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ABNORMAL LABOR
DYSTOCIA
TUJUAN =
Mahasiswa kedokteran klinik
Mampu menginterpretasikan temuan atau
informasi yang ditampilkan oleh Partograf.
Mampu mengklasifikasikan persalinan normal
dan abnormal berdasarkan temuan dari
partograf.
Mampu menilai penyebab persalinan abnormal.
Mampu menjelaskan keputusan klinik sebagai
upaya penyelesaian persalinan berdasarkan
diagnosis klinis.
Abnormal labour = dystocia
4 jam kemudian =
his 5x dalam 10 menit, lama 50 detik, djj
150x/mnt
penurunan kepala 2/5
periksa dalam vagina =
pembukaan 8 cm, portio edem
kepala turun HIII
moulage sulit dinilai, caput +
Gambaran apa yang
ditunjukkan oleh
partograf?
Kesimpulan apa yang
anda buat
berdasarkan temuan
partograf tersebut ?
Dari kesimpulan yang
anda buat, apakah
persalinan ini
termasuk normal ?
Apakah penyebab dari
kelainan ini ?
Bagaimana jika
dilakukan augmentasi
pada kasus ini ? Apa
resikonya ?
Tindakan apa yang
paling rasional untuk
menyelesaikan
persalinan ini ?
PERSALINAN PATOLOGIS
ABNORMAL LABOR
DYSTOCIA
Abnormal labour = dystocia
PONEK
Progress in labor
POWER PROGRESS
PASSAGES PASSENGER
IN LABOR
Alarm course
Assessment of labor
abnormalities
Hypotonic uterine
dysfunction
Hypertonic uterine
dysfunction
Incoordinate uterine
dysfunction
Assessment of uterine
activity
Ideally = Uterine activity can be quantified by
measurement of intrauterine pressure (IUP).
High risk pregnancy, 2006
3 or 4 contraction every 10
Frequency minutes
Minimum 40 seconds
Duration
Good progress
Result
High risk pregnancy, 2006 and alarm course
Reported causes of uterine
dysfunction
Epidural analgesia
Chorioamnionitis
Maternal position during labor
William obstetrics 24 ed.
Management of hypotonic
dysfunction
Check maternal condition
Informed consent
Assess maternal pelvis and fetal presentation.
Remember =ineffective labor is generally
accepted as apossible warning sign of CPD
Amniotomy in case of intact membrane.
Oxytocin augmentation
Because of
1. Clinical and x ray pelvimetry have poor
predictive values.
2. the mechanism of labor that relies on
flexion, rotation, molding and even pelvic
compliance.
High risk pregnancy, 2006
Caused by
Alarm course
Generally assume
Malpresentation
Face, brow, shoulder/arm, breech, compound
presentation
Malposition
Persistent occipito transverse
Malformations
Hydrocephalus, abdominal tumors, cystic higroma,
conjoined twins
Alarm course
Macrosomia
Shoulder dystocia
William obstetrics 24 ed
Clinical feature of CPD
Excessive moulding
Failure of presenting part to engage and descent.
High risk pregnancy 2006
Temuan sebagai indikasi CPD
Pemeriksaan abdomen
- ukuran janin besar ( > 4 kg )
- kepala janin diatas PAP
Pemeriksaan panggul
- serviks mengecil setelah amniotomi
- edema serviks
- caput
- molase bera
- defleksi kepala (fontanella anterior mudah dipalpasi.
- asinklitismus (sutura sagitalis tidak tepat di tengah panggul)
PONEK
Alarm guideline for
management dystocia
Arrest without CPD = amniotomy and
oxytocin augmentation.
Arrest withCPD = CS
Remember
A lack of descent in the absence of moulding
or caput is likely due to inadequate
contractions.
Alarm guideline for
prevention of dystocia
Accurate diagnosis of labor
Management of prolonged latent phase (PLP)
Deff PLP = time > 20 hours
Labor preparation (prenatal education)
Birth companion (continuous support)
Not only health discipline
Ambulation
Choice of labor position
Active management of labor
Analgesia
Alarm guideline for active
management of labor
Correct diagnosis of labor.
Close surveillance of progress of labor by
partogram.
Continuous support in labor.
Early intervention to correct inadequate progress
of labor with appropriate intervention = such as
amniotomy, oxytocin augmentation.
Maternal effects of dystocia
Intrapartum infection
Uterine rupture
Pathological retraction ring
Fistula formation
Pelvic floor injury
Postpartum lower extremity nerve injury
Wiiliam obstetrics 24 ed
Fetal effects of dystocia
Caput succedaneum
Fetal head molding
Asfiksia
Wiiliam obstetrics 24 ed