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PRESSURE
11ou
VOLUMES
rrssti#ftft
ruMoRS
-CEREBRAL EDEMA
(Increase water content in brain's
white matter)
-TRAUMA
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{csrt
-tpnoDucrroN
. BLOCKAGE OF
CIRCULATION
tnr"?}ilt8*' voLUME
VASODILATION FROM
t co2
COMPENSATORY
MECHANISMS
FAIL QUICKLY
THIS RESULTS IN
INCREASED PRESSURE
QvoLUME -t**EssuRE)
TT{E RESULTS OF
INCREASED
INTRACRANIAL
PRESSURE ARE:
-TISSUE ISCHEMIA
.TISSUE COMPRESSION
IF PRESSURE IS NOT
RELIEVEI)O THE BRAIN
HERNIATES AND THE PERSON
DIES.
THERE ARE TWO \TAYS THE
BRAIN CAN HERNIATE
?k?k*First sign is
unilateral
pupil dilation
(Oculomotor nerve:
3td cr n.)
HERNIATION HAPPENS
WHEN THE BRAIN PRESSES
THROUGH THE ONLY
OPENING IN THE SKULL:
THE FORAMENMAGNUM
SIGNS AND SYMPTOMS OF
INTRACRANIAL PRESSURE
-changes in behavior
then
-changes in level of
consciousness
STAGES:
#l Diencephalon
Impairment:
?k
?k ?k ?k ?k
FifSt Sympt0m :
changes in loc
(RAS)
-subtle behavioral A
agitation, apathy, lethargy
(stupor, coma)
restles sn es s : dis o rien tatio n
-I?
headache
projectile Yomiting
(without nausea)
Eyes: oculomotor nerve
*****changes in vision:
r increase in blind spot
-pupils small but react
to light
-roving ocular movement
-rray have diplopia
-papilledema (optic disk
a
choked)
STAGE #2: MIDBRAIN
As pressure further f,
midbrain and upper pons
function decreases.
Brain-stem failure
-Respirations:
Cheyne-Stokes progress
to central neurogenic
hyperventilation
-Pupils:
Fixed and dilated
Fixed at midpoint
*****Changes in respirations
(medulla & pons)
-eventually C heyne-Stokes
POSTURING:
-flexion: decorticate
7"os