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HEALTH COMMITTEES https://app.box.

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1.BHORE(1946)-
Integration of preventive & curative services,
Primary health Centre- PHC per 40,000 population,
3 million plan, Social physician-3 month training.

2.MUDALIAR- Health survey and planning committee,
All India Health Services.

3.CHADAH- Multipurpose Health Worker(1 per 10,000),
Vigilance in malaria programmes,
Combined Malaria & family planning.

4.MUKERJI 1st- Separate staff for family planning,
5.MUKERJI 2nd- Basic health services,
To examine National Health Programmes

6.JUNGALWALLA- Integration of health services,
Seniority,No private practice,Equal pay for equal work,
Importance of extra qualifications.

7.KARTAR-
Multi purpose wokers and Female health worker(which replaced ANMs),
1 PHC per 50,000 population,divided into 16 subcentres.

8.SHRIVASTAV-
Referral services complex,
“ROME scheme”-Reorientation of medical education,
Rural health services,
Two cadres of health workers-MHW,HA.

9.KRISHNAN- Urban health.

10.BAJAJ- Health manpower.
HUNTERS TONGUE/MOELLERS TONGUE------ PERNICIOUS ANAEMIA

BALD TONGUEOF SANDWITH-----------NIACIN DEFICIENCY(PELLAGRA)

MAGENTA OR BEEFY RED TONGUE-----------RIBOFLAVIN DEFICIENCY

STRAW BERRY(EARLY)AND RASBERRY(LATE)------SCARLET FEVER

SCROTAL TONGUE-----FISSUREE TONGUE

BLACK HAIRY TONGUE------PROLONGED ORAL USE OF ANTIBIOTICS.

CEREBRIFORM TONGUE-----A FEATURE OF PEMPHIGUS VEGETANS.

List of Monoclonal Antobodies
Author: aminhars, Posted on Thursday, December 27 @ 02:27:53 IST by RxPG
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• Abciximab ReoPro 1994 chimeric inhibition of glycoprotein IIb/IIIa Cardiovascular disease

• Infliximab Remicade 1998 chimeric I inhibition of TNF-a Inflammatory diseases Adalimumab Humira 2002 human inhibition of TNF-a signalling
Inflammatory diseases

• Natalizumab Tysabri 2006 humanized T cell VLA4 receptor Inflammatory diseases . (esp autoimmune-related multiple sclerosis therapy)

• Omalizumab Xolair 2004 humanized Ig E Inflammatory diseases . (mainly allergy-related asthma therapy)

• Eculizumab Soliris 2007 humanized complement C5 Inflammatory diseases . (PNH)

• Efalizumab Raptiva 2002 humanized CD11a Inflammatory diseases . (psoriasis)

• Alemtuzumab Campath 2001 humanized CD52 CLL Gemtuzumab Mylotarg 2000 humanized CD33 AML . (with calicheamicin)

• Ri - tuximab Rituxan, Mabthera 1997 chimeric CD20 NHL Ibritumomab tiuxetan Zevalin 2002murine CD20 NHL (with yttrium-90 or indium-111) Tostumomab Bexxar
2003 murine CD20 NHL

• Daclizumab Zenapax 1997 humanized IL-2 receptor Transplant rejection Basiliximab Simulect 1998 chimeric IL-2 receptor Transplant rejection MuromonabOrthoclone
OKT3 1986 murine CD3 Receptor Transplant rejection

• Bevaciz umab Avastin 2004 humanized vascular endothelial growth factor Colorectal cancer Panitumumab Vectibix 2006human epidermal growth factor receptor
Colorectal cancer Cetuximab Erbitux 2004 chimeric epidermal growth factor receptor Colorectal cancer

• Ranibizumab Lucentis 2006 humanized vascular endothelial growth factor Macular degeneration

• Palivizumab Synagis 1998 humanized epitope of F protein of RSV Viral infection (espRSV)

• Trastuzumab Herceptin 1998 humanized ErbB2 Breast cancer
-mab suffix monoclonal antibody.

if immediately before mab----u--its humane derived
. ---mo--its murine
. ----i----its chimeric.

