is elevated.
The left hemidiaphragm and chest bones are intact.
Impression: Consider Subdiaphragmatic lesion.
Clinical coyrrelation and further evaluation is
recommended.
GORDON’S ASSESSMENT
C. Sleep/Rest
Usually sleeps between 11-12 midnight and wakes up between 6-7
am. Awaken when pain is felt.
III. Mobility
With regular exercise
IV. Cognitive/Perception
Speaks Bicol, Tagalog and English
Has abdominal discomfort
X. Sexuality/Reproductive
Married
Competencies:
Has many friends
Alteration in physical ability
Nursing Physical Assessment
Age: 47
TPR: 36.7, 74bpm, 21cpm
BP: 140/100 mmHg
Skin pale
Pale conjuctiva
Facial edema
drowsy
Dry oral mucous membrane
Cracked lips
Nonproductive cough
Abdominal pain (pain scale = 5/10)
Decreased peristaltic sound heard upon auscultation
Muscle twitching
(+) Trousseaus’s sign
1. Acute Pain
6. Make time
to listen to
and maintain - Helpful in
frequent alleviating anxiety
contact with and refocusing
patient. attention,
which can relieve
7. pain.
Administered
analgesics as
indicated - Relief of pain
facilitates
cooperation with
other
therapeutic
interventions,
2. Fluid Volume deficient
- Skin and
mucous
3. Provide membranes
skin and are dry, with
mouth care decreased
elasticity,
because of
vasoconstricti
on and
reduced
intracellular
water.
- promotes
hydration.
4. Increase
fluid intake - Relieves
thirst and
5. Ascertain discomfort of
patient’s dry mucous
beverage membranes
preferences, and augments
and set up a parenteral
24- replacement.
hr schedule
for fluid
intake.
Encourage
foods with - Reduces
high nausea and
fluid content. prevents
vomiting.
6. Administer
antiemetics,
e.g.,
prochlorperazi
ne
(Compazine)
as ordered by
the physician.
Post-operative NCP
3. Knowledge Deficit
- Prevents/limits
4. Instruct recurrence of
patient to gallbladder
avoid attacks.
food/fluids
high in fats
(e.g.,
whole milk, ice
cream, butter,
fried foods,
nuts, gravies,
pork), gas
producers
(e.g., cabbage,
beans, onions,
carbonated
beverages), or
gastric irritants
(e.g., spicy
foods,
caffeine, - Promotes gas
citrus). formation, which
can increase
5. Suggest gastric
patient limit distension/discom
gum chewing, fort.
sucking on
straw/hard
candy, or
smoking.