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Wolfgang Lutz: Dismantling a Myth: Chapter VII
DISMANTLING A MYTH: The Role of Fat and Carbohydrates in our Diet
by Dr. Med. Wolfgang Lutz
ISBN 3-921500-24-9 © 1986 by Selecta-Verlag Dr. Ildar Idris GmbH & Co. KG Planegg V. Muenchen, West Germany Original title "Leben ohne Brot", translated by Beatrice IdrisDuncan and Joy Wieser. All rights reserved. No part of this text may be reproduced in any form or by any means electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the publisher.
Service to Danish readers: Danish translation of certain difficult words and medical terms
(Excerpt 2: page 125-180)
Chapter VII: GASTRO-INTESTINAL TRACT
To anyone holding the view that carbohydrates are un-natural and harmful components of our diet it is quite obvious that the gastrointestinal
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Dr. med. Wolfgang Lutz: Dismantling a Myth: Chapter VII
tract, being in the front line, is exposed to the most danger. The idea of introduction of a low-carbohydrate diet as a general mode of therapy for disorders of this system is therefore logical, even if contrary to what has hitherto been practiced. Although it has been recommended that refined sugar and white flour be avoided in some gastrointestinal conditions (*12), and gliadin-containing types of grain in another (*3-6), carbohydrates as a whole so far have not been considered as undesirable energy sources.
Too Much Acid Distressing heartburn is often the first symptom to disappear following withdrawal of carbohydrates from the die However severe, and even if made worse by factors like the back-flow of gastric juice into the esophagus in hiatal hernia, the chances of success are good. If patients come back with the complaint that the diet is no longer effective and their heartburn has returned, a closer look usually reveals that some carbohydrates have again crept into the diet. Or a gastrointestinal infection cam be suspected - and treated. In some way carbohydrates appear to disturb acid regulation, i.e. the normal state of affairs in which acid is produced by the stomach only when it has something to digest. Only a sick stomach produces digestive juices when empty. This so-called 'fasting secretion' is the reason for the auto-digestion seen in gastric ulcers, or at least in ulcers near the pyloric sphincter, the duodenum or at the artificial exit of a resected stomach. Excess gastric acid is responsible for or provides the right conditions for, for development of a gastric ulcer, which is deducible from the fact that a typical gastric ulcer is found only in sites where contact with gastric juice is possible. If the stomach is resected so that no more acid can be produced a gastric ulcer is permanently cured. In rare cases, however, yet another ulcer develops even if further portions of the stomach have been removed in a second and even third operation. Surprising quantities of acidic gastric juice can be obtained from such patients by means of a stomach tube even in the absence of a test meal or any food whatsoever. Laboratory tests on such juice reveal that the fasting secretion of acid, just as in patients with a duodenal ulcer, approaches the so-called maximum secretion. By this, we mean the quantity of hydrochloric acid produced by the gastric glands when they are strongly stimulated by food or histamine. Under these circumstances the Zollinger-Ellison syndrome (*7-10), should be suspected, a condition first described many years ago by two Americans, Zollinger and Ellison. Tumors or tumor-like growths are found either in the islet organ itself or in its immediate vicinity, and can be removed surgically.
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Dr. med. Wolfgang Lutz: Dismantling a Myth: Chapter VII
Gastrointestinal Hormones In histological preparations of resected material from the above cases, cells were found closely resembling the alpha-cells of a normal islet (pancreas' Islets of Langerhans organ (*11-12). Large quantities of a substance extracted from them proved to be highly effective in causing acid secretion, and was finally identified as 'gastrin' (*13). Gastrin is the hormone responsible for gastric secretion. Although not the sole agent eliciting acid secretion it is certainly the most important. I do not intend to go into the physiology of gastric secretion in detail since it would take us too far from our main theme. However, it should be mentioned that apart from nervous influences, which for example cause our mouths to water at the sight or smell of food, or even at the sound of the dinner gong, it is gastrin which sets off the flow of gastric juice at meal-times (*14-15). The substance is produced at the base of the mucous glands of the pylorus when the latter is in contact with food. It enters the blood stream and thus arrives at the glands in the stomach's body where hydrochloric acid and protein-splitting enzymes (pepsin, cathepsin) are produced, and where it can directly and indirectly influence the stomach movements, particularly the emptying mechanism. Thus the stomach is provided with a kind of self-regulating mechanism. Gastrin is formed when food enters, and this leads to secretion of the enzymes and hydrochloric acid necessary for digestion in the stomach. When the stomach contents have been sufficiently acidified the sphincter opens and the acid contents enter the duodenum. Here substances are produced (secretin, cholecystokinin, and enterogastrin for fatty foods) which stimulate the activity of the pancreas and also affect the neutralization of the acid from the stomach, at the same time arresting the efflux of further acidic stomach contents. The important point to note is that too much gastrin is produced in all cases of hyperacidity, in gastric ulcers and particularly duodenal ulcers, and in extreme quantities in the Zollinger-Ellison syndrome. But this gastrin arises in the pancreas and not in the stomach (*13), (*16-23). In recent years the so-called Verner-Morrison (*24-29), syndrome has been considered to be a mirror image of the Zollinger-Ellison syndrome. The former is associated with diarrhoea, low potassium levels in the blood, and with a deficiency in free gastric acid. Small tumors contain abnormally-large amounts of secretin (a hormone similar to gastrin but with a different effect) may be found in the pancreas. The two syndromes, however, are not sharply distinguishable from one another as this description would suggest. The symptoms can occur in a variety of combinations, and may be accompanied by disturbances of a diabetic type such as elevated blood sugar or too little sugar (hypos), just as in the case of tumors of the beta-cells. The patients often complain of headaches and hot flushes in connection with eating, and in fact at such times it is possible to detect abnormallyhigh amounts of serotonin in the blood and urine. The serotonin is a hormone believed to be formed in the so-called light cells of the islet
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Dr. med. Wolfgang Lutz: Dismantling a Myth: Chapter VII
organ of the pancreas. Some investigators consider them the source of all Islet cells (*30-33).
The Pancreatic Duct Syndrome
In order to understand how the activity of the various segments of the gastrointestinal tract is controlled we have to go back to what we said earlier about neurosecretion in connection with the hypophysis. The nerve cells of the hypothalamus secrete polypeptide substances which reach the hypophysis via small blood vessels and are then either released into the blood when needed, or stimulate the hypophysis itself to produce protein hormones such as the gonadotropins, ACTH, human growth hormone, thyrotropic hormone, etc. The closer we delve into the problem of neurosecretion the clearer it becomes that it is not confined to the hypothalamus and hypophysis but is in fact a widespread phenomenon. Communication between nerve cells is effected by means of chemical substances known as neurotransmitters. In this way one nerve cell can contact another, its messages can be conveyed either within an organ by means of peptide hormones, or over larger distances by means of the proteohormones which are produced in certain endocrine organs at the behest of peptide hormones. The gastrointestinal tract seems to be especially influenced by peptide hormones formed on the spot by neurosecretion. The neurosecretory centre is situated in the pancreas in round, glandular structures termed "islets" (of Langerhans), which apparently secrete insulin directly into the blood since there is no sign of effluent ducts. Insulin is an antagonist of glucagon. The large number of different types of cells in the islets would suggest that they form still other, as yet unrecognized, incretions. In addition to the hormones of the pancreas a larger number of more or less well-known polypeptides with their own regulatory functions is produced in the stomach and intestine. Gastrin is one of them, and although normally produced in the cells of the stomach and reaching its target organ, the gastric mucosa, via the blood stream, it can under certain circumstances also be produced in the small intestine and in the islets of the pancreas. As has already been mentioned, the islets are also the source of secretion. Since the development of a highly sensitive method for detecting polypeptides, the RIA or Radio Immune Assays, more of this group of substances have been discovered. They are apparently concerned with governing the activity of the gastrointestinal tract and its glands, and include cholecystokinin or pancreokinin, enteroglucagon (an insulin antagonist like pancreatic glucagon), and the gastroinhibitory polypeptide (GIP) from the duodenum and upper small intestine, which is chemically very similar to secretin and glucagon. GIP inhibits the formation of
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as well. Some members of the clan have built larger houses (the islets of the pancreas) whereas others still live in the open or in very primitive shelters. They respond to too much or too little blood sugar. primarily regulate carbohydrate metabolism. It leads to hot flushes and diarrhoea if produced in excessive amounts. In the course of further developments. Not only is insulin or glucagon produced.html[24/1/2013 1:49:49 μμ] . That the disturbances already discussed are due to the carbohydrates in the diet.scdiet. Originally. Nonetheless the clan sticks together through thick and thin. Even though insulin and glucagon are apparently produced exclusively in the islets the other gastrointestinal hormones can be produced elsewhere too. med. In the embryo the primitive pancreatic duct from which the pancreas develops appears to harbour the primitive form of every kind of cell producing gastrointestinal hormones. the entire gastrointestinal tract was probably able to produce these regulatory polypeptides. so as to serve the body in the best possible way. VIP) has an insulinotropic action. however. Yet another gastrointestinal hormone is the socalled Vasoactive Intestinal Polypeptide (VIP). In the course of this transition glandular organs have fused. And least one of the gastrointestinal polypeptides (GIP. the neurosecretory portion of the pancreas is concentrated in the islets. secretin. Neurosecretion is nevertheless not confined to the pancreas and its islet organ. and their sense of loyalty is such that if one of the members is injured. in order to produce the liver and pancreas. like gastrin. and is thus insulinotropic. duodenum. insulin and glucagon. at least under pathological conditions. is probably due to the fact that the two chief hormones of the islet organ. in primitive forms of life. Esophagus. serotonin. whose only present connection with the intestine is via the ducts through which their secretions enter its lumen. i.Dr. http://www. but gastrin. but still plays a role over the whole of the gastrointestinal tract. the various parts took on a specialized function. Wolfgang Lutz: Dismantling a Myth: Chapter VII gastric acid and promotes insulin production in carbohydrate digestion. and the presence of carbohydrates in the intestine. With this in mind I suggested the term "pancreatic duct syndrome" for all disorders triggered by gastrointestinal peptides. and probably others as yet unrecognized. an outcry will be heard from the rest. and that they disappear if carbohydrates are restricted. stomach. As has already been mentioned. The cells producing gastrointestinal hormones can be thought of as belonging to a clan dating back to primitive times. differ not only in structure but also in function. small and large intestine. Apparently a strong stimulus to one gastrointestinal polypeptide or to its site of production involves to greater or lesser extent all of the others. Emanation or irradiation is a familiar idea to members of the medical profession. GIP.org/7archives/lutz/lutz7. It has already been mentioned as representing the probable starting point of diabetes and the reason for hyper-insulinism in carbohydrate eaters.e. VIP.