High yield facts in Mycology
Author: drpsg, Posted on Tuesday, July 13 @ 17:26:29 IST by RxPG
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1) Vascular invasion is characteristic of: mucormycosis, aspergillus

2) Inhalation of spores of which fungus is commonest: aspergillus

3) Only pathogenic yeast in man: C. neoformans

4) Sclerotic bodies with septate hyphae characteristic of: blastomycosis

5) Asteroid bodies are a characteristic feature of: sporothrix

6) Fungal infection is characterised by its capability to disseminate via lymphatics: sporothrix

7) Sporulation occurs in which phase: phase of decline

8) Prominent feature of paracoccidiomycosis: ulcerative granuloma of buccal and nasal mucosa

9) Characteristic feature of coccidiomycosis: spherule formation

10) Fungal infection which causes arthralgia / valley fever / desert rheumatism:
Coccidiomycosis

11) Thick walled barrelled shaped spores: arthrospores characteristic of coccidiodes immitis

12) Fungus which is urease positive: C. neoformans

13) Copper pennies appearance in tissue: paracoccidiodes

14) Most characteristic clinical feature of all cases of paracoccidiodomycosis: lymph node enlargement


Named fevers in microbiology
Author: psr4z4, Posted on Wednesday, January 21 @ 00:15:09 IST by RxPG
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Pontiac fever---Legionella pneumophila

Shanghai fever---Pseudomonas

Brazilian purpuric fever---Hemophilus aegyptius
(pink eye)

Havernhill fever---Streptobacillus monoliformis

Oroya fever---Bartonella bacilliformis
(Carrion's d/s)

Q fever---Coxiella burnetti

Colorado tick fever---Orbivirus

Trench fever---Rochalimaea quintana
(five day fever)

LIST OF FUNDOSCOPY FINDINGS
******************************

1) CHERRY RED SPOTS :
--------------------
- TAY SACHS DISEASE GM2 GANGLIOSIOSIS TYPE 1
-NIEMANN PICK
-CRAO
-COMOTIO RETINAE( BERLINS OEDEMA)
-FABERS LIPO GRANULOMATOSIS
-METACHROMATIC LEUCODYSTROPHY
-GM1 GANGLIDOSIS INFANTILE FORM
-SANDHOFF DISEASE
-SIALIDIOSIS

2) ROTHS SPOTS
-----------------

-CANDIDIAL CHORIORETINITIS
-SABE
-LEUKEMIC DEPOSITS
-ANAEMIC RETINOPATHY
-AIDS

3)GOLDEN RAIN APPEARENCE
-------------------------

-SYNCHYSIS SCINTILLANS

4)DOME OF CHAMPAGNE CORK APP..
-----------------------------

-PAPILLOEDEMA

5)SPLASHED TOMATO APP..
--------------------------

-ISCHAEMIC CRVO

6) CIRCULAR RING REFLEX
---------------------------

- CENTRAL SEROUS RETINOPPATHY

7) HONEY COMB APP...
----------------------

- CME

8) TOMATO KETCH UP APP...
--------------------------

- STURGE WEBER SYNDROME

9) CANDLE WAX APP..
----------------------

- SARCOIDOSIS

10) SALT PEPPER APP..
----------------------



-1) cong. rubella
2) cong. syphilis
3) early stages of leber's amaurosis
4) maculo cerebral familial disease( batten mayou disease)
5) ? cmv retinitis.


