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The Role of Excitotoxins in Autistic Type Behavior

The Role of Excitotoxins in Autistic Type Behavior

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Published by Antonio Sonrise

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Published by: Antonio Sonrise on Jun 21, 2012
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12/29/2013

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Dr.
Amy
6/16/02
Page1
TheRoleofExcitotoxinsinAutisticTypeBehavior
Dr.
AmyA.
VaskoIntroduction
IhavebeenresearchingandworkingwithinflammatorypathwaysinthebodysincemydoctoralworkatAlbanyMedicalCollege.Ihavealwaysfeltthatitiscriticaltounderstandwhysomethingishappeninginordertomakeinformedchoicesdirectedatcorrectingtheimbalance.IbelieveverystronglythatthepathwayofexcitotoxindamageasdescribedbrilliantlybyDr.RussellBlaylock,definitelyleadstoneurologicalinflammation.InapplyingtheknowledgeofthisprocesstomyworkwithindividualswhohaveALS,Parkinson's,MSandAlzheimer's,Ihavehadconsiderablesuccessinreversingsymptomsofthesediseases.Morerecently,Ihaveusedandextendedtheconceptsofexcitotoxindamageastheypertaintoautism.Ihavefoundthatbyunderstandingtheprocessthatleadstothetypeofneurologicalinflammationthatweknowas"autism",Iamabletomakesignificantstridesinhelpingthesechildren.ThefollowingrepresentswhatIbelieveisoccurringthusfarwithrespecttotheprocessofneurologicalinflammationthatresultsinautisticlikebehavior.Itisaworkinprogress,anddoesnotanswereveryquestionwithregardtoautistictypeneurologicalinflammation.Itdoeshowever,provideanexplanationformanyoftheobservedbehaviorsandsymptomsthatareassociatedwithautism,aframeworkfromwhichtohelpreversemanyofthesetraits,andarationaleforwhymanyofthecurrentlyutilizedtherapiesareeffective.AlthoughIhavenevermet,norcorrespondedwithDr.RussellBlaylock,Ithankhimforhisexplanationanddescriptionofexcitotoxindamage.Itisfromusing,andextendinghisinsightsthatIhavebeenabletosuccessfullytouchandchangemanylivesthathavebeenafflictedwithneurologicalinflammation.
 
Dr.Amy
A.
6116/02
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Overview
Throughherconsultingpractice,Dr.AmyArrowVaskohasworkedwithalargenumberofindividualswithneurologicalinflammation.ThisincludesindividualswithALS,MS,Parkinson'sDisease,Alzheimer'sDisease,andautism.Dr.Amyhasfoundthattheprogramshehasimplementedtosuccessfullybreakthecycleofneurologicalinflammationwithothertypesofneurologicaldisordersisusefulinhelpingtoreversetheneurologicalinflammationthatmanifestsasautism.Inaddition,shehashadsuccessinreversingsymptomsofCrohn'sdisease,whichhasbeenpostulatedtooccurviachronicmeaslesinfectionasisoftenseenwithautism.Dr.Amyviewsautismasasubsetofneurologicalinflammation,andassuchhasappliedavariationoftheapproachthatshehasutilizedwiththeseotherinflammatorydisorderstothetypeofneurologicalinflammationthatresultsinautisticlikebehavior.Whileautismhasavarietyofsimilaritiestootherformsofneurologicalinflammation(Parkinson's,ALS,MS,andAlzheimer's),italsohasuniquefeaturesthatcontributetotheautistictypeofinflammation.Basically,Dr.Amyapproaches
neurologicalinflammation
ingeneralwithathree-stepprogramwhichcanbeimplementedsimultaneously.First,itiscriticaltoremoveexcitotoxintriggersfromthesystem.Thisinvolvescloselymonitoringfoodandsupplementintaketoavoidexcitotoxins.Excitotoxinsareneurotransmitterssuchasglutamateoraspartatethatcanexcitethenervestodeathwhentheirlevelsarenotregulatedproperly.Excessexcitotoxinscauseanimbalanceintheflowofcalcium,whichleadstoactivationofacomplexinflammatorycascade,releaseofinflammatorymediators,andultimatelycausesthedeathofneurons.FoodsorsupplementsthatcontainexcitotoxinsincludeMSG(monosodiumglutamate),glutamicacid,glutamine,nutrasweet,aspartate,aspartame,andcysteine.Mercuryandaluminumcanalsoservetotriggerglutamaterelease.Next,itisimportanttostoptheinflammatoryprocesscreatedbytheexcitotxintriggers.Thisisachievedwithanumberofsupplementsknowntomitigateinflammatorymediators.Finally,thethirdstageistorepairthedamage,generatenewneurons,andsupporttheliver.Thisisaccomplishedwithanumberofsupplements,whichserveasantioxidantsaswellastohelpincreaseglutathionelevels,restoreliverfunction,
 
6/16/02
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promotenervegrowth,restorevitaminKlevels,decreaseglutamatelevels,andbalanceGABAlevels.Dr.Amyhasfoundthatinworkingwithautisticneurologicalinflammationinparticular,thereareadditionalfactorstoconsider.Theseincludedealingwithpotentiallychronicviral,bacterial,andyeastinfectionsinthebody(whichcangeneratereleaseofadditionalinflammatorymediators),balancingtheacid/alkalineratioandrestoringnormalfloratotheGItract,andmetaltoxicity(whichalsogeneratesexcitotoxindamage).Thereareseveralunderlyingfactorsthatmaypredisposecertainindividualstoautistictypeneurologicalinflammation.Theseincludepersonsofbloodtype0,(andtoalesserextentbloodtypeA),earlyrecurrent
streptococcal
infections,excessstomachacid,predominantlymalesex,earlymultiplevaccinations,andwellabovenormalintelligence.Theremayalsobeacorrelationwithafamilialhistoryofliverdysfunction/disease,vitaminKdeficiency,acidreflux,andautoimmuneproblems.
Excitotoxins
Glutamateisthemainexcitatoryneurotransmitterinthebody.Itisessentialforlearning,andforbothshort-termandlong-termmemory.Itisalsotheprecursortotheinhibitoryneurotransmitter,GABA.GABAisacalmingneurotransmitter,andisessentialforspeech.Problemsoccurifthenormalprocessofregulationofglutamatemalfunctionsandiftoxiclevelsofthisexcitatoryneurotransmitterbuildupinthesynapticjunctions.Thebrainrequiressufficientlevelsofoxygenandenergytoremoveexcessglutamate.However,glutamatereleaseleadstothereleaseofinsulin,whichresultsindecreasedglucoselevels.Theamountofglucoseinthebrainregulatesthe
removal
ofexcessglutamatefromthesynapses.Therefore,adropinbloodglucosedisruptsthisremovalprocessandallowsthebuildupoftoxicglutamate.Infact,conditionsofhypoglycemia,orlowcalorie/starvationconditionsinducethereleaseofglutamateandreducetheabilityto
remove
excesslevelsofglutamatefromthebrain.Thisexcessglutamatedepletesglutathione.Glutathioneisoneofthemostpowerfulantioxidantsfoundinthe

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