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ASKEP KLIEN DG DIABETES MELITUS TIPE 1

NURSISWATI, MKep., Sp.KMB

6. 4. PENGERTIAN ETIOLOGI PATOFISIOLOGI MANIFESTASI KLINIS PENATALAKSANAAN ASKEP . 2.OUTLINE 1. 5. 3.

. and was previously known as juvenile diabetes. In type 1 diabetes.PENGERTIAN • Approximately 5% to 10% of people with diabetes have type 1 • Type 1 diabetes is usually diagnosed in children and young adults. the body does not produce insulin. and was previously known as judiabetes is usually diagnosed in children and young adults.

As a result. in which the insulin-producing pancreatic beta cells are destroyed by an autoimmune process. they produce little or no insulin and require insulin injections to control their blood glucose levels .• diabetes.

.DAN FAKTOR LINGKUNGAN (SEPERTI VIRUS) • FAKTOR GENETIK DIHUBUNGKAN DG TIPE HUMAN LEUCOCITE ANTIGEN (HLA) TERTENTU. IMMUNOLOGIK. • 95 % RAS CAUCASIAN DG DM TIPE 1 MENUNJUKKAN HLA-DR3 ATAU HLA-DR4 • RESIKO DM TIPE 1 MENINGKAT 3-4X PD ORG DG KEDUA TIPE HLA TSB.ETIOLOGI • DESTRUKSI SEL BETA PANKREAS TERJADI AKIBAT KOMBINASI DR FAKTOR GENETIK.

CONT • AUTOIMUN MRP RESPON ABNORMAL DIMANA ANTIBODI MENYERANG SEL2 NORMAL KRN DIANGGAP SBG SEL ASING.ETIOLOGI .. • VIRUS ATAU TOXIN DPT MENGINISIASI KERUSAKAN SEL BETA PANKREAS .

PATOFISIOLOGI .

CONT .PATOFIS..

MANIFESTASI KLINIK • ANAK2 MENGOMPOL • TANDA DEHIDRASI • KETOASIDOSIS (NAUSEA. ABDOMINAL PAIN) . VOMIT.

PENATALAKSANAAN • • • • • TERAPI NUTRISI EXERCISE MONITORING INSULIN TH/ EDUCATION .

1020% PROTEIN REGULARLY INSULIN . 20-30% LEMAK.ASKEP DIET: 50-60% KARBOHIDRAT.