Bilateral

Miosis – pupillary constriction Iridocyclitis, miotic eye drops (pilocarpine)

Mydriasis – pupillary dilation Iridocyclitis, mydriatic or cycloplegic drops (atropine); midbrain (reflex arc) lesions or hypoxia; oculomotor CNIII damage; acute angle
glaucoma

Failure to respond (constrict) with light stimulus Iridocyclitis; corneal or lens opacity; retinal degeneration; optic nerve CNII destruction; midbrain synapses involving
afferent pupillary fibers or oculomotor nerve; impairment of efferent fibers that innervate sphincter pupillae muscle

Argyll Robertson pupil Bilateral, miotic, irregularly shaped pupils that fail to constrict with light but retain constriction with convergence; pupils may or may not be
equal in size; commonly caused by neurosyphilis or lesions in midbrain


Unilateral

Anisocoria – unequal pupils Congenital or caused by local eye medications, amblyopia or unilateral sympathetic or parasympathetic papillary pathway destruction

Iris constrictive response Acute uveitis is frequently unilateral; constriction of pupil accompanied by pain and cicumcorneal flush

Oculomotor nerve damage Pupil dilated and fixed; eye deviated laterally and downward; ptosis

Adie pupil (tonic pupil) Affected pupil dilated and reacts slowly or fails to react to light; responds to convergence; caused by impairment of postganglionic
parasympathetic innervation to sphincter pupillae muscle or ciliary malfunction; often accompanied by diminished tendon reflexes (diabetes or alcoholism)


CHROMOSOME 1
Alzheimer disease, type 4
Gaucher disease
Prostate cancer
Glaucoma
Rh blood type
TSH, beta chain
Amylase
Histone proteins

CHROMOSOME 2
Antibody, light chain
Myosin, light chain
Glucagon
Mismatch repair 2 (hereditary
nonpolyposis colon cancer, type 1, HNPCC)
Waardenberg syndrome

CHROMOSOME 3
Rhodopsin (retinitis pigmentosa, partial color blind)
Mismatch repair 1
Colon cancer,
nonpolyposis, type 2
Small cell lung carcinoma
Von Hippel-Lindau syndrome

CHROMOSOME 4
Alcohol dehydrogenase
Red hair color
Achondroplasia (dwarfism)
Ellis-van Creveld syndrome
Huntington disease

CHROMOSOME 5
Diastrophic dysplasia
Steroid 5-alpha-reductase-1
Achondroplasia (dwarfism)

CHROMOSOME 6
MHC (Major Histocompatibility Complex)
HCG, FHS, LH, TSH, alpha chain
Beta tubulin
Juvenile onset diabetes
Spinocerebellar atrophy

CHROMOSOME 7
Collagen 1, alpha 2 (Osteogenesis imperfecta)
Trypsin
Blue cone pigment (partial color blind)
Cystic fibrosis
Obesity

CHROMOSOME 8
Burkitt lymphoma
Werner syndrome

CHROMOSOME 9
ABO blood group
Ultraviolet repair (xeroderma pigmentosum & skin cancer prone)
Malignant melanoma
Tuberous sclerosis

CHROMOSOME 10
Hexokinase (hemolytic anemia)
Gyrate atrophy of the choroid and retina
Multiple endocrine neoplasia

CHROMOSOME 11
Hemoglobin, beta chain (sickle cell anemia, thalessemia)
Insulin (mutation NOT usual cause for diabetes)
Parathyroid hormone
Catalase
FHS, beta chain
Tyrosinase (Albinism)
PAX6, Aniridia
Ataxia telangiectasia
Harvey ras oncogene
Long QT syndrome

CHROMOSOME 12
Tirose phosphate isomerase
Keratin
Phenylketonuria (PKU)
Zellweger syndrome

CHROMOSOME 13
Breast Cancer
Retinoblastoma
Wilson Disease

CHROMOSOME 14
Ribosomal RNA
tRNA proline, leucine, threonine
Antibody, heavy chain
Alzheimer disease, type 3

CHROMOSOME 15
Prader-Willi Syndrome,
Angleman Syndrome
Tay-Sachs (N-acetyl-hexosaminidase)
Marfan syndrome (fibrillin)

CHROMOSOME 16
Hemoglobin, alpha chain
Chymotrypsin
Polycystic kidney disease

CHROMOSOME 17
p53 Tumor Suppressor Protein
Charcot -Marie-Tooth-Syndrome
Myosin, heavy chain
Neurofibromatosis
Collagen 1, alpha 1 (Osteogenesis imperfecta)
Growth hormone - Dwarfism
Glucose transporter
Familial breast & ovarian cancer, type 1

