Professional Documents
Culture Documents
EPISIOTOMY
STUDENT NAME—Miss Sushruta Mohapatra
YEAR OF STUDY—2018
IDENTIFICATION DATA
AGE— 25years
SEX— Female
RELIGION— Hindu
EDUCATION— Graduation
DISCHARGE DATE—
OCCUPATION— Housewife
WEIGHT— 70 kg
HIGHT—152cm
CHIEF COMPLAINS—
FAMILY HISTORY—
She belongs to a joint family having 8 numbers . Her husband &2 Brother-in-law are the supporting person in her family. The monthly income
of her family is nearly about Rs 45,000. There is no history of any disease like TB,HTN, DM & hereditary disease , twin pregnancy in her family.
There is a CHC in her village at a distance of about 6 km. Transportation facility available like bicycle & motorcycle
HOUSING —
She lives in a pucca house having 8 numbers of rooms with adequate ventilation. They use sanitary latrine for toileting. Electricity supply is
available. They use municipality water supply taps as well as own borewell for drinking.
PERSONAL HISTORY—
PERSONAL HYGIENE—She is maintaining her oral hygiene by brushing daily and taking bath once daily with soap & normal water.
DIET—She takes both vegetarian & non-vegetarian diet & She takes meals 4-5 times a day. She don’t have any addiction of alcohol &
tobacco. She drinks about 3-4 lts of water per day. She takes rest of about 2 hrs at day time & 8 hrs during night time. She takes no
drugs for sleep.
ELIMINATION—She has a regular bowel & bladder habits
MOBILITY & EXERCISE—No regular walking habits. Only moderate activity with normal house hold work.
MENSTRUAL HISTORY—
She got menarche at 14 year of age with regular cycles of 28-30 days interval & 3-4 days duration with average amount of bleeding. Her LMP is
16/4/18 and EDD- 23/01/19.
She is married since 1and 1/2 years & She has satisfactory relationship with her spouse. General health of her spouse is good.
OBSTETRICAL HISTORY—
INVESTIGATIONS—
Hb=7.9gm%
FBS=85mg/dl
Urine for HCG=positive
Blood group— ‘B’ positive
Sickling -- Negative
Urine test=Albumin-- Not Present
=Sugar--Not Present
VDRL=Negative
HIV=Non reactive
HbsAg =Non reactive
HCV =Non reactive
USG= done on 11/12/18 showing single live intra-uterine fetus in cephalic presentation.
PHYSICAL XAMINATION—
VITAL SIGN—
Temp –98.4 F
BP—130/80mmhg
Pulse –88beat/min.
Resp –22 braeth/min.
OBSTETRICAL EXAMINATION—
INSPECTION—
PALPATION—
P/V EXAMINATION—
INTRODUCTION— vaginal delivery is a normal procedure of child birth which due to the changes occurs in the female genital organs that can
able to push the viable products of a conception out of the maternal uterus.
DEFINITION— Delivery is the expulsion or extraction of a viable fetus out of the womb.
Labour: Series of events that takes place in the female genital organs in an effort to expel the viable products of a conception out of the womb
through the vagina in to the outer world is called labour.
STAGES OF LABOUR
IN BOOK IN PATIENT
First stage: It starts from the onset of true labour pain & ends The duration of 1st stage is 9hr 30min.
with full dilatation of cervix. Its duration is 12 hour in
primigravida & 6 hour in multipara.
Second stage: It starts from full dilatation of cervix & ends The duration of 2nd stage is 1hr.
with expulsion of fetus from the birth canal. Its duration is 2
hour in primigravida & 30 min in multipara.
Third stage: It starts after expulsion of fetus & ends with The duration of 3rd stage is 10min.
expulsion of placenta & membrane. Its duration is 15 min in
both primigravida & multipara.
Fourth stage: It is the stage of observation for at least 1hour The duration of 4th stage is 1hr.
after expulsion of after birth products.
MECHANISM OF LABOUR
To accommodate itself to the maternal pelvic dimensions, the fetus must undergo a series of changes in the attitude of its presenting part. This
is required for fetal descent through the birth canal.
