P. 1
Non Carious Lesions and Their Management

Non Carious Lesions and Their Management

|Views: 900|Likes:
Published by DrApplu Atrey

More info:

Published by: DrApplu Atrey on Jul 17, 2012
Copyright:Attribution Non-commercial


Read on Scribd mobile: iPhone, iPad and Android.
download as PPTX, PDF, TXT or read online from Scribd
See more
See less








Normal physiologic process that occurs throughout life.

Problems arise when the rate of loss becomes
excessive causing functional or esthetic problems or sensitivity for the patient.

Enamel is one of the few tissues in the body that
does not regenerate or replace itself in the way that skin, blood cells, and fractured bones do. Fortunately, the dentine does show some reparative mechanisms as reactionary or reparative dentine which is laid down in the pulp chamber as a response to tooth wear

Traditionally, the terms erosion, abrasion and attrition were used to describe non carious pathologic loss of tooth structure.

Clinically tooth wear appears to progress very slowly over years  .  Tooth wear is a cumulative lifetime process which is irreversible .  Tooth wear is defined as the surface loss of dental hard tissues other them by caries or trauma . Smith and Knight advocated the use of term tooth wear .

Traditionally. Tooth wear has the multi-factorial aetiology. hard surface. but certain clinical features may suggest a major contributory factor. The lesion may become more rounded and shallow if there is an element of erosion present   . cervical lesions caused purely by abrasion have sharply defined margins and a smooth.

  . then dentine tends to wear at the same rate as the surrounding enamel. If wear is primarily attritional. Erosive lesions cause „cupping‟ to form in the dentin. the clinical appearance is determined by the relative contribution of the etiological factors. Once dentine is exposed. Flattening of cusps or incisal edges and localized facets on occlusal or palatal surfaces would indicate a primarily attritional aetiology.

     Change in appearance of teeth Exposure of dentin normally covered by enamel Dentin Hypersensitivity Loss in occlusal vertical dimension Loss in posterior occlusal stability resulting in   Mechanical failure of teeth or restorations Hypermobility and drifting   Exposure of pulp Pulpitis and loss of vitality .

In-vitro measurement • Macroscopic changes • Polarized light microscopy • Surface profilometry • Microhardness tests • Scanning electron microscopy In-vivo • Microradiography measurement • Digital image analysis • Iodine permeability • Macroscopic • Synthetic hydroxyapattite changes powders/discs • Replica • Calcium and phosphorus technique dissolution • Intra-oral carcinogenicity test Newer methods •Scanning tunneling microscope •Atomic force microscope •Finite element analysis .

1. Attrition Erosion Abrasion Abfraction Localized non hereditory Enamel hypoplasia Localized non hereditory enamel hypocalcification Localized non hereditory dentin hypoplasia Localized non hereditory dentin hypocalcification Discolourations Malformations Amelogenesis imperfecta Dentinogenesis imperfecta Trauma . 7. 8. 2. 6. 12. 5. 3. 4. 13. 11. 9. 10.

ATTRITION Derived from Latin word ATTRITIM meaning “action of rubbing against something” .

1998 . The physiologic wearing of the teeth resulting from tooth to tooth contact as in mastication.  Shafer  Wear caused by endogenous material such as microfine particles of enamel prisms caught between two opposing tooth surfaces.  Every (1972)  Loss by wear of surface of tooth or restoration caused by tooth to tooth contact during mastication or parafunction  Milosevic.

  Attrition occurs at an ultra structural level It can be caused by direct contact between surfaces or the action of an intervening slurry  Attrition can be hastened by coarse diet and abrasive dust  Some para-functional habits like Bruxism may also contribute to attrition .

 Distribution of attrition is influenced by the type of occlusion. it is considered pathologic.  If occlusal wear occurs at a rate faster than compensatory physiologic mechanisms. Vertical loss of enamel of 50-68 µm/year is considered physiologic  . the geometry of stomatognathic system and grinding pattern of the individual.


4. 3.1. (unlike erosion) . 2. Shiny wear facets with well defined borders The surface of wear facet is flat and flush with the opposing tooth on contact Enamel and dentin wear at the same rate Possible fracture of cusps or restorations Pure attrition shows equal wear on both arches. 5.





EROSION Derived from Latin verb EROSUM meaning “to corrode” .

 Imfeld T (1996)  Erosion is defined as the chemical dissolution of teeth by acids  Martin .  Every (1972)  Erosion is process of gradual destruction of tooth surface. Erosion is defined as superficial loss of hard tissue due to chemical process not involving bacteria. usually by a chemical or electrolytic process.

of Contemporary Dental Practice 1999. Dental erosion is defined as the progressive. Truelove EL. 1(1): 1-17 . irreversible loss of hard dental tissues due to a chemical process not involving bacteria Gandara BK. Diagnosis and management of dental erosion. J.

The critical pH of enamel is 5.    .5. erosion is primarily a surface phenomenon The solubility of enamel is pH dependent The rate at which apatite precipitates depends on factors such as calcium binding in saliva. any solution with a lower pH may cause erosion if the attack is lengthy and intermittent over time. Clinically.

increased incisal translucency Rapid process may lead to sensitive teeth due to dentin exposure while slower progressive lesions may be asymptomatic   . erosion effects enamel resulting in smooth. glazed surfaces In advanced cases. restorations may project above the occlusal surfaces and exhibiting concavities known as cupping. In early stages.

