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List of hematologic conditions

From Wikipedia, the free encyclopedia

This is an incomplete list, which may never be able to satisfy certain standards for completion. There are many conditions of or affecting the human Hematologic system the Biological system that includes plasma, platelets, leukocytes, and erythrocytes, the major components of blood and the bone marrow.[1]

Contents
1 Anemias 1.1 Nutritional anemias 1.2 Haemolytic, Aplastic and other anemias 2 Blood cancers 2.1 Lymphoma 2.2 Leukemia 2.3 Myeloma 2.4 Malignant immunoproliferative diseases 3 Coagulation, purpura, and other hemorrhagic conditions 4 Infection-related 4.1 Bacterium-related 4.2 Protozoan-related 5 Immune system regulation-related 5.1 Immunodeficiency with predominantly antibody defects 6 References

Anemias
Main article: Anemia An anemia is a decrease in number of red blood cells (RBCs) or less than the normal quantity of hemoglobin in the blood.[2][3] However; it can include decreased oxygen-binding ability of each hemoglobin molecule due to deformity or lack in numerical development as in some other types of hemoglobin deficiency. Anemia is the most common disorder of the blood. There are several kinds of anemia, produced by a variety of underlying causes. Anemia can be classified in a variety of ways, based on the morphology of RBCs, underlying etiologic mechanisms, and discernible clinical spectra, to mention a few. The three main classes of anemia include excessive blood loss (acutely such as a hemorrhage or chronically through lowvolume loss), excessive blood cell destruction (hemolysis) or deficient red blood cell production (ineffective hematopoiesis). Based on 20052006 estimates, the Centers for Disease Control and Prevention, has stated that approximately 5.5 million Americans a year are either admitted to a hospital or seen by a physician, with some form of anemia as their primary diagnosis.[4]

The pale hand of a woman with severe anemia (right) in comparison to the normal hand of her husband (left)

Nutritional anemias
A nutritional anemia refers to a type of anemia that can be directly attributed to either a nutritional disorder or a nutritional deficiency. Medical Subject Headings

Condition name

ICD-10 coding number

Diseases Database coding number

Iron deficiency anemia

D50. 6947 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb6947.htm) gd50.htm+d50)

Iron deficiency anemia (or iron deficiency anaemia) is a common anemia that occurs when iron loss (often from intestinal bleeding or menses) occurs, and/or the dietary intake or absorption of iron is insufficient. In such a state, hemoglobin, which contains iron, cannot be formed.[5] Plummer-Vinson syndrome D50.1 10134 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb10134.htm) gd50.htm+d501)

Plummer-Vinson syndrome (PVS), also called Paterson-Brown-Kelly syndrome or sideropenic dysphagia presents as a triad of dysphagia (due to esophageal webs), glossitis, and iron deficiency anemia.[6] It most usually occurs in postmenopausal women. Vitamin B12 deficiency anemia E53.8 13905 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb13905.htm) ge50.htm+e538)

Vitamin B12 deficiency anemia occurs when a "lower-than-normal" amount of the vitamin B12 is available within the body, leading to a decreased production of healthy red blood cells.[7]

Vitamin B12 deficiency anemia due to intrinsic factor D51.0 deficiency (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d510) vitamin B12 deficiency is caused by a lack of intrinsic factor, as seen in pernicious anemia, causes a vitamin B12 deficiency Pernicious anemia D51.0 9870 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb9870.htm) gd50.htm+d510)

Pernicious anemia (also known as Macrocytic achylic anemia, Congenital pernicious anemia, Juvenile pernicious anemia, and Vitamin B12 deficiency) is one of many types of the larger family of megaloblastic anemias. It is caused by loss of gastric parietal cells, and subsequent inability to absorb vitamin B12. Pernicious anemia is the result of inadequate production of the protein intrinsic factor needed by the body to absorb vitamin B 12, causing a reduction of new red blood cells.[8] Vitamin B 12 deficiency anemia due to selective vitamin B 12 malabsorption with proteinuria Vitamin B 12 deficiency anemia due to selective vitamin B 12 malabsorption with proteinuria (also known as Imerslund-Grsbeck syndrome) is a rare autosomal recessive disorder which requires the indefinite administration of Vitamin B12 injections.[9] D51.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d511) , D52.0 29507 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb29507.htm) gd50.htm+d520) , D53.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d531)

Megaloblastic hereditary anemia

Megaloblastic anemia (or megaloblastic anaemia) is an anemia (of macrocytic classification) that results from inhibition of DNA synthesis in red blood cell production.[10] Transcobalamin II deficiency D51.2 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d512)

Transcobalamin II deficiency (TCII) (also known as hereditary transcobalamin II deficiency) is a rare autosomal recessive disorder that results in neurological dysfunction.[11] Transcobalamin II are a type of carrier proteins which bind with plasma vitamin B12 (cobalamin) in the production of red blood cells.[12] D52. (http://apps.who.int/classifications/apps/icd/icd10online/? 4894 gd50.htm+d52) E53.8 (http://www.diseasesdatabase.com/ddb4894.htm) (http://apps.who.int/classifications/apps/icd/icd10online/? ge50.htm+e538)

Folate-deficiency anemia

Folate-deficiency anemia (also known as dietary folate-deficiency anemia) is a condition that develops when the body does not have the adequate supply of folic acid available that is needed for the production of new healthy blood cells.[13] D51.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d511) , D52.0 29507 (http://apps.who.int/classifications/apps/icd/icd10online/? D000749 (http://www.diseasesdatabase.com/ddb29507.htm) gd50.htm+d520) , D53.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d531)

Nutritional megaloblastic anemia

Nutritional Megaloblastic anemia is an anemia (of macrocytic classification) that results from inhibition of DNA synthesis in red blood cell production.[10] Drug-induced folate deficiency anemia D52.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d521)

Protein deficiency anemia Protein deficiency anemia is an anemia that results from an inadequate intake of dietary protein.[14] E54. 13930 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb13930.htm) ge50.htm+e54)

Scurvy

Scurvy is a disease resulting from a deficiency of vitamin C,[15] which is required for the synthesis of collagen in humans.

Haemolytic, Aplastic and other anemias


Condition name Acanthocytosis Acanthocytosis (also known as Abetalipoproteinemia, Apolipoprotein B deficiency, and Bassen-Kornzweig syndrome) is a rare, genetically inherited, autosomal recessive ICD-10 coding number Diseases Database coding number Medical Subject Headings

condition that causes the inability to fully digest dietary fats in the intestines. The gene responsible for this condition is the microsomal triglyceride transfer protein gene.[16] D62.0 Acute posthemorrhagic anemia (http://apps.who.int/classifications/apps/icd/icd10online/? gd60.htm+d620) Acute posthemorrhagic anemia (also known as acute blood loss anemia) is a condition in which a person quickly loses a large volume of circulating hemoglobin. Acute blood loss is usually associated with an incident of trauma or a severe injury resulting in a large loss of blood. It can also occur during or after a surgical procedure.[17] 448 (http://www.diseasesdatabase.com/ddb448.htm) , D56.0 33334 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb33334.htm) gd55.htm+d560) , 33678 (http://www.diseasesdatabase.com/ddb33678.htm)

Alpha-thalassemia

Alpha-thalassemia (-thalassemia) is a form of thalassemia involving the genes HBA1 [18] and HBA2.[19] It is condition that causes a reduction of hemoglobin production. There are two types of Alpha-thalassemia named hemoglobin Bart hydrops fetalis syndrome (also known as Hb Bart syndrome) and HbH disease.[20] D50. (http://apps.who.int/classifications/apps/icd/icd10online/? 663 gd50.htm+d50) -D64. (http://www.diseasesdatabase.com/ddb663.htm) (http://apps.who.int/classifications/apps/icd/icd10online/? gd60.htm+d64)