The body produces not only insulin and glucagon but secretin. Viewed in the light of such observations it would appear that gastric surgery is being performed in the http://www. as a result of prolonged excessive intake of carbohydrates. Although I have not yet had the opportunity to put my idea into practice I am convinced that the Zollinger-Ellison syndrome would. insulin production is stepped up much more rapidly and is more persistent than if the sugar is given intravenously. med. reacts "allergically" to the kind of food that was introduced with grain cultivation.Dr. Similarly. And it is in this fasting state that the tendency to hypoglycemia becomes manifest. the opposite holds true and sugar in fact is much more effective if it enters the body via the intestines. They are the outposts which serve to warn the headquarters that sugar is on its way. pancreozymin. i. It might be that these outposts represent the seat of our civilIzation diseases (see also chapter XIII). if not the result of a malignant tumor. Like a beleaguered garrison it finally resorts to producing ammunition in workshops originally intended for quite different purposes. Surprisingly. and even more to the large quantities of sugar that we have consumed since the development of sugar-beet cultivation. with its resultant need for increased glucagon (and gastrin) production. Wolfgang Lutz: Dismantling a Myth: Chapter VII It was previously assumed that sugar injected direct into the blood necessarily has a greater effect than the same quantity administered orally. This was discussed in the chapter on diabetes and hypoglycemia. vasoactive intestinal polypeptides. gastrin is finally produced by the pancreas as well as by the stomach. and above all gastrin as well. The reason for this is I think that when sugar enters the intestine insulinotropic substances are produced the mucosa and as their name suggests they stimulate the pancreas to produce insulin. via the above-mentioned outposts.org/7archives/lutz/lutz7.e. That this somehow describes the situation is clearly borne out by the observation that all symptoms connected with the pancreatic-duct syndrome disappear upon withdrawal of carbohydrates from the diet.scdiet. There is little doubt that in addition to GIP or VIP the typical fluctuations in the blood-sugar level and the attendant counter-regulation so typical or carbohydrate consumers play a special role. Our primitive ancestors learned to live without carbohydrate over the course of millions of years and now our body. just as does Morbus Cushing. eventually respond to a low-carbohydrate diet if continued long enough.html[24/1/2013 1:49:49 μμ] . It is striking that in cases of hyperacidity and duodenal ulcer the maximum acid production in connection with eating is the same as in normal subjects. The 'clan' reacts as to an enemy against which it is in fact no longer armed. whereas the socalled basal secretion in the fasting state is higher than normal. HIstological similarities between the gastrin-producing cells in tumours from Zollinger-Ellison patients and the glucagon-producing alpha-cells of the islet organ support this theory. It throws in all the resources at its command even if the weapons involved are not suited to the purpose. and that insulin has to be produced for its breakdown.
e.Dr.html[24/1/2013 1:49:49 μμ] . At this stage the mucosa presents a fairly normal aspect under the microscope. X-ray pictures show the so-called fasting secretion. at any rate less common than gastritis. only a form of mutilation leaving the root of the trouble untouched. i. The frequent mention of ulcers in connection with gastro-intestinal disorders does not necessarily mean the condition is as commonly encountered in a physician's practice as in a hospital. after all. X-ray pictures of children with gastritis almost invariably http://www. indicating that adequate substance is still available. That this type of gastritis represents the initial stages of the disease and is not another variation of it is demonstrated by the observation that it is the common form in children and young subjects where the disorder is not long-standing. An operation is. Enteritis. Even the duodenal ulcer is a relatively rare condition. It was originally believed that mucosal irritations could be detected by means of an X-ray. A so-called callous gastric ulcer. These ulcers are stress-induced and a transition to a new diet entails further stress. however. We know that the story begins with excessive irritation of the mucosa and increased production of gastric acid and digestive enzymes. Their histological examination has provided us with more detailed knowledge as well as some surprises. where there is simply an accumulation of cases that cannot be treated at home. In my experience the only permanent relief for an ulcer lies in consistent adherence to a low-carbohydrate diet. enteritis and colitis. is full of juice. whilst leaving the cells that are active between meals to produce the fasting secretion and ulcers. Ulcers The above ideas have been fully confirmed by my experience on the effect of a low-carbohydrate diet in patients with hyperacidity and duodenal ulcers. Gastritis. even an 'empty' stomach.scdiet. The removal of the lower part of the stomach (antrum) deprives the organism of a gastrin production geared to the requirements of the digestive tract. biopsy methods have been developed in which small pieces of mucosa are removed from the stomach or intestine. The mucosal folds are either normal or swollen.org/7archives/lutz/lutz7. however. Both gastritis and ulcers heal if carbohydrates are restricted. med. Therefore one has to give small amounts of cortisol first until the ulcer has healed and then slowly introduce a low-carbohydrate diet. More recently. Wolfgang Lutz: Dismantling a Myth: Chapter VII wrong place. Lack of discipline in this respect usually leads to the operating table. I am convinced that we would spare many of our patients the need of undergoing surgery if we were to put them on a low-carbohydrate diet. should not be treated immediately by simple restriction of carbohydrates.
html[24/1/2013 1:49:49 μμ] . in the absence of adequate amounts of acid or pepsin. This is no great misfortune and the patient can easily avoid such foods if made aware of the necessity.org/7archives/lutz/lutz7. In my opinion we are dealing with nutritional diseases provoked by the high carbohydrate content of the diet of civilized man. VIP) production which might to be responsible for the deficiency of acid. Although no-one believes today that gastritis and enteritis are expressions of bacterial or viral irritations. the vitamin is not absorbed but is lost in the stool.scdiet. however. Raw connective tissue. secretin and so on. In this manner the passage of further acid contents is delayed until the acid has been neutralized by the alkaline juices of the duodenum (bile and pancreatic juice). renders it absorbable. and perhaps with other so far unknown substances. The sphincter is open and the stomach is seen to empty its contents prematurely into the small intestine. until finally it http://www. results in malfunction of the emptying mechanism of the stomach. is insufficiently broken down and even the juices from the liver and pancreas in the small intestine are unable to complete the process. Wolfgang Lutz: Dismantling a Myth: Chapter VII reveal normal or enlarged mucosal folds and a copious fasting secretion. med. and in the initial stages the mucosa is stimulated to produce excessive quantities of digestive enzymes. Heartburn and other symptoms of hyperacidity should not be allowed to mislead one. An experienced eye recognizes this in an X-ray picture. what was initially an excess of gastric acid and pepsin is now too little and in some cases NO gastric acid can be demonstrated at all. Since vitamin B12 is essential for growth of the entire organism as well as for maturation of the red blood corpuscles it is understandable that patients of this type suffer from a variety of disorders including pernicious anaemia.Dr. Carbohydrates elicit production of gastrin. Such patients often suffer from diarrhoea if they eat raw or smoked meat. but it becomes very obvious in pieces of mucosa examined under the microscope that atrophy of the mucosal layers has set in and large numbers of mucosal cells have perished. But breakdown of food is not the only function of the stomach. If the mucosa is atrophic and unable to form intrinsic factor. and this at the wrong time. or --. Even with such a thin mucosa the stomach can still produce acid in quantities sufficient to cause a severe burning sensation in the lower oesophagus. In the end. The mucosal atrophy. The longer the process has time to develop the less fasting secretion is seen. it produces a substance. whereas the normal stomach moves its contents in a downward direction (except in vomiting). which by combining with vitamin B-12 (Cobalamine) in the food. a diseased stomach can in some cases regurgitate its contents into the oesophagus. The deficiency of acid. whether due to chronic damage. Normally the gastric sphincter shuts as soon as the acid content in the duodenum rises. The stomach thus loses its function as a depot and the rest of the digestive tract is overburdened by receiving insufficiently-digested food. If the stomach produces too little acid the point of neutralization is reached sooner.to excessive secretin (GIP. we still have no exact idea as the cause of these common complaints.as mentioned earlier --. For example. with the result that it empties too rapidly. the Intrinsic Factor. leads to a series of consequences for the digestive apparatus.
I have described these processes in detail because their understanding is of vital importance in treating such patients. In fact the diet that I recommend for patients can later include large quantities of fat.scdiet. Wolfgang Lutz: Dismantling a Myth: Chapter VII succumbs. for unknown reasons. often occur together. the duodenum and small intestine (enteritis). low-protein diet. enlarged mucosal folds and other signs of mucosal swelling and hyperacidity. rumblings. nausea and vomiting in the fasting state. and the patients have a pot-belly similar to that seen in primitive peoples whose diet consists mainly of carbohydrates. usually tine morning and following breakfast. a disease characterized by a state of malnutrition. A typical disorder of this kind is celiac disease in infants which results from oversensitivity to the protein gliadin (*3-5). med. The process is enhanced by the tendency to tissue deterioration seen in carbohydrate-eaters. the sooner can the patient expect to be relieved of his complaint by means of a low-carbohydrate diet. and resulting from their onesided. the more fasting secretion. The patients feel distinctly better even within a few days and usually are completely well again within a couple weeks. inability to digest fats and colicky stomach pains. swollen belly and oedema.html[24/1/2013 1:49:49 μμ] . Fat is not the Culprit Because fat in the diet evokes unpleasant symptoms in many patients. although in most cases the symptoms of one or another of the organs or hormones of the pancreatic duct system usually dominate the picture. the opinion has arisen that a gastric diet has to be low in fat. The better-established the process. In very severe cases of enteritis the small intestinal loops are knotted and swollen. and large intestine (colitis). Every organ has to make a contribution to the toll of protein that is constantly demanded of carbohydrate-eaters by the mechanism of the adrenocortical hormone. Even in civilized countries we find equivalents to this disease in which. The process is the same as that already discussed in connection with the striae. The characteristic symptoms of gastritis are a feeling of sickness. But an improvement can be expected in any case. gas. Chronic inflammation of the stomach (gastritis). But a low-fat diet never brings about a cure. dietary fat cannot be utilized but is eliminated in the stool. choking. The children often already suffer from Kwashiorkor. But if they attempt to eat carbohydrates they quickly notice that these were responsible for the discomfort and not the fat or other foods. Fasting pains that are usually improved by food to return as after-pains a couple of hours later suggest disease of the gastric pylorus or duodenum.org/7archives/lutz/lutz7.Dr. a substance present in http://www. the thinner the mucosa and the more rapid the stomach emptying the longer it takes to effect a cure. Enteritis is usually accompanied by a full feeling after meals. a lump in the throat and a burning sensation the stomach. and in some cases the less complete will this cure be. The more recent the process the earlier the patient comes for treatment.