 Class I – superficial lesion involving enamel only Class II – localized lesions that involve dentin or less than 1/3rd of the surface Class III – generalized lesions involving dentin and more than 1/3rd of the surfaces Eccles et al. 1979   .

vinegar Medication – aspirin. bleaching agents     Intrinsic – conditions that lead to chronic vomiting or persistent gastroesophageal reflux    Anorexia and bulimia nervosa Chronic alcoholism Morning sickness associated with pregnancy . carbonated drinks. swimmers Diet – citrus fruits. vit-C. Extrinsic (exogenous)  Environmental – by acid fumes and aerosols in occupational. calcium chelators Life style – fruits and diet drinks.

 Clinical severity    SUPERFICIAL EROSION Superficial Localized Generalized  Activity of progression   Active or manifest Inactive or latent GENERALISED SEVERE EROSION .

Mannerberg described 2 types of erosive lesions as viewed under SEM  Active lesions – shows distinctive etched enamel prisms resembling honeycomb  Inactive or latent lesions – faint with unrecognizable characteristics .

 Enamel erosion appears smooth and rounded and the surface contour is lost Broad concavities within smooth surface enamel Cupping of occlusal surfaces.   (incisal grooving) with dentin exposure   Increased incisal translucency Wear on non-occluding surfaces  There is a difference in wear in opposing arches .

pH Acquired pellicle – diffusion limiting properties and thickness Tooth composition and structure Dental anatomy and occlusion Physiologic soft tissue movements    . composition. buffering capacity.  Saliva – flow.

.   pH Total acid level Type of acid (pKa)    Calcium chelating properties Calcium phosphorus and fluoride content Physical and chemical properties affecting adherence to the enamel surface and stimulation of saliva flow.

 Medical History     Excessive vomiting. swallow?)  Dental History     Dietary History   . rumination Gastroesophageal reflux disease (Symptoms of reflux) Frequent use of antacids Alcoholism History of bruxism (grinding or clenching) Morning masticatory muscle fatigue or pain? Use of occlusal guard Acidic food and beverage frequency Method of ingestion (swish.

 Occupational/Recreational History     Regular swimmer? Wine-tasting? Environmental work hazards? Sports energy drinks Tooth brushing method and frequency Type of dentifrice (abrasive?) Use of mouthrinses Use of topical fluorides  Oral Hygiene Methods     .

Early recognition of erosion is important to successfully manage and prevent disease progression. Identification of the etiology Preventive measures Patients compliance.    .


refer to a physician. A patient with alcoholism should be assisted in seeking treatment in rehabilitation programs.   Saliva buffering capacity resists acid attacks.   Enhance the defense mechanisms of the body (increase salivary flow and pellicle formation). Saliva is also supersaturated with calcium and phosphorus. which inhibits demineralization of tooth structure . If undiagnosed Gastroesophageal reflux is suspected.   Decrease amount and frequency of acidic foods or drinks. Diminish the frequency and severity of the acid challenge.

Dietary components such as hard cheese (provides calcium Improve chemical protection. Neutralize acids in the mouth by dissolving sugar-free antacid tablets. Apply fluoride in the office 2-4 times a year. Enhance acid resistance.   A fluoride varnish is recommended.   and phosphate) can be held in the mouth after acidic challenge .   Daily topical fluoride at home. remineralization and rehardening of the tooth surfaces.

Do not brush teeth immediately after an acidic challenge to  the mouth.  Use soft toothbrushes and dentifrices low in abrasiveness in a gentle manner. Decrease abrasive forces. Rinsing with water is better than brushing immediately after an acidic challenge. as the teeth will abrade easily.  .  Provide mechanical protection. Construction of an occlusal guard is recommended if a Bruxism habit is present.  Consider application of composites and direct bonding where appropriate to protect exposed dentin.

ABRASSION Derived from Latin verb ABRASUM meaning “ Scrape off ” .

 Imfeld T (1996)  Abrasion is the wearing of tooth substance that results from friction of exogenous material forced over the surface by incisive. masticatory and grasping functions  Every (1972)  Loss by wear of dental tissue caused by abrasion by foreign substance  Milosevic. 1998 . Abrasion is wearing away of tooth substance or structure through mechanical process.

carpenters. and use of abrasive dentrifice.The location and pattern of abrasion may be dependent on the cause  Most common area is cervical area. musicians  Proximal tooth abrasion due to improper flossing and use of tooth picks . nail biters.  Notching of incisal edges in pipe smokers. hair pin biting  Notching of incisors in Tailors. zealous and vigorous methods. related to improper tooth brushing technique.

it may be scooped out because it is softer than enamel. An abrasion area is generally not well defined unlike in attrition. Where dentin is exposed.   PIPE SMOKERS . Abrasion tends to round off or blunt tooth cusps or cutting edges.

  Tooth surface will have a pitted appearance. Microscopically an abraded surface shows haphazardly oriented scratch marks and numerous pits . .

You're Reading a Free Preview

/*********** DO NOT ALTER ANYTHING BELOW THIS LINE ! ************/ var s_code=s.t();if(s_code)document.write(s_code)//-->