Anemia

Anemia is a general medical term used to describe a number of conditions that result in a decrease in the number of red blood cells (RBCs) or less than the normal quantity of hemoglobin in the blood.[2] Anemia of chronic disease Anemia of chronic disease (ACD) (also known as anemia of inflammatory response) is a condition where the body converts iron into unused ferrin, causing a drop in hemoglobin production, and as a result; decreased red blood cell production and count. This is caused by a natural defense mechanism initiated by an inflammatory response in response to the underlying chronic disease.[21] Anemia in Kidney Disease and Dialysis Anemia in Kidney Disease and Dialysis results from the diseased kidney's inability to produce enough of the hormone erythropoietin. Erythropoietin is used to stimulate an adequate production of red blood cells from the bone marrow.[22] Anemia of prematurity P61.2 (http://apps.who.int/classifications/apps/icd/icd10online/? gp50.htm+p612)

Anemia of prematurity refers to a form of anemia affecting preterm infants[23] with decreased hematocrit.[24] D60. (http://apps.who.int/classifications/apps/icd/icd10online/? 866 gd60.htm+d60) -D61. (http://www.diseasesdatabase.com/ddb866.htm) (http://apps.who.int/classifications/apps/icd/icd10online/? gd60.htm+d61)

Aplastic anemia

Aplastic anemia is a condition where bone marrow does not produce sufficient new cells to replenish blood cells.[25] D59.0 (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d590) -D59.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d591)

Autoimmune hemolytic anemia

Autoimmune hemolytic anemia (AIHA) is a type of hemolytic anemia where the body's immune system attacks its own red blood cells (RBCs), leading to their destruction (hemolysis).[26][27] Types of AIHA include Warm autoimmune hemolytic anemia, Cold agglutinin disease, and Paroxysmal cold hemoglobinuria. Beta-thalassemia D56.1 3087 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb3087.htm) gd55.htm+d561)

Beta-thalassemia (-thalassemia) is an autosomal dominant blood condition that results in the reduction of hemoglobin production. The cause for the disorder is related to a genetic mutation of the HBB gene. This gene is responsible for providing the instructions to produce beta-globin; one of the major components of hemoglobin. The two classification types of beta thalassemia are thalassemia major (also known as Cooley's anemia) and thalassemia intermedia.[28] Diamond-Blackfan anemia D61.0 29062 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb29062.htm) gd60.htm+d610)

DiamondBlackfan anemia (DBA), (also known as BlackfanDiamond anemia and Inherited erythroblastopenia) [29] is a congenital erythroid aplasia that usually presents in infancy.[30] Congenital dyserythropoietic anemia D64.4 (http://apps.who.int/classifications/apps/icd/icd10online/?

gd60.htm+d644) Congenital dyserythropoietic anemia (CDA) is a generically inherited autosomal recessive (types I and II) or autosomal dominant (type III) blood disorder that effects the normal maturation process of red blood cell production. Mutations to the CDAN1 gene (type I), SEC23B gene (type II), and a currently unknown gene for type III causes a disruption in the normal formation of erythropoiesis, thereby causing a reduction of circulating healthy mature red blood cells.[31] Drug-induced autoimmune hemolytic anemia D59.0 (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d590)

Drug-induced autoimmune hemolytic anemia is a type of hemolytic anemia in which a mediated immune response triggers IgG and IgM antibody production in regards to the presence of high doses of penicillin via the hapten mechanism causing the reduction of red blood cells in the spleen.[32] Drug-induced nonautoimmune hemolytic anemia D55.0 19674 (http://apps.who.int/classifications/apps/icd/icd10online/? D005955 (http://www.diseasesdatabase.com/ddb19674.htm) gd55.htm+d550) D58.2 5037 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb5037.htm) gd55.htm+d582)

Glucose-6-phosphate dehydrogenase deficiency

Hemoglobinopathy

Hemoglobinopathy is a kind of genetic defect that results in abnormal structure of one of the globin chains of the hemoglobin molecule.[33] Hemoglobinopathies are inherited singlegene disorders; in most cases, they are inherited as autosomal co-dominant traits.[34] Hemoglobinopathies imply structural abnormalities in the globin proteins themselves.[35] Hemoglobinopathy variants include sickle-cell disease.[36] D55. (http://apps.who.int/classifications/apps/icd/icd10online/? 5534 gd55.htm+d55) -D59. (http://www.diseasesdatabase.com/ddb5534.htm) (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d59)

Hemolytic anemia

Hemolytic anemia (also known as haemolytic anaemia) is an anemia due to hemolysis, the abnormal breakdown of red blood cells. A number of different mediating factors can cause this condition; either from within the blood cell itself ((intrinsic factors) or outside of the cell (extrinsic factors).[37] Congenital hemolytic anemia D61.0 4745 (http://apps.who.int/classifications/apps/icd/icd10online/? D005199 (http://www.diseasesdatabase.com/ddb4745.htm) gd60.htm+d610)

Fanconi's anemia

Fanconi's anemia is a rare genetic autosomal recessive aplastic anemia that involves chromosomes 9q and 20q.[38] Hereditary spherocytosis D58.0 5827 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb5827.htm) gd55.htm+d580)

Hereditary spherocytosis is a genetically-transmitted (autosomal dominant) form of spherocytosis, an auto-hemolytic anemia characterized by the production of red blood cells that are sphere-shaped rather than bi-concave disk shaped (Donut-Shaped), and therefore more prone to hemolysis.[39] Hereditary elliptocytosis D58.1 4172 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb4172.htm) gd55.htm+d581)

Hereditary elliptocytosis (HE) (also known as ovalocytosis), is an inherited blood disorder in which an abnormally large number of circulating red blood cells are elliptical or cigar shaped rather than the typical biconcave disc shape. It is caused in part by mutations in the formation of specific Spectrin tetramers or proteins responsible for giving the red blood cell its shape and elasticity causing continued deformation as the cell matures.[40] Subtypes of this condition include southeast Asian ovalocytosis and spherocytic elliptocytosis. Hereditary pyropoikilocytosis Hereditary pyropoikilocytosis (HPP) is an autosomal recessive form of hemolytic anemia which typically presents at infancy or early childhood, characterized by abnormal red blood cell morphology including "budding red cells, fragmented red cells, spherocytes, elliptocytes, triangular cells, and other bizarre-shaped red cells." [41] Acquired hemolytic anemia D59.1 2949 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb2949.htm) gd55.htm+d591)

Cold hemagglutinin disease

Cold hemagglutinin disease (also known as cold agglutinin disease and autoimmune anemia due to cold-reactive antibodies)is an autoimmune disease characterized by the presence of high concentrations of circulating antibodies, usually IgM, directed against red blood cells.[42] It is a form of autoimmune hemolytic anemia, specifically one in which antibodies only bind red blood cells at low body temperatures, typically 28-31C. Paroxysmal cold hemoglobinuria D59.6 9679 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb9679.htm) gd55.htm+d596)
[43]

Paroxysmal cold hemoglobinuria (PCH) (also known as Donath-Landsteiner syndrome) is a rare condition characterized by the sudden presence of hemoglobin in the urine (called

hemoglobinuria), typically after exposure to cold temperatures.[43] Hemolytic-uremic syndrome D59.3 13052 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb13052.htm) gd55.htm+d593)

Hemolytic-uremic syndrome (HUS) (also known as haemolytic-uraemic syndrome) is a disease characterized by hemolytic anemia, acute renal failure (uremia) and a low platelet count (thrombocytopenia). It predominantly but not exclusively affects children. Most cases are preceded by an episode of diarrhea caused by E. coli O157:H7, which is acquired as a foodborne illness.[44] Hereditary persistence of fetal hemoglobin Hereditary stomatocytosis | |D58.8 29710 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb29710.htm) gd55.htm+d588) Hereditary stomatocytosis describes a number of inherited autosomal dominant human conditions which affect the red blood cell, in which the membrane or outer coating of the cell 'leaks' sodium and potassium ions, causing cell lyses and eventual haemolytic anaemia. Hexokinase deficiency D55.2 (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d552)

