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Mantak Chia - Darkness Technology - Darkness Techniques for Enlightenment DL-BL05

Mantak Chia - Darkness Technology - Darkness Techniques for Enlightenment DL-BL05

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Published by: BigDipperTao on Aug 31, 2010
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08/27/2014

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An “alarm system” is built into the brainstem, to wake us up and
bring us to waking consciousness, called the reticular activating sys-
tem (RAS) (Fig. 9). Sight and hearing are two major pathways of
incoming sensory information, providing cues which maintain our
state of alertful wakefulness. The optic and auditory nerves stimu-
late brainstem centers, which, in turn, activate higher cortical cen-
ters in the brain though the RAS.

Intermediate Mass of Thalamus

Mammillary Body

Corpus Callosum

Pituatary Gland (Posterior and Anterior Lobes)

Pineal Gland (in
Epithalamic Nucleus)

Suprachiasmatic
Nucleus

14

Fig. 9. Reticular Activating System (RAS): Fibers that project from the
reticular formation through the thalamus to the cerebral cortex are
responsible for maintaining consciousness, muscle tone, and awakening
from sleep, with stimuli from the ears, eyes, and skin, but not the
olfactory system, which explains why people die in house fires3
.

Many of the functions of waking consciousness are maintained
by the neurotransmitter seratonin. Seratonin is a chemical messen-
ger, traversing the synapse, or the gap, between two nerve cells
(Fig. 10). Some of the important nerve pathways assisted by seratonin
begin in a region of the brainstem called the raphe nuclei and ex-
tend upwards into the cerebrum (Fig. 11). Seratonin plays an impor-
tant role in maintaining cortical arousal, concentration, and suppress-
ing distracting stimuli, as well as a role in sleep.

Thalamus

Cerebral Cortex

RAS projections
to cerebral cortex

Visual impulses from eyes

Pons
Recticular Formation
Medulla Oblongata
Spinal Cord
Somatic Sensory impulses
(from nociceptors, proprioceptors,
and touch receptors)

Auditory and vestibular
impulses from ears and
vestibular appeatus.

Cerebellum

15

Fig. 10. Synapse: Neurotransmitter seratonin bridges the gap between
nerve cells. After completing its “mission”, the seratonin is reabsorbed
into the nerve cell and decomposed by MAO into inactive by-products4
.

Fig. 11. Seratonin Pathways. Many of the important nerve pathways
assisted by the neurotransmitter seratonin begin in the brainstem
and extend upwards into the cerebrum5

.

Pre-synaptic
neuron

Post-synatic
neuron

Inactivated by
MAO

Reuptake

Seratonin

Trytophan

Inactivated by-Products

Receptor

Seratonin
Projection Pathways

16

Seratonin is implicated in a wide variety of psychological phe-
nomena, including depression, anxiety, obesity, and LSD hallucina-
tions. The anti-depressant Prozac, for example, elevates seratonin
levels in the synaptic cleft by blocking re-uptake of seratonin into
pre-synaptic neurons. (Seratonin levels cannot be raised by inges-
tion, because the molecule is too polar to pass through the blood-
brain barrier.) As another example, LSD mimics the shape of the
seratonin molecule and redirects nerve impulses down unfamiliar
and unstructured neural pathways, giving rise to hallucinatory per-
ceptions and experiences. In short, seratonin is the most important
neurotransmitter governing states of waking consciousness.

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