CHROMOSOME 18
Niemann Pick Disease
Pancreatic cancer
Suppressor of pancreatic carcinoma
?Tourette Syndrome
(may not be this chromosome)

CHROMOSOME 19
Familial hyperchlolesteralemia (LDL receptors)
Brown hair color
Blue/green eye color
LCG, beta chain
LH, beta chain (Polio virus sensitivity)
Apolipoprotein E
Myotonic dystrophy

CHROMOSOME 20
Severe combined immunodeficiencydisease
(SCID)

CHROMOSOME 21
Ribosomal RNA
Amyotrophic lateral sclerosis

CHROMOSOME 22
Ribosomal RNA
Antibody, light chain
Myglobin
DiGeorge syndrome
Neurofibromatosis, type 2

CHROMOSOME X
Red/green color blindness
Hemophilia A
Pseudoautosomal region X
Interleukin 2 receptor gamma chain (“boy in the bubble”)
Adrenoleukodystrophy (ALD-“Lorenzo’s Oil”)
Duchenne muscular dystrophy -
Becker muscular dystrophy (dystrophin)
Menkes syndrome
Fragile X (X-linked mental retardation)

CHROMOSOME Y
Testis-determining factor

Contraindications to Use of Antimalarials

* Allergy to 4-aminoquinoline derivatives
* Retinopathy or visual field deterioration

Contraindications to Use of Anti-Tumor Necrosis Factor (Anti-TNF) Agents

* Sepsis or clinically manifest infections and/or abscesses
* Past medical history of hypersensitivity to infliximab or other murine proteins

Contraindications to Use of Azathioprine

* Known neoplastic disease

Contraindications to Use of Cyclophosphamide

* Pregnancy
* Chronic or active infection
* Liver disease
* History of neoplasia
* Renal failure is a relative contraindication that requires adjustment of dosage.

Contraindications to Use of Cyclosporin A

* Coexisting cancer (except non-melanoma skin cancer)
* Uncontrolled hypertension
* Renal dysfunction
* Uncontrolled infections
* Primary and secondary immunodeficiency

Contraindications to Use of D-penicillamine

* Kidney disease
* Blood disorders (e.g., leukopenia, thrombocytopenia)

Contraindications to Use of Leflunomide

* Serious immunodeficiency
* Dysplasias
* Uncontrolled infection (due to the theoretical possibility of immunosuppression)
* Moderate or severe renal failure (there is no experience in this group of patients)
* Liver function disorder
* Significant bone marrow disorder
* Severe hypoproteinemia

Contraindications to Use of Methotrexate

Absolute contraindications

* Pregnancy
* Alcohol abuse
* Hepatitis B or C
* Cirrhosis of any origin

Relative contraindications

* Renal failure
* Chronic pulmonary disease
* Active infection not associated with Felty’s syndrome

Contraindications for Use of Gold Salts

* Severe liver or kidney disease
* Blood and marrow disorders

Contraindications for Use of Sulphasalazine

* Allergy to salicylates or sulfonamides
References:
1. Clinical practice guideline for the management of rheumatoid arthritis.
2. GUIPCAR Group. Clinical practice guideline for the management of rheumatoid arthritis. Madrid: Spanish Society of Rheumatology; 2001. 170 p. [430 references]


I. Acute Leukaemias
-----------------------------------------------------------

A. Lymphoblastic:
---------------------
-Accounts for 20% of all acute leukaemias.
-Accounts for 80% of childhood leukaemia.
-Peak incidence at age 4 years.

Most patients present with an acute illness of several days or less frequently, with tiredness or non-specific aches over several weeks or months.

-Clinical features can be grouped into:

1. Consequences of bone marrow failure - anaemia, bleeding, vulnerability to infection.
2. Consequences of accumulation of leukaemic cells.

Important Points to remember:

-The patient with acute lymphoblastic leukaemia is usually a child, whereas the patient with acute myeloid leukaemia is rarely a child.