Hexokinase deficiency (also known as human erythrocyte hexokinase deficiency) is an anemia-causing condition associated with inadequate hexokinase.[45] Hyperanaemia Hypochromic anemia Hypochromic anemia is a generic term for any type of anemia in which the red blood cells (erythrocytes) are paler than normal.[46] This is caused by a proportionally reduced amount of hemoglobin present in relation to the size of the red blood cell. Ineffective erythropoiesis Ineffective erythropoiesis is an anemia caused by the premature apoptosis of the body's mature red blood cells [47] and subsequent reduction in an adequate production and full maturation of new healthy red blood cells.[48] Macrocytic anemia D51.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d511) , D52.0 29507 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb29507.htm) gd50.htm+d520) , D53.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gd50.htm+d531)

Megaloblastic anemia

Megaloblastic anemia (or megaloblastic anaemia) is an anemia of macrocytic classification that results from inhibition of DNA synthesis in red blood cell production.[10] Microangiopathic hemolytic anemia Minkowski-Chauffard syndrome D61.9 (http://apps.who.int/classifications/apps/icd/icd10online/? gd60.htm+d619)

Myelophthisic anemia

Myelophthisic anemia (also known as myelophthisis) is a severe kind of anemia found in some people with diseases that affect the bone marrow. Myelophythisis refers to the displacement of hemopoietic bone-marrow tissue into the peripheral blood,[49] either by fibrosis, tumors or granulomas. Neuroacanthocytosis 29707 D054546 (http://www.diseasesdatabase.com/ddb29707.htm)

Neuroacanthocytosis (also known as Levine-Critchley syndrome) are a group of rare, genetic conditions that are characterized by movement disorders and acanthocytosis.[50] Chorea acanthocytosis 29707 D054546 (http://www.diseasesdatabase.com/ddb29707.htm)

Chorea-acanthocytosis (ChAc)(also known as Levine-Critchley syndrome, acanthocytosis with neurologic disorder, neuroacanthocytosis, and choreoacanthocytosis)[51] is a rare hereditary disease caused by a mutation of the gene that directs structural proteins in red blood cells. It belongs to a group of four diseases characterized as neuroacanthocytosis.[52] Non sideropenic hypochromic anaemia Normocytic anemia A normocytic anemia is a general term for an anemia with an Mean corpuscular volume (MCV) of 80-100. Paroxysmal nocturnal hemoglobinuria D59.5 9688 (http://apps.who.int/classifications/apps/icd/icd10online/? (htt // d55 h d595)

di

d t b

/ddb9688 ht )

gd55.htm+d595) Paroxysmal nocturnal hemoglobinuria (also known as Marchiafava-Micheli syndrome) is a rare, acquired Pyruvate kinase deficiency

(http://www.diseasesdatabase.com/ddb9688.htm)

D55.2 11090 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb11090.htm) gd55.htm+d552)

Pyruvate kinase deficiency, also called erythrocyte pyruvate kinase deficiency,[53] is an inherited metabolic disorder of the enzyme pyruvate kinase which affects the survival of red blood cells and causes them to deform into echinocytes on peripheral blood smears. Rh deficiency syndrome Rh deficiency syndrome is a type of hemolytic anemia that involves erythrocytes whom membranes are deficient in Rh antigens. It is considered a rare condition.[54] Sickle-cell disease D64.0 (http://apps.who.int/classifications/apps/icd/icd10online/? 12110 gd60.htm+d640) -D64.3 (http://www.diseasesdatabase.com/ddb12110.htm) (http://apps.who.int/classifications/apps/icd/icd10online/? gd60.htm+d643)

Sideroblastic anemia

Sideroblastic anemia or sideroachrestic anemia is a disease in which the bone marrow produces ringed sideroblasts rather than healthy red blood cells (erythrocytes).[55] It may be caused either by a genetic disorder or indirectly as part of myelodysplastic syndrome.[56] Southeast Asian ovalocytosis D58.1 9416 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb9416.htm) gd55.htm+d581)

Southeast Asian ovalocytosis (also known as Stomatocytic ovalocytosis, stomatocytic elliptocytosis, and Melanesian ovalocytosis) is a form of hereditary elliptocytosis common in some communities in Malaysia and Papua New Guinea, as it confers some resistance to cerebral Falciparum Malaria.[57] Spur cell hemolytic anemia Spur cell hemolytic anemia is a form of hemolytic anemia that results when free cholesterol binds to the red blood cell's membrane increasing its surface area, causing later deformities such as rough or thorny projections on the erythrocyte named acanthocytes. This condition is caused by the deceased liver's decreased ability to esterificate cholesterol.[58] Thalassemia D56. (http://apps.who.int/classifications/apps/icd/icd10online/? gd55.htm+d56) D55.2 30116 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb30116.htm) gd55.htm+d552) D013789

Triosephosphate isomerase deficiency

Triosephosphate isomerase (TPI) deficiency is a genetically inherited autosomal recessive condition "characterized bychronic hemolytic anemia, cardiomyopathy, susceptibility to infections, severe neurological dysfunction, and, in most cases, death in early childhood." [59] Warm autoimmune hemolytic anemia D59.1 29723 (http://apps.who.int/classifications/apps/icd/icd10online/? (http://www.diseasesdatabase.com/ddb29723.htm) gd55.htm+d591)

Warm autoimmune hemolytic anemia is an autoimmune hemolytic anemia (AIHA) characterized by formation of antibodies that attack the body's own red blood cells in a destructive immune system response.[60]

Blood cancers
A blood cancer or hematological malignancy is a type of malignant cancer that originates, affects, or involves the blood, bone marrow, or lymph nodes.[61] These cancers include leukemias, lymphomas, and myelomas. These particular types of cancers can arise as defected mature cell types that have differentiated from hematopoietic precursor cells (often in the bone marrow) and begin to quickly proliferate through the bloodstream where it can then often infiltrate other organs and tissues. Others can involve the formation of tumors from lymphoblasts from within the lymphoid tissue.[62] Incidences of affected people with a form of blood cancer has been steady increasing over recent years; however, due in part to early detection methods and subsequent advancements in the treatment of the diseases, mortality rates have continued to decrease.[63]

Lymphoma

Seven-year-old Nigerian boy with Burkitt's lymphoma presenting with a severely ulcerated and swollen jaw.

Condition name

ICD-10 coding number

Diseases Database coding number

Medical Subject Headings

C81

Hodgkin lymphoma

C81. (http://apps.who.int/classifications/apps/icd/icd10online/? 5973 gc81.htm+c81) C82. (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c82) -C85. 9065 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c85)

Non-Hodgkin lymphoma

The non-Hodgkin lymphomas (NHLs) are a diverse group of blood cancers that include any kind of lymphoma except Hodgkin's lymphomas.[64] Anaplastic large cell lymphoma C84.4 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c844)

Angioimmunoblastic T-cell lymphoma

Angioimmunoblastic T-cell lymphoma (AILT) (also known as Angioimmunoblastic lymphadenopathy with dysproteinemia) [65]:747) is a mature T-cell lymphoma with systemic characterized by a polymorphous lymph node infiltrate showing a marked increase in follicular dendritic cells (FDCs) and high endothelial venules (HEVs) and systemic involvement.[66] It is also known as immunoblastic lymphadenopathy (Lukes-Collins Classification) and AILD-type (lymphogranulomatosis X) T-cell lymphoma (Kiel Classification) [66] Hepatosplenic T-cell lymphoma Hepatosplenic T-cell lymphoma is a systemic neoplasm comprising medium-sized cytotoxic T-cells that show a significant sinusoidal infiltration in the liver, spleen, and bone marrow.[67] It is a rare and generally incurable form of lymphoma.[68] B-cell lymphoma reticuloendotheliosis reticulosis Microglioma Diffuse large B-cell lymphoma C82. (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c82)

