Welcome to Scribd, the world's digital library. Read, publish, and share books and documents. See more
Download
Standard view
Full view
of .
Look up keyword
Like this
3Activity
0 of .
Results for:
No results containing your search query
P. 1
Checkpoint to Eat or Not to Eat

Checkpoint to Eat or Not to Eat

Ratings: (0)|Views: 346|Likes:
Published by tvalentine99

More info:

Published by: tvalentine99 on Mar 07, 2011
Copyright:Attribution Non-commercial

Availability:

Read on Scribd mobile: iPhone, iPad and Android.
download as DOCX, PDF, TXT or read online from Scribd
See more
See less

05/12/2014

pdf

text

original

 
Running head: TO EAT OR NOT TO EAT 1To Eat or Not to EatTiffany Nicole Cooper University of PhoenixJwaundace Belcher January 21, 2011PSY/240
 
TO EAT OR NOT TO EAT 2To Eat or Not to EatHello class. My name is Nicole Cooper, and today we are going to be discussing some of the physiological factors that cause hunger and satiety, and we will also be taking a look at someof the misconceptions regarding these issues. There will be plenty of time at the end of our session today for questions so as I talk about these issues; please, be thinking about somequestions that you may want to ask about this subject.
Fact and Myth:
I believe that it is very important for everyone to understand the physiological factors,and how the research is opening doorways for the scientific community. First, I want to start bytalking about some common misconceptions regarding hunger and satiety. The most common belief is that hunger is caused by an empty stomach and that satiety is caused by feeling full. ³Astudy showed that even people who have had their stomachs removed still experience thesefeelings (Pinel, 2007).´ The most logical explanation for this would be that the gastrointestinaltract is the source of these feelings of satiety. A fact that I find most interesting is that ³thegastrointestinal tract and the stomach release chemicals called peptides, which can interact withthe hormones and neurons in the brain. There is a well-known peptide that is solely responsiblefor increasing appetites in the hypothalamus´ (Pinel, 2007). This recent discovery has establishedan interest in the ³hypothalamus¶s´ role in hunger and satiety. An important notion to keep inmind regarding this issue is that the hypothalamus is what controls the energy metabolism andnot your actual eating.A second misconception is the set-point assumption. This is when people think that
hunger 
(or the motivation to eat) is caused by the presence of an energy deficit, and they ³vieweating as the means to which the energy resources of the body are returned to their optimal level
 
TO EAT OR NOT TO EAT 3(also known as their energy set-point.)´ (Pinel, 2007) There are either set-points or settling pointsand to understand those, may help with understanding weight. Everyone has a set point and justlike you do not have any control over what color your eyes or hair will be, or how tall you will be. You also have no control over what your set point will be. Your body is genetically and biologically determined to weigh within a certain weight range. Set points vary for eachindividual person. That is why it is not a good idea for people to go by the weight charts that onemay see in medical books or hanging in your doctor's office. It is more of a discussion you andyour doctor need to have. Just as your metabolism will slow down when you go under your  body's set point, it will also increase if you go above it. The body will try to fight against theweight gain by increasing the metabolism and raising the body's temperature to try and burn off the unwanted calories. The settling point theory refers to the idea that weight tends to driftaround the level at which the constellation of factors that determine food consumption andenergy expenditure achieve an equilibrium.The third misconception that we are going to talk about is how hunger and satiety is thatour blood glucose levels affect our hunger and satiety. ³Research tells us that this is very muchuntrue. Glucose levels remain fairly stable and do not fluctuate very often´ (Pinel, 2007). Thistheory is referred to as the ³
 glucostatic theory
´. The lipostatic theory is another set-point theorythat says ³every person has a set point for body fat, and deviations from this set point producecompensatory adjustments in the level of eating that return the levels of body fat to their set points.´ (Pinel, 2007).
P
atient Question and Answer:
 Now, we are going to open the floor for discussion.

Activity (3)

You've already reviewed this. Edit your review.
1 thousand reads
1 hundred reads
Nell Estocado liked this

You're Reading a Free Preview

Download
scribd
/*********** DO NOT ALTER ANYTHING BELOW THIS LINE ! ************/ var s_code=s.t();if(s_code)document.write(s_code)//-->