It can hardly be assumed that the defect is of genetic origin in these patients but rather that the damaged mucosa permits the passage of antigens which elicit the production of antibodies in the blood. it is quite a simple matter to rear the patients on a gliadin-free diet and at the same time ensure their normal development.g. vegetables. My explanation is that the diet gives the intestine a chance to heal. Therefore bread Is forbidden to such patients. This would mean that the mucosal damage represents the first step in the disease and can thus be regarded as the cause and not the effect of autoimmunization. When the condition due to low-carbohydrate has improved to the point where the patients can safely begin to add again some carbohydrate to their diet it is bread and other flour-containing foods that are least-well tolerated. potatoes and pulses. which they report as occurring after wetting of the skin (bathing or sweating). but also antibodies to hens' eggs. rice and potatoes contain none of it. and even to rabbit protein (*34-36). the mucosa recovers and no longer lets through (or completely breaks down) the food constituents that could previously enter the blood and elicit an allergy. rye. Itching (Pruritus) I have another reason for believing that this is indeed the situation. and for this reason the argument that such antibodies are found in 'healthy' subjects and has nothing to do with enteritis does not hold water. It is small wonder that the gliadin-free diet automatically prescribed for adult enteritis is not particularly successful. barley and oats. They may replace the carbohydrate with fruits.scdiet. http://www.Dr.html[24/1/2013 1:49:49 μμ] . The absence of the necessary enzyme for splitting gliadin from the intestinal villi suggests that the gliadin sensitivity is of genetic or immunological origin. as in other forms of chronic enteritis. and also after eating certain food. I believe that this is not because gliadin is responsible for the enteritis but because it has a greater allergic effect than other unsplit proteins if it gets into the blood. med. Only when closely questioned do they admit their discomfort. A gliadin-sensitive type of enteritis is also encountered in adults in the condition known as endemic (as opposed to tropical) sprue (*5). Since I have been so much concerned with the clinic and radiological signs of enteritis I have come to realize just how many apparently clinically healthy patients suffer in this way. Wolfgang Lutz: Dismantling a Myth: Chapter VII wheat. Since corn. A large number of patients visit the doctor on account of itching.org/7archives/lutz/lutz7. not only can antibodies to gliadin or its individual fractions (e. milk. In this disorder. fraction III) be demonstrated. If these patients are put onto a lowcarbohydrate diet the itching usually disappears after a couple of months.
For the latter disorder he recommended other diets. Wolfgang Lutz: Dismantling a Myth: Chapter VII The fact that carbohydrates have something to do with gastro-intestinal ailments was already known to Prof. who recommended restricted carbohydrate intake for the so-called fermentation dyspepsia (when stools are acidic and smell peculiarly acidulous). Colon Disorders Constipation On the whole. Had he continued on this path.html[24/1/2013 1:49:49 μμ] .Dr.scdiet. In the long run they are most likely health-detrimental since they cause an evacuation of the bowel by irritating it. and because they have been demonstrated to disturb the mineral metabolism (loss of potassium). The gases produced during this process can he taken up by the blood and detoxified in the liver. But without laxatives the patients have no stool. Without laxatives the stool of chronically constipated patients is broken down by putrefaction bacteria. he surely would have observed that not only "fermentation dyspepsia" but also "putrescent dyspepsia" responds to carbohydrate restriction. but it is mostly restricted to the female gender since the female hollow organs. Billions of dollars are realized by producers of laxatives but the ailment is treated only very superficially. Experience shows that the effect of laxatives diminishes with time so that the dosage has to be increased or the preparation has to be changed. tend to retain their contents in order to preserve the fetus. Lampert. When these patients come to the physician. but less susceptible to ureteric colics. Now the last motivating force for the bowel abolished because the fermentation process stops and the entire gastrointestinal tract calms down. and for abdominal flatulence (*36). Even the female patients who are not normally constipated react to a low-carbohydrate diet with an indolent bowel. H. This constant irritation of the mucosa finally leads to the most predominant complaint among chronically constipated patient: they feel constipated although their colon is totally empty and laxatives would not be necessary. he justly recommends the discontinuation of the medication. constipation is the most common illness of the colon. That is why I have not initially treated constipation with this diet. med. However. Or the tormented gut finally rids itself of the stools by the so-called paradox diarrhoea. instead of expelling them. If they are put on a low-carbohydrate diet then they definitely have no evacuation. which include the uterus. over the years I solved this problem by instructing my patients to continue the laxatives and by prescribing them instead daily cleansing enema with one and a half litres of warm water without additives in order http://www. Therefore women are also more susceptible to enervation and infections of the urinary tract.org/7archives/lutz/lutz7.
These "buttons" are covered by peritoneum on the outside and are therefore fixed in place so that they can no longer retract. The number of these so-called diverticula can vary greatly. sooner or later the stool will normalize.html[24/1/2013 1:49:49 μμ] . an Sshaped loop between the rectum and descending colon on the left side. but undoubtedly it will be successful sooner or later. Here an inflammation creates symptoms like an like an appendicitis would be on the right. although his original diet may have contained somewhat moreundigestable food residues. however. at times it necessitates immediate surgery. In children it can take one or two days. the inflammation. Here the prevention of an initial constipation through the regular application of cleansing enemas as described in the previous chapter on constipation is particularly important. The diverticula are most-commonly localized in an area of the large bowel called the pelvic colon. Finally. do not believe in this fiber hypothesis which has become very popular over the last few years. which very often exists in the gastro-intestinal canal of consumers of normal diets. namely the muscular weakness with the enlarged spaces http://www. Only when they become inflamed does the resulting peritonitis create problems. med. These finger-like mucosa protrusions get under the peritoneum. Under the continued diet. and which spreads to the diverticula. Usually a diet rich in fibers is recommended for the treatment of diverticulosis because years ago scientists observed that African Negroes who eat fiber-rich diets have no diverticulosis and also get few other colon diseases (cancer). which covers most of the colon and becomes deformed into button shapes. Diverticulosis The wall of the colon consists of several muscle 1 which cross each other and. On the inside they contain stool or other bowel contents. and in older persons a few months.org/7archives/lutz/lutz7. They generally do not cause typical symptoms. The existing diverticula will not disappear but no new ones will form. like a lattice. leave open spaces through which blood vessels and nerves pass. After a few months the patients will have normal stools and no further complaints relating to their diverticulosis. When these muscle layers weaken. the spaces enlarge to the point that mucosa can protrude through the meshwork. I also treat diverticulosis with a low-carbohydrate diet very successfully. Therefore. in young adults one or two weeks.scdiet. Most of all. I. the bowel musculature will strengthen and the original cause of the diverticula. Man is a hunter and gatherer and not a herbivore. will heal. There are patients with one or two diverticula and others with 20 to 50. Wolfgang Lutz: Dismantling a Myth: Chapter VII to bridge the period of initial change for the worse.Dr.
because I was sure that the problem would have resolved by then. the foreman of a Mercedes-Re service station. med. http://www.html[24/1/2013 1:49:49 μμ] . Particularly viruses multiply in the gut. I remember one patient. it is not able to absorb water and to produce stool of a normal consistency. then chronic diarrhoea surely cannot be considered solely as motility disturbance. Whether this was not so in medicine? No. this illness is evaluated differently by various people. On long trips one enters regions with unfamiliar germs to which one is not yet resistant and promptly becomes ill. the bowel evacuates because it does not want to contain tts contents. Wolfgang Lutz: Dismantling a Myth: Chapter VII between the bundles. A frequent traveller knows that "Montezuma's Revenge" is not restricted to Mexico. This was indeed the case. Some have had diarrhoea for years and have become so used to it that they do not perceive their condition as unusual anymore. The many "outsiders" among the medical scientists will surely agree with me wholeheartedly. Here I mean the chronic diarrhoea.Dr. which is much more common than is generally assumed. and finally lead to a general illness. disappears. I had to admit: we have the so-called scholarly medicine with certain dogmatic views. not one knew how to help him. He came. Rather. all service stations in the whole world would be informed and instructed how to repair the defect.org/7archives/lutz/lutz7.scdiet. Diarrhoea I am not talking about the short periods of liquid stool which occur several times a year due to infections with various bacteria or viruses. who came to me years ago and told me that he has had diarrhoea for over 15 years now and that he had consulted at least ten physicians without any success. thanked me and told me that of the many doctors he consulted previously. However. when he had to out anyway. he would immediately look for a rest stop since at times he could hold his stool not even for one minute. and other views are not tolerated. which is unable to move its contents ahead (which Is hardly the heart of the problem). When chronic constipation is to be understood as disturbed motility of the bowel due to an insufficiently-developed colon musculature. enter the blood stream. I prescribed him a low-carbohydrate diet and told him to see me again in a fortnight. He did not dare to leave his house anymore. One just has to visit larger toilet facilities in a restaurant or train station to hear the familiar noise in one of the neighboring stalls. He did not quite understand that. because if there is mechanical trouble with one the Mercedes-Benz models. therefore it releases the contents of the small intestine in a more or less unaltered form.
When diarrhoea after an acute gastrointestinal inflammation does not heal and persists for years. therefore we are dealing here with a so-called "auto-aggressive disease". believing it to be inappropriate. nothing else but the extrapolation of the chronic enterocolitis. Here the illness is not limited to the mucosa alone but Involves all levels of the http://www. by weakening it with its defensive forces.scdiet. The body tries not only to fight the infectious organism. see page 239) and. as described above. the body starts to mount an immune reaction not only against the infectious organisms but also against the affected organ. The myocardial inflammations. most of all. a "Crohn's" always has a pro-dromal phase in which the patient has diarrhoea over some period. The former is less harmful and therefore should be tried first. can most easily be explained in this way. chronic diarrhoea will always heal under the above-mentioned circumstances and with a low-carbohydrate diet. develops into a Morbus Crohn (English: Crohn's Disease). Frequently an immunosuppressive therapy is not even necessary. These affect not only the gastro-intestinal tract but also the nervous system (multiple sclerosis. Furthermore. One can hardly understand the multitude of existing auto-immune diseases in any other way. by subduing the immune reaction. but also to destroy its basis for existence. One has to know this in order not to break off the diet. which frequently occur even among young people. acute diarrhoea is followed by a lengthy period of liquid or soft stool. one not only has to restrict carbohydrate intake.html[24/1/2013 1:49:49 μμ] . It is. For reasons which are not yet known in detail. rebels against having to work again. which should be treated with cleansing enemas. but also to slow down the immune reaction. This can be done with cortisone or gold preparations. med.org/7archives/lutz/lutz7. Crohn (*37) is not clear. I got the Impression that initially this mechanism does not necessarily have to be considered morbid. so to speak.e. this type of diarrhoea can be halted by giving gold or cortisone for several days. After one or two weeks the stools normalize although often accompanied by abdominal pain. I prescribe 10 mg Prednisolon or 8 mg Triamcinolon (early in the morning for eight to ten days) and repeat the same treatment in case of a relapse. Apparently the large bowel. the affected organ. which has not actively worked for months or years as it has discharged the contents coming from the small intestine in a more or less unaltered form. Ultimately. Crohn's Disease Why chronic diarrhoea. because in my experience. Sometimes even a period of constipation follows.Dr. that the attack against the affected organ is part of the immune reaction itself. Wolfgang Lutz: Dismantling a Myth: Chapter VII In my experience. although through therapy and the elapsed time interval the Infectious organism has been killed off and the Infection has been overcome. since laxatives should be avoided at all cost. the heart. named after the American physician Dr. i. Apparently there are connections between the two.