Nine discrete cardinal movements of the fetus occur over the course of labor and delivery: engagement, descent, flexion, internal rotation,
crowning, extension, external rotation, restitution, expulsion of trunk & shoulder.
Engagement: Engagement is the descent of the widest part of the fetus through the pelvic inlet. This normally occurs 2-3 weeks before labour
in nulliparous women and may occur any time before or after onset of labour in multiparous women. In this the antero-posterior diameter or
biparietal diameter (9.5cm) of head coinsides with the transverse diameter of maternal pelvis.
Descent: Descent refers to the downward passage of the presenting part through the pelvis. Descent of the fetus is not a steady, continuous
process. The greatest rate of descent occurs during the deceleration phase of the first stage and during the second stage of labor.
Flexion: While descending through the pelvis, the fetal head flexes so that the fetal chin is touching the fetal chest. This functionally creates a
smaller structure to pass through the maternal pelvis. When flexion occurs, the occipital (posterior) fontanel slides into the center of the birth
canal and the anterior fontanel becomes more remote and difficult to feel. The fetal position remains occiput transverse.
Internal Rotation: Internal rotation is the 2/8th rotation of the presenting part from its original position (usually transverse with regard to the
birth canal) or 1/8th if the presenting part is in oblique diameter to the anteroposterior position as it passes through the pelvis. As with flexion,
internal rotation is a passive movement resulting from the shape of the pelvis and the resistance of the pelvic floor musculature.
Crowning: In this the biparietal diameter of head stretches the vulval outlet without any recession of head even after the contraction is over.
Extension: Extension occurs once the fetus has descended to the level of the introitus. This descent brings the base of the occiput into contact
with the inferior margin of the symphysis pubis. At this point, the birth canal curves upwards. The fetal head is delivered by extension and
rotates around the symphysis pubis. The forces responsible for this motion are the downward force exerted on the fetus by uterine
contractions and maternal expulsive efforts along with the upward forces exerted by the muscles of the pelvic floor.
External Rotation: After the fetal head deflexes (extends), it rotates to the correct anatomic position in relation to the fetal torso; left or right
rotation depends on the orientation of the fetus. This is again a passive movement resulting from a release of the forces exerted on the fetal
head by the maternal bony pelvis and its musculature and mediated by the basal tone of the fetal musculature.
Expulsion of shoulder & trunk: Expulsion refers to delivery of the body of the fetus. After delivery of the head and external rotation, further
descent brings the anterior shoulder to the level of the symphysis pubis. The anterior shoulder rotates under the symphysis pubis, after which
the rest of the body usually delivers without difficulty.
EPISIOTOMY:
Definition:
Episiotomy, also known as perineotomy, is a surgical incision of the perineum and the posterior vaginal wall generally done by a midwife or
obstetrician. Episiotomy is usually performed during second stage of labor to quickly enlarge the opening for the baby to pass through. The
incision, which can be done at a 90 degree angle from the vulva towards the anus or at an angle from the posterior end of the vulva (medio-
lateral episiotomy), is performed under local anesthetic (pudendal anesthesia), and is sutured after delivery.
1. Medio-lateral: The incision is made downward and outward from the midpoint of the fourchette either to the right or left. It is directed
diagonally in a straight line which runs about 2.5 cm (1 in) away from the anus (midpoint between the anus and the ischial tuberosity).
2. Median: The incision commences from the centre of the fourchette and extends on the posterior side along the midline for 2.5 cm (1
in).
3. Lateral: The incision starts from about 1 cm (0.4 in) away from the centre of the fourchette and extends laterally. Drawbacks include
the chance of injury to the Bartholin's duct, therefore some practitioners have strongly discouraged lateral incisions.
4. J-shaped: The incision begins in the centre of the fourchette and is directed posteriorly along the midline for about 1.5 centimetres
(0.59 in) and then directed downwards and outwards along the 5 or 7 o’ clock position to avoid injury to the external & internal anal
sphincter.
