TO EAT OR NOT TO EAT 2To Eat or Not to EatHello class. My name is Nicole Cooper, and today we are going to be discussing some of the physiological factors that cause hunger and satiety, and we will also be taking a look at someof the misconceptions regarding these issues. There will be plenty of time at the end of our session today for questions so as I talk about these issues; please, be thinking about somequestions that you may want to ask about this subject.
Fact and Myth:
I believe that it is very important for everyone to understand the physiological factors,and how the research is opening doorways for the scientific community. First, I want to start bytalking about some common misconceptions regarding hunger and satiety. The most common belief is that hunger is caused by an empty stomach and that satiety is caused by feeling full. ³Astudy showed that even people who have had their stomachs removed still experience thesefeelings (Pinel, 2007).´ The most logical explanation for this would be that the gastrointestinaltract is the source of these feelings of satiety. A fact that I find most interesting is that ³thegastrointestinal tract and the stomach release chemicals called peptides, which can interact withthe hormones and neurons in the brain. There is a well-known peptide that is solely responsiblefor increasing appetites in the hypothalamus´ (Pinel, 2007). This recent discovery has establishedan interest in the ³hypothalamus¶s´ role in hunger and satiety. An important notion to keep inmind regarding this issue is that the hypothalamus is what controls the energy metabolism andnot your actual eating.A second misconception is the set-point assumption. This is when people think that
(or the motivation to eat) is caused by the presence of an energy deficit, and they ³vieweating as the means to which the energy resources of the body are returned to their optimal level