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The anterior surface of the (left) outer ear cartilage and outer ear

muscles.
Translated by: Ronald A. Bergman, PhD and Adel K. Afifi, MD, MS
Peer Review Status: Internally Peer Reviewed

a) Helix.
b) Spina helices.
c) Cruraanthelicis.
d) Crus superiusanthelicis.
e) Crus inferiusanthelicis.
f) Fossa triangularis (s. fossa innominata).
g) Scapha (s. fossa navicularis).
h) Tragus.
i) Antitragus.
k) Incisuraintertragica (s. incisuraauriculae).
l) m. Concha auris.
m) External auditory meatus.
n) m. Auricularis superior (s. m. attolens).
o) m. Auricularis anterior (s. m. attrahans).
p) m. Auricularis posterior s. m. retrahentes).
q) m. Helicis major.
r) m. Helicis minor.
s) m. Tragicus.
t) m. Antitragicus.

http://www.anatomyatlases.org/atlasofanatomy/plate31/01antouterear.shtml

or heat absorbed/T (i. the heat of fusion.mcb. Likewise the heat of vaporization is measured when Hbonds are broken on the liquid-gas transition.edu/anim_innerlife.harvard. namely. to break H-bonds and cause melting. an increment of heat is required.html ) Lipid Conformational Transitions If a liposome preparation is placed in a sensitive calorimeter and the temperature slowly increased. the heat capacity) vs temperature are shown below for the melting and evaporation of water. breaking H-bonds. and for liposome transitions. These transition are associated with non-covalent processes. This is analogous to what would happen if solid water was heated. At the melting point of water. it is observed that the liposome preparation absorbs a significant amount of heat at a temperature characteristic of the PL which compose the liposome.Figure: Lipid Rafts enriched in SM and Cholesterol (screen capture from: http://multimedia.e. . Graphs of heat absorbed (Q) as a function of temperature.

urticarial plaques.http://employees. Although most patients are asymptomatic. Louis Duhring at the University of Pennsylvania. and papules with vesicles.medscape. Dermatitis herpetiformis is exquisitely pruritic. The disease was described and named in 1884 by Dr.ht ml Background Dermatitis herpetiformis (DH) is an autoimmune blistering disorder associated with a gluten-sensitive enteropathy (GSE). Classic vesicles of dermatitis herpetiformis. Diagnosis requires direct immunofluorescence of a skin biopsy specimen showing deposition of immunoglobulin A (IgA) in a granular pattern in the upper papillary dermis. as shown in the image below.edu/hjakubowski/classes/ch331/lipidstruct/oldynamicves. The classic location for dermatitis herpetiformis lesions is on the extensor surfaces of the elbows. knees. buttocks. Among patients with celiac disease. The mainstays of treatment are dapsone and a gluten-free diet.1Dermatitis herpetiformis is characterized by grouped excoriations. http://emedicine.com/article/1062640-overview . erythematous. and back. greater than 90% have an associated gluten-sensitive enteropathy upon endoscopic examination. and the vesicles are often excoriated to erosions by the time of physical examination.csbsju. 15-25% develop dermatitis herpetiformis.

epiblastic cells migrate anteriorly through the node to become the longitudinally running cellular rod called the notochord. A collection of cells at the end of the primitive streak is the primitive node (Hensen's node) . the methods available for the removal or improvement of acne scars and for the correction of wrinkle lines increased exponentially with the advent of new skin filler substances. Under the influence of the underlying is seen lying notochord.temple.edu/neuroanatomy/lab/embryo_new/nt/1/ Introduction Over the past 30 years. gastrulation begins and mesoderm appears (also originating from epiblast). the dorsal ectodermal surface of the early embryo thickens and elongates to form neural plate. Subsequent changes convert the plate into a neural tube which will give rise to the CNS. improved . the streak is a linear thickening on the dorsal surface of the epiblast in which cells of the epiblast form endoderm and mesoderm. which develops from the axial mesoderm. Gastrulation begins with the formation of the primitive streak at the posterior (caudal) end of the embryo. http://isc. Beginning at the third week.Neural Tube Development: Formation and Closure (Page 1 of 4) 16 20 22 Days 24 Days Days Days Prior to implantation early in the 2nd week the inner cell mass converts to an epiblast (primitive ectoderm) and a hypoblast (primitive endoderm).

and technology for abrading and resurfacing facial contours. the basic mechanisms remain the same. Before and after images are below. a new and improved filling agent or laser. and (3) contouring the surface of scars.techniques for elevating existing scars. (2) filling depressed scars. Seemingly every month. In the midst of these technological breakthroughs. . which will be the best of all available methods. Photograph before collagen injection. Three categories of techniques are presently available to improve acne scars: (1) scar removal and revision. becomes available.