they worsen acutely. The distinction between Crohn's and ulcerative colitis is of significance.) must be removed surgically if they do not heal spontaneously. often it affects only the lower small intestine. is characteristic. in contrast to ulcerative colitis. and the stomach can be affected as well.org/7archives/lutz/lutz7. those with involvement of the large and small Intestine. its frequency Increases upward. if possible. etc. A form of InflammatIon. also narrowings in the bowel may require surgical intervention. However. http://www. Crohn's disease. through which the bowel contents can enter the bladder. Potential remnants (fistulas. until iron level. but they also can melt down. When I started to treat my colon-patients with a low-carbohydrate diet 25 years ago. There are intermediate forms between ulcerative colitis and Crohn's disease: patients with intestinal bleeding and fistulas. fever and so on. exit next to the rectum or through the abdominal wall. Wolfgang Lutz: Dismantling a Myth: Chapter VII bowel. its frequency decreases continually toward the small intestine. called granulomatous. Crohn's disease becomes more common from year to year. one should wait until the underlying illness has resolved. or. Morbus Crohn differs from ulcerative colitis in that there often is no intestinal bleeding. have diarrhoea. However. Often they can only be removed surgically and often without ideal results. Infiltrations around the bowel in the peritoneal cavity can result. Crohn's rarely occurs in the rectum. There are individual patients who have difficulties changing from the high-carbohydrate standard diet to the lowcarbohydrate diet. heals with great probability within six months to one year and usually without complications. erythrocyte sedimentation and stools have normalized. the gall bladder. Ulcerative colitis. most frequently. med. this rarely lasts longer than a few weeks. form abscesses. and in its different localization. the duodenum.Dr. at least sporadic bleeding occurs and no fistulas form. there was no Crohn's patient among them. and the lymph nodes. Lately they have become almost as frequent as patients with ulcerative colitis. on the other hand. prefers the rectum. the vagina. and leave fistulas. the mesentery.html[24/1/2013 1:49:49 μμ] .scdiet.
med. I do not believe this. Crohn's disease generally thought of being incurable can so be shown to be very well accessable to dietary measures. which Is shown in the lowest line. 16: 103 patients suffering from Crohn's disease were treated by a low-carbohydrate diet. After a quarter of year most patients (85 percent) showed remarkable improvement in their health. This corresponds to today's widespread conception that only the refined carbohydrates are harmful. Perhaps then also a breakthrough regarding other diseases can be reached. Starch turns into sugar in the bowel anyhow. perhaps somewhat slower than if one eats sugar and only dextrose. sugar and white flour. more than 60 percent were asymptomatic. In March 1985 I published 103 Crohn's patients (Fig. and with this information I hope to be able to convince my colleagues to first try to treat these unfortunate patients with diet instead of cortisone and scalpel. In recent years German and English authors (*38-39) have found that Crohn's patients experience a certain improvement after relinquishment of sugar and so-called refined carbohydrates. At least this http://www.org/7archives/lutz/lutz7.html[24/1/2013 1:49:49 μμ] . After half a year. 16) which I had treated up to then (*74-75). i. I am convinced and have seen again and again that bread and particularly whole meal bread is not tolerated well by patients with gastro-intestinal ailments. This is in contrast with ulcerose colitis. Wolfgang Lutz: Dismantling a Myth: Chapter VII Fig. after one year more than 70 percent and after one and a half year about 85 percent.Dr. To the contrary. improvement of which runs much slowlier on the same diet and often is interrupted by relapses. but in principle the result is the same.e.scdiet.
based on the fact that in France and in the WalIonic part of Belgium. Wolfgang Lutz: Dismantling a Myth: Chapter VII recommendation points in the right direction: restriction of carbohydrates. However. Only the mucosa and the underlying muscle layer are affected. and no flstulas. A toxic megacolon can develop at any time as long as the ulcerative colitis is active. definitely less natural than carbohydrates. so that their metabolites enter the lymphatic vessels and cause the described granulomatous inflammations. but I have none who has eaten no carbohydrates. Instead he sees a reddened mucosa which starts bleeding at the slightest touch or when the overlying purulent layer is removed. when carbohydrates are consumed in large quantities. Recently the consumption of margarine and other artificial fats as cause for Crohn's disease has been discussed. This could be quite true. flat-cake-shaped stools. but I have among my patients cases of Crohn's disease who have only eaten natural fats and never artificial fats. Artificial fats are. But I have seen many times that grain protein is not the cause of this colon disease. There are no infiltrates. but responded to carbohydrate restriction despite of gluten. and impaired development. considering human evolution. The physician diagnoses hemorrhoids which cannot be found with the rectoscope. It may begin one day quite harmlessly with the appearance of blood in the stool. which is completely motionless and has lost most of its function. no perforations. unsuccessfully. if the disease would not become life-threatening by turning into the so-called toxic megacolon or by degenerating malignantly. the duodenum. Again and again patients were on a gluten-free diet for months or years. Crohn's disease occurs less frequently (*40-41). In mild cases this process is restricted to the rectum. in time both are destroyed to an extent that the whole colon contracts into a short cylinder. which is accompanied by pulpy.scdiet. One speculates that the artificial fats are not broken down and absorbed properly. Yet I can imagine that the consumption of artificial fats supports the tendency to develop the disease. and diarrhoea. the gallbladder. the increase in Crohn's disease in the German-speaking countries in the last years correlates with an increase in margarine consumption. Indeed. At least these patients still have their natural anus and not an artificial abdominal one. because the alcohol-soluble fraction of the grain protein causes the so-called Celiac Disease in children. particularly for chronic diarrhoea and Crohn's disease. and the stomach are never involved. In contrast to Crohn's Disease the small intestine. abdominal pain.html[24/1/2013 1:49:49 μμ] . Only three http://www.Dr. where less artificial fat and more butter is consumed.org/7archives/lutz/lutz7. Often a gluten-free diet is tried. the more severe the disease the further it extends upward. med. Ulcerative Colitis Ulcerative colitis is located at the lower end of the continuum "chronic diarrhoea ---> Crohn's Disease". All this is bad enough but the patients would and actually do put up with their bloody stools.
Surgical Therapy Of all patients with ulcerative colitis 20 to 30% sooner or later have to be operated on if they do not eat a low-carbohydrate diet.scdiet. Of course. a stage of the disease so advanced that the transition to a new diet is not tolerated or effective anymore. It can also alter the hormonal status. died two months after operation of thrombo-embolism. which can compromise sexuality.org/7archives/lutz/lutz7. and the joints. it inhibits their activity and thereby improves immune diseases. since usually the question is whether the colon can be preserved or not. in severe cases of ulcerative colitis one will have to ignore these side. Of course I cannot be certain that one or the other who was lost to follow up (who did not answer my repeated inquiries) did not have to have his colon removed after all. One was a relatively young man who had recovered quite well under a low-carbohydrate diet and had gained 20 kg of weight. Wolfgang Lutz: Dismantling a Myth: Chapter VII patients of a total of 285 that I have treated up to the end of 1984 have died. but with it all other symptoms of a chronic colitis which can occur in the eyes. skin. in men sexual potency often declines. The constant loss of blood and iron and the continuously-deteriorating general health necessitate the removal of the colon and the creation of an artificial anus (*42). Another one underwent surgery without my approval. the white blood cells and their progeny. However.Dr. Not only the colitis is resolved because the diseased organ is gone. I have observed that the gonadotropin secretion decreases (see page 94). sometimes even advantageous. With the surgical removal of the entire colon the disease is finally overcome. the third. Certainly there is a limit even to what a lowcarbohydrate diet can do.html[24/1/2013 1:49:49 μμ] . They manifest due to an outward radiation of the immune reaction from the colon to other organs. a very old lady. or other reasons necessitated surgical intervention. Only two of my patients had to have their colon removed. In women this is not so bad. because menstrual breast pain disappears. Gold is directly taken up by the immune-competent cells. Gold is a well-known anti-rheumatic drug which was used extensively to treat joint inflammations before the newer non-steroidal anti-inflammatory drugs and cortisone preparations became available. http://www. med. and that is gold therapy. Suddenly he became acutely ill with high fever and abdominal pain. He was admitted to a clinic but was operated on too late because he had fiercely refused this operation. There is still one other possibility before the operation. The side effects usually disappear with time.effects. Gold therapy should only be applied to severe cases of ulcerative colitis because sometimes it causes side-effects like week-long fever or urticarial skin rashes. Maybe the patients gave up after months or years of dieting. The leukocytes are not able to attack the colon's mucosa anymore.
they disappear after several months of a low-carbohydrate diet. and a normal-looking rectal mucosa (figure 17.Dr. approximately 60% were without complaints after two years. top).scdiet. until the bleeding stopped. Alone the number of more than 200 patients is unusual for a practitioner. http://www. Many patients came to me from hospitals.html[24/1/2013 1:49:49 μμ] . eight years." Of the first 74 patients. (*48). iron levels normalIzed. Take your medication and come back to us when you have a relapse. Wolfgang Lutz: Dismantling a Myth: Chapter VII Low-Carbohydrate Nutrition I can only report positive about my ulcerative colitis patients (*43-47). It indicates that one patient recommends him to another. whom I have treated until 1979 with a lowcarbohydrate diet (*46). see also color plate following page 348). The remaining 40% took longer. Eventually you may have to be operated anyway. had normal laboratory values. four. six.org/7archives/lutz/lutz7. Then the intestinal mucosa looks like that of healthy individuals. One commonly sees yellow spots at the beginning of treatment. Eat whatever you can tolerate. Flashlight photographs yield interesting findings (color plate following page 348. sum of 12 cases average per case kcal per 24 hours Percent calories of main 20 nutrient sources protein 1241 103 422 fat 1866 156 1395 66 carbohydrate 868 72 295 14 kcal 25422 2118 - kilojoule 106391 8866 - Table 1: Values for actually consumed nutrient components of twelve patients with ulcerative colitis after a lowcarbohydrate diet for several months. which are interpreted as superficial mucosal ulcers. I had only two patients who took longer than eight years until their disease calmed down. diarrhoea and abdominal pain subsided. med. where these unlucky people congregate and are told "There is no diet for ulcerative colitis or Crohn's Disease.
