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Entire Study Guide for Fitness

Entire Study Guide for Fitness

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Published by tonomos26

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Published by: tonomos26 on Apr 01, 2012
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01/21/2013

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1Eddie Cabrera, skype user eddie.cabrera6 |1
S
TUDY GUIDE
N
º
4
Steiner holistic approach on the detoxification
I.- Escoja la alternativa correcta
1.
La propuesta de steiner para mejorar la salud incluye los siguientes tópicos
a)
 
Desintoxicación, ejercicios y alimentaciónb)
 
Desintoxicación, ejercicios y nutriciónc)
 
Desintoxicación, ejercicios y dietad)
 
Todas las anteriores
2.- Una de las principales herramientas de educación y por tanto de preparaciónpara el cliente sobre el barco es:
a)
 
Los seminarios y charlasb)
 
Las clases de spinningc)
 
La evaluación del estado físicod)
 
Clases de baile entretenido
3.- Uno de los principales riesgos del consumo de asparthame es
 a)
 
Es un producto carcinogénicob)
 
Es un gran endulzante pero produce diabetesc)
 
No está aprobado por el FDAd)
 
Ninguna de las anteriores
4.- Con respecto al endulzante splenda, las personas debieran saber que:
a)
 
Es natural y por lo tanto no precisa de conservantesb)
 
Es artificial y suprime a las bacterias benéficas del tracto digestivo, interfiriendo enla correcta asimilación celular de nutrientesc)
 
Es similar al asparthame pero mejor que la azúcar blancad)
 
Ninguna de las anteriores
5.- Las toxinas pueden causar diversas enfermedades crónicas en nuestro cuerpopor acumulación, una definición correcta de toxinas sería:
a)
 
Son productos de desecho (catabolitos secundarios)b)
 
Son diversos tipos de venenos (metales pesados: mercurio, plomo, amalgamas…)
 c)
 
Son diversos tipos de sustancias que pueden encontrarse fuera y dentro del cuerpohumano, provocando diversos tipos de enfermedades crónicas. Estos productospueden ser orgánicos o inorgánicos y varían en su grado de toxicidad.d)
 
All previous
6.- The four ways in which toxins enter our bodies are:
a)
 
Digestion, inhalation, absortion, reactionb)
 
Digestion, contact, dietary, reactionc)
 
Absortion, inhalation, contact, foodd)
 
Ninguna de las anteriores
 
2Eddie Cabrera, skype user eddie.cabrera6 |2
II.- Connect the organ with symptoms that correspond
Colon
- Skin Problems - Poor Complexion - Lack of Vigour -Hyperactivity
Liver and gallbladder
- Difficult or Frequent Urination - Lower Back Pain - Weak Joints - Cold to Touch
Lungs
- Easily Fatigues - Headaches, including migraines -Insomnia - Food Sensitivities
Kidney and bladder
- Frequent Colds & Infections - Tiredness - Lethargic Body -Tender Lymph Nodes
Lymph
- Constipation - Mental Dullness - Tired for no Reason - BadBreath
Blood and skin
- Shortness of Breath - Coughing - Cold Hands - Congestion
III. - Preguntas de desarrolloa)
 
Resume las funciones del hígado en seis líneasb)
 
¿De qué forma procesa el hígado las grasas? Explica el mecanismo de formación delos cálculos biliaresc)
 
Explica tres de las funciones del sistema linfáticod)
 
Si el sistema linfático, a diferencia del circulatorio, no posee un corazón paraimpulsar su flujo, de qué manera circula por el cuerpo?e)
 
Como afecta la flora intestinal a la salud integral del cuerpo?f)
 
¿Cuánta agua es necesario ingerir a diario y como afecta al colon la cantidad dehumedad?g)
 
¿Cómo se conecta el sistema circulatorio con el linfático?
 
3Eddie Cabrera, skype user eddie.cabrera6 |3
R
EAD THE FOLLOWING TEXT AND ANSWER THE QUESTIONS BELLOW
 
How to eliminate 50 percent of all coronary events
By Dr. Barry Sears, founders of zone diet 
The European Society of Cardiology estimates a 50 percent reduction of coronaryevents if you can stabilize soft, vulnerable plaques (1). We are often led to believe
that plaques you can see on an angiogram are “killer” plaques. It’s true that if they
are large enough to obstruct blood flow, they will decrease oxygen transfer to theheart muscle cells making them more tired with less effort.This is the definition of stable angina. It simply means it takes less effort to over-exert the heart muscles before they fatigue. However, you need approximately a 90percent total obstruction of the blood vessel to develop stable angina. Theseplaques account for most of the plaques you might find in an angiogram. This iswhy if you take an angiogram, you are often immediately wheeled into theoperating room to have a stent put into the artery with the belief you are onlyseconds away from an immediate heart attack and death.
However, the same angiogram can’t see a few plaques (because they are so small),
known as the soft, vulnerable ones. When soft, vulnerable plaques rupture (like apimple), then you have the death and disability (i.e., damaged heart tissue) that truly characterizeheart disease.Technically, this is called an acute coronary event,and it has very little to do with the stable plaques that can cause angina. It is this
small number of “rogue” soft, vulnerable plaques that are the true killers in heart 
disease (2,3).The ultimate cause of plaque rupture is cellular inflammation inside the plaque.Cellular inflammation degrades the fibrous external coating of the plaque. Usuallyinside these soft, vulnerable plaques are also a lot of macrophages engorged with
lipids. This is called the “necrotic core”. When the plaque bursts, these lipid pools
are released into the bloodstream causing platelet aggregation and the rapidblockage of the artery resulting in a complete restriction of blood flow (as opposedto a limited restriction of blood flow with a typical stable plaque that will neverrupture). It is estimated that about 75 percent of all coronary events are caused byruptures of the soft, vulnerable plaques (2).As I mentioned above, the really scary part of this story is that there is no type of imaging technology that can detect dangerous soft, vulnerable plaques. In essence,
you don’t know if you have them or not. This is why the prediction of impeding
cardiovascular events remains a guessing game. Even more interesting is that these soft, vulnerable plaques seem to form rather quickly (in about 10 years) asopposed to growing slowly over a lifetime (4). Moreover, the rate of growth of these soft, vulnerable plaques is strongly correlated with increasing insulin levelsin the blood (4).
So what does this mean for people who don’t want to die from a s
udden rupture of 
soft, vulnerable plaques that can’t be detected? The first thing is to reduce the
inflammation within the plaque. Surprisingly, there is only one clinical study that has ever been published that addressed this question, and it used fish oil (5). Thisstudy indicated that if you give patients relatively high doses of fish oil, you could

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