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EFFECT OF MANEUVER

G FORCE
ACCELERATION :
LINIER : Vt = Vo + at , S= Vo t + ½ a t ²
ANGULER : a = V ² / R
G=a/g

PH = h d G
PH = HIDROSTATIC PRESSURE GRADIENT
d = SPECIFIC DENSITY = 1/ 13.6
h = DISTANCE -GZ
+GX

CENTRIFUGAL -G Y +G Y

- GX
+GZ
GENERAL AFFECT
MOBILITY : +2G SAGGING SOFT TISSUE OF FACE
+ 3G DIFFICULT RAISE FROM
SITTING

VISUAL SYMPTOM : - TUNNEL VISION


- GRAY OUT
- BLACK OUT

UNCONSCIOUSNESS : TOTAL LOSS OF MUSCLE TONE SO


THAT THE HEAD & TRUNK SLUMP
AT MODERAT LEVEL 5-6 +Gz
AT HIGHER ACCELERATION
WITHOUT ANY VISUAL SYMPTOM
AT VERY ROR  AFTER 4-6 SEC
GENERAL
AFFECT
CARDIOVASCULAR
EFFECT :

- BARORECEPTOR REFLEX
- CARDIAC ARRYTMIA
- 3 SPECIFIC DYSRITHMIA CAUSE PARTICULAR
CONCERNS: SA BLOCK, AV DISSOCIATION,
VENTRICULAR TACHYCARDIA

PULMONAR EFFECT :
POLLING BLOOD IN ACCELERATION ATLECTAISIS :
LOWER PART OF LUNG DRY COUGH, SUBSTERNAL
RESULT VENTILATION DISCOMFORT OR CHEST PAIN,
PERFUSION RATIO EXACERBATED BY DEEP
DIFFERENCE IN UPPER INSPIRATION, DYSPNOE
& LOWER PART
G TOLERANCE
PH = h d G
EXAMPLE : h= DISTANCE OF EYE to COR = 300 mm
PH FOR 1 G = h d G = 300 x 1/13,6 = 22 mmhg / 1 G
IF MEAN ARTERIAL PRESSURE /MAP = 120 mmhg
INTRA OCULAR PRESSURE 25 mmhg
RELAX G TOLERANCE IN GRADUAL ONSET

VISUAL SYMPTOMS OCCUR


AT ( 120 –25) : 22 = 4,7 G
- TUNNEL VISION
- GRAY OUT
- BLACK OUT
G LOC OCCUR AT 120 :22 = 5,45 G
AT HIGH G ON SET VISUAL
SYMPTOMS NOT APPEAR/OCCUR
HIGH G ONSET
GRADUAL
ONSET

PROTECTIVE STRATEGIES :
- CENTRIFUGE TRAINING
- ANTI G SUIT & G VALVE
- INCREASED SEAT BACK ANGLE
/LEAN
- AGSM L1/M1 MANEUVER
SYMP/PARASYMP BARORECEPTOR
TONE 6 – 12 SEC
GOR - TV (PLL)
ROR - GRAY OUT
RECLINING
+ SEAT - BLACK OUT
SV Ph= h d G
CARDIAC HEAD ± 0.8 G
CO +
+ - RESERVE 02 G LOC
BLOOD << 4 – 6 SEC
HR MAP - BONY BOX
RECOVARY
+ - ACTIVE
ROR > GOR
VASODILATE
- SIPHON 24 SEC 37 SEC
RESISTENSI +Gz EFECT
PERIFER

AEROBIC PPB
TRAINING APEX V/P RATIO >>
PULMO
r HC
a= V²/r BASAL V/P RATIO <<
ANGULAR
ACCELERATION

ABDOMEN AGSM

a= V/t=V²/2S=2S/t² BLOOD >>


LINIER ACCELERATION
ARM AGS
WEIGHT
TRAINING
LEG PALE
dY/dt CAHNGES IN ONSET RATE

Brain Heart
dY/dt dY/dt

R.O.R
G.O.R R.O.R G.O.R

High increase
in intracranial Diastole systole
mechanical stress

Brain C.S.F. Pressure Increase in Decrease in


+ - mechanical end diastolic end distolic
stresses volume volume
Blood shift toward Increase in vessels
lower limb surfaces pressure
Heterometric
myocsrdisl
Low brain blood Venous Transmural arterial sdjusment
depletion collaps pressure inversion (Starling)

+ + Supercritical
+ flow

I.C.H
+ Dynamic
+ shock

“Hydraulic Ram”
effect
+ +
if too early 1 st Systole
+ pressure

- Anti G Suit
M1 , Li maneuvers P.P.B Homeometric
increased in blood pressure + O2
myocardial
adjusment
_
+
HYPOXIA
NEGATIVE G z

- OPPOSITE TO + Gz
- - 2,5 Gz : SENSE OF FULLNESS, OEDEM EYE LIDS, PETECHIAL
HAEMORRHAGE IN SKIN OF FACE AND NECK
- - 2,5 Gz to – 3Gz : FEEL AS IF EYE ARE POPPING OUT OF THE EYE
- -4 to – 5 Gz : LONGER THAN 6 SEC -> MENTAL CONFUSION AND
UNCONSCIOUSNESS

CARDIOVASCULAR EFFECT :
- HYDROSTATIC EFFECT
- CAROTID SINUS& CARDIAC OUTPUT  BRADY CARDIA 
CARDIAC ARRYTHMIA  ARTERIOLAR VASODILATATION
- CEREBRAL CIRCULATION : NO SIGNIFICANT RISK OF
RUPTURE OF VESSEL WITHIN SKULL
PULMONARY EFFECT: DIAPHRAGMA PUSH TO UPWARD VITAL
CAPACITY & FUNTIONAL CAPACITY DECREASE

TOLERANCE NOT WELL TOLERANCE


- 2Gz ABOUT 5 MIN, - 5Gz FOR SEVERAL SEC (MAX 5 SEC),
-3Gz IN 10 TO 15 SEC
RED LAMP RED LAMP

RED LAMP

LIGHT BAR

RED LAMP