By: Sytrose Morales Tabaco

Mgt. Pathogenesis Clinical features prevention .definition Etiologic agent Developmental stage Clinical manifestations Nursing.

but may spread to soft tissues. most commonly to the liver or lungs. . by contiguity or hematogenous of lymphatic dissemination.Amoebiasis is a protozoal infection of human beings initially involves the colon.

to the liver. to the portal vein. . It passes to the mesenteric veins.Entamoeba Histolytica Prevalent in unsanitary areas Common in warm climate Acquired by swallowing Cyst survives a few days outside of the body Cyst passes to the large intestine and hatch into the trophozoites. thereby forming amoebic liver abscess.

 This is considered as the infective stage in the life cycle of E. Trophozoites/vegetative form  These are facultative parasites that may invade the tissues or may be found in the parasitized tissues and liquid colonic contents. Cyst  It is passed out with formed or semi-formed stools and are resistant to environmental conditions. histolytica.Two Developmental Stages 1. 2. .

colon.Ingestion of Bacteria Multiplication in mucosa Endotoxin production affecting the lining of the small intestines. and capillary Necrosis of the mucosal layer Ulceration Gangrene TOXEMIA .

Acute Amoebic Dysentery  Slight attack of diarrhea.Clinical Manifestations 1. watery and foul-smelling stool often containing blood-streaked mucus  Colic and gaseous distention of the lower abdomen  Nausea. altered with periods of constipation and often accompanied by tenesmus  Diarrhea. abdominal distention and tenderness in the right iliac region over the colon . flatulence.

loss. scattered ulceration with yellowish and erythematous border Gangrenous type (fatal cases) is characterized by the appearance of large sloughs of intestinal tissues in the stool accompanied by hemorrhage . Chronic Amoebic Dysentery           Attack of dysentery that lasts for several days. and weakness Liver may be enlarged The stool at first is semi fluid but soon becomes watery.2. flatulence. constipation or irregularity of bowel. bloody and mucoid Vague abdominal distress. wt. and lassitude Abdomen loses its elasticity when picked-up between fingers On sigmoidoscopy. Mild toxemia. usually succeeded by constipation Tenesmus accompanied by the desire to defecate Anorexia. constant fatigue.

c.3. e. b. d. pain at the URQ with tenderness of the liver Jaundice Intermittent fever Loss of weight or anorexia Abscess may break through the lungs. Extraintestinal forms Hepatic a. patients coughs anchovy-sauce sputum .

Clinical Features 1. Abscesses in the skin 4. Onset is gradual 2. Diarrhea increases and stool besomes bloody and mucoid 3. In extreme cases liver abscesses if the infection goes to the bloodstream .

Provide health education and instruct the patient to: a.Nursing Management 1. Wash hands after defecation or before eating. Observe isolation and enteric precaution 2. e. Cover left-over food. d. Avoid washing food from open drum or pail. and Avoid ground vegetables (lettuce. carrots. b. and the like) . Boil water for drinking or use purifies water. c.

Sanitary disposal of feces 2.Prevention Methods 1. Observe scrupulous cleanliness in food preparation and food handling 4. Detection and treatment of carriers 5. chlorinate and purify drinking water 3. Protect. Fly control .