= ulcers and fibrin Absolute years 0 2 4 6 8 <8 number of patients 74 74 54 46 35 28 without findings . The most severe symptoms disappear first. . . Fig.org/7archives/lutz/lutz7. contact hemorrhaging last. One can see that the inflammatory process.4 Table 2: http://www. the many white blood cells in the mucosa. .. Simultaneous increase of percentages of symptom-free patients _____ = without findings. .scdiet. . disappear.= spontaneous hemorrhaging.Dr.. 17: Graphic depiction of the values of table 2. . = only contact hemorrhaging.54 76 85 91 93 contact hemorrhaging 32 26 11 9 6 4 spontaneous hemorrhaging 31 10 7 4 3 ulcers and fibrin 36 5 6 4 .html[24/1/2013 1:49:49 μμ] . when patients live on the diet for some time. Wolfgang Lutz: Dismantling a Myth: Chapter VII The same can be seen on fine-grained pictures of small pieces of mucosa removed during rectoscopy.. med.40 41 39 32 26 contact hemorrhaging 24 19 6 4 2 1 spontaneous hemorrhaging 23 7 4 2 1 ulcers and fibrin 26 4 3 2 .. .1 Percent years 0 2 4 6 8 <8 number of patients 100 100 100 100 100 100 without findings .
org/7archives/lutz/lutz7. Increase of rate of healing to over 90 percent. Every other one of my patients had undergone previous psychiatric treatment. 1980 I want to thank SELECTA for referring me to Dr. The commonly-recommended diets did not help at all. patience from side of the physician. They thought they were happily married. sufficient income. But one has to see the success of the diet in relation to its alternatives. Whoever believes he can get rid of his colitis in a few months is mistaken. nocturnal sweating. None was able to help me.html[24/1/2013 1:49:49 μμ] . by no means the feeling that something psychological had Influenced or even caused their disease. It requires cooperation and sacrifice from the side of the patient. I am totally convinced now that this illness is in no respect psychosomatic but purely a nutritional problem. Certainly this is not an easy form of therapy. Now." The second alternative is the removal of the entire colon and the creation of an artificial anus. swollen knee joints. At that time I had suffered from this disease for several years. sleeplessness and Cushing's symptoms. which I became acquainted with in November 1974. I had rectal ulcers and crypt abscesses. Due to the lack of success of conventional therapies it was commonly assumed that colitis must be a psychosomatic disorder. med. October 23. I felt I was stuck on a dead-end road. after two-and-a-half years of low-carbohydrate diet. had no problems. and time. I was allergic to drugs and experienced unpleasant side effects from cortisone. and for his book "Leben ohne Brot (life without bread). I only regret that most colitis patients will go on to be considered psychosomatically ill and will not be treated with the only effective therapy. These patients asked me how they had given the impression of psychological difficulties. Wolfgang Lutz in Salzburg and his methods of treating ulcerative colitis. Decline of hemorrhage and ulcers during two to eight years.scdiet. everything has changed. that is carbohydrate restriction. Certainly there are cases where this is finally the last http://www. edemas of the legs. Wolfgang Lutz: Dismantling a Myth: Chapter VII Progress of ulcerative colitis of 74 patients' on a low-carbohydrate diet. Criteria: rectoscopic findings. Psychotherapy For one there Is psychotherapy. Not only the colitis has healed but also the stomach problems have disappeared. just because doctors do not believe in it. The following letter of a doctor is characteristic for this situation.Dr.
Basically what Prof. This time the infection manifests itself in the form of shingles. others just have fever but everybody has had chicken-pox. Resides this one. represented by white blood cells and bone marrow cells. he will never get rid of the disease again. There is a good analogy to this: we all contract chicken-pox during childhood. med.scdiet. even after the resolution of an ulcerative colitis there can be a relapse. Wolfgang Lutz: Dismantling a Myth: Chapter VII possibility to cure a chronic colon illness.org/7archives/lutz/lutz7. knows what that means. namely that once a person has ulcerative colitis. Immune reactions are supposed to protect us from external enemies. Patients are usually capable to put up with anything their fate imposes on them. but his colon becomes more resistant and can cope better with the immune system's attacks. however. The immune person will never fall ill of scarlet fever again. a child will only contract them once and then never again. The first encounter with them causes scarlet fever. however. With increasing age the immunity against this pathogen wears off. Because the colon does not bleed or otherwise react to the attacks. Rut this is different under a low-carbohydrate diet. Some develop a typical rash.html[24/1/2013 1:49:49 μμ] . I did not have to refer one single patient of mine to surgery and I hope this will be so in the future. Two of my patients have had a relapse after ten years. As mentioned before. Let us consider the hemolytic streptococci which are dangerous germs." Demling meant this in a different context. still holds true: "Once colitis. so that we can be infected with it again. it also does not forget if it ever has been sensitized to the body's own organs. An Immune Disease This is because ulcerative colitis is distinctly an immune disease (*4957). Most other childhood diseases proceed similarly.Dr. but whoever has had an artificial anus. The recurrent disease was usually mild and lasted only a few weeks. his immune system will remain primed toward his colon. a specialist of gastrointestinal diseases. knows the germs. usually it passes without fever and is restricted to small areas of the skin. Sometimes this reinfection can be very extensive. After a while the immune system loses interest in such attacks. http://www. one after six. the immune system is less stimulated and its reactions are less intense. The immune system never forgets. In contrast to Crohn's Disease. I have lost one patient who was operated on too late because he absolutely refused to have an artificial anus. During the progression of the disease the organism develops immunity toward the pathogen. always colitis. From year to year these relapses become less probable and less severe. Now the immune system. said. and one after four. Ludwig DemIing. Once a person has colitis.
I have a whole file of thank-you letters from patients who had tried every possible therapy unsuccessfully and finally were left with surgery as the only option. Eventually we operated anyway.e. the development of colon cancer. the disease had resolved to that extent under the diet. also regarding her leukemia. med. before they came to me. Usually it takes ten to fifteen years for a cancer to develop. First one should try to wean the patient from cortisone since these artificial adrenal cortex preparations generally inhibit protein synthesis. Generally these patients have had their colitis since their youth. is missing.Dr.html[24/1/2013 1:49:49 μμ] . She was operated in 1980 and is well now. Between 1965 and 1981 I published five scientific http://www. that a patient with ulcerative colitis has to face. Certainly a patient with an undiagnosed lactose intolerance will have diarrhoea after drinking milk. Eventually one will be able to discontinue all medication. which splits the milk sugar molecules. Now his surgeon also uses a low-carbohydrate diet for colitis patients in his hospital.080 cases of Crohn's disease and ulcerative colitis alone in the Germanspeaking countries. one can temporarily take up the medication again. In these cases the enzyme lactase. just to come back to me with a relapse of his colitis. it was cancer. Postoperatively the patient started to eat everything again. But this has nothing to do with ulcerative colitis. Why are they not put on a low-carbohydrate diet? Why does not this method become established? I can only say that I have tried everything. Therefore milk sugar cannot be absorbed by the intestine and transferred to the blood. A mild relapse does not require medication. This was after more than ten years of the diet. That means they do not only suppress the production of immune cells and thereby subdue the symptoms. Whether or not patients who have been on a low-carbohydrate diet develop cancer. One woman simultaneously had leukemia. Wolfgang Lutz: Dismantling a Myth: Chapter VII Lactose Intolerance There have been claims that milk may be harmful for a patient with ulcerative colitis (*42). Medication The medication cannot be discontinued immediately since it takes a little while until the diet takes effect.org/7archives/lutz/lutz7. The second case was an older man. I have never observed anything of this kind. Certainly there are more than 50. We found a suspicious area in his colon but a biopsy taken at that site was negative. but also interfere with the healing of the mucosa. It reaches the colon in an undigested form and causes diarrhoea.scdiet. Among my first 100 patients I had two cases of cancer. hemorrhages recurred. a malignant blood disease. i. The surgeon told me later that he had not found any indication of ulcerative colitis in the colon. Should there be a relapse in the first few years. Another danger. (*49). After he had been well for over a decade. is malignant degeneration. and colon cancer. often cancer develops at multiple sites. I cannot say.
http://www. Wolfgang Lutz: Dismantling a Myth: Chapter VII papers in respectable medical journals. The well-known Swedish physician Axel Munthe a long time ago associated this kind of colitis with a whole variety of hysterical traits and thereby turned it into a fashionable disease. There Is no limit to the arrogance of orthodox medicine toward all outsider methods. homeopathy. diarrhoea. One would have clung to the hysterical symptoms and would have looked for another physiological substrate. toward acupuncture. although the hysterical connotation would not have convinced the upper class at that time. in my opinion is irresponsible. chiropractic. Caution is partially justified but none even had the courage to use these precise methods of this orthodox medicine to prove the outsiders wrong. that this is simply a carbohydrate-associated disease.scdiet. irritations. To ignore something that comes from outside. For this condition the term colitis or colica mucosa was coined. But surely it is not an isolated disease but rather an irritation of the colon which leads to an excessive stimulation of the mucous glands and the excretion of the mucus. However. Irritable Colon This entails complaints which the patient associates with his colon. Too bad.org/7archives/lutz/lutz7.Dr." I hope this will happen for the sake of the many tenthousands of colon patients who today can still "eat what they can tolerate". abdominal cramps. They are worried because the mucus looks like pieces of tissue and they believe they are excreting the actual mucosa. neural therapy. and the like. of all the letters the postal service handles. it has lost exactly these. med. The term "colitis" slowly became removed from the colon and became a disease of those social circles who were able to afford to have it. Some patients excrete with their stools large amounts of mucus. from a physician who "only" practices and does not teach at a university. This can simply be concluded from the fact that they resolve quickly on a low-carbohydrate diet. because they commonly pertain to bowel movements and are felt along the colon. I do not believe that. In my office I have posted a saying by Mark Twain: "A person with a new idea is a crank until the idea turns out to be right.html[24/1/2013 1:49:49 μμ] . the irritable colon actually exists as a mixture of all the conditions which have been discussed so far: constipation. I have written over 40 letters to editors of scientific journals. All these conditions are caused by an excessive intake of carbohydrates. we do not live during Axe Munthe's times. I contacted the societies for patients of Crohn's disease and ulcerative colitis and have not even received a reply.
however. But even the former have bloodnegative stools on a meatless diet so that it can be concluded that the blood-positive reaction previously obtained was due to the meat in their diet. even though they now are eating meat. 100 mL (1/10 L) of blood contains 14 to 16 grams of haemoglobin. It is therefore not surprising that the iron levels in the blood rise when the haemorrhages associated with colitis ulcerosa are stopped. med. of which 0. an important component of the red blood pigment haemoglobin. They even return to normal in patients who have never had haemorrhages and never suffered from colitis ulcerosa (*62). http://www. by means of the benzidine reaction) it turns out that on a normal diet some patients show a positive and some a negative reaction. Our approximately 5 litres of blood therefore contain about 2. The frequent intestinal haemorrhages in ulcerous colitis cause large losses of iron. What is the reason for this improvement in the blood iron on a lowcarbohydrate diet? Cessation of haemorrhage is certainly not the only reason because we have seen that the diet is also effective in cases where no bleeding occurred.org/7archives/lutz/lutz7. On a low-carbohydrate diet such patients lose their blood-positive stool. If the stools are chemically analysed for blood (e.34% is iron. the haemorrhages do not stop at once.Dr. but the blood iron values nevertheless begin to rise. Wolfgang Lutz: Dismantling a Myth: Chapter VII Iron and Calcium I have observed that the low iron levels in the blood of patients with colitis ulcerosa may return to normal an a low-carbohydrate diet.g.8 g of iron (an additional gram is present as tissue iron in enzymes and iron depots). In many cases.html[24/1/2013 1:49:49 μμ] .scdiet. The process of meat digestion must have improved in some way.
