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Promised Infonnation Page 1 of 1

(b)(6)

From: Kratz, Kurt, ,OSD-ATL


Sent: Wednesday, November 19,20038:19 AM
To: Van Brocklin, Connie H Ms ACSIM
Cc: Newsome, Richard E Mr ASA..;I&E; Cotter, Sandra, Ms, OSD-ATL
Subject: FW: Promised Information
Attachments: Perchlorate_ActiveBRAC sites. pdf; Sites using USEPA Data.xls; DoD Sites with RB Ltrs.xls

Connie,

Enclosed is the list of installations that should have received the letters. We promised on Weds, last, to provide

an ETA to CA on the answers.

Thx,

Kurt

-----Original Message----­
From: Brausch, Rick [mailto: • rll.n•••••••

Sent: Monday, November 17, 2003 7:18 PM

I
To: 'Kratz, Kurt, ,OSD-ATL ;ri1MftrEfiRlaHI• • • • • •

Cc: Ijames Giannopoulos (E-mail)'; 'Lisa Babcock (E-mail)'; 'Linda Dorn (E-mail)'; 'Ton Vorster (E-mail)'; 'Rick

Moss (E-mail)'; • a Schuparra, Kurt; Spagnole, Jim

Subject: Promised Information

Kurt and Sandy ­

Attached are two spreadsheets with information we promised at our video conference on Wednesday. The first is

the list of California installations/sites that received letters from a RegionalBoard requesting perchlorate

information. The second is the list of sites represented in the spreadsheet that was sent with the maps last week

(also attached for your convenience).

«000 Sites with RB Ltrs.xls» «Sites using USEPA Data.xls» «Perchlorate_ActiveBRAC sites. pdf»

In addition, we checked with the Department of Health Services regarding access to the data overlays for the

supply well information used to generate the maps. They indicated that they require a signed agreement with any

government entity that desires the information. I suggest you contact David Spathrl~ftniTif3k;J~• • • •

, • or Steven Book , for additional information.

Looking forward to hearing from you soon on when we can expect responses to the Regional Board letters.

Rick

Active and BRAC FUD Sites** Perchlorate Sites with


Sites** Manufacturing Confirmed
Sites** Perchlorate
Releases ***
Total Sites 85 1,257 52 28
Statewide
Number of sites 5 47 6 3
within 1-mile
radius of DHS
public supply
wells having at
least one 4 PPB
perchlorate
detection *
Number of sites 16 227 24 15
within 5-mile
radius of DHS
public supply
wells having at
least one 4 PPB
perchlorate
detection *

* 5-mile buffer zone includes those sites within the 1 mile buffer zones from the impacted DHS
wells. DHS well locations and perchlorate concentration data from DHS database (10/2002)

** Data source: USEPA.612oo3

*** Data source: SWRCB, 2003


Perchlorate

000, FUDs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

000 Sites ONE MILE FROM MUNtCIPAL SUPPLY WELL


Branch Base Name
Air Force DOD Mather Air Force Base
Army DOD Fort Ord Military Reservation
Air Force DOD Edwards Air Force Base
Air Force DOD Norton Air Force Base
Navy DOD San Nicolas Island Naval Reservation

000 Sites FIVE MILES FROM MUNtCIPAL SUPPLY WELL


Air Force DOD McClellan Air Force Base
Air Force DOD McClellan Air Force Base
Air Force DOD Mather Air Force Base
Navy DOD Stockton Naval Communications Station
Army DOD Sharpe General Depot (Field Annex)
Navy DOD Moffett Field Naval Air Station
Army DOD Fort Ord Military Reservation
Navy DOD Lemoore Naval Air Station
Army Corps of Engineers DOD Success Lake
Air Force DOD Edwards Air Force Base
Air Force DOD Norton Air Force Base
Air Force DOD March Air Force Base
Army DOD Los Alamitos Armed Forces Reserve Center
Navy DOD Seal Beach Naval Weapons Station
Marine Corps DOD Tustin Marine Corps Air Station
Navy DOD San Nicolas Island Naval Reservation

Manufacturers ONE MILE FROM MUNICtPAL SUPPLY WELL


Site City
Olin Safety Flare Morgan Hill
US Rocket Pomona
Dynamic Propellant Ontario
Jet Propulsion Laboratory (NASA) Pasadena
San Gabriel Valley Baldwin Park
Astro Pyrotechnics Rialto

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 1
, Perchlorate
000, FUOs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

Manufacturers FIVE MILES FROM MUNICIPAL SUPPLY WELL


Mather AFB Rancho Cordova
Olin Safety Flare Morgan Hill
Trojan Fireworks Palmdale
Kinetech Corp Rancho Cordova
Lawrence Nat! Lab Tracy
Starflight Space Technologies Windsor
US Rocket Pomona
San Fernando Valley Glendale
U.S. Navy Firing Range San Nicolas Island
Stri.ngfellow Glen Avon
Aerojet General Corp. Azusa
Dynamic Propellant Ontario
McKesson Chemical Co. Riverside
Jet Propulsion Laboratory (NASA) Pasadena
Aerojet General Corp. -Rancho Cordova Rancho Cordova
San Gabriel Valley Baldwin Park
Lockheed Propulsion Company (Former) Redlands
Astro Pyrotechnics Rialto
B.F. Goodrich Aerospace & Defense Products Rialto
NATIONAL TECHNOLOGIES SYSTEM (NTS)
Hi-Shear Technology Corp. Santa Clarita
Whittaker Bermite Division. Saugus
WYLE LABS
Denova Environmental

FUDs FIVE MILES FROM MUNICIPAL SUPPLY WELL


ANAHEIM AIRPORT
STOCKTON ORDNANCE DEPOT
MARCH AFB RIFLE RANGE
MARCH FIELD
SAN NICHOLAS ISLAND NAVAL RES.
SANTA ANITA RECEPTION CENTER ARMY CAMP
STOCKTON MIL AF
POLLOCK STOCKTON SHIPBLDG
SUNNYVALE
HANFORD WAREHOUSE

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 2
Perchlorate

000, FUOs and Manufacturers Within i-mile and 5-miles of a Municipal Supply Well

SPADRA GENERAL HOSPITAL


MCCLELLAN AFB OUTER MARKER
SAN NICHOLAS AIRFIELD
NORWALK AF POL RETAIL DIS #2
SAN ANTONIO DAM
MIRA LOMA ENGR SUB-DEPOT
LA RIVER IMPR SEC VII
ADVANCE Q.M. DEPOT
ARMY AIR CORPS SUPPLY DEPOT
ONTARIO ARMY AIRFIELD
ORDINANCE BACKUP STORAGE DEPOT
RYAN AIRCRAFT SCHOOL
OBSERVATION SQUADRON ONTARIO ARMY AIRPORT
QUARTERMASTER SUPPLY DEPOT
WEST COAST AIR TRAINING CENTER
NAVAL RESERVE ARMORY
VULTEE AIRCRAfT CO.
VEGA AIRCRAFT
ALHAMBRA AIRPORT
ARLINGTON STAGING AREA CAMPANZA
WESTERN STOVE BOMB PLANT
HAYWARD BOMB PLANT
ARMY SUPPLY BASE GENERAL DEPOT
VEGA AIRCRAFT
VULTEE TRAINING FIELD
PALMDALE AIRPORT
STATE GUARD AIR FIELD
NAVAL AIR STATION
IRVINE PARK- ARMY CAMP
Af PLANT #14
CAMP KOHLER
LOS ANGELES ORDNANCE DEPOT
PRODUCTION FLiGH TEST AF PLT42
NIRF (UNDERSEA CENTER)
FORMER NIKE SITE LA-14 LAUNCHER AREA
NORTH AMERICAN AVIATION
SAN BER ENGR DEPOT

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 3
Perchlora~e
DoD, FUDs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

MC SUPPLY FORWARDINGANNEX
NAAS SANTA ROSA
SANTA ROSA AAF
LONG BEA MUNICIPAL AIRPORT
PALM SPRINGS ARMY AIR FIELD
GRAND CENTRAL AiR TERMINAL
CPANZA
BREA FLOOD CONTROL BASIN
SANTA ANITA ORD TRAINING CTR
CLAREMONT RIFLE RANGE
MARCH TVOR ANX
WINDSOR POW LABOR CAMP
NIROP SACRAMENTO
NAS MOFFETT FIELD
NSC ROUGH & READY ISLAND
SHARPE GENERAL DEPOT
AF PLANT #71(AIR PROD&CHE
CLASSIFICATION YARD
KINSBURY AUX FLD #1
TRACY AUX FLD #5
PORTERVILLE ARMY AF
AZUSA DUMP SITE OWL 4X PL
NIKE BATTERY 14 - SILOS
SAN BARNARDINO ARMY AIRFIELD
MOFFETT MIL AF
MARCH MILITARY AIRFIELD
SANTA ROSA FEDERAL CENTER
GEN ENGRG & DRYDOCK CO
AF PLANT #42 K9ANX
AIR FORCE PLANT #16 (NASA)
MCCLELLAN COMM FAC ANX
NORWALK AF POL DIST STATION #1
MARQUARDT RAM JET DEV FACILITY
AIR FORCE PLANT 15 (NAA)
MARCH COMMUNICATION ANNEX #2
KINNER MOTORS
DOWNEY FAMILY HOUSING

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 4
Perchlorate

DoD, FUDs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

CHEll AFS
AIR FORCE PLANT #76
VAN NUYSAAF
NORTON AFB ILS OUTER MARKER AN
MC CLELLAN SRC AUX
BAKERSFIELD MUNICIPAL AIRPORT
CAL-AERO AIRPORT
CARBON CANYON FLOOD CONT BASIN
FACTORY TRAINING SCHIDOUGLAS
DEL MAR-ENCINITAS FCS #1
GRIFFITH PARK PHOTO CENTER
GRIFFITH PARK EXPERIMENTAL LAB
HANSEN FLOOD CONTROL BASIN
FULLERTON DAM-E FULLERTON CREE
LA DEF AREA NIKE BTRY 96
KOBE INC
KAISER SHELL CASING PLANT
JET PROPULSION LAB
LONG BEACH CA BRIGADE SITE
LOCKHEED AIR TERMINAL IDI
LA DIST ENG CON PROJ OFF
LA DIST MAINT YARD
MIRA LOMA AIR FORCE STATION
MIRA LOMA QUARTERMASTER DEPOT
MARCH WATER SYSTEM ANNEX
LOS ANGELES MEDICAL DEPOT
LOPEZ FLOOD CONTROL BASIN
ORANGE COUNTY ARMY AIRFIELD
ONTARIO AN.G. TRAINING SITE
NORTON AIR FORCE BASE
PRADO FLOOD CONTROL BASIN
POMONA QUARTERMASTER DEPOT
PASADENA AREA SUPPORT CENTER
POMONA ORDNANCE DEPOT
PALMDALE CANT AREA
PALMDALE ARMY AIR FIELD
SAN ANTONIA/CHINO CREEKCHANNEL

The data prOVided in this table is from the USEPA The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 5
Perchlorate

000, FUOs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

SALVAGE SEGREGATION CENTER


SAN BERNARDINO BASE GEN D RR#1
RIALTO AMMUNITION STORAGE POIT
LOS ALAMITOS RADAR BOMB SCORIN
RADAR BOMB SCORING
QUARTERMASTER STORAGE
SAN BERNARDINO ENG SUB-DEPOT
SAN BERNARDINO CWS PL
SAN BERNARDINO BASE GEN D RR2
SEPULVEDA FLOOD CONTR"OL BASIN
SANTA FE DAM
SAN ANA AIR NG RADAR RELAY
SANTA ANITA ORO TRNG CT RIF RG
SAN GABRIEL RIVER IMPROVEMENTS
SAN FERNANDO PLAYGROUND
U.S. ARMY RES CENTER PACOIMA
USARC BELL
USAR OUTDR TRAINNG AREA WH NAR
U.SAR. HUNTINGTON PARK
TUSTIN NATIL GUARD RIFLE RANGE
TORNEY GEN HOSPITAL
VAN NUYS MET AIRPORT
USARC SANTA ANA
WHITTIER NARROWS FC BASIN
WEST LA AREA STATION HOSPITAL
STO DIV CAN & CAL
SHARPE ARMY DEPOT
BOMB MANUFACTURING/STORAGE PLT
CAMPONOPOW
USNAS LOS ALAMITOS
NAVAL WEAPONS INDUST RES PLT
MCAS HOLFMILE FIELD
CALIF REHABILITATION CENTER
NAVAL ORDNANCE LAB
TAYLOR FORCE & PIPE CO
WESTERN GEAR WORKS OF CA
NAVAL INDUSTRIAL RESEARCH AP

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 6
Perchlorate

000, FUOs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

MCAS SANTA ANA


CORONA ANNEX
BRAZIL STREET DEPOT
LOCKHEED-CALIFORNIA
STEARNS-RODGERS
ONTARIO ARMY AIRFIELD
NINTH SERVICE COMMAND
AF PLANT #14 STORAGE ANNEX
NORTON COMMUNICATION FACILITY
SANTA ROSA FEDERAL RESERVE CENTER
NIKE BTRY #09-MT DISAPPOINTMENT
NIKE BTRY - LOS PINETOS
AIRSTRIP N SAN BERNARDINO
PASADENA ARMY HOSPITAL
NORCO USARC
PYRITE CANYON
ARMY FIELD (OPERATIONS HOSPITAL)
ADEL PRECISION PRODUCTS CORP.
AEROJET ENGINEERING CORP.
ASSOCIATED MANUFACTURING CO.
AVIATION GASOLINE FACILITIES
AXELSON CORP.
BAASH-ROSS TOOL COMPANY
BAKERSFIELD GAUYULE PROCESSING MILL
BAKERSFIELD VEGETABLE DEHYDRATION
CAL-AERO FIELD
CALIFORNIA INSTITUTE OF TECHNOLOGY
CAMP COMMANDER
CAMP RUBIDOUX - ARMY
CHINO PRISONER OF WAR CAMP - ARMY
CHRYSLER MOTORS
COMPAK FOODS
FIRESTONE TIRE & RUBBER CO.
GENERAL METALS CORP.
GLOBE ASBESTOS CO.
HIGHLAND AVE. AUXILIARY FIELD
HQ SOUTH DIST. 9TH SERVICE COMMAND

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 7
Perchlorate

DoD, FUDs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

JOSHUA HENDY IRON WORKS


KERN COUNTY DEHYDRATING"PLANT
KINNEY ALUMINUM CO.
LA QUINTA
LINCOLN FOUNDRY CORP.
LOS ANGELES SIGNAL DEPOT
LOS ANGELES U.S.N. REPAIR DEPOT
MAGNOLIA HOUING PROJECT
MENASCO MANUFACTURING CO.
MERCOCO.
METROPOLITAN AIRPORT
NATIONAL CYLINDER GAS CO.
NAVAL HOSPITAL
NAVAL HOSPITAL UNIT 6
NAVY TRAINING SCHOOL
NEWHALL AIRFIELD
NORTHERN TRANSPORTATION CO.
NORWALK AIRPORT - ARMY
PACIFIC PUMP WORKS
POMONA AIRPORT
RHEEM MANUFACTURING CO.
ROSS FIELD
SAN FERNANDO VALLEY AIRPORT
SAN NICOLAS ISLAND ARMY AIR BASE
SOUTHERN CAL. GAS CO.
VARDS INC
WEBER SHOWCASE AND FIXTURE CO.
WILSHIRE OIL CO.
MCCLELLAN AFB TEMPORARY HOUSING
TEMECULA BOMB TARGET #107
OUTER LANDING FIELD COTATI
LAKE HODGES SCAR ROCKET RANGE

FUDs ONE MILE FROM MUNICtPAL SUPPLY WELL


STOCKTON MIL AF
MIRA LOMA ENGR SUB-DEPOT
ORDINANCE BACKUP STORAGE DEPOT

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 8
Perchlora~e
000, FUOs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

WEST COAST AIR TRAINING CENTER


VULTEE AIRCRAFT CO.
VEGA AIRCRAFT
HAYWARD BOMB PLANT
VEGA AIRCRAFT
VULTEE TRAINING FIELD
STATE GUARD AIR FIELD
NIRF (UNDERSEA CENTER)
NORTH AMERICAN AVIATION
GRAND CENTRAL AIR TERMINAL
AZUSA DUMP SiTE OWL 4X PL
SAN BARNARDINO ARMY AIRFIELD
SANTA ROSA FEDERAL CENTER
AIR FORCE PLANT #16 (NASA)
MARCH COMMUNICATION ANNEX #2
CHEll AFS
JET PROPULSION LAB
LA DIST ENG CON PROJ QFF
ORANGE COUNTY ARMY AIRFIELD
NORTON AIR FORCE BASE
POMONA QUARTERMASTER DEPOT
POMONA ORDNANCE DEPOT
SAN ANTONIA/CHINO CREEKCHANNEL
RIALTO AMMUNITION STORAGE POIT
QUARTERMASTER STORAGE
SAN ANA AIR NG RADAR RELAY
USARC BELL
USAR OUTDR TRAINNG AREA WH NAR
TORNEY GEN HOSPITAL
STO DIV CAN & CAL
SHARPE ARMY DEPOT
NAVAL WEAPONS INDUST RES PLT
NAVAL INDUSTRIAL RESEARCH AP
CALIFORNIA INSTITUTE OF TECHNOLOGY
CAMP RUBIDOUX - ARMY
CHINO PRISONER OF WAR CAMP - ARMY
HQ SOUTH DIST.

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 9
Perchlorate
000, FUDs and Manufacturers Within 1-mile and 5-miles of a Municipal Supply Well

JOSHUA HENDY IRON WORKS


LINCOLN FOUNDRY CORP.
LOS ANGELES U.S.N. REPAIR DEPOT
NAVY TRAINING SCHOOL
NEWHALL AIRFIELD
PACIFIC PUMP WORKS
VARDS

The data provided in this table is from the USEPA. The USEPA collected data from various sources (USGS, USACE, California DHS). The SWRCB is not
responsible for its accuracy. Page 10
DRAFT Emergent ChemicallPerchlorate Letter Requests as ofSeptember 12, 2003

Regional ISSUE DUE REVISED RECEIVED RESPONSE TRACKING MMRP


BUSINESS NAME Board DATE DATE DUE.OATE DATE DESCRIPTION REVIEWED NUMBER Inventory
USSRAAF 1 712112003 913012003 SOUfCEl Evaluation Report NONE
A1amedaNAS 2 611112003 9/1512003 Source Evaluation. Report NONE
Alameda Naval SUpply Center (NSC) Annex 2 6/1112003 9/1512003 Source Evaluation Report .NONE
Concord Naval Weapons Station 2 6/27/2003 912712003 Source Evaluation Report 2119.1058
Concord PFC Bacciglieri Armed Forces Reserve Cente 2 71112003 10/112003 Source Evaluation Report 2119.1238
Golden Gate National Recreational Area 2 6127/2003 912712003 Source Evaluation Report 2159.5145
Hayward Army Airfield 2 7/112003 10/112003 Source Evaluation Report 2199.9441
San Jose AMSA 12sUB 2 71112003 101112003 Source Evaluation Report 2119.1238
SANDIA NATIONAl lABS DOE 2 612712003 912712003 911712003 Source Evaluation Report 2119.91

Travis AFB 2
Vallejo Young USARC 2 71112003 101112003 Source Evaluation Report. 2129.2086
Camp Stoneman 2 11112003 101112003 Source Evaluation Report 2199.1239
OAB Heroic War Dead USAR (B1086, 1060, 1064, 1070, 2 71312003 101112003 Source Evaluation Report 2199.944

Camp Roberts 3 81612003 1016/2003 Source area evaluation report NONE

Fort Hunter Liggett 3 81112003 101212003 Source area evaluation report NONE

LOMPOC, BRANCH U.S. DISCIPLINARY BARRACKS 3 81612003 101612003 Source area evaluation report NONE

Monterey Peninsula Airport 3 91312003 10128/2003 Source area evaluation report NONE

Presidio of Monterey 3 91312003 1012812003 Source area evaluation report NONE

Vandenberg Air Force Base 3 41912003 51912003 111112003 512012003 Source area evaluation report YES NONE

San Nicolas Istand (ind. With PI. Mugu NAWS) 4


Naval Information Research Foundation (J09CAI05200 4 911112003 1113012003 SOl.lfCe'area evaluation NONE
Naval Marine CoMilitarys Reserve Center, los Angel 4 512912003 9130/2003 source area evaluation NONE
Oxnard Air Base, Camarillo Site (J09CAOI2600) 4 611712003 913012003 source area evaluation NONE
San Pedro, Fuel Terminal DFSP 4 5129/2003 913012003 source area evaluation NONE
Bakersfield Municipal Airport 5
Source Area
Beale Air Force Base 5 611112003 913012003 Characterization Report NONE
SOUfCEl Area
Beale Air Foree Base - Titan 1A 5 611112003 913012003 Characterization Report NONE
Source Area
Beale Air Force Base - Titan 1B Sutter Co. 5 611112003 9130/2003 Characterization Report NONE
Source Area
Beale Air Foree Base - Titan 1C, Butte Co. 5 6111/2003 913012003 Characterization Report NONE
Source Area
CasUe Air Foree Base 5 611112003 913012003 Characterization Report NONE
Source Area
Defense Distribution San Joaquin CA - Sharpe 5 6111/2003 913012003 Characterization Report NONE
Source Area
Defense Distribution San Joaquin CA - Tracy 5 611112003 913012003 Characterization Report NONE
Source Area
Deganawidah-QuetzalcoaU University 5 611112003 913012003 Characterization Report NONE

DoD Sites WIth RB LlI> xis


DRAFf Emergent Chemical/PerchJorate Letter Requests as of September 12,2003

Regional ISSUE DUE REVISED RECEIVED RESPONSE TRACKING MMRP


BUSINESS NAME Board DATE DATE DUE DATE DATE DESCRIPTION REVIEWED NUMBER Inventory
SotirceArea
Dixon Naval Reserve Training Facifity 5 6/11/2003 913012003 Characterization Report NONE

Source Area
Mather Air Force Base 5 6/1112003 9130/2003 Characterization Report NONE
Source Area
McClellan Air Force Base 5 6111/2003 913012003 Characterization Report NONE

Riverbank AAP 5 611112003 913012003 Source Area Evaluation Repar NONE


Source Area
Sacramento Army Depot 5 6111/2003 913012003 Characterization Report NONE
Source Area
Slockton NCS 5 611112003 913012003 Characterization Report NONE

EdwardsAFB 6
Azusa Dump Site 8

EI Toro MCAS 8 6/12/2003 913012003 Source Area· Evaluation Repor NONE

Los Alamitos Armed Forces Reserve Center 8 6/12/2003 913012003 Source Area Evaluation Repor NONE

March Air Force Base 8 9111/2003 913012003 Source Area Evaluation Repor NONE

March Air Reserve Base 8 6/12/2003 9130/2003 Source Area Evaluation Repor NONE

Norton Air Force Base 8 6/12/2003 9130/2003 Source Area Evaluation Repor NONE
Rialto Ammo Storage 8

Seal Beach Naval Weapons Station 8 6112/2003 913012003 Source Area Evaluation Repor NONE
Borrego Springs 9
Coronado NAVPHIBASE 9 612012003 1013012003 Source Evaluation Report NONE
Imperial Beach Naval Auxiliary Landing Field 9 6/2012003 10130/2003 S!>urce Evaluation Report NONE
MCAS Miramar 9 6120/2003 1013012003 Source Evaluation Report NONE
North ISland Naval Air Station 9 612012003 1013012003 Source Evaluation Report NONE
Poinl Loma Naval Complex SPAWAR - PLC 9 612012003 1013012003 Source Evaluation Report NONE
San Diego FASWTC PAC 9 6/2012003 1013012003 Source Evaluation Report NONE
San Diego FISC (NSC) 9 6/20/2003 10130/2003 Source Evaluation Report NONE
San Diego MCRD 9 612012003 1013012003 Source Evaluation Report NONE
San Diego Naval Station 9 6120/2003 10130/2003 Source Evaluation Report NONE
San Diego NAVSUBASE 9 6120/2003 10130/2003 Source Evaluation Report NONE
San Diego NCTS 9 6/2012003 10130/2003 Source Evaluation Report NONE
San Diego NISE-West (NOCCSC Old Town Campus)(former1y AF PIt#19) 9 612012003 1013012003 Source Evaluation Report NONE
San Diego NTC (BRACIII or DERA?) 9 6120/2003 1013012003 Source Evaluation Report NONE
Fallbrook Naval Weapons Station 9 612012003 1013012003 Source Evaluation Report NONE
Warner Springs SERE camp 9 6120/2003 1013012003 Source Evaluation Report NONE
CALIFORNIA AIR NATIONAL GUARD ?? 6/2712003 912712003 Source Evaluation Report. 2199.9154

DoD Sites WIth RB Ltrs xIs 2


DRAFT Emergent ChemicaUPerchlorate Letter Requests as of September 12, 2003

DOD identified Facilities

uIUUIIUft'G,er,.-:I1 e IlL. 0 expusure paulvvay urowluwa ernot


usedas a drinking water source

.....u uun, v.uU.~•••• ,


site B, SLC4 in TCE plume Soil. tn Groundwater, maximum
, detected Perchlorate was 517 ug/L
IUIUUnUWiUer IQ ppu. nu exposure pi:lmway. Impa\;lCIU wens
taken out of service. RO provides all dnnking water.

Perchlorate max detect 492 uglL in gw at site 16, 08100


monitoring well (000 Charter Mtg)

DoD Sites Vo1th RB Ltrs.x:ls 3


J?RAIT Emergent ChemicallPerchlorate Letter Requests as of September 12,2003

DOD identified Facilities

site, $0 we are perlonmng environmental cleanup under the


CERCLA process WIth State and Federal regulator overslte We
have a groundwater plume under Mather as a result of
AeroJeVBoemg past practice

GW and Soil: Rocket Research 300 ppb deteded in GW plume.


Ion exchange perchlorate is t:urrently being demonstrated. North
Base is contaminated by perchlorate and organic solvents.
Detected in soil (max detect 36,000 ug/L In plume from jet

Groundwater 380 ppb

Drinking water >4 ppb to 811 ppb

DoD Sites with RB Ltrs.xls 4


Page 1 of 1

(b)(6)
-... --- ­

From: Kratz, Kurt, , OSD-ATL


Sent: Friday, December 06,20024:15 PM
To: Woodley Jr., John, Mr, OSD-ATL
Cc: Cornell, Jeff, Lt. Col., SAFIIE; Choudhury, Shah Mr OSD-ATL; Cotter, Sandra, Ms, OSD-ATL
Subject: FW: Risk Assessment Relevant Documents
Attachments: Chapter 3.pdf; Executive Summary.pdf

Sir,

The information that Dan Rogers promised during the last perchlorate brief.

Kurt

-----Original Message----­
From: Rogers, Daniel, Lt Col, AFLSA/PR

Sent: Wednesday, December 04, 2002 1:11 PM

To: Kratz, Kurt, , OSD-ATL; Choudhury, Shah, IVlr, OSD-ATL

Subject: Risk Assessment Relevant Documents

It took me a little while to find the most relevant portions of the risk assessment that
speaks to EPA's methodology for determining the lowest levels but t,he executive
summary and chapter 3 tell the story.

vIr

Lt Col Dan Rogers


Air Force Office of Professional Responsibility
AFLSAIPR
112 Luke Avenue, SuitemD
Bolling AFB, DC 20332
DSN:rtftWJW Fax: DSN:fiftWJ_
Commercial: rmtii Fax:flrnlft~~rI1l'..I:1l __•
EXECUTIVE SUMMARY
2
3
4 The purposes of this document is to present an assessment that updates previous
5 provisional values issued by the U.S. Environmental Protection Agency (EPA) for an oral
6 reference dose (RID) for perchlorate and revises the assessment previously released as a draft
7 external review document (U.S. Environmental Protection Agency, 1998d). The objective of this
8 assessment is to derive a human health risk estimate, based on an evaluation of its potential to
9 cause toxicity or cancer, and to provide a screening-level ecological risk assessment for
10 perchlorate based on all toxicity data that recently have become available to the Agency as of fall
11 2001. Another important objective was to evaluate the evidence for indirect exposures, Le.,
12 those exposures not by direct ingestion of contaminated water. This revised assessment
13 incorporates data from new studies and analyses in response-level to recommendations made at a
14 previous peer review of the 1998 draft (Research Triangle Institute, 1999). Most of these data
15 were obtained as results of a testing strategy that was designed with knowledge of the mode of
16 action for perchlorate toxicity that identified major data gaps in the data available prior to 1997.
17 This executive summary concisely presents key findings from the present assessment.
18

19 SUMMARY FINDINGS
20 Sources of Perchlorate Contamination and Occurrence
21 • Perchlorate is an oxidizing anion that originates as a contaminant in ground and surface waters
22 from the dissolution of ammonium, potassium, magnesium, or sodium salts. Perchlorate is
23 exceedingly mobile in aqueous systems and can persist for many decades under typical ground
24 and surface water conditions.
25 • Ammonium perchlorate is manufactured for use as the oxidizer component and primary
26 ingredient in solid propellant for rockets, missiles, and fireworks. Because it is a reducing
27 agent, it can undergo a variety of intramolecular redox reactions that lead to the release of
28 gaseous products. Through such reactions, it acts as a thrust booster.
29 • Perchlorate salts are also used on a large scale as a component of air bag inflators. Perchlorate
30 salts are also used in nuclear reactors and electronic tubes, as additives in lubricating oils, in

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tanning and finishing leather, as a mordant for fabrics and dyes, in electroplating, in aluminum
2 refining, and in rubber manufacture, as a mordant for fabrics and dyes, and in the production of
3 paints and enamels. Chemical fertilizer had been reported to be a potential source of
4 perchlorate contamination, but new investigations by the Agency have determined that this is
5 not an issue for agricultural applications.
6 • Large-scale production of perchlorate-containing chemicals in the United States began in the
7 mid-1940s. Because of its shelflife, perchlorate must be washed out of the United States'
8 missile and rocket inventory to be replaced with a fresh supply. Thus, large volumes have been
9 disposed of in various states since the 1950s.
10 • Perchlorate began to be discovered at various manufacturing sites and in well water and
11 drinking water supplies within the months following the April 1997 development of an ion
12 chromatography analytical method that achieved a method detection limit (MDL) of
13 approximately 1 ppb and a minimum reporting limit (MRL) of 4 ppb. There are 20 states with
14 confirmed releases in ground or surface water. There are 40 states that have confirmed
15 perchlorate manufacturers or users based on BPA Information Request responses.
16 In California, most of the locations where perchlorate has been detected are associated with
17 facilities that have manufactured or tested solid rocket fuels for the Department of Defense or
18 the National Aeronautics and Space Administration.
19 • To date, there has not been a systematic national survey of perchlorate occurrence and a
20 National'Primary Drinking Water Regulation for perchlorate does not currently exist.
21 Perchlorate was placed on the Contaminant Candidate List (CCL) in March 1998. The CCL
22 lists priority contaminants (defined as either known or anticipated to occur in public water
23 systems) in need of research, guidance development, regulatory determinations, or monitoring
24 by the states. Perchlorate was listed as a contaminant that required additional research and
25 occurrence information before regulatory determinations could be considered.
26 • Perchlorate was placed on the Unregulated Contaminants Monitoring Rule (UCMR) in March
27 1999 (Federal Register, 1999) to gather needed exposure information. Under the UCMR, all
28 large public water systems and a representative sample of small public water systems were
29 required to monitor for perchlorate beginning in January 2001. This effort does not extend to
30 investigating potential sources in ground and surface water that have not migrated into public
31 water supplies. Identification of the magnitude and extent of perchlorate occurrence in the

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environment is important in assessing the routes of exposure to humans and for determining the
2 different types of organisms and ecosystems that may be affected.
3 • In early 2000, an analytical method to detect perchlorate in drinking water (EPA Method 314.0)
4 using ion chromatography was published as a direct final rule (Federal Register, 2000). The
5 EPA Method 314.0 was also approved as a monitoring method for the UCMR (Federal
6 Resister, 2000). The MDL for the method is 0.53 ppb and the MRL is 4 ppb. Improvements
7 developed commercially in the analytical capabilities may lower the MRL to the sub-part per
8 billion level in the near future.
9 • Adequate exposure characteristics of transport and transformation in the environment are also
10 absent. Preliminary biotransport studies at six contaminated sites indicate a potential for
II uptake into plant and animal tissues in ecosystems. Extension of analytical methods to detect
12 perchlorate in plant and animal tissues awaits validation before a conclusive determination can
13 be made.
14
15 An Integrated Approach to Comprehensive Risk Characterization
16 • Perchlorate is of concern for several reasons. First, there were uncertainties in the toxicological
17 database available that could be used to evaluate the potential for perchlorate to produce human
18 health effects when present at low levels in drinking water. The purpose of the targeted
19 toxicity testing strategy was to develop a database to address key data gaps. Secondly, the
20 actual extent of the occurrence of perchlorate in ground and surface waters is not known at this
21 time. Additionally, the efficacy of different treatment technologies for various water uses (such
22 as drinking water or agricultural applications) and different scales (i.e., large or small volumes)
23 is still being determined. Finally, the extent and nature of ecological impact or transport and
24 transformation phenomena in various environmental media have only, as yet, been studied
25 superficially.
26 • To adequately and comprehensively characterize the risks posed by perchlorate contamination
27 and to develop scientifically';'based management strategies that effectively mitigate the potential
28 risks posed by perchlorate contamination, several advances are essential. The analytical
29 methods used to characterize various exposures must be accurate and precise. The exposure
30 estimates cannot be gauged with respect to their risk unless robust health and ecological risk
31 estimates are available. Treatment technologies should be targeted to levels of concern and

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tailored to the intended water use. Technology transfer is necessary so that all affected parties
2 and concerned citizens are apprised of accurate and reliable information that is up to date with
3 the evolving state of the science.
4 • The toxicity testing strategy was expedited through a unique partnership between the
5 Department of Defense and EPA, together with members of an Interagency Perchlorate
6 Steering Committee (IPSC), which includes other governmental representatives from the
7 National Institute for Environmental Health Sciences (NIEHS) and affected state, tribal, and
8 local governments.
9 • The charge of the IPSC is to facilitate and coordinate accurate accounts of related technological
10 issues (occurrence surveys, health assessment, ecotoxicology assessment, treatability, waste
11 stream handling, and analytical detection). This assessment is intended to address the need for
12 evaluation of perchlorate's potential to cause human health effects or impact on ecological
13 systems, based on currently available data.
14
15 Physicochemical Characteristics
16 • As an oxidant, perchlorate is kinetically nonlabile. This means the reduction of the central
17 chlorine atom from an oxidation state of +7 (perchlorate) to -1 (chloride ion) occurs extremely
18 slowly. Sorption is not expected to attenuate perchlorate because it absorbs weakly to most soil
19 minerals. Natural chemical reduction in the environment is not expected to be significant.
20 These two factors account for perchlorate being both very mobile in aqueous systems and
21 persistent for many decades under typical ground and surface water conditions.
22 • The activation energy to perchlorate reduction is so high that it cannot be expected to act as an
23 oxidant under human physiological conditions (Le., dilute solution, unelevated temperatures,
24 neutral pH). This is supported by absorption, distribution, metabolism, and elimination studies
25 that show perchlorate is excreted virtually unchanged in the urine after absorption.
26
27 Hazard Identification and Mode of Action Testing Strategy
28 • The health effects and toxicity database available in the spring of 1997 was determined to be
29 inadequate for quantitative risk assessment by an independent (non-EPA) peer review. A
30 testing strategy was developed based on a hazard identification using the available data and the
31 suspected mode of action for perchlorate to target testing on potential effects of perchlorate.

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Data from this effort was used to support the previous EPA draft assessment and this revised
2 assessment in 2002.
3 • To design a testing strategy based on the mode of action for a chemical, it is necessary to
4 understand its toxicokinetics and toxicodynamics. Perchlorate is readily absorbed from the
5 intestinal tract, and oral uptake is considered to be the major route of exposure. Because of its
6 high charge, perchlorate does not pass readily through the skin. Exposure via inhalation is
7 expected to be negligible because the vapor pressure of perchlorate salts and acids is expected
8 to be low at room temperatures. Droplet size during showering likely would preclude
9 inhalation of perchlorate-contaminated water as an aerosol. Perchlorate is known to inhibit the
10 uptake of iodide in the thyroid at the sodium (Na+)-iodide (J-) symporter, or NIS, thereby
11 causing a reduction in the hormones thyroxine (T4) and triiodothyronine (T3). When these
12 hormones enter the blood circulation, they are bound to plasma proteins. There may be other
13 locations of inhibition of iodide transport in the gland. Perchlorate itself is not metabol ized in
14 the thyroid or peripheral tissues.
15 • Control of circulating concentrations of these hormones is regulated primarily by a negative
]6 feedback known as the hypothalamic-pituitary-thyroid axis or feedback system involving three
17 organs: (1) the thyroid, which produces T4 and T3; (2) the pituitary gland which produces
18 TSH; and (3) the hypothalamus, which also responds to and helps to maintain optimal T4 and
19 T3 levels. The hypothalamus stimulates the pituitary gland through thyrotrophic-releasing
20 hormone (TRH) to produce thyroid stimulating hormone (TSH), which then prompts the
21 thyroid to produce T4 and T3. Cells in the hypothalamus and pituitary gland respond to the
22 levels of circulating T4 and T3, such that when thyroid production levels are low, there is a
23 signal to increase the output ofTRH and TSH. Circulating hormone levels (T4, T3, and TSH)
24 can be monitored readily to serve as biomarkers of exposure and effect of agents that disrupt
25 the status of this negative feedback system.
26 • The hypothalamic-pituitary-thyroid feedback system for regulation of thyroid hormones is
27 conserved across species. Differences in plasma protein binding between rats and humans
28 account for differences in the circulating half-life ofthe hormones and in thyroid response to
29 TSH between the species. New studies since 1999 have confirmed that the inhibition of iodide
30 uptake by perchlorate at the NIS is essentially the same sensitivity across species. This is

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important when considering decrements in T4 as important to neurodevelopmental effects
2 versus neoplasia that results in the gland due to stimulation by TSH.
3 • Given its mode of action as an inhibitor of iodide uptake that results in disturbances of the
4 hypothalamic-pituitary-thyroid axis, concerns arose about the potential for perchlorate to cause
5 carcinogenic, neurodevelopmental, developmental, reproductive, and immunotoxic effects.
6 Further, there is concern for ecotoxicology effects on various aquatic and terrestrial plants and
7 animals.
8 • The human health testing strategy for perchlorate developed in 1997 originally included eight
9 different recommended studies to address data gaps and enhance the mechanistic information
10 on the mode of action. The goal of these studies was to provide a comprehensive database on
II which to arrive at a revised human health risk assessment with greater confidence than previous
12 recommended provisional values. These studies are described briefly below.
13 (1) A 90-day oral bioassay to identify other target tissues in young adult rats; to provide data
14 on the effects of repeated exposures to perchlorate on T3, T4, and TSH levels; to
15 evaluate recovery of effects after 30 days; and to screen for some reproductive
16 parameters. A genotoxicity assay also was performed on rats from the terminal sacrifice.
17 (2) A neurodevelopmental study in rats to evaluate the potential for functional and
18 morphological effects in offspring from the mother exposed during pregnancy and
19 lactation.
/
20 (3) A Segment II developmental study in rabbits to evaluate the potential for perchlorate to
21 cause birth defects and to provide data on thyroid hormone effects in a second species
22 other than the rat.
23 (4) A two-generation reproductive toxicity study to evaluate the potential for perchlorate to
24 cause deficits in reproductive performance in adult rats and for toxicity in the young
25 offspring.
26 (5) Absorption, distribution, metabolism, and elimination (ADME) studies to characterize
27 the pharmacokinetics of perchlorate in laboratory animals and humans and to provide
28 data necessary to allow construction of models for quantitative description of different
29 internal dose metrics and interspecies extrapolation.