-Although the marrow is usually extensively replaced by leukaemic cells, elated symptoms are uncommon. Rarely there may be bone tenderness in
childhood ALL.

-A common presentation of childhood ALL is with joint and muscle pain. ALL does not infiltrate extra-medullary tissues - lymph nodes, spleen, liver,
meninges - as much as AML.

-T cell ALL may present with superior vena caval obstruction.

B. Myeloid:
---------------------
-Accounts for 80% of acute leukaemias
-Peak incidence between 15 and 39 years

It is important to distinguish between AML and ALL:

-Morphological - Auer rods pathognomic of AML
-Histo-chemical stains for myeloid enzymes such as peroxidase or chloroacetate confirm AML
surface markers characteristic of primitive lymphoid cells identify ALL, e.g. terminal deoxynucleotide transferase present in 95% of cases of ALL
-Primitive B lymphocyte antigens such as CALLA, B1, BA1 may help to identify ALL
-T cell ALL diagnosed by rosette formation with sheep erythrocytes or identification of cell markers by monoclonal antibodies such as Leu-1 or Leu-9


II. Chronic Leukaemias
------------------------------------------------------

CLL usually has an insidious onset. Approximately 25% of patients are diagnosed incidentally when they are found to have lymphocytosis. Rapidly
progressive CLL occurs occasionally and is characterised by larger, less mature - appearing lymphocytes - "prolymphocytic" leukaemia.

Typical clinical features of CLL include:

-Non-specific symptoms - lethargy, weight loss, fever and sweating, and infections.
-Attributable to anaemia and thrombocytopenia - which may be autoimmune.
-Lymphadenopathy - occurs in 80% of cases. Typically, moderate enlargement, affecting nodes in the neck, axilla and groin. Classically,
symmetrical with non-tender, rubbery nodes. May have developed over a period of months or years.
-Hepatosplenomegaly - in 50% of cases.
-Skin lesions - pruritus, Herpes zoster, generalised infiltration (l'homme rouge, usually associated with pruritus), vesibullous lesions

The clinical features of chronic myeloid leukaemia may be classified into:


non-specific systemic features:
-anaemia
-malaise & fatigue
-weight loss
-sweating

common features:
-bleeding diathesis
-discrete ecchymoses
-menorrhagia
-splenomegaly
-hepatomegaly

rare features:
-abdominal pain or pleuritic pain due to splenic infarction
-gout
-retinal haemorrhages
-priapism
-fever

comparison of CML and CLL

-lymphadenopathy
CML: uncommon
CLL: common

-hypogammaglobulinaemia
CML: not feature
CLL: may occur

-positive Coomb's test
CML: not feature
CLL: may occur

-development of blast
CML: common
CLL: not common crisis

-prognosis
CML worse than CLL
II. SIGNATURE DRUG TOXICITIES
Agranulocytosis Clozapine
Aplastic Anemia Chloramphenicol
NSAIDs
Benzene
Atropine-like Side Effects Tricyclics
Cardiotoxicity Doxorubicin
Daunorubicin
Cartilage Damage in children Fluoroquinolones (Ciprofloxacin & Norfloxacin)
Cinchonism Quinidine
Cough ACE Inhibitors
Diabetes Insipidus Lithium
Disulfiram-like effect Metronidazole
Sulfonylureas (1
st
generation)
Extrapyramidal Side Effects Antipsychotics (Thioridazine, Haloperidol, Chlorpromazine)
Fanconi's Syndrome Tetracycline
Fatal Hepatotoxicity (necrosis) Valproic Acid
Halothane
Acetaminophen
Gingical Hyperplasia Phenytoin
Gray Baby Syndrome Chloramphenicol
Gynecomastia Cimetidine
Azoles
Spironolactone
Digitalis
Hemolytic Anemia in G6PD-deficiency Sulfonamides
Isoniazid
Aspirin
Ibuprofen
Primaquine
Hepatitis Isoniazid
Hot Flashes, Flushing Niacin
Tamoxifen
Ca
++
Channel Blockers
Induce CP450 Barbiturates
Phenytoin
Carbamazepine
Rifampin
Inhibit CP450 Cimetidine
Ketoconazole
Interstitial Nephritis Methicillin
NSAIDs (except Aspirin)
Furosemide
Sulfonamides
Monday Disease Nitroglycerin Industrial exposure ----> tolerance during week ----> loss of tolerance during weekend --
--> headache, tach, dizziness upon re-exposure
Orange Body Fluids Rifampin
Osteoporosis Heparin
Corticosteroids
Positive Coombs' Test Methyldopa
Pulmonary Fibrosis Bleomycin
Amiodarone
Red Man Syndrome Vancomycin
Severe HTN with Tyramine MAOIs
SLE-like Syndrome Procainamide
Hydralazine
Tardive Dyskinesia Antipsychotics (Thioridazine, Haloperidol, Chlorpromazine)
Tinnitus Aspirin
Quinidine