INCIDENCE –
ETIOLOGY —
IN BOOK IN CLIENT
In majority causes are not known. Idiopathic
Possible causes are - ( Causes are not known)
Increased friability of the membranes
Decreased tensile strength of the membranes
Polyhydramnios
Cervical incompetence
Multiple Pregnancy
Infection – Chorio – amnionitis, Urinary tract infections
and lower genital tract infection
Cervical length < 2.5 c.m
Prior preterm labour
Low BMI ( < 19 kg/m2)
IN BOOK IN CLIENT
Head reeling Present
Weakness Present
Edema Present
Mild :- rise of blood pressure of more than 140/90mmhg but less than 160 mmhg systolic or 110 mmg diastolic without significant protienuria.
Severe :- 1) a persist systolic blood pressure above or equal to 160mmhg or diastolic pressure above 110mmhg. 2) protien more than
5g/24hrs. 3) oliguria 4) HELLP syndrome. 5) cerebral or visual disturbance.
DIAGNOSIS –
Immediate :-
During pregnancy :- accidental hemorrhage ,oliguria and anuria , dimness of vision and even blindness, preterm labour, cerebral haemorrhage.
IN BOOK IN CLIENT
Consistent BP recording of 140/90 mmhg or more . Done
Physical examination may reveal the long term effect of HTN such as – Done
retinopathy, ischemic heart disease & renal damage
COMPLICATION
FETAL :
Fetal risk is related to the severity of preeclampsia ,duration of the disease and degree of protienuria a) IUD b) intrauterine growth
retardation c) asphyxia d) prematurity.
MANAGEMENT—
IN SEVERE CASES :-
IN BOOK IN CLIENT
Hospitalization Done
Antihypertensive drug (when BP > 160/100 mm/hg) Done
Spontaneous labor at term in mild cases Not Done
Termination done after 38 weeks in severe cases Not Done
Treatment of mild to moderate chronic HTN neither benefits the fetus nor prevents pre eclampsia .Excessively lowering BP may result
in decreased placental perfusion & adverse perinatal outcomes.
When a patient’s BP is persistently > 150-180 (systolic) & 100-110 mm/hg(diastolic), pharmacologic treatment is needed to prevent
maternal end organ damage
Methyldopa, Labetalol & Nifedipine (Procardia) are oral agents commonly used to treat pregnancy induced hypertension.
Angiotensin converting enzyme inhibitors angiotensin-II receptor antagonists are not used because of teratogenicity ,IUGR, neonatal
renal failure .
The beta blocker , atenolol (Tenarmin) has been associated with IUGR
Women in active labour with uncontrolled severe chronic HTN require treatment with IV labetalol or Hydralazine.
Advice
Foetal growth may be assessed by serial fundal height measurements supplemented by USG at every 4 weeks starting at 28 weeks of gestation
DAY – 1 (27/11/2017)
DAY 2 (28/11/17)
GENERAL CONDITION OF ADVICE NURSING INTERVANTION
MOTHER--
DAY 3 — (29/11/17)
ADVICE ON DISCHARGE :
SUMMARY—
Mrs rasmita is a multigravida having GA 36 weeks & with pregnancy induced hypertension, is taken to improve nursing care. The care giver
established a good IPR with the client & her trust & confidence was gained. The client revealed all her problems, thus the care giver was able
provide care to meet the need up to an optimum. During this period she gains knowledge on different aspects like care of herself, regarding
possible complications, regular follow up, which makes her more confident & her anxiety was reduced & due to this she is now able to cope to
any stressful situation . She was also educated on nutrition, personal hygiene, antenatal exercise & regular follow up.
CONCLUSION—
Effect of my care—
After providing nursing care, the client has improved her self confidence .She feels relaxed & no anxiety is there. The client & family members
are very co-operative & they have trust on me.
BIBLIOGRAPHY—
1. DUTTA.DC “ Text book of obstretics “jaypee brothers medical publisher ,new delhi ,(2016).pgno-255-270.
2. Jacob annamma, A text book of midwifery ang gynaecological nursing, jaypee brothers medical publishers, new delhi 3rd edition (2012)
pg no- 572.
SUBMITTED BY : Miss Madhusmita Nayak
SUBMITTED ON :04/04/2014