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(6) Mechanistic studies that characterize the effects of perchlorate on the iodide uptake
2 mechanism across species as a link with the ADME studies to aid in the quantitative
3 extrapolation of dose across species.
4 (7) A battery of genotoxicity assays to evaluate the potential for carcinogenicity by
5 evaluating the potential for direct effects on deoxyribonucleic acid (DNA).
6 (8) Immunotoxicity studies to evaluate the potential for perchlorate to disrupt immune
7 function, including cell-mediated and humoral toxicity.
8 • After the External Peer Review in 1999, additional studies were performed to replicate the
9 neurodevelopmental study (Le., changes in brain morphometry and motor activity); determine
10 the developmental toxicity potential in rats versus rabbits; investigate additional aspects of
11 immunotoxicity; and develop a consistent nomenclature and scoring system for the
12 histopathological lesions in the thyroid gland. Additional pharmacokinetic data was also
13 developed into physiologically-based pharmacokinetic (PBPK) models of perchlorate and
14 iodide distribution.
15 • A battery of eco logical screening tests as part ofthe 1997 testing strategy was conducted as
16 part of the 1997 testing strategy in laboratory organisms representative of ecological receptors
17 across soil, sediment, and water to evaluate dose-response relationships. These were
18 considered to be a tier of tests to give an idea of gross toxicity that would determine the need
19 and types of tests to be performed in the next tier. The tests did not measure the amount of
20 perchlorate in the tissues of the species being tested. Based on stakeholder input and the need
21 for a more focused battery of tests, lettuce was substituted for duckweed because of Tribal
22 concerns regarding the sizable lettuce crop along the Colorado river. The following species
23 were selected for the first round of testing:
24 (1) Daphnia magna (water flea) to represent an aquatic invertebrate
25 (2) Ceriodaphnia magna (water flea) to represent an aquatic invertebrate
26 (3) Lactuca sativa (lettuce) to represent a vascular plant
27 (4) Pimephales promelas (fathead minnow) to represent an aquatic invertebrate
28 (5) Eiseniafoetida (earthworm) to represent a soil invertebrate
29 (6) Microtus pennsylvanicus (meadow vole) to represent an herbivore
30 • Other studies in the set of tests included the Frog Embryo Teratogenesis Assay: Xenopus
31 (FETAX) and a phytoremediation study to examine uptake, distribution, and degradation in

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experimental systems with rooted cuttings of woody plants, including willow, Eastern
2 Cottonwood, and eucalyptus.
3 • Additional studies, some of chronic duration, on effect levels in aquatic animals, an aquatic
4 plant, a terrestrial plant, and a soil invertebrate have been performed since 1999. A study of
5 perchlorate occurrence in six selected sites with known or suspected contamination also
6 examined perchlorate concentrations in site media and in various ecological receptors.
7
8 Human Health Assessment
9 • The testing strategy confirmed that the target tissue for perchlorate toxicity was the thyroid
10 gland. Anti-thyroid effects included iodide uptake inhibition, perturbations ofT3, T4, and TSH
II hormones, and thyroid histopathology in adult, fetal, and postnatal rats across studies with a
12 range of experimental design. Thyroid weight in these studies was also effected. Other than
13 effects in the thyroid, no effects were observed in rabbits of the developmental study, but the
14 developmental study in rats identified 30 mg/kg-day as the lowest observed adverse effect level
15 (LOAEL).
16 • Competitive inhibition of iodide uptake at the NIS by perchlorate is the key event leading to
I7 both potential neurodevelopmental and neoplastic sequelae. The decrement in iodide uptake
18 leads to subsequent drops in T4 and T3 that can lead to permanent neurodevelopmental
19 deficits. Because of strong correlations between changes in iodide uptake inhibition with
20 decrements in T3 and T4; between T3 and T4 with changes in TSH; and between changes in
21 T3, T4, or TSH with thyroid histopathology, an assessment model was proposed that used the
22 changes in T3, T4, and TSH as the precursor lesions to subsequent effects that potentially could
23 lead to thyroid tumors or to altered neurodevelopment. This assessment approach essentially
24 harmonizes noncancer and cancer approaches because it is presumed that the no-observed­
25 adverse-effect-level (NOAEL) for the precursor lesions will preclude any subsequent sequelae
26 at higher doses.
27 • Thyroid tumors were observed in previous studies in rats exposed in long-term bioassays at
28 high doses. Thyroid tumors were more recently also diagnosed in the first-generation (FI)
29 adults (second parental generation [P2]) at 19 weeks in a two-generation reproductive study.
30 Both the latency and incidence of these tumors were significant relative to the entirety ofthe
31 National Toxicology Program data base for this type of tumor and in this strain ofrat. These

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effects and the demostration of a progression with duration of effects on hormones and thyroid
2 histopathology in the 90-day study raised the concern that extended exposures to perchlorate
3 may change the hypothalamic-pituitary-feedback system or the cellular sensitivity and demand
4 for thyroid hormones.
5 • The rat model is considered relevant yet conservative for human health risk assessment of
6 potential thyroid neoplasia because of the differences in thyroid structure and hormone
7 half-lives. Perchlorate was demonstrated to be nongenotoxic in the testing battery employed,
8 suggesting the antithyroid effects are an indirect mode of action for thyroid tumor formation.
9 • Due to the age- and time-dependent nature of the key event of perchlorate toxicity and its
10 anti-thyroid effects, the revised RID was based on weight-of-the-evidence approach to the
11 entire data base. The RID is proposed to be protective of both neurodevelopmental and
12 neoplastic sequelae. An administered dose of 0.01 mg/kg-day was supported as a lowest­
13 observed-adverse-effect level (LOAEL) based on effects on brain morphometry in pups from a
14 PND21 sacrifice in a neurodevelopmental study that repeated similar observations made in a
15 similar 1998 study, hormonal effects indicative of hypothyroidism (decreased T4 and increased
16 TSH) in the dams of those same pups on GD21, thyroid histopathology and hormone changes
17 in these same pups at various developmental stages (GD21, PND4, PND9, and PND21),
18 thyroid histopathology and hormone changes at the 14- and 90-day sacrifice dates in a
19 subchronic study, and indications of immunotoxicity (dermal contact hypersensitivity).
20 • A human equivalent exposure (HEE) was calculated using physiologically-based
21 pharmacokinetic (PBPK) models for interspecies adjustment based on the area under the curve
22 (AVC) of perchlorate in the serum as the dose metric. The HEE for the maternal dams was
23 chosen for operational derivation because brain morphometry effects may have been
24 programmed in utero and because the dams of effected pups were hypothyroid.
25 • A composite uncertainty factor of 300 was used to address uncertainties in the extrapolations
26 required for the RID derivation. A three-fold factor for intraspecies variability was retained
27 due to the variability observed in the data and PBPK modeling for the adult humans and
28 because the subjects used to develop the models did not provide kinetic data for the potentially
29 susceptible population. There was also uncertainty in the parallelogram approach to extending
30 the adult structures to predict doses for different life stages in the human. A full factor often
31 was applied to extrapolate the LOAEL for the adverse effects (brain morphometry, colloid

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depletion and hormone changes) observed in various studies at the 0.0 I mglkg-day dosage
2 level. A three-fold factor for duration was applied due to the concern for the biological
3 importance of the statistically significant increase in tumors observed in the F I-generation pups
4 (second parental, P2 generation) at 19 weeks and the evidence for progression of effects with
5 extended exposure in the 90-day study. The finding of tumors at 19 weeks raised concern for
6 in utero programming, i.e., that disruption of thyroid hormones in the developing fetus may
7 predispose the developing neonate and adult to future insults to the thyroid gland. This factor
8 can also be viewed as part of a data base deficiency since there are no adequate long-term
9 bioassays of perchlorate. Finally, a three-fold factor was applied for inaccurate characterization
10 of immunotoxicity since recent studies reinforced concern for this potential endpoint. Be\cause
II the test article was ammonium perchlorate, an adjustment factor of 0.85 was made for the
12 percent of molecular weight of the salt from ammonium (15.35%), so that the RID is expressed
13 for perchlorate as the anion alone. This was done to be compatible with the analytical methods
14 that measure the anion in environmental samples and because most perchlorate salts readily
15 dissolve in water. The resultant revised RID value for perchlorate is 0.00003 mglkg-day.
16 Confidence in the principal study, the data base and the RID were all designated as medium.
17
18 Screening Ecological Risk Assessment
19 • A secondary acute value of 5 mglL (as perchlorate) was derived to be protective of95% of
20 aquatic organisms during short-term exposures with 80% confidence. The secondary chronic
21 value of 0.6 (as perchlorate) likewise was derived to be protective of95% of aquatic organisms
22 during short-term exposures with 80% confidence. These values were derived based on
23 sodium perchlorate and are probably protective even if ammonium perchlorate is the
24 contaminant released. Calculated ammonia-nitrogen concentrations corresponding to those
25 values are below the acute and chronic ambient water quality criteria for ammonia, regardless
26 of pH.
27 • For terrestrial plants, the quartile inhibitory concentrations for growth in soil and sand were
28 78 mglkg (293 mg/L) and 41 mglkg (160 mg/L), respectively. A factor of 10 was applied to
29 account for interspecies variance to obtain a screening benchmark of 4 mg/kg.

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• Because of limited data on effects for soil invertebrates, a conservative estimate of a threshold
2 for soil community effects was derived at I mglkg. The equivalent aqueous phase benchmark
3 is 2.8 mglL.
4 • A factor of 10 for interspecies variance and LOAEL to NOAEL extrapolation was applied to
5 the human health risk LOAEL estimate based on rat data (0.01 mglkg-day) to obtain a
6 screening benchmark of 0.001 mg/kg-day for the representative herbivore (meadow vole)
7 because it also is a rodent. The population-level implications of this effect are unknown, but it
8 seems likely that such effects on the thyroid could diminish survivorship and fecundity, which
9 would diminish population production.
10 • Data are available showing that perchlorate accumulates in the tissues of exposed fish,
11 amphibians, and invertebrates. However, data are insufficient to determine whether perchlorate
12 is concentrated in those tissues to levels exceeding the levels of exposure. By contrast, several
13 studies have shown that perchlorate is taken up and concentrated in aerial plant parts, especially
14 leaves, although studies designed for the purpose of quantifying plant concentration factors
15 have not yet been conducted.
16
17 Uncertainties and Assessment Research Needs
18 • Accurate exposure information is a requisite for risk characterization for both human and
19 ecological assessments. These data should include transport and transformation processes,
20 notably the fate of perchlorate in irrigated soils because of the potential for evaporative
21 concentration.
22 • Research concerning the human health risks of perchlorate needs to better characterize the
23 dose-response for perchlorate inhibition of iodide uptake in adults, fetuses, and neonates. More
24 definitive studies linking iodide uptake inhibition and the degree of perturbation of the
25 hypothalamic-pituitary-thyroid axis (i.e., changes in T3, T4, and TSH levels) and association
26 with neurobehavioral problems, thyroid changes, and neoplastic sequelae may continue to
27 improve the confidence in the assessment. Understanding the relative sensitivity of laboratory
28 animal assays of neurodevelopmental effects versus epidemiological studies of
29 neuropsychological development also needs to be advanced. Research on potential factors
30 influencing sensitivity is critically requisite. Animal models of thyroid impairment such as
31 iodide deficiency and "womb to tomb" exposure designs should be explored.

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• Because only a screening tier of tests has been performed, the major uncertainty derives from
2 data gaps. Data on bioaccumulation in aquatic biota would allow evaluation of exposure of
3 organisms that feed on fish and other aquatic organisms. Effects of perchlorate on algae and
4 aquatic macrophytes are required to estimate risks to aquatic primary producers. Data on
5 bioaccummulation in aquatic plants are necessary to assess direct impact to primary consumers
6 (Le., planktonic and benthic invertebrate communitie's). Data on accumulation in terrestrial
7 vascular plants also should be investigated further. The factor applied for the use of subchronic
8 data in fish could be addressed by chronic effect testing. Effects also should be determined in
9 nondaphnid invertebrates and of dietary exposure in birds and herbivorous or litter-feeding
10 invertebrates.
11
12 Risk Characterization
13 • As noted above, the lack of exposure information precludes comparison with the human health
14 and ecological toxicity assessment for accurate characterization of risk. Indirect human
15 exposure pathways can be addressed best by a new EPA document, Methodology for Assessing
16 Health Risks Associated with Multiple Pathway of Exposure to Combustor Emissions, which is
17 scheduled for final release in January 2002.
18 • Noncancer neurobehavioral effects have been shown at lower doses. The estimate for
19 perchlorate has been based on precursor effects considered protective for both the thyroid
20 neoplasia and neurodevelopmental effects. It is appropriate for comparison against direct oral
21 exposures. The frequency and magnitude of exposure are key attributes for characterization
22 compared with those assumptions of continuous lifetime exposure assumed in the derivation.
23 The degree to which the particular suspected population at risk fits with the assumptions used
24 in the RID derivation should be kept in mind when performing any risk characterization.
25 Further, RID estimates are not intended to serve as a "bright line" because, by definition, there
26 is an order-of-magnitude uncertainty around the estimate. This typically translates into a range
27 of threefold below to threefold above the RID.
28 • Ecological risk could not be precluded nor accurately characterized because ofthe significant
29 data gaps described above.
30

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3. TOXICOKINETICS/TOXICODYNAMICS AND
2 MODE-OF-ACTION TESTING STRATEGY
3
4
5 This chapter explains the rationale that was the basis of the testing strategy which was
6 designed to evaluate the potential critical targets for perchlorate and to establish a database
7 robust enough to support a quantitative risk assessment. Aspects of the toxicokinetics and
8 toxicodynamics of perchlorate and its interaction with the thyroid are discussed as the basis for
9 the development of a testing strategy based on the mode of action of perchlorate. Mode ofaction
to is defined as a chemical's influence on molecular, cellular, and physiological functions (Federal
II Register, 1996; Wiltse and Dellarco, 1996). Understanding the mode of action helps to interpret
12 the relevancy of the laboratory animal and human data to inform the most appropriate
13 dose-response procedure (see Chapter 7).
14 As discussed in Chapter 2, perchlorate salts dissolve readily in water. The resultant anion
15 is easily absorbed from the gastrointestinal tract. However, because of its high charge, neither
16 perchlorate, nor other electrolytes applied from aqueous solution or aqueous media penetrate the
17 skin readily (Scheuplein and Bronaugh, 1983). Uptake of inorganic ions such as perchlorate
18 through the skin is typically less than 10% and frequently less than 1%. Exposure via inhalation
19 of fumes or vapors is considered negligible because the vapor pressure of perchlorate salts and
20 acids is low at room temperatures. The risk from exposure to particles would depend on the
21 particle size distribution. Thus, the ingestion route is the major concern for the risk posed by the
22 perchlorate contamination and is the focus of this characterization.
23
24

25 3.1 ABSORPTION, DISTRIBUTION, METABOLISM, AND


26 ELIMINATION OF PERCHLORATE
27 Limited absorption, distribution, metabolism, and elimination (ADME) studies were in
28 existence prior to the testing strategy discussed in Section 3.5. Although experimental studies in
29 laboratory species and humans had been perfo~med using radiolabeling techniques, most were at
30 high concentrations, and the published data were expressed simply as thyroid:blood ratios of

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radioactivity counts that provided no information on internal dose to biological tissues. Oral
2 drinking water administration, the most relevant to the contamination issue, was not the norm.
3 Time-course studies were very limited and essentially nonexistent for repeated administration.
4 More importantly, no data existed on the co-administration of iodide (I") and perchlorate even
5 though data were necessary to develop a physiologically based pharmacokinetic model (Fisher,
6 1998a). The following section describes the limited pharmacokinetic information that was
7 considered when the data gap was highlighted during the development of protocols for the testing
8 strategy. The development of physiologically-based pharmacokinetic models that describe
9 ADME for perchlorate with data from the testing strategy will be discussed in Chapter 6.
10 Perchlorate appears to be eliminated rapidly, primarily in the urine (>90%), and virtually
11 unchanged from both rats (Eichler and Hackenthal, 1962) and humans (Anbar et ai., 1959).
12 Durand (1938) measured urinary elimination from two human subjects who ingested 794 mg of
13 sodium perchlorate in 100 g of water. Urinary elimination accounted for 50% of the
14 administered dose within 5 hr and 95% within 48 hr. Half-lives have been reported for the rat
15 ranging from <8 hr (95% in 60 hr) to "'20 hr (Wolff, 1998). Stanbury and Wyngaarden (1952)
16 reported that perchlorate appears in the urine within 10 to 15 min of oral dosing and that peak
17 plasma levels occur within 3 hr. Perchlorate was reported to undergo a two-phased urinary
18 elimination process in rats and calves. In rats, the first phase accounted for approximately 96% .
19 of the administered dose and had a half-life of 1 to 2 hr. The second phase accounted for 4% and
20 had a half-life that ranged from 72 to 80 h. In calves, the first-phase half-life was reported to be
21 2 to 2.5 hr, and the second 23 to 27 hr (Selivanova et aI., 1986, as cited in Allred, 1998). The
22 kinetics of long-term administration of perchlorate have not been characterized. The distribution
23 and metabolism of perchlorate and its relevance to potential toxicity in the thyroid will be
24 discussed in greater detail in Section 3.3 following discussions of iodine metabolism and thyroid
25 physiology in Section 3.2.
26

27 3.1.1 Human Studies


28 The majority of the human data on perchlorate ADME prior to the strategy was comprised
29 of the therapeutic case and clinical studies of Graves' disease patients described in Section 4.2.2.
30 These studies established the effect of perchlorate on the sodium (Na+)-iodide (I') symporter
31 (NIS) but were of limited use in establishing quantitative dose-response relationships.

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Anbar et al. (1959) demonstrated that perchlorate was not metabolized in humans, Four
2 patients were administered 200 mg (approximately 2.9 mglkg using a default body weight of
3 70 kg) double-labeled K36C11804' and urine was collected 3 h after dosing. Perchlorate was found
4 to be excreted at approximately 200 ,ug/min in the urine. Total urine radioactivity was
5 distributed between 36Cl , 36CI1804", 36Cl04" and 36Cl' and indicated that perchlorate was excreted
6 unchanged in the urine. No human data existed with which to adequately characterize the
7 pharmacokinetics of perchlorate during steady-state, low-dose, repeated administration.
8

9 3.1.2 Laboratory Animal Studies


10 Although the perchlorate discharge test has been performed in rats (Atterwill et aI., 1987),
II the procedure is very different than that used in humans and does not readily allow for
12 comparison or extrapolation. Rats are dosed intraperitoneally (ip) with 100 ,uL (1 ,uCi) 1251", then
13 dosed ip with potassium perchlorate at 5, 10, 25, or 50 mglkg body weight from 1 to 6 h
14 afterwards. Results are expressed as thyroid:blood ratios, which differ from how most human
15 data are expressed. Additionally, the time points at which uptake is measured are highly
16 dissimilar to those used in human studies.
17 Anbar et al. (1959) also attempted to confirm the lack of perchlorate accumulation and lack
18 of metabolism in the thyroid in rats. White rats were injected ip with 36KCl04, and the specific
19 activity per gram of tissue was measured at 30 min, 4 hr, and 12 hr. The activity was greatest in
20 the thyroid and peaked at 4 h. The salivary and adrenal glands also had high activity levels.
21 Rabbits also were tested; the thyroid activity levels were again the highest of any tissue and
22 peaked at 2 h. Rabbit testes had the next highest specific activities.
23 In one of the only co-administration studies, Anbar et al. (1959) simultaneously
24 administered 1311" and 36Cl04' in equimolar concentrations. The thyroid:blood specific activity for
25 iodide was slightly higher than the ratio for perchlorate (1.80 and 1.69, respectively).
26 Halmi et al. (1956) examined iodide uptake in male Sprague-Dawley rats when active
27 transport was completely blocked via sodium perchlorate. The rats were first administered 6 mg
28 of propylthiouracil (PTU) subcutaneously to prevent iodide organification. Iodide uptake was
29 prevented by administration of 100, 200, or 400 mg sodium perchlorate with half of each dose
30 administered along with the PTU and the other half administered 45 min later with 5 to 50 ,uCi
31 1311". The rats were sacrificed 1.0 to 1.5 h after the iodide administration. Perchlorate reduced the

January 16, 2002 3-3 DRAFT-DO NOT QUOTE OR CITE


thyroid:blood ratio from 22.7 to 0.45; radioiodide was found to account for 30% of the thyroid
2 gland volume when it entered the gland by diffusion alone. Rats sacrificed 4.0 to 4.5 h after
3 iodide administration produced similar results, indicating that equilibrium is reached prior to
4 1.0 to 1.5 h. The distribution of radioiodide in other tissues also was examined. Perchlorate did
5 not affect the organ:serum iodide ratios in the following organs: submaxillary salivary gland,
6 parotid salivary gland, pituitary gland, adrenal glands, testes, spleen, kidneys, lung, skin, or
7 diaphragm. However, perchlorate administration did affect the stomach wall:serum and gastric
8 juice:serum iodide ratios (0.36 and 0.75, respectively) compared with the ratios for controls
9 administered sodium chloride (1.45 and 15.8, respectively). This suggested a gastric iodide
10 pump subject to inhibition by perchlorate and, as will be discussed in Chapter 6, the
11 gastrointestinal tract is another tissue with NIS.
12 Goldman and Stanbury (1973) administered 0.1 ,uCi of the potassium salt of 36CI-labeled
13 perchlorate (K 36 CI0 ) by ip injection to male Sprague-Dawley rats that had been maintained on a
4

14 low-iodine diet for 4.5 to 5.0 weeks prior to dosing (approximately 40,ug stable perchlorate per
15 injection). The radionucleide retention in the thyroid, expressed as percent of dose per gram of
16 tissue, was recorded at 2 h (6 rats), 4 h (5 rats), 8 h (6 rats), 24 h (6 rats), 48 h (6 rats), and 96 h
17 (5 rats). The peak was reported to appear around 4 h and then to fall to approximately 5% of this
18 peak value after approximately 96 h. An exponential function was used to estimate a half-life of
19 20 h. Urinary excretion data indicated that the disappearance rate from the plasma and thyroid
20 and the appearance rate in the urine corresponded closely although the question was raised as to
21 whether there is some curvilinearity to the urinary excretion, which may suggest limited
22 saturation. The retained dose and its standard deviation in tissues at 96 h were reported as
23 0.142 ± 0.1, 0.125 ± 0.09, 0.098 ± 0.03, 0.048 ± 0.04, and background for the thyroid, kidney,
24 spleen, liver, and brain, respectively.
25 Chow et a!. (1969) compared the uptake of radiolabeled perchlorate and iodide ions with
26 stable ions in normal and thyroid-impaired rodents. Intact male Sprague-Dawley rats were
27 injected ip with 0.1, 0.2, or 5.0 meqlkg stable potassium perchlorate (14, 28, or 690 mglkg,
28 e
respectively) 2 h prior to sacrifice. The specific activity of the chlorine label 6Cn was
29 25.2,uCi/mmo!. Thyroid impairment was affected by pretreatment with thyroid-stimulating
30 hormone (TSH) (1 international unit [IV] TSH in 0.9% saline solution ip 18 h prior to
31 perchlorate administration), hypophysectomization (removal of the pituitary), TSH and

January 16,2.002 3-4 DRAFT-DO NOT QUOTE OR CITE


hypophysectomization, or PTU (0.1 % PTU in drinking water for 2 weeks prior to perchlorate
2 administration). Perchlorate at the 0.1- and 0.2-meq/kg dose levels was found to preferentially
3 concentrate in the'rat thyroid as compared to the plasma, and the concentration was related
4 inversely to dose. The high dose level did not result in the concentration of radio labeled
5 perchlorate in the thyroid. Rats pretreated with TSH or PTU also concentrated perchlorate at the
6 lower dose levels. At the two lower levels, hypophysectomized rats were not able to concentrate
7 perchlorate compared with intact rats, but the thyroid perchlorate concentration at the high dose
8 level did not differ between intact and altered rats. In a second subset of the same study, rats
9 were exposed to 0.005, 0.01, 0.02, 0.05, or 0.10 meq/kg perchlorate (0.69, 1.4,2.8,6.9, or
10 ]4 mg/kg, respectively) under the same general conditions. The concentration of radiolabeled
11 perchlorate in the thyroid again was related inversely to perchlorate dose. Male albino guinea
12 pigs also were exposed to the same doses. The guinea pigs displayed the same relationships as
13 the rats, but concentrated more perchlorate in the thyroid compared to plasma levels.
14 Chow and Woodbury (1970) demonstrated that perchlorate is actively sequestered by the
15 thyroid gland at low doses but that the capacity of the symporter to actively sequester perchlorate
16 is exceeded at higher doses. Male Sprague-Dawley rats were functionally nephrectomized by
17 ligating the renal pedicle of both kidneys 24 h before the rats were sacrificed. PerChlorate was
18 administered as the radio labeled potassium salt (K36Cl04) in solution by ip injection at 0.005,
19 0.1, or 2.0 mmollkg stable potassium perchlorate (0.69, 14, and 280 mg/kg body weight,
20 respectively, assuming 0.266 kg body weight; actual weight 226 ± 4 g) 2 to 240 min before
21 sacrifice. A group of control rats received [14C]-insulin, 35S04·2 or 36CI" 2 h prior to sacrifice to
22 determine thyroid follicle volume and intrafollicular membrane potential. Concentrations of
23 perchlorate in the thyroid and plasma were measured at 0.033, 0.067, 0.13,0.2,0.50, 1.0,2.0,
24 and 4.0 h after sacrifice. Again, perchlorate was actively sequestered by the thyroid gland at the
25 low dose, but the capacity of the symporter to actively sequester perchlorate was exceeded at the
26 higher doses (e.g., the thyroid:plasma [milligrams per gram:milligrams per liter] ratios at 15 min
27 or 4 h post-dosing were 6.4, 0.69, and 0.36 or 13.8,0.93, and 0.44 at the 0.5, 14.0, or
28 280.0 mg/kg doses, respectively). These data suggest that maximal inhibition by perchlorate of
29 active uptake of iodide probably occurs below 14 mg/kg potassium perchlorate (l 0.0 mg/kg as
30 perchlorate). Ifperchlorate-induced inhibition of active iodide uptake is substantial, iodide still
31 may enter. the thyroid by diffusion, but in a smaller amount. Likewise, if inhibition of iodide

January 16, 2002 3-5 DRAFT-DO NOT QUOTE OR CITE


uptake by perchlorate is incomplete, then iodide still may be actively sequestered into the thyroid,
2 again in a smaller amount. Thus, perchlorate-induced thyroid hormone perturbations may
3 plateau in adult rats dosed with perchlorate greater than approximately 5 to 10 mglkg of
4 perchlorate (Fisher, 1998a).
5 Wolff and Maurey (1962) demonstrated the competitive nature of the perchlorate inhibition
6 in sheep thyroid tissue slices incubated at 37°C for 100 min. This study showed that the
7 Km constants for anion accumulation and the K; constants for inhibition of accumulation were
8 identical within the error of the method.
9 Eichler and Hackenthal (1962) presented perchlorate elimination data for male and female
10 Wistar rats dosed subcutaneously with 0.2, 1.0, or 6.0 of the 36Cl- sodium perchlorate salt
11 (Na36 CI04) per 100 g body weight (2, 10, or 60 mglkg). The elimination curves showed nearly
12 linear, rapid excretion of perchlorate until 6 hr, at which time the curve slope started to decrease.
13 The rate of excretion increased with dose. The elimination rates of the different doses prior to
14 24 h were significantly different from each other but were similar after 24 h. Over 60 hr, 93.4 to
15 97.4% of the administered dose was recovered, again suggesting that perchlorate was not
16 metabolized.
17 In a recent review (Von Burg, 1995), perchlorate elimination curves in rats and calves were
18 described as biphasic in both species. For rats, 96% of administered perchlorate is eliminated
19 with a half-life of I to 2 hr. The second portion ofthe curve accounts for 4% of the dose, with
20 half-life of 72 to 80 hr. Calves have a faster overall rate of elimination, but the initial elimination
21 is slower. The first-phase half-life was 2.0 to 2.5 hr, and the second-phase half-life ranged from
22 23 to 27 hr.
23 An intravenous (iv) study performed at AFRL/HEST in Sprague-Dawley rats with
24 perchlorate to characterize its inhibition of iodide uptake supports the conclusion that there is
25 inhibition at low concentrations and there is a gradual plateau at higher concentrations (Meyer,
26 1998). Rats were dosed once by iv tail-vein injection with either 0.01,0.1, 1.0, or 3.0 mg/kg of
27 cold (i.e., not radiolabeled) ammonium perchlorate in saline. Perchlorate was administered as
28 ammonium perchlorate, and the data are presented as milligrams perchlorate per kilogram body
29 weight. Two hours after dosing with perchlorate, the rats were dosed again by iv tail-vein
125
30 injection with 33 .uglkg 1 dissolved in saline. Rats were sacrificed at selected times (n == 6 per
125
31 time point) up to 24 h. Total and free 1were measured in serum, thyroid, and urine.

January 16, 2002 3-6 DRAFT-DO NOT QUOTE OR CITE


Perchlorate serum, thyroid, tissue, and urine analyses began in January 1999 and are reported in
2 Chapter 6. For control comparison, rats were dosed once by iv tail-vein injection with 33 ,ug/kg
3 nonradiolabeled iodide and 125 1 mixed in physiologic saline. Rats (n = 6) were sacrificed at the
4 same selected time points up to 24 hr.
5 Table 3-1 shows the percent of inhibition of 1251uptake as measured by bound 125 1 in the
6 thyroid. Inhibition of 125 1uptake into the thyroid by perchlorate was measured by bound or free
7 1251 in the thyroid at various time points after the singl"e-dose of perchlorate. Because the 125 1was

8 administered 2 hr after dosing with ammonium perchlorate, these time points correspond to 4, 8,
9 and 11 h after dosing. The most profound inhibitory effects were found at the 1.0- and 3.0-mg
10 perchlorate/kg dose group; however, the trend for 1251 inhibition is evident at the 0.01- and
11 O.I-mg/kg levels (Meyer, 1998). By 24 h (26 h after dosing with perchlorate), inhibitory effects
12 on 125 1uptake were still observed at the 1.0- and 3.0-mg/kg dose groups.
13 Recovery of 125 1 in urine 24 hr after dosing with 125 1(26 h after ammonium perchlorate) was
14 between 79 and 88% for control 1251-dosed rats and perchlorate-dosed rats. The control125l-dosed
15 rats excreted 79.5% (SO ± 5.50) of the 125 1dose over the 24-hr period; whereas, the perchlorate­
16 dosed rats excreted 87% (SO ± 7.84),86% (SO ± 4.47),87.8 (SO ± 20.20) and 79.3 (SO ± 10.58)
17 of the 125} dose in urine at the 0.01-, 0.1-, 1.0-, and 3.0-mg/kg dose levels, respectively. The
18 amount of 1251 in serum was elevated in the perchlorate-dosed animals compared to the control
19 125}_dosed rats for up to 6 hr in all dose groups, suggesting that thyroid function was altered by
20 perchlorate and that a transient "discharge" of organified 125} occurred as reported in studies
21 summarized in Chapter 3. Free 125 1 levels in serum were similar between perchlorate-dosed and
22 control 1251-dosed rats (Meyer, 1998). These results are consistent with those of Chow et at.
23 (1969) and Chow and Woodbury (1970). The pattern for the inhibition of iodide uptake, albeit
24 only after a single dose, is strikingly similar to the patterns shown for the thyroid hormone
25 decreases. Consequently, data on the species differences (i.e., rat versus human in particular) in
26 perchlorate inhibition of the symporter will provide a basis for evaluating the degree of
27 uncertainty that should be applied when utilizing laboratory animal data as the model for humans
28 (see Chapter 7).
29 Repeated dose studies in rats (Fisher, 1998a) and in humans (Channel, I998a) to establish
30 the kinetics of perchlorate at steady-state performed by AFRL/HEST to further characterize the
31 inhibition of iodide uptake by perchlorate are discussed in Chapter 6.

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TABLE 3-1. PERCENT INHIBITION OF IODIDE UPTAKE IN THE

THYROID GLAND OF SD RATS DOSED WITH PERCHLORATE (Meyer, 1998)

Dose [Iodide] Percentage of


Time Points8 (mg perchlorate/kg) (,ug/g) Inhibition
2 hr Control b 24.4
0.01 21.3 13
0.1 18.6 24
1 7.4 70
3 2.99 88
b
6 hr Control 46.5
0.01 36.7 21
0.1 32.0 31
19.2 59
3 9.13 80
9 hr Controlb 55
0.01 49.2 11
0.1 39.2 29
1 24.7 55
3 10.0 82

125
"Time points correspond to dosing with 1 and to 4, 6, and 11 hr after dosing with ammonium perchlorate.
bOosed with only iodide (33 J.lglkg).

3.2 IODINE METABOLISM AND THYROID PHYSIOLOGY


2 Iodine plays a central role in thyroid physiology as both a constituent ofthyroid hormones
3 and a regulator of thyroid gland function. Like perchlorate, iodine is absorbed efficiently from
4 the gastrointestinal tract. Iodine in organic form is converted mostly to iodide before absorption
5 (Cavalieri, 1997). The kidneys account for about two-thirds of the iodide cleared from plasma
6 and more than 90% of the iodide cleared from the body. Sweat and breast milk account for
7 various fractions of iodide loss, and fecal elimination constitutes approximately 1% of total body
8 iodide clearance.

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1 The thyroid gland concentrates iodide against an electrochemical gradient by a carrier-
2 mediated mechanism driven by adenosine triphosphate (ATP). The activation energy required
3 for perchlorate reduction is so high that it cannot act as an oxidant under physiological conditions
4 (i.e., dilute solution, moderate temperatures, and neutral pH). Plasma membrane experiments
5 indicate that the sodium cation (Na+) and iodide cotransport are electrogenic, with a
6 thermodynamically downhill transport of approximately two Na+ ions driving one iodide ion
7 against its electrochemical gradient into the cell. The transport is sensitive to ouabain, an
8 inhibitor of ATPase. The molecule responsible for the transport of iodide has been named the
9 sodium (Nat)/iodide (I) symporter or NIS. The thyroid thus has a specialized ability to
10 concentrate iodide selectively from the surroundings where the concentration is very low (10-8 to
11 10-7 M) and where the concentration of chloride ions is in the order of 0.0 1 to 0.1 M. The
12 transport is "active," not only by electrochemical criteria, but also by metabolic ones: it does not
13 occur in the cold, it requires oxygen, and, as mentiOrled, it is a function of the ATP level.
14 In addition to the thyroid, other organs that can concentrate iodide include the salivary glands,
15 gastric mucosa, choroid plexus, mammary glands, and the placenta. Iodide secreted into the
16 saliva and gastric juice is reabsorbed in the small intestine (Cavalieri, 1997).
17 Nevertheless, it is essentially only in the thyroid that the newly concentrated iodide can be
18 metabolized further to form thyroid hormone; and, only in the thyroid, does TSH regulate the
19 process. Thyroid hormones play numerous and profound roles in regulating metabolism, growth,
20 development, and maintenance of homeostasis. It is generally thought that these actions result
21 from the effects of the thyroid hormones on protein synthesis (Hill et aI., 1989).
22 Figure 3-1 shows a schematic representation of thyroid hormone biosynthesis and secretion
23 in a single thyroid follicular cell. The thyroid hormones are stored as amino acid residues in
24 thyroglobulin (Tg), a protein constituting most of the colloid in the thyroid follicles. In situ, the
25 follicular cell displays functional and structural polarity: the vascular space is at the bottom, and
26 the lumen of the follicle is at the top. The striated circle straddling the basolateral membrane
27 represents the iodide transporter. The process of thyroid hormone biosynthesis is first stimulated
28 by TSH binding to the follicular cell TSH receptor and cyclic adenosine monophosphate (cAMP)
29 activation (Hard, 1998). The protein portion ofTg is synthesized on rough endoplasmic
30 reticulum (ER), and carbohydrate moieties are added by the Golgi apparatus (GA).
31 Thyroglobulin proceeds to the apical surface in secretory vesicles (small open circles) that

January 16,2002 3-9 DRAFT-DO NOT QUOTE OR CITE


Follicular Lumen
(Colloid)
Apical
Surface

Thyroid
Follicular
Cell

Basolateral
Membrane

Extracellular
Space
MIT

Figure 3·1. Schematic representation of thyroid hormone biosynthesis and secretion in a


single thyroid follicular cell. (Modified from Hill et aI., 1989; Cavalieri, 1997;
and Fisher, 1996.)

fuse with the cell membrane and discharge their contents into the follicular lumen. Iodide enters
2 the cell by active transport, and then, at the apical surface, is oxidized by thyroid peroxidase
3 (TPO). The hydrogen-peroxide-generating system is represented by hydrogen peroxide (HP2)'
4 Organification occurs at or near this apical cell-colloid interface; the oxidized iodide is
5 incorporated into tyrosine residues in peptide linkage in Tg. Two iodinated tyrosyl groups couple
6 in ether linkage to form tetraiodothyronine (T4), which initially remains trapped in Tg. Hormone
7 secretion first involves pinocytosis of colloid-containing iodinated Tg (large open circle) at the
8 apical border of the follicular lumen and resolution into vesicles that fuse with Iysosomes (LY,
9 dark circle). Lysosome proteolysis (striated circle) then converts Tg to amino acids, T4,

January 16,2002 3-10 DRAFT-DO NOT QUOTE OR CITE


triiodothyronine (T3), diiodotyrosine (DIT) and monoiodotyrosine (MIT). Iodotryosine
2 dehalogenase regenerates iodide from MIT and DIT for reuse within the thyroid or release into
3 the blood, accounting for the iodide leak in the chronic state of iodine excess in certain thyroid
4 disorders. Type I iodothyronine deiodinase converts a fraction of the free T4 to T3. Both
5 hormones (T4 and T3) are released into the blood circulation by a process that is not well
6 understood. The thyroid also releases Tg, of which some is iodinated and some uniodinated as
7 newly synthesized protein.
8 Although T4 is by far the major hormone secreted by the thyroid (typically at 8 to 10 times
9 the rate ofT3), T4 is considered a prohormone because about 33% of the T4 secreted undergoes
10 5'-deiodination to T3 in the peripheral tissues and T3 is about fourfold more potent than T4.
11 Another 40% undergoes deiodination of the inner ring to yield the inactive material, reverse
12 triiodothyronine (rT3), which recently has been postulated to play an inhibitory role on the
13 conversion ofT4 to T3. T3 is regarded as the active hormone because it is the form that appears
14 to activate a response by nuclear DNA. Upon entering the circulation, both T4 and T3 are bound
15 and transported in strong, but not covalent, association with plasma proteins.
16 The major plasma-protein carrier in humans is thyroxine-binding globulin, a glycoprotein
17 with a very high affinity for T4 and a lower affinity for T3. In rats, the T4 and T3 are bound to
18 prealbumin (PA) or albumin with a weaker attachment. Control of the circulating concentrations
19 of these hormones is regulated primarily by a negative feedback involving three organs: (1) the
20 thyroid, which produces thyroid hormone, and (2) the pituitary gland and (3) hypothalamus,
21 which respond to and help maintain optimal T3 and T4 levels (Hill et al., 1998). Figure 3-2
22 shows the schematic for this hypothalamic-pituitary-axis and the feedback mechanisms.
23 The hypothalamus stimulates the pituitary gland through thyrotropin-releasing hormone
24 (TRH) to produce TSH, which prompts the thyroid to produce T4 and T3. Once secreted into the
25 blood, T4 and T3 are bound to plasma proteins (thyroid-binding globulin [TBG] in humans or
26 prealbumin [PA] and albumin in rats). In addition to the aforementioned conversion ofT4 to T3
27 in peripheral tissues, thyroid hormone also is metabolized irreversibly in the liver by uridine
28 diphosphyl glucuronosyl transferases (UDPGTs) to either glucuronic (T4) or sulfate (mainly T3)
29 conjugates that are excreted in bile. A portion of the conjugated material is hydrolyzed in the
30 intestine, and the free hormones thus released are reabsorbed into the blood via enterohepatic
31 circulation. The remaining portion of the conjugated material is excreted in the feces.

January 16, 2002 3-11 DRAFT-DO NOT QUOTE OR CITE


~ +- Influences from Periphery
{~.<

.
via Nervous System

i
( •) - - - - - -.....'"'1I Hypothalamus
(Paraventricular Nuclei)
I
I
ITRHI
(+)

(.) I - - - - - - . . . . - . t Pituitary
(Thyrotroph)

I TSHI (+)

Thyroid

Thyroglobulin Plasma/Blood
~~~peroxidase ,. --MIT ...
f1;- • 12+TyrOSine-.DIT;+T4,T3/*"-....j~T4,T3 "';SAG, PP-TH
II \----- Deiodination---~~__ --""" [S dt]
_ _ _
~=============~
.... ..............-_- J ___
~~~mao
Proteins

Proteolysis
r Trapping Organic Binding-coupling T4, T3 Release

ThiOCyanate)
(+)
Thiourea, PTIJ ) Iodide-)
( Perchlorate 5 ulfonam ides ( Excess

( Methimazole

Aminotrazole

1
Liver
Certain Tissues Target Tissue
UD+GTs
T4-+T3
(Nuclear Receptor)
T4-GLUC

~
Bilary
Excretion

Figure 3-2. Schematic of the hypothalamic-pituitary-thyroid axis and feedback


mechanisms (PP-TH = plasma protein-thyroid hormone, PTU =
propylthiouracil, UDPGT = uridine diphosphyl glucuronosyl transferase,
T4 GLUC = T4-glucuronide conjugate). (Modified from U.S. Environmental
Protection Agency, 1998a; Hill et aI., 1998; and Capen, 1997).

January 16, 2002 DRAFT-DO NOT QUOTE OR CITE


- ------ -------------

Cells in the hypothalamus and pituitary gland respond to levels of circulating T4 and T3
2 such that when thyroid production levels are high, there is a signal to reduce the output of (TRH)
3 and TSH. Similarly, when thyroid hormone levels are low, the pituitary is prompted to deliver
4 more TSH to the thyroid in order to increase the output ofT4 and T3. This negative feedback
5 loop helps the body respond to varying demands for thyroid honnone and to maintain hormone
6 homeostasis. Thus circulating T4, T3, and TSH are monitored readily in experimental animals
7 and humans and so may serve as biomarkers of exposUre to and indicators of the effects from
8 agents that disrupt the status of the hypothalamic-pituitary-thyroid axis (U.S. Environmental
9 Protection Agency, 1998a).
l O I n the absence ofthyroid-binding globulin, as in the rat and mouse, a greater fraction of
11 thyroid honnone is free of protein binding and subject to metabolism and removal from the body.
12 As a consequence, the half-life ofT4 in the rat is only about 1 to 24 hr, in contrast to the 6 to
13 7 day half-life in humans. Rats compensate for the increased turnover rate by secreting more
14 TSH from the pituitary gland. Table 3-2 provides the interspecies and intraspecies differences in
15 both thyroid hormone and gland structure between rats and humans. The consequences of
16 disrupting the status of the hypothalamic-pituitary-axis will be discussed in Section 3.4.
17
18

19 3.3 TOXICOKINETICS OF PERCHLORATE


20 Because of the complex anatomy of the thyroid follicle, all of the locations where
21 perchlorate inhibition is exerted remain to be established (Wolff, 1998). Perchlorate has been
22 established as a competitive inhibitor of iodide uptake across the basolateral membrane (i.e., acts
23 by the inhibition at NIS). Figure 3-3 shows a comparison of the molecular dimensions of
24 perchlorate and iodide. The following potency series was constructed for monovalent anion­
25 based inhibition of iodide transport in thyroid slices: Tc04' ~Cl04" > Re0 4" > SCN" > BF4' > r>
26 N0 3" > Br' > Cl" (Wolff, 1998). However, it is not clear whether this anion sequence, measured
27 at very high concentrations, has any mechanistic relation to what occurs at low concentrations in
28 the thyroid. It is important to determine which solution properties ofthe anions determine this
29 sequence (e.g., crystal radius, hydrated radius, hydration enthalpy, charge density). Strong base
30 anion-exchange resins (usually a large cation with a weak field) exhibit a marked preference for
31 Cl04"(e.g., compared to Cn; thus, it seems likely that selectivity for iodide or perchlorate in the

January 16,2002 3-13 DRAFT-DO NOT QUOTE OR CITE


TABLE 3-2. INTERSPECIES AND INTRASPECIES DIFFERENCES IN

THYROID STRUCTURE AND T3, T4, AND TSH HORMONES

(U.S. Environmental Protection Agency, 1998a)

Parameter Human Rat


Thyroxine-binding globulin Present Essentially absent
T4 Half-life 5 to 6 Days 0.5 to 1 Day
T3 Half-life 1 Day '0.25 Day
T4 Production rate/kg body weight 1x lOx that in humans

TSH 1x 6 to 60 x that in humans

Follicular cell morphology Low cuboidal Cuboidal


Sex differences
Serum TSH Ma=F a M s2xF
Cancer sensitivity F =2.5 x M M>F

aM = male, F = female.

Perchlorate (CI0 4-):


Iodide (1-):
Max. Dimension:
Max. Dimension:
(xyz) = 5.420
(xyz) == 4.400
angstroms
angstroms

Min. Dimension:
Min. Dimension:

(xyz) = 4.724
(xyz) = 4.400

angstroms
angstroms

Figure 3-3. Comparison of the molecular dimensions for the perchlorate (left) and iodide
(right) anions.

January 16,2002 3-14 DRAFT-DO NOT QUOTE OR CITE


1 thyroid may be based on an anion-exchange mechanism using a large cation such as a quaternary
2 amine (e.g., arginine) (Wolff, 1989).
3 Perchlorate also has been used to stimulate the efflux of iodide already stored in the
4 follicular lumen of the gland (Atterwill et aI., 1987). The exact nature of the mechanism for this
5 effect has not been established, however. Transport of iodide out of the cell is downhill
6 electrically, but this could be accounted for by the high concentration gradient that is established
7 from follicular lumen (iodide stored in the colloid) to the basolateral and extracellular space.
8 This may be the rate-limiting aspect for perchlorate efflux effect. Perchlorate added to the apical
9 side of a polarized thyroid cell monolayer is substantially less effective than when added to the
10 basolateral side (Wolff, 1998). Moreover, perchlorate rapidly increases the secretory response to
11 TSH, and TSH increases iodide efflux before it increases iodide influx, suggesting that additional
12 control points may exist.
13 Thus, perchlorate appears to have no effect on the iodination process itself but, rather,
14 displaces iodide by competitive uptake at the NIS. Perchlorate is concentrated by thyroid tissue
15 in a manner similar to iodide, but it is not significantly metabolized in the gland nor peripherally,
16 as mentioned previously. It is not unequivocally established whether there are additional effects
17 of perchlorate on iodide transport within the thyroid. Pharmacokinetic studies with perchlorate,
18 both acute and particularly once steady state has been achieved, have provided some useful data
19 with which to gain insight on this issue. The potential impacts as health endpoints of interest for
20 human health risk assessment of this perturbation in the hypothalamic-pituitary-thyroid axis and
21 hormone economy will be discussed in Section 3.4.
22
23

24 3.4 TOXICODYNAMICS OF THYROID HORMONE PERTURBATIONS


25 Given the established mode of action for perchlorate as the inhibition of iodide uptake at
26 the NIS, it is important to distinguish the temporal aspects with respect to potential adverse tissue
27 response.
28
29 3.4.1 Carcinogenic Effects
30 In higher organisms, when demands for more thyroid hormone are small, existing thyroid
31 follicular cells can meet the demand. With increased need, as a result of certain chemical

January 16, 2002 3·15 DRAFT-DO NOT QUOTE OR CITE


----------------.-------- ---------

exposures or iodine deficiency, the thyroid responds by increasing the size (hypertrophy) and
2 number (hyperplasia) of thyroid follicular cells to enhance hormone output. With continued TSH
3 stimulation, there is actual enlargement of the thyroid (goiter) and, at least in rodents, eventual
4 neoplasia of the thyroid follicular cells. Because TSH~producing pituitary cells also are
5 stimulated, they too sometimes undergo hyperplasia and neoplasia (U.S. Environmental
6 Protection Agency, 1998a; Hill et aI., 1998). The EPA Assessment of Thyroid Follicular Cell
7 Tumors (U.S. Environmental Protection Agency, 1998a), as well as reviews recommended
8 therein, provides details about thyroid follicular cell carcinogenesis. Figure 3~4 shows
9 schematically the possible antithyroid effects that could influence carcinogenesis. Note that
10 effects, not only in the thyroid but also in peripheral tissues and the liver, may cause demand on
11 thyroid hormone production such that the TSH stimulation of the thyroid to produce more
12 hormone is enlisted. Table 3-3 lists mechanisms of antithyroid~mediatedneoplasia in rodents.
13 The potential for an indirect effect of perchlorate has been established, but genotoxicity
14 information was required to evaluate its potential for direct effects. As will be discussed in
15 Section 3.5, a battery of genetoxicity assays was included in the testing strategy.
16 Long-term perturbations in the hypothalamic-pituitary~thyroid axis by the various
17 influences listed in Table 3-3 are more likely to predispose the laboratory rat to a higher
18 incidence of proliferative lesions (Capen, 1997). One factor that may playa role in this
19 interspecies quantitative difference in sensitivity to thyroid stimulation is the influence of protein
20 carriers of thyroid hormones in the blood (Table 3-2). Both humans and rodents have
21 nonspecific, low-affinity protein carriers of thyroid hormones (e.g., albumin). However, in
22 humans, other primates, and dogs, there is a high-affinity binding protein, thyroxine-binding
23 globulin, which binds T4 (and T3 to a lesser degree). This protein is missing in rodents and
24 lower vertebrates. As previously indicated, T4 is bound to proteins with lower affinity in the
25 rodent and is more susceptible to removal from the blood, by metabolism, and through excretion
26 than in dogs and primates.
27 In keeping with this finding, the serum half-life ofT4 is much shorter in rats (less than
28 1 day) than it is in humans (5 to 9 days); this difference in T4 half-life results in a 10-fold greater
29 requirement for exogenous T4 in the rat with a nonfunctioning thyroid than in the adult human.
30 Serum T3 levels also show a species difference: the half~life in the rat is about 6 hr; whereas, it is
31 about 24 hr in humans. High thyroid hormone synthetic activity is demonstrated in thyroid

January 16, 2,002 3~16 DRAFT-DO NOT QUOTE OR CITE


Thyroid Gland Peripheral Tissue
1. Partial thyroidectomy -TH--+ 7. Inhibition of 5'-mono­
2. Iodine deficency deiodinase
3. Inhibition of iodine pump
4. Inhibition of thyroid peroxidase
5. Toxicity to cells
6. Inhibition of TH release TH
t
TSH TH/
I

Pituitary Gland Liver


9. Transplantation of 8. Enhance TH conjugation
THS-secreting tumor and excretion

Figure 3-4. Schematic of antithyroid effects that influence thyroid carcinogenesis. (V. S.