Follicular lymphoma

Follicular Lymphoma also known as (indolent follicular lymphoma) is a type of non-hodgkin's lymphoma that involves both large and small B-cell lymphocytes that spreads from the lymphatic system and into the blood, bone marrow, and internal organs. Approximately 20 to 30 percent of non-hodgkin's lymphomas are diagnosed as follicular lymphoma, with a majority of cases involving those 60 years of age or older.[69] Mucosa-Associated Lymphatic Tissue lymphoma 31339

Mucosa-Associated Lymphatic Tissue lymphoma (as known as MALT lymphoma and extra-nodal marginal zone lymphoma) is a condition in which lymphatic tissue abnormally presents outside the lymphatic system (extra-nodular) and instead within the mucosa of the gastrointestinal tract, typically as a lesion in the stomach.[70] B-cell chronic lymphocytic leukemia C91.1 (http://apps.who.int/classifications/apps/icd/icd10online/? 2641 gc81.htm+c911)

B-cell chronic lymphocytic leukemia (also known as small cell lymphocytic lymphoma) is a blood cancer that involves the B-cell lymphocytes; responsible for the creation of antibodies. Of the two general types of chronic lymphocytic leukemias (the other involving T-cells), B-cell chronic lymphocytic leukemia accounts for approximately 95 percent of the diagnoses.[71] Mantle cell lymphoma C85.7 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c857)

Mantle cell lymphoma (MCL) is a type of B-cell lymphoma an and one of the rarest forms of non-Hodgkin's lymphomas comprising approximately 6% of diagnosed cases.[72] C83.7 (http://apps.who.int/classifications/apps/icd/icd10online/? 1784 gc81.htm+c837)

Burkitt lymphoma

Burkitt lymphoma (also known as Burkitt's tumor, Burkitt lymphoma or malignant lymphoma, Burkitt's type) is a type of B-cell lymphoma that is categorized into one of variant types. These are variants are endemic (occurring in equatorial Africa), sporadic ("non-African"), and immunodeficiency-associated (usually associated with HIV). Mediastinal large B cell lymphoma C88.0 (http://apps.who.int/classifications/apps/icd/icd10online/? 14030 gc81.htm+c880)

Waldenstrm's macroglobulinemia

Waldenstrm's macroglob line (also kno n as l mphoplasmac tic l mphoma) is a l mphoproliferati e disease that in ol es an abnormal increase of l mphoc tes ithin the bone

Waldenstrm's macroglobuline (also known as lymphoplasmacytic lymphoma) is a lymphoproliferative disease that involves an abnormal increase of lymphocytes within the bone marrow, creasing disruption of normal red blood cell production.[73] Nodal marginal zone B cell lymphoma Splenic marginal zone lymphoma Splenic marginal zone lymphoma (SMZL) (also known as well-differentiated lymphocytic lymphoma, small lymphocytic lymphoma, and splenic lymphoma with circulating villous lymphocytes) is a lymphoma made up of small B-cells that replace the normal architecture of the white pulp of the spleen. The neoplastic cells are both small lymphocytes and larger, transformed blasts, and they invade the mantle zone of splenic follicles and erode the marginal zone, ultimately invading the red pulp of the spleen. Frequently, the bone marrow and splenic hilar lymph nodes are involved along with the peripheral blood.[74] Intravascular large B-cell lymphoma Primary effusion lymphoma Primary effusion lymphoma is a condition caused Lymphomatoid granulomatosis Nodular lymphocyte predominant Hodgkin's lymphoma by Kaposi's sarcoma-associated herpesvirus (KSHV) [75] 33904

Leukemia
Diseases Database coding number Medical Subject Headings D007952

Condition name

ICD-10 coding number C90.1 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c901)

plasma cell leukemia

Plasma cell leukemia (PCL), a lymphoproliferative disorder,[76] is a rare cancer involving a subtype of white blood cells called plasma cells.[77] Acute erythraemia and erythroleukaemia Acute erythremic myelosis C94.0 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c940)

Acute erythroid leukemia

Acute erythroid leukemia (also known as Guglielmo's disease, and acute Di Guglielmo syndrome) is a rare form of acute myeloid leukemia. Heilmeyer-Schner disease Acute megakaryoblastic leukemia D007947

Acute megakaryoblastic leukemia (AMKL) is a form of acute myeloid leukemia where megakaryoblasts account for approximately 30% of the nucleated cells within the bone marrow.[78] Acute megakaryoblastic leukemia Mast cell leukemia C94.4 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c944) C94.4 (http://apps.who.int/classifications/apps/icd/icd10online/? gc81.htm+c944)

Panmyelosis

Acute panmyelosis with myelofibrosis

Acute panmyelosis with myelofibrosis (APMF) is a poorly-defined disorder that arises as either a clonal disorder, or following toxic exposure to the bone marrow. Lymphosarcoma cell leukemia Acute leukaemia of unspecified cell type Blastic phase chronic myelogenous leukemia Blastic phase chronic myelogenous leukemia is a phase of chronic myelogenous leukemia in which more than 30% of the cells in the blood or bone marrow are blast cells. Stem cell leukemia Chronic leukaemia of unspecified cell type

Subacute leukaemia of unspecified cell type Accelerated phase chronic myelogenous leukemia C92.0 (http://apps.who.int/classifications/apps/icd/icd10online/? 203 gc81.htm+c920)

Acute myeloid leukemia

Acute myeloid leukemia (AML), also known as acute myelogenous leukemia, is a cancer of the myeloid line of blood cells, characterized by the rapid growth of abnormal white blood cells that accumulate in the bone marrow and interfere with the production of normal blood cells.[79] Acute promyelocytic leukemia Acute basophilic leukemia Acute eosinophilic leukemia Acute lymphoblastic leukemia Acute monocytic leukemia Acute myeloblastic leukemia with maturation Acute myeloid dendritic cell leukemia Adult T-cell leukemia/lymphoma Aggressive NK-cell leukemia B-cell prolymphocytic leukemia B-cell chronic lymphocytic leukemia B-cell leukemia Chronic myelogenous leukemia Chronic myelomonocytic leukemia Chronic neutrophilic leukemia Hairy cell leukemia

Myeloma
Condition name Multiple myeloma Kahler's disease Myelomatosis solitary myeloma Plasma cell leukaemia Plasmacytoma, extramedullary Malignant plasma cell tumour NOS Plasmacytoma NOS Solitary myeloma ICD-10 coding number Diseases Database coding number Medical Subject Headings

Malignant immunoproliferative diseases


Di M di l

Condition name Monoclonal gammopathy Angiocentric immunoproliferative lesion Lymphoid granulomatosis Angioimmunoblastic lymphadenopathy T-gamma lymphoproliferative disease Waldenstrm's macroglobulinaemia Alpha heavy chain disease Gamma heavy chain disease Franklin's disease Immunoproliferative small intestinal disease Mediterranean disease Malignant immunoproliferative disease, unspecified Immunoproliferative disease NOS

ICD-10 coding number

Diseases Database coding number

Medical Subject Headings

Coagulation, purpura, and other hemorrhagic conditions


Condition name Disseminated intravascular coagulation (DIC, Defibrination syndrome) Protein C deficiency Protein C deficiency is a rare genetic trait that predisposes to thrombotic disease.[80] Protein S deficiency Thrombocytosis D69.3 (http://apps.who.int/classifications/apps/icd/icd10online/? 6673 gd65.htm+d693) ICD-10 coding number Diseases Database coding number Medical Subject Headings

Idiopathic thrombocytopenic purpura

D016553

Idiopathic thrombocytopenic purpura (ITP) is the condition of having a low platelet count (thrombocytopenia) of no known cause (idiopathic). Recurrent thrombosis Hemophilia Hemophilia A Hemophilia B Hemophilia C Von Willebrand disease Antiphospholipid syndrome Thrombocytopenia Glanzmann's thrombasthenia