The iron contained in our food varies in quantity and absorbability. in which the mean value drops to 140 mu. whereas meat contains large amounts of iron. med. We can thus partially explain the rise in iron in the blood of irondeficient patients on a low-carbohydrate diet. The main effect of the carbohydrate restriction is seen in the first four weeks. a substance known as haeme. 18: The behaviour of (a) hypo. Whereas iron is normally completely dissolved out of the food in the intestines and traverses the intestinal mucosa in molecular form. The figures of "n =" show how many pations were investigated at each point.html[24/1/2013 1:49:49 μμ] . In in average a normal iron level is attained after six months.Dr. iron is present in meat in combination with the organic complex protoporphyrin. b: 38 cases with hypersiderosis approaching normal within 10 weeks. Further.scdiet. Thus the iron in meat is in this respect superior to that in vegetable http://www.and (b) hypersideroses on a low-carbohydrate diet (72 g) without other therapeutic measures. a fact that considerably facilitates its uptake by the body (*58-61). a: 38 cases with manifest hyposiderosis. it is absorbed as a complex in haeme. both in the blood remaining in the blood vessels as well as in myoglobin. an iron-containing pigment of the muscle cells. Vegetable matter is relatively low in iron and even this is not readily digestible. Wolfgang Lutz: Dismantling a Myth: Chapter VII Fig.org/7archives/lutz/lutz7.
Lack of iron therefore cannot have been the reason for the low iron levels in the blood. takes up the iron. It appears that carbohydrates are cause of both disorders and. c) Improvement of meat digestion in the intestine.scdiet. The low-carbohydrate diet must exert its effect at some other site (*62). The Bantus use only iron vessels for cooking and their diet therefore contains adequate quantities of iron. in this case a lowcarbohydrate diet. Cases of siderosis (too much iron in the blood) are even more common than sideropenia (too little iron) and are important since they may lead to deposition of iron in the tissue and thus to severe metabolic disturbances (haemochromatosis). as yet unrecognized. is involved. thus leading to increased uptake of iron. accompanied by large amounts of carbohydrate. rob us of our ability to keep our iron balance in order. Among my patients were cases that had already received large quantities of iron. 18 includes 38 cases in which an elevated iron level responded to restriction of carbohydrate intake. eating food from iron pots. med. can restore abnormal to normal from both directions strongly suggests a common causal effect. Fig. The Bantus could probably change this. The elevation of low iron levels in the blood which results from a lowcarbohydrate diet may be brought about in three ways: a) The food contains more iron (meat) b) This iron is more readily absorbed.Dr. Too Much Iron in the Blood (Hyper-siderosis) The same diet also has a beneficial effect on abnormally high iron levels (*62). A diseased intestine permits the loss of undigested food and thus of the iron in it. and the stools give a bloodnegative reaction. some by injection.org/7archives/lutz/lutz7. As will be seen the same holds for calcium. http://www. without any improvement having been achieved. Anyone.html[24/1/2013 1:49:49 μμ] . whereas the healthy intestine breaks down the food. either by giving up their iron pots or their carbohydrates. like the Bantus. Joking apart. in a manner as yet unknown to us. Wolfgang Lutz: Dismantling a Myth: Chapter VII foodstuffs. may suffer from siderosis. Although these three factors undoubtedly play am important role I believe that still another factor. the fact that one and the same measure.
Several of my patients had already tried treatment of various kinds in clinics or hospitals without any success. med. Low blood calcium levels are mainly seen in disorders of the thyroid or parathyroid glands. Wolfgang Lutz: Dismantling a Myth: Chapter VII An Example Provided by the Kung Iron deficiency disease serves as an example emphasizing that we were originally hunters and meat-eaters. The difference between the two groups.the so-called calcinosis factor or vitamin D. Recent reports on investigations made among the Knng. The general improvement in intestinal function on a low-carbohydrate diet is reflected not only in the normalization of the low iron levels in the blood but also of calcium levels. the situation is different and cannot be alleviated by administering parathyroid hormone -. and not until I put them on a low-carbohydrate diet did their blood calcium levels become normal. I have observed a number of such cases of idiopathic hypocalcaemia (for which no particular cause could be discovered) over a number of years.5 mg/dL. If the parathyroids. As long as we consume meat everything is all right. He came to me with the symptoms of tetany and blood calcium values of 6 mg/dL (instead of the normal 10. If we think back to the cases of low iron levels discussed above it is tempting to adopt the view that here. On a low-carbohydrate diet he recovered within a few months.000 years the Kung lived a nomadic existence in the same region. some of them have changed to a sedentary way of life and have taken on the nutritional habits of the neighbouring Bantus (but not their iron cooking pots). that our metabolism is geared to the preferential treatment of the iron from meat and has neglected the utilization of iron from vegetable matter. a primitive tribe inhabiting the Kalahari desert In South Africa. They are shown in Fig.which usually has the same effect --.Dr. however. The latter show signs of iron deficiency (as well as obesity and high blood pressure). but if we go against our nature and eat only vegetable matter we get into difficulties. In most cases it looks as if the ability of the Intestine to take up calcium from the food is impaired. the blood calcium values returned to normal although no medicinal calcium had been prescribed. conditions which are not found among the nomads (*63).org/7archives/lutz/lutz7. 19. are damaged or erroneously removed during a goitre operation the blood calcium level may drop radically and so-called tetany with muscular spasm may ensue. it is the intestine that is responsible for the disorder.scdiet. Although these troubles can often be circumvented by means of iron pills the solution is not an ideal one. My first case of this kind was a colleague suffering from chronic inflammation of the pancreas and the small intestine. The nomadic Kung live as they always have done on neat. is in the meat and carbohydrate content of their diet. three on each side. seen to confirm the ideas put forward above. of which we usually possess six. In my patients.html[24/1/2013 1:49:49 μμ] . In recent decades. too. however. just as between us and our forefathers. http://www. For 11. vegetables and nuts whereas those who have settled down live on cereals and soya milk.
Bile and Liver In 1960. 19: Abnormal low calcium levels normalize with carbohydrate restriction.Dr.scdiet. when I began to use a low-carbohydrate diet in treating patients with abdominal complaints. experience shows that gastritis does not heal without the addition of fat to the low-carbohydrate nutrition.org/7archives/lutz/lutz7. Among my successfully-treated gastrointestinal patients more and more cases have later turned out to have been suffering from gall bladder http://www.html[24/1/2013 1:49:49 μμ] . supposedly for slimming purposes. In fact. see later) the fat intake had been kept down. When a patient tells me (which is rarely the case) that the prescribed diet has brought no relief to his gastrointestinal troubles I look not only for chronic constipation but also try to discover whether. Wolfgang Lutz: Dismantling a Myth: Chapter VII Fig. and without fat it is impossible to prescribe a low-carbohydrate diet. It was an accepted fact that fat and roasted products were bad for them. med. (or on account of the cholesterol level. I excluded those with gall bladder troubles.
In spite of this they had responded well to the lowcarbohydrate diet and its accompanying additional fat. Wolfgang Lutz: Dismantling a Myth: Chapter VII disease as well.Dr. I gradually began to include gall-bladder patients in my dietary programme. given a low-carbohydrate diet. where he often ate nothing for days on end and then quite suddenly consumed large amounts of meat and fat. a process which is inevitably painful if gall stones are present since the latter cannot be expelled (otherwise they would no longer be inside the gall bladder). This involves contraction and the expulsion of its contents.org/7archives/lutz/lutz7. All patients who in the end had to have an operation despite a lowcarbohydrate diet. For us. suffices for the process of digestion. When to Operate Instead of recommending patients with a pathological X-ray diagnosis of the bile system to consider an operation in the immediate future. Post-Operative Complaints http://www. in which no contrast shadow appeared) had to he operated upon. Apparently a low-carbohydrate diet leads to an improvement in the situation along the entire length of the digestive tract. which filled with contrast agent during the X-ray examination and was capable of contraction at the appropriate stimulus. It should be borne in mind that the size of the gall bladder and the quantity of concentrated bile stored within it are adapted to a human being in his natural state.e. ought in fact to experience some symptoms as a result of eating fat. The function of the gall bladder is. and of the so-called liver bile secreted by the liver.scdiet. I cannot recall a single case in which a gall bladder already totally out of action (i. gall stones. med. so that a colic becomes less likely. This is why it is possible to live comfortably after operative removal of the gall bladder. had stones in a partially-functioning gall bladder. which is not usually difficult. I now prescribe a low-carbohydrate diet plus medication aimed at slightly subduing the intestinal flora. I endeavour to see the patients through the first couple of weeks. and thus also in the region of the bile ducts.g. It appears that. it is the contractile gall bladder with stones and not one totally tilled with stones and unable to contract and empty that requires operation. e. A patient with any kind of gall bladder complaint. the organ is in fact oversized and even the volume of bile at the disposal of a gall bladder containing stones. and if they feel an improvement then they no longer press for an operation. after all.html[24/1/2013 1:49:49 μμ] . to make available a large quantity of concentrated bile at the beginning of the digestive process. eating three times daily.