Environmental Protection Agency, 1998a; and Hill et ai., 1998).

TABLE 3-3. MECHANISMS OF ANTITHYROID-MEDIATED

NEOPLASIA IN RODENTS (V.S. Environmental Protection Agency, 1998a).

• DNA Directed
-X rays
_ 131 1

- Genotoxic chemicals

• Indirect
- Partial thyroidectomy
- Transplantation ofTSH-secreting pituitary tumors
- Iodide deficiency
- Chemicals inhibiting iodide uptake
- Chemicals inhibiting thyroid peroxidase
- Chemicals inhibiting TH
- Chemicals inhibiting conversion ofT3 and T4
- Chemical inhibiting hepatic thyroid hormone metabolism and excretion

January 16, 2002 3-17 DRAFT-DO NOT QUOTE OR CITE


~_._---------------

follicles in rodents, where the follicles are relatively small and are surrounded by cuboidal
2 epithelium. Follicles in primates demonstrate less activity and are large with abundant colloid,
3 and follicular cells are relatively flattened (low cuboidal) (McClain, 1992).
4 The accelerated production of thyroid hormones in the rat is driven by serum TSH levels.
5 that are probably about 6- to 60-fold higher than in humans. This assumes a basal TSH level in
6 rats and humans of200 ng/mL and 5 ,uU/mL, respectively, and a potency of human TSH of 1.5 to
7 15 IU/mg of hormone (U.S. Environmental Protection Agency, 1998a). Thus, it appears that the
8 rodent thyroid gland is chronically stimulated by TSH levels to compensate for the increased
9 turnover ofthyroid hormones. It follows that increases in TSH levels above basal levels in rats
10 could more readily move the gland towards increased growth and potential neoplastic change
11 than in humans. In addition to considerations about the influence of serum thyroid hormone
12 carrier proteins, there are differences between humans and laboratory animals in size and life
13 span and in the pharmacokinetics and pharmacodynamics of endogenous and exogenous
14 chemicals. Any comparison of thyroid carcinogenic responses across species should be
15 cognizant of all these factors.
16 A.number of goitrogenic compounds, those that either interfere with thyroid hormone
17 synthesis or secretion, have been demonstrated to result in thyroid follicular cell adenomas in
18 rats. Excessive secretion ofTSH alone has been reported to produce a high incidence of thyroid
19 follicular cell adenomas. The pathogenic mechanism ofthyroid follicular cell tumor
20 development in rodents involves a sustained excessive stimulation ofthe thyroid by TSH. In the
21 multistage model of this pathogenesis, the proliferative lesions often begin as hyperplasia, may
22 proceed to the development of benign tumor (adenomas), and infrequently develop into
23 malignant tumors (Figure 3-5).
24 The precise molecular steps in the carcinogenic process leading to thyroid follicular cell
25 cancer have not been elucidated totally although significant insights into the problem have been
26 described (Farid et aI., 1994; Said et aI., 1994). Normal cell division in the thyroid seems to be
27 affected by an interplay among several mitogenic factors, namely TSH, insulin-like growth
28 factor-l (IGF-l), insulin, epidermal growth factor (EGF), and possibly fibroblast growth factor
29 (FGF). Additionally, other factors, such as transforming growth factor p, certain interferons, and
30 interleukin 1, may inhibit growth.
31

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- - - - _ .. _-~--_ .. ~ - - - - - - - - -

Morphologic Continuum II ~

Normal

Hyperplasia .1

IAdenoma I
I Carcinoma
Significance in Risk Assessment

Figure 3-5. Proliferative changes involved in the multistage characterization of thyroid


follicular cell neoplasia in rodents represent a morphologic continuum.
Although these lesions typically are classified as discrete entities, the overlap in
morphologic features should be emphasized because only imprecise criteria to
separate borderline proliferative lesions exist. Thyroid neoplasia in rodents is
considered relevant to human risk assessment (U.S. Environmental Protection
Agency, 1998a) but thought to be protective (Capen, 1997).

1 Figure 3-6 shows the possible molecular events in human thyroid follicular carcinogenesis.
2 In spite of the potential qualitative similarities, there is evidence that humans may not be as
3 sensitive quantitatively to thyroid cancer development from thyroid-pituitary disruption as are
4 rodents. Rodents readily respond to reduced iodide intake with the development of cancer;
5 whereas, humans develop profound hyperplasia with "adenomatous" changes with only
6 suggestive evidence of malignancy. Even with congenital goiters from inherited blocks in
7 thyroid hormone production, only a few malignancies have been found in humans. Thus, despite
8 a common physiology in regard to the thyroid-pituitary feedback system, the role of disruption of
9 this axis in human cancer development is much less convincing. EPA has adopted the following
10 science

January 16, 2002 3-19 DRAFT-DO NOT QUOTE OR CITE


I Thyroid follicular cells

TSH, insulin, IGF-1, EGF, FGF


~Ir

I Nodular hyperplasia

! ras, gsp Chromosome aberrations


of 5, 7, and 12

Follicular adenoma I
! Loss of heterozygosity at 3p

Follicular carcinoma I
p53
, r

Anaplastic carcinoma

Figure 3-6. Possible molecular events in human thyroid follicular carcinogenesis (ras = ras
protooncogene, gsp = GTP-binding protein mutation, p53 = p53 tumor
suppressor gene) ( U.S. Environmental Protection Agency, 1998a and Hill
et aI., 1998).

policy that recognizes the role of mode-of-action information regarding thyroid-pituitary


2 disruption and mutagenesis to potential thyroid carcinogenesis (U.S. Environmental Protection
3 Agency, 1998a).
4 • It is presumed that chemicals that produce rodent thyroid tumors may pose a carcinogenic
5 hazard for the human thyroid.
6 • In the absence of chemical-specific data, humans and rodents are presumed to be equally
7 sensitive to thyroid cancer caused by thyroid-pituitary disruption. This is a conservative
8 position when thyroid-pituitary disruption is the sole mode of action because rodents appear to
9 be more sensitive to this carcinogenic mode-of-action than are humans. When the thyroid
10 carcinogen is a mutagenic chemical, the possibility that children may be more sensitive than
II adults needs to be evaluated on a case-by-case basis.

January 16,2002 3-20 DRAFT-DO NOT QUOTE OR CITE


• Adverse rodent noncancer thyroid effects (e.g., thyroid enlargements) follOWIng short- and
2 long-term reductions in thyroid hormone levels are presumed to pose human noncancer health
3 hazards.
4 The new data on the antithyroid activity of perchlorate that has resulted from the testing
5 strategy will be evaluated in Chapter 7 according to criteria provided in the guidance (U.S.
6 Environmental Protection Agency, 1998a) to determine the likelihood that the chemical would
7 act indirectly, via disruption of the thyroid-pituitary axis, or directly on DNA.
8
9 3.4.2 Neurodevelopmental Deficits and Other Potential Adverse Effects
10 Resulting from Thyroid Hormone Disruption
11 As expressed by the external review panel convened by Toxicology Excellence for Risk
12 Assessment (TERA) in 1997, there was concern about other potential adverse effects of
13 perchlorate-induced hypothyroidism. Humans respond as do experimental animals in regard to
14 short- and mid-term disturbances in thyroid functioning from various anti-thyroid stimuli such as
15 iodide deficiency, partial thyroidectomy (surgically or 1311" induced), and goitrogenic chemicals
16 such as thionamides (U.S. Environmental Protection Agency, 1998a). For instance, thyroid
17 hormone is critical to normal brain and physical development. This dependency begins in the
18 uterus and extends to 3 years of age in humans. Thus, there was concern that hypothyroidism
19 during pregnancy could result in neurodevelopmental effects.
20 The role of the placenta in thyroid hormone metabolism is shown in Figure 3-7. Although
21 the fetus is initially dependent on maternal thyroid hormone levels, the potential for disruption of
22 fetal hormone production remains once the fetal thyroid assumes this function because
23 perchlorate can cross the placenta. Disruption of circulating thyroid hormones can have
24 drastically different effects on fetuses and infants than on adults, depending on the developmental
25 stage at exposure (Table 3-4). It is important to emphasize that even transient disruption may
26 lead to permanent effects in the developing organism.
27 Chemical-induced alterations in thyroid hormone homeostasis are known to adversely
28 affect the development of many organ systems, including the nervous and reproductive systems
29 (Porterfield, 1994; Jannini et aI., 1995). Severe developmental hypothyroidism caused by iodine
30 deficiencies or a congenital condition has devastating effects on fetal and postnatal development,
31 including mental deficiencies and hearing, speech, and motor deficits (Porterfield, 1994; Sher

January 16, 2002 3-21 DRAFT-DO NOT QUOTE OR CITE


Mother Placenta Fetus

+TBG
+T4 ....... Estrogens
+T3
+T4 .....
-.... hCG ~

+TSH
Iodine
...
TRH ... TRH
TRH ...

TSH "­
rT3
..... I
T4 ~? rT3
rT3
...
~
T4
....
T3 I\. ~ T2 ...
~ T3
PTU
.
Methimazole ...

CIO4 ­ .

Figure 3-7. Schematic representation of the role of the placenta in thyroid hormone
metabolism during human pregnancy. The placenta produces estrogens and
heG that increase maternal TBG levels and stimulate maternal thyroid
hormone production, respectively. Both activities tend to increase maternal
T4 and T3 concentrations and to inhibit maternal TSH secretion. Iodide and
TRH readily cross the placenta, and the placenta itself synthesizes TRH. The
placenta is impermeable to TSH and only partially permeable to T4 and T3.
Placental Type III iodothyronine monodeiodinase enzymes degrade T4 to rT3
and T3 to 3,3'-diiodothyronine (T2). Propylthiouracil and methimazole
readily cross the placenta. Given its physicochemical characteristics and
similarity to iodide, perchlorate also is anticipated to cross readily. (Modified
from Fisher, 1996 and Underwood, 1998).

January 16, 2002 3-22 DRAFT-DO NOT QUOTE OR CITE


TABLE 3-4. MAIN SYMPTOMS AND EFFECTS OF HYPOTHYROIDISM

Developmental Adult
(Transient disruption leads to permanent effects.) (Transient disruption leads to transient effects.)

• Delayed reflex ontogeny • Run down, slow, depressed


• Impaired fine motor skills • Sluggish, cold, tired
• Deaf-mutism, spasticity • Dryness and brittleness of hair
• Gait disturbances • Dry and itchy skin, constipation
• Mental retardation • Muscle cramps
• Speech impairments • Increased menstrual flow
• Thyroid tumors in rodents

et aI., 1998). It is important to emphasize that these effects are caused by a lack of thyroid
2 hormones alone, rather than by tumor development or thyroid hypertrophylhyperplasia due to
3 increases in TSH. Thus, the important species comparison may be perchlorate's action of iodide
4 uptake inhibition at the NIS. In fact, data discussed in Chapters 5 and 6 show that the sensitivity
5 of the NIS is quite similar across species.
6 During development, thyroid hormones regulate cell proliferation, migration, and
7 differentiation. IntraceHularly, THs bind to thyroid hormone receptors that interact with thyroid
8 response elements to alter expression of messenger ribonucleic acids (mRNAs) and subsequent
9 protein synthesis. The pituitary-thyroid TSH feedback loop mayor may not be activated during
10 development, depending on the mechanism of action of the chemical. The adversity of
I1 congenital hypothyroidism, usually less severe than endemic cretinism, can be ameliorated via
12 early postnatal thyroxine therapy" In contrast, the effects of developmental iodine deficiency can
13 not be corrected with only postnatal therapy, indicating that iodine deficiency during pregnancy
14 is the causative action (Cao et aI., 1994). Clearly, xenobiotics that contribute to fetal or maternal
15 hypothyroidism or hypothyroxenemia are of concern.
16 Since the previous external peer review, studies reported in the clinical and epidemiological
17 literature have reinforced concerns for deficits in neuropsychological 'development related to
18 maternal thyroid deficiency. Haddow et al. (1999) showed an effect on IQ scores in children
19 (ages seven to nine) who had normal thyroid function at birth but were born to women with
20 abnormal thyrotropin levels versus children born to a matched cohort of women with normal
21 thyrotropin levels as controls. Haddow et al. (1999) concluded that even mild and probably

January 16, 2002 3-23 DRAFT-DO NOT QUOTE OR CITE


asymptomatic hypothyroidism in pregnant women can adversely affect their children's
2 subsequent performance on neuropsychological tests.
3 Pop et al. (1995) noted an average impairment of 10.5 IQ points in the offspring of mothers
4 with high thyroid peroxidase antibody (TPO-Ab) titers during pregnancy. In a later prospective
5 study these same researchers evaluated developmental indices at 3 weeks, 10 months, 1 and
6 2 years of age and demonstrated that a maternal free T4 blood level that was less than the loth
7 percentile of first trimester values (10.4 pmol/L in their study series) was associated with
8 distinctly impaired psychomotor development whether or not TSH and TPO-Abs were elevated
9 (Pop, et al., 1999). Smit et al. (2000) reported a similar relationship between free T4 and early
10 neurodevelopment of children born from treated hypothyroid women.
11 Morreale de Escobar et al. (2000) evaluated epidemiological, clinical, and basic research
12 data to ascertain if the principal factor leading to neurodevelopmental deficits in children was
13 related to maternal hypothyroidism, whether clinical or subclinical (as defined by TSH higher
14 than the 98 th percentile of the normal population); or ifthey were instead related to maternal
15 hypothyroxinemia per se (decrement in T4 without concomitant increase in TSH). These
16 researchers concluded that conditions resulting in hypothyroxinemia alone (a low for gestational
17 age circulating maternal free T4 level whether or not TSH was increased) poses an increased risk
18 for poor neuropsychological development of the fetus. T4 is the required substrate for the
19 ontogenically-regulated generation ofT3 in the amounts needed for optimal brain development,
20 both temporally and spatially. Normal maternal 13 concentrations did not seem to prevent the
21 potential damage of a iow T4 supply (Morreale de Escobar et aI., 2000). Hypothyroxinemia
22 seems to be much more frequent in pregnant women than either clinical or subclinical
23 hypothyroidism and autoimmune thyroid disease (AlTO), especially in regions where the iodine
24 intake of the pregnant woman is inadequate to meet her increased needs for T4 (Morreale de
25 Escobar et al., 2000).
26 Figure 3-8 illustrates the windows of susceptibility for insults to the brain resulting from
27 hypothyroxinemia. A similar map has been developed for rats, and time lines have begun to be
28 compared and correlated (Rice and Barone, 2000), as shown in Figure 3-9. Morreale de Escobar
29 et al. (2000) reported findings that altered early migration of cortical cells can be observed in rats
30 with severe iodine deficiency. Porterfield (2000) has also discussed the potential for

January 16, 2002 3-24 DRAFT-DO NOT QUOTE OR CITE


.. ­
iodine-deficiency cretinism
mixed neurological and hypothyroid manifestations

_-1------­ •
neurological

maternal thyroidal autoantibodies congenital hypothyroidism

M.raa@M
cerebral cortex
@tiilii.
ISubarachnoid pathways

o 23456
Gestational age in six months
_.---­ 7 8

Prematurity

Figure 3-8. Approximate timing of major insults to the brain resulting from
hypothyroxinemia, superimposed on major neurodevelopmental events in
humans. Conditions resulting in early maternal hypothyroxinemia, combined
to later impairment of the fetal thyroid, are the most damaging, with central
nervous system (CNS) damage that is irreversible at birth. The most frequent
cause is maternal iodine deficiency (ID) and the presence of maternal
autoimmune thyroid disease (AITD). Unless ID is also present, the CNS
damage in congenital hypothyroidism is preventable by early postnatal
treatment because the normal maternal thyroxinemia has avoided damage to
the brain until birth. If maternal hypothyroxinemia persists, normal maternal
concentrations of T 3 do not protect the fetal brain because of its dependence
on intracerebral regulation of local T3 availability by deiodinating pathways
using T 4 as a substrate. Interruption of the contribution of maternal T 4 in
premature infants with an immature thyroid may also underlie their increased
risk of neurodevelopmental problems, the more severe the earlier their birth.
The question mark indicates that it is unknown whether very early CNS
development, corresponding to a period when the general morphogenesis of
the pros encephalon (neurolation and segmentation) is being determined, is
thyroid hormone sensitive or not (Morreale de Escobar et aI., 2000).

January 16,2002 3-25 DRAFT-DO NOT QUOTE OR CITE


i----·~--·,-----·------------ Birth

r----E-m-b-ry-o-n-i-c--------F-e-ta-I----r Postnatal

I
Functional 'organization

Histogenesis
I
I
i Adolescen.cll.
GO 21·22 PND 15 PND 35-45
Neurulation

Proliferation and migration


!
Differentiation and synaptogeniSiS

Apoptosis I
Gliogenesis
!
i
(A) Myelination

Cell Migration
(B) proliferation of SUbplate Synapse
neurons neurons formation Myelination
0 Radial glia 8rain and Marginal -£l
iii
.r.
1:
1 and neurons spinal cord zone
1
0
2 I
~
0 SUbplate
.§. 3
~
Q)
e?
'C
0
4 ~ +
E .~
'I: 5 Hippocempus
J .2
~ ~
~
CI) E
Q.
II
.,
.l!l
6 Reticular
'"

! ~ ~ +
~ 7
formation c.>
l5
II
I'll
l:
8 Visual .lii
,.,B i "'"'"
l!!
Q. 9
External
granular jrtr ~ .1 ~ j

Gila

I!!
I'll
CI)
~
3
1
2 V
U
Layer
cerebellum
Associatkm
~I
I I

!c:
i
S I~l
1
I~
I
'C
0
'I: 4
cortex
I
8­ 5 '"'15.I!l 'f5
.Ti
~ ~
-
ell
l:
I II
0
Q.
10
'"

Figure 3-9. Timelines of developmental processes in the nervous system of rats (a) and
humans (b). Rat timeline is compared to timing of fertilization, organogenesis,
and histogenesis. Human perinatal period is scaled in months and the
postnatal development is scaled in years (Rice and Barone, 200,0).

January 16,2002 3-26 DRAFT-DO NOT QUOTE OR CITE


neurotoxicity and altered brain development that may result from exposure to environmental
2 chemicals that disrupt thyroid function even on a transient basis.
3 These concerns for the potential adverse effects of perchlorate on T4 and T3, especially
4 during pregnancy, are compounded by the growing appreciation that women of childbearing age
5 have relatively low iodide intake. A January 2001 report by the National Academy of Sciences
6 (NAS). concerning the dietary reference intake of trace-mineral nutrients, including iodine,
7 indicated that less than 25% of the total population was below the estimated average requirement
8 for iodide and stressed a need to look at levels of adequacy for susceptible age groups and status
9 during pregnancy and lactation. The higher requirements during this time indicate a potential
10 susceptibility as shown in Table 3-5. The NAS also cautions against using urinary iodine as a
11 biomarker for iodine status unless the data are from 24-hour collections or are normalized against
12 creatinine. Other reports suggest that the level of iodide intake is less than a third of the range
13 recommended for pregnant women by the World Health Organization (WHO) (Caron et aI.,
14 1997).
15
16
TABLE 3-5. DIETARY REFERENCE INTAKES (DRI) FOR IODIDE
(National Academy of Sciences, 2001)
Estimated Average Recommended Dietary
Adequate Intake (AI) Requirement (EAR) Allowance (RDA)
Age or Status ,ug/day ,ug/day ,ug/day
0-6 months 110
6-12 months 130
1-3 years 65 90
4-8 years 65 90
9-13 years 73 120
14-18 years 95 150
19-15 years 95 150
51 + years 95 150
Pregnancy 160 220
Lactation 209 290

January 16, 2002 3-27 DRAFT-DO NOT QUOTE OR CITE


The prevalence of abnormal thyroid function continues to be debated and this is
2 confounded by the variable definitions of the disease state as well as the different measures of
3 thyroid function (Canaris et aI., 2000). Most reports are still defined by TSH levels rather than
4 for hypothyroxinemia per se, but recent presentations suggest that TSH is a poor test to assess the
5 severity of tissue hypothyroidism (Meier et aI., 2001), and recommendations in the epidemiologic
6 literature are proposing that screening of pregnant women should include the determination of
7 free T4 (Morreale de Escobar et aI., 2000). Age, sex and dietary iodine levels are confounding
8 factors, although virtually all studies report higher prevalence rates for hypothyroidism
9 (as defined by increased TSH) in women with age (Canaris et aI., 2000). Rates as high as 24%
10 among women older than 60 years have been reported. Suppressed TSH levels have been
11 associated with decreased bone density, increased risk of atrial fibrillation, premature atrial beats,
12 and effects on serum lipids notably elevated serum cholesterol levels.
13 Together these findings strongly suggest that a susceptible population of particular concern
14 for perchlorate exposure is pregnant women with hypothyroxinemia and that the iodine
15 deficiency represents an additional potential insult that could exacerbate the effects of perchlorate
16 toxicity. The elderly, especially women, represent another potentially susceptible population, as
17 well as people with cardiac dysfunction or risk factors such as elevated serum cholesterol.
18 As mentioned above, reproductive toxicity was also a concern as a potential effect of
19 perchlorate's mode of action. In females, thyroid hormones appear to have a role in stimulating
20 the onset of human chorionic gonadotropin (hCG) production by the placenta early in pregnancy.
21 Human chorionic gonadotropin is essential for the maintenance of pregnancy. Therefore, a
22 hypothyroid condition has potential to interfere with normal placental function and fetal
23 survival, as well as the potential to interfere with lactation. Suppression of thyroid hormone
24 secretion with radioactive iodine or goitrogens reduces milk yield in lactating animals. This
25 effect may be caused by suppression of placental lactogen production. Thyroid-releasing
26 hormone is known to playa role in prolactin release during the estrous cycle. Additionally, the
27 thyroid is necessary for the transition to the anestrus state in seasonally breeding species.
28 In summary, effects on thyroid hormone levels have roles in estrous cycle regulation, pregnancy
29 maintenance, fetal growth, and lactation.
30 In males, the primary effects of hypothyroidism appear to occur during testicular
31 development. The testis is responsive to thyroid hormones only during a limited time during the

January 16,2002 DRAFT-DO NOT QUOTE OR CITE


perinatal and prepubertal periods. Thyroid hormone is a major regulator of seminiferous
2 epithelium development by inducing the normal differentiation of Sertoli cells, gonocytes, and
3 Leydig cells, and by limiting the proliferation of those cell types. In the hypothyroid condition,
4 those cells proliferate beyond the norm, and the steroidogenic function of the Leydig cells, on a
5 per-cell basis (but not necessarily in total), is impaired. Secretory activity of the Sertoli cells also
6 appears to be impaired. In boys, untreated hypothyroidism is associated with marked and
7 precocious testis enlargement, but low androgen activity. In a small study, hypothyroid men had
8 complaints of reduced libido that was probably related to a defective leutenizing hormone
9 response to gonadotropin-releasing hormone.
10 The inclusion of an immunological evaluation of mice exposed to perchlorate was
II warranted because of evidence from earlier clinical studies that indicated a link between the
12 treatment of Graves' disease with perchlorates and serious hematological effects that may be
13 linked to immune mechanisms. A small number of patients undergoing perchlorate therapy have
14 been reported to develop aplastic anemia, agranulocytosis, lymphadenopathy, leukopenia, or skin
15 rashes. The antithyroid drugs propylthiouracil and methimazoles are reported to exert their
16 effects on the hematopoietIc system through immune mechanisms. Because the use of these
17 antithyroid drugs by a small number of patients also resulted in sequelae similar to that of some
18 patients under perchlorate treatment, it has been postulated that perchlorate also may act via the
19 immune system.
20
21

22 3.5 DEVELOPMENT OF A TOXICITY TESTING STRATEGY BASED


23 ON MODE OF ACTION
24 Because the RID is intended to be a lifetime dose-response estimate, the typical objective
25 of a database to support such a quantitative assessment is to evaluate a comprehensive array of
26 testing endpoints that represent various life stages during which potential effects could occur
27 (e.g., the developing fetus through adult) and for effects on reproductive capability (shown
28 schematically in Figure 3-10). As discussed in the previous sections, thyroid hormone
29 deficiencies, such as those induced by perchlorate, can affect normal metabolism, growth, and
30 development. No robust data existed prior to this time to evaluate other potential target tissues or
31 effects. There were limited data on effects caused by long-term exposures and no data with

January 16,2002 3-29 DRAFT-DO NOT QUOTE OR CITE


Reproductive

Developmental General Toxicity

Figure 3-10. Schematic illustrating that a high confidence RID is based on data that
address all potentially critical stages over a lifetime.

which to evaluate the effects of perchlorate in potentially susceptible populations such as in


2 developing fetuses, nor were there data on the effects of perchlorate on the reproductive capacity
3 of male or female laboratory animals. Table 3-6 shows the minimum database for derivation of
4 an RID with low confidence (a 90-day bioassay) and the rationale for other tests typically
5 included to bolster the confidence in the derivation-the same suite oftests that has been
6 discussed for perchlorate. These data typically also reduce the uncertainty for which uncertainty
7 factors are applied (see Table 3-7), either because the absence of data on a suspected endpoint
8 (e.g., developmental toxicity) has been addressed or because mechanistic data provide insight on
9 the relevance of the laboratory animal model, including the magnitude of interspecies and
10 intrahuman variability in toxicokinetics and toxicodynamics. Any individual chemical database
II may fall in between this range of high and low certainty, depending on the quality of the
12 individual studies and whether the dose response for suspected endpoints is characterized well.
13 The objective ofthe testing strategy was to provide a comprehensive database that
14 described the mode-of-action-based pathogenesis in quantitative terms so that the resultant
15 estimate could be more predictive and ultimately support the development of a robust RID
16 estimate that reduced the uncertainties inherent in the provisional, presumably protective values
17 (see Figure 3-11).
18

January 16, 2002 3-30 DRAFT-DO NOT QUOTE OR CITE


TABLE 3-6. MINIMUM DATABASE FOR DERIVATION OF AN

ORAL REFERENCE DOSE

Mammalian Database" Confidence Comments

Two chronic oral bioassays in different Minimum database for high confidence
species
One two-generation reproductive study
Two developmental toxicity studies in
different species

One subchronic oral bioassay Low Minimum database for estimation of an RID

"Rationale is to use different species to evaluate variability in species sensitivity unless a particular laboratory
animal model is more appropriate.
bRationale is to address all potentially critical life stages.

TABLE 3-7. FACTORS FOR UNCERTAINTIES IN APPLIED EXTRAPOLATIONS


USED TO DERIVE REFERENCE DOSESa
10H - Human to sensitive human
°
1 A - Experimental animal to human
lOs - Subchronic to chronic duration
lOL - LOAEL(HEE)" to NOAEL(HEE)"
10D - Incomplete to complete database
MF - Modifying factor. Professional assessment of scientific uncertainties of the study and database not
explicitly addressed above. Default for the MF is l.0 (e.g., applied for small sample size or poor
exposure characterization).

"HEE = human equivalent exposure.

As illustrated in Figure 3-11, it is ultimately desirable to have a comprehensive


2 biologically-based dose-response model that incorporates the mechanistic determinants of
3 chemical disposition, toxicant-target interactions, and tissue responses integrated into an overall
4 quantitative model of the pathogenesis (Jarabek, 1995a). Because the internal tissue dose of the
5 chemical or its toxic moiety in a target tissue is not always proportional to the applied dose of a
6 compound, emphasis has been placed on the need to distinguish clearly between the exposure
7 concentration and the dose to critical target tissues. Consequently, the term "exposure-dose­
8 response" has been recommended as more accurate and comprehensive (Andersen et aL, 1992).
9 This expression refers, not only to the determination of the quantitative relationship between
10 exposure concentrations and target tissue dose; but also to the relationship between tissue dose
11 and the observed or expected responses in laboratory animals and humans. The process of

January 16, 2002 3-31 DRAFT-DO NOT QUOTE OR CITE


Chemical
Exposure "Dose" Toxicological
Concentration Response

Protective Exposure • -----...... Response Qualitative

Exposure
, Mechanisms
T
Disposition Models
I
..
.1
L-....-Tissue
~
- _......-..... Response

Exposure ... TIssue


Dose
I .. I Toxicant
TIssue
Interaction
Response

, Mechanisms , Meehanisms ,

T T

Disposition Models Toxieant·Target Models

Predictive Exposure

, Mechanisms
• TIssue
Dose I -.. --

, Mechanisms,
Toxicant
TIssue
Interaction
J - ...-.....

, Mechanisms
Response Quantitative

Dlsposlti~n Models Toxicant-Ta~etModels Tissue Resionse Models

Figure 3-11. Schematic characterization of comprehensive exposure-dose-response


continuum and the evolution of protective to predictive dose-response
estimates (U.S. Environmental Protection Agency, 1994 and Jarabek 1995b).

determining the exposure-dose-response continuum is achieved by linking the mechanisms or


2 critical biological factors that regulate the occurrence of a particular process and the nature of the
3 interrelationships among these factors. This can be especially important for interspecies
4 extrapolation and to understanding intrahuman variability.
5 Dose-response estimates based on characterization of the exposure-dose-response
6 continuum at the rudimentary ("black box") level necessarily incorporate large uncertainty
7 factors to ensure that the estimates are protective in the presence of substantial data gaps. With
8 each progressive level, incorporation and integration of mechanistic determinants allow
9 elucidation of the exposure-dose-response continuum and, depending on the knowledge of model
10 parameters and fidelity to the biological system, a more accurate characterization of the
11 pathogenesis process (Jarabek, 1995a). Because of the increase in accuracy ofthe
12 characterization with each progressive level, dose-response estimates also progress from more
13 protective to factually-based (predictive).

January 16, 2002 3-32 DRAFT-DO NOT QUOTE OR CITE


Eight new studies were recommended as part ofthe original testing strategy after the May
2 1997 external peer review to provide such a comprehensive array of endpoints. These studies are
3 described below along with the role they were anticipated to play in informing the revised health
4 risk assessment (see Table 3-8).
5
6
TABLE 3-8. PERCHLORATE PEER REVIEW RECOMMENDED
STUDIES SUMMARY
Study Description Potential Use in Assessment
90-Day subchronic bioassay Tests for other target tissues; Minimum database for RID dose­
+ TH a + reproductivity + evaluates effect on TH in young adult response for TH in young adult rats;
genotoxicity + recovery rats; reproductive parameters added; additional information on others; may
mouse micronuclei and a recovery allow decrease in uncertainty factor
group (UF) for database deficiencies
Developmental Evaluates nervous system in fetal and Potentially critical effect; comparison of
neurotoxicity + TH postnatal rats; TH in does developmental versus adult effects on
(PO-generation) and pups TH
(F I-generation)

Developmental study + TH Evaluates birth defects in rabbits; Potentially critical effect; data in second
TH in does at end of gestation species for TH effects; may reduce UF
for database deficiencies
Two-Generation Evaluates fertility of adult rats and Potentially critical effect; may reduce
reproductive toxicity + TH toxicity in offspring over two UF for database deficiencies
generations; TH in parents
(Fa-generation) and offspring
(FI- and F2-generations)

ADME studies Characterize absorption, distribution, Interspecies extrapolation


metabolism, and elimination in rats
and humans; iodine inhibition and
perchlorate kinetices and hormone
homeostasis
Mechanistic studies Evaluate mechanism of TH response Interspecies extrapolation; determine
and sensitivity in rats and humans susceptible subpopulation
Genotoxicity assays Test for toxicity to DNA Mode-of-action information for thyroid
neoplasia; may reduce UF for database
deficiencies
Immunotoxicity Evaluates immune system structure Potentially critical effect; may reduce
and function UF for database deficiencies

"Thyroid hormones (T4 and T3); Thyroid stimulating hormone (TSH), a pituitary hormone, was also assayed in

those studies.

January 16,2002 3-33 DRAFT-DO NOT QUOTE OR CITE


(1) 90-Day Subchronic Oral Bioassay Study. This study was considered the minimum data
2 requirement for derivation of an oral RID. The study aimed to identify other target tissues,
3 to test young adult rats, and to provide data on the effect of repeated exposure to perchlorate
4 on thyroid hormone levels. The 30-day recovery phase, I.e., evaluation of the thyroid status
5 30 days after perchlorate was stopped, would provide data necessary to characterize its
6 anti-thyroid effects with respect to carcinogenicity (U.S. Environmental Protection Agency,
7 1998a). These data were collected to allow reduction of the uncertainty factor applied for
8 database deficiencies.
9
10 (2) Developmental Neurotoxicity Study. This study was designed to evaluate the potential for
11 developmental neurotoxicity of perchlorate by assessing functional and morphological
12 endpoints in offspring from the mother exposed during pregnancy and lactation.
13 Neurotoxicity endpoints were likely to be a critical effect, and the developing organism a
14 sensitive subpopulation. It was hoped that these data would allow reduction of the
15 uncertainty factors applied for intrahuman variability and database deficiencies.
16
17 (3) Segment II Developmental Study. This study was conducted to evaluate the potential for
18 perchlorate to cause birth defects in rabbits and to evaluate a potentially critical effect and
19 subpopulation. This study also was conducted to provide data on the thyroid hormone
20 effects in a second species (in addition to rats). These data might allow reduction ofthe
21 uncertainty factor applied for database deficiencies.
22
23 (4) Two-Generation Reproductive Toxicity Study. This study was designed to evaluate the
24 potential for perchlorate to cause deficits in reproductive performance in adult rats and for
25 toxicity in the young offspring. The primary goal of this study was to identify a potentially
26 critical effect and to allow for reduction of the uncertainty factor applied for database
27 deficiencies.
28
29 (5) Absorption, Distribution, Metabolism, and Elimination Studies. These ADME studies
30 aimed to understand the pharmacokinetics (I.e., how perchlorate is absorbed, distributed,
31 metabolized, and excreted) of perchlorate in test animals and humans. These data were to

January 16,2002 3-34 DRAFT-DO NOT QUOTE OR CITE


provide information to support construction of quantitative extrapo'lation of dose across
2 species (e.g., rat to human).
3
4 (6) Perchlorate Mechanism Studies. These studies provided a link to the pharmacokinetic
5 studies and were conducted via a comparison of existing literature and of new in vitro and
6 in vivo data that evaluated the effects of perchlorate on the iodide uptake mechanism across
7 species to aid in the quantitative extrapolation of dose.
8
9 (7) Genotoxicity Assays. These studies evaluated the potential for carcinogenicity by
10 evaluating mutations and toxic effects on DNA. These data were useful to determining
11 whether the benign thyroid tumors were likely to be a result of the proposed threshold
12 pathogenesis process.
13
14 (8) Immunotoxicity Studies. These studies were planned to evaluate the potential for
15 perchlorate to disrupt immune function and identify a potentially critical effect. These data
16 would help to reduce the uncertainty factor applied for database deficiencies. Because
17 concern was raised for these potential adverse effects based on the previous clinical
18 experience with treatment of Graves' disease patients, these studies were considered
19 necessary to a comprehensive database for perchlorate.
20
21 In the 1998 external review draft (U.S. Environmental Protection Agency, 1998d), a model
22 based on mapping the events of the mode of action for perchlorate was proposed as shown in
23 Figure 3-12. The key event was identified as the inhibition of iodide uptake at the NIS, followed
24 by decreases in thyroid hormones and increases in TSH. Both the potential neurodevelopmental
25 and neoplastic sequelae ofthis perturbation in thyroid hormone economy were proposed as
26 downstream adverse health outcomes. The conceptual model was endorsed by the external peer
27 review panel in 1999 (Research Triangle Institute, 1999), and additional studies were
28 recommended to reevaluate indications of developmental and neurodevelopmental in rats for
29 effects observed in the 1998 database. Delineating the continuum of histopathological changes
30 in the thyroid was also recommended. The results of all the studies in the testing strategy (both
31

January 16,2002 3-35 DRAFT-DO NOT QUOTE OR CITE


Effect
Exposure
Noncancer Cancer

8 I..
Blologlcall Early Altered Prognostic
Internal Clinical
Exposure "~--..l
_Dose
Effective .. Biological .. Structure Disease Significance
Dose Effect Function

Children's Health
RIsk

Human Health
Risk

Figure 3-12. Mode-of-action model for perchlorate toxicity proposed by the U.S. EPA
(U.S. Environmental Protection Agency, 1998d). Schematic shows the
exposure-dose-response continuum considered in the context of biomarkers
(classified as measures of exposure, effect, and susceptibility) and level of
organization at which toxicity is observed (U.S. Environmental Protection
Agency, 1994; Schulte, 1989). The model maps the toxicity of perchlorate on
this basis by establishing casual linkage or prognostic correlations of
precursor lesions.

"old" 1998 and "new" 2001), as well as additional studies now available in the literature, will be
2 reported together with EPA's interpretation and evaluation in Chapter 5.

January 16, 2002 DRAFT-DO NOT QUOTE OR CITE


b)(6)
/ 5
From: Kratz, Kurt, , OSD-ATL ­
Sent: Friday, June 06, 2003 11 :04 AM

To: Meehan, Patrick, Mr, OSD-ATL

Cc: Ferrebee, Patricia, Ms, OSD-ATL; Cotter, Sandra, Ms, OSD-ATL

Subject: FW: the leak to Lenny S continues FW: [CPEO-MEF] Interim Guidance on Perchlorate

Activities

fyi

-----original Message----­
From: Koetz, Maureen, SES, SAF/IE
Sent: Friday, June 06, 2003 10:15 AM
To: Woodley Jr., John, Mr, OSD-ATL; Cohen, Ben, Mr, DoD OGC; Fatz, Raymond J Mr ASA-I&E;
Schregardus, Donald, Mr., SECNAV
Cc: Kratz, Kurt, ,'OSD-ATL; Cornell, Jeff, Lt. Col, SAF/IE; Rogers, Daniel, Col,
AFLSA/JACE
Subject: FW: the leak to Lenny S continues FW: [CPEO-MEF] Interim Guidance on Perchlorate
Activities

We should probably talk about this at a Thursday ...

-----Original Message----­
From: Cornell, Jeff, Lt. Col, SAF/IE
Sent: Friday, June 06, 2003 7:55 AM
To: Koetz, Maureen, SES, SAF/IE
SUbject: the leak to Lenny S continues FW: [CPEO-MEF] Interim Guidance on Perchlorate
Activities

-----Original Message----­
From: Moessner, Phillip, Maj, AF!ILEVQ
Sent: Thursday, June OS, 2003 5:16 PM
To: McCann, Robert, Civ, SAF/IEE; Cornell, Jeff, Lt. Col, SAF/IE
SUbject: FW: [CPEO-MEF] Interim Guidance on Perchlorate Activities

-----Original Message----­
From: CPEO Moderator [mailto:I.I·~II""""
Sent: Thursday, June OS, 2003 2:01 PM
To: tk\thl
subject: [CPEO-MEF] Interim Guidance on Perchlorate Activities

The following document outlines DOD's Interim Guidance on perchlorate.


As far as we can determine the letter appears genuine, however, we are
trying to confirm whether it is a final document or a draft. If any of
the information below has changed or is incorrect we would appreciate
knowing right away.

Aimee

MEMORANDUM FOR ASSISTANT SECRETARY OF THE ARMY


(INSTALLATIONS AND ENVIRONMENT)
ASSISTANT SECRETARY OF THE NAVY
(INSTALLATIONS AND ENVIRONMENT)
ASSISTANT SECRETARY OF THE AIR FORCE
(INSTALLATIONS, ENVIRONMENT, AND LOGISTICS)
STAFF DIRECTOR, ENVIRONMENT AND SAFETY,
1
DEFENSE LOGISTICS AGENCY SUPPORT SERVICES
(DSS-E)
SUBJECT: Interim Guidance on Perchlorate Activities
Currently the National Academy of Sciences is conducting a study to
determine the best science and model to use for determining standards
for perchlorate. The result will be used by EPA to finalize a risk
assessment that will likely lead to the development of an oral reference
dose (RfD) for perchlorate. Once established, the RfD can be used for
conducting risk assessments for environmental restoration purposes and
for setting a Federal drinking water standard (i.e.! maximum contaminant
level under the Safe Drinking Water Act). A few states are also taking
actions on provisional non-enforceable perchlorate risk or action
levels. In preparation, and to allow us to plan and then respond when
standards are set, I am directing the Services to conduct a record
search for past and current perchlorate use and sample to ascertain
perchlorate occurrence at our installations, both active and closed, as
well as at Formerly Used Defense Sites (FUDS) as we investigate each of
those sites. At a minimum, all installations will be required to sample
for perchlorate at the installation public water systems, and
down-gradient from active and/or closed open burn/open detonation (OBOD)
sites. Additionally to the extent that monitoring wells are located
down gradient of active or closed ranges, sampling should also be
conducted at those locations. I am issuing this interim guidance under
authority of DoDD 4715.1, Environmental Security. I believe this will
position the Department to react faster and more efficiently when
standards are set.

When perchlorate is detected at a level above 1 part per billion, the


installation is to immediately inform its' chain of command, through
channels, the local community and consult with leadership on what
actions to take. The installation will evaluate each incident based on
the unique circumstances to determine the next step and report up the
chain both the occurrence and the action to be taken. The Components
will plan and program for future cleanup. .

This interim policy is part of a larger effort within the Department of


Defense. We are currently working to improve test and analytical
methods for determining concentrations of perchlorate. We are
developing and testing new techniques for cleanup, and we are
researching substitutes for perchlorate in solid rocket systems.
This approach in planning and budgeting for perchlorate sampling and
analysis will put DoD in a better position to comply with regulatory
standards when the are established. My points of contact are Mr. Shah
A. Choudhury at for environmental restoration, and Mr. Ed
Miller at • for compliance.

John Paul Woodley, Jr.


Assistant Deputy Under Secretary of Defense
(Environment)
Enclosures
Sampling and Analysis

Funding Sampling and Analysis. Components are to plan, program, bUdget,


and use appropriate environmental restoration or compliance funding for
future perchlorate sampling and analysis requirements. Environmental
restoration funding can only be used for sites that meet Defense
Environmental Restoration Program (DERP) eligibility requirements in the
current version of the DERP Management Guidance. Under DODI 4715.6,
"Environmental Compliance," perchlorate sampling and analysis would be
considered Environmental Quality Status Class III.