Wiskott Aldrich syndrome

Infection-related
Hematological disorders may be caused by a number of infection-related conditions involving the introduction of microorganisms into the host, such as bacteria and protozoa.[38]

Bacterium-related
Condition name Clostridium infection Cholera infection E. coli 0157:H7 infection Typhoid fever ICD-10 coding number Diseases Database coding number Medical Subject Headings

Protozoan-related
Condition name Leishmania infection Malaria infection (Plasmodium infection) toxoplasmosis ICD-10 coding number Diseases Database coding number * Medical Subject Headings

Immune system regulation-related


Immunodeficiency with predominantly antibody defects
Condition name Hereditary hypogammaglobulinemia Nonfamilial hypogammaglobulinemia Selective deficiency of immunoglobulin A [IgA] Selective deficiency of immunoglobulin G [IgG] subclasses Selective deficiency of immunoglobulin M [IgM] Immunodeficiency with increased immunoglobulin M [IgM] Antibody deficiency with near-normal immunoglobulins or with hyperimmunoglobulinemia Transient hypogammaglobulinemia of infancy ICD-10 coding number Diseases Database coding number Medical Subject Headings

References
^ Saladin, Kenneth S.; Miller, Leslie (2004). "18". Anatomy & Physiology: The Unity of form and Function (3rd ed.). Mc Graw-Hill. pp. 679. ISBN 0-07-242903-8. ^ a b MedicineNet.com --> Definition of Anemia (http://www.medterms.com/script/main/art.asp?articlekey=15491) Last Editorial Review: 12/9/2000 Retrieved March 27, 2011 ^ merriam-webster dictionary --> anemia (http://www.merriam-webster.com/dictionary/anemia) Retrieved on March 27, 2011 ^ "Anemia or Iron Deficiency" (http://www.cdc.gov/nchs/fastats/anemia.htm) . Centers for Disease Control and Prevention. Last reviewed April 15, 2010. http://www.cdc.gov/nchs/fastats/anemia.htm. Retrieved 28 March 2011. 5. ^ Brady PG (2007). "Iron deficiency anemia: a call for" (http://meta.wkhealth.com/pt/pt-core/template-journal/lwwgateway/media/landingpage.htm? doi=10.1097/SMJ.0b013e3181520699) . South. Med. J. 100 (10): 9667. doi:10.1097/SMJ.0b013e3181520699 (inactive 2010-01-09). PMID 17943034 (http://www.ncbi.nlm.nih.gov/pubmed/17943034) . http://meta.wkhealth.com/pt/pt-core/template-journal/lwwgateway/media/landingpage.htm?doi=10.1097/SMJ.0b013e3181520699. 6. ^ Novacek G (2006). "Plummer-Vinson syndrome" (http://www.ojrd.com/content/1//36) . Orphanet J Rare Dis 1: 36. doi:10.1186/1750-1172-1-36 (http://dx.doi.org/10.1186%2F1750-1172-1-36) . PMC 1586011 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=1586011) . PMID 16978405 (http://www.ncbi.nlm.nih.gov/pubmed/16978405) . http://www.ojrd.com/content/1//36. 7. ^ Mayo Clinic staff (March 4, 2011). "Vitamin deficiency anemia" (http://www.mayoclinic.com/health/vitamin-deficiency-anemia/DS00325) . Mayo Foundation for Medical Education 1. 2. 3. 4.

8. 9. 10. 11.

12.

13. 14. 15. 16. 17. 18. 19. 20. 21. 22. 23.

24. 25. 26. 27. 28. 29. 30.

31. 32. 33. 34. 35. 36.

37. 38. 39. 40. 41.

42. 43. 44.

45.