Inflammation of the Bile Ducts (Cholangitis) Since liver and gall bladder are to a large degree functionally involved with one another it is to he expected that the same success obtained by dietary treatment of patients with diseases of the gall bladder and bile ducts can also be achieved in diseases of the liver. however. then some other foodstuff has to be cut down.) in the smaller bile ducts within the liver. a well-known Viennese physician. highprotein diet for patients with cirrhosis of the liver and abdominal dropsy (*64). http://www. And at this point I am obliged to digress considerably. If a patient is given more protein. in order to maintain a normal glycogen content. In cases of a swollen liver resulting from an infection (by bacteria etc. was already of the opinion that sufferers from liver disease should not be put on a very low-fat diet even if they had jaundice. and heals when the enteritis itself is cured. An unhealthy liver tends to lose its glycogen and to store fat in its place. If a patient who has had an operation to alleviate symptoms of this kind or for gall bladder trouble tries to eat anything he likes. Such cases are by no means rare. During the Second World War Patek in the USA propagated a high-calorie. e. In the meantime. in which the liver cells themselves are inflamed. The infection of the bile ducts probably comes from the inflamed and infected small intestine. They have been termed the "post-cholecystectomy syndrome". and complaints of post-operative complications are frequent. Inflammation of the Liver (Hepatitis) It was to be expected that the situation would not turn out to be so simple in hepatitis (termed "infectious jaundice" in its acute stage).html[24/1/2013 1:49:49 μμ] . Alleviation is to be found solely in a restriction of dietary carbohydrates.org/7archives/lutz/lutz7. it has become clear that this strategy is not successfulI. from which was concluded years ago that patients with liver complaints require plenty of sugar and other carbohydrates but little fat. It is known that common enteritis often leads to colicky sensations in the upper right abdomen. the enteritis or inflammation of the bile ducts and thus the accompanying symptoms often persist. supplemented at first by the above-mentioned antibiotic measures.scdiet. Sometimes patients are not satisfied with the result of a gall bladder operation. diet and antibiotics in fact bring relief as readily as in the case of gall bladder disease. med. Wolfgang Lutz: Dismantling a Myth: Chapter VII A further point seems to me to be of importance.Dr. Professor Eppinger. The Patek diet nevertheless contained large amounts of carbohydrate so that in the long run it was not successful.g. In contrast to a chronic hepatitis. We know that animals and humans possess a small depot of carbohydrate in the form of glycogen in the liver and musculature. the enlarged liver is remarkably sensitive to pressure.
the more so since drastic nutritional measures are not considered necessary in routine cases. and for this reason it has been suggested that the principle of treating liver diseases with a low-fat. which has a poor prognosis due to its tendency to develop into cirrhosis and for which all measures so far tried. the serum bilirubin (bile pigment in blood) and the electrophoretic separation of the serum into its individual fractions (serum electrophoresis). Statistics from infectious jaundice have revealed that a high-protein. whilst electrophoresis gives a measure of the individual protein fractions.html[24/1/2013 1:49:49 μμ] .org/7archives/lutz/lutz7. dietary therapy seems to warrant closer attention. As a rule acute hepatitis almost always subsides. and above all of the gamma globulins. so that it is not easy to judge the advantages and disadvantages of a particular type of diet. at least for a time. however. high-fat. In the chronic stage. restriction of carbohydrates in favour of more protein and fat is also beneficial to a diseased liver. in this case the liver. The GOT which is found in the liver cells and their energy sources. low-fat type of nutrition (*65-72). The LAP (like alkaline phosphatase) indicates the degree of involvement of the bile capillaries. the mitochondria. http://www. high-carbohydrate diet should be discontinued (*69-72). perhaps more importance would be attached to the treatment of the latter. contrary to what has previously been accepted. If it were to be borne in mind. I have personal access to many patients with pathological liver tests. and additional fat has to be permitted. Wolfgang Lutz: Dismantling a Myth: Chapter VII carbohydrates. A variety of observations suggest that. The proportion of the blood proteins accounted for by the gamma globulins shows to what degree the organism has so far mobilized immune reactions against its own organs. The tests in regular use are the so-called GOT (glutamate-oxalatetransaminase). med. the LAP (leucine-arylamidase). particularly all types of medication.Dr. that chronic hepatitis often develops from an incompletely healed acute condition. high-fat diet brings somewhat better and in no case worse results than a high-carbohydrate. have proved to be fairly ineffective. Fat is not Harmful The question as to whether a low-carbohydrate.scdiet. high-protein diet would be more beneficial than the general practice of prescribing a low-fat nutrition has recently been taken up again: at least a fatty liver responds favourably. serves as an indicator of the extent of the inflammation of the liver cells (hepatitis). We have already encountered these immune reactions on several occasions and in view of the size of the organ involved it is not difficult to conceive of their playing a particularly important role in liver diseases.
I already have 190 cases in which the GOT has been consistently recorded. a parameter indicative of the state of the liver.html[24/1/2013 1:49:49 μμ] . a low-carbohydrate diet is successful. Even in severe cases (C) the results are impressive. too. but the laboratory values also show an improvement. Following jaundice the two abovementioned liver tests often remain positive and.Dr. The slighter cases (GOT up to 20 mU) returned to normal within two weeks. in addition. but even in more severe cases a response is generally seen although this understandably requires longer. 20: The behaviour of GOT (ASAT). 20). the blood frequently contains elevated quantities of bile pigments. The speed with which a normal value is obtained on a low-carbohydrate diet depends upon the initial value (A.B. "n" = number of cases investigated at each date. so that the results are statistically significant (Fig. In 23 of the cases LAP was also determined.org/7archives/lutz/lutz7. 21 shows.C). This is a disorder which is usually considered to be very resistant and for which so far no effective mode of treatment has been devised. med. http://www. Wolfgang Lutz: Dismantling a Myth: Chapter VII Enzyme Reactions Improve If a patient with a diseased liver is put onto a low-carbohydrate diet not only does he soon begin to feel better. As can be seen from Figure 22 however. as Fig. These values. returned rapidly to normal. Fig.scdiet.
the development of a chronic inflammatory process in the liver. Chronic hepatitis is the classical example of an auto-aggressive disease. 21: The LAP level (leucine-arylamidase) is characteristic of the function of the bile capillaries and the unimpeded flow of bile. is due to the production of antibodies by the organism to its own liver.scdiet. Defence reactions of this kind are being attributed an http://www. the body reacts to one of its own parts as if they were foreign and tries to reject it.html[24/1/2013 1:49:49 μμ] .Dr. or --. A similar situation is encountered in heart transplants.org/7archives/lutz/lutz7. Just as has been observed in diseases of other organs. Even elevated LAP values rapidly returned to normal on a low-carbohydrate diet. Wolfgang Lutz: Dismantling a Myth: Chapter VII Fig. thyroid and gastrointestinal tract. med. Even if electrophoresis does not indicate that the gamma fraction of the blood proteins Is higher than normal it can be assumed that the failure of Infectious jaundice to heal.in its absence --. particularly the heart.
html[24/1/2013 1:49:49 μμ] . Normally the gamma globulins account for 12 to 15% of all proteins circulating in the blood. Wolfgang Lutz: Dismantling a Myth: Chapter VII ever-increasing role in the normal theory of disease. however. The larger the diseased organ the more immunoglobulin is formed in autoimmunological disease. In autoimmune diseases aimed at smaller organs no changes in these values can be detected.Dr. They assume even greater significance In my experience with a changeover to a lowcarbohydrate diet. med. http://www.scdiet. Fig. the reasons for which will be gone into in a later chapter. shows that even high values for bile pigments in the blood can be lowered. and if this organ is of the size of the liver a rise in the gamma fraction becomes noticeable. 22: Chronically-elevated serum bilirubin levels are generally considered to be incurable.org/7archives/lutz/lutz7. Observation of such patients on a low-carbohydrate diet. "n" = number of cases investigated at each date.
23: http://www.html[24/1/2013 1:49:49 μμ] .Dr. If the GOT improves on the diet although the gamma globulins remain elevated this means that the vulnerability of the liver cells. There seems only a non-specific increase in all fractions and thus also of the total serum proteins. But there are limits of course. decreases despite the fact that the organism continues to bombard them with antibodies. 23). just like the intestinal mucosa. bones and cartilage of the joints. but no change was noticeable on a low-carbohydrate diet. Wolfgang Lutz: Dismantling a Myth: Chapter VII Cirrhosis of the Liver On an average we observed the gamma globulins to be 40% above the normal values (21% instead of 15% of the total blood proteins) in our liver patients. since this enhances the formation of protein of all kinds including the immunoglobulins (Fig.scdiet. the number dying off in unit time. teeth. and continued to rise on a low-carbohydrate diet.e. heart.org/7archives/lutz/lutz7. i. They become more resistant to auto-aggression. med. In three cases of chronic hepatitis and cirrhosis of the liver the gamma globulins exceeded 30% when I began treatment. Fig. skin.
After a few weeks to months the initial improvement gave way to a deterioration in the already-existent ascites so that the low-carbohydrate diet had to be discontinued or at least supplemented. This is an example of a detrimental effect not planned by nature. suffers most. In heart cases with primary weakness of the right chamber and chronic obstruction of the liver. These patients often have a swollen and coarse liver. Alcohol is high in calories (7 calorie/gram) and contains almost as much energy per unit weight as fat (9 calories/gram). although this means relinquishing the hope of final success. aggravation of the ascites is common. Heavy drinkers therefore have little appetite.org/7archives/lutz/lutz7. In such cases I now prefer not to try the diet at all. and thus protein intake.Dr. Wolfgang Lutz: Dismantling a Myth: Chapter VII Gammaglobulin as percent of total protein in a case of liver cirrhosis with ascites. The Drinker's Diet In the introduction we mentioned that a low-carbohydrate diet can to a certain extent replace the restriction of alcohol in alcoholics. End of Chapter 7 http://www. about which we shall have more to say later. Such patients therefore do not have such a good prognosis as other heart cases on a low-carbohydrate diet.html[24/1/2013 1:49:49 μμ] . If such patients are deprived of carbohydrates they still receive enough calories In their alcohol. and frequently show pathological liver tests. often with elevated GOT and LAP and sometimes accompanied by a marked increase in blood fat values. essential for building-up and maintaining the body.scdiet. The rise on a lowcarbohydrate diet reflects the general anabolic effect of the diet and indicates the formation of antibodies. In my opinion the 'drinking man's diet' is one of the more important lay contributions to medical knowledge (*73). med. the desire for food returns and protein loss is made up.