2
4

Analysis Method. At present, EPA Method 314.0 is the only approved test
method for the analysis of Perchlorate in water. This method was
promulgated under the Unregulated Contaminants Monitoring Rule of the
Safe Drinking Water Rule. The method requires that each laboratory
establish and verify with every analysis batch a minimum reporting level
(MRL) for perchlorate (Section 9.2.7 of Method 314.0). If a regulatory
agency desires a lower MRL for monitoring under the Safe Drinking Water
Act or other regulatory program, then that agency needs to identify an
alternate method that meets or exceeds the performance criteria of
Method 314.0.
Analytical Quality Assurance. A.laboratory chosen to perform
perchlorate analysis shall demonstrate proficiency to perform the test
method through an accreditation process established by the Component.
In addition, the laboratory shall posses the requisite state
certification(s). All analyses for perchlorate must be performed in
accordance with the current DoD Quality Systems Manual for Environmental
Laboratories (DoD QSM). The current ~rsion of the DoD QSM is available
on the Defense Environmental Netwbrk ~d Information eXchange (DENIX).
Conducting Sampling. Any perchlorate sampling plan will be based on
specific site conditions using the quality assurance and analytical
method described above. The sampling plan will include the reasonable
basis for suspecting the presence of perchlorate, sampling approach, and
decision criteria for potential further action to be taken based on the
analytical results.

Sampling Request from Regulatory Agencies. A regulatory agency may


request the installation to conduct perchlorate sampling or allow the
regulatory agency access to an installation to conduct sampling at the
agency's own expense. In either case, installations will not consider
any regulatory agency request for perchlorate sampling unless the
agency's written request clearly states the reason for the sampling
request (such as evidence stating that perchlorate was released into the
environment at the installation), risk posed (such as the pathway for
perchlorate that either exists on the installation or risk of exposure),
the regulatory structure for the sampling and the evaluation of
analytical results, and decision criteria for and expectation for
potential further actions. This requ~t will then be the basis of the
written agreement between the regulatory agency and the installation
prior to sampling. This written agreement. will clearly describe an
acceptable sampling plan as described above. Split sampling with
regulatory agencies is strongly recommended.

Actions Beyond Sampling and Analysis. Follow-on action can only be


taken when proper environmental restoration or compliance funding
resources are identified and available.

You can find archived listserve messages on the CPEO website at


http://www.cpeo.org/newsgrp.html
If this email has been forwarded to you and you'd like to subscribe, please send a blank
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3
Page 1 of2

Unknown

From: Cotter, Sandra, Ms, OSD·ATL


Sent: Thursday, June 19, 2003 11:01
To: Rogers, Daniel, Col, AFLSAIJACE; Kratz, Kurt, ,OSD-ATL
Cc: Kowalczyk Daniel (E-mail);Cornell.Jeff.Lt.Col.SAFflE;Cohen.Ben.Mr. DoD OGC
SUbJect: Perchlorate data

Kurt,
In response to discussion with George last week about concems with a list furnished by Jeff to OGC, for use with
Sen. Inhotrs staff, I asked Jeff to reconcile lists, in a master list that we can use in all future requests. This list
would include congressional districts, as requested. I believe George said that the list was needed no later than 1
July. How are you coming with that effort Dan?

SC

·----Original Message----­
From: Rogers, Daniel, Col, AFLSNJACE

sent: Friday, June 13,20032:45 PM

To: Cotter, Sandra, Ms, OSD-ATL; Cornell, Jeff, Lt. Col, SAFflE

Cc: Kratz, Kurt, , OSD-ATL; Kowalczyk Daniel (E-mail)

Subject: RE: Master survey data

Dan,

Can you come over Monday morning When I start briefing the PA folks coming to develop the plan?

vIr

DANIEL E. ROGERS, Colonel, USAF

Professional Responsibility Administrator and

Chief, Environmental Law and Litigation

(b)(2) (PR Office)


(E-Law Office)

DSN:"'ra5~tft~:tI!l-.:E-Law Office)

• • (Cell Phone)

-----Original Message----­
From: Cotter, Sandra, MS,.OSD-ATL

sent: Friday, June 13, 2003 11:11 AM

To: Cornell, Jeff, Lt. Col, SAFflE; Rogers, Daniel, Col, AFLSAjJACE

Cc: Kratz, Kurt, , OSD-ATL; Kowalczyk Daniel (E-mail)

Subject: RE: Master survey data

OGC said we need the list by the 1st of July for EPW staffers.

---·-Original Message---­
From: Cornell, Jeff, Lt. Col, SAFflE
Sent: Friday, June 13, 2003 9:33 AM
To: Rogers, Daniel, Col, AFLSNJACE
Cc: Kratz, Kurt, , OSO-ATL; Cotter, Sandra, Ms, OSD-ATL; Cullison, Geoffrey D; Van
Brocklin, Connie H Ms ACSIM; Carrillo, DaVid, Civ, AFfILEVR; Kowalczyk Daniel (E-mail)
Subject~ Master survey data
Page 2 of2

Dan - a couple of thoughts about "lists" ...there are at least five that I can think of:

1. the 2001 occurrence survey


2. the Apr 2003 congressional district survey
3. the May 2003 list from the services PWG reps (Dan K compiled and provided
to Kurt)­
4. The NPL list put together for the Dingell-Solis letter
5. EPA's list(s)

In a telecom this morning Sandy indicated that she and aGe feel it an urgent
matter to review these lists and reconcile them into one master spreadsheet that is
current and coordinated (confidence in the data is another thing). As you know I'm
essentially out of pocket the next 3 weeks (although I'll try my best to check email
occasionally remotely b Please talk to Sandy about how to accomplish this
reconciliation - either the PWG or BAH or perhaps one of the service reps.

vr
jeff

9/21/2005

Page 1 of1

Unknown

From: Kratz, Kurt, Mr. OSD-ATL


Sent: Friday, April 30, 2004 16:35
To: m.:\TR• • • • • • • • • •
Subject: Perchlorate Data - TRl/Munitions Constituents Emissions - IPR - BRAC Survey

Jim,
Love these all in one emails.
Thanks for the support the last two days. It does me good to get out and remember why I am doing this stuff.
1) Enclosed is the perchlorate data, minus AF for the IRP section. That will follow soon. I would request
that you don't pUblish on the web until we provide the final AF data. I will wait until Mon to send to states.
If I don't have info by then I will send them the same message.
2) Army ;s preparing an info p~per on the TRl/Munitions Constituents actions. It should be ready by Friday.
3) Barry Breen and I talked yesterday during a break. Understand he is pushing hard for the m~etlng on
BRAC. One issue I wanted to discuss with you was/is the BRAC survey. When we had the last (PR, it
was my understanding that instead of trying to match up the EPA/DoD data base, EPA was going to do a
scrub of the existing data we developed for the last IPR. EPA would highlight the sites with information
that was not consistent between our two organizations and work at the installation level to come to
agreement. If agreement could not be reached, those sites would be elevated through the chain. If not
rectified by the next IPR meeting, those sites would be addressed at the meeting. Vic told me that my
understanding may not be shared by you. That is why' asked Vic to set up a meeting between you, I, Vic
and your pac, so we can get this back on track.

Kurt
S_lc. I IMtalllItIon
Neme
18tlIte I EPA t Sempllng
Dat.. I ow SCIUCW
(Surfec:. or
Region Condue:ted . GrOWlClwet.r)
sample CoIIKtJon
Location Drlver Cited
PIrC~
Regultllory 8empIing Resultal
(ppb)
8IImpilngf I
An.lytlc:el Method
ConwnenIa or R.-..­

.l Qwy , ........ eorp. ~8ufwy. ..,_~)_U8OlI ..


._ .....,_ _lor_NAW.Cl*loLaaonl NOlF... _ _ aeo LoU .. _
.... _
_~_LoUuoIrvEPA3t4 ... _ ~......... -*9

.. c.A:omWO....._ ..

UCMR and Drlnldng w.tw ThI_..-. __


~ ....__. . -_ _ _ oI .. .. CI*IoLoU..., ..._ ..........
\fflOllor... _ _ Io:CAJt_
~ _ -..~

Navrf
Mama CorpsIBase Corpe I VA I
MarIneQuanllco 3
I 2J25I2OO3
I s.n- W_
I Water Treatment Plan
(Plarj Ef!IIJIn POE)
I NO
I IFederal; StateI <.4'
I
EPA 314 IMarine Corps Base QuariIco. 3.b.4. SOWA-UCMR Dale /I Sanlple No.:
021251200311 03-02626 Quantification laYeI: 4 ugIL ReeUt: -< 4 UlIIl

t s.n- Water I Water Corps


::'Corpst=~1 I Treatmenl PIari MarIne Base Quar1Ico. 3.b.4. SOWA-UCMR Date II San1p/8 No.
VA I 3 5/1312003 (PIarj Ef!IIJIn POE) NO Federal: State
.<4' EPA 314
051131200311 03-01104 QolnIficationLevel: 4 U(JIL R~: <.4 UlIIl

Marine Corps
:='.Corpsl::a:=ol VA I 3 I 8f26I2OO3 I s.n- W8l8r I Waler Treatment Plarj Base Quar1Ico. 3.b.4. SOWA-UCMR Date// Sample No.
NO Federal; State <.4" EPA 314
(PIarj EfIII.ent POE) 08I26l2OO3//02·13683 QuIIrtIlIcaIIon 1..IMll: 4 ugIL ReaUt: < 4 UlIIl

Navyt
MarIne Corps IMarIne Corpe VA 3 11/1812003 s.n-W8l8r
W8l8r Treatment PIari
NO Federal;'State <.4" EPA 314
MarIne Corps Base QuanlIco. 3.b.4. SDWA-UCMR Date /I Sample No.:
Base Querjk;o (Plari EfIII.ent POE) 11/1812003/1 03-18333 Querdlcetlon 1..IMll: 4 ugIl. R~: <4 UlIIl

Navyt
MarIne Corps MCSCamp
Pendleton
CA 9 2/2112002 Gnlund Water Wei 12202 YES 1210112003 Federal; Slate
ND EPA 314

Navy!
MarIne Corps MCSCamp
PendIelon
I CA I 9 I 5I2l2OO2 I Gnlund W8l8r I Wei 12202 I YES I 1210112003 IFederal; Statel
NO I EPA 314

Navyt
MerIne Corps MCSCamp
PendIelon
I CA I 9 I 2/1212003 , Gnlund Water' WeI~2 I YES I 1210112003 IFederal; Slatel
NO I EPA 314

Navyt
MarIne Corps MCScemp
Pendleton
I CA , 9 I 419/2003 I GnlundWater I WeI~2 I YES I 1210112003 IFederal; Slatel
NO I EPA 314

Navy/
MerIne Corps MCBCamp
PendIelon
I CA I 9 I 7I23l2OO3 I Gnlund Water I Wei #2202 I YES I 1210112003 IFedenII; Statel
NO I EPA 314

Navy/ MCBCamp
I
MarIne Corps PendIelon
, CA 9 I 12/112003 I Ground Water I WeI~2 I YES I 1210112003 IFederal; Stalel
NO I EPA 314

Navy!
MarIne Corps MCBCamp
P9IlIlIelon
I CA I 9 , 5I29l2OO2 I Ground Water I Wei #2301 I YES I 1210112003 I Federal: Stalel
NO I EPA 314

Navyi MCBCamp
MarIne Corps Pendleton I CA I 9 , 112712003 I Ground Water I Wei #2301 I YES I 1210112003 I Federal; Statel
NO I EPA 314

Navy! MCBCamp
MarIne Corps PendIelon
, CA I 9 I 2/1212003 I Ground Water I Wei #2301 I YES I 1210112003 IFederal; Slatel
NO I EPA 314

NavyI MCBCamp
Marlne Corps Pendleton I CA I 9 I 411612003 I Gnlund Water I Wei #2301 I YES I 1210112003 IFederal; Statel
NO I EPA 314

NavyI MCBCamp
MalIne Corps PendIelon I CA I 9 I 7/1712003 I Ground W8l8r I Wei #2301 I YES ·1 1W112OO3 I Federal; Statal
ND I EPA 314

NavyI ~MCB Camp


MIme CorpsPendlelon
~ 12/112003 I Ground Water I Wei #2301
I YES !1WII2l103 I Federal; Statal
NO I EPA 314

, Method 314 reporting Urnlls 4 ppb


Fie: ConeoIIdalecI ServiceoUJCaofl and NPOES_26Ap04_OSDII ""Urn. of detecIlon by rnetIQf 314 .. stated by 1aboraIoly.

SIWII: ~ and DrlrQ1g Water +J value Is stallslicaI estimate Page 1 of 28

O.tes ow Source . Expectlld "-rchlorate


8ervIc:e I IMtallalion state- EPA Sampling (SOO_ or S.mple Collec:llon Sampling Date ofNut Regulatory Sampling RQU/tel 8ampIingI I CClINMflIe or R.r.r.nc-

=-
N_ Region Conducted Groundwater) Location Planned Sampling Ormr Cited (ppb) Analytical Method

Corps ~p CA 9 5I9l2OO2 Grolnf Water Well23063 YES 1210112003 Federal; Slale NO I EPA 314

:z. Corps ~C:P I CA I 9 I 11»'2003 I Grolnf Water I Well23063 I YES I 1210112003 IFederal; Slatel NO I 300.0a

:::'. Corps ~detC:P I CA I 9 I 5I8l2OO3 I Grolnf Water I Well23063 I YES I 1210112003 I Federal; Slalel NO I EPA 314

: :'.Corpa =P I CA I 9 I 712412003 I GrolnfWater I Well23063 I YES I 1210112003 IFederal;Slatel NO I EPA 314

:t. Corps = P I CA I 9 I 121112003 I Grolnf Water I Well23063 I YES I 1210112003 IFederal; Slatel NO I EPA 314

:t. Corpa ~C:P I CA I 9 I 1I23l2OO3 I Grolnf Weier I Wei 123073 I YES I 1210112003 IFederaI; Statel NO I EPA 314

:t. Corps ~ CA 9 211212003 Grolnf Water Wei 123073 YES 1210112003 Federal: St8le NO EPA 314

:t. Corpa ~p CA 9 41312003 Ground Water Wei 123073 YES 1210112003 Federal: State NO
EPA 314; Prob:oI
Stat. CA DHS SRL
RAwlsionO

:t.CorpaI~P I CA I 9 I 7/1712003 I GroundWater I Wei 123073 I YES I 1210112003 IFederal;Stalel NO


EPA 314; Protocol
IStaI~=~SRL

EPA 314; Protocol


:t. Corps I~C:p I CA I 9 I 121112003 I Grolnf Water I Wei #23073 1 YES 11210112003 IFederal; Statel NO lStal~~~~ SRL!

:::'. Corpsl~p I CA I 9 I 3/1312002 I Grolnf Weier I Wei 112393 I YES 11210112003 IFederaI: Statel NO
EPA 314; Protocol
IStala CA DHS SRL
.RevlsIonO

:z. Corps I~p I CA I 9 I 2/1212003 I Grolnf Waler I Wei 112393 I I


YES 11210112003 Federal; Statel NO
EPA 314; Protocol
IState CA DHS SRL
Revision 0

=-Corpsl~p I CA I 9 I 41312003 I GrolnfWaler I Welt12393 I YES 11210112OO31FederaI:StateI NO


EPA 314; Protocol
IStalaCADHSSRLI
RevlslonO

EPA 314; Protocol


=-Corpel~";:p I CA I 9 I 519/2003 I GroundWeIer I Welt12393 I YES 11210112003 Federal; I Statal NO IStalaCADHSSRLI
RevlllonO

:t.CorpeI~p I I ICA 9
----l
7I23l2OO31 GroundWater
.
I. Wei 112393 I. . YESj1210112OO3jFederaI:Stale!
au FI'Q".~IIIllM."'.""'4~_,... .
NO
--'._.
EPA 314; Protocol
IStat~~SRL
_
FIe: ConIoIIdaled S.vlc81UJCt-Mt and NPDES_26Ap04_0s011 "lint 01 deledIon by meIhad 314 8I8taI8d by laboratory.

St1eel: UCMR and llrW*Ing Weier +J value II stalIstlcaI 8Ilmate Page 2 of 28

EPA se";;~
Servlce I Installation lltMe Reglon_ I I (S~; I a_pie Collection 18Ml pII"" I dN- I Regul8tory 181mP1\till ReeuItsI• __L~_..........
PI_ned D~ •. ext DrIvw Cited ~
I
Commenta or Rem.­
Neme ... _ ..• location •• _ •••••L •

Navy{ MeBCamp CA 9 12/112003 GnuldW_ WelI23Il3 YES 1210112003 Federal; State NO


MarlneCape Pendleton

EPA 314; Protocol


Navy! MCBCamp CA 9 5I9l2OO2 GnuldW_ Welt2e03 YES 1210112003 Federal; Slate NO State CA OtiS SRlI
MarlneCape Pendleton
RevllllonO

IMeB Camp I I EPA 314; Protocol


Navyf
MarIne Cape Pendleton 1 CA I I 9 1/2112003 I Grculd W" I Welt2e03
I YES 112101/2003 Federal; Statel NO State CA OtiS SRl
RevtslonO

I I I I EPA 314; Protocol


Nevyl IMCB Camp
Mame Cape Pendleton I I I
CA
9 211912003 Grculd W_ Welt2e03
I YES 1'210'12003 Federal; Statel NO State CA OtiS SRl'
RevtslonO

EPA 314; Protocol


NavyI IMCB Camp CA 9 4I9l2OO3 GrculdWlIllIr Welt2e03 YES 1210112003 Federal; State NO State CA OtiS SRl
MIme Cape Pendleton
RsvIeIonO

Navy{ MCBCamp 9 1/2412003 GrculdWlIllIr WeI~ YES 1210112003 Federal; State NO EPA 314
CA
MIme Cape Pendleton

EPA 314; Protocol


Navy{ MCBCamp CA 9 121112003 Grculd Wster Wellf2l103 YES 1210112003 Federal; State NO Stale CA OtiS SRl'
MIme Cape Pendleton RsvIeIonO

IMCB Camp I I I EPA 314; Protocol


Navyl
MIme Cape Pendlelon I I CA
9
I 1I23l2OO3 Grculd W.. well2tlOn
I YES 1'210'/2003 Federal;I Statel NO State CA OtiS SRl,
RsvIeIonO

IMCB Camp I I I EPA 314; Protocol


NavyI
MIme Corps Pendleton I I CA
9
1
2/1212003 1 Grculd Wat.­ Well2tlO12
I YES 1'210112003 Federal; Statel NO State CA DHS SRl'
RsvIeIonO

I I I I EPA 314; Protocol


NavyI IMCB Camp
Marlne Cape Pendleton I CA I 9
I 4/1012003 Grculd Watw well2tlOn
I YES 1'210'12003 Federal; Statel NO State CA OtiS SRll
Revl8lonO

NavyI IMCB Camp


I I 9 5I9I2l103 I Grculd Watw I WelI2lI012 I
YES 1'210'12003 Federal; Statel NO
I f>nlloca State CA
OtiS SRL RllVl8Ion
Marlne Corps Pendlelon CA I I 0
MCBCamp f>nlloca State CA
NavyI
CA 9 71Z317OO3 Grculd Wster WelI2lI012 YES 1210112003 Federal; Slate NO OtiS SRl Revtslon
MarIne Cape Pendleton 0

NavyI MCBCamp 12/112003 GrculdWat. WelI2lI012 YES 1210112003 Federal; State NO EPA 314
CA 9
MarIne Carps Pendleton

I I I I I I I I
NavyI MCBCamp 9 Wei 12671 NO NO I EPA 314
MIIrIne Cape Pendleton 5I9l2OO2 Grculd W_ Federal; Statel
CA

• MeIhDd 314 ~ Iml III 4 ppb


Fie: ConliolllIlItIlServlces_~ and NPDES_28Apr04_0s011 -llmI d d8teetIon by method 314 .. 8llIl8d by 18bor8lory.
Shell: ~Ind~W.. +J value III atalIlIllcaIll8limale Page 30128
ServIce
H_
I I Dat..
I InetallatJon Slate EPA I Sampling
Region Conduct.cl
I I DW Source
(Surf_ or
GrouncIwllter)
Slim"" Collec:tlon
Location
I I 8mnpling O ::=xt
Plenned Sampling
Regulatory Perchlorate
DrtwrClted
5ampllngl
Sempllng RMUlta Anelytlcal Method'
(ppb)
C_m.orR____

EPA 314; Protocol


Navy( IMe8 camp CA II 1/2312003 Ground Water WeJ #2671 NO F8denII; Slate NO Stale CA DHS SRL
Marine Corpc Pendleton
RevtslonO

Navy! MeBC8mp
CA II 411012003 Ground Water WeJ#2671 NO Federal; State NO EPA 314
Marine Corpc Pendleton

Navyf MCBcamp
CA II 5I9l2OO2 Ground Water WeJ #2673 YES 1210112003 F8denII; State NO EPA 314
Marlne Corpc Pendleton

EPA 314; ProIOcOI


Navy/ MCBcamp
CA II 1I22l2OO3 Ground WlJIer WeJ#2673 YES 12101/2003 Federal; Slate NO Slate CA DHS SRl
MarIne Corpc Pendlelon
Revision 0
---
Navy/ IMCS Camp
MarIne Corpc. Pendleton I CA I I II 2/1I/l2OO3 I Ground WlJIer I WeJ#2673 YES 11210112003 1Federal: Statel ND
I EPA 314; Protocol
State CA DHS SALI
I Revision 0

IMCS camp IEPA 314; Protocol


Navy{
MarIne Corpc Pendleton I CA I
II
1 4I9i2OO3 I Ground WlJIer I WeJ#2873
I YES /1210112003 I Federal; Slatel NO State CA OtiS SALI
RevlllonO

IMCS 1EPA 314; Protocol


Navy/ camp
MarIne Cope Pendleton 1 CA I II
I 7/1ll12OO3 I Ground Water I WeJ#2673
I
YES /1210112003 IFederal; Statel NO Slate CA DHS SRL
Revision 0

EPA 314; Protocol


Navy{ IMCB camp CA II 12/112003 Ground Water WeJ#2873 YES 1210112003 Federal; Slate NO State CA DHS SRLI
Marlne Corpc Pendleton
RevlllonO

NavyI MCBC8mp
MarIne Cope Pendleton
CA II 2/1312llO2 Ground Water WeJ 11330923 YES 1210112003 Federal; Stllle NO - EPA314

Navy/ MCBCamp
MarIne Corpc PlIndIeIon I CA I II I 5I'lII2OO2 I Ground WlJIer I WeJ#330923 I YES I 1210112003 I Federal; Statel NO I EPA 314

Navyf MCBCamp
Marlne Corpc Pendlelon I CA I II I 2/812003 I Ground Water I WeJ#330923 I YES I 1210112003 I Federal: Stalel NO I EPA 314

Navyf MCBC8mp
Merlne Corpc Pendleton I CA I II I 4I23l2OO3 I Ground Water I Well 11330923 I YES I 1210112003 IFederal: Statel NO I EPA 314

Navy! MCBCamp
Merlne Corpc Pendleton I CA I II I 8/2712003 I Ground Water I WeJ#330923 I YES I 1210112003 IFederal; Statet NO I EPA 314

Navyf MCBCamp
Merlne Corpc Pendleton I CA I II I 12/112003 I Ground Water I WeJ 11330923 I YES I 1210112003 I Federal; Statel NO I EPA 314

Navyf
MarIne Cope :a..:,mp I I I I
CA II 2/1312llO2 Ground Water I WeJ tI330925
1
YES 11210112003 I Federal; Slate! NO I EPA 314

• Method 314 rlIpllIting Urn. Ie 41lPb


FIe: ConsoIId8led s.w-_UCMR and NPDES_28Apr04_OSDII "LImI fA deleclion by rnelhod 314 .. stated by 18Ilor8loIy.
ShMt UQ"fi 8IlII ~ Water +J YlIUe Ie ItatislIcaI aatmaIlI Page 4 al28
Oat.. DWSource Expected Perchlorate I
IMUllatlon
SlIIlIPllngResultal~~1
EPA SlIIlIpie CoIlec:tlon Sampling ReglJlatory
ServIce Slate (Surf_or
Sampling Date of Next Commenta or rter.rencn
Name Region Loc:atIon Pl8nned Driver Cited
Conducted Groundwater) 8ulpIlnlJ (ppb)

Navy{ MCBCamp
Federal; Stale
MarIne Corps Pendlelon
CA 9 5I9l2OO2 Ground WaI8r Wei #330925 YES 1210112003 NO I EPA 314

Navy{ MCBCsmp
MarIne Corps Pendleton
I CA I 8 I 21612003 I Grlu1d Water I Well33Oll25 , YES , 1210112003 'Federal; Stalel NO I EPA 314

N8vyI MCBCsmp
MarIne Corps Pendlelon
I CA I 8 I 411l112OO3 I Grlu1d Water I Welt330925 I YES , 1210112003 !Federal; StaleI NO , EPA 314

Navy{ MCBCamp I CA
MarIne Corps Pendleton
, II I 7/1812003 I Grlu1d Walet I Well33Oll25 I YES I 1210112003 IFedersI; Slata' NO I EPA 314

Navy{ MCBCamp
MarIne Corps Pendleton
, CA I II I 12/112003 I Grlu1d Water I Wei *330825 , YES I 1210112003 IFederal; Slatal NO , EPA 314

Navy{ MCBCamp
MarIne Corps Pendleton
, CA , II , 2/1312002 I Grlu1d Walet I Wei *331124 , YES , 1210112003 IFederal; Slatal NO I EPA 314

Navy{ MCBCamp
MarIne Corps Pendleton I CA I II I 5I9f2OO2 , Grlu1d Waler I Wei #33924 I YES I 1210112003 JFederal; Stalel NO I EPA 314

Navy{ MCBC8mp
MarIne Corps Pendlelon
I CA I II I 2J2112OO3 I Gnulll Water , Wei *331124 , YES I 1210112003 IFederal; Stalel NO I EPA 314

Navy{
Marine Corps
MCBC8mp , CA
Pendleton
I 8 I 411412003 I Grlu1d Water I Wei #33924 , YES , 1210112003 IFederal; Stalel NO I EPA 314

Navyf MCBC8mp
Marine Corps Pendleton I CA I II I 711712003 I Gnulll Water I Wei 1133824 I YES I 1210112003 IFederal; Stalel NO I EPA 314

Navyf MCBCamp , CA ,
Marine Corps Pendleton
II I 121112003 I Gnulll Water I Wei #33924 I YES I 1210112003 IFederal; Slatel NO I EPA 314

Navy{ MCBCamp
Marine Corps Pendleton I CA I II I 5I8l2OO2 , Gnulll Water I Wei 1141611 I YES I 1210112003 IFederal; Stale' NO I EPA 314

Navy{ MCBCamp , CA
Marine Corps Pendleton
I II , 216/2003 , Ground Water I Wei 1141611 I YES I 1210112003 IFederal; S1atel NO I EPA 314

Navyf MCBCamp
MarIne Corps Pendleton
I CA I 9 , 4f2J2OO3 I Gnulll Water I Wei 1141611 I YES I 12101/2003 IFederal; Stalel NO I EPA 314

Navy{ Mq!Camp
MarIne Corps Pendleton
I CA I 8 , 7f24f2003 I Ground Wa!flIt I Well 1141611 I YES I 1210112003 'Federal; Stalel NO I EPA 314

Navy{ MCBCamp IFederal; Stalel


MarIne Corps Pendleton I CA I 9 I 121112003 I Gnulll WaI8r , Wei 1141611 I YES , 1210112003 NO I EPA 314

Navyt
Marine Corps ~..;a;p I I ICA II 5Illf2OO2 I Gnulll Water I Wei 1141613
1
YES 11210112003 IFederal; Stalel NO I EPA 314

" Method 314 reporlIng lint Ie 4 ppb


FIe: ConsoIld8led SflItvlc81U~ and NPOES_~_OSD11 ""lint 01 delection by method 314 .. elaIed bylabonllllry.
Sheet: IJCMR and ~ W" +J V8lJe Ie stati1t1ca18lItImaIe Page 5 of 26
. . . .
Il'IlltaIIatJon
N_
EPA
0....
State Region s.npllng (Surf_or
ConcIuctllCl Groundwater)
Locdon
I I I
DW80urce I 8ample Collection IS_piing Expec:tllCl Regulatory f'en:hlome 88mpIl1llll I
PIannlICI Old. of Next Drtv. CItlICI Sempliog R..u1~ AnalytIcal Method I
Sampling (ppb)

C_nta orRee.­

INBvy/ MCBCamp

MarIne Corps Pendlelon


CA 9 2JlII2OO3 GrotnI WeIM I WellU1613 I YES I 1210112003 I FederlIl; Statel NO I EPA 314

NBvy/ MCBCamp

I CA I II I 4I2l2OO3 I GrotnI WeIM I WellU1613 I YES I 1210112003 IFederal; Statel NO I EPA 314
MarIne Corps Perdeton

NBvy/ MCBC8mp

MerIne Corps Pendlelon I CA I II I 8IZ712.OO3 I GrotnI WeIM I WellU1613 I YES I 1210112003 I FedenII; Statel NO I EPA 314

NBvy/ MCBC8mp

MerIne Corps Pendleton I CA I II I 12/112.003 I GrotnI Water I WellM1613 I YES I 1210112003 IFederal; Statel NO I EPA 314

N8vyI • MCBCamp

I CA I II I 5IlIi2OO2 I GrotnI WaIIIr I WellM1621 I YES I 1210112003 IFederlIl; Statel NO I EPA 314
MerIne Corps Pendleton

N8vyI MCBc.mp

I CA I II I 2I8l2OO3 I GrotnI WeIM I WellM1621 I YES I 1210112003 I Federal; Statel NO I EPA 314
MarIne Corps Pendleton

N8vyI MCBc.mp
...... Corps Perdelon
I CA I II I 4I2l2OO3 I GrotnI WeIM I WellM1621 I YES I 1210112003 I Federal; Statel NO ,
EPA 314

N8vyI MCBc.mp

MarIne Corps PendIelon


I CA I II I 7I24l2OO3 I GrotnI W.. I WellM1621 I YES I 1210112003 IFedenII; SlateI NO I EPA 314

Navy( MCBC9mp

MarinI Corps PendIelon


I CA I II I 12/112.003 I GrotnI WeIM I WellM1621 I YES I 1210112003 I Federal; Statel NO I EPA 314

NBvy/ MCBCamp

I CA I 9 I 5I8l2OO2 I GrotnI WeIM I Wei 152013 I NO I IFedenII; Statel NO I EPA 314


MarinI Corps Perdeton

Navy( MCBC8mp

...... Corpe PendIelon


I CA I 9 I 51812002 I GrotnI Water I WfII152023 I YES J 1210112003 I Federal; S1atel NO I EPA 314

Navy! MCBC8mp

MarinI Corpe PendIelon


I CA I 9 I 2I2l2OO3 I GrotnIWater I Wei 152023 I YES I 1210112003 I Federal; Slate' NO I EPA 314

NavyI MCBC9mp

I CA I 9 I 4I3l2OO3 I GrotnI WeIM I Wei 152023 I YES I 1210112003 I Federal; Statel NO I EPA 314
MarinI Corps PendIelon

Navy( MCBCamp

I CA I 9 I 1/1112.003 I GrotnI Water I Wei 152023 I YES I 1210112003 I Federal; Slatel NO I EPA 314
MIme Corpe Pendleton

NavyI MCBC9mp
MarinI CClrpa Pendleton I CA I 9 I 12/112003 I Groupd Water I Wei #52023 I YES I 1210112003 I Federal; Statel NO I EPA 314

NavyI MCBC8mp
MarIne CClrpa PendIelon
I CA I 9 I 2l2Ol2OO2 I GrotnI WeIM I Wei #52028 I YES I 1210112003 IFederal; S1atel NO I EPA 314

NavyI MCSC8mp
MameCClrpa PerllIeton
Wei 152028 Federal; Statel NO I EPA 314

" MBIhod 314 repatlng 11m. is 4 Pllb

File: ConsolIdated Servic81UJCt.lA end NPDES_26Ap04_0s011 ""Urn. 01 datecllon by melhod 314 as ataled by Iaboralay.

Sheet: UCt.lA end ~ WaIIIr +J v8lue II tdaIIIticaI eatImaIe


Page 6 0128
s.rvtc. I InstlI/llItlon
N_
Slllte
EPA
Region
Sempllng
ollt..
(Surf_ Of'
ow Source hmple Collection
LocatJon
hmpllng
Planned
Expected
Det8 of Nut
Regulatory P~hIDl8ta I Sempllngl
Driver Cited Sempllng Reeults AMIytlcaJ Method'
CommentsOf'R...-­
Conducted - GroundwatMl hmpllng (ppbl
I
Navyl MCeC8mp
Marine Corps Pendleton
CA II 2I5l2OO3 GrcudW.... Well #52028 YES 1210112003 Federal; State NO I EPA 314

Navyi MCBCamp
Maflne Corps Pendleton
I CA I 9 I .ci3f2OO3 I GrcudW.. I Well #52028 I YES I 12101f2003 IFederal; Slatel NO I EPA 314

l~vyI MCBCamp
Marin8 Corps PendleIon
I CA I II I 7/1712003 I Ground W.. I WelllII5202B I YES I 1210112003 JFederal; Slatel NO I EPA 314

Navyi MCBCamp
Marin8 Corps Pendleton I CA I II I 12/112003 I Grcud Wilt« I Well #52028 I YES I 1210112003 IFederal; Statel NO I EPA 314

NavyI MCBC8mp
Marin8 Carps Pendleton I CA I II I 5I8l2OO2 I Grcud Wilt« I Well 1/61511 I YES I 1210112003 IFederal; Slatel NO I EPA 314

MCBC8mp
1:=,.Carps Pendleton I CA I II I 2I5l2OO3 I Ground Wilt« I Wei 1/61511 I YES I 1210112003 IFederal; Statel NO , EPA 314

NavyI MCBCamp
Mertne Corps Pendleton I CA I II I 4J2I2OO3 I Grcud Wilt« I Wei 1/61511 I YES I 1210112003 IFederal; Stalel NO I EPA 314

NavyI MCBCamp
Marine Corpa Pendleton I CA I II I 7/1812003 I Grcud Wilt« I Well 1/61511 I YES I 12101f2003 IFederal; Statel NO I EPA 314

MCBCamp
I:.Carps Pendleton I CA I II I 12/112003 I Ground W.... I Well 1/61511 I YES I 1210112003 IFederal; Statel NO I EPA 314

NavyI MCBCamp
MIme Corps Pendlelon I CA I 9 I 2nOI2OO2 I Ground Wilt« I Wei 1/61521 I YES I 1210112003 I Federal; State/ NO I EPA 314

NavyI
MIme Carps MCBCamp
Pendleton I CA I II I 5I8f2OO2 I Ground Wilt« I Wei 1/61521 I YES I 12101f2003 IFederal; Statal NO I EPA 314

NavyI MCBCamp
MarIne Corps Pendleton I CA I II I 2/512003 I GroundW.... I Wei 1/61521 I YES I 1210112003 IFederal; State' NO I EPA 314

NavyI MCBCamp
MIme Corps Pendlelon I CA I 9 I 4I2l2OO3 I GroundWIJI.eI I Wei 1/61521 I YES I 12101/2003 I FedlIraI; Statal NO I EPA 314

NavyI MCBCemp
MarIne Corps Pendlelon I CA I II I 7/1612003 J Ground Water I Wei 1/61521 I YES I 12101f2003 IFedlIral; stalel NO I EPA 314

Navy! MCBC8mp
MIme Corps Pendleton I CA I II I 12/112003 I Ground Water I Wei 1/61521 I YES I 1210112003 IFederal; stalel NO I EPA 314

Navy! MCBC8mp
MIme Corps Pendlelon I CA I 9 I 5tllI2OO2 I GroundWIJI.eI I Wei 1/62621 I YES I 12Jll112OO3 IFederal; Slatel NO , EPA 314

NavyI
MIme Carps :onmp I I I I CA 9 2/512003 Ground Water I Wei 1/62621
I YES , l2Jlllf2003 JFederal; Statel NO I EPA 314

• Meltod 31418p1lrt1ng lim•• 4 PIlb


Fie: Conadldated ServlclllUJCMR IWId NPOES_26Ap04_0s011 "lAnI cA detection by meIhDd 314 ul1al8d by IabonllOly.
Sheet: lJCW{ IWId ~ Wf/bII +J value • sta1ist1ca18lIlImaIe Page 7 cA 28
Dates OW Source Expected Perchlom. .

Servtce Installation State EPA Semplll1lI (Surf_ or 88mple Collec:tlon SamplII1ll Date of Next Regulatory SlIRlplll1ll RMUItsI SamplI"liI J CocrwMnta or R.m.nc..

Name Region Conducted Groundwater) location Plannecl 8amp"11lI DrIver Cited (ppb) Analytical Mathod

=.. Corpe :;=" CA II 4I2l2OO3 Groo.nI Water Wellll2621 YES 1210112003 Federal; Slate NO I EPA 314

:z. Corpe =:P I CA I II I 7/1612003 I Groo.nI Water I Wei 162621 I YES I 1210112003 IFederal; Slatel NO I EPA 314

:;:'. Corpe ~P I CA I II 1 12/112003 I Groo.nI W8ler I Wei f62621 I YES I 1210112003 IFederal; Slatel NO I EP" 314

MamBCorpe
:;:'.Corpa logllbB8ae1 CA I 9 I 5131J:lOOl I Groo.nIW8ler I YermoDrlnlWlgweltI4l NO I I Slate I NO I EP"314
Berataw

=. M8rln8 Corpe
Corpallogllb B8ae I CA I 9 I 811llf2OO2 I Groo.nI W_. I Yermo Dr1nkk1g Wei tI4 I NO I I SIale I NO I EP" 314

=- E:;
Corpat~"""a::., CA I II I 811212003 I Groo.nI W_ I Yermo Dr1nkk1g Wei tI4 I NO I I Slate I NO I EP" 314
Berataw

=- MamBCorps
I
Corpe II.oIllItica B8ae CA
Il8r&Iow
I II I 5131f.2Ol11 I Groo.nI W8ler I Yermo Dr1nkk1g Wei #5 I NO I I Slate I NO I EP" 314

:z. Marlne Corps


CorpeIloglsIIcs B8ae I CA
Il8r&Iow
I II I 811ll12OO2 I Groo.nI W8ler I Yermo Dr1nkk1g Wei #5 I NO I I Slate I NO I EP" 314

=.. MamBCorps
Corpe\loQlsIIcs Base CA
IBarstow
9 811212003 Groo.nI Water Yermo Dr1nkk1g Wei #5 NO Stale NO EP" 314

lMamB Corps There Ia no known acuce 01 percIbaIe for MCl8 Baratow. Semp\IrG
:::'. CorpsllOlllstlca B8ae CA 9 10I29l2OO2 Groo.nI W8ler Yermo Dr1nkk1g Wei..,... NO St8le NO EP" 314 _ StIle 01 CalIfornIa requIremenI per Tile 22 CaIlfo<ria Code Of
IIl8r&Iow ReglAtlona SeclIon 84450 for ~ chemteal monltoIlrQ.

IMamBCorpe
~CorpellOlll8llcsB8ae CA 9 2/412003 Groo.nIWater YermoDr1nkk1gWeI..,... NO St8l8 NO EPA 314
Barstow

MamBCorps
:::'. Corps ILool8llcs B8ae CA 9 5/1312003 Groo.nI Water Yermo Dr1nkk1g Wei tI6A NO St8l8 NO I EPA 314
l&nlow .~

IMarlne
Corps
=:'. Corps I~ B8ae CA II 811412003 Groo.nI Water Yermo Drlnldnll Wei tI6A NO St8l8 NO I EPA 314

MCBCamp
:::'.Corps\~~ 7fl512OO1 I Groo.nIWat« I~~::oIRl I NO l.:-ederaI I NO I EP"314

• Melhod 314 reporting lImala 4 ppb


Fie: ConsoIldaled Servlc.-_lJCI,fl and NPDES_26Ap04_0s011 -Lmll 01 delection by IIMIthod 314 ealtal8d by laboralay.
Sheet: UCMR and ClrInkk1g W8ler +J value Ia st8lIsIIcal88llmale Page 8 01 28
Service
I IftI!lall.uon State EPA
"_ Region
D....
Sempllng
Conducted
DWSource
(SurfKeor
Groundw.....)
8MIple Collection
LOC8lIon
I I
Sempllng Expected Regul"ory Percnlome I
SampllngI
Planned Dm of Next Drfver Cited Sempllng R..ulla AnalytIcal
S8mpllng (PPb)
Method' Comments or Refwencea

NaVfl I~ Ne 4 7I25l2OO1 GroundWater


Outftow Of Holcomb BIv
TI'll8\IJIlri PIart
NO I Federal I NO I EPA 314
Marine Corps New Riv.

