PREDISPOSING

FACTORS:

Family Hx of
DM (both

PRECIPITATING
FACTOR:

Energydensed diet
Sedentary
Lifestyle
HTN

EXCESS GLUCOSE
MOLECULES
ATTACHES TO THE
HGB
Metformin
500g 1tab
BID

INSULIN
DEFICIENCY

DECREASED
GLUCOSE
INDUCED
INSULIN

ALPHA CELLS

Diagnostic
Results
Signs and

GLUCAGON

Nursing
Diagnosis
Medications/
Treatments

INCREASE
GLUCAGON
SECRETION

Unknown
Etiology

GLYCOGENOLYSIS
INGESTED
GLUCOSE
CANNOT BE

CBG q
6H
D5050 1 Vial
for CBG = or <
80g/dL

KIDNEY TUBULE
KIDNEY
CELLS
CANNOT
EXERTS OSMOTIC
REABSORB
PRESSURE
IN FAST
THE
GLUCOSE
BEGINS
ENOUGH
FILTRATE
POLYURIA
TO SPILL
INTO THE

INSULIN
RESISTANCE

GLYCOGEN

BLOOD GLUCOSE LEVEL RISES:

HYPERGLYCEMIA

RI 5 units for
CBG = or >
180g/dL

GLUCUSURIA

PANCREAS

INCREASE
HEPATIC
OUTPUT
DECREASED
GLUCOSE
UTILIZATION

DECREASED
TISSUE RESPONSE
TO INSULIN

INCREASE FAT
MOBILIZATION

FAT
METABOLISM

INCREASE
PROTEIN
UTILIZATI
ON

MUSCL
E

WIGHT
LOSS,D
RY SKIN
IMPAIRED SKIN
INTEGRITY

FATTY ACIDS
BREAKDOW
N

INCREASE
CHOLESTEROL
PACKED
CHOLESTEROL
WITH PROTEIN AND
AND
LIPOPROTEIN
PLAQUE
DEPOSITION
LOSS
OF
WATER
POOR
LARGE
AND SMALL
PROLONGED
INCREASE VENOUS
AND
ELECTROLYTES
CIRCULATION
ACCELERATED
TO
BLOOD
CELLS
PRESSURE

THIRST

INCREASE OF
FLUIDIMMUNO
INTAKE
POLYDIPSI
IMPAIRED
A
FUNCTION

DEHYDRATION

RISK FOR
IMBALANCED FLUID
AND ELECTROLYTES

LEAFLETS OF THE VENOUS

VALVES ARE STRETCHED AND
PREVENTED FROM CLOSING
COMPLETELY

RISK FOR
INFECTION
Cefepime 1gm IV
q 12

TISSUE
INJURY:
WOUND ON
THE RIGHT
FOOT

ALLOWING BACKFLOW OR
REFLUX IN THE VEINS
EDEMA,SKIN
DISCOLORATION ON
THE RIGHT FOOT

VENOUS INSUFFICIENT

INEFFECTIVE TISSUE
PERFUSION
RELATED TO
DECREASED Hgb
Hgb: 109; Hct: 0.32;
RBC: 3.41

IMPAIRED SKIN INTEGRITY

RELATED TO
INFLAMMATORY

Mupirocin
Ointment TID

REDNESS, SWELLING,
ERYTHEMA, SKIN

PRESENCE OF
PORTAL OF

ETIOLOGIC AGENT:
STAPHYLOCCOCUS
AUREUS

BACTERIA RELEASE THEIR

TOXINS IN THE
SUBCUTABEOUS TISSUE

CELLULITIS

ACUTE
PAIN

Pain scale of 5-6/10,

localized on the wound
area, sudden and

Ketorolac 30g IV q 8 for

pain