hi friends !! BODIES are asked frequently in exams so i had collected some of its and i think it will be very useful to you in different entrance exams.

Aschoff Bodies - rheumatic fever

Asteroid body - sporotrichosis

BABES - ERNST Bodies - metachromatic granules

BALBIANI'S Bodies – yolk nucleus

Bamboo bodies - asbestosis

Bodies OF ARANTIUS - aortic valve nodules

BODY OF HIGHMORE - mediastinum testis

Bollinger bodies - fowlpox

Brassy body – dark shrunken blood corpuscle

found in malaria

Call exners bodies – granulosa theca cell tumour

Chromatid bodies - entamoeba histolytica precyst

Citron bodies - cl. Septicum

Civatte bodies – lichen planus

Councilman bodies – hepatitis B

Coccoid X bodies – psittacosis

Creola bodies - asthma

Cystoid bodies – in degenerated retinal nerve

fibers ( seen in Cotton wool

spots)

Donnes bodies – colostrums corpuscles

Donovan bodies – granulose inguinale (LGV)

Ferruginous bodies – asbestosis

Gamma gandy bodies – congestive splenomegaly

Guarnieri bodies - inclusion bodies of vaccinia

Henderson Peterson bodies - molluscum

contagiosum

Harting bodies – calcospheritis in the cerebral

capillaries

Heinz bodies – G 6 PD deficiency

Herring bodies – neurohypophysis

Heterophil antibodies – infectious mononucleosis

Hirano bodies – alzheimer’s disease

Lewy bodies – parkinsonism

Levinthal coles lille bodies - psittacosis

Mallory bodies – alcoholic hepatitis

Masson bodies – rheumatic pneumonia

Michelis guttman bodies – malakoplakia

Mooser bodies – endemic typhus

Moot bodies – multiple myeloma

Negri bodies – rabies

Odland body – keratinosome

Oken’s body - mesonephros

Paschen bodies – vaccinia / variola

Pick bodies – picks disease

Psamomma bodies –1. papillary carcinoma of

thyroid ,

- 2.serous papillary carcinoma

of ovary

- 3. meningioma

- 4. mesothelioma

Reilly bodies – hurler’s syndrome


Rokitansky bodies - teratoma

Ross’s bodies – syphilis

Rushton bodies – odontogenic cyst

Sclerotic bodies - chromoblastomycosis

Sandstorm bodies – parathyroid glands

Schillar dual bodies - yolk sac tumour

Schaumann bodies - sarcoidosis

Verocay bodies – schwanoma

Winkler bodies – syphilis

Zebra bodies – metachromatic leukodystrophy

Note: Compiled by Dr Prem really tired of typing by now !..... but would like to add a few things.......

MUDIT AND ASHISH- very useful books, try to cover as much as possible.

PHYSIO(GANONG)- really pithy book. can ask from anywhere..... proof- AI06 paper !!! try to covr as much as possible.

BIOCHEM(harper)- really imp subj....can reallly make or break ur day. but very time consuming . can be done effectively by group study.

PHARMAC- (KDT)dont miss anticancer, antiarryhtmics and newer drugs from harri.