Nsvyl I
MeB
Camp
Uarine Corps lejeunB/MCAS
I NC I I 4 7I25l2OO1 I Ground W... I Outftow Of UCAS
Treatmerl PIart
I NO I I Federal I NO I EPA 314
NewRiv8f

Navyi
MameCorps
MeBC8mp
lejeunBIMCAS
I NC I I 4 512112003 I Ground Water I Outftow Of UCAS
Treatmelt PIart
I NO I I Federal I NO I EPA 314
New RIver

NsvyI tMGTF29 , CIt ,


II J 12/1212001 t GroundWater t Scuoe WIIl10A I NO I 'Federal; Slate\ ND I EPA 314
MarIne Corps Pelma
I

tMGTF29 , CIt ,
II I 0/1212002 t Ground Water t Scuoe WIIl10A t NO I IFederal; Stalel NO I EPA 314
i=.C<wps Palme

NsvyI tMGTF211
MarIne Corps Palma I CIt t II I 12/1112002 I Ground Willer I Scuoe WIIl10A I NO 1 I Federal; Slatel NO I EPA 314

NsvyI tMGTF211 I
MarIne Corps Palma
I CIt I II 8I2llI1991 , Ground W8l8r , Scuoe Wel11A I NO t t Federal; Slatel NO I EPA 314

NsvyI tMGTF211 , CIt


Marine Corps Palma
t II t 1~2 I Ground W8l8r I ScuoeWel11A , NO t t Federal; Slatel NO I EPA 314

Navyi tMGTF211
I Federal; Slatel
MarIne C<wps Palmi I CIt I II I 811212002 I Ground Water t ScuoeWel11A t NO I NO I EPA 314

Navyi tMGTF211 t I Federal; Slatel


Marine C<wps Pelma
t· CIt I II 1211112002 , Ground Wet..- , ScuoeWel11A I NO I NO I EPA 314

Navy! tMGTF29
Marine C<wps Palmi I CIt t II , 8IW11191 I Ground Wet..- , ScuoeWel2A , NO J I Federal; Slatel NO \ EPA 314

Navy! tMGTF29 I
MlmeC<wps Palmi
CIt , II I 8IW11197 I Ground Wet..- I ScuoeWel3A I NO I IFederal; Slatel NO I EPA 314

NavyI tMGTF 211 , CIt t


MlmeC<wps PalmI
II I 1211212001 I Ground Water I ScuoeWel3B I NO t IFederal; Slatel NO I EPA 314

NavyI tMGTF 211


MarIne Corps Palmi
I CIt I II I 811212002 I Ground Wet. , Scuoe Wei 38 I NO t IFederal; Slatel NO I EPA 314

NavyI tMGTF211
Marlne COrps PalmI t CIt t II I 1211112002 I .GroundWater I Scuoe Will 38 I NO I I Federal; Slatel NO ,
EPA 314

Navy! tMGTF211
Marlne Corps Palma
t CIt I II I 8128/1991 I Ground Water I ScuoeWel4A I NO I I Federal; Slatel NO I EPA 314

Navy! ~lfMGTF 29
Mame Corps Palms I~ 12112flOO1 I GroundW.. I ScuoeWel4A
I I NO ~Slalel NO I EPA 314

• Med10d 3141'llpD11irQ 11m. ill 4 ppb


Fie: Consolidated Ser1taI_lJCY{ and NPOES_26Apr04_0SD11 ·'LIm. of detection by method 314 aallallld by laboratory. P8ge 9 of 28
Sheet. lJCt,fl and Dmkng W.. +J V8kJ8 ilIlaIIItlcaI8lIlImate
, Servlc.
,
Installation
N_
SIM.
EPA
Region
Oat..
SamplJIll/
DWSource
(Surf_or
Sempl. Collection
Locetlon
Samplllll/
Planned
Expected
DlIta ofN.llt
Regulatory
Dm.rCItad
Pen:hloreta I s-Piingi
Sampl~~"UIta Analytical M9thocII Commenta or Refw•.-.
,
Conducted Groundwater) Sunplllll/

Navy( MAGTF29
Marine Corps Palma
CA 9 6/12J2002 Ground Water Source Wel4A NO Federal; Stale NO I EPA 314

Navyl MAGTF29
Marine Corps Palma
I CA I 9 I 1211112002 I Ground Water I Source Wei 4A I NO I I F_al; Statel NO I EPA 314

Navy( MAGTF29
Marine Corps Palma
I CA I 9 I 6/26/1997 I Ground Weter I Source Wel5A I NO I I Federal; Statel NO I EPA 314

Navy( MAGTF29
MlmeCorps Palma
I CA I 9 I 12112fZOO1 I Ground Water I Source Wel5A I NO I IFederal; StaleI NO I EPA 314

Navy( UAGTF29
MlmeCorps Palms
I CA I 9 I 6/12J2002 I Ground Water I Source Wei 5A I NO I IFederal; Statel NO I EPA 314

Navy( MAGTF29
Marine Corps Palms
I CA I 9 I 1211112002 I GIourd WallW I Source Wel5A I NO I I Federal; Statel NO I EPA 314

Navy( MAGTF29
MarIne Corps Palms I CA I 9 I 6/28/1997 I GIourd w.. I Source Wei 8A I NO I I Federal; Statel NO I EPA 314

Navy( W.GTF29
MarIne Corps Palms I CA I 9 I 1211212001 I GIourd WallW I Source Wei SA J NO I IFederII; Statel NO I EPA 314

Navy( W.GTF29
Marine Corps Palma I CA I 9 J 6/12J2002 I GIourd Weter I Source Well SA I NO I IFederal; Statel NO I EPA 314

Navyl MAGTF29
MarIne Corps Palma I CA I 9 I 12/1112002 I GIourd Weter I Source Wel8A I NO I I Federal; Statel NO I EPA 314

Navyl MAGTF29
Marine Corps Palma I CA I 9 I 6/26/1997 J GIourd Water I Source Wei 7A I NO I IFederal; Stalel NO I EPA 314

Navy( MAGTF29
Marine Corps Palms I CA I 9 I 12/12J2001 I Ground Water I Source Wei 7A I NO I IFederal; Stalel NO I EPA 314

I~avyf MAGTF29
I CA I 9 I 6/1212002 I Ground W.. I Source Wei TA I NO I I Federal; Statel NO I EPA 314
IMarine Corps Palms
INavyf MAGTF29
MarIne Corps Palms I CA I 9 I 12/1112002 I Ground Water I Source Wei 7A I NO I I Federal; Stalel NO I EPA 314

Navy! MAGTF29
Marine Corps Palms I CA I 9 I 8/28/1997 I Ground Weter I Source Wei SA I NO I IFederal; Stalel NO I EPA 314

Navyf MAGTF29
MarIne Corps PaJms I CA I 9 I 12/1212001 I Ground Water I Source Wei 8A I NO I I Federal; Stalel NO I EPA 314

I I I I I I I I
Navy( MAGTF29
MarIne Corps Palma 9 6/1212002 GroundW.. Source wei SA NO Federal; Statel NO I EPA 314
CA

o MBhld 314 I8jlOl1Ing om. Is 4 ppb


Fie: ConsoIJdaIIldS."tC81UJCMR and NPDES_26Ap04_OSDl1 "UmI 01 delect10n by meIhod 314 as staled by 1aboraIory.
Sheet UCMRend ~ W.. +J Yalue Is alalIsIlcal eaIImale Page 10 of 28
Neme
I
8erv1ce I InBtallation Sl8tel EPA I Dam
S....pllng
Region Conducted
I OW Bourn
(Surf8ce or
Ground__)
88mple Collection
l.oc:8tIon
Expected Perchlorate
S_pllng D.e of Next Regul8tOry Sempllng Reeulta Sempllngl
P18nlHlCl s.mpllng Drtvw CItecI (ppb) AnaIytJc:al Method

C_nts or Rem.-

NavyI MAGTF 29 CA 9 12112J2001 GroInl W'" Sotne Wel9A NO Federal; Slate NO EPA 31. MAGTF 29 Palms Is scheduled 10 ......mple SOU'C8 weIIa for pen;Norate
MarIne Corps Palms 2004 calendar year.

Navy( MAGTF 29 CA II 6/1212002 GroInlW... Sotne Wel9A NO F-..· Slate NO EPA 31.
Marln8 Corps Palms '

:t. Corps ::~F 29 I CA I II I 12/1112002 I GroInl W... I Sotne Wel9A I NO I I Federal; Stalel NO I EPA 31.

Navy( PENSACOlA FL • 212712001 GroInl WlIl8r NTTe Drir'tkq Water I NO I I FederlII I NO I EPA 31.
Marln8 Corps HAS PIlIIt

NavyI CORONADO
Mlnle Corps NAVSASE
CA " 1I181ZOO3 GroInlWlItlIf seRECsmpBaclwp
Wei
I NO I IFederaI·SlateI

NO I EPA 31.

NavyI CORONADO
Mlnle Corps NAVSASE
CA " 517flOO3 GroInl WlItlIf SERE Camp
Wei
Backup I I NO IIFederal'' SlatelJ NO I EPA 31..
I
INAB Coronado, seRE Camp: Drir'tkq water weIs thet Id groundW8blr
110 _the baae. Sle Is • sm8llralnng baae In the East PMt r:A san
::::'Corpsl~~ I CA I " I 11nflOO3 I GroInlW... I seRE C:e:Backup I NO I /F8dlnl; Slatel NO I EPA 31. IDidueegoCoony· NASHI 5189: SampIrQ for percIjoralewas perfonn8d
10 lrUrest b, !he Slate l'IIQlJaIory egendes and noI due to lIllY
speclfic reqWmert.

I:;t.Corps ~~ ~I "181200' I GroInlWaler I seRE Camp MltIn Wei \ NO \ IFederaI:StateI NO I EPA 31. I
!:;t.Corps ~~ I CA I 9 I 517flOO3 I GroInlWater I SERE CampMltlnWei I NO I IFederal;S!atel NO I EPA 31..

I=-
"

'.
Corps ~~ I CA I 9 I 11n12OO3 I GroInl Water I seRE Camp MaIn Wei I NO' IFederal; Statel NO I EPA 31.

:z. Corps :~:~ I MO I 3 I 1f29(2Q()2 I GroInl W... I polrj rA Enlry Bldg 525 I NO I I Federal I NO I EPA 31.

NavyI PATUXENT MO 3 lf29(2Q()2 GroInl Water PoIrt rA Enlry Bldg 526 I NO I I FederlII I NO I EPA 31..
MarIne Corps RIVER HAS

!NavyI ELCENTRO
IMarine Corps NAF CA 9 311312001 StriaceWater OuIftowfromdrlnldr1g
water plan
I NO I I Slate I NO I EPA 31. II~todrlnldr1gwaterplanlisColoracloRJverWlll8r.
CorilmancIer

IMarine Corps Forces.


!Navyf lu,s, NlMIII GU 9 1()(1212OO3 GroInl W... Groundwaler
Wei " NCS 3
MOOIor1ng I NO I I Federal I NO I EPA 31..

MarIanas
Commander
Navyf
MarIne CorpsluMarlanIIlI
'S.
Forces.
NlMIII I GU I 9 I o4IllI2OO3 I GroInl Water I Groundwater Monitor1ng I
Wei" NCS 5
NO I I Federal I NO I EPA 31.

Commander

:t.CorpsJ~:-~ 1~1'()(1212OO31 GroInlWamr ~~or1ngl


MarIanas
I NO 1 1 Federal I
I" 314 Flp rlliR IlIA. II .. IlPIl
NO I EPA 31.

fie: ConsoIldated Servicea_UQ,fl and NPDES_2llAp04_05011 "Um. rA detection b, rneChod 31. as atal8cI b, laboratory.
Sheet UQ,fllllld Drir'tkq W... +J vabl Is IIIalistIcaI esttmate Page 11 of 28
0... OWSoun:e ExpectMt r>.n:hIorlte
I
R"ubl~~1
lnatallallon EPA a.mpIe Collection SMIJI/ing Ragulatory
Servlce lltIte Raglan s.mpt111ll (Surf_or Dace of Next CommwIta or Referencn
[Name Loc:MIon PI8J1ned OrMrCbd SMIJI/Ing
Conducted GrounclwatM) Sampling (ppb)

ICommander
tuForc:ea,
.s. IAt aI Commander U.S. N8vlII Forces. MartaIlll8 8Ile&, c:uhc......alIon
NavyI
Martne Corps
Naval

IMartaIlll8
GU 9 1011212003 GroIn:IW. Grot.nIwater ~
WaI.NCS 7 NO Federal NO
I
EPA 314 resuta __ less than Ihe detection 11m•. So U- ~ -
sa "0". (NO)
reported

Navyi
ICommander
IU's.I I I
Naval
Martne Corps Forc:ea, GU 9 121712003
, GroIn:IW.
I Groundwater Mlriorfng
WeUNCS9' NO ,
, Federal I NO ,
EPA 314
Martanaa
Commander
NavyI r.s.
MIlrlne Corps Forc:ea.
I I
Naval , GU 9 1Q/1l12OO3 I GroIn:I W.. I~~ I NO I I Federal I NO I EPA 314
MariaIlII8
Commander
NavyI • lu,s.
N8v8I
MBme Corps Forcee, I I9I
GU 31'3112003 I GruI.Ild W.. I~~·1lI1 NO I I Federal I NO I EPA 314
Marl8Na
Cocnmand.-
I I~~ , ,

....
I I I
NavyI IU's.
MarIne Corps Forcee,
N8v8I
GU 9 3I'31rJ003 GroIn:IW.. I NO I I Federal NO EPA 314

Commander
NavyI \u.s. I I I
MarIne Corps Forcee,
N8v8I
GU 9 10I28l2OO3 I GroIn:I W.. I~tINCS~ I NO I I FedenII I NO I EPA 314
MartaIlII8
Commander
lU's. Naval ~ W"(ClearweI"
, ,
NavyI
M8rIne Corps I=' GU 9 31'3112003 Sufec:aW. F_ W..TreaImenI ,
PIari)
NO I Fedar8l NO I EPA 314

NavyI {Navy
PIAJIic Pearl twbor DrinkIng
GroIn:IW. WaterSyalem-~' NO
M8rln8 Corps Worb Canter, HI 9 51712002 I 'Federal; Slatel NO I EPA 314
Pearltwbor F'lInp Station
---
MIme (BVY
NavyI Corps WorbPoM:
Canlar,
Pearltwbor
II I I I
HI 9 51712002 Pearl System
GroIn:I W. Water twbor DrinkIng
- Red HII
F'lInp Slatton
NO
,
'Federal; Slate' NO ,
EPA 314

NavyI ,Navy
PoM:
MIme Corps Worb cemr.
PearlH.oor
II I I I
HI 9 51712002 Pearl twbor
GroIn:I WaIBt WaIBt:" DrinkIng
~Iawa NO Federal; Slate NO EPA 314

NavyI ILEMOORE
MIme Corps HAS 1 CA I 9 I 211212003 I I DrinklngW.
Sufec:a Water TreaImenI PIari Eftluert
flIdg722
NO Slate NO EPA 314
HAS lemoore. TIwe.. no rangee, no demoIIIon and no expostn to
Ihe c:hemlt:ala Il'IIhe mlrtilns @ Lemoore. Thera Is no reason to suspect
lI1ere Is sui or groundwater c:untamlnlIlIon
DrinkIng W.
NavyI !LEMOORE
MIme Corps HAS I I CA I 9 5/1412003 1 Sufec:a W. ITreaImenI PIari Eflluert
Bldg 722
NO State NO EPA 314

I I I I I DrinkIng Water
NavyI ILEMOORE 9 8f2Q/2OO3 Sufec:a Wetar TreaImenI PIari Eflluert I NO I I StIle I NO I
EPA 314
MIme Corps HAS CA
Bldg 722

NavyI ~ILEMOORE ~ 11/12120031


Marine Corps HAS Sufec:a WaIBt IT~~: I Hol

DrinklngW.
Eftluert
·~a14 ..,.-ti;
I StaIa I NO I
EPA 314

FIB: ConsolIdated ServIc:a8_uaR and NPOES_26Apr04_0SD11 -LImI 01 detec:tIon by melhod 314 sa etatad by 1aburaIory.
ShaeI: Ua.fl and DrinkIng W.. +J YfIIua Is atatiIlk:aI 8Illm8le Page 12 0/ 28
8eM~ I
I Installation StM81 EPA
N_
I 0...
S....pllng OWS-I I
ISurfKe or
Region Conducted . Groundwater)
S....ple CoIlec:tlon
I.ocMIon .
Sampling
Planned
Expected
O.e of Next
Regul.OIY
Drlwr CItecI
S-P'1ng
Perchlorate
SamplIng
(ppb)
RMUltsI~:mc.1 ComInent8 or R....._

Tap 01 - WfIb1t
INavy{ Navy PubliC
MarIne Corps Works Center
IL 5 211012003 Sdface Water Tre8lmenl PIllnt EfIIuent, NO FBdaraI <4" I EPA 314
Bulding 12

Navy! BANGOR
Marine Corps NAVSUBASE
WA 10 5I28l2OO3 GrounI WfIb1t Ctb1nalion 8.1009 I YES I 12I26l2OO3 I FBdaraI; Statel NO I EPA 314

Navy! BANGOR
td8rine Corps NAVSUBASE' WA ,
10 I 5/2812003 I GrounI WfIb1t I Ctb1nalion 8.1051 I YES t 12I26l2OO3 I Federal; Stalel NO I EPA 314

Navy!
I.t8rine Corps
Navy PublIc
Works Cef1ter I FLI 4
JackIlOrNlle
I 211012003 I GrounI WfIb1t I
WTP I, NAS JAX
(2181212) I I NO I Feder8I: I
Stat8; Loc8I
NO I EPA 314

N8Y)f ravy PublIc


I.t8rine Corps Works Center FL 4 211012003 GrounI WfIb1t
WTP 2, NAS JAX
NO
Feder8I;
NO EPA 314
(2181212) Stat8; Loc8I
Jack8cJnvII8

A seca-d IOUI1I 01 &llI1Iples _ taken Navy PubIlc Works een­

~
MIrInlI Corps
r~=-c:-
avy PublIc II I I I
FL
4 21101'2003 Gnu1d WfIb1t WTP, NS MAYPORT
12180734)
I I I I I
NO Federal;
State; Loc8I
NO EPA 314
Jack8orwIIeon.uy 22 ancI23, 2003, In lICCOl'dlInce wllh the EPA'.
1d8l8 cal far ... conI8mln8nts. Ths seca-d rounr:I Of SlIIl\JllInlI- found
to have the same "non-detect" -..s. The same EPA method (314.0)
_ USlId. alleveI8 of cllItect ancI f1IIlClI1Inll- the _. Urd bth8r
nolIce. IIww • no Int8nt to sample far pen:tbate at tt-.1clcalIOlW.

CA SNI • ZllnIc Sprtng NO EPA 314

SNI • ZInIc Sprtng NO EPA 314

Slriey Inck.cIe ~ fa' both NAWS CNna Lake ant NOlF Sen
NIcoIaIIsI8nd. CNne Lake • Ioc8ted In C8IlfomIa', MoI8V8 Dew11n a

Navyf INAVAlRWPNS\
Marine Corps TA CHINA
lAKECA
CA
II I I
9 s ample
1012312003 from ~ ocA8a8d
water at SN'-5WTP (Raw)
/
YES I
/'2/'0/2003 Federal; S1ale1 NO
I EPA 314
dry lake (pIayII) . . .,Ironmenl. USGS hBlIteetlId.-by s-1eILake
lusing EPA 314l1fld detected high concentrationl of rwUaIy 0CCIA1l1l
perctbate In the 8\I8POIlI8 depoe" found Ihwe. Same of the pen:Horalel
delectlone at CNna Lake may be due to nab.nIy occurIrV percIb8le.
The FBdaraI F8CII1y IderCiflcallon Number IFFlo) far San NIcoIae .eland
18: CA1f10090208.

NevyI INAVAIRWPNS sample cdected


MarIna Corps TA CHINA CA 9 11/1812003 from raw water at SNI-5WTP (Raw) NO EP" 314
lAKECA SWTP

NevyI INAV"'RWPNS sampled collected


Marine Corps TA CHINA CA 9 10I23l2OO3 81 firishBd water I SNI-5WTP (lr8ated) I YES , 12/tO/2OO3 IFederal; Slatel NO I EP" 314
lAKECA fromSWTP

NevyI ,NAVAlRWPNS
Marine Corps TA CHINA
lAKECA
CA 9 11/1812003
5ampled

8a~SW:-1 SNI-SWTP (1r8at8d) I YES LOO!OO3 IFeder8I; Statel NO I EP" 314

" Melhod 314 reporting 11m•• 4 P\lb


Fie: Consolidated Servlcee_uct.fl ancI NPOES_26Ap1G4_05011 "-una of detection by nIlllhod 314 81 etaIed by laborlIlory.
Sheet: ua.fl ancI 0r1nIWlg Wat« +J valull • el8lIlltlcaI8Itlmate Page 130128
I .na....latlon EPA
0... DWSource
&.npI. Collection
ExPllCted
Sempl'''ll Ollt. of Next Regu'IItOty Pen:h1oAte I8arnp111llll

8efvIc:e
I N_
&Ute
Region
(SurfKeor
8empll "ll
Conductecl Groundwllterl
loc:lItIon Planned
a_piing
DthlerClted Samplll1ll RHuIts AM/ytIcaIIhthodI
(ppbl
Comrnenta or R......~

N f !NAVAIRWPNS
~ Corps TA CHINA CA II 1on312OO3 SufaceWater SNI-TholMend Springs YES 12/1012003 Federal; State NO I EPA 314
I.Al<ECA
Nevyf jNAVAIRWPNS
Marine Corps TA CHINA I CA I II I 11/1812003 I SufaceWater I SNI-TholMendSprings I YES I 12/1012003 IFederal; Statel NO I EPA 314
I.AI<E CA
NAVAlRWPNS
:.::'.CorpsITACHINA I CA I II I 10I23l2OO3 I Suface Water I SNI-WRlm. Spring I YES I 01/1012004 IFederal; Statel NO I EPA 314
I.AI<E CA
NAVAlRWPNS
::t.CorpsITACHINA I CA I II 1 11/18120031 Suface Water I SNI·W~ Spring I YES , 01/1012004 t Federal: Slatel NO I EPA 314
I.Al<ECA
NAVAlRWPNS
::'.CorpsITACHINA I CA , II I 101112003 t Grculd W.. , Wel1S I YES , 0710112004 'Federal; Slate' NO I EPA 314
I.Al<ECA
NAVAlRWPNSI
::'CorpsITACHINA 'CA' II t 101112003 , Grculd W.. t Wei 18 I YES , 0710112004 'Federal; State' NO , EPA 314

=- ,lAKECA
INAVAlRWPNS

Corps TA CHINA 'CA'


lAKECA
II I 101112003 I Grculd WabIr t Wei 215 I YES I 0710112004 'Federal; Statel NO I EPA 314

=. NAVAlRWPNSI
CorpsITA CHINA
LAKECA
CA , II I 10I2l2OO3 , Grculd W... I Wei 77 I YES , 0710112004 t Federal; Statel NO I EPA 314

NAVAlRWPNS
::. CorpsITA CHINA I CA J II I 101112004 I Grculd W_ I Wei 28 t YES , 0710112004 ,Federal; State! NO I EPA 314

=- I.AI<E CA
NAVAIRWPNS
Corps ITA CHINA J CA , II

Navyl
LAKECA
rAVAlRWPI
, 10I2l2OO3 I Grculd W_ I Wei 30 , YES I 0710112004 IFederal; Statel NO I EPA 314

Manne Corps TA CHINA , CA I II I 10I2l2OO3 I Grculd Watf1t I Wei 31 I YES I 0710112004 IFederal; Statel NO I EPA 314
lAKECA
NAVAlRWPNS
:.::'.CorpsITACHINA I CA , II I 101112003 J Grculd Watf1t , WelSS1 J YES I 0710112004 IFederal; Statel NO I EPA 314
I.AI<E CA
NAVAIRWPNS
:.::'.CorpsITACHINA I CA , II I 101112003 J Grculd W.. , WelSS3 I YES I 0710112004 IFederal; Slalel NO I EPA 314
LAKECA
NAVAlRWPNS

=,-Corpsl~~ ,L I
II I 101112003 , Grculd W_ , Wei SII I YES ( 0710112004 (Federal; State' NO I EP 10l1S

Amrr ~ IurvlIy ° UCMR'" Drtr*I"ll w.ter :".


! I "
"

I
ARMY IFort Huaclu:al AZ. I II I l I3OI2OO3 I GIl I poIrj 0( emyfwIJII head yes 312412004 UCMR < 1 ppll EPA 314 r.R.1s 1 ppb (8 wei ~ Ie8ndlrV 10 the poIrj 0( erUy __ 88lIIpledl

SPIA monIlorlng wels


ARJI( I Fort Devens ( MA I 1 I ~a I GIl
I and Wei 0-1 (1rII1l81an1 Y.. Apr~ EPA reqlaJlt ~a ~a
EPA reqlaJ8t 10 sample Impact Ivea monIlorlng WIllIs and nearby
noncommlftty supply IranaIenI noncommlftty auppIy weJI 0-1
wei)

• Method 314 reporting lImii is 4 ppb


Fie: C<noIIdalsd ServIces_UCMR and NPOES_26Ap04_0s011 o°Unil 0( detection by rnelOOd 314 .. stated by laboratory,
Sheet: UCMR and ~ W_ +J value Is etalIsticaI8Itimale Page'40(28
0 ... DWSoun:e Expected PWcIIlorlIte
IMbIIldIon EPA 8ample Collection Sempllllll Reguilltory llampIlng/
8ervic4I N_ Slide Region SlIII1pllllll (8""-01 o.teofNext llampIlng R..uIts Comme..... or R....-.­
location P1MMd DriverCIUd AnaIytIc:aI MlIthod
Conducted Groundw"') 8lIII1p111ll1 (ppb)

Vtglnla
Radford Atmy 0epartmef1l 01 EPA 314.0 arel
f'IIW wat... lnIakea at two Req~ arel recelved approval from HODA 10 sample for pelcI40iates
ARMY AmmunllJon VA 3 ria SIN Vea lbel Env nla LCIMSIMS
Dmkng water plarU at drinkng waI8' plant wakes
Plent Resources verHicalJoo
reqtMSl
-
Fort George W8lIIr plant -plant tap.
ARMY KI 3 1011612002 GWISW )"lIS quarl8l1y UCMR <4.0' EPA314-M
Meade poW ofenlry
Fort George W8lIIr p1ant..pant tap.
ARMY KI 3 01/1312003 GWISW )"lIS quarl8I1y UCMR <4.0' EPA 314-M
Meade IlOinI of eRrI
Fort George W8lIIr pIant-planl '*P.
ARMY KI 3 4IW2OO3 GWISW )"lIS quarl8l1y UCMR <4.0' EPA314-M
Meade polnIdeRrl
Fort George W8lIIr planl-plant tap,
ARMY KI 3 71312003 GWISW )"lIS quarl8l1y UCMR <4.0' EPA314-M
Meade polnI d enIry
WlII.er pIant-plant tap. lab det8c:tIon 1tmI-• ugIl; planl doeed fa' repaInl CU1ng tnt hIIIf of
ARJKY FortMomle VA 3 12/412001 SIN )"lIS 5ep-02 UCMR <4.C· EPA314-M
DOlnl of enIry 2002
WaIIltr plant ..... tap.
ARMY FortMomle VA 3 911112002 SIN )"lIS Oct-02 UCMR <4.0' EPA 314.0
DOlnl of eRrI
WaIIltr plant -plant '*P.
AAMY Fort Momle VA 3 10l3Ol2OO2 SIN )"lIS Apr..()3 UCMR <4.0' EPA 314.0
llOinIofenlry
Water planl-planllap.
AAMY Fort Momle VA 3 04125/2003 SIN )"lI8 JI.n.03 UCMR <4.0' EPA 314.0
pOintofenlry
Water planl-planl tllP.
ARJKY FortMomle VA 3 6l3Ol2OO3 SIN yes 5ep..()3 UCMR <4.0­ EPA 314.0
IlOinI of 8nlrv
IlDI-. ­ plant
Water pIant-planl tllP.
AAMY Fort Momle VA 3 QI3I2OO3 SW no doeedon26 UCMR <4.0' EPA 314.0

----
pOint of III1Iry
SepOJ
ARMY Fort Dlx NJ 2 1212002 GW well head UCMR <4.0' EPA314-M
auartel "
ARMY FortDlx NJ 2 0212003 GW WIllI head auart81 Y UCMR <4.0' EPA314-M
ARMY Fort Dlx NJ 2 0512004 GW well head UCMR <4.0' EPA314-M

-
auart81 '"
ARMY Fort Dlx NJ 2 0812004 GW well head auart81 Y UCMR <4.0' EPA314-M
ARMY Fort Drum NY 2 1012Q12OO3 GW WIllI head 01I04I04 UCMR <4.0' EPA314-M
water treatmenI plant;
ARMY Fort~ GA 4 10/2112002 SW )"lIS 2/2412003 UCMR <4.0' EPA 314.0 samplng ... 8na1ys1a conducted by GA EPD-WPB
filratIon
water treatmenI plant;
ARMY Fort~ 'GA 4 2/2412003 SW )"lIS 0512003 UCMR <4.0' EPA 314.0 sampling and anaIysla CXlIlducted by GA EPD-WPB
filratIon
AIU6Y Fort~ GA
• 0512003 SW
water treatmenI plant;
filratIon
)"lIS 8I8l2OO3 UCMR <4.0' EPA 31•.0 S8mpIIng and 8na1ys1a CXlIlducted by GA EPD-WPB

AAMY Fort~ GA
• 81812003 SW
water treatmenI plant;
filratIon
no ria UCMR <4.0' EPA 314.0 sampling 8re1 anaIysla conducled by GA EPP-WPB

SlriIce Water Plant 8rd


ARMY Fort Gordon GA 4 41112003 GWISW )"lIS 611012003 UCMR <4.0' EPA 314.0' sampling and anaIysla conducl8d by GA EPD-WPB
Rang8 Comof Will

&.dlIce W8lIIr PIenl and


AAJKY Fort Gordon GA 4 611012003 GWISW
Range Control Will
)'88 9I9l2OO3 UCMR <4.0' EPA 31•.0 sampling 8re1 anaIysla conducl8d by GA EPD-WPB

&.dlIce W8lrIIr Plent 8rd


ARMY Fort Gordon GA 4 9I9l2OO3 GWISW
Range Comof Will
yes 121llt2OO3 UCMR <4.0' EPA 314.0 samplng arel anaIysla CXlIlducted by GA EPD-WPB

SlriIce Water PIenI arel


ARMY Fort Gordon GA 4 121Q12OO3 GWISW
Range Comof Will
no ria UCMR <4.0­ EPA 314.0 samplng 8rd anaIysla conducled by GA EPD-WPB
•••.-£1.. , ~.,

FIe: Consclldaled ServlcelUJCMR ... NPOES_26Apr()4_0s011 "UnI of det8c:tIon by meltIxI 314. 8lal8d by lallCltlItoty.
Sheet: UCMR 8rd ~ W8lrIIr +J value II stalistica/ estimate Page 15 0128
..me. 1'....N.....
I'..on Slat.
EPA
Region
Del..
S8mp/lng
DW80urce
(Surf_or
8unple Collection
location
S_pIIng
Planned
Expected
Dat.ofNed
Reguletory Pen:hIorel. Sampllngl I
Driver Cited SlIIIIPIlng Results AnalytIcal Method'
Comments or References
Conducted Groundwater) S_pllng (ppb)
Huntlll" ArmV
ARMY
AtfieId
GA 4 None GW ~ water WIllIs yes Ibd UCMR 0'8 I EPA 314.0 IPlamed for fuI\n operatlona

ARMY Fort Stewart GA 4 None GW ~ water W8IIa fulu'e Nol known UCMR 0'8 EPA 314.0 IPlamed for fubIe operatlona
operationa
ARMY Fort Riev KS 7 5/1612002 GW water IlIant Wake • VIIS 11/612002 UCMR <40· EPA 314
ARMY Fort Riev KS 7 1116/2002 GW water Dlant Wake no noll'8QUired UCMR <40" EPA 314
Fort Leonard
ARMY KS 7 8/112002 f!m SW lrBatmert plant yes 11/2112002 UCMR <4.0· EPA 314
Wood
ARMY I Fort Leonard
Wood
KS 7 11/2112002 f!m SW lrBatment plant I yes 1 3/112003 I UCMR I <4.0· I EPA 314

ARMY 1.Fort Leonard


Wood
KS 7 3/112003 f!m f!m lrBatment plant I yes I 8/112003 I UCMR I <4.0· I EPA 314

ARMY I Fort Leonard KS 7 8/112003 f!m f!m lrBatment plant no nolreqlWed UCMR <4.0· EPA 314
Wood

Aberdeen MlE Advisor) Finished ~ water I8II\PI8d for cay of Aberdeen. Water lICU'Ce"
ARMY 1 Proving Me 3 11/1212002 (Nt Finished water Yea 11/1912002 leYelln <11lPb EPA314-M GW ~ from aquifer Ird..- APG Ir8Ing field. APG lIlllI cay
GrolnI IiWhlId watlll" alternate sample coIecIIon. MRL .. 1 ppb.

FInIshed ~ water sempled for Cly of Aberdeen. W8I8r lICU'Ce ..

ARMY I
Aberdeen
ProIIklg
GrotnI II I I I
Me
3 1111llnOO2
(Nt FInIshed water
I Yea
1"~21::::1 0.81J IlPb +
I
IGW emanallng from aquifer Ird..- APG Ir8Ing field. APG lIlllI cay
EPA314-M alternate sample coIecIIon. MRL III 1 ppb.

+ J vaka ilia alallatlcal ettJmate


FlnIIIhed ~ water sempled for cay of Aberdeen. Water 8CU'C8 ..
IGW 8ll18Il8lIng from aquifer Ird..- APG lraq field. APG arQ cay
I I t 11~ t I I lm12OO2IMDE:~
I
Aberdeen
ARMY I Proving 3 (Nt Finished water Yea 0.81J ppb + EPA314-M alternate semple CClIec:tion. MRL III 1 ppb.
GrolnI Me I water
ftnIahed
+J vaka III a III8lIstiCal estimate

I I
FIMhed ~ water I8II\PI8d for cay of Aberdeen. W8I8r lICU'Ce ..
Aberdeen
ARMY I ProIIklg
GrolnI II I I I
Me
3
lm12OO2 (Nt Finished water Yea 1211Q/2OD2 !MDEAdv~
~:'er 0.76J ppb +
I EPA314-M
IGW ernanatk1g from aquifer under APG Ir8Ing field. APG arQ cay
aItem8le sample coIecIIon. MRL" 1 ppb.

+ J velue III • atatIlItIcaI eatlmat8

rlMhedern8Il8lIng
ARMY I
EII I MD
3 /1211Q/2OD2 1 GW Finished watlll"
I Yea
1,211712002/::::1
< 1 ppb
1
EPA314-M (Nt
~ WIller sampled for CIty of Aberdeen. Watel8CU'C8 III
from aquifer under NlG Ir8l1rc1f1e1d. APG CIty
alternate aample collection. MRL III 1 ppb. .
arQ

rinlshed ~ water sampled fa' CIty of Aberdeen. W8I8r 8CU'C8 ..


ARMY
II
Aberdeen
I Provtng
GrolnI I MD
3 11211712002/ GW Finished water
I I Yea
12fl8120021::::1
< 1 ppb
I EPA314-M GW ern8Il8lIng from aquifer under APG lniInllfleld. APG and CIty
alternate sample collection. MRL III 1 ppb.

Fintshed ~ water sampled fa' CIty of Aberdeen. Water 8CU'C8 is

ARMY I Proving
GrolnI I I I1~2 I I
Aberdeen
MD
3 GW Ftmhedwater
I Yea 12/3112002
MOE Advisory
leYelln
IiWhlId water
0.8OJ~ EPA 314-M
GW ernanalIng from aquifer under APG Ir8lIng field. APG am Cly
aItemate sample collection. MRL" 1 ppb.

+J vaka Is a sla1IstIcaI ettJmate

• Method 314 f9IlOIIIng lim... 4 ppb


FIe: CoosoIIdaled Servi:es_UCMR lIlllI NPDES_28Ap04_0SD11 ••L.inI of det8ctIon by rnelhod 314 as sIalad by Iaboratay.
St..-: UCMR 8nd ~ W8I8r +J velue III sIaIiIIiclIl eatImate Page 16 of 28
Service
.......

I I I
I Installation Slate EPA
Oat..
Sampling
Region Conducted
IOW Source
(8urf_ «
Groundwater)
Sample Con.ctJon
locatlon
Regulatory
Drlwr CJtecI
Perchlorate
Sampling Results ......:::'~
(PPb)
Comments or RIIfenInc:ft

Finished ~ water aampled for ClIy of Aberdeen. Wfb1t IQlI'C8Ia

fIIRr.fY I
Aberdeen
Promg
Grotnl II
M)
3
1'2m~ 1 GW
I Finished W8ler
I I Y. 1nJ2003
IMOEleYelln
AdviSOry
fll1ishedwater
O.61J ppb + EPfII314-M
GW em~ from aquifer tnler M>G traing field. M>G and cay
altemaltl aample collec1lon. MAl Ie 1 ppb.

+ J vlllue Ie e stallstlcal estmaIe

ARlit I Proving
Grotnl
IM) I I
Aberdeen
3 11712003
I I GW Finished W8ler
I
Y.
I
I
MOE AdviSOryl
1/1412003 , ..:: :m. < 1 ppb
I EPA314-M
IFIriahed ~ water sampled for cay aI Aberdeen. Water 1QlI'C81s
GW emanating from 8llulfer tnler APG tnliing tIeId. APG and cay
alternate 8lIIIlpie collection. MAl Is 1 ppb.

FIriahed ~ W8ler sampled for cay aI Aberdeen. Water IQlI'C8 Is

ARlIt I Promg

Grotnl
Aberdeen
I M) I I 3 1/1412003 I GW.
I
Finished W8ler
I I 1~112OO31:="!:1
Y. O.59Jppb+
I
rwemanatlng from aquifer tnler APG IralIng tIeId. APG and cay

EPA314-M aRemate sample coIeclIon. MAlls 1 ppb.

+ J value Ie • statisllclII estim8le


,Finished ~ water sampled for ClIy aI Aberdeen. Water 1QlI'C81s

ARlit I

E I M)I I 3 1/2112003 I GW
f
FIriahed W8ler
II Y.
112812OO31:="!.:1
O.7OJ ppb+
I
EPfII314-M
IGW 8I1l8I18lIng from 8lluller tnler APG lr8IIng field. APG and cay

alternate sample ~ MAlia 1 ppb.

+ J VIlIue Is e 818lIatIcaI88tlmete

ARlIt I IM) I I
Aberdeen
ProvIng
Grotnl
3 I I
1I28l2OO3 GW FIriahed W8ler
I I Y. 2/412003
I:="!.:I
< 1 ppb
I EPA314-M
rlGWnished8I1l8I18lIng
~ water sampled lor cay of Aberdeen. W8tfK IIClU'C8 Ie
from equlfer Iftfer APG tnlilng field. APG and ely
aIIem8le IIlII'Ilpie coIecllon. MAlis 1 ppb.

rlGWnished8I1l8I18lIng
ARlIt I
E IMl\ I 3
I I
2/412003 GW FIriahed water
II Y..
2/
11I2OO3
I:="!:1
< 1 ppb
I
EPA314-M
~ water sampled for cay of Aberdeen W81erllClU'C8le
from 8llulfer Iftfer APG tnlilng tIeId. APG end CIty
aIIem8le sample coIecllan. MAlis 1 ppb.

Finished ~ W8ler sampled for cay of Aberdeen. WBler IIClU'C8Ie

I I I IMOE AdviSOry GW emanating from equIfer under APG 1r8llng field. APG end cay

I I
Aberdeen
ARlIt I ProvIng
Grotnl
I M) I 3 2/1112003 GW Finished water YIl8 2/19I2l103 leYelln
fltjshed water
O.86J ppb + EPA314-M aIlemIItel8mple cdlectlon. MAlis 1 ppb.

+ J value Is • statisllclIIll8lImlllll

Aberdeen M) MOE AdviSOry


ARlIt I Proving 3 2/1Q12O()3 GW FkWhed water Yes 2I25l2OO3 leYelln 1.0ppb EPA314-M Delected BlliTlt aI MOE 8dvl8ory. MRlls1 ppb

Grotnl filished water

Aberdeen MOE Advisor) Finished ~ water sampled for cay aI Aberdeen. Weter source Is
ARlIt I ProvIng
Grotnl
Ml 3 212512003 GW Finished W8ler Yes 31412003 leYelln
filished water
< 1 ppb EPA314-M GW emanating from equlfer under APG IralIng tIeId. APG e'" cay
alternate sample coIeclIon. MRL Is 1 ppb.

Aberdeen
ARlIt I ProvIng
Gro&rd
IM) I I I
I
3 31412003 GW Finished water
I I YIl8 3I1112OO3IMOE:tj
IIriIhed weier
< 1 Rlb
I EPfII314-M
rlGWnishedemanaltng
~ water sampled for ely aI Aberdeen. Weter source Ie
from equIfer under APG tnliing tIeId. APG and CIIy
alternate sample cdIectIon. MRL Ia 1 ppb.

• Melhod 3141'l1JlO1tinll IImt Is 4 ppb


FIe: ConaoIId8led S8fvIGeIUJCMR .... NPDES_2Mpr04_0s011 ""Unt aI d8llIcllon by method 314 .. statad by Iaborlllory.
Shllet UCMR end ~W.. +J vlllue Is 11ati1tica188llmal& Pege 170128
8erYIce I
NMM
I I I I I
0..
InstaII8tIon Stile EPA Sampling
~1On Conducted
ow Source
(Surface or
Groundwlller)
Sampla CoIlKtIon
location
I Io:::'xtIR~ulItory Is.::=.
Sampling
Plenned s.mpllng Orlvllr CItH (ppb)
88mpIlngi
Analytical Method
e-rdsorRmr-

Finiahed 0drti1g water aampIed for cay 0I~. Water llOln& Is

~
Aberdeen
I ProvIrG
Grotnl IM> I I ~1100)3 I I
3 GN Finished water
I I ~1~31:h~1
Yes O.39J ppb + EPA314-M
GN ~ from aquifer lftIer 1'S'G traIIng field. APG IIIld cay
allemate I8IIlple colIectlon MRL Is 1 ppb.

+ J value Is a staliatk:al estlmllla


Finished 0drti1g water aampIed for CIy 0I~. Wafer llOln& Is

~ I
E M>I I 3
I ~1~ 1 GN
I FInIshed water
I I Yes
3rl5I2YJ3 I==~l O.5OJppb ..
I EPA314-M
IGN emanatlrG from aquifer lftIer APG lr8llng field. APG and cay
alternate sample colIectlon MRL Is 1 ppb.

+ J vabl Is a 8latlslk:aI esllmale

ElM> I I~I GW
IFInIshed 0drti1g water I8IIlpled for cay 0I~. W/JIBIllOU'C8 Is
~ I 3
I Finished water
I I Yes 41112003
l:h~ < 1 ppb
I EPA314-M GW emanating from aqulf8-lftIer 1'S'G Iraflng field. 1'S'G and cay
altwnata sample ooIIectlon. MRL Is 1 ppb.

E 1M>t GW I I /::':~I GW
rlnlahad 0drti1g water aampled for cay 0I~. W/JIBIllOU'C8"
ARMY I
I 3 41112003
f I FInIshed water V.. 4I8l2OO3 O.8ppb
I EPA314-M emanating from lIqlMw lftIer APG traIIng field. APG and cay
aItwnata sample ooIIectlon. MRL Is 1 ppb. .

Flnlahad 0drti1g water sampled for cay 0I~. WafM llOU'C8 Is


IGW
ElM> I I GW I I I 4I1~ I
8IIllVllIltng from aqulf8-lftIer APG Iraflng field. APG and cay
ARMY I 3 4I8l2OO3 Finished water V.. 0.34J ppb+ EPA314-M altwnata I8IIlple ooIIectlon. MRl" 1 ppb.
t 1:h'!:1
.. J value Is a sIatiIIIcaI estJmale
Flnlahed 0drti1g water I8IIlpled for cay 01 Aberdeen. WafM llOU'C8 Is
IGW
ARMY I
E M>\ 14I1~I 3
I I GN FInIshed water
I V..
\ 4rnmYJ31:h~1 O.4OJppb +
1
EPA314-M
amanatlng from 8qlMer lftIer APG Iraflng field. APG and cay
allwnata I8IIlple ooIIectlon. MRl Is 1 ppb.

.. J vakla Is 8 ItallslIc8I estimate

Aberdeen
ARMY I Promg
GRlInI IM> I I I I
3
4f22J2OO3
GN FInIshed water
I I ~ 1:=: 1
Vas < 0.5 ppb
I EPA314-M
IFlnished 0drti1g water 88mpled for cay 0I~. WatW llOlII'C8"
GW amarwllng from aquifer lftIer APG trallrQ 1ieId. APG and CIy
aItwnata I8IIlple coIectIon. MRL Is 1 ppb.

rlnlshed 0drti1g water .....pIed for cay 01 Aberdeen. Water llOlII'C8 ..


I I 1:1: :1
Aberdeen
ARMY I Promg
Grotnl I I M> 3
/
4fNlOO3
1
GW Finished water
I
Yes 5I6l2OO3 < 0.5 ppb
t
EPA 314-M GW llfII8I1lIlIng from aqUfer lftIer APG traIIng field. APG and CIty
8llemate I8IIlple coIecIIon. MRlIs 1 ppb.

Finished 0drti1g water I8IIlpIed for cay 01 Aberdeen. Water llOlII'C8 1&

E IM>I I I~1YlOO31::':'!:1
IGW 8Ill8flIItinlI from aqUfer lftIer APG traIIrQ field. APG and CIty
~ I
I I 3
I
5I6l2OO3 GW FInIshed water Vas 0.45J ppb"
I EPA314-M alternate I8IIlple coIecIIon. MRlIs 1 ppb.

+ J value Is a stalIsticaI astImata


Finished 0drti1g water ...-npled lor cay 01 Aberdeen Water sou:ce Is

EIM>I I ~1YlOO3 I I
MOE AdvI80Iy GW em8f1lllng from aquifer lftIer 1'S'G tralirV 1ieId. APG and City
ARMY I 3 GW Finished water
I Yes 5l2Ol2OO3 level In
&1IstllId water
O.48Jppb+ EPA 314-M 8llemate I8IIlple coIecIIon. MRL Is 1 ppb.

+ J value Is a statIstlcaI estimate

• MeIhod 31418pOf11ng IlrnI .. 4 ppb


fie: ConBolid8led ServIl»a_UCMR end NPOES_2Mp04_0SD11 -Unl 01 detecIIon by III8lhod 314 81 8IBl8d by taboraIory.
Sheat UCMR andOdrti1g Water +J ~ Is 8laIIIIticaI8IItImale Page 18 of 28
--­ _.. _--­
Servfce I
N.me
I I
lnatalilltion Stale EPA
Region
S8II'IpIlng (Soo-or
S8mpIe Collection
ue.tJon
S8II'Ipling
P"nned
O-w of Nut ·"'-.----1 S8II'IpIIng RMUhs
Drtver Cited ~Method
--
.......... Comments Of' R.rw.nen
ConcIue:Ud . Groundw-wrl Sampling IJlPbI

Finished Dt1nImg W8l1Ir B8l1\llIl d for ely of Aberdeen. W.. SOlmlIl


E'I~I
M>EAdvIsory
AAt6f I 3 5l2Ol2OO3 GW Finished W8l1Ir V.. 5/2711003 IlMIIIn 0.5ppb EPA314-M GW emMllting from equIfw undllr N'G lrlIIIng field. N'G and cay
I nI8hed water alternate sample collection. MRL II 1 ppb.

