ALOPECIA

WHAT IS ALOPECIA?

IT IS ABSENCE OR LOSS OF HAIR

FORMS OF ALOPECIA   AREATA-BALD SPOTS PATCHY HAIR LOSS TOTALIS-COMPLETELY BALD OR WITH LITTLE VISIBLE HAIR ON SCALP UNIVERSALIS-NO VISIBLE HAIR ON BODY CICATRICIAL-SCARRING HAIR LOSS   .

. thyroid disease. As a result. and ulcerative colitis.ALOPECIA AREATA Current evidence suggests that alopecia areata is caused by an abnormality in the immune system. This particular abnormality leads to autoimmunity. the immune system attacks particular tissues of the body. What causes this is unknown. lupus. the body's own immune system attacks the hair follicles and disrupts normal hair formation. for unknown reasons. rheumatoid arthritis. Sometimes. alopecia areata occurs within family members. vitiligo. suggesting a role of genes and heredity. In alopecia areata. Biopsies of affected skin show immune cells inside of the hair follicles where they are not normally present. Alopecia areata is sometimes associated with other autoimmune conditions such as allergic disorders.

ALOPECIA AREATA .

heredity plays a role. someone else in the family also has it.Is alopecia areata hereditary?  Yes. In one out of five persons with alopecia areata. . Those who develop alopecia areata for the first time after the age of thirty years have less likelihood that another family member will have it. Those who develop their first patch of alopecia areata before the age of thirty have a higher possibility that other family members will also have it.

Its causes are unclear. Stress is sometimes thought to be a contributor to the hair loss caused by alopecia. however many people leading relatively stress-free lives have experienced the symptoms. but it is an autoimmune disorder. .ALOPECIA TOTALIS  Alopecia totalis is the loss of all head hair.

Fortunately. The affected hair follicles become small and drastically slow down hair production. .ALOPECIA TOTALIS (CONT’D)  In alopecia totalis. immune system cells called white blood cells attack the rapidly growing cells in the hair follicles that make the hair. the stem cells that continually supply the follicle with new cells do not seem to be targeted. So the follicle always has the potential to regrow hair.

.

Steroids may help but superficially. Steroids do not address internal autoimmune disorder. Our experience suggests that by use of steroids. eyes lashes is called Alopecia totalis. including eyebrows. a few patches of hair loss is called Alopecia areata. That is. while total hair loss all over the head. The treatment approach is different and the prognosis is also difference. while Alopecia totalis is not curable using homeopathy.IS THERE A DIFFERENCE BETWEEN AREATA AND TOTALIS?   For all practical purposes. Alopecia Areata is treatable with great success. . they are very useful during acute attacks. But. most patients get more spots elsewhere on the skin.

the only difference between Alopecia Universalis and it's variants is the extent of hair loss.ALOPECIA UNIVERSALIS  The most advanced form in a series of conditions all related to the same disease. A member of the group of hair loss conditions called Alopecia Areata. . Alopecia Universalis is characterized by total a loss of body hair.

but then begin losing it very quickly. Those with vitiligo (patchy loss of skin color) may also develop AU in time. but have more thyroid disease and vitiligo than the general population.2 that is the human homologue of the mouse "hairless" gene -. The disorder is inherited as an autosomal recessive trait. . Many individuals with Alopecia Universalis are born with some hair. It is caused by a mutation in a gene dubbed HR in chromosome band 8p21.ALOPECIA UNIVERSASLIS (CONT’D)  Patients are usually otherwise healthy.the human version of the gene in the mouse that is responsible for hairless mice.

Based on the known research. we can safely assume that only individuals with this rare and severe form of Alopecia Areata carry the gene. .ALOPECIA UNIVERSALIS (CONT’D)  Is the "hairless" gene only found in people with Alopecia Universalis? Most likely. there have not been enough studies done to verify that this is true of all those afflicted. Unfortunately.

all the way to severe distortion of the entire nail. as the scalp. The nail involvement may be limited to pinprick indentations. . and other potentially harmful elements. The possibility of regrowth does remain however. According to the National Alopecia Areata Foundation. Predicting when regrowth may occur is not currently possible. nasal cavity and eyes are not protected. or remain permanently. other than the hair. It is important that those with it are careful to protect themselves from the sun.ALOPECIA UNIVERSALIS (CONT”D)  Aside from genetic tendencies. even for those with 100% hair loss for many years. Alopecia Universalis may be acute and short-lived. bacteria. the contributing causes of Alopecia Universalis are not known. nails can also be affected.

.

but for the vast majority of children.sometimes up to a year or more. which can be quite frustrating both for the parents and the child affected by this often cosmetically embarrassing condition. .ALOPECIA IN CHILDREN  Hair loss in children is actually not very common. it will spontaneously resolve. The bad news is that 40% wont have such luck. however it is significant enough that nearly 2 Million children suffer from at least one form of Alopecia (hair loss) or another in the United States alone. As with all forms of Alopecia. The good news is that at least 60% of children with Alopecia will "outgrow" the condition without need for treatment. a reversal and complete restoration of hair takes time .

Some patients with this condition will also develop a grid-like pitting of the nails. poor scalp circulation. hyperactive disorders. caused by foods.CHILDREN WITH ALOPECIA  Please keep in mind: hair loss in children is not due to vitamin deficiencies (unless extreme malnutrition is present). or the result of nervousness. or psychological stress. Why the hair falls out from the roots is still a mystery. headbands. or cold weather. Alopecia is not life-threatening. What is known is that the condition is not contagious.  . Diagnosis is typically as simple as an evaluation of the risk factors a visual examination of the type of loss. In 20% of cases another family member has been affected. and children who have it are otherwise healthy. and some tests your doctor can perform. hats.

If the stem cells are destroyed. and the sebaceous gland as well.CICATRICIAL ALOPECIA  The cause of the various cicatricial alopecias is poorly understood. usually the upper part of the follicle where the stem cells and sebaceous gland (oil gland) are located. there is then no possibility for regeneration of the hair follicle and permanent hair loss results. . all cicatricial alopecias involve inflammation directed at the hair follicle. However.

ALOPECIAWORLD.WEBSITES AND SUPPORT GROUPS WWW.HAIRLOSSTALK.  .COM  CHILDRENS ALOPECIA PROJECT  NATIONAL ALOPECIA AREATA FOUNDATION  WEBMD FOR INFO  WWW.COM  GOOGLE – ALOPECIA AND FIND OTHER RELATED SITES.

DEDICATED TO JOSE CHARQUENO MY PELONSITO .

Sign up to vote on this title
UsefulNot useful