You are on page 1of 3


Who says smoking cigarettes are so bad ... well, aside from the World Health Organization, Food
and Drug Administration, the Centers for Disease Control and Prevention, and every medical
board and association on the face of the Earth?
But should smokers be fortunate enough to dodge all that cancer, heart disease, emphysema etc.,
they will be uniquely protected for reasons unexplained by science against a handful of
diseases and afflictions.
Although long-term smoking is largely a ticket to early death, here are five possible benefits
from smoking.
1. Smoking lowers risk of knee-replacement surgery
While smokers might go broke buying a pack of cigarettes, they can at least save money by
avoiding knee-replacement surgery. Surprising results from a new study have revealed that men
who smoke had less risk of undergoing total joint replacement surgery than those who never
The study, from the University of Adelaide in Australia, appears in the July issue of the journal
Arthritis & Rheumatism. What could be the connection? Knee-replacement surgery was more
common among joggers and the obese; smokers rarely jog, and they are less likely to be
morbidly obese.
After controlling for age, weight and exercise, the researchers were at a loss to explain the
apparent, albeit slight protective effects of smoking for osteoporosis. It could be that the nicotine
in tobacco helps prevent cartilage and joint deterioration.
2. Smoking lowers risk of Parkinson's disease
Numerous studies have identified the uncanny inverse relationship between smoking and
Parkinson's disease. Long-term smokers are somehow protected against Parkinson's, and it's not
because smokers die of other things earlier.
The most recent, well-conducted study was published in a March 2010 issue of the journal
Neurology. Far from determining a cause for the protective effect, these researchers found that
the number of years spent smoking, more so than the number of cigarettes smoked daily,
mattered more for a stronger protective effect.
Harvard researchers were among the first to provide convincing evidence that smokers were less
likely to develop Parkinson's. In a study published in Neurology in March 2007, these
researchers found the protective effect wanes after smokers quit. And they concluded, in their
special scientific way, that they didn't have a clue as to why.
3. Smoking lowers risk of obesity
Smoking and, in particular, the nicotine in tobacco smoke is an appetite suppressant. This
has been known for centuries, dating back to indigenous cultures in America in the pre-
Columbus era. Tobacco companies caught on by the 1920s and began targeting women with the
lure that smoking would make them thinner.
A study published in the July 2011 issue of the journal Physiology & Behavior, in fact, is one of
many stating that the inevitable weight gain upon quitting smoking is a major barrier in getting
people to stop, second only to addiction.
The relationship between smoking and weight control is complex: Nicotine itself acts as both a
stimulant and appetite suppressant; and the act of smoking triggers behavior modification that
prompts smokers to snack less. Smoking also might make food less tasty for some smokers,
further curbing appetite. As an appetite suppressant, nicotine appears to act on a part of the brain
called the hypothalamus, at least in mice, as revealed in a study by Yale researchers published in
the June 10, 2011, issue of the journal Science.
No respectable doctor would recommend smoking for weight control, given the toxic baggage
accompanying cigarettes. This recent Yale study, however, does offer an inkling of hope for a
safe diet drug to help obese people control their appetites.
4. Smoking lowers risk of death after some heart attacks
Compared with non-smokers, smokers who have had heart attacks seem to have lower mortality
rates and more favorable responses to two kinds of therapy to remove plaque from their arteries:
fibrinolytic therapy, which is basically medication; and angioplasty, which removes the plaque
by inserting balloons or stents into the arteries.
There's a catch, though. The reason why smokers have heart attacks is that smoke scars the
arteries, allowing fat and plaque to build up in the first place. So, one theory as to why smokers
do better than non-smokers after such therapies is that they are younger, experiencing their first
heart attack approximately 10 years before the non-smoker.
A study published in an August 2005 issue of the American Heart Journal, however, states that
age alone is not enough to fully explain the survival differences and that "the smoker's paradox is
alive and well." No alternative theories have been put forth since.
5. Smoking helps the heart drug clopidogrel work better
Clopidogrel is a drug used to inhibit blood clots for those patients suffering from coronary artery
disease and other circulatory diseases leading to strokes and heart attacks. Smoking seems to
help clopidogrel do its job better.
A study by Korean researchers in the October 2010 issue of the journal Thrombosis Research
builds upon work by Harvard researchers published in 2009 that demonstrates the benefit of
smoking at least 10 cigarettes a day. It seems that something in cigarette smoke activates certain
proteins called cytochromes, which convert clopidogrel into a more active state.
Again, no respectable doctor is encouraging patients to start smoking to get the most out of
clopidogrel. But this and the other four "benefits" of smoking reveal how tobacco perhaps not
unlike other potentially toxic plants might contain certain chemicals of real therapeutic value.