y ( ) y ( p y y ) y f and Research. Mayo Clinic. http://www.mayoclinic.com/health/vitamin-deficiency-anemia/DS00325. Retrieved 5 March 2011. ^ "Pernicious anemia" (http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001595/) . National Center for Biotechnology Information (NCBI). U.S. National Library of Medicine. November 23, 2008. http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001595/. Retrieved 1 March 2011. ^ Grsbeck, R. (May 19, 2006). Imerslund-Grsbeck syndrome (selective vitamin B(12) malabsorption with proteinuria). National Center for Biotechnology Information, U.S. National Library of Medicine. PMID 16722557 (http://www.ncbi.nlm.nih.gov/pubmed/16722557) . ^ a b c "Megaloblastic Anemia: Overview - eMedicine Hematology" (http://emedicine.medscape.com/article/204066-overview) . http://emedicine.medscape.com/article/204066overview. Retrieved 2009-02-07. ^ Kaikov, Y.; L. D. Wadsworth, C. A. Hall and P. C. J. Rogers (1991). "Transcobalamin II deficiency: Case report and review of the literature" (http://www.springerlink.com/content/u31t710158288166/) . European Journal of Pediatrics. European Journal of Pediatrics. pp. Volume 150, Number 12, 841843, DOI: 10.1007/BF01955004. http://www.springerlink.com/content/u31t710158288166/. Retrieved 2 March 2011. ^ Hall, C. A. (November, 1975). Transcobalamins I and II as natural transport proteins of vitamin B12. (http://www.pubmedcentral.nih.gov/articlerender.fcgi? tool=pmcentrez&artid=301974) . The Journal of Clinical Investigation. pp. 56(5): 11251131. doi: 10.1172/JCI108187. PMC 301974 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=301974) . http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=301974. ^ "Folate-deficiency anemia" (http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001578/) . National Center for Biotechnology Information. U.S. National Library of Medicine. reviewed January 31, 2010. http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001578/. Retrieved 2 March 2011. ^ Shahidi, Nasrollah T.; Diamond, Louis K; Shwachman, Harry (October 1961). "Anemia associated with protein deficiency" (http://www.jpeds.com/article/S00223476%2861%2980237-0/abstract) . The Journal of Peditrics. Elsevier Inc. http://www.jpeds.com/article/S0022-3476%2861%2980237-0/abstract. Retrieved 28 March 2011. ^ Vorvick, MD, Linda (Updated March 14, 2009). "Scurvy" (http://www.nlm.nih.gov/medlineplus/ency/article/000355.htm) . Medline Plus. U.S. National Library of Medicine. http://www.nlm.nih.gov/medlineplus/ency/article/000355.htm. Retrieved 1 March 2011. ^ Chambers, Diana; Zieve, David Zieve (Last reviewed August 11, 2009). "Bassen-Kornzweig syndrome" (http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0002631/) . Pub Med Health. National Center for Biotechnology Information, U.S. National Library of Medicine. http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0002631/. Retrieved 23 March 2011. ^ Turgeon, Mary (2005). "8". Clinical Hematology: Theory and Procedures (4th ed.). Philadelphia: Lippincott Williams & Wilkins. pp. 117. ISBN 0-7817-5007-5. ^ Online 'Mendelian Inheritance in Man' (OMIM) 141800 (http://omim.org/entry/141800) ^ Online 'Mendelian Inheritance in Man' (OMIM) 141850 (http://omim.org/entry/141850) ^ "Alpha thalassemia" (http://ghr.nlm.nih.gov/condition/alpha-thalassemia) . Genetics Home Reference. U.S. National Library of Medicine. February 27, 2011. http://ghr.nlm.nih.gov/condition/alpha-thalassemia. Retrieved 3 March 2011. ^ "Anemia of Chronic Disease" (http://www.irondisorders.org/anemia-of-chronic-disease) . Iron Disorders Institute. 2009. http://www.irondisorders.org/anemia-of-chronic-disease. Retrieved 3 March 2011. ^ "Anemia in Kidney Disease and Dialysis" (http://kidney.niddk.nih.gov/kudiseases/pubs/anemia/) . The National Kidney and Urologic Diseases Information Clearinghouse. National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health. October 2008. http://kidney.niddk.nih.gov/kudiseases/pubs/anemia/. Retrieved 28 March 2011. ^ Widness JA (November 2008). "Pathophysiology of Anemia During the Neonatal Period, Including Anemia of Prematurity" (http://www.pubmedcentral.nih.gov/articlerender.fcgi? tool=pmcentrez&artid=2867612) . Neoreviews 9 (11): e520. doi:10.1542/neo.9-11-e520 (http://dx.doi.org/10.1542%2Fneo.9-11-e520) . PMC 2867612 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=2867612) . PMID 20463861 (http://www.ncbi.nlm.nih.gov/pubmed/20463861) . http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=2867612. ^ "Anemia of prematurity" (http://www.uptodate.com/patients/content/topic.do?topicKey=~4ppbbmoywE7y17P) . http://www.uptodate.com/patients/content/topic.do? topicKey=~4ppbbmoywE7y17P. Retrieved 2010-05-31. ^ Aplastic anemia (http://www.mountsinai.org/Patient%20Care/Service%20Areas/Cancer/Diseases%20and%20Conditions?citype=Disease&ciid=Aplastic%20anemia) at Mount Sinai Hospital ^ [1] (http://www.mountsinai.org/patient-care/health-library/diseases-and-conditions/autoimmune-hemolytic-anemia) at Mount Sinai Hospital ^ "Hemolytic Anemia: Overview - eMedicine Hematology" (http://emedicine.medscape.com/article/201066-overview) . http://emedicine.medscape.com/article/201066-overview. Retrieved 2009-02-07. ^ "Beta thalassemia" (http://ghr.nlm.nih.gov/condition/beta-thalassemia) . Genetics Home Reference. U.S. National Library of Medicine, NIH. March 13, 2011. http://ghr.nlm.nih.gov/condition/beta-thalassemia. Retrieved 21 March 2011. ^ Tchernia, Gilbert; Delauney, J (2000-06). "DiamondBlackfan anemia" (http://www.orpha.net/data/patho/Pro/en/BlackfanDiamond-FRenPro429.pdf) . Orpha.net. http://www.orpha.net/data/patho/Pro/en/BlackfanDiamond-FRenPro429.pdf. Retrieved 1 January 2010. ^ Cmejla R, Cmejlova J, Handrkova H, et al. (February 2009). "Identification of mutations in the ribosomal protein L5 (RPL5) and ribosomal protein L11 (RPL11) genes in Czech patients with DiamondBlackfan anemia". Hum. Mutat. 30 (3): n/a. doi:10.1002/humu.20874 (http://dx.doi.org/10.1002%2Fhumu.20874) . PMID 19191325 (http://www.ncbi.nlm.nih.gov/pubmed/19191325) . ^ "Congenital dyserythropoietic anemia" (http://ghr.nlm.nih.gov/condition/congenital-dyserythropoietic-anemia) . Genetics Home Reference. U.S. National Library of Medicine, National Institutes of Health. Reviewed July 2009. http://ghr.nlm.nih.gov/condition/congenital-dyserythropoietic-anemia. Retrieved 22 March 2011. ^ Dhaliwal, Gurpreet; Cornett, Patricia, Tierney, Lawrence (2004). "Hemolytic Anemia" (http://www.aafp.org/afp/2004/0601/p2599.html) . American Family Physician. 2004 Jun 1;69(11):2599-2607. American Family of Family Physicians. http://www.aafp.org/afp/2004/0601/p2599.html. Retrieved 28 March 2011. ^ "hemoglobinopathy (http://www.mercksource.com/pp/us/cns/cns_hl_dorlands_split.jsp?pg=/ppdocs/us/common/dorlands/dorland/four/000048231.htm) " at Dorland's Medical Dictionary ^ Weatherall DJ, Clegg JB. Inherited haemoglobin disorders: an increasing global health problem. Bull World Health Organ. 2001;79(8):704-712. ^ Hemoglobinopathies and Thalassemias (http://web2.airmail.net/uthman/hemoglobinopathy/hemoglobinopathy.html) ^ "Sickle Cell Trait and Other Hemoglobinopathies and Diabetes: Important Information for Physicians" (http://diabetes.niddk.nih.gov/dm/pubs/hemovari-A1C/index.htm) . National Diabetes Information Clearinghouse. National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health. November 2008. http://diabetes.niddk.nih.gov/dm/pubs/hemovari-A1C/index.htm. Retrieved 3 March 2011. ^ Reviewed by Vorvick, Linda J.; Chen, Yi-Bin (Reviewed January 31, 2010). "Hemolytic anemia" (http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001597/) . Pub Med Health. National Center for Biotechnology Information, U.S. National Library of Medicine. http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001597/. Retrieved 24 March 2011. ^ a b Turgeon, Mary Louise (2004). "12". Clinical Hematology (4th ed.). Lippincott Williams & Wilkins. p. 161. ISBN 0-7817-5007-5. ^ Cotran, Ramzi S.; Kumar, Vinay; Fausto, Nelson; Nelso Fausto; Robbins, Stanley L.; Abbas, Abul K. (2005). Robbins and Cotran pathologic basis of disease. St. Louis, Mo: Elsevier Saunders. pp. 625. ISBN 0-7216-0187-1. ^ Tavares, Simone; Sills, Richard (Last updated January 3, 2011). "Pediatric Hereditary Elliptocytosis and Related Disorders" (http://emedicine.medscape.com/article/955827-overview) . eMedicine. http://emedicine.medscape.com/article/955827-overview. Retrieved 28 March 2011. ^ Yawata, Yoshihito (2003). Cell membrane: the red blood cell as a model (http://books.google.com/books? id=_HFyKIAN2p8C&pg=PA227&lpg=PA227&dq=Hereditary+pyropoikilocytosis&source=vrt&ots=115KXLCnCa&sig=Xgdw21VBHTRInD8CwkTkzWaTdl4&hl=en&ei=gmeiTaejH8 OftwepuayQAw&sa=X&oi=book_result&ct=result&resnum=11&ved=0CGMQ6AEwCg#v=onepage&q=Hereditary%20pyropoikilocytosis&f=false) . Wiley-VCH. p. 227. http://books.google.com/books? id=_HFyKIAN2p8C&pg=PA227&lpg=PA227&dq=Hereditary+pyropoikilocytosis&source=vrt&ots=115KXLCnCa&sig=Xgdw21VBHTRInD8CwkTkzWaTdl4&hl=en&ei=gmeiTaejH8 OftwepuayQAw&sa=X&oi=book_result&ct=result&resnum=11&ved=0CGMQ6AEwCg#v=onepage&q=Hereditary%20pyropoikilocytosis&f=false. Retrieved 11 April 2011. ^ "Cold Agglutinin Disease: Overview - eMedicine Pediatrics: General Medicine" (http://emedicine.medscape.com/article/954954-overview) . http://emedicine.medscape.com/article/954954-overview. Retrieved 2009-02-07. ^ "Paroxysmal cold hemoglobinuria (PCH) e" (http://www.nlm.nih.gov/medlineplus/ency/article/000557.htm) . Medline Plus. U.S. Department of Health and Human Services, National Institutes of Health. Last updated March 28, 2010. http://www.nlm.nih.gov/medlineplus/ency/article/000557.htm. Retrieved 28 March 2011. ^ "Hemolytic Uremic Syndrome in Children" (http://kidney.niddk.nih.gov/kudiseases/pubs/childkidneydiseases/hemolytic_uremic_syndrome/) . National Kidney and Urologic Diseases Information Clearinghouse. National Institutes of Health of the U.S. Department of Health and Human Services. January 2009. http://kidney.niddk.nih.gov/kudiseases/pubs/childkidneydiseases/hemolytic_uremic_syndrome/. Retrieved 4 March 2011. ^ Rijksen, G; Staal, G E. (August 1978). "Human erythrocyte hexokinase deficiency. Characterization of a mutant enzyme with abnormal regulatory properties." (http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=371766) . The Journal of Clinical Investigation (National Center for Biotechnology Information, U.S. National Library of Medicine) PMC 371766 (http://www pubmedcentral gov/articlerender fcgi?tool=pmcentrez&artid=371766) http://www pubmedcentral nih gov/articlerender fcgi?