EI~I I:=~
I
I
FInishlId Dt1nImg water sampled for.eIy of Aberdeen. W.. SOlmlIl
AAt6f I 3 ¥l7a003\ GW FinIshed water V.. 8I3l2OO3 <04ppb EPA314-M GW 8III8llIItIng from equIfw undllr N'G lrIIiIng lIekI. N'G and ely
I I I \ altemate sample CXlIecIIon. MAL II 1 ppb.

FInishlId DItnlrcI water sampled for ely of Aberdeen. W.. lOIRe II

I I~1~ I:E~ I
IGW 8III8llIItIng from 8QUIfer undllr N'G baling lIekI. N'G and ely
AfU6'( I
E f~1 I I 3
I
8I3l2OO3 GW FinIshed water V.. <O.4Jppb + EPA314-M allernaIe sample CXlIecIIon. MRlla 1 ppb.

+ J YlIIkJe II a 8IatIstIcaI estlma\e


Finished DItnlrcI water sampled lor ely of Aberdeen. Water lOIRe II

AfU6'( I
E t~l I I 3 1111012003 GW
I
FinIshed W8l1Ir
II~17a0031:==:1 t
V.. 0.81Jppb+ EPA314-M
IGW 8Ill8IlIItIng from aquifer undllr N'G baling 1IaIcI. APG and ely
alternate sample CXlIecIIon. MRl. 111 ppb.

+ J value Ia a IlatIItIca/ eatlmate

E I~' I I 17fm I I
IArished DItnlrcI water sampled lor ely of~. WtbllOIRe II .
AfU6'( I 3 GW FInishlId water
I
V..
I~~I:=~ <0.5ppb
I
EPA314-M GW ~ from aquIler Inter APG lrIIiIng 1IaIcI. APG and cay
8IIemate sample caIectIon. MAL Ia 1 ppb.

AfU6'( I
E I~I I ~4n0031 3 GW
I
FinIshed W8l1Ir
I V.
I I
7/112003
I:=~ < 0.5ppb EPA314-M
rlGWnishlemanalIng
ld Dt1nImgfromwateraquIlersampledInterlorAPGelybaling
aIlemale _ple CXlIecIIon. MRlls 1 ppb.
of~. WtbI eotR8 Is
1IaIcI. APG and cay

Finished Dt1nImg water sampled lor ely of Aberdeen. WtbI eotR8 Is


IGW ~ from aquIler Inter APG lrIIiIng field. APG and cay
AAt6f I
EI~I 3
I I I7/111003 GW Finished water
I I 1:=:1 1
V. 7/812003 0.3OJ ppb+ EPA314-M aIlemaIe sample CXlIecIIon. MRL Is 1 ppb.

+ J value II a IlatIItIca/ 88llmale


FIniIIled Drtr*Ing W8l1Ir lllWIlpled for ely of Abardeen. Water eotR8 II

I I7/1~
IGW
AAt6f I
E I~I 3
I I I7I8l2OO3 GW FiMhed water
I
Vee
1:==:1
0.57 J ppb+ EPA314-M
emanating from aquifer ur-der N'G lrIIiIng field. APG and ely
8IIernale sample CXlIecIIon. MRlls 1 ppb.

+ J vaILe II a lIlallatlcal 8Sllmale

AAt6f I
EI~I 17I1~ 3
I I GW Finished water
I
Vee
17~1:E~ <0.5 ppb
I EPA314-M
IFInIshed Dr1nk1ng W8l1Ir sampled for ely of Aberdeen. Water IOln:8Is
GW emanallng from aquifer ur-der N'G IralIng lIekI. APG and cay
altemale sample collection. MRL II 1 ppb.

AfU6'( I
EI~I 3
I I GW I
7rnmm
Finished water
I
Vee
I 7aw2003I==~ <O.Sppb
I EPA314-M
rlGWnl hemMllting
ed Dr1nk1lgfromW8l1Ir sampledur-derforN'Gelybaling
aquifer
aIlemale sample coIec;tlon. MRL II 1 ppb.
of Aberdeen. Water IOln:8Ia
field. /\PG and cay

• Mslhod 314 reportI1g limit Ia 4 ppb


FIa: ConIolldaled Servlc8IUJC:MR and NPOES_26Apr04_0SD11 ""lim. of cIelection by method 314 • Itated by 18boralDIy.
Sh8et: UCIofl .... DmIdng W. . +J value IlIlat11t1ca/ aatImaI8 Page 19 of 28
$ervIce I N_
I I I --- I _.-_.-. I
.....lIatlon EPA
SUIte. R...Ion hmpllng
"' __..11••__ ..1
(Sumc. or
__ ••_ ........... -.&..._"
SMIpleCollectlon
locatlon
I I.. . . -- I R~u1ato1y I
Sampling
Plllnned Date
Perc!IIorate
Of Next Driver CItMI SempJlIlll
D ___ n__ (ppb)

8ampl1ngt

R..uIta AneIytl~1 Method COIIWMI1ta or Refefe_

FinIshed Drlr*lr1I"er ..mpled for ely of Aberdeen. W_ IOUIOIJ Is

ARMY I
EILCI 3
I I I7Q9J2003 GW FinIshed... , I I:=~
V.. 8/512003 O.50J ppb +
EPA314-M
GW emanattrv from eqUfer under APG traIInll field. APG em CIy

aItemale sample coIection. MRl III 1 ppb.

+ J value III 8 st8tistlc8IlIlIllmete

IFIn8tled Drlr*lr1I wet« .sampled for ely of Aberdeen. W_ IOUIOIJ Is

E\LCI I I I:=~I I
GW emaIl8llng from eqUfer under APG traling field. APG em CIy

ARM( I 3
I I I 8I5l2OO3 GW FInl8hed water V..
8/14Q003
O.54Jppb+ EPA 314-M alI8m8le sample collection MRL" 1 ppb.

+ J value III 8 st8tistlc8I88IImaIe


FinIshed 0rIItilg water sampled for ely of Aberdeen. W... acuce Is

ARM( I
E ILCI 3
I I I8/14Q003
GW FinIshed water
I 18/1~ I:=rl I
V.. O.40Jppb+
IGWemlll'lllllng from equIfer under APG traling field. APG end CIy

EPA 314-M alIemlIte sample collection MRL Is 1 ppb.

+ J value III 8 st8tistlc8I eslfm8le


FinIshed Drlr*lr1I.... sampled for CIy of 1dlerdeen. WfIIter acuce Is

E ILCI 18/1~ I I I~31::~


IGW
I
emanalllV from eqUfer under APG traIInQ lIllId. APG 8Ild CIy
ARMY I 3 GW FInl8hed weter Vee 0.34 J ppb + ·1 EPA314-M 8IlemlIIe sample collection MRlIs 1 ppb.

+ J velue Is e st8tistlc8I 88IImaIe

FlrMhed Drlr*lr1I wet« sampled for ely of Aberdeeo. W... acucels

ARMY I
EILCI 3
I~I I GW FinIshed water
I f I:=~I
Vee llIlII2OO3 O.llOJ ppb +
I
EPA314-M
IGW emaIl8llng from lIQlM8r under APG traling lIllId.. APG 8Ild ely
a118m8le 88Il1p1e collection MRL III 1 ppb.

+ J velue Is • 8l8llsIIc8I88lIm8te

ARMY I
EILCI 3
I I QI9/2OO3 GW
I
FinIshed wat..
I IWl~ 1~=::1 I
Ves <O.50ppb EPA314-M
IFInl8hed Drlr*lr1I wet« sampled for ely of Aberdeen. Water acucels
GW 8llI8nItIng from lIQlM8r under APG traIInll field. APG end CIy
aIlem8Ie pmple collection MRL Is 1 ppb.

ARMY I
E IMl\
3
\
9/1eJ2003 I I GW Fnshsd water
I I I::rl I
Ves
W2412OO3
<1 EPA314-M
rlnl8hed Drlr*lr1I.... sampled for ely of Ab8rII8en. Weter acuce Is
GW emaIl8llng from lIQlM8r uncI8r APG lr8llng lIllId. APG end ely
8lIem8t818111p1e coIectIon. MRlIs 1 ppb.

EI I I I I~AdvIlayI IFInl8hed 0rIItilg wet« 88Il1p1ed for ely of Ab8rII8en. Weter acuce Is
ARMY I
Ml \
3 W24Q003 GW FIn8tled wat.
I I Ves
I 10I9l2OO3 level In
....hedWlller
<1 EPA314-M GW emlII'llIllng from aquifer under APG traling field. APG end ely
alIernete pmple collection. MRlIs 1 ppb'

FlrMhed 0rIItilg weter sampled for ely of Aberdeen. Wat. acuce Is

EI I I I I 1 1:=rl I
IGW em8nallfV from aquifer under APG 1r811nD field. APG and ely
ARMY I 3
10I9l2OO3 GW Finished WlIler Vea 1W14flOO3 O.39J ppb+ EPA314-M alIernete sample colIecllon. MRL Is 1 ppb.
Ml/
+ J velue Is a 81811stlCal eslfm8le
Finished Drlr*lr1I water sampled for ely of Aberdeeo. Wet. acuce Is
GW emaIl8llng from aquifer under APG 1r8i1ng field. APG and City

E
~Advllay

ARJG( I
IMl!
3
!IWI4Q0031 GW
I Finished waI.
I Ves
1 10121r.ioo3 level In
flnished weter
O.5OJ ppb+ EPA314-M alIernete sample coIectIon. MRllII 1 ppb.

+ J velue Is 8 statl8tlc8l estimate

• Method 314 reporting \Iml Is .. ppb


fie: ConsoIldaI8d Servk;81UJCMR em NPOES_26Apr04_0s011 ··Lml of detection by method 314 sall8l8d by laboratory.
Sheet. UCMR end Drlr*lr1I WfIIter +J value Is st8tistlc8I 8Itim8Ie Page 20 of 28
service
I Ins:: Is"'l R:~ I p~~~~~ , ,,~~~~~-' ,
~ Collection
LocatIOn
I I I
ISlIftlJlIlno ~...-
Plan'*' Date
Regulatory
of Next Orlwr Cited Sampling
a ___,,_
Pen:hlonltl
(""blResulta
I I
~lngI
Melhod
Comments or Rn.re-

Finished ~ watw sampled for ely of AbenIeen. W" tIOlR8.

ARMY I
I~ I
Aberdeen

Proving

Gnu1d
3 11tV21~ I I GW FlriahBd WIller
I Yes
11tV2812OO31~~1 IlnIahed WIller
0.37J ppb+
I EPA314-M
lGW en18nal1nQ from aquWer lJl"C8" APG trd1g liBId. APG and cay
alternate sample collection. MRL. 1 ppb.

.. J value is a lllaIlslIcaI estimate

u
FiRahad DrinkIng watw sampled for ely of Abenleoo. Water tIOlR8 •

ARMY I
EIMlI 3 1tV2812OO3
I GW FIrished water
I Yes \1:& IMlE 0.21Jppb+
I EPA314-M
tGW emanating from equIfer lJl"C8" APG lnlllrg field. APG end cay
alamate ~ collection. MRL. 1 ppb.
1 1

r-m- _ . . _.. . -•. .,


IherellIIer flnIshed water
+J value •• alaIIslicaI asllmate
ProducIIon WIlls thIt ~ the elyof Aberdeen Wels 7, 8.9. end 10

IMl I I I I 1 I=~ IMlE AdW«y1

I
Abenleoo flI2OO2· 10.
ARMY I Provlng 3 GW Wei mad for ely
Production (CAP) Wei 1
Yea weeIdy 8InCe level In NO EPA314-M
pr--.t

r-m--.. _.. . _..


Gnu1d then flr*had water
NOTE: MRL has ranged from 5 ppb to 1 ppb. lppb MRI. was at8rted In
JUy 23, 2002. NO. < MRl
Productionwell& thBt ~tha ely of Aberdeen. Wels 7, 8. 9, end 10

E IMlI I I t~~
weeIdy 8InCe IMlevel
>E:In : ]
ARMY I 3
812002 •
~
GW
I
Wei mad for ely
Production CAP 2
I Y8S
then flr*had walIlr
NO

I EPA314-M
10.

NOTE: MRl he& rarved from 5 ppb to 1 ppb - 1ppb MRL__ 8lIIIted In

E IMlI I
weeldyurdl
MlEMvIsory
I-_m-_ . ._.. . -. ,.
July 23. 2002. NO. < MRI.
Production weII& lIlIIl ~ tha ely of AberdeerI. Wela 7. 8. 9. end 10

ARMY J 3
812002 ­
pr--.t
I GW
I Wei mad for ely
Production CAP 3
/
Yee / mid 2003. bI­
weeIdy 8InCe
level In
Iri&hed walIlr
NO to 1.8 ppb EPA314-M
10.

\__m__.. ._.. . -. .
then NOTE: MRl he& ranged from 5 ppb to 1 ppb - lppb MRlwas allIrted In
JUy 23. 2002. NO. < MRL
Production weII& thIt ~ tha ely of AberdeerI. WeI& 7. 8. 9. end 10

ARMY I
E IMll 3
1
&2002 ­
pr--.t
I I GW
Wei mad for City
Production CAP 4
I I~~ IMlE
Yee weeIdy 8InCe level In Advlsoryl NO

I EPA 314-M
10.

r-
then fV*hBd walIlr NOTE: MRllvIs rariged from 5 ppb to 1 ppb - 1ppb MRl was starI8d n

E IMl/ I ! I I~~ IMlE AdvIaayI m- _ . ._.. h_..·.


JUy 23. 2002. NO 1& < MRL
ProductIon weII& thIt ~ tha CIty of Aberdeen WeI& 7.8.9, end 10

ARMY I 3
6/2002.
pr--.t
GW
Wei mad for ely
Productlon CAP 5
/
Yes weeIdy since level n
then fV*hBd water
NO

I EPA 314-M
10.

NOTE: MRlIvI& ranged .-om 5 ppb to 1 ppb - 1ppb MRL _ alerted In

I-_m-- . .-. . .-,..,.

July 23. 2002. NO 1& < MRL


Productlon weII& thBt ~ tha ely of Aberdeen. Wela 7. 8. 9. end 10

AAMY J
E IMl! I I I 3
&2002 ­
pra&ert
GW
Wei mad for ely
Production CAP 6
I I~~ IMlE Advlsoryl
Yee weeIdy 8InCe level In
then Ilrishad water
NO

I EPA 314-M
10.

NOTE: MRl.IvI&~ed.-om5 ppbto 1 ppb.1ppb MRL _ started In

EIMl/ /~~IMlE~1
I-_m-_ . ._.. . _. . ,
July 23, 2002. NO Is < MRL
Productlon_s thal~thaelyofAberdeM. Wells 7.8.9, and 10

AAMY I 3
/
6/2002.
pra&ert
I IGW
Wei '-d for ely
Production CAP 7
I Yes weeIdy 8InCe level In
then fV*hBd water
NO

I EPA314-M
10.

NOTE: MR( hall ranged .-om 5 ppb to 1 ppb - 1ppb MRL was atarted In
JUy 23. 2002. NO Is < MRl

"Malt¥ld 314 reporting lima. 4 ppb


Fie: Cor1BolIdated Servlce&_UCMR and NPOES ~ OSDl1 ""lina of deIaclIon by melhod 314 as slatad by laboratory.
5heat. UCMR and ~ W" -.- +J value Is statlslIcaI estimaIe Page 21 of 28
Servlc. I InstaHatlon
N.,...
I I I EPA
Stat. R-V
lon
DN.
Sampling
Conduc:t.
I I
DW Sourc.
(Surf.... or
Groundwater!
a_phi CoIlec:tlon
LocaIIon
I I I I
S~I Ex~.
P1.n~ D:"o:.~xt P_h~
;:::~:Zt S8mpl~~..uItI Ane=II~ COfIlrltefQ or Rm-

or
Production.... that eupplythe City AberdeM. Web 7. 8. 9. end 10
Allen*n
ARMY I Proving
Ground I~I 3
I I I
612002 -
presenI
OW
Wfil head for City
Production CAP 8
I
I ~wBl
Ves
mid 2003. Ill- MOE Advisory
~ sInCe Jevelln
then flRshed water
I N0-1.5 ppb EPA31~
are IoC8ted on In&taIlatIon. Detections have 00... found In WllIs 3. 8. 9.
10.

NOTE: MRL has ranged from 5 ppb lo 1 ppb - 1ppb MRL _ started In
JlAy 23. 2002. NO is < MRL

ProductIon WIlIlsthat euppIy the City aI Aberdeerl. Web 7.8. 9. end 10

I=~I~E~I
I~ Iocaled on lnslaIIatIon. Detections have 00... found Inweh 3.8.9.
Allen*n
ARMY I Pnwing
Grnmd I I I I I
hIl
3
612002-
pr-.t OW
Wfil head for cay
ProductIon CAP 9
I
Vee W88kIy since Jevelln

water

then flRshed
NO-5ppb
I
EPA 314-M
NOTE: MRL has ranged from 5 ppb lo 1 ppb - 1ppb t.fiL _ aI8r1ed In
J<Ay 23. 2002. NO ill < MRl CAP 9 has 00... OCC8SIonlllly taken 011 line.

E Ihili I I .I I:
ProductIon.... that euppIy the City or Aberdeen. Web 7. 8. 9. en! 10
Iocaled on InstaIallon. Oelections have 00... found Inweh 3. 8. 9.
ARMY I 3
1
612002 -
pr-.t
I I OW
Wfil head for City
Production CAP 10
I Vee
2003.mlIll- MOE AdvISOry
W88kIy
mid
W88kIy sInCe Jevel WI
then fWshlld water
NO - 2.1 ppb
I EP"31~

NOTE: MRL has ranged from 5 ppb lo 1 ppb - 1ppb t.fiL _ aI8r1ed WI
JUy 23. 2002. NO Is < MRl
ProductIon .... that euppIy the cay or Aberdeen. Web 7. 8. 9. and 10

I I. I I~~I~~I
,_ IoC8ted on InstaIallon. Oelections have been found In weh 3 8 9
ARMY I
E (hili 3
l I2OO2 -
~
OW
Wfil head for cay
Production CAP 11
I Vee W88kIy sInCe Jevel WI
then ftnshed WIller
NO
I EPA31~
10.

NOTE: MRL hes ranged from 5 ppb lo 1 ppb - 1ppb MRL _ aI8r1ed WI
• ••

JUy 23. 2002. NO ill < MRl


AIr F_ PM:Nonte 8&mII8W •. NOTE FOR ALl AIR FORCE DATA: OSD r.quest for tIN- . 1nfonnalkIIl ........ by

IM.IC<lMa ~ ...... ~. wNch Inc:UIIe tIN. GW 8Ild ~ Some MAJCOMa


UCIIR and Drinldng WsW ......beck NIA - ttl"", not been delNd
8aIlcsdale EPAlUCMR;
AIlFace LA 6 3f2S~3 SW Narthple No NlA <4.0· EPA 314.0
AFB stale Barksdale does NOT have lo do the UCMR 8llIllIllI"lI per Fed8r8I
Barksdale EPAlUCMR; requAmeata. LA DHH produced a I8tter (rooo-speclflc: to Barksdale) that
All Face LA 6 4129~3 SW Narthple No NlA <4.0· EPA 314.0
AFB stale sald INs sampling WOtJd be handled at the wat.lreatmenl pIarD.
EPAlUCMR; HcMvvar. WI typlcaI state faaNon, the letter Is v-v vague. Barksdale he.
All Face Barkadale AFB LA 8 7/181113 SW Nor1Ilgate No N/A <4.0' EPA 314.0 accompllahed the sam~ lIf¥VlIy. At IeIlIt _ wi twve Ih8m WI case
state
EPAIUCMR; anyone (whllIIw state 01 ESOHCAMP 1n8pecIor) _lISka.
AIlFace Barkadale AFB LA 8 10I30I03 SW Nor1hgale No N/A <4.0" EPA 314.0
state
EPMJCMR;
AIlFace BealeAF8 CA 9 7129/1997 OW Wei" 1 No N/A NO EPA 300.0
DOHSUCMR
EPAlUCMR;
All Face BealeAFB CA 9 3I22J2OOO OW Wfil ", No N/" NO EPA 300.0
DOHSUCMR
EPAIUCMR;
AIlFace BealeAFB CA 9 7129/1997 OW WfiM"2 No N/A NO EPA 300.0
DOHSUCMR
EPAIUCMR;
AIlFace BealeAFB CA 9 3I22J2OOO OW WeI/"2 No N/A NO EPA 300.0
DOHSUCMR
EPAIUCMR;
AIlFon:e BealeAFB CA 9 712911997 OW WeI"3 No N/A NO EPA 300.0
DOHSUCMR
EPAlUCMR;
NrFace BealeAFB CA 9 3I22J2OOO OW Wfil "3 No N/A NO EP"3OO.0
DOHSUCMR
EPAIUCMR;
NrFon:e BealeAF8 CA 9 712911997 GW Wfil 114 No N/A NO EPA 300.0
DOHSUCMR
NrFon:e EPAlUCMR;
BealeAFB CA 9 3I22J2OOO OW W.. II4 No N/A ND EPA 300.0
DOHSUCMR
• Melhod 31418pOft1ng IImIIs 4 ppb
fie: ConsolIdaled 5ervIces_UCMR and NPOES_26Apr04_0s011 --unt or detection by melhod 314 as IIat8d by Iaboralory.
Sheet UCMR and Orlr*q W•• +J value ill ItalIslicaI estinal8 Page 22 28or
Oat.. DW80urce EJcpectecI Pen:lllorat8
lnetallatton EPA Sample ColIKtIon sampling RltlIulalory S8mpltnot
lIervlce State Samplll1ll (Sume. or Oat. ofN.xt 8ampllng R..ulta Comrnenb or Rer.,..­
Neme RltlIlon LOCIItIon P1anlMlCl DrlvwCIt8d AnaIytlc.l Method
Conducted Groundwater) SempUIllI (ppb)

EPAlUCMR:
AiFore;e 8eBI8AFB ell II 81512001 GW WeI.5 No HlA NO EPA 300.0
DOHSUCMR
EPAlUCMR:
AiFore;e Beale AF8 ell II 712911997 GW We" 8 No HlA NO EPA 300.0
OOHSUCMR
EPAlUCMR;
AiFore;e 8eBI8AFB ell II 312212000 GW WeM.8 No HlA NO EPA 300.0
OOHSUCMR
ell
EPAlUCMR:
AiFore;e 8eBI8AFB II 712911997 GW Wei, 7 No HlA NO EPA 300.0
OOHSUCMR
EPAlUCMR:
AiFore;e IleaIeAFB ell II 3l2212OOO GW Wei 11 No HlA NO EPA 300.0
OOHSUCMR
EPAlUCMR;
AiFore;e IleaIeAFB ell II 112911991 GW WeI.S No HlA NO EPA 300.0
DOHSUCMR
EPAlUCMR:
AiFore;e 1leaIeAF8 ell II 3l2212OOO GW WeI.S No HlA NO EPA 300.0
DOHSUCMR
AiFore;e
EPAIUCMR;
1leaIeAF8 ell II 112911997 GW WeI.1I No HlA NO. EPA 300.0
OOHSUCMR
EPAlUCMR;
AiFore;e 1leaIeAF8 ell II 3l2212OOO GW Wei, II No HlA NO EPA 300.0
OOHSUCMR
EPAlUCMR;
AiFore;e CemonAF8 NM 8 3I23I9lI GW 2 Yes Jan.Mar 04 46 EPA 314.0 S8mp/es taken after cNcllhl8liol L Resampled
atate
EPAlUCMR;
4I2llIlI9 GW Yes Jen.Mer 04
AiFore;e CemonAF8 NM
" 2

Yes
.tate
EPAlUCMR;
< 5.00' EPA 314.0 S8mpling IequHd by state of NM

S8mpling IequHd by state of NM


AiFore;e CemonAFB NM
" 4I2llIlI9

411ll1llO
GW

GW
2

Yes
J&n-Mar 04
state
EPMJCMR;
<5.0" EPA 314.0

AiFore;e CemonAFB NM
" 2 Jen-Mar04
alate
EPAIUCMR;
5.7 EPA 314.0 S8mp1ing requAd by 8llIle of NM

Ai Fore;e C8monAFB NM 8 7/1103 GW 2 Yes Jan-Mer04


,tale
<4.0' EPA 314.0 Sempling IequHd by 8llIle of NM

EPAIUCMR;
AiFore;e CemonAFB NM 8 3I23I9lI GW 3 Yes Jail-Mar 04
IIlate
< 5.0·· EPA 314.0 S8mpling IequHd by IIlate of NM

EPAIUCMR;
AiFore;e C8monAFB NM 8 4I28lOO GW 3 Yes Jail-Mar 04 <5.0" EPA 314.0 Sempling IequHd by 8late of NM
alate
EPAlUCMR;
AiFore;e C8monAFB NM e 411101 GW 3 Yes Jan-Mar 04
alate
<4.0' EPA 314.0 Semplq IequHd by state of NM

EPAlUCMR;
AiFore;e C8nn0nAFB NM e 315102 GW 3 Yes Jan-Mar04
alate
<4.0' EPA 314.0 ~ IequHd by 8llIle of NM

EPAIUCMR;
AiFore;e CemonAF8 NM e 2I20I03 GW 3 Yea Jan-Mar 04
state
<4.0' EPA 314.0 Sampling IequHd by 8llIle al NM
EPAIUCMR;
AiForce CemonAF8 NM 8 3I23I9lI GW 5 Yea Jail-Mar 04
.tate
< 5.0·· EPA 314.0 S8mpling J8llI*ed by alate al NM

EPAlUCMR;
AiForce CemonAFB NM 6 4I28lOO GW 5 Yes Jan-Mar04 <5.0·· EPA 314.0 S8mpling IequHd by alaIe al NM
slata
EPAlUCMR;
AiFore;e CemonAFB NM 8 2I20I03 GW 5 Yea Jan-Mar04 <4.0' EPA 314.0 S8mplq IequHd by IIlate al NM
state
EPAlUCMR;
AiFore;e CemonAFB NM 8 411101 GW 5 Yea Jan-Mar04 <4.0' EPA 314.0 S8mpling IequHd by state al NM
state
EPAlUCMR; S8mples taken after cNorlnalIon. Resampled; sampling required by state
AiForce CamonAFB NM 8 3I23I9lI GW 7 Yea Jan-Mar04 21 EPA 314.0 ,
state ofNM

AiFore;e CemonAFB NM 8 4/20199 GW • 7 Yes Jan-Mar04


EPAlUCMR;
state
<5.0·· EPA 314.0 Sempling requAd by alata al NM

EPAlUCMR;
AiForce C8monAFB NM 8 4I2llIlI9 GW 7 Yea Jan.Mar04
state
<5.0" EPA 314.0 S8mp/q IequHd by alaIe of NM

• MaItJod 314 reporting Ilmllis 4 ppb


FIe; ConsoIIdaIed servtces_UCMR and NPDES_28Apr04_0s011 "Urn. al deleclIon by melhad 314 .. 'tatad by laborlIlOry.
ShBel: UCMR and ~ W" +J vahJels atalIstlcaI8Illimate Page 23 of 28
DatM DWSou~ Expected Perel\lor.w
IMwlletion EPA Sample Collection Sampl1"ll R8fillu18tOfy hmpllngl
8eMca S~ hmpllng (Surface Of' Date of Next SempUng R..ults C-maOf'R......­
NIII1llI R8filllon L~on Planned Drtvw CItacI AnalytIC8l Melhod
ConcIuet8d GroundwatM) Sampling (ppbl
EPAlUCttfl; .
All Force CamonAFB NM 8 4I28lOO GW 7 Yea Jan-Mar04 < 5.0" EPA 314.0 Sampl~ I8Cl\*8d by staIll 01 NM
slllte
EPAlUCttfl;
All Force CllmonAFB NM 8 2J2O/03 QN 7 Yes Jan-Mar04
slllte
<4.0' EPA 314.0 Sampling requied by staIll 01 NM
EPAlUCMR;
All Force CamonAFB NM 8 411/01 GW 7 Yea Jar>-Mar04 <4.0' EPA 314.0 Sampling requilld by staIll 01 NM
state
EPAlUCttfl;
All Force CamonAFB NM 8 318102 GW 7 Yea Ja....Mar04
state
< 4.0' EPA 314.0 5ampl~ requilld by 8lale 01 NM

EPAlUCt.fi;
All Force CamonAFB NM 8 3123199 GW 8 Yes JaM.4ar04 <5.0" EPA 314.0 Sampling requhId by state 01 NM
state
EPAlUCMR;
All Force CamonAFB NM 8 6/15/00 GW 8 Yes Jan.Ma-04
stale
<5.0" EPA 314.0 Sampling requhId by illite 01 NM
EPAlUCMR;
All Force CamonAFB NM 8 2J2O/03 GW 8 Yes JaM.4ar04 <4.0' EPA 314.0 Sampling requied by stale 01 NM
state
EPAlUeMR;
All Force CamonAFB NM 8 411101 GW 8 Yes Jan.Ma- 04
state
<4.0' EPA 314.0 5arnpllng requHd by alate 01 NM
EPAlUCMR;
All Force CamonAFB NM 8 316102 GW 8 Yes Jan.Ma-04 <4.0" EPA 31•.0 5ampllng requied by state 01 NM
state
EPAlUCMR;
All Force CamonAFB NM 8 3123199 GW 12 Yes J8Il-Mar04 <5.0" EPA 314.0 sampling requied by state 01 NM
illite
EPAlUCMR;
All Force CamonAFB NM 8 6/15/00 GW 12 Yes J8Il-Mar04
state
< 5.00' EPA 314.0 Sampling requied by state 01 NM
EPAIUCMR;
All Force C8m0nAFB NM 8 411101 GW 12 Yes .Jan.Mlw 04 <4.0' EPA 314.0 Sampllng requied by state 01 NU
state
EPAlUCMR;
All Force C8m0nAFB NM 8 316102 GW 12 Yes .Jan.Mlw 04 <4.0' EPA 314.0 ~ TflClI*8d by state 01 NM
staIll
EPAlUCMR;
All Force C8m0nAFB NM 8 7/1103 qw 12 Yes Jan.Ma- 04 <4.0' EPA 314.0 sampling requied by state 01 NM
illite
EPAlUCMR;
All Force C8m0nAFB NM 8 5/5/00 GW 4A Yes JaIl-Mar 04
&late
8.4 EPA 314.0 sampling requhld by state 01 NM
EPAlUCMR;
All Force C8m0nAFB NM 8 411/01 GW 4A Yes Jar>-Mar 04
slate
<4.0' EPA 314.0 sampling requAd by state 01 NM
EPAlUCMR;
All Force C8m0nAFB NM 8 318102 GW 4A Yes Jan-Mar04
illite
<4.0' EPA 314.0 sampling requWed by state 01 NM
EPAlUCMR;
All Force CIImonAFB NU 8 2I20I03 QN 4A Yes Jan-Uar04 <4.0' EPA 314.0 sampling I'8IlI*!id by state 01 NM
state
DavI&-Mortban POE 18. Base Tower
All Force AZ 9 1015/00 GW No HlA HlA <4.0' EPA 314.0 sampled 5 OCt 00. net mandated by anv sI8l8 c.loc8I ~
AFB (Main Resorvoir)
All Force SMwAFB SC 4 10l22I03 GW WeIIOOadl#1 Yes 12/1103 EPAlUCMR 4.9 EPA 31•.0
All Force ShBwAFB SC 4 10l22I03 GW WeIlI\ead #3 Yes 12/1103 EPAlUCMR 5.5 EPA 314.0
All Fon:e ShBwAFB SC 4 10l22I03 GW Wellhead .. Yes 12/1103 EPAIUCMR <4.0' EPA 314.0
All Force ShawAFB SC 4 10l22I03 GW W8IIhead#5 Yes 12/1/03 EPAIUCMR <4.0' EPA 314.0
All Force ShawAFB SC 4 10l22I03 GW WeIIOOad If6 Yes 1211/03 EPAlUeMR <4.0' EPA 314.0
All Fon:e ShawAFB SC 4 10122J03 GW Welheadll7 Yes 1211103 EPAlUCMR 4.2 EPA 314.0
All Force
AIIFon:e
ShawAFB
ShawAFB
SC
SC
•• 12I29l2OO3
12J2912OO3
GW
GW
Wellhead III
Wellhead 1#3
No
No
HlA
HlA
EPAlUCMR
EPAlUCMR
5.1
5.3
EPA 314.0
EPA 314.0
All Fon:e SMwAFB SC 4 12I29l2OO3 GW Wellhead .. No HlA EPAlUCMR <4.0' EPA 314.0
All Force SMwAFB SC 4 12I29l2OO3' GW Welhed#5 No HlA EPAlUCMR <4:0' EPA 314.0
All Force ShawAFB SC 4 12I29l2OO3 GW Wellhead tI6 No HlA EPAlUCMR <4.0' EPA 314.0
AIIFon:e SMwAFB SC 4 12/29/2003 GW Welheadl#7 No HlA EPAlUCMR <4.0­ EPA 314.0
All Force LackIand AFB TX 8 511512001 GW WELLI#I Bldg 1013 No HlA EPAUCMR <4.0' EPA 314.0 Emypold

• MelhocI314 ~ Ilmk Is 4 ppb


fie: ConaoIIdated Servk:es_~ end NPOES_26Ap1l4_OSD11 "Urn. 01 delectIon by method 314 lIS stated by Iaboretory.
Shlllll: UCttfl end DrW<ing WaIw +J value Is statIatIcaI eatlmele Page 24 0128
Installation EP...
D.... DWSource
8ampIe CoIlKtIon SlIIlpllng
Exl*ted Regul«ory PwchIorale
BarnpllngI
Service Stat. SlIIlpllng ISurf_or DateolN.... SempllngRMub C-maorR.m-
N_ Region l.OC8tIon Pl8ll1llld Driver Cltecl Analytical Method
Conduc:Wd . Groundw.....) Sampling IPPb)

NtForce UCklandAFB TX e 11/512001 GW WELLn Bldg 5106 No NI... EP... UCMR <4.0· EPA 314.0 EtCrypoW

NtForce Lackland AFB TX e 5/2112003 GW WELLn Bldg 5706 No NlA EP... UCMR <4.0· EPA 314.0 ErCrypoW

NtForce Lackland AFB TX 6 11/512001 GW WE1.1..#3 Bldg 3106 No NI... EP... UCMR <~.O· EP... 314.0 ErCrypoW

NtForce Lackland AF8 TX 6 5/2112003 GW WE1.1..#3 Bldg 3106 No NI... EP... UCMR <4.0· EP"'314.0 ErCrypoW

NtForce Lackland AF8 TX e 11/512001 GW WELL14 Bldg 4070 No NlA EPAUCMR <4.0· EPA 314.0 erCrypoW

Nt Force Lackland AFB TX e 5/2112003 GW WELL14 Bldg 4070 No NlA EPAUCMR <4.0· EP... 314.0 ErCrypoW

Nt Force Lackland AFB TX e 5/2112003 GW WElU5 Bldg 4360 No NI... EPAUCMR <4.0· EPA 314.0 ErCrypoW

NtForce LacIdand AFB TX e 11/1012003 GW WELlIS Bldg 4360 No NlA EPAUCMR <4.0· EPA 314.0 ErCrypoW

NtForce LacIdand AFB TX 8 l /1512OO1 GW WELU16 Bldg 9062 No NI... EPAUCMR <4.0· EPA 314.0 ErCrypoW

NtForce lacldand AFB TX 8 11I5l2OO1 GW WELU16 Bldg 9062 No NI... EPAUCMR <4.0" EPA 314.0 erCrypoW

NtForce EdwardaAFB CA 9 1/16102 SW AVEKUNE No NI... EPAIUCMR NO EPA 314.0


NtForce £dwardaAFB CA 9 7/11W2 SW AVEKUNE No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardaAFB CA 8 1116102 . SW AVEKSUPPlY No NlA EPAlUCMR NO EP... 314.0
NtForce EdwardsAFB CA 9 1/1812002 GW c-3 No NlA EPAIUCMR NO EPA 314.0
NlForce EdwardsAFB CA 9 7/10/2002 GW c-3 No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 11 1/2412002 GW c... No NlA EPAlUCMR NO EP"'314.0
NtForce EdwardaAFB CA 9 7/1012002 GW c... No NlA EPAlUCMR NO EPA 314.0
NtForce Edward.AFB CA 11 1/1812002 GW NST·1 No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 11 7/10/2002 GW NST·1 No NlA EPAlUCMR NO EP... 314.0
NtForce EdwardsAFB CA 11 1/16102 GW NST·2 No NlA EPAIUCMR NO EPA 314.0
""Force EdwardsAFB CA 9 7110102 GW NST·2 No NlA EPAlUCMR NO EP... 314.0
NtForce EdwardsAFB CA 9 112412002 GW 5-2 No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 7I9l2OO2 GW 5-2 No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 1/1812002 GW 5-3 No NlA EPAIUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 7I9l2OO2 GW 5-3 No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 1/16102 GW 5-4 No NlA EPAIUCMR NO EP... 314.0
""Force EdwardsAFB CA 9 71fJl(J2 GW 5-4 No NlA EPAlUCMR NO EP... 314.0
""Force EdwardsAFB CA 9 1/1612002 GW s-s No NI... EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 7I9l2OO2 GW 5-S No NlA EPAlUCMR NO EP... 314.0
""Force EdwardsAFB CA 9 1/1812002 GW ~ No NlA EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 7I9l2OO2 GW ~ No NI... EPAlUCMR NO EPA 314.0
NtForce EdwardsAFB CA 9 1/1812002 GW 5-7 No NI... EPAlUCMR NO EP... 314.0
NtForce EdwardsAFB CA 8 911812002 GW 5-7 No NlA EPAIUCMR NO EPA 314.0
NtForce WIlIl SAFBlAF CA 9 1116J02 GW .. No NI... EPAlUCMR NO EPA 314.0
NtForce WIlIl ....FBlAF CA 9 7116J02 GW .. No 'NlA EPAIUCMR NO EPA 314.0
NtForce wan sAFBlAf CA 9 7110102 SW AVEKSUPPLY No NlA EPAlUCMR NO EPA 314.0
NtForce WIlIl sAFBlAF CA 9 1/16J02 GW B No NlA EPAlUCMR NO EPA 314.0
NtForce WIlIl sAFBlAF CA 9 1116102 GW C No NI... EPAlUCMR NO EPA 314.0
A'lForce WIlIl sAF8lAF CA 9 7/16J02 GW C No NlA EPAlUCMR NO EPA 314.0
A'lForce WIlIl sAF8lAF CA 9 1/16102 GW 0 No NlA EPAlUCMR NO EPA 314.0
A'lForce WIlIl sAF8lAF CA 9 7116102 GW 0 No NlA EPAIUCMR NO EPA 314.0
NtForce HanscomAFB MA 1 311/01 SW ErCryPolrC
.
No NlA EPAIUCMR <4.0" EPA 314.0

fie: Consolldated Servk:es_tJa.fi and NPOES_26Apr04_0SD11 ."Urn. or detecllon by methDd 314 as slated by IabaralAlry. Page 2Sof28
Sheet. UCMR and D1rti'lg W8llIr +J value Is staIlsticlII estlmate
DetM DW80un:. Expecl8cl P.n:ltlorat.
I..lallatlon EPA aMlple CohctIon hmpllng R-eutatory s.mpllngl
lIltrvlce SllU s.mptlng (Surf_or D8180fNut s.mpllng R..wta CClIIIIl*Ita or ReflnncM
N_ R-elon location Planned OrhrMCIMd AMIytIcal MlIthod
Conduct" GroundwlltM) Sampling (ppb)

All Force HlInscom AFe MA 1 8/14101 SW EntryPt*t No NtA EPAIUCMR <4.0' EPA 314.0

AIlForce HlInscom AFe MA 1 9(1101 SW EntryPt*t No NtA EPAlUCMR <4.0' EPA 314.0

AIlFaroe HaIllCXllll AFB MA 1 12/4101 SW Entry Pt*'t No NtA EPAlUCMR <4.0· EPA 314.0
AIlFaroe HIAFB UT 8 10I23l2OO1 GW WflMtI2. U· 782 NlA NlA EPAIUCMR <4.0· EPA 314.0
AltFaroe HIAFB UT 8 512112002 GW WflMlI2. U·782 NlA NlA EPAIUCMR <4.0· EPA 314.0
AltF_ HIAFB UT 8 10122/2001 GW Well #8. g.1570 NlA NlA EPAlUCMR <4.0· EPA 314.0
AltF_ HlAFB UT 8 10I23l2OO1 GW weatll. Ia. 7'» NlA NlA EPAlUCMR <4.0' EPA 314.0
AltF_ HIAFB UT 8 5l2Ol2OO2 GW WflMtll. 111·727 Nil. Nil. EPA/UCMR <4.0· EPA 314.0
AltF_ KIrIIand AFB NM 8 712712001 GW ERntPt*'t NlA Nil. EPA/UCMR NO EPA 314.0
AIt Faroe KIrIIand AFB NM 8 1/2412002 GW ERntPOlnt NlA Nil. EPAlUCMR NO EPA 314.0
Wrlght-
AIt Faroe
P----.AFB
Ott 5 2J8I02 GW Bldg 10855 (EP) Yes Jtj..()2 EPA/UCMR <4.0' EPA 314.0

WrtghI­
AIt Faroe Ott 5 1f29/02 GW Bldg 1.0855 (EP) No Nil. EPA/UCMR <4.0­ EPA 314.0
P8I\erwon AFB

WrtghI­
AIlFaroe
PlIlI8r8On AFB
Ott 5 2J8I02 GW Bldg 10857 (EP) Y.. Jtj..()2 EPAIUCMR < 4.0· EPA 314.0

Wrlgtt­
All Faroe Ott 5 7f2fM02 GW Bldg 10857 (EP) No Nil. EPAIUCMR <4.0­ EPA 314.0
P8ltlQOn AFB

Wrlgtt­
All Force OH II 2J8I02 GW Bldg 200851. (EP) Yes Jtj..()2 EPAIUCMR < 4.0­ EPA 314.0
P8ltlQOn AF8

Wrlgtt-
All Force
PlItterson AFB
OH II 7129/02 GW Bldg 2OO85A (EP) No NlA EPAlUCMR <4.0­ EPA 314.0

Wrigtt­
AIlFaroe
P8ltlQOn AFB
OH 5 216102 GW Bldg 20453 (EP) Yea J~2 EPAIUCMR 17.2 EPA 314.0

Wrlgtt­
AIlFon:e OH 5 7129102 GW Bldg 20453 (EP) No NlA EPAlUCMR <4.0­ EPA 314.0
P8ltenon AFB

Wrlgtt-
AIlFon:e
PatIerIlon AFB
OH 5 2IIlI02 GW Bldg 30172 (EP) Yes Jtj..()2 EPAlUCMR <4.0­ EPA 314.0

Wrlgtt-
AIlFon:e
Patterson AFB
Ott 5 7129102 GW Bldg 30172 (EP) No Nil. EPAIUCMR <4'.0­ EPA 314.0

WrtgIt­
AIlFaroe
Patt_AFB
OH 5 2IIlI02 GW Bldg 31229 (EP) Yes Jtj..()2 EPAIUCMR <4.0' EPA 314.0

Wrlgtt-
AIlFaroe Ott 5 7129102 GW Bldg 31229 (EP) No NlA EPAlUCMR <4:0­ EPA 314.0
Patterson AFB

Wrlgtt-
AltFaroe
Patterson AFe
Ott 5 2J8I02 GW Tap 1 (Area B) Yes .hI-02 EPAIUCMR <4.0· EP,o\314.0

• MeIhod 314 reporting Jimlls 4 ppb


fie: CoosoIldaIed ServiceIl_UCMR and NPOES_26Ap()4_0s011 ·'Unt of detedlon by melhod 314 as sllIl8d by Iaboralory.
Sheet. UCMR and Drlr1IOOg WBt.er +J vaILe Is statislIcaI 8Illm8Ie Page 26 of 28
Dat.. DW8ourc. Expected P.n:hloraw
l..llIIl8tlon EPA S_pleColf~1on 8wftpllng RegulatOlY Sampllngl
Service Stat. Sampling (Surf8ceOf Dale of Next 8wftpllng RMuIts COOlIMIIIa Of R~
Name R~ Location Planned DrlvwCIted Analytlcal MelhocI
ConductN Groundwat8r) Sampling (ppb)

Wrlght­
Ai Force OH II 1129102 GW Tap 1 (Alwa B) No HlA EPAIUCMR <4.0­ EPA 314.0
PalIllr1IOlI AFB

Wrlght-
Nt FllI'C8 OH II 2JlW2 GW Tap 19 (Alwa C) Y.. JtA.02 EPAlUCMR <4.0­ EPA 314.0
PIIlIlnon AF8

Wright-
Nt FllI'C8 OH 5 112902 GW Tap 19 (Alwa C) No HlA EPAIUCMR <4.0' EPA 314.0
PIIltersoo AFB

Wright-
Nt FllI'C8 OH II 218102 GW Tap 20 (...... A) Y.. J~2 EPAlUCMR <4.0' EPA 314.0
PIIltersoo AF8

Wrlght­
Nt Fan:e OH 5 1/29102 GW Tap 20 (Alwa A) No HlA EPAlUCMR <4.0­ EPA 314.0
PlIll8rMln AFB
V-.IenIlerg
Nt Fan:e CA 9 1111101 GW SanAdoBD W~ ... No HlA EPAlUCMR NO EPA 314.0
AF8
V-.IenIlerg
All FllI'C8 CA 9 12/1102 GW San AdoBD Welhead ... No HlA EPAlUCMR NO EPA 314.0
AFB
V8Ildenberg
Nt FllI'C8 CA 9 511103 GW SanAnlonlo W~'" Y.. Nov~1 EPA/UCMR NO EPA 314.0
AFB
V8Ildenberg
Nt FllI'C8 CA 9 511103 GW San AnIonIo WeIhead ... No HlA EPAIUCMR NO EPA 314.0
AFB
Vandenberg
Nt FllI'C8 CA 9 1111101 GW San AnIonIo Welhead IS No HlA EPAlUCMR NO EPA 314.0
MB
VandenIl8rg
Nt FllI'C8 CA 9 12/1102 GW San AnIonIo Welhead IS No HlA EPAlUCMR NO EPA 314.0
AFB
Vandenberg
Nt FllI'C8 CA 9 511103 GW San AnIonIo WeIhud IS Y.. Nov~1 EPA/UCMR NO EPA 314.0
AFB
VIIIldenbelv
Nt FllI'C8
AF8
CA 9 511103 GW San AnIonIo WeIhud IS No HlA EPA/UCMR NO EPA 314.0
VandenIlelg
Nt FllI'C8
MB
CA 9 1111101 GW san AdoBD Welhead lJ8 No HI" EPA/UCMR NO EPA 314.0
Vandenberg
Nt FllI'C8 CA 9 12/1102 GW San Aftcno WeIhud lJ8 No HlA EPA/UCMR NO EPA 314.0
AFB
VandenIlelg
Nt FllI'C8
AFB
CA 9 511103 GW san AnIonIo WeIhud lJ8 Y.. Nov~1 EPAIUCMR NO EPA 314.0
V8Ildenberg
Nt FllI'C8 CA 9 May-03 GW San AnlonIo WeIhud tI6 No HlA EPAIUCMR NO EPA 314.0
AFB
Vandenberg
Nt FllI'C8
AFB
CA 9 Nov~1 GW san Antonio WeIhud tIT No HlA EPMJCMR NO EPA 314.0
Vandenberg
NtNlrce
AFB
CA 9 Dec-02 GW san Antonio WeIhud tIT No HlA EPAIUCMR NO EP"314.0·
Vandenberg
Nt FllI'C8 CA 9 May-03 GW SanAnlcno Welhelld tIT Y.. Nov~1 EPAIUCMR NO EPA 314.0
AFB
Vandenberg
Nt FllI'C8 CA 9 May-03 GW San AnIonIo Weu--s #1 No HI" EPMJCMR NO EPA 314.0
AF8
scmA
Gel&- Reav & Wellhelld
Nt FllI'C8 F8IrchIId AF8 WA 10 3/13101 GW Y.. ~1 EPAlUCMR <4.0 314,515.3.524.2,
#2 No fIriw ......pllng reqund • coneiIlts of orly 2 roums for dsta
625.2
coIIectian PI'JlC)lIM for lUn ~ f they blIcQme R"lloAated
Geiger Resv & Wellhelld SOW" CoftamlrwliB.
Nt FllI'C8 F8IrchIId AF8 WA 10 8121101 GW No HI" EPAIUCMR < 4.0 314.515.4.624.2,
#2
. . ".­ 525.2
FIe: ColwolicIlQd Servicea_uet.fl and NPDES_26o/Ipr04_OSD11
Sheet: UCMR and ~ Watt.
· Lin. of deIsction by method 314 ...... bylaboralory.
+J valle II etalIBticaI estm818 Page 21 of 28
8ervlce I 1.-u.tIon

I I
lWM Sute REPAIon 8unpIing
0.. f
(Sum- or
Conduc:t4d Groundw8t8r)
lOWe- I Sample Collection
Locetlon
1=lo::::..l=l..:::-n - I
llampHng (ppb' AnlIIytIceI Method
C~orR-'-"

1sl rcxnI of 88lI1p1fng _ COI1lb:ted In JU 01. 2nd roond of eampIIng


Ale Foroe I Tr8YII AFB I CA I 9 I 1118101 I GW I 3-Wellheads I No I NfA IEP~;I <4.0· I CAOHSIEPA314 Iwa6 waived on 4 Sep 02 by DHS. exceplIon of C/mmun VI. No ftriW
68Illpllng requhld • consI6ts of oRy 2 rourdlI for data cdIectiOO fU1lOlI8lI
for future U68 If lhIIy become Reo*ed CorQmlnarts.