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com/article/1271282-overview Oesophagus. http://emedicine. This irregular border aids in anchoring of the epithelium to the connective tissue. human . Basal cells often form a well defined layer at the border of the epithelium to the underlying connective tissue. The border between epithelium and connective tissue may appear quite irregular because of the papillae. If these extensions are not cut exactly along their long axis.H&E The oesophagus is lined by a stratified squamous epithelium consisting of many cell layers. Draw the stratified squamous epithelium of the oesophagus and label your drawing. Try to draw a little schematic illustration which shows how the plane of section would effect the appearance of the .medscape. The underlying connective tissue forms fingerlike extensions towards the lumen of the oesophagus.Photograph after collagen injection. which are called papillae. they may look like isolated small islands of connective tissue and blood vessels within the epithelium.

com/article/758322-overview an adenovirus Genus Aviadenovirus fowl adenovirus 1 Image reconstruction reveals the complex molecular organization of adenovirus . there are no missed beats.lab. Firstdegree heart block.1First-degree heart block is considered "marked" when the PR exceeds 300 msec. http://emedicine. http://www. ECG in a patient with first-degree heart block. this interval should be between 120 and 200 msec. or first-degree atrioventricular (AV) block.anhb.au/mb140/ On an electrocardiogram (ECG).uwa.edu. is defined as prolongation of the PR interval on the ECG to more than 200 msec. ECG of a patient with first-degree heart block is shown below. Normally. the PR interval is defined as the time interval between the initial deflection of the P wave to the start of the QRS complex.connective tissue extensions.2While the conduction is slowed.medscape.

animal adenovirus From Stewart McNulty at Veterinary Sciences.avian adenovirus From Milan Nermut from the UK's National Institute for Biological Standards and Control. The sample was freeze-dried and shadowed with Pt/C.html Choroidal melanoma is the most common primary malignant intraocular tumor and the second most common type of primary malignant melanoma in the body.virology. Queen's University. .net/Big_Virology/BVDNAadeno. Color photograph of a dome-shaped choroidal melanoma. Egg Drop Syndrome Virus http://www. It is nevertheless an infrequently found tumor. Belfast.

Usually. Urate crystals are easily seen with polarized light. Gram stain. Gram stain and culture. but the apatite crystals typical of calcific tendinitis appear only as shiny chunks that are not birefringent. ciliary body. fluid is withdrawn from the bursa using sterile techniques.com/article/1190564-overview Superficial bursitis should be suspected in patients with swelling or signs of inflammation over bursae. bursitis can be diagnosed clinically. analysis includes cell count. palpation. or warm or if the olecranon or prepatellar bursa is affected. when the tumor arises in the iris. and in posterior uveal melanomas. It is primarily a vascular tissue. The uvea is subdivided into iris.Choroidal melanoma is a subtype of uveal melanoma. The choroid underlies the retina and its pigment epithelium throughout the ocular fundus. These tests are done to confirm or exclude a suspected diagnosis. although helpful. and choroid. with fenestrated capillaries and stroma containing melanocytes. Ultrasonography or MRI can help confirm the diagnosis when deep bursae are not readily accessible for inspection. red. These imaging techniques increase the accuracy of identifying the involved structures. X-rays should be taken if bursitis is persistent or if calcification is suspected. is a densely pigmented layer that forms part of the wall of the eye. Intraocular melanomas simultaneously can involve more than one uveal structure. Uveal melanomas can be classified in anterior uveal melanomas. infection and crystal-induced disease should be excluded by bursal aspiration. or aspiration. and microscopic search for crystals. The main function of the uvea is to provide oxygen and other nourishment to the highly metabolically demanding retinal photoreceptors. the uvea. http://emedicine. Deep bursitis is suspected in patients with unexplained pain worsened by motion in a location compatible with bursitis. Prepatellar Bursitis . may not be specific. After a local anesthetic is injected. If bursal swelling is particularly painful.medscape. and WBC counts in infected bursae are usually lower than those in septic joints. The ocular tissue where these tumors arise. when it arises in either the choroid or the ciliary body.

cellulitis overlying the bursa is a relative contraindication to bursal puncture through the cellulitis. Fluid in traumatic bursitis is serosanguinous. Epidemiology • Up to 25% of patients with metastatic carcinomas • Lung and breast predominate • Also GI tract.html Metastatic Adenocarcinoma • The gray white nodules which obliterate normal architecture are metastatic carcinoma. General Microscopic Description • Resembles primary site . • Yellow tissue is normal cortex. but if septic bursitis is strongly suspected. tan with or without hemorrhage or necrosis.com/mmpe/sec04/ch040/ch040b. by: UCHC ) Metastatic Adenocarcinoma Click on Image to Enlarge it Metastatic Adenocarcinoma Etiology • Unknown.D. (Description By:Melinda Sanders. kidney. which can cause similar manifestations because blood is inflammatory.Acute bursitis should be distinguished from hemorrhage into a bursa. Cellulitis can cause signs of inflammation but does not normally cause bursal effusion. aspiration must occasionally be done. other adrenal General Gross Description • May be cortical or medullary. ) (Image Contrib. thyroid. Pathogenesis • Arrive via systemic circulation • Unknown why lung carcinoma has predilection for adrenals. M. brown gray normal medulla.merck. http://www.

Philadelphia: Lippincott-Raven. 585-6.Clinical Correlation • Requires chemotherapy to treat. Metastatic Adenocarcinoma http://radiology. • May be associated with mild adrenal failure. 2d edition. pp.1996.edu/eAtlas/Endo/299. Sternberg SS (ed).htm .uchc. References • Diagnostic Surgical Pathology.