46.

47. 48. 49. 50. 51. 52. 53. 54.

55.

56. 57. 58. 59.

60. 61. 62. 63. 64. 65. 66. 67.

68.

69.

70.

National Library of Medicine). PMC 371766 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=371766) . http://www.pubmedcentral.nih.gov/articlerender.fcgi? tool=pmcentrez&artid=371766. ^ Wilson, William C.; Grande, Christopher M., Hoyt, David B. (2007). "Trauma: Critical Care" (http://books.google.com/books? id=3H3AIEtvc8YC&pg=PA962&lpg=PA962&dq=Hypochromic+anemia+pale+cells&source=bl&ots=p4J4yC2eRe&sig=mhgZnqQ65fbr25e7y2bbt0JD98A&hl=en&ei=T0GVTabbJom9 0QGotanxCw&sa=X&oi=book_result&ct=result&resnum=7&ved=0CEAQ6AEwBg#v=onepage&q=Hypochromic%20anemia%20pale%20cells&f=false) . Google books. CRC press. p. 962. http://books.google.com/books? id=3H3AIEtvc8YC&pg=PA962&lpg=PA962&dq=Hypochromic+anemia+pale+cells&source=bl&ots=p4J4yC2eRe&sig=mhgZnqQ65fbr25e7y2bbt0JD98A&hl=en&ei=T0GVTabbJom9 0QGotanxCw&sa=X&oi=book_result&ct=result&resnum=7&ved=0CEAQ6AEwBg#v=onepage&q=Hypochromic%20anemia%20pale%20cells&f=false. Retrieved 1 April 2011. ^ Rivella, S. (May 2009). Ineffective erythropoiesis and thalassemias. National Center for Biotechnology Information, U.S. National Library of Medicine. PMID 19318943 (http://www.ncbi.nlm.nih.gov/pubmed/19318943) . ^ "Ineffective Erythropoiesis" (http://www.nejm.org/doi/full/10.1056/NEJM196209132671113) . The New England Journal of Medicine. September 13, 1962. http://www.nejm.org/doi/full/10.1056/NEJM196209132671113. Retrieved 22 March 2011. ^ "Hematopathology" (http://library.med.utah.edu/WebPath/HEMEHTML/HEME030.html) . http://library.med.utah.edu/WebPath/HEMEHTML/HEME030.html. ^ "NINDS Neuroacanthocytosis Information Page" (http://www.ninds.nih.gov/disorders/neuroacanthocytosis/neuroacanthocytosis.htm) . National Institute of Neurological Disorders and Stroke. National Institutes of Health. Last updated March 16, 2009. http://www.ninds.nih.gov/disorders/neuroacanthocytosis/neuroacanthocytosis.htm. Retrieved 11 April 2011. ^ "CHOREOACANTHOCYTOSIS; CHAC" (http://www.omim.org/entry/200150?search=Chorea%20acanthocytosis) . Online Mendelian Inheritance in Man. Johns Hopkins University. http://www.omim.org/entry/200150?search=Chorea%20acanthocytosis. Retrieved 11 April 2011. ^ "Chorea-acanthocytosis" (http://ghr.nlm.nih.gov/condition/chorea-acanthocytosis) . Genetic Home Refrense. U.S. National Library of Medicine, National Institutes of Health. May 2008. http://ghr.nlm.nih.gov/condition/chorea-acanthocytosis. Retrieved 11 April 2011. ^ Online 'Mendelian Inheritance in Man' (OMIM) 266200 (http://omim.org/entry/266200) ^ Cherif-Zahar, B.; Raynal, V., Le Van Kim, C, D'Ambrosio, AM, Bailly, P., Cartron, JP and Colin, Y. (April 6, 2011). "Structure and expression of the RH locus in the Rh-deficiency syndrome" (http://bloodjournal.hematologylibrary.org/content/82/2/656.full.pdf) . Blood. Blood Journal. http://bloodjournal.hematologylibrary.org/content/82/2/656.full.pdf. Retrieved 7 April 2011. ^ Caudill JS, Imran H, Porcher JC, Steensma DP (October 2008). "Congenital sideroblastic anemia associated with germline polymorphisms reducing expression of FECH" (http://www.haematologica.org/cgi/pmidlookup?view=long&pmid=18698088) . Haematologica 93 (10): 15824. doi:10.3324/haematol.12597 (http://dx.doi.org/10.3324%2Fhaematol.12597) . PMID 18698088 (http://www.ncbi.nlm.nih.gov/pubmed/18698088) . http://www.haematologica.org/cgi/pmidlookup? view=long&pmid=18698088. ^ Sideroblastic Anemias: Anemias Caused by Deficient Erythropoiesis (http://www.merck.com/mmpe/sec11/ch130/ch130c.html) at Merck Manual of Diagnosis and Therapy Professional Edition ^ "Prevention of cerebral malaria in children in Papua New Guinea by southeast Asian ovalocytosis band 3 -- Allen et al. 60 (6): 1056 -- American Journal of Tropical Medicine and Hygiene" (http://www.ajtmh.org/cgi/reprint/60/6/1056) . http://www.ajtmh.org/cgi/reprint/60/6/1056. Retrieved 2008-02-26. ^ "Spur-cell hemolytic anemia in severe alcoholic cirrhosis" (http://bloodjournal.hematologylibrary.org/content/101/1/3.full) . Journal of the American Society of Hematology. The American Society of Hematology. January 1, 2003. http://bloodjournal.hematologylibrary.org/content/101/1/3.full. Retrieved 10 April 2011. ^ Ralser, Markus; Heeren, Gino; Breitenbach. Michael; Lehrach, Hans; and Krobitsch, Sylvia (December 20, 2006). "Triose Phosphate Isomerase Deficiency Is Caused by Altered DimerizationNot Catalytic Inactivityof the Mutant Enzymes" (http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=1762313) . PLoS ONE (National Center for Biotechnology Information, U.S. National Library of Medicine). PMC 1762313 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=1762313) . http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=1762313. ^ "Autoimmune Hemolytic Anemia" (http://www.merckmanuals.com/home/sec14/ch172/ch172f.html) . The Merck Manuals: Online Medical Library. Merck Sharp and Dohme. http://www.merckmanuals.com/home/sec14/ch172/ch172f.html. Retrieved 25 March 2011. ^ "Hematologic (Blood) Cancers" (http://www.cdc.gov/cancer/hematologic/) . Centers for Disease Control and Prevention. http://www.cdc.gov/cancer/hematologic/. Retrieved 25 January 2011. ^ McPhee, Stephen; Ganong, William (2003). "Chapter 5: Neoplasia". Pathophysiology of Disease (5th ed.). New York: Lange Medical Books/McGraw-Hill. pp. 106 110. ISBN 0-07144159-X. ^ "Blood Cancers: Leukemia, Lymphoma, and Myeloma" (http://www.cdc.gov/Features/HematologicCancers/) . Centers for Disease Control and Prevention. http://www.cdc.gov/Features/HematologicCancers/. Retrieved 5 April 2011. ^ "non-Hodgkin lymphomas (http://www.mercksource.com/pp/us/cns/cns_hl_dorlands_split.jsp?pg=/ppdocs/us/common/dorlands/dorland/nine/000954456.htm) " at Dorland's Medical Dictionary ^ James, William D.; Berger, Timothy G.; et al. (2006). Andrews' Diseases of the Skin: clinical Dermatology. Saunders Elsevier. ISBN 0-7216-2921-0. ^ a b [2] (http://www.iarc.fr/WHO-BlueBooks/BBwebsite/bb3.html) Jaffe E.S., Harris N.L., Stein H., Vardiman J.W. (eds): World Health Organization Classification of Tumors. Pathology and Genetics of Tumours of Haemopoietic and Lymphoid Tissues. IARC Press: Lyon 2001 ^ Elaine Sarkin Jaffe, Nancy Lee Harris, World Health Organization, International Agency for Research on Cancer, Harald Stein, J.W. Vardiman (2001). Pathology and genetics of tumours of haematopoietic and lymphoid tissues (http://books.google.com/?id=XSKqcy7TUZUC) . World Health Organization Classification of Tumors. 3. Lyon: IARC Press. ISBN 92-832-2411-6. http://books.google.com/?id=XSKqcy7TUZUC. ^ Mackey AC, Green L, Liang LC, Dinndorf P, Avigan M (February 2007). "Hepatosplenic T cell lymphoma associated with infliximab use in young patients treated for inflammatory bowel disease" (http://meta.wkhealth.com/pt/pt-core/template-journal/lwwgateway/media/landingpage.htm?an=00005176-200702000-00019) . J. Pediatr. Gastroenterol. Nutr. 44 (2): 2657. doi:10.1097/MPG.0b013e31802f6424 (http://dx.doi.org/10.1097%2FMPG.0b013e31802f6424) . PMID 17255842 (http://www.ncbi.nlm.nih.gov/pubmed/17255842) . http://meta.wkhealth.com/pt/pt-core/template-journal/lwwgateway/media/landingpage.htm?an=00005176-200702000-00019. ^ OConnor,, Owen A.; Vose, Julie M. (March 2008). "Getting The Facts: Indolent Follicular Lymphoma" (http://www.lymphoma.org/atf/cf/%7B0363cdd6-51b5-427b-be48e6af871acec9%7D/INDOLENT%20FOLLICULAR.PDF) . Lymphoma Research Foundation. http://www.lymphoma.org/atf/cf/%7B0363cdd6-51b5-427b-be48e6af871acec9%7D/INDOLENT%20FOLLICULAR.PDF. Retrieved 2 April 2011. ^ Storey, Rowland; Gatt, Marcell, Bradford, Ian (January 6, 2009). [; "Mucosa associated lymphoid tissue lymphoma presenting within a solitary anti-mesenteric dilated segment of ileum: a case report"]. Journal of Medical Case Reports. BioMed Central. Archived from the original (http://jmedicalcasereports.com/content/3/1/6) . Error: If you specify |archiveurl=, you must also specify |archivedate=. . Retrieved 2 April 2011.