PATHO (robbns)- great book.....worth reading most of the chapters. group study very useful.

MICRO - another difficult subject..... can never be a champ...... stick to ananthnarayan... cover it as much as possible retro....and dont aim to be a champ!!!

HARRISON(Medicine !!)-

most controversial subj. my warning - never try to read tht book pro. u will never steer to the other end of that sea....and if u do , wont know where u have landed!!!!
meaning to say tht even if u manage to read it ..will never remember all.... not cost effective.
best thing acc to me - solve as many mcqs as possible and just have a look in harri about each of those topics.......DONT CRAM ALL .... ONLY READ 'THAT' TOPIC.build
database gradually
secondly, HARRISON TABLES ARE POWER PUNCHES.. DONT MISS ANYONE U COME ACROSS.............THERE ARE THOUSANDS OF HIDDEN MCQS IN THERE!!!!

Surgery- difficult subject to cover. reading L&B is fruitless. instead solve as many mcqs as possible. IF HAVE TIME ... do schwartz pretest (recommended).

PSM -scoring sujbect. group study v.imp to prevent sleep.

SKIN - harrison best acc to me. few pages will cover most+ mudit ashish ......enough.

Anaesthesia - kdt for drugs, coaching class pamplhlet +mudit khanna........enough(NO LEE).
RADIO- mudit ashish enough
RADIO THERAPY- DONT MISS THIS ONE. there are questions scattered all place in recent exms. me did not read. if u can come across some basic book...read it now
only.......will definitely give u an edge.

1) Viral Hepatitis –Repeated 20 times
Refer 16th edition Harrison Vol 2 page no 1845-1851

2) Acid base balance – Repeated 15 times
Refer 16th edition Harrison Vol 1 page no 263-270

3) CA lung – Repeated 14 times
(Ref: 16th edition Harrison vol 1 page no 506-514)

4) Migraine/Temp Arteritis/Cluster Head Ache – Repeated 14 times
(Ref: 16th edition Harrison vol 1 page no 87-93)

5) Intracranial Haemorrhage – Repeated 13 times
(Ref: 16th edition Harrison vol 2 page no 2387)

6) Hyperparathyroidism – Repeated 11 times
(Ref: 16th edition Harrison vol 2 page no 2252)

7) Cardiac Tamponade -Repeated 11 times
(Ref: 16th edition Harrison vol 2 page no 1415)

8) CSF in meningitis –Repeated 10 times
(Ref: 16th edition Harrison vol 2 page no 2476-Table 360-1)

9) HOCM – Repeated 10 times
(Ref: 16th edition Harrison vol 2 page no 1410)

10) Antiphosholipid antibody syndrome – Repeated 10 times
(Ref: 16th edition Harrison vol 2 page no 1681-1682)

11) Multiple myeloma- Repeated 10 times
(Ref: 16th edition Harrison vol 1 page no 656)

12) Coarctation of aorta – Repeated 9 times
(Ref: 16th edition Harrison vol 2 page no 1387)

13) HSP – Repeated 9 times
(Ref: 16th edition Harrison vol 2 page no 2010)

14) Psoriatic/Osteo/Rheumatoid Arthritis – Repeated 9 times
(Ref: 16th edition Harrison vol 2 page no 1998/2036/1968)

15) Thymoma leads to Hypogammaglobulinaemia – repeated 8 times
(Ref: 16th edition Harrison vol 2 page no 1946)

16) MEN – Repeated 8 times
(Ref: 16th edition Harrison vol 2 page no 2231)

17) Phaeochromocytoma – Repeated 8 times
(Ref: 16th edition Harrison vol 2 page no 2148)

18) No infective endocarditis in ASD- Repeated 8 times
(Ref: 16th edition Harrison vol 2 page no 1385)

19) Polycythaemia – Repeated 7 times
(Ref: 16th edition Harrison vol 1 page no 335)

20) Vit B12 deficiency – Repeated 7 times
(Ref: 16th edition Harrison vol 2 page no 2404)