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Benign Paroxysmal

Positional Vertigo (BPPV)
Amy Flory PT
NAUPT 04/20/09

PERIPHERAL VESTIBULAR
ANATOMY
 Semi-circular canals
– 3 SSC, oriented at right angles to
each other.
– Membranous, filled with
endolymph, surrounded by
perilymph.
– The ampulla contains sensory cells
projecting into the gelatinous
cupula.

.

PERIPHERAL VESTIBULAR ANATOMY  Otolith organ anatomy – Utricle and saccule are oriented at right angles to each other. called otoconia. – The otolithic membrane is embedded with calcium carbonate crystals. or otoliths. – Sensory hair cells contained within the macula project into the otolithic membrane. .

.PERIPHERAL VESTIBULAR ANATOMY  The vestibular nerve is composed of the superior and inferior nerves exiting the vestibular apparatus. – The superior nerve carries information from anterior and lateral SSCs and the otolith organs. – The inferior nerve carries information from the posterior SSC and the saccule.

 The six SSCs are paired such that each pair is sensitive to motion within one plane of 3D space. . AFFERENT PHYSIOLOGY  The sensory receptors of the vestibular nerve are directionally oriented. VEST.PERIPH.  Matched pairs respond to angular acceleration in an “equal- opposite” fashion.

resulting in gaze direction same as endolymph movement. – Enables gaze stabilization on a stationary target during head movement (image stabilizer) . then through MLF to Abducens and Oculomotor nuclei.PERIPH. VEST. AFFERENT PHYSIOLOGY  Vestibulo-ocular reflex (VOR) – Vestibular apparatus afferents synapse in vestibular nuclei.

CENTRAL VESTIBULAR ANATOMY  The vestibular nuclear complex: 4 “major” nuclei and >7 “minor” nuclei.  The nuclei are connected with cerebellum. . reticular formation. spinal cord. and eye movement systems.

descending vest.CENTRAL VESTIBULAR EFFERENT ANATOMY  Medial vestibulospinal tract: originates from medial. superior. nuclei and descends bilaterally to the cervical spinal cord in the MLF  Mediates postural change in response to SSC input (stabilizes head position) .

nucleus and descends to the anterior horn cells in the spinal cord  Mediates postural changes in response to cerebellar and otolithic input (compensates for head tilts and body movements) .CENTRAL VESTIBULAR ANATOMY  Lateral vestibulospinal tract: originates from lateral vest.

LM. British Journal of Hospital Medicine. . Vertigo: new approaches to diagnosis and management.CENTRAL VESTIBULAR ANATOMY  Cervical spinal proprioceptive input – Mostly from facet joints and capsules – Less so from deep paraspinal muscles – None from superficial muscles and skin Luxon.

g.CENTRAL VESTIBULAR PHYSIOLOGY  Varied and widespread vestibular nuc. the postural correction used when the otolithic organs are stimulated is different when the head is forward vs. connections enable postural corrections in different situations (e. when it’s turned to the side) .

Example: head tilt to one side results in extensor activity ipsilaterally and flexor activity contralaterally while in stance.CENTRAL VESTIBULAR PHYSIOLOGY  Vestibulo-spinal reflex – Actually many different reflexes. . dynamic). tonic. depending on state of system (static.

3-10 seconds . visual object to the fovea in shortest possible time – Subject fixates on target 40° from center on command. CENTRAL VESTIBULAR PHYSIOLOGY  Smooth pursuit – Maintains gaze on a moving target – Subject follows one finger in 30-40° arc for 2-3 seconds in each direction  Saccades – Ballistic eye movements to bring a periph.

– Fast phase (central component) is in same direction of perceived head- on-body rotation.CENTRAL VESTIBULAR PHYSIOLOGY  Nystagmus – Slow phase (peripheral component) is in opposite direction of perceived head-on-body rotation. unless <1 week since onset .  Spontaneous nystagmus usually a central finding.

BENIGN PAROXYSMAL POSITIONAL VERTIGO (BPPV)  Cause of 40% of peripheral vestibular cases  Diagnostic criteria – Nystagmus is predictably produced – Paroxysmal nystagmus (immediate to <10 sec onset with provoking position) – Symptoms experienced while nystagmus is present – Fatiguing symptoms and nystagmus – Latency of several seconds – Positionally provoked .

BPPV: ETIOLOGY & PRESENTATION  Cupulolithiasis – Canaliths adhered to the cupula – Immediate onset of symptoms in provoking position – May continue >1 minute – Eases with return to upright .

BPPV: ETIOLOGY & PRESENTATION  Canalithiasis – Canaliths floating freely in the SSC endolymph – Latent onset of symptoms in provoking position – Usually fatigues within 1 minute – Repeats (and reverses) on return to upright .

head 30° below horizon  Hold position up to a minute without symptoms .BPPV: HALLPIKE-DIX TEST  Patient in long-sitting  Head turned to testing side  “Lie back as quickly as you can”  End position: supine.

may not be provoked with H-D test (use supine head turns) . BPPV: VARIANTS  Posterior canal – Most common form – Nystagmus is torsional and geotropic toward involved ear in Hallpike-Dix test  Horizontal/lateral canal – 10% of BPPV cases – Nystagmus is horizontal.

etc. Modified Liberatory.BPPV: TREATMENT  Canalith repositioning – Maneuvers by the therapist in the clinic. BBQ roll. inc. inc. BBQ roll and variants . Epley.. Brandt-Daroff exercises. TID. Semont. – Exercises done by the patient at home.

BPPV: TREATMENT  Cupulolithiasis – Semont maneuver – Other repositioning maneuver with mastoid vibration or oscillation – Brandt-Daroff exercises  Canalithiasis – Canalith repositioning maneuver – Brandt-Daroff exercises .

CANALITH REPOSITIONING MANEUVERS  Goal is to float debris from SSCs and cupula back into utricle  Timing strategy applied to positioning sequence to approximate migration rate of debris  All are based on the Epley maneuver .

affected side down.MODIFIED LIBERATORY MANEUVER (MODIFICATION OF EPLEY MANEUVER)  Patient is quickly moved into H-D position.  Turn head slowly (30-45 sec) to opposite H-D position.  Return slowly to sitting. supported there for 4 minutes. for 30 seconds. . chin slightly tucked.  Patient rolls onto unaffected side. Hold 4 minutes. then rotate head to face forward. nose pointed to floor.

POST-MANEUVER INSTRUCTIONS  For 48 hours following maneuver – Avoid looking up or down – Avoid lying below 30 degrees above horizontal  For 7 days following maneuver – Avoid lying on affected side – Avoid turning head toward affected side while supine .

com 928-556-9935 .corebalancetherapy.THANK YOU! Amy Flory PT CoreBalance Therapy LLC www.