• Melhod 314 repa1Ing lIml III 4 JlIlb


fie: Con6oIiIaled Servlces_~ and NPDES_26AplO4_0s011 -uma of delectIon by III8Ihod 314 88 8lBted by Iaboralory.
SI1IlIt ~ and ~ W.. +J vBlue lll1la1lslica18l1l1ma1e P8II8 28 of 28
Regutetory

8trvIce I F.cllity I 81tt I


Sourc. of PerdlIond. (Oper8tlona,
Equipment, ActMU"1 I
Locrion
Detected
(IIteII_1
I I81m.. . I I~ I
~
Nwnbe of Numbw of
Colltcttd
Rang. of
c_nnuem.
Detected (ppbl
M-UW: Potential Pethw-rl-I of ~~aea~
I permIIlnQ'
~.
Fundl"ll TVII'

sampling
~)
I I I I

Nlrty I M...... Corpe Ptrc:NorMe llIIWr· EnvIfvnnlent8I R-*or.uon • Clunup. Other


I I I I I I
Navy' IALLEGANY
MarIne Caps LABORATORY
8ALUSTI~ SIll 1: 1EW08 ~-u.. Test and
EvaUIIIon: MatU8cUIng;
DeveIopnert,
cI dIspotaI Grou1dw8ler 2 2 14.0 -76.2
NQne. Groundwater II lrikely III
EPA 314 dv8Ioped for ~Ing water. 8nd IFeder8I; SlaIe I
oeRP
waste product 08811 conIroIed by prql8rty_.

Nevy' IALLEGANY 8ALUSTICS


MarIne Caps LABORATORY
I I~' Development, Test 8nd
Site 1: 1EW10 Evablllon; M8RJfIIcIu1ng; disposal cI Grou1dw8ler 4 4 1-48.0 - 329.0
Nona. Groundwate
EPA 314 dY8IooBd for ~ _er. and IFederaI: Slate I
DERP
-product

Navy' IALLEGANY BALUSTICS


Mlwna Caps LABORATORY
I I~' ~ Test 8nd
SIll 1: 1EW12 EYIIkAIlIon; M8RJfIIcIu1ng; dIspotaI cI Grou1dw8ler 3 3 315.0 - 5,910.0
None. GroundwaIer II LriI<eIy to
EPA 314 dv8Ioped lor drIridng water, and )Federal; SlaIe I
DERP
waate product use Is conIroIed by prql8rty_.

Nevy' IALLEGANY BALUSTICS


Mlwna Caps LABORATORY
I r-u.
,-product
DlMllopmeri. Test and
SIll 1: lEW14 EvaIu8Ilon; ~; diIpoBaI cI Grou1dw8ler 4 4 626.0 - 7.930.0
None. Grotnlwater II urlikAlly to
EPA 314 dv8Ioped for drIridng water, and IFederaI; SlaIe
.... 11 conIroIed by PfOI*tY-.
I
DERP

Navy' lALLEGANY8ALUST1CS
Maril8 Caps LABORATORY
I I~ ~ T..and
i

SIte 1: 1EW16 EYllIuaIIon; ~; dIspotaI cI Groundwater 7 7 3.500.0 - 34,900.0


None. GraundWllltr Is IriIuIIy to
EPA 314 dYeIoped for drIridng water. and IFederaI; Slate I DERP
-product use II conlIoIed by PfOI*tY-.

NIlYy' IALLEGANY BAWSTICS ISIll 1: 1EW18 1Research. Developmert, Test and


Nona. GrounlIw8ler Is ~ to
I

=
MarIne Caps LA80RATORY Evalu8llon; Marofacblng; disposal cI Groundwater 4 4 34.2-72.0 EPA 314 dYeIoped for drIridng _ter, and Ifederal; Stale OERP
waate product use II conlIoIed by PfOI*tY _.

IResearch' Development, TelIl and Nona. Groondwater II unIIk8Iy III


CorpsI~:USTICS ISile 1: 1EW21 Evaluation; Marofacllmg; disposal at Groundwater 1 2 NO-8.9' EPA 314 dYeIoped for drIridng water. and Ifederal; Slate
U8e II conlIoIed by PfOI*tY _.
I DERP
waate product

Navy I JALLEGANY BALUSTICS


Mlwna Caps lABORATORY
I research. Development, Test ard
SIe 1: 1EW30 Evaluallon; Marofacllmg; disposal at Groundwaler 4 4 190.0 - 1,110.0
Nona. Groondwater II unIIk8Iy III
EPA 314 dYeIoped for drIridng water, and IFederal; SlaIe I DERP
lwasle product Ul811 conlIoIed by PfOI*tY _.

=CapaI~:STICS ISile1:IEW31
research. DeveIopmenI. Test and
Evaluation; Mardacllmg; cI
dIspotaI Groundwater 3 3 14.0 -50.0
None. Grolntwater Is unlkely to
EPA 314 dYeIoped for drIridng water, and IFederaI; Stale I DERP
UI8 II conlIoIed by PfOI*tY _.
lwaste product
Navy' IALLEGANY BAlUSTICS ISite 1: I~ Development, Test and Groundwater 1 1 26.7
None. Groundwater It IriIuIIy to
EPA 314 dYeIoped for drIridng water, and IFederal; SlaIe I DERP
MarIne Corps lABORATORY lGroundwater10 Evaluatlon; Marofacllmg; disposal at
Iwaste product UI8 III conIrolIed by prql8rty _.

Navy' .1~GANY8ALUST'CS IS1Groundwater11


MIme Caps lABORATORY
f.'­-dl,Developmenl,
le 1: EvaIuatlon; ManJfactI.mg;TelIland
dIspotaI cI Grou1dw8ler 8 8 7.5- 55.0
Nona. Groundwater III IriIuIIy to
EPA 314 dv8Ioped for drIridng water, and IFederaI; Stale I DERP
waate product UI8 III conIrolIed by PfOI*tY-.

Fie: PercDallu~.S_j\plt2217_
Sheet: Env Restorallon-CIea Olher • Melhod 314 ~ lint Is 4 ppb Page 1 of 73
Regulatory

8ervIce I Facility 1 .. I ofP~


SourceEqu\pmlnl, ActMtIM)
(OperatloM, I loc.uon
Detec:ted
(Media'
I I I
Number of Numberof
DstactI_ Range of
SampMeCOllCllfltlatlon.
CoIlec:tecI DetacMd (ppb)
I ~ I Potential P......ay<.) of ~lr~.
Im-st (CIeInJp
equested I FUlldIft\l Type
~,
sampling
requIremerts)

Navy / IAlLEGANY BAUJST1CS


MarQ Corps I.AflORATORY
ISle
1:1'~ Dev~. T.... ard
1Gmnlwater32 EvaIulItIon; MarI.JIadI.mg; dIspou\ af Grot.ndwater 4 4 14.8 • 33.0
None. G<oundwater ill ooikely 10 bel
EPA 314 dveIoped far ~ water. and Federal; State I OERP
iwate product use ill conlrolled by property owner.

Navy/ IAllEGANYBAUJsncs ISle1:AS01. liR8eealch.DevaIopmert. Test and


MarQ Corps I.AflORATORY S803 EV8IuaIIon; MarI.JIadI.mg: dIspou\af Sol 1 1 cao EPA 314 None. IFederaI: StaIB I DERP
wate product
NlIV)' / AllEGANY BAUJSTlCS SIe 1: AS01- ~ DevaIopmert. Test and
Marina Corps lABORATORY S804 Evabltlon; ~; dIspou\ af ISoi 1 1 I 1 1 98.0 I EPA 314 INone IFederaI; SlaI8 I OERP
- WlIIt8 product
Navy / AU.J;GANY BAl..USTlCS SIe 1: ASC1- R-m, Clev8Iopmert, T88l and
Marina Corps lABORATORY S805 ~~; dIspou\ af ISoi 1 1 1 1 1 880.0 1 EPA 314 INone IFederaI: State I DERP

Navy / 'AllEGANY BAUJSTICS JSIs 1: AS01.


M8rlne Corps lABORATORY
IR~ D8v8loprrIed, Tell 8f1lI
SB08~MenAcl1n'G; dIspou\ af ISoi I 1 I 1 1 95.0 I EPA 314 INone IFederaI; State I OERP

NlIV)' / fAllEGANY BAUJ5nCS ISlte1: ASC1-


MerlneCorpslABORATORY 5814
IR.-clI.
~~;dIspou\af
Developmert Tell end
ISoi I 1 1 1 1 120.0 1 EPA314 INone IFederaI:Slate I OERP

NaYy/ IAllEGANYBAUJSTlCS ISlte1:AS01- J~~'Tell end


MerlneCorps LABORATORY 5818 1~~;dIspou\af ISoi 1 1 \ 1 1 62.0 I EPA314 INone IFederaI;SIele I OERP

Navy / IAllEGANY BAlLISTICS ISIs 1: AS01-


M8rlne Corps I.AflORATORY 5817
IR. -ch. DeveIopmeIt, Tell end
EvaIulItIon; ~; dIspou\ af Sol 2 2 220.0 - 2.500.0 EPA 314 None IFederaI; Stet. I OERP
If'W8lIt=·.::::product=:::.....------t----+---+----lf-----+--~-----
Navy / IAllEGANY BAWSTICS ISIs 12: AOCN-
M8rlne Corps I.AflORATORY 31
'II~DeveIopmer1, T88l and
EvakJ8llon: ManJIIilcluing; disposal af Groundwater 1 1 18.0
Nona. GroInIwat8r ill t.nIl8Iy 10
EPA 314 dv8lopadfar ~ water. end lFederaI; Stal8 I OERP
W8lIt8 product use II conlrolled by property owner.

Navy/ IAU..EGANYBAlUSTlCS /Sls12:AOCN-I R-m.DeveIopmert, T88land None. GnJundwalerllt.nll8lylo


Mama Corps t.ABORATORY 33 Evllluallon; ~; dlsposal af Groundwater 1 1 (g.0 EPA 314 dveIoped lor ~ water, 8f1lI Ifederal: Slate I OERP
IW8lIt8
product use II conlrolled by property owner.

NaYy / IAllEGANY BAWSTICS ISle 12: AS12.


Mama Corps t.ABORATORY 12MW08
'1IRasearch.
DeveIopmert. Test and
EvaIUalIon; MaraJfaclu1ng; disposal af GrounlIwat. 1 1 13.0
None. Groundwel.1I t.nIl8Iy 10
EPA 314 dv8lopad far ~ water. end \Fadar8I: Stal8 I OERP
waste product use II conlrolled by property owner.

Navy / AllEGANY BAUJSTICS SIe 2: R-m. DeveIopmert. T9Sl and . Nona. GroundwlIIer 18 t.nIl8Iy 10
MlnlaCorps I.AflORATORY 2GroIRIwaler02 Evllluallon; Maf'llfactuirV; disposal af G<oundwater 1 1 10.8 EPA 314 dveIoped far ~ water, end IFederaI; State I DERP
wast8 product use II comolad by property owner.

Navy/
MarQ Corps CORONADO NAVBASE NASHI IR SiI8 9 DemIlalizallolYDls Groundwater 0 5 NO EPA 314 None State OERP

INavy / CRANE Navy Sin- Sampled wIh FY


MarQ Corps Water Career Old Jeep Tral DemIllarizalIcn Sol 5 5 24.0·470.0 EPA 314 None Federal 2001 DERP
funding

fie: Pen:OllbLAISvca_AprI2211_

Shaet Env ResloraIIon-Cle. 0Ihar • Malhod 314 reporlq 1im11I4 PJJb Page 2 af 13

Regul8tory

8ervIce Feclllty 81W


SOurce 01 ~chlor8te (Opendlona,
Equipment, ActIvItIM)
location
o.tec:t.d
(MedIa)
~ I NlIIIIWoI
hmp/ee
CoilectecI
f C~
Rangeol
Detected IPPO)
I I
~ PclIenlIaI Pldhwaria) of ~~~
pennIIlno I Funcllng Tyaw
~.
aampIrQ
requi'emenlll)

Navy I ICROWS LANOING 51. 11 DIsposal /DemllarlzBtionlDllposal


Marnl Corps FUGHf FACIUTY PIs
Groundwater o 11 NO EPA 314 INone Stale BRAC

CADHS

NtNy I IFormer
MIme Corps Slallon, EI Toro
lEI
MarIre CarPI At foro
Basewkfe
None 1delUled. Bes8wIde grotnlw8l8r
ClIIl8IlItIlIt wtlh olf-lllllllcn grotnIwaIer Groundwater 73 188 NO -111.0'
Cl04METH; SoI.rce: ProductIon Wela MedIa
ClCMAETH; Exposl.nt: Groundwater Human
EPA 300.0: Receplar: Producticn Wei
Federal: State BRAC
COl It:8IlIJ lIlIoI.
EPA 314; Coneumer
EPAM300.0

None 1derdIed. a-Ide (jfOlnlwlIl8r


NINII IFormer ~ CarPI At lEi Toro ClIIl8IlItIlIt wtlh olf.-.uon groundwater Sol o 3 NO EPA 314 INone :FederaI; State BRAe
MarIne CarPI St8lIon, EI Toro BeeewIde
WI...............

N6Ny I IFormer MarInI CarPI At


Mertne CarPI Slallon, EI Toro IAnomaIy ....... 3 IPelcNoral8 nat deleclllChtth..IB. GrolnIwIIter o 25 NO EPA 314 INone Federal: Stale BRAC

Navy I IFormer MarInI CarPI All


Mama Corps StalIon. EI foro IIRP Sb 2 Sh Ie a 1endII. Graundwater 118 217 NO- 20:7'
CADHS I
Source: PolenII8I. ProduclIon Wei
MediI
01 Exposl.nt: Shallow
Cl04METH: Grocntwal« Human Receptor:
ClO4METH: Pot..... Water Coneumer. It Ie nat
EPA 314; mtcIpeted l!Bl groundwater wIlhIn jFederaI: State
BRAe
EPA M3OO.0 1hB ... bound8Iy wit be ll88d lor
benIlIcIaIln IhB a-tlIe fubn.

Sol contamlnlltlon QQ1 poterCt8Iy


'leech ~ groundwater. SheIow
ClCMAETH; (GlOundwater c:cUS be ll88d for
N6Ny I IFormer MarIre CarPI All IRP SIte 1 TralrirQ: DemIlarlzalloWDlsp Sol 4 85 ND- 320.0' EPA 300.0; drlr*Ing water. It III net lIf'Ilk:lpaled IFederaI: State e.~
MarInI CarPI StatIon. EI Toro
EPA 314 thlIl groundwater wIlhInlhB ...
boundery wit be ll88d for ben8IlcllII
ta8 In IhB mr-ble fW.re.

SotIce: PoteniaI Production Wei


CADHS Medla 01 Expo8ln: Shallow
ClO4METH' Groundwater
Navy I IFormer Marine CarPI All
MIme Corps Station. EI Toro IlRP SIB 1 TreirW"Q: DemlilarizallofVDlsposaI Groundwater 52 106 ND- 398.0'
Cl04METH: Human Recepor: PotenIilII W...
EPA 300 0: Coneumer. IFederaI; Stale BRAe
EPA 31.4:' "Ia net artlcipllled lhaI
EPA M3OO.0 grotnIw8I8r wIIin IhB .18
boundery wit be U88d for beneIIcI8I
use In IhB foresMable fW.re.

~I Iindlan He8d ~ Hog OW DemDarlzaltolYDlsp Groundwater 72 72 5.0 - 278.000.0


EPA 314:
EPA 314
Groundwater diIcharges to
Mattawoman Cleek. RecrealionlII
None
I""""
bioremedla\lOCl
MlIrtne Colpe Warfare CerDr .-. 01 the <:nl8k coUd poteItIaIy fleId
(modlfled)
be exposed. denIOI18tr8IlOl

Fie: P8rcOlIIa_AlSYca_Aprt 2217_


SIwlet: Etw ~ 0Itw , Me4hod 314 reporting Imlla 4 ppb Page 3 01 73
Regul.eDry

8erYIc:a I Fecility I Site I


8oIR. of Perchlorate (OperatIons.
equipment, ActIvItIM) I
LOCIltIon
OetecWd
I~)
I-~I"-~I -~ I
.DelKtIOnS
.
8ampIea Concentrations
CoIlechIct Oetected (ppb)
~ I -1=~I
Potemt.l Pethwllri.) of pa1IIlIlIrv'
requfrerlIerU,
Fundlllll Type

$8Rl1lfinO
1'lIQUhlmenIs)

Navy/ IndIIn Head ~ NO DERP


None FedenII I
MameCorps WIritIs Certer

Navy/ IndBlHead~
MerInII Corps W.... Certer I~ CteekINone ISurface Willet I 0 I 2 I NO I EPA 314 INone IFedenII I DERP

Navy/ Indlan Head Surfac:e


MerInII Corps WIIIfln Certer
ISle 12 INone IGrourdwat8r I 0 I 7 I NO I EPA300 INone IFedenII I DERP


Navy/ indian Head s..tece
M8rlne Corps W.... CwUr
ISb28 INone ISedImert I 0 I 10 I NO I EPA 314 INone IFedenII I OERP

Navy/ indian Head s..tece


MerInII Corps WIIIfln Certer
ISIle28 INone ISoI I 0 I 86 I NO I EPA 314 INone IFedenII I OERP

Navy/ lndien Head s..tece


MerInII Corps W.... CwUr
ISb28 INone IGroI.nIwIIIer I 0 I 22 I NO I EPA 314 INone IFeder.. I OERP

Navy/ lndien Head s..tece


M8rln8 Corps W-r- Certer
ISIle28 INane 1Surfac:e Willer I 0 I 4 I NO I EPA 3\4 INone IFlldenII I OERP

Navy/ Indlan Head Surfac:e


SIle39 None Sol 0 47 NO EPA 314 INane IFlldenII I DERP
M8rln8 Corps Werfenl Certer

Navy I Indian Head s..tece ICIGP


SlIe41 None GrolnIwaIer 0 4 NO INane IFIIder8I I OERP
Merlne Corps Werfenl Center M8IhDcI

Navy / I'ndlan
Head s..tece
Mama Corps Warnn Center ISl e 44 INone \Grooodwaler I 0
I 1
I NO
I IClGP
MBlhod
INone IFeder.. I OERP

Navy/ indian Head Surface


Merlne Corps Werfenl Certer
Isae45 INone ISeclinItri I 0 I 5 I NO I EPA 314 INone IFIIder8I I OERP

Navy/ Indlan Head Surface


Merlne Corps Wmfln Certer
Isae45 INone ISoI I 0 I 11 I NO I EPA 314 INane IFIIder8I J OERP

Navy/
-­ I.ndlan Head Surface
ISite 45 INone IGroundwater I 0 I 5 I NO I EPA 314 INone IFIIder8I I DERP
MameCorps W.mre Certer

Navy/ Indllln Head Surface


MamaCorps Wmfln Certer
151&45 INone 1&I1ace Waler I 0 I 3 I NO I EPA 314 INone IFedenII I OERP

Navy / llndlan Head &.d8ce Isae47 INone ISedimenI I o. I 2 I NO' I EPA 314 INone IF11der81 I DERP
M8rlnI Corps Werfenl Certer

Navy / ~l'ndIlIn Head Surfac:e


Merlne Corps Wmfln CerUr ~47 INone ISOI I 0 I 10 I NO I EPA 314 INone IFIIder8I I OERP

fill: PercOabLAISVca_Aprt 2217_


ShMt:Env~. 0Iher • MBlhod 314 repartIrQ IImI Is 4 ppb PlIge4 of 73
Regul8lOry

I I I I":::c: I
InterHt (CIe8nJp

I~'
LOCIIlIon
~ of Pen:hIonde l0perlltJona.
SeMc:e I FeellIty I 8b I Equipment, ActIvIUn) I Delected
(MedIa)
Number '" Nwnb«
8MIpIMof C-mr.tJona
Range of
Det8cIJorw CoIlectM DelectM lPPb)
Potential P......ay(.) '" ExpoIIut8 perm....
requArnem,
I FundlllllType

samplWlg
requArnem)
J I I J I J I I
Navy I
~Corps
Indian Head ~
W...... cemr ISle 41 I~ I~· I I I 0 18 NO 1300.3140; EPA INone Feder8I I DeRP

Navy I Indian Head ~


Marine Corps W.-ecemr ISle4T j~ 15I.r1ace We#M I 0 I 3 I NO I EPA314 I~ IFeder8I I DERP

Navy I indian Heed ~


~Corps W......cemr
ISle 53 I~ I~ I 0 J 18 I NO I EPA 314 INane IFeder8I I DeRP

Navy I 1nd18n Heed ~


Marine Corps W.-ecen_ ISle 53 I~ j5l.r1ace We#M I I I
0 2 NO I EPA 314 INone IFeder8I I DeRP

I I I I I I IEPAs..taee

Navy I 1Indian Head s..taee


Marine Corps W.-e cemr IS'<TTP MW-41 1== 8lnl Thermal Treatment 1501 1 1 2 2 1921 .0 • 11.800.0
Wat_-&48

183=~ --at
PdeIilaI4IlqlOlIlI"e1o AICl eatIoIllII

the Maltawomen Creek


ttwough eoII oonI8mhItlon IeecNng Stale
to groundwater. lind llrQllllllwlnr
I lo&MOlDefelll8
WortOOg CapIllI
Fund
eel) IC 01
dilchBlgea 10 the CIlIlIk.
LCIMS

Navy I IIndian Heed 5I.r1ace


M8rtnB Corps W.-e cemr ISATTP MW-41 1=~ 8lnl Thermal Treatment IG!oIRIwater
I 2
I
8
1 NO"·35.4 I EPA314 --
PotenIiIII exposure 10 recreetiolllll
at the MaItawomllIl Creek Slat.
ltrough groundwater diIchBIgn 10
I
lo&MOlDefenle
WortOOg CapIllI
Fund
the creek.

.-.atthe~Creek I

EPA~
PolertIaI exposure 10 recreatIonlII
Navy I 1Indian Head s..taee
M8rtnB Corps War1In cemr
ISATTP MW-42 ISubpart X Open 8lnl TIlennal Treatment
Irterlm &talUe
1501 I I I m.o· 183::'!COl:"
2 2 Ieacl*'ll
4.710.0
We#M-&48
ttwough eoII corUmlnlIlion Stale
/o&MorDefenle
WortOOg CapIllI
10 groundwater, lind gtOUIlllwater FIR!
eel)
dIIcI1aI'gef 10 the CIlIlIk.
LCIMS

Navy I IIndian Head s..taee


M8rtnB Corps Warfare cemr ISATTP MW-42 I:=' 8lnl Thermal Treatment IGrooodwaler I 1
I
7
1 NO" ·25.1
Polenllal tIllllOBlH 10 ~1lII
I EPA314 .-.at the Mattawoman Creek Stale
ltrough groundwater cIIschlIrges 10
I
IO&MOlDerenee
WortOOg C8pIaI
FIR!
the creek.

Navy I Iindian Head s..taee


M8rtnBCorps W.-ecen_ ISATTP MW-43 I :~ 8lnl Thermal Treatment 1501 I
3
1 3
1 258.0 •823.0 183=~ to
EPASlri8ce
We#M-&48
PolenllaI8Xp08Ift 10 AICl eatIoIllII
user. at ,he MaIfawoman Creek I I
ttwough eoII conamlnlilllon Ieacl*'ll Slate
groundwal_, .nd groundwater
o&M at Defen&8
WortOOg C8pIaI
Fund
eel) IC or
dIIcI1aI'gef 10 lh8 creek.
LCIMS

PolertIaI ~e to~1lII J
N8vy1 indian Heed s..tace Subpart X Open 8lnl Thermal Treatment .-.of MatlaMlmen Creek Jo&M or Defense
Marine Corps W..-cemr
SATTPMW-43 Groundwelet 8 9 NO"·483.0 EPA 314 ltrough groundwater diIchBIgea to State WorklnQ C8pIlal
InI8rim &lalUe
Fund
thacreek.

FIe: Pen:Oala_AlSla_Apt 2217_

ShMtEIW~OU- • MeIhod 314 ~ lin. Ie 4 ppb Page 5 at 13

R-eu!*'Y

IeMce I F.cllity I alta I


8ou~ ~ Perchlonde (Openllons.
Equipment, AcllvItIaa) I
lOAllon
o.tected
(Medla)
I I I I I
Number ~ Numbw
8empIes ~
Oltec:tiotle CoIlec:ted
Rang. ~
C~
o.tected (ppb)
AIIaIytIcm PotentIlII """811_) ~
Melhod
~~ed.
,...... (Clearop

pennItWlg I Funding Type


requIrernerD.

I~plllv.l
requirements)

I I
EPASOO-
Water-848
Potnla/8XJlOSIA to recreetIonaI
t'ndIan Heed ~ ISATIP MW~ 1 = : 'Iltm Thenn8l TreatmenI tSCI
N8'j,
Malle Corps w... cerar I 3
I 3 130,400.0 - 54.400., Melhod -­
8321a(mocIIfI
ed) ICar
oIlte MaIlawoman Creek
tIYough loll conIamlnllllon I8acting State
to groundwater, lIIld If/OUIldwaI8r
O&M 01 DeIenBe
Waldng CapI8I
Fund
lCIMS
to lte CflIllk.

N8Y1! l'ndIan Heed soo­


Ma1e Corps Wen- Certer ISATTP MW~ I~=:' Iltm Thermal Treatment IGroundwater I I I 1 7 NO' -763.0 I PotertIal8XJlOSlA to reerealIonaI
.... oIlte Mallawoman Creek
EPA314 tIYough groundwater diIchargeI to State
I I O&M 01 DeIenee
Waldng CapI8I
the creek.
Fund

Navyl I'' ' 'Heed ~


M8I1re Corps Warfare cerar
ISATIPMW~ t=~IltmThwm8lTI8lIlrll4ri ISCI I 2
I 2
EPAStnece
WlIler-848
127•liOO.O- 40.000.01 83218{modlll
Uelhod usen
PotertIal expoeure to rectelIIlol..
oIlte Mallawoman Creek
lInlugh loll COIUmInIIIon Ieachi1g State
to llroundw11ler. 8I1d QI'lUldw8ler
I I O&M Of DeIenee
WorlOOg CapbI
Fund
ed) 'COl
diIctwges to lte CflIllk.
lCIMS

~vy I
~Cclrpa
l'Werfant
ndIan HeadCenIer~ ISATTP MW~ 1= =' Ilt.m Thenn8l Treetmenl IGroundwater
I 3
I 8
1
PoleBt81 expcllIUrll to rectelIIloll8I
NO' -1111.0 1 EPA 314 ....01 the Mallawoman Creek State
though groundweter dIadIqee to .
I 100MOIDeI8nIe
WorlOOg CapIaI
the CflIllk.
Fund

EPAStnece
PoterCIaI expcllIUrll to reereallon8I
W8Ier-848
usen 01 the Mattawoman Creek /
I 183~
Navy! l'ndIan Heed Stnece /O&MorDel8nle
!SATTP MW-66 1 = : 'Ilt.m Thennal Treetmenl ISoi 2 2 58.2 -98.2 tIYough loll conIamlnllliDn Ieachi1g State WoBilg CapIaI
M8rir8Corps Werfant CenIer 1 1
ed) ICOI
to groundwater. and fPOI,RIwaler Fund
dlsclwges to the CflIllk.
lCIMS
I
O&MOf~i
Navy 'iindian Heed Stnece
MarIne Corps Warfare CenIer
ISATTP MW-66 1=:'Ilt.m Thennel Treatmert IGroundwater I 0
I I 7 NO I EPA314 None State WoBilg CapIaI
Fund

Navy I IIOOIan Heed ~


MIme Caps Warfare Certer ISATTP MW.{)7 I::' Ilt.m Thennel Treetmenl ISoi
1
1
1
2
1
NO-79.S
EPA Surface
Water-848

1832~ lInlugh
Potenllal exposure to recnNIIloll8I
usen 01 the Mattawoman Creek
loll corternnIIIon IeacNng Stale
to groundwater. and If/OUIldwaI8r
f IO&M 01 Defense
WllIidng C8pIal
Fund
ed)lCor
clisc:hlJfges to the CflIllk.
lCIMS

O&MOIDerense
ISATTP MW.{)7 1=~c:-' Ilt.m Thennal TreatmenI 1 EPA314
Navy' ea.p.'1WlWfare
MIme nd18n Heed ~
CenIer IGroundwater I I 0 8
I
NO None SIale Working Csplal
Fund

fie: PercD8la_AlSvl:a_Ald2217_
Sheet Env~.Other , Melhod 314 I1IIXlItinllImlIl 4 ppb Page 6 0173
Regulldory

8ervIc. I Fac:llity I 8lw I


Soun:. of PercIIIome (OperatIons,
Equipment, ActIYItIn) 1
Locetlon
Detected
IMtdIa)
Sam.. . ...~ I-I
I-~r-~I
Detecti_
CollectllCl
eonc.ntratlona
Detected lppb)
Method PotentIal Pllthway(a) of ExpoaUr8 pennlltlng'F-" 1'8qIftmenIa,
sampling
I Funding Tn-

11'8qIftmenIa)

Navy / Iindian Head St.rfaoa ISATTP 1AW-08 1:=~!UnThBrm8l Treatmenl ISoi


Marine Corps Waftn een. I t I 2
I NO· 82.0
EPA Surface
wm.-&48

183~
PclenlllII exposII'8 to recreaIIonaI
t.-s of !he MaIlawoman Creek I
through sol coriatnftttIon Ieact*'U StaI8
to groundwater, and grOUldwatw
100MorDattne
WorkrG CapItal
Fund
ed) IC or
diectIwge& to !he creek.
LCIMS

Navy / flndlan HeadSt.rfaoa ISATTP 1AW-08


Mama Corps W..rar. een.
1=~:"!Un TtwmeI Treatment IGrculdwaI. I 0
I 8
I NO 1 EPA314 None state
O&MocDattne
WorkklQ CapIIaI
Fc.nI

EPA Surface
wm.-84e
NlIYy / Iindian Head Surface ISATTP 1AW.()9
Mertne Corps W... een. 1=~!Un ThBrm8l Treatment fSol I 1 I 0 2 NO
1832~od~INone
ed)ICoc
1&Me
!O&MOCDerense
WorkklQ CapIaI
Fc.nI
lCIMS

NIIvy / IlndIan
Head Surface
Mertne Corps Waftn een. ISATTP 1AW.()9 1=:' fUn ThBrm8l Treetment IGIoc.nMaler 1 0
I 1
I NO 1 EPA314 None Slate
O&MorDattne
WorkklQ CapIaI
FIRl

Navy / Iindian Head Surface


Mertne Corps Warfln CeR. ISATTPIAW-to 1=~BtmTlBmlllTreatment ISoi 1 I 3 3
EPA Surface
WaIW-&48
PotenIlaI8lq)08<ft to rec:reetlocllll
I..-s of !he M8llawoman Creek
I 243.0 - 645.0 1832~. throughaol COlilall1il8lionleacNng. Slate
to groundwater, and groundwater
I I O&M oc oer.­
Wa1dng CapIIaI
Fc.nI
ed) IC or
dlachBtgea to !he creek.
lCJMS

NIIvy / I'ndlan
Head Surface
Marine Corps Warfln c:ert.
ISATTP 1AW-10 1=~ Btm Thermal Treatment IGIoc.nMaler I 0
1 1
I NO
I EPA 314 None Slate
O&MorDattne
Wa1dng CapIaI
Fund

l'ndIan 1 I -
O&M or Defense
NIIvy / Head Surface
Marine Corps Warfare c:ert. lpotable Well#1SIThwe is None. IDrIr1kq WaIW
I I 0 1
I NO EPA300M INone INone Workklg Caplal
Fund
--­
IO&M
NIIvy / IIndIan
Head Surface
Marine Corps Warfln CeRer
(PoIallle Well
#1C1a
IThwe is None. IDrIr1kqWm.
I 0
I I 1 NO 1EPA300M INone INone
or Defense
Working CapIaI
Fund
--­
O&MorDefen&e
Navy/ Indian Head St.rfaoa PoIallle Well
Thwe is None.
Marine Corps W.-farec:ert. #2012
OrWdngWater 0 1 NO EPA300M None None Working CapIaI
Fund

Navy/ Indlan Head St.rfaoa


Marine Corps W.mreCeRer
Sle11 U""'"'-' Other 2 2 NO EPA 314 None Federal DERP

fie: PercOaIa_AlSvca_AprI2211_

SI8It: Erw RealoraIion-Cle, QIher • MBlhod 314 repor1i1g Iinl III 4 ppb Page 1 of 13

lIervIce I FtclJlty I 'Ite I


Source of PIIdIlorMe (Openl/ona,
Equlpmenl, ActIvItlMI I ....
Location
0etee:t8d
( )

1-"1-"1 _of I I
oet.ctIonI ........

CofleCUd
C~ ~
Dstec:t8d (ppbl
PotenIW P8Ihwarislof -.,:=. . .
Regullltofy

permmv' .
AIquiemerD,
samplirll
~)
I Fundllll T"e

Polertlal exposure to Sol for


I
I EPA 314 ICOllSlructlon
Navy 1 I'ndlan He.! &.face
tM1ne Corps W.we Celt. ISla 11 Illntrown ISedimelt I 7
I 8
1
NO' -230.0
WlllIt. ., malrtllnance
wart<ers or Irespaas«s. P<JlsriIaI (Federal
8l<p08lJl9to .00- W8l8r and
I OERP

ll4IdInMn for rSCleallolllll ~.

Navy I· l'ndIan
He.! &.face
tM1ne Corpa W.we Cefter ISla 11 Jllntrown I
GrotnJwster
I I I
11 15 NO'·4.0
_.
Groundw8ler recharges Into
I EPA 314 lwwatateer.r endPoter&I exposure to -'-:'lFederal
aedimeIt for rect8lIIIoIllII
I DERP

PotertIaI8lqlOIIft to Sol for

Navy 1 Ilndlan soo­


He.!
t.AIItn Corpa W... Cefter ISla 11 Illntrown I~W'" I 7
I I 11 NO' -4.0
I EPA 314 lCwart<ers
OOll nJetor~.
lon WlllIt.., llIIIlnl_nce
P<JlsriIaI IFedenli
8l<p08lJl9 to .00- W8l8r and
, DERP

Hdlmenl for IlICl88l1oual_.

---
fUvy 1 11nd18n He.!
tM1ne Corpa W... Cefter
soo- ISla 11 Ium- ISoi 1
45
I 86 I NO" - 480,000.0 1 EPA 314 1COOll nJetlon wart<ers,to Solma.......
Pot8RlaIIlllIlOIlft for IFedenli , DERP

_.
wakera or trespeasln.

Navy 1 Indian He.! Sl.riace


PotertIaI exposure to .00- w8lBr
SIe 17 llntrown Sl.riace Water 7 7 4.0 EPA 314 and Hdlmenl for r8Ct8lIllonal IFedenIl I DERP

_.
~Corpa W.weCefter

Indian He.! Sl.riace


PotertIaI 8lqlOIIft 10 .00- W8l8r
Navyl
MameCorps Warfa-e Cefter
SIa 17 Ur1uIDwn Sedlment 6 6 86.0 -180.0 EPA 314 and sedImenl for recreatJonaI JFedenIl I DERP

PoleR\Bl exposure to Sol for


fUvyl Indian He.! Sl.riace
SIa 17 Ur1uIDwn Sol 24 35 NO" -130.0 EPA 314 COfllllructIon ~ millnler8nce IFedenIl I DERP
MameCorpa W"'Celter
wakera or trespeMin.
I
'Navy 1 indian He.! Sl.riace
SIa 17 Ur1uIDwn GroLnlwater 0 8 NO EPA 314 None IFedenIl I DERP
MameCorps Warfa-e Celter

Individuals (conslructlon WlllIter)

Navy 1 I'ndian
He.! &.face
Mame Corps W.we Cefter ISla 21 lur1uIDwn ISoi I I I 23 30 NO'o130.0
I l hrlt
come In COlUd will
EPA 314 ~ Sol or groundwater at IFedenII I DERP
this ale could pollnlaIIy be
exposed.

,
Individuals (conslructlon WlllIter)
Navy 1 I'ndian
He.! Sl.riace
tM1ne Corpe Wadln Cefter Islte 21 lur1ulDwn IGnuxtwater I 20
1 23 I NO' 2.900.0
0
I EPA 314
lhat
Iaubeu1ace
come InSol
COlUd will
or groundwater at IF8d8raI DERP
this .Ie could poler*aIIy be
exposed.

fie: F'en:08Ia_AISvaI_Ap12217_

Sheet Env ~ Olher


• MelhQd 314 ~1Im1 It 4 ppb Page 8 of 73
Servtc:. I F8CIUly I Site I Source 01 PeRhIorMe (C)penItIclM,
Equipment, AcUvItln)
I location
OeWCted
(lhdle)
Number 01
Humber 01 8amplH C-mrau-
Range 01

Detaetlonl Collec:tad DelM:ted (ppb)


~ PoIentIei Pelhwlly(a) 01 Exposure!permlltlng I Fundi,. Type

The groundwater dlsch8rges 10 8


"8.
EPA 314: 8Ir8Bm 80uIh of lhe PolenliaI
Navy I IlndlanHead~ ISlle42 IUnknown IGroundwater I 9 I 15 1 NO' -4.0 1 E~; 8llpll8In8by~and IFederai I OERP
MarIna ~ Warfare Certer Meltod ~ personnel dlgQlng II
lhe ....
The grotnlwaler cllecI-vea 10 8
8Iream 80uIh of lhe lite. PolenliaI
Navy I Iindian Head ~ ISle 42
MarIna ~ W.... Certer
lu!WIown ISoi I 10 I 12 I NO' - 88.2
I EPA 314 exposurllS by ~ and
1
maIrterw1ce personnel dIgQIng 81
IFederai I DERP

= __
lhe ....

PolertI8I exposurllS by reall8llol181


N8vy1 IlndilnHead~ ISle 57 U!WIown Groundwater 1 8 NO-8.8 fromgnxnlwaler~IFederal I OERP
MarIna Corpa Werfln Certer 10 lhe Maltawlxnan Creek.

Navy I IMarin
M8rIna ~ YUIIllI
a ~ A; S18IIon, ICScuca
hocolaW".
te
BII Mect8t
Motrtalna
CoIcndo Rlver Backgromd ~ ~ W8ler 1 1 4.2
Scuca ....... to
EPA 314...­
'.-...
drtr*lr4I
....... I I
None
O&M or DeNnse
Wor1lrG CapIaI
Fund
Rqe

=-~-~-I~~ I~~-·-- 1'-1' I' I I 1­ I'" NO ,"'OW

To Confoml Will
I~=
The 29
September 2003

=CorpaI=CorpaA;~ l::er·~I~T~ IGroundwater I 0 I 2 I NO I EPA314/None I=~I~~=


PwcWorate
Sampling

To Ccriorm Will
The 29

Navo/I IMarinaCorpaA;StatIon. I=:e,.


