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Brady & Tachy Pathophysiology

Brady & Tachy Pathophysiology

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Published by Jam Knows Right
things about cardio
things about cardio

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Published by: Jam Knows Right on Sep 18, 2013
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Arrhythmias  Too fast (tachycardia) or too slow (bradycardia); normal: 60-100BPM  Irregular BRADYCARDIA PATHOPHYSIOLOGY CAUSES: Less sympathetic

impulse/increased parasympathetic impulse Damaged pacemaker Other pacemakers take over (junctional), approximately 50bpm Ventricular escape (25bpm) Blockage due to ischemia, fibrosis, infection, MI, etc.) – heart block (3types) Left Bundle Branch Block – block the conduction of the heart – no passage of conduction to the left side – get action potential from the RV to LV – wide QRS complex but does not necessarily slow the HR

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SA Node is slowed/absent action potential

TACHYCARDIA: 3 major causes of tachycardia: 1. Increased pacemaker activity (SA node) Causes: increased sympathetic activity; decreased parasympathetic activity; SA node dysfunction (Sick Sinus Syndrome or SSS) Sinus tachycardia 2. Re-entry tachycardia – example: Atrial Fibrillation, V. Tach (paroxysmal), Wolf-Parkinson-White (W.P.W) syndrome Cause: scar tissue Quick/fast depolarization-repolarization happening around the scar For WPW syndrome: deformity of the muscle of the valve create alternating pathway between the atria and the ventricles Waveforms from the AV node depolarize the ventricles then go back to the atrium too quickly (short PR interval)

some meds. Delayed repolarization Causes: ischemia. electrolyte imbalances Refractory period is already pver but the myocardial cells remain hupopolarized An R on T phenomenon Cells repolarize the cells next to them .3.

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