Ass. A. Elliot.L.: Clin.T.H. 37. H.scdiet. R.: Lancet 1959.. A. J. Utrecht. Kh. Herkovic. Wschr. M. B. 149 (1959) 58... H. G.. W. Lancet 1960.L..A. Morrison: Am J. Lancet 1960. E. 156 (1962) 572.E. 69 (1961) 143. Tracy: Gut 5 (1964) 115. Biol. Thompson. T. Wschr.. H.D.J. E. et al. T. Med. Med. 142 (1955) 709. 9 (1960) 753. M. H. French. Uppsala. 5) Ross.K. W. Exper.M. D. Wschr.: Klin..A. F. 25 (1958) 374. M.M. 26) Priest. John Wright & Sons. Erpenbeck: Dtsch. 30) Feyrter. 12) Osborne. 22) Robinson.N. II.M. 5 (1964) 1903. et al. M. N. J.G. 27) Chears. W. H.: Proc.: Der Zucker als pathogenetischer Faktor. M. Med. 13) Gregory. 271 (1964) 769. J.A. 7) Zollinger.. Ch. Coronary Thrombosis and the Saccarine Disease. Wschr. 96 (1957) 518 23) Salter. 32) Dollinger. Med... Physiol. Pernokas. 24 (1953) 71. Rev. L. I. Int..H. B.html[24/1/2013 1:49:49 μμ] . 122 (1925) 271.H. Smeller: Lancet 1955. R. 42 (1953) 34. 11) Greider. 1054. Brown: New Engl. 40 (1962) 1085. (1962) 572. Tracy.A. R. B. Kearns.. R... 25) Martini. R. R. 24) Verner..M.W. W.J.J. F. H.. A.A.. Bad Homburg 1962. M. Eyrin.. B.Dr. Goosard.A. J. Mathews. 1966.: Ann. M. M. J. C. med. R.. Schwabe & Co. 10) Zollinger. L.: Klin.M.C.V. H. 886 18) Gregory. R. Weijers. I. C. 2) Cleave. M.C. 1950. R.P. Rohner: Schweiz.: Med J. Soc. 86 (1962) 847. R. J.. L. P. 33) Aron. D. Blackbourne: Arch.P. I. R.. Haug..M.A. 31) Dengler. Med. J.E.A. W. R. 28) Bergoz. J.. M. G. J. 91 (1966) 634. Med. Ellison: Ann. Med. Liebowitz: New Engl. 9) Zollinger.M. Wakin. Tracy: Gut. Surg. E. A. D. J. Shamolan. Med. Bristol. 22 (1962) 179.N.. Forell. C. Grant: JAMA 190 (1964) 181. H. R. . G. 8) Zollinger. R. P.M.D.org/7archives/lutz/lutz7.H. McPhedran: Canad. A. 15) Gregory. C. 37 (1959) 401. 17) Billington. 1145. 21) Aylett. 29) Zenker. Surg. R. W. 137. 3) Dicke. R. Strohmeyer. Fekete: Presse Med. 4) Dicke. J. Gusek: Dtsch Med. F. F. Wschr. J. C. O..W. 16) Edit.O. 35) Alarcon-Segovia. Sammons. French. II. Zollinger: JAMA 186 (1963) 566. 6) Kivel. J. 1087.: Thesis Univ. Fraser. Hutcheson. Med. Campbell: Diabetes. Green. 268 (1963) 465.C. G. Leidlaw: Metabolism. R. K. H.: Gastroenterol. 19) Gregory. 11. 120 (1967) 575.H.K. D. Leger. http://www. Alexander: Lancet 1957. Sircus. J.. a. van de Kamer: Acta paediat. 14) Dragstedt. 1151.J. M. Ellison: Gastroenterology. G. R. Vlg. Ratmer. 97 (1967) 413. J. Lancet 1957. J. 34) Anderson. (1959) 1245. Tracy: J. Patterson: Am. Wolfgang Lutz: Dismantling a Myth: Chapter VII References (Chapter VII) 1) Bruker.M. J. Sci. 20) Edmeads. 89 (1964) 313. 29 (1960) 529.
P. H. Med.. C.. Fortbildung.: Aetiologie des Morbus Crohn. Med. 44) Lutz. Wild: Die Höhe der Nährstoff Cholesterinund ballaststoffzufuhr unter kohlenhydratarmer Diät bei freir Wahl der Fett. 107 (1982) 71. und Nahrungsfette. 45) Lutz. 38) Thornton. Wschr. 42) Fahrländer. Klin.. I. Münch. Cambridge Mass..A. L. 54) Knick. J. 16/17 (1976) 221. J.P. Bericht uber 40 Fälle. Bad Neuenahr (1965). S. Aktuelle Ernährungsmedizin 4 (1979) 155. Wschr. T.. 16 (1965) 464.. Foy: Human absorption of hemoglobin iron. 57) Gebbers. R. 122 (1925) 271. Med.: Brit. Durham: JAMA.: Konstiution und Blähsucht. Otte: Zur Immunpathogenese der Colitis ulcerosa.und Hypersiderosen unter kohlenhydratarmer Diät. B.: Brit. DeVore. J. 47) Lutz. Gastroenterology 53 (1967) 5. Health 36 (1946) 1278. Jr. B. 53) Klavins. L. 41) Guthy. Med. W. M. B. Hemat. 36) Lampert. ärztl. Nutr. Diab. N. S. Grayson: Brit.. 58) Callender. 37) Crohn. 66) Gertzen. Vol. B. 64) Patek. W. Hippokrates-verlag. H. et al.. Gastroent. R. Ges.: Hypo. D. A. Med. 37 (1962) 497. 60) Conrad. H.. (1961) 160. Wschr. Moeller: Amer.F. Brit. 36 (1964) 485.. F. I (1950) 1166. J. Characteristics of the Pre-Illness Diet. Sölwell: Absorption of hemoglobin iron in man. R. 18 (1968) 615.eine prospektive Studie. 51) Anderson. New Jersey Rev. 183 (1963) 547. C. after Science.Dr. Clin. Wschr. Darm 12.: Die Behandlung der Colitis ulcerosa durch Kohlenhydratebeschränkung. E. Med. B. J. K. Clin. 43) Lutz. Wien. Nr.. Brit. Amer..D. Press. M. Harris: Lancet 250 (1964) 881. Kebleu: Hunter-Gatherers: Harvard Univ. M. 40) Guthy. 55) Koffler. Invest. 20 (1941) 481.: Erste Tagg. V. J.: Kohlenhydratarme Diät bei Colitis ulcerosa. et al. Med.und Proteinzufuhr. A. Layrisse. L. I (1961) 154. Wschr. J. Med. J. H. Mallett. Med. C. 60 (1965) 1. (1965) 516. J. 46) Lutz. Lutz. J. Med. Wschr.C.html[24/1/2013 1:49:49 μμ] .: Am J. 52) Dick. Post: J. Publ. Med. L. R. 39) Brandes. D. J . IntervallBehandlung in der Remissionsphase des Morbus Crohn . 28 (1979) 953. W. Rothmann. 185 (1974) 932. M. 3 (1957) 186. Wolfgang Lutz: Dismantling a Myth: Chapter VII Scudamore: Am. B.: Zuckerfreie Diät als Langzeit. I.. 108 (1983) 1229. J. W. C. J. Garlock: Amer. G. Leber. Williams. J.J. Wschr. A.scdiet. 102 (1977) 400. A. Smith: Absorption of hemoglobin iron.: Wien. 48) Kasper. 59) Hallberg. Eds. W.. Walker. Dtsch.. Finch: Nutritive value of food iron. Minkovitz. Benjamin. Oppenheimer: JAMA 99 (1932) 1323. 65) Hoagland. H.: Morbus Crohn. Polok. Scand. W.: Monatskurse f. L. Ginzburg. Dtsch. Shalev: Dtsch. Path. E. 50) Rieder.D. 62) Lutz. M.: Med.. Clark. 56) Müller-Wieland. R. J...org/7archives/lutz/lutz7. http://www. 67) Wilson. H. Coloprocology 3 (1981) 349. I. O. Dtsch Med. R.bzw. Stuttgart 1943. B. S. 63) Lee. W. J.. M. 181 (1965) 335. E. A. 49) Truelove. Marquardt & Cie. 99 (1974) 2141. W. Med. 25/26 (1967) 660. 41 (1962) 733.. Wien. E. med.: Kohlenhydratarme Diät bei Colitis ulcerosa und Morbus Crohn. A. Acta.: Diet and Crohn¹s Disease. 61) Hussain. Magen.C. Dtsch. Wschr. Vortrag. Med. J. 365 ( 1979) 74. J. 6 (1982) 225.W.
Kanzler: Dtsch. Inn. In the meanwhile.scdiet. Service to Danish readers: Danish translation of certain difficult words and medical terms The English version of the book is unfortunately presently out of stock. Oette. translated by Beatrice IdrisDuncan and Joy Wieser. 70) Phlippen. R. G. G. you can order the German version of the book. 72) Phlippen. Oette: Verh. A new print is on its way. All rights reserved.Dr. West Germany Original title "Leben ohne Brot". Wolfgang Lutz: Dismantling a Myth: Chapter VII 68) Chalmers. 74 (1968) 239. T. San Francisco 1965.html[24/1/2013 1:49:49 μμ] . Can.: J. Magen.13th edition (1995). H. Williams: The drinking man¹s diet. Med. E.: Med. Dtsch. Trotz. 69) Knick. Ottenjann. Invest. R. C.) (1974) 1916. K. [price: DM 44. med. Georghiu: Leber.. 16 (1971) 298. Ges. Th.. R. R. 34 (1955) 1163. J. Welt 25 (N. Wolfgang Lutz: http://www. B. KG Planegg V. Muenchen.org/7archives/lutz/lutz7. K. Ildar Idris GmbH & Co. Med. "Leben Ohne Brot" . Darm 1 (1971) 146.. et al.F.. 73) Jameson. 254 pages. Wschr.Immobilien & Werbe GmbH Fachagentur fur Medizin Irminfriedstrasse 31 D-82166 Graefelfing Germany Tel: (+49) 89 85 42 088 Fax: (+49) 89 85 42 766 Other books by Dr. Gruner.00] at: Informed . ISBN 3-921500-24-9 © 1986 by Selecta-Verlag Dr. Cameron & Co.. 71) Phlippen.
(1988).scdiet. Canada. July 1997 http://www. DM 6.html[24/1/2013 1:49:49 μμ] . was wirklich hilft" . 96 pages. med. What's the Solution? ") .dk]. June-July 1997 Input and "massaging" of references: Barbara Mills. downloaded.to be ordered at: Socio-medico Verlag GmbH P.July 1997 This page has had has had visitors since July 4th.80] ("Sick Stomach.org/7archives/lutz/lutz7. recording.1st edition (1995). May-July 1997 Editing and "massaging" of scanned copy: Dietmar [dhmx@vcn. without permission in writing from the publisher.O.80] . including photocopying. 112 pages. [price: DM 29. We remind you of the copyright: No part of this text may be distributed.to be ordered at: Informed (address above) "Cholesterin und tierishe Fette. kranker Darm. or by any information storage and retrieval system. HTML translation. Canada. eine Neubewertung" . Sick Gut. reproduced in any form or by any means electronic or mechanical. This excerpt was scanned from the original book by Rolf. and web-siting: Mik [mik@inform-bbs. Did you read the introductions of the book? (Page 2-16) On-line excerpt production: May . Denmark.Dr.Box 1311 D-82155 Graefelfing Germany Tel: (+49) 89 85 20 46-47 Fax: (+49) 89 85 41 599 Postage is not included in the prices mentioned above. 1997.00 for postage (within Europe). central coordination.ca]. [price: DM 22.bc. May 1997 Handling. Denmark. Expect app. Wolfgang Lutz: Dismantling a Myth: Chapter VII "Kranker Magen.
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