MarIna ~ YUIIllI Tracker Buldlng
100000000000Tesling IGroundwater I I I 0 1 NO I I EPA 314 None
September 2003

/=::n~k11
On
Polley
~~= Fund

PtlIcNora1e
Sampling

fie: PeR:Dala_AlSYcaj'llrl 2217_

SheIII: Em Re8lonItian-CIe. OCher • Method 314 IllIJClI1Ing llmlia 4 ppb P8Q89of73

RegulmKy
Inte.....(CIeBrup

8ervIce I FIICIIIty I Site I ~quIpmll11t. ActMtles)


Sou of P«l:hlorate (OperatJona, I LOClItIon
Detected
(Media)
IDetec:tIo
Number of I Number of I Ranga 01 I AnaIytlcm I 11'l qUll ted
a_pies Conc:entratfoM Method PoIentf.. Pathw-ri.) of Expos permlllnlJ
na CollllCted DetKt8d (ppb) requQmenIa.
f Funding Type
sampl~
~)

I
To eonrorm WIh
The 29

Navy'
MarIna Corps IMarIna Corpa A'I St8IIon.
y~
ClImp Bt.n 2.
IEastam
Chocolate
0peraII0nel Tesllrv Sol 0 7 NO EPA 314 None Depar1menI Of
Defense IrUnnln
woo:.
$ejllember 2003\ O&M Oefwee
CapIIaI
Fund
MonI8lnlI PolIcy On
PercNarate
Sampling

To Conform WItl
The 29

ISuppIIlId from CoIor8doRJverwNch twas IGrolnIwlIler I I I


tau.
10 lha IWgI'Iy aokJbIlly 01 $ejllember 2003\ O&M Oefwee
I EPA 314 Ipel(:hkllllte.lha ~tike rotleolDepar1menl Of W.....~ CapIIaI
Navy'
MarIna Corps IMerIna Corpa A'I &.uon. IBeckelt Hlue.
y~ Up-Gradiart blIckground '-vel 014-8 ppb.
2 2 33.35'
.. . • < MRL tunan Illlp06lft Ie tITough Defense IrUnnIn
consuntIon 01 Groundwater. Policy On

-'F:m
P8I"eNara\e

I Sam~

Navy'
MarIna CorpsIy~
,
'MSluceCAS-Water.
MarIna Corpa A'I &.uon. Water Sya1em SuppIIlId from Colorado River wNch twa a
8IICkground Level 014-8 ppb.
~ Water 1 1 4.6 EPA 314 water ~ lha
"'"---"- 10 Water
TI'88tmenl Platt.
None
r"".-.
Worldng CapIIaI
Fund

To ConIonn WIh
The 29

INavy'
a.rycIwaiM er
IMarIna Corpa A'I St8IIon. IGol ITr8~ ISol I 1 I 2 I NO· • 34.8 I lau.
10 lha ligh
EPA 314 percNarate. mosllbly01 rotle 01
lhaaokJbIlly woo:.
Of O&M Oefwee
DapertmenI2003
5epIember CapIIaI
MarIne Corps YUIlIlI AuxIIary II tunan Illlp06lft Ie through DeflInse IrUnnln
1
Fund
consuntIon 01 GroInIwater. PolIcy On
Perctbala
sampling

= Corps!: Corpa A'I &.uon. Iwestern


Camp BII
Machen,

Chocolate
~m
Tra~ Sol 0 6 NO EPA 314 None
ToConlonn WIh
The 29
$ejllember 2003 O&M Oefwee
~"":In
PolIcy On
PercNarate
Sampling
1
woo:. CapIlsI
Fund

To Conform wah

Navy , Msrine Corpa A'I St8IIon. Eastern


MsrineCorpslyUIlIlI
Camp Bt.n 1.
IChocoIate T~ Sol 0 9
I NO EPA 314 None
The 29
$ejllember 2003
Depar1menI Of O&M or Defense
1
Defense IrUnnln WorIdng Capllal
~ Policy On Fund
Perctbale
Sampling
I I I I I I I I I I
fie: PercOallu'.SVClI_AprI2217_
Sheat Em Raaklration-CIe. au.. • Method 314 AIpOrting liml Ie 4 ppb Page 100173
Service FecHIIy 81te
Sourc. of ParddCKllle (Operations.
Equipment, ActIvltIM)
Location
Detec:t8d
(Media)
Datactlona
I
Nunbar of Numbat of I
Range dI
S8mpIas C_ntratJons AnaIytlce/
Collected Dat.ctad (llPb) MethodI Funding Type

To Conform With

Nevy I
Merlne Corpa Yuma
c.mpDavld.
IMerlne Corpa"" Slatton, IWestern
Chocolate
MoIriaInI
Tmq Sol 0 7 NO ,. ,. I.. The 29
September 2003 O&M Der8nIe
Oepartmert or or
oere.- Intermin Wor1<Ing Cepllal
Polley On
Perd*lrate
1 Fund

Sampllng
---
To Conform With

Navy I
Merlne Corpa Yuma
IrlI Wah,
IMIme Corpa"" Slatton, IWestern
Chocolate
MoIriaInI 1- l~ 0 2 NO
IEPA", Nane 1=':'IO&M~DoIooo
The 29

Der8nIe lnhlmlln WorldrQ CeplIaI


PoIlcy On
P&"CID8Ie
Fund

SampIlng

To Conform With

I
The 29
Cue to the high eoUlIIIy of September 2003
Nevy I IMIme Corpa Ilk Slatton, IY~' e.ry ITI'lIlrq
Merlne Corps Yuma M. Gddwater ISoi 1 1
I I 10 NO··93.9
I
EPA 314 !perctbaIe,
the IIlOIIIIkety FO\ft of Department or O&M or 0eIenIe
tuna" expolIIft Is though Defense 1rU'm1n Wor1<Ing CeplIaI
COf1IUIlplion of Groundw8ter. Polley On Fund
P&"CID8Ie
SampUng
I

I~Tmq
Range ShothoIe Cue to the high eoUlIIIy of
Nevy I IMerlne Corpa"" SIaIIon. Iloc8Ied
On The Tralr*'g; Dem~ Sol 1 1 150.0 EPA 314 percHoraIe, the IIlOIIlIkely FO\ft of INane
1O&M or 0eIenIe
Wor1<Ing CeplIaI
Merlne Corps Yuma Bany M. Iun8n expolIIft Is though
Goldwater COf1IUIlplion of Groundwater.
Fund
Range

Methods
3OO.08llln:l
Navy I
Merlne Corps
IHAS ScUh W8yIIlOIAh \=.Gate ISom:e Is unknown. IGroundw8Ier
I
0
I 10
I NO
I
350.2; INane
Methode
IFederaI; State I BRAC
3OO.08n:l
350.2
I
DetectIons located
81 Norn_ I.lInd
Island wIich • Ioc8Ied about 3

Navy I
MlmeCorpa
IHAS ScUh W8yIIlOIAh I~L.an:l ITraDng
Island ISadktKMt I
2
I 2
mleal<Uh MIt of Marthe'.

urinheblate~
I 240.0 - 1,935.0 I EPA 314 IVIneyard· Island Is an
d Ialanduaecl ea a IState I BRAC
wIdlIfe refuge ani percHoraIe does
not pr--.t a Itn8I to Iunan
. , , , . , , , I188lIh.

fie: P8rcData_AlSvca_Ap12217_

Sheet Env R8lIIorlIIIon-Cl. au.. • Method 314 reporIlng lint. 4 PIlb Page 11 of 73

Location Number of Range of


Source of P.n:hlorW (OperatiOM. Number of
Anelytlcal
Service I Fcility I Site I Detected 8empI.. Concentratlona Method PcMntIaJ Pathw8J(a) of Expoaurejpennlltlng I Funding Type
Equipment, ActlvItIM) I (Media)
Detec:tJons

Collected Detected (ppb)

None. Groundwat~ Ie not used for

I;~ IRP 10peralI0nal T88lIng; Trair*lg;


I~~

tunan constmjllion. 5•• Is

Navy' INAVAIRWPNSTA CHINA EPA 314


1 1 15.0 separated from dr1nIlDJ W8l~ IFederal; State I OERP
MarIne Corps lAKE CA l8lIch field Oem~ (modified)
lIOlI"C8 weIs by mlea end ttick cI8y
equiIard.

NlIVy' INAVAlRWPNSTA CHINA


MarIne Corps lAKE CA
I148CI*l0Indl

e lake IRP IR.-ch. Development. Teal end

EV8Iu8tIon; ~econfigUllllon; I~~ I I I I 2 2 58.1 -421.0


None. Groundw8t8r Is not used for
EPA 3141tunanconsumpllon. SMeIs
(modified) separated from dr1nIlDJ W8l~ IFederal; State I DERP

I _....-.. . . . .

Dr8lnIIge 0Ilch 0em1ll8rlz8lllW[)l lIOlI"C8 W8IIs by mlea end ttick cI8y

I
equiIard.

IGroundwat~
CI*le Lake IRP
I I
Navy' INAVAlRWPNSTA CHINA In t.Ich8Iaon R-m, Developmert Teat end EPA 31411un8n consumption. SIe~
from dr1nIlDJ Is IFederal',Sl8le'
.
Manne Corps lAKE CA lab 0r8In8ge EVIIIu8lIon; M8IU8clImg
4 4 111.0- 720.0 sepereted I OERP
(modlIled) lIOlI"C8 weIs by mlea end ttick cI8y loc8I
0Ilch I equiIard.
None. Groundw8ler Is not used for
NlIVy' INAVAlRWPNSTA CHINA 1#8an~J
&.n- R.-d\ 0eveIcJpmert. Teat end
Groundw8Ier 3 3 24.1-344.0
EPA 314
Iun8n consumption. SIe Is
sepereted from dr1nIlDJ ~ IFederal; Sl8le I OERP
MarIn8 Corps lAKE CA W8l8rPL EVlIIulItIon; Meruf8ctu1ng; (modlIled)
Dr8lnIIge Dem~ lIOlI"C8 W8Ils by mles end ttick cI8y
equlard.
None. Groundw8ler Is not used for
Navy, INAVAlRWPNSTA CHINA
M8rIn8 Corps lAKE CA
IM3CI*lMInIdeck
e lake IRP ISuapect th8l11s IIIhnIy occmtng IGroundw8ler I 2
I 2
I
34.8- 50.0
I l EPA 314
human
conaumpllon. SIe Is
(modified) sepIWlIled from ch*q W8ler IFeder8I; Sl8l8 I OERP
lIOlI"C8 W8Ils by mles end ttick cI8y

,
equIIard.
STL

Navy , f8Val Weepons InduslrleI


M8rIn8 Corps R_ Pl8nt. McGregor Anle T
Texas
I I~&mm ISOO8ce Waf«
I
3
I I I 7 NO-".0
PerchIor8le
bylC IAnle T 1nduelrt8l ...; Groundw8l_ 1Sl8le
m8thod
b88ed ~ to SIn- W.-1llIlhw8Y
I OERP

CALOHS
11197
--
EPA 314;

STL

Navy' I Naval
Weeporl8 InduslrleI
Manne Corps R888lVe PI8nt, McGregor Anle T
Texas
I IDelivery StatIon IGroundw8ler I I I I eo 64 NO' -140.0
PereI*lnIle

by IC IAnle T 1nduelrt8l ...; Groundw8l_ 1Sl8le


method. to SIn- Waf« IllIlhw8Y
I OERP
b88edon
CALDHS
11197
-

I
I~&mm I~
I STL
PercIb8te
by lC Anle T 1nduelrt8l ...; ~
NavalWeepons InduslrleI
:::. CorpsIR_ PlIri, McGregor Anle T
Texas I
I 2 32
I
NO-2,300.0
. conllImlnaIIon Ie8l:'-s to
method. groundwater which leeds to
b88edon
"CAL OHS W.~ IllIlhw8Y
SOO8ceISl8le I OERP

. . , , . , , 11197

FIe: PercOllhLAISvca_AprI2217_
Sheet: Em ResIclr8lIon-Cl. Other • Method 314 repoltflg IImIIs 4 ppb Page 12d73
r
Regul8toly

8eMce I hefilly I Site I


Source of Perchlor8te (OpemJons.
Equipment, Acttvlttea) I
LocMlon
Detected
(Media)
I ~
N ber of
-
wn
Samples I 1 I
• of Concentrations
Range of
Collected DetectecI(ppb)
M.u:::1 PotenIIet Pathw~e) of Expwure permlltlrv
AnmyU
1'lIqIftrnenIs.
I==~I Funding Type

sampling
reqt*-n)
I I
sn
Navy I t
Naval
Weapons IndusIrlat
MarIne Corps R_ Plant. McGnlQor Area T
Texas
I I~~ l5edknMt I I I I0 2 NO
PllIl:I*lrate
bylC t Area T Irdustr181 BIe; Groundwater IState

method
based ~ to Striace Water patIJo¥ay

I DERP

CALDHS
1997
---

= Corps!~ Pten. McGnlgor


Naval Weapons fnduslrtal I I
Area A l8ndfII . DemIIartzatIolY I~er I
10
I I I 12 .NO"· 110.0
EPA 314:
STL
Perclbate
by IC IArea A L8ndIII; Groundwater 10
Swf8ce WIller;
melhod. c:omm.1calIlndulb1lll worIuIr

based on
CALDHS
1997
---
IState
I OERP

= Naval Weapons ~I
Corps!==- PIart, McGnlgor
I
.we 0 l8ndfII DemIl artzalloWOtl IGroundwater I I 21 24
I
NO"·240.0
EPA 314;
STL
I pe"oiIlcUN,Area 0 L8ndIII; Groundwater to
by IC Swf8ce Water;
melhod. Commertcalllndultr worIuIr
based on
CALDHS
1997
-STL
IState I DERP

:::. Corps!~=-
Naval Weapons IndusIrlat I
Plant. McGnlgor Area 0 Landfll
I
DemI~ I~ I 0
I
6
I I NO
Pel ctIIclr8le Area 0 l.anlft; Pd8nIIlII ftv sol to
by IC IeedI to ll/OUfIdwaI Groundwater
method. 10 ur- water ~ pettway; IState I DERP
based on Comrnarlcalllndulb1lll worIuIr
CALDHS
1997
---
EPA 314;

=-
STL
PercIIIclrate
I
I I Groundwater to IState
I Naval Weapons InduslrIal
CorpstReserve PIart, McGnlgor Area E Landfll IDemIIlarlzatlonl[)l IGnxmwater I 6 16
I
NO" -90.0
by IC IArea E L8ndIII;
melhod Swf8ce Water; I DERP
Texas based ~ Comrnarlcalllndu8IrIBI worIuIr
CALDHS
1997
-STL

Navy I !Naval Weapons IndustJ1lII I


MarIne Corps R_ Plant. McGregor Area E Landlll
Texas
IDem~ ,~
I I I 0 5 NO
I PercIIIclrate Area E L8ndIII; Pd8nIIlII ftv sol to
bytC
leach to groundwater. ~er
melhod. to Swf8ce Water 8lqlOSlft
based on pettway; Comrnarlcalllndu8IrIBI
IState I DERP

CALDHS worIuIr
1997

fie: Pen:Dala_AlSvcs_"PrI2217_

Sheet: Env RestonIlioo-CIea. Olher " MeIhod 314 ~ IImlis 4 ppb Page 13 of 73

servlce I FIlCIIIty I SIte I


Source of Pen:bIonIte (Operations.
Equipment. ActIvltJ..) I
Location
oetected
(Media)
. .: _.­E
I·-~I·--~I -~
.. CoIIact.cI Datactad (ppb)
_I--.~_. I Funcllng Type

EPA 314;

I I
STL

I lo.n~
PercI*lrale

I I
l
Navy I Naval WlIlIPOIlI nduatrl8l
MlwIna Corps R_ Plait, McGregor Area P ISuface Water 22 48 Noo-42.0
by IC
melhod
IArea
Suface
0 Landfil; Groundwater to
Water pathway; IStale I DERP
T_ 1 balled ~Commerk:alIlnduslrIaI worller
CALDHS
1997
--­
EPA 314;
STL

Navy I INaval
WlIlIPOIlIlnduslrIaI
Mama CorpsR_ PIart. McGregor AI-. P
T_
I lo.nl~ I~ f
74
I I 110 NO' -98.0
I PercI*lrale IArea 0 lMdM; Groundwater to
by IC Suface Water paIhway;
melhod, CommerlcaVInduslrtaI worller
IStale I OERP
bBsedon
CALDHS
1997
-STL

=~
Naval WlIlIPOIlIlnduslrIaI
CorpsIR_ PIart, McGregor AI-. P
T_
I I~ ISedmR I I I
0 2 NO
PerdbaIa
I melhod,
by IC IArea 0 Landfil; Groundwater to
Suface Water pathway;
balled on Commerlcalllnduslrlal WOI1<er
IState I DERP

CALDHS
1997
-
STL

Navy I INaval
WlIlIPOIlIlnduatrlaI
MlwIna Corps R..-ve Plant, McGregor
T_
IArea P
lo.n~ I~ I
0
I I I 6 NO
PeldDale Area 0 lMdM; PollnlaI for 101
bylC
cortamlnatlon to leach i*l
melhod, groundwater. Groundwater to
balled on Suface Water expIllIIH pathway;
IStale I DERP

CALDHS CommerIcalIlnduatrlal WOI1<er


1997

EPA 314;
Area
Z InduatrlaIArea; PollnlaI for

I
STL

Navy I I
NavaI

T_
WlIlIPOIlIlnduatrlaI
Mama Corps R_ PIart, McGregor Area Z lo.n~ I~ I 33 .f 43 I NO' - 91,000.0 I
PercI*lrale 101 cortamInatIon to leach i*l
bylC
groundwater. Grolnlwaler wNch
melhod. leads to Suface Water expIllIIH
balled on pathway; Commerlcalllnduslrtal
IStale I DERP

CALDHS WOI1<er
1997
-
STL

Navy I INavai
WlIlIPOIlIlnduslrIaI
Mama Corps R_ PIart, McGregor
T_
IArea Z lo.nl arlzatlorVDlsp I~ I
0
I I I 6 NO
Pen:tbate
bylC
melhod.
balled on
INone IState I OERP

CALDHS
1997

fie: PercDala_AI&«:s_AplI2217_

Sheet Enw RestoralIon-CIee. 00- • ~ 314 ~ 11m. II 4 ppb Page 14 of 73

Regulatory
IntelllSt (Clearup
of ~of I~I
Method Potential Pdlwayfa) of ~~
LOC8Uon
Service I F8C1l1ty I 81te I
8oun:e Pen:hlcnte (OperlltloM.
Equipment, ActJvItIM) I Detected INwnbeofrwnWOf\
Detectl r 8ampIes C~ntr.u- perm~' I Funding T".
(Mad") - CoIlec:ted Oee.ctecl (ppb) requiBmenls,
sampling
requhlmeIU)
I I
EPA 314;

Navy I r aval
Weapone IIldu*llII
MarIne Corps Reserve PIaft. McGnIgor
T_
I
Area F I~~ ISurf- Water
I I 84 125
I
NO" - 1,800.0
STL

I perd'b'ate
by IC
method,
based on

F Industrial Area;
ea to sm-wcrbr
lAGroundwater
rCommerlcaLllndustrlal Water; IState I DERP

CAlDHS

1997

---
EPA 314;

= Naval Weapone IIldu*llII


CorpsIReserve PIaft. McGregor
T_
I
Area F I~ IGroundwater
I
386
I
458 I NO"· 17,000.0
I~e
STL

by IC IArea F tnduIlrIaI Area;


method Groundwater to sm- Water; IState
I DERP
besed ~ CommerlcaLllndullrl8l wcrbr

CALDHS

1997

---
EPA 314;
STL

=.:. Corpsl~ Naval Weapone InduIIrIlII

T_
Plan, McGregor Area F
I IMalrUfwncelReconIIgInlIon
1
501
I 41
I
184 I NO" - 98,000.0 IPerctbate Area F InduIlrl8I Area; Pl:lllnIaIIor
by IC
method
eoI COIUmInatIon to I8lIch 11m
gtOlnlwater. Groundwater to
ISlate
I DERP
based ~ sm-Water 8lqlO8U'8 ~

CAl DHS Commerlclllllndullrl8l wcrbr

1997
-
STL

Perctbate

=.:. Naval Weapons IIldu*llII


CorpsIR_ P1arC, McGnIgor
T_
I
Area F IMalrtenanceJReconflgUlltlon ISedknM \
0
I
2
\
NO
I method,
byte
based on

I None ISlate
I DERP

CAlDHS

1997
-STL

Navy I r aval
Weapons Industrial
MarIne Corps Reserve Plait, McGregor
Texas
I
Area M IMalrtenancelR~; Storage 501
1 I 153
1
328 I I NO - 1,800,000.0
Perct*lrate Area M tnduIlrIaI SIte; PoleItIalIor

eoI corumlnatlon to I8lIch to

bylC gtOlnlwater. Groundwater to


~
sm-
Water exposure patlMay;

IState
I DERP

CAlDHS Commerlcalllndusb1al Worker

1997

-
STL

Navy I r aval
Weapone Industrial
MarIne Corps Reserve Plait, McGregor
T_
IArea M IMalrtenanceJRllCOl'Ifl jl 'atlon; Storage ISedknert
I
1
I
9
I
I NO· 310.0
Perd'b'ate
by IC
method, 10
IArsm-
ea MInduIlrl8I 51e; Groundwater IState

Water patIMay;
based on Commerlcalllndullrl8l Worker

I DERP

CAlDHS

1997

FIe: Pen:OellLAlSvcs_AprI2217_

SIwIat Erw Re&toratlon-CIe, Other • MBlhod 314 reporting 1m. Is 4 ppb Page 150173

I·-~I-~I -~f IPc*ntI••


Loc:atIon
8etvIc:e I FllClllty I Site I
8ol.wce of Perc:hlor* (0pemI_.
Equipment, Ac:tMtIft) I DelIlCted
(Media)
Dlltec:tJons hmplea
CoIfec:tad
Conc:entntIoM
DelIlCted (ppb)
Method PllthwllYfa) of ExpoauraJ~ I Funding T".

STL

N I l
N8val
Weapona IncUIriaI
~ Corps R-.ve Plant. McGnIgor Area M
Texas
I IMlIlrtllIlIIllCel'ReconIlgtnIlon; Storage ISlriacaW..
I I 267 344
I
NO-5,300.0
I
PercIiorate
by IC IArea
M Induslrial SIte; Grou1dwaJer
meIhod. 10 Slriaca W.. paIlway;
based on CommerlcaUlndustrla Worker
IStale I OERP

CALDHS
1997

EPA 314;

I
STL

I I
I Peiclbate IArea M IncUIriaI Sle; Grou1dwaJer
twvy r I N8vaI Weapona IndustrtIII
Mame Corps R-.ve Pln. McGnIgor Area M
Texas
I~Storage IGrou1dwaJer I 354 445 NO· - 85.000.0
m
~ to Slriaca W.. paIlway;
• Commertc:8lllncUlrial Worker
IStale I OERP
based on
. CALDHS
1997
-STL

=:. N8vaI Weapona InlUtrilII


CorpsIR_ Pln. McGnIgor Area B
Texas
I I~ I~ I I I I1 2 NO-35.0
Peiclbate Area B IndusJrtal Area; PoI8r&I for
bylC
meIhod.
sol to leech 10 groundwater.
Grou1dwaJer
10 Sw'- w.. IState
based on expoIU'8 paIlway;
I OERP

CALDHS Commerlcal/lnduslrial worker


1997
-
STL

twvy I l N8val
Weapona Induslrial
Mame Corps R_ Plant. Mc:Gr8gor
Texas
I
Area B IMa~ IGrou1dwaJer
I I 0 2
I
NO
I
Perctbate
bylC
meIhod. lNone
based on
ISlate I OERP

CALDHS
1997
-STL
twvy I I
Naval Weapons Induslrial
Mame Corps R_ Plart. Mc:Gregcr
Texas
I
Area L 10peraII0naI Testlng IGrou1dwaJer I I I 10 33 NO-160.0
I
Perd1Iorate
by IC IArea
L IncUIriaI ...;
meIhod. 10 Slriaca Water;
Grou1dwaJer IState
based on Commeric:aIIInduslrial worker
I DERP

CALDHS
1997
-
STL

twvy I (&Val
WeaponslncUlrial
Marlne Corps R_ Plant. McGregor Area L
Texas
I 10peraII0naI Teating I~ I I I I0 29 NO
Perclbate
bylC
meIhod. I None
based on
I Slate I DERP

CALOHS
1997

fie: PercOata_AlSVCI_AprI2217_

Sheet Erw RIlIIIDrlIIion-O. 0lhBr • Melhod 314 reporting 11m. Is 4 ppb Page 18 of 73

Reguletoly

8ervIc:e I F8clllty I a.. I


Source of PerchlorMe (O~.
EqulpoMllt, ActMtIes) I
locMlon
Detected
(Medl.)
IN~of I~berof j I~I I bngeot
DeWctJona 8emples Concentratlone
Collect8d Detected (ppb)
Method PotentI8l P8thwey(e) of ~:=~I permItIng
AIqlQmerts.
Fundi.. Type

eampltng
reqlkemenla)
I I
STl

~
NlIv8I WlIlIIXJI1Ilnduelrl8l I
CorpeIR_ PIn. McGAIgor AnIlIl
Texas
/Operallonel T~ l&riace Willer I I I 0 4 NO
I PercIbate
bylC
melhod.
baled on
INone ISlat. I DERP

CALOHS
1997
--­
EPA 314;
STl

tWvy I INev
I
.. WlIlIIXJI1Ilndueb1lII
Mame Corpe R.-w PIn. McGregor AnIlI R
T_
10peral 0neI Teetk1g I~er I
27
I
41
I NO -110.0
I Perctbllle IArelI R IrdulIlrilIIde; GnxnIwater
~ to &riace Willer patIway;
baled ~ Commerlcal/lrdulllrilll War1ter
ISlate I OERP

CALOHS
1997
-STl
Perctbale
I
NlIv8I
I~T~ I IArelI
tWvy I
Mame Corps R.-w PIn. McGregor Nee R
T_
I
WlIlIIXJI1Ill1lUlrlIIl
l&riace Willer I I 4 7
I
NO-23.0
by IC
melhod. to
R II1lUlrlIIl ...; Groundwater
&riace
Willer pelIway;
baled on Commertcal/lnduelrl8l Worker
CALOHS
ISlate I DERP

1997
-STl

tWvy I INevel
Weapone 1nduelrl8l I
MarIne Corpe R~. PIart. McGAIgor Area R
Texas
10peratklnaI TesIIng I~ I I 1 21
I
NO-59.0
PerchlOlllle
I melhod.
bytC
ArelI R 1nduelrl8l de; Pot..- for
101 to leech to 1JI'OUIldw8ler.
Groundwater to &riIce Water
baled on 8lqlOlI\n patIway;
ISlate I OERP

CALOHS Commerlcal/lrdulllrilll Worker


1997
-
STl
Perct1IorlIle
l"avaI by~ INone
Navy I
. Texas
Weapone IrdulIIrilII I
Merine Corps R~. Plenl. McGAIgor Area R 10peratklnaI Testing ISedknft I
0
I 2
/
NO I melhod.
baled on
CALOHS
IState I DERP

1997
-STl

Navy I I
NlIv8I
I
WeaporelncUlri8l
MarIne Corps R.-v. PIart. McGAIgor OZ Ofrah
Texas
l0peratlonal Testing I~ I 1
I
33
I I ND-370.0
Perct1IorlIle Varlou& III8I1UfactIPlg .... ons••;
bylC
PoterftIl for 10110 leech Into
melhod. grllInIweler. Groundwater to
baled on &riace Water8lqlOlI\n patIway;
ISlate I OERP

CALOHS Resldentual
1997

Fie: PercOala_AlSvca_Apt 2217_

ShIel: Env Reslorafion.CIe 0lIw • Method 314 reporting lin. 114 ppb Page 17 of 73

lIervIce I Fllell", I 81ta I


Source of Pen:hIome (Opendlons.
Equipment, Ac:tlvItJes) I
Loc:.tIon
Detected
(Ihdla) I"o:c"~I-~I

u_ -~E 8M1P'"
Collected
Co~
Detectad (ppb)
Method IPocemgJ Pethwlly(e) of Exposure/permitting I FUndl1lll Type

r
EPA 314;

I
STL
arlous
Navy I
NaYlII Weapona IrWIlJIaI
I~ PlaIt. McGnlgor
MarIne Corps T_
OZ 0IlsIe 10penIli0nBI TMIIng I St.tace Water I 4
I 21
I
NO' -370.0
I Perctbale
by IC Groundwat
method.
based on
CALDHS
er to St.tace-Waler;
rnaoofactlmg
R86IdenluaI
- onsIe·'Istate
I DERP

1997
---
EPA 314;
STl

=:. NaYlII W88POI1IIrWIlJIaI


CorpsIR_ PIart McGnlgor
T_
I
OZ 0IlsIe 10pe-8lI0nlII T86llng IGroundwater I 389
I
804 I NO - 56.000.0
I PelcNor8Ie IVarious III8IIlfaclImg - - onsIe;i
by IC Groundwater to St.tace Waler; lstate
method. Re8lcIlImJaI
based on
I DERP

CALDHS
1997
-STl

=.:. I I~TMIIng I~
P8rl:NonIle
IVarious ~
NaYlII W88POI1I1nlll.IetrWI
CorpsIR_ PIart McGnlgor
T_
OZ 0IlsIe
I
2
I
19
I NO-720.0
I by IC
method. Groundwater to
based on Re8lcIlImJaI
CALDHS
St.tace--Waler;onsIe;(Stale I DERP

1997
--
EPA 314;
STL

I I PeI:;:-IDIfnkIng water ~ pIarC;


Navy I
MarIne Corps ~ Plart. McGnlgor (Olf Post)
T_
I
NaYlII W8lIPOIlIlrWIlJIaI Bell Co. Irtake 10perati0naI
T86llng;
Marn-ncelReconIlgIntion; Staage;
Demillarlzation'[)l
1St.tace Wal_
I I I 1 45 NO' - 4.1
method.
based on
CALDHS
AllIidertI8I.­
(state I DERP

1997
--
EPA 314;
STl

I I~I~
I I I
Navy I NaYlII Weapona Indu8IriaI IEIIuBbonnet
rJ*lItionai T86llng;
Marlne Corps R.-ve Plart. McGnlgor Irtake (Olf Post) Malrt8llllflC6/ReconIlgLl1ltlon; Staage;
T_ Dem~
1St.tace Water 0 44 NO
method.
(state I DERP
based on
CALDHS
1997
--­
EPA 314;

Navy I I
NaYlII Weapona Industrial
MarlneCorps R_PIart McGnlgor (OIfPosl)
T_
Irtake IGatesvIe 10perat1onal
T86llng;
~ Storage;
Dem~
1St.tace Water I I I I
0 46 NO
STL

. PelbylC
cNor8Ie INone
method.
lSt8le I DERP
based on
CALDHS
. , I I , I I
1997

fie: Pen:D8Ia_AISvc8_Apl2217_

Sheat E"" R8lItonIlion-CI. 0lIw ' Method 314 reporting Imlle 4 ppb Page 18 af73

RlIgulatoly

service I Fec:HIty I Site I


SouR. of Perchlonte (OpeqtJona.
Equlpment, ActIvltIea) I
Location
Detected
(Medlll)
IN"-"'I-"I . . . 1-'1
. Detec:tlone S.."pl..
CollllCtM
Concentrattone
Detec:Wd (ppb)
~ I='~ I
PotentI8l Pathway(lI) of Expoaure permlltlng
requlremenls,
FunclllllI Type

sampllng
1'8q\kenleIU)
I I
EPA 314;
STL

I
N8VlII
lHarril (10pend10nB1
ISI.rf- 1~81e
I /~ water; agrleubnl;
Navy I WeeponIlrWalrl8I CnIek TN1lrV; by IC Groundwater to SI.rface water;
Marn Corps R_ PIert. McGregor Poat) MalrDInenceo'ReconItglnlIon; Storage; . 21 62 NO' -17.0 IState I DERP
Waler method.
T_ . DemIllarlzallooY[) I I based on
CALDHS
1997
--
EPA 314;

= Nllvlll WeeponIlrWalrl8I
Corps/R_ PIart. McGregor
T_
I lOpendlonBl TN1lrV;
~ Belton (011 MIilo".lC8JR~ Storage; ISI.rface W8lfJI
~ I I
1110 790
I
NO' - 210.0
1P~e
STL

bytC 1000water~~
melhod resa'VOir; resldeRlalllnd
based ~ agrIc:UbnI receptors
CALDHS
IState I DERP

1997
-
STL

Navy I I
NllvIll
WeeponIlrWalrl8l
Marn Corps ~ PIart. McGr8gor Poat)
T8llIIS
1'"
Belton (OII/DpenItlonII TN1lrV;
~ Storage;
DemIllarlzallooY[) ISedDMwt I I I 39 153 NO-540.0
I
~
by IC 100* water ~ 18cn'IIIIoIIII
melhod. resa'VOir; I'lIIIdenIeI ~
based on egrIcUIInI receptors
I StaIB I DERP

CALDHS
1997
-
STL

I I I I ~ I~
P~

Navy I I
NllvIII
Weepona IrWalrl8I
Marn Corps R_ PIart. McGr8gor Po8l)
Weco (011 ILeke Testing;10perali0neI
MeInl_nceJR~S\onIge; ISI.rface W8lfJI 1 291 NO -17.4 ~
weier I8Cl1IIIlIoIWII ISlet. I DERP
Texas Demlilerlzatlonl[)l based ~ resa'VOir; I'lIIIdenIeI receptors
CALDHS
1997
-STL

Navy I I
Navlll

ILeke Weco (Off /0MeInl_nceJReconflgtnllon;


Weepona Induslriel
Marn Corps RMfJlVe PIart. McGnIgor Po8l)
T8llIIS
peralI0naI Testing; Sknge;
DemlilarizationlDisposai
ISedm-t I I I I
0 69 NO
PerchIoret8

bylC
melhod. I None
based on
IState I DERP

CALDHS
1997
---
EPA 314;
STL

I~=
Navel Weepons Induslriel ILeon River (Off 10000etional Testing;
I IP8I~8le river thet 8lqlI1es Leke Belton Is

I I
Navy I
MImI Corps PIart. McGnIgor Po8l) ===~tnlIon;Storage; SI.rface W8lfJI 14
I
124 NO- - 27.0 melhod water; I'lIIIdenIeI ~ agrIcUturaI IStaIB I OERP
based ~ receptors
CALDHS
1997

Fie: PercOIIte_AlSvce_AprI2217_

ShI8t Env Restoration-Oee. au.. • MBlhod 314 f8PllIllnlIlmIllI 4 ppb Page 19of73

Regulatory

8ervlc4I I Facility I 81te I


Soun:e of ~ (Oper.u-,
Equlpmenl, ActIvItIeII) I
Location
Detected
(Media)
I : n.: I Semples C~
of Nwnberof I I· I
Rangeof
one Coilect8cI Detect8cl (ppb)
~ PoWntlal Pathw8y(a) of ~:=~~
~.
I permlltlng. Funding Type

sampling
ruquiremefts)
I I
EPA 314;

Navy I I
Neval
W....
InduItrIal
Marine Corps R_ PIln. McGregor (011 Post)
T_
ISCUh
Boequa R./Opemlon8I T88tIng;
Malmlc8llC8lRtICOI'IIlgmIlIo Slor8ge;
Dem~
l&nace Water
I I I I
39 129 NO" -13.0
STl

~ate r~~erw~_water.
meIhod egrtcUh.nI and I'8SkIeftI8I
based ~ receplorI
IState I DERP

CALDHS
1997
--­
EPA 314;

I
STL

Navy I INaviI Waepons


InduItrIal ISlIIlion
Creek
Marine Corps R_ Plart. McGregor (011 Post)
T_
(MalrUrwIC8IRecclI'IIIgtnIIo
)peqtDW T88tIng; Slor8ge;
Dem~
I&naceW" I 58
I
85 I NO" - 8,600.0
P8:::,ate IGnxnlwaler to &nacewater. ISlate
meIhod, Strlace
based on
water. agrIcuIlInI receplor I DERP

CALDHS
1997
--­
EPA 314;

I
STl

I W.... 10pa-aI 0nsI


I I I
NlIVII
Navy I InduItrIal ITempIae II'UIce T88tIng; P8:;':;14e INone
Marine Corps R_ Plait. McGregor (011 Post)
T_
MalrHnIInceIReconIIgtnlIon; Slor8ge;
D8mIIarlzlIIIon \&naceW'" 0 44 NO
meIhod.
IStale I DERP
based on
CALDHS
1997

EPA 314;

Navy I INlIVII Weepons tndultrlal Iwacos Irtake


Marine Corps R_ Plarl McGregor (Off Post)
T_
I
Dem~
()peratIon8I Testing;
Malrt8nlInceIReconIIglDllon; Storage; ISui_Water
I I I I
0 44 NO
STL

PfJA:::,ate I None
meIhod,
IStale I DERP
based on
CALDHS
1997

EPA 314;
STL

INaval Weepons InduslrlaI IwlIdlkMvs I~ Testing; 1~ l&nace


N I
,;;:. Corps ~=- Planl. McGregor Inlake (Off Post) =,lDIIon; Storage; l&nace Water
I I I 2 45 NO" -8.0
meIhod, receplorI
based on
CALDHS
water: irrigation; reslcIllnlal lslate I DERP

1997
--­

= Nav8I Weapons tnduslrlal I


CorpsjR_ Plait. McGregor Area 0
T_
!Slorage f~ /
82
I 88
I NO" -180.0
EPA 314;
STl

I byte Worker
Perchlorate Area 0 IndusIrtaI Area;
Gnxnlwaler wStrlace water
meIhod, palIway; Commertcal/1nduab1al
based on
CALOHS
1997
IStale I DERP

fie: Pen:Data_AlSYcs_Aprt 2217_

Sheet: Em RlIlItorallooOea. Olher "Melho;d 314 ~ Imlls 4 JlIlb Page 20d 73

lOClltlon Number 01 RHgeof


ServIca I Fac:llity I Site I
Boun:e 01 Pen:1IIome (OperMloM.
Equipment, ActMtIeaI I 0aWc:tecI
(Medial
Number 01
Detectlona
SIdIp/ft
CoIIact8cI
C~
0aWc:tecI (ppbl
~ IPofantW PIllhw8y(.1 01 ExpoaunjJ*llllllrV I Fundlng type

r
STl

Navy /
lMll

T_
I
WeeponIlnduIlrlIII
MarIne Corpa ~ Plant. McGnIgor Arrta 0 1&cAge I~ I I 1 7
I I ND-2S.0
PercIbate
bylC
IVea
0 InduIlrlIIIlVea; f>olertIaI for
sal 10 leach InIo groundwater.
method. GrOlRlw8ler to Ilrlace WlII8r
based on 8llPOI'A~
ISlate I DERP

CAlDHS Commerlc8lllndullrllll Worlter


1997
-
STl

Navy /
Marlne Corpa ~ Plan. McGregor
T_
I
(Naval WeeponIlnUlrl8I Arrta G 1&cAge I~ I 0
I
2
I
NO IPercIbate
by~
mathod.
based on
INone ISlate I DERP

CAlDHS
1997
--
STl
PerchIorlIht
I I~ I I I
Nlwal
Navy / Weapons InUlrl8II I mathod.
bylC
INone
MarIne Corpa ~ PIart McGregor Arrta G
T_ ISknue 0 4 NO
based on
ISlate I DERP

CAlDHS

-­ 1997
EPA 314;

I I
STl

I I~
PercIbate
by ~
I I
Naval
Navy / Weapons Industrial
MarIne Corpa R_ P1anl. McGregor Area H 9aege 15 32 NO· -41.0 method to
IArSwface
ta
H aIonIge - ; GroundwlIl..
W8I8r; IStat. I DERP
T_ 1 1 based ~ Commertcal/lndullrllll Worlter
CAlDHS
1997
-STl
INaval I Perctb1Ile
by ~ IIVea H aIonIge .-; Grcu1r:IwlD­
Navy / Weapons Industrial
MarIne Corpa Reswve Plant. McGregor Area H
Texas
19aege ISwface Wat.. I I I 2 4 NO-9.7
I m8lhod. to Swface WtAer. 1St8t8
based on Commerlc8lllndullrllll Worlter
CAlDHS
I DERP

1997
-
STl

Navy / I Navai
Weapons Industrial I
MarIne Corps R_ P1anl. McGtegor Area H
Texas
ISknQe I~ I I I I10 51 NO - 93.000.0
Perctb1Ile Area H s1aege 8IlIII; f>olertIaI for
bylC
sal 10 leach InIo groundwater.
method. GroundwlIl.. to
based on exposu-e~
Swface WBtw IState I DERP

CAlDHS Commerlcal/lndustrilll Worlter


1997

FlIt: Pwdlata_AlSvce_AprI2217_

Sheet. E"" Restor1IlIon-Cl. ou. • MMhod 314 repartlng lint Is 4 ppb Page 21 of 73

Service I FKIIIty I Site I


8oun:e of pen:hlome (OpemJona.
Equipment, ActIvItIeaI I
LOC8tIon
Oetectect
(1hcI1al
=
1 1-"" _.. 1 I &.mplM
Collect8d
Concentrau­
Oet8c:tecl (ppbl
~ PotentIal pethwllYCalof -1="-
Reguletory

permlltlng
requftmenla,
sam~
requQrmris)
I Fundlng Type

I I
STL

Navy / INaval
W8lIpOllIindusb1aI
MIwlne Corps RM«Ve Planl. McGIegor Nee H
T_
I \&uage I~ I I 0 2
1
NO
I Perctbale
bylC
1IIlllhod. I None
b8sedon
IStale I OERP

CALDHS
1997
-­EPA 314;
I
STL

I ISoi 1 Peu:tuale
I Nee 0 InduIlrlaI Area; PoteniaI for
Navy /. Naval W8lIpOI1B IndustrtaI
MIwlne Corps ~ PIart. McGregor Nee S
T_
l&uage I I 44 119 NO· 1.500,000.0
by IC
elhod
m •
based on
. CALDHS
sol conlamlnation 10 leech lnIo
groundwater. Groundwater 10
Nface water exposure ~
Commartcalllndusb1al WOI1<8'
ISlate I OERP

1997
-STL

=~ Corpsl~
T_
N8vlII W8lIpOllIindustrtaI
PIa,., McGIegor
I
Nea S I~ I~ I 1
I
4
I NO-120.0
I"~"F=-
1IIlllhod. paltMay; Commerlc8l/lndustrtal
b8sedon '
IStaIe I OERP

CAL DHS WOI1<8'


1997

EPA 314;

I STL

I I
I I
Perd1Iorale Nee 0 Indusb1aI Ar88;

I~
Naval
IGroundwater I
Navy / W8lIpOllIindusb1aI by IC Groundwater 10 Nface water
Mama Corps R-.ve Planl. McGregor Ar88 S 117 140 NO'· 13,000.0
1IIlllhod. ~ CommartcalIII1llJItrI8I
ISlate I OERP
T8lC8lI
based on Wortcer
CALDHS
1997

EPA 314;
STL

I
Naval
1 Perd1Iorale Nee 0 II1llJItrI8I Ar88;
Navy / W88pOI1B Induslrlal
Mama Corps RMerve Plant. McGregor Ar88 S
T_
Istorage . ISuface Watel I 31
I 97
I
NO' - 810.0 J bylC Groundwater 10 Nface water
1IIlllhod. ~y. CommartcalIInduslrlal
ba88don WOI1<8'
CALDHS
IStale I DERP

1997
-
STL

Navy / IN8vlII
Weapore Indusb1aI
MIwlne Corpe R-.ve Plant. McGregor Ar88 A
T8lC8lI
I I ISoi I I I I0 1 NO
PercNorale
bylC
melhod. INone
b8sedon
IStale I DERP

CALDHS
1997

fie: PercDala_AlSvca_AprI2217_

St&ll: Env ReetoratIon-CIe. 00.. , Melhod 314 repoItIng Iinlls 4 ppb Page 220173

8aMce I F8ClIlty I 8.. I


Source of ~ (0peratI0na,
Equipment, ActIvltIaa) I
LOC8tlon
DatectH
(Media)
=
I I I Nwnberof Rang. of
&.npIea ConcantratJona
CoIlac:ted Datected (ppb)
I~ I PotentIal Pllthway(.) of

I
~=~
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