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Granny's List July 2011

Granny's List July 2011

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Published by: renaxgade81 on Aug 25, 2011
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01/11/2013

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 1
Granny Storm Crow's MMJ Reference List- July 2011
This year‘s message to all of you is a little different.
I am going to be explaining a majorscientific discovery- the Omega 3 / CB1 connection, and how it affects your healing withcannabis! But to understand fully this discovery, we need to revisit Biology 101.
Every cell in your body has tiny chemical receptors all over the cell‘s ―skin‖ or cell membrane.
These receptors work kind of like an ignition switch-
you put the right type of chemical ―key‖into a receptor and it ―turns on‖ some kind of action. The type 1 cannabinoid receptors (CB1s)
are the ones we are interested in looking at. They are found in the body and the brain.
―Turning on‖ a CB1 receptor with either an endocannabinoid that your 
body makes, or aphytocannabinoid like THC, can result in many different things occurring. A cancer cell may be
―told‖ to die through a process called
apoptosis,
it may activate a basic instinct such as nursing,soothe an irritated digestive tract, or simply ease your pain. The CB1 receptors in your brain are
the ones to blame, or praise, for the cannabis ―high‖.Every time a cell divides, whether it is a brain cell, or a body cell, it needs to make new ―skin‖ to
grow back to its full size, and that involves making a whole bunch of new receptors.And this is where the cutting-edge science starts-- to make functional CB1 receptors, youabsolutely need Omega 3! In
“Nutritional omega
-3 deficiency abolishes endocannabinoid-
mediated neuronal functions” 
 ,
the Omega 6-
rich ―western diet‖ is implicated in our decliningmental and physical health. The ―ideal‖ proportion of Omega 6 to Omega 3 is around 3 to 4 parts Omega 6 to 1 part Omega 3. Our ―western diet‖ can deliver up to a 50 to 1 ratio!
When no Omeg
a 3 is available, our bodies will ―jury
-
rig‖ a new receptor with an Omega 6 where
there should be an Omega 3. This results in a small, but important chunk, the Gi/o effectorprotein, not getting attached. As with a machine, the pieces need to be assembled right to work!
A drop in the number of working CB1 receptors is an early clinical sign in Parkinson‘s, coloncancer, Huntington‘s, and heralds a high risk for premature birth. Mice bred to be low in CB1
receptors have more severe heart attacks and strokes. Cancers ravage them. They age andbecome senile earlier than normal mice. They are used to study neurological conditions andbowel disorders. They often seem depressed. They sound a lot like many modern Americans.The three most common sources of Omega 3 are fish oil, flax seed oil and hemp seed oil.Cannabis is an effective and safe herbal medicine, but we need functioning CB1 receptors for itto work its miracles. Virtually every person needs more Omega 3 in their diet, but none as muchas the medical users of cannabis! Cannabis heals us using our cannabinoid receptors, and alsoprovides the Omega 3 we need to make healthy CB receptors, so we can heal. And that is thesimple, but scientific truth.
If the truth won‘t do, then something is wro
ng!
 
 2
It Is Time for Marijuana to Be Reclassified as Something Other Than a Schedule I Drug!(2005)
ACEA/ ARACHIDONYL-2'-CHLOROETHYLAMIDE
- endocannabinoid, CB1 agonist
 In vivo effects of CB1 receptor ligands on lipid peroxidation and antioxidant defensesystems in the rat brain of healthy and ethanol-treated rats. (full
 – 
2006)
Arachidonyl-2'-chloroethylamide, a highly selective cannabinoid CB1 receptor agonist,enhances the anticonvulsant action of valproate in the mouse maximal electroshock-induced seizure model. (abst
 – 
2006)
Opposing control of cannabinoid receptor stimulation on amyloid-beta-induced reactivegliosis: in vitro and in vivo evidence. (full - 2007)
Ultra-low dose cannabinoid antagonist AM251 enhances cannabinoid anticonvulsanteffects in the pentylenetetrazole-induced seizure in mice. (abst
 – 
2007)
Attenuation of Experimental Autoimmune Hepatitis by Exogenous and EndogenousCannabinoids: Involvement of Regulatory T Cells (full - 2008)
Cannabinoid modulation of cutaneous Adelta nociceptors during inflammation.(full
 – 
2008)
Cannabinoid-mediated antinociception is enhanced in rat osteoarthritic knees.(full
 – 
2008)
Cannabinoid receptor activation induces apoptosis through tumor necrosis factor alpha-mediated ceramide de novo synthesis in colon cancer cells. (full
 – 
2008)
Endogenous cannabinoids induce fever through the activation of CB1 receptors.(full
 – 
2009)
The effects of intracerebroventricular AM-251, a CB1-receptor antagonist, and ACEA, aCB1-receptor agonist, on penicillin-induced epileptiform activity in rats. (full
 – 
2009)
 
 3
Involvement of nitrergic system in the anticonvulsant effect of the cannabinoid CB(1)agonist ACEA in the pentylenetetrazole-induced seizure in mice. (abst
 – 
2009)
Involvement of nitric oxide in the gastroprotective effect of ACEA, a selectivecannabinoid CB1 receptor agonist, on aspirin-induced gastric ulceration. (abst
 – 
2009)
Effect of arachidonyl-2'-chloroethylamide, a selective cannabinoid CB1 receptor agonist,on the protective action of the various antiepileptic drugs in the mouse maximalelectroshock-induced seizure model. (abst
 – 
2009)
Role of cannabinoid CB1 receptors on macronutrient selection and satiety in rats.(abst
 – 
2009)
Regulatory Role of Cannabinoid Receptor 1 in Stress-Induced Excitotoxicity andNeuroinflammation (abst - 2010)
Alkamides and a neolignan from Echinacea purpurea roots and the interaction of alkamides with G-protein-coupled cannabinoid receptors. (abst
 – 
2011)
ADD/ ADHD
ADHD by Ryan P (anecdotal - no date)
Marijuana and ADD Therapeutic uses of Medical Marijuana in the treatment of ADD(no date)
How Cannabis Compares to other treatments (no date - 2008)
 Recipe For Trouble (anecdotal/ news - 2002 )
Association between cannabinoid receptor gene (CNR1) and childhood attentiondeficit/hyperactivity disorder in Spanish male alcoholic patients (full - 2003)
Cannabinoids effective in animal model of hyperactivity disorder (abst - 2003)

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