71. ^ "Leukemia - Chronic Lymphocytic - CLL" (http://www.cancer.net/patient/Cancer+Types/Leukemia+-+Chronic+Lymphocytic+-+CLL) . Cancer.Net. American Society of Clinical Oncology (ASCO). Last Updated: December 02, 2010. http://www.cancer.net/patient/Cancer+Types/Leukemia+-+Chronic+Lymphocytic+-+CLL. Retrieved 2 April 2011. 72. ^ Turgeon, Mary Louise (2005). Clinical hematology: theory and procedures. Hagerstown, MD: Lippincott Williams & Wilkins. pp. 283. ISBN 0-7817-5007-5. 73. ^ "Waldenstrm Macroglobulinemia" (http://www.mayoclinic.org/waldenstroms-macroglobulinemia/) . Mayo Clinic. Mayo Foundation for Medical Education and Research. http://www.mayoclinic.org/waldenstroms-macroglobulinemia/. Retrieved 4 April 2011. 74. ^ Franco,, Vito; Florena, Ada Maria, Iannitto, Emilio (November 21, 2002). "Splenic marginal zone lymphoma" (http://bloodjournal.hematologylibrary.org/content/101/7/2464.full.html) . Journal of the American Society of Hematology. American Society of Hematology. http://bloodjournal.hematologylibrary.org/content/101/7/2464.full.html. Retrieved 4 April 2011. 75. ^ Jones, Dan; Ballestas, Mary E., Kaye, Kenneth M., Gulizia, James M., Winters, Gayle L., Fletcher, Jonathan, Scadden, David T., and Aster, Jon C. (August 13, 1998). "PrimaryEffusion Lymphoma and Kaposi's Sarcoma in a Cardiac-Transplant Recipient" (http://www.nejm.org/doi/full/10.1056/NEJM199808133390705) . The New England Journal of Medicine. Massachusetts Medical Society.. http://www.nejm.org/doi/full/10.1056/NEJM199808133390705. Retrieved 4 April 2011. 76. ^ Chan SM, George T, Cherry AM, Medeiros BC (February 2009). "Complete remission of primary plasma cell leukemia with bortezomib, doxorubicin, and dexamethasone: a case report" (http://www.casesjournal.com/content/2/1/121) . Cases J 2 (1): 121. doi:10.1186/1757-1626-2-121 (http://dx.doi.org/10.1186%2F1757-1626-2-121) . PMC 2644294 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=2644294) . PMID 19192311 (http://www.ncbi.nlm.nih.gov/pubmed/19192311) . http://www.casesjournal.com/content/2/1/121. 77. ^ Kim SJ, Kim J, Cho Y, Seo BK, Kim BS (May 2007). "Combination chemotherapy with bortezomib, cyclophosphamide and dexamethasone may be effective for plasma cell leukemia" (http://jjco.oxfordjournals.org/cgi/pmidlookup?view=long&pmid=17538191) . Jpn. J. Clin. Oncol. 37 (5): 3824. doi:10.1093/jjco/hym037 (http://dx doi org/10 1093%2Fjjco%2Fhym037) PMID 17538191 (http://www ncbi nlm nih gov/pubmed/17538191) http://jjco oxfordjournals org/cgi/pmidlookup?

(http://dx.doi.org/10.1093%2Fjjco%2Fhym037) . PMID 17538191 (http://www.ncbi.nlm.nih.gov/pubmed/17538191) . http://jjco.oxfordjournals.org/cgi/pmidlookup? view=long&pmid=17538191. 78. ^ Gassmann W,; Lffler H. (1995). Acute megakaryoblastic leukemia. National Center for Biotechnology Information, U.S. National Library of Medicine. pp. Leukemia and Lymphoma. 18 Suppl 1:6973.. PMID 7496359 (http://www.ncbi.nlm.nih.gov/pubmed/7496359) . 79. ^ Lowenberg, M.D., Bob; Downing M.D., James R., Burnett M.D., Alan (September 30, 1999). "Acute myeloid leukemia" (https://www.evalue.net/ndfiles/EvalueMProd_Collections/155/155Article_Library/Heme_Onc_Required_Readings_1.pdf) . The New England Journal of Medicine. Massachusetts Medical Society. pp. page 1051 Volume 341 Number 14. https://www.e-value.net/ndfiles/EvalueMProd_Collections/155/155Article_Library/Heme_Onc_Required_Readings_1.pdf. Retrieved 8 March 2011. 80. ^ Griffin JH, Evatt B, Zimmerman TS, Kleiss AJ, Wideman C (1981). "Deficiency of protein C in congenital thrombotic disease" (http://www.pubmedcentral.nih.gov/articlerender.fcgi? tool=pmcentrez&artid=370934) . J. Clin. Invest. 68 (5): 13703. doi:10.1172/JCI110385 (http://dx.doi.org/10.1172%2FJCI110385) . PMC 370934 (http://www.pubmedcentral.gov/articlerender.fcgi?tool=pmcentrez&artid=370934) . PMID 6895379 (http://www.ncbi.nlm.nih.gov/pubmed/6895379) . http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=370934. Full text at PMC: 370934 (http://www.ncbi.nlm.nih.gov/pmc/articles/PMC370934/)

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