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Chronic Obstructive Lung Disease Chronic Airway Limitation
Description
A group of diseases that include: Chronic Bronchitis- chronic inflammation of bronchi unrelieved in 3 consecutive months and in 2 consecutive years Chronic Asthma (Status Astmaticus)- S/sx of allergic attack unrelieved within 24 hours of adequate therapy
Bronchiectasis- dilation of bronchioles r/t chronic airway obstruction Pulmonary Emphysemaoverdilatation of alveoli (compliance) and resulting in Recoil
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Basis
Chronic
airway blockage Airway resistance Progressive airflow limitations both ways Irreversible alveolar distention air trapping alveolar damage ABG imbalances: Low pO2, High pCO2
Possible Complications
Pulmonary hypertension Respiratory insufficiency or Respiratory failure Cor Pulmonale CO2 Narcosis Alveolar Rupture Atelectasis
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Bronchial Asthma
Description
Also
called RAD (Reactive Airway Disease) and ROAD (Reversible Obstructive Airway Disease) A complex inflammatory process that results to increased airway resistance and later, alveolar damage Airway inflammation r/t hyperresponsiveness (hypersensitivity) to allergens
Etiology
Intrinsic allergens
Fatigue Stress / anxiety
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Types of Asthma
Immunologic asthma
Occurs in childhood r/t allergens; Allergic asthma or atopic asthma; heredity; high lgE
Non-Immunologic
Occurs in adulthood, usu. Associated with URTI or LRTI Non-allergic asthma or nonatopic asthma; onset usually > 35 years
Mixed Asthma
Any age; any allergen; nonspecific stimuli
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Pathophysiology
Allergens Release of IgE by B-lymphocytes IgE + mast cells (respiratory tract) Damage to mast cells
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Vasodilation Hypotension
Capillary Permebility
Shock
Escape of Colloids BV
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Edema
Other
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Nursing Interventions
Administer medications, as ordered Administer nebulizer as ordered Provide patient teaching about preventing attacks and proper use of medications
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Pharmacotherapy: Bronchodilators to relieve bronchospasm Beta-Adrenergic agents: rapid onset of actions when administered by aerosol
Corticosteroids to relieve inflammation and edema Antibiotics if secondary infection Cromolyn sodium not used during acute attack; inhaled; inhibits histamine release in the lungs and prevents attack
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Chronic Bronchitis
Is
May
be o Acute when the bronchus becomes inflamed o Chronic results when inflammation occurs several times a year; can be diagnosed by the presence of cough that persists for 3 months a year for 2 years
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Etiology
Exposure
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PATHOPHYSIOLOGICAL PROCESS
Causes : Cigarette Smoking
RTI
Environmental Pollutants
INFLAMMATION
Bradykinin
Capillary Permeability
Histamine
Prostaglandin
Hypersecretion of Mucus
Signs
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Nursing Interventions
Eliminate
/ minimize patients exposure to irritants and people with RTI. Clear airways with chest physical therapy or suctioning as ordered. Mucolytics as prescribed. Deep-breathing exercises. Patient teaching about adequate nutrition and medication therapy.
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Pulmonary Emphysema
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Description
Terminal
stage of COPD Overdilated alveoli and bronchioles Damage to alveoli and failure of alveolar diffusion NSg. Dx: Imp. Gas Exchange ABG: paO2 paCO2
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Etiology
Predisposing Fxs:
A-ge H-eredity (low alpha1 antitrypsin) A-uto-Immune tendency
Precipitating Fxs:
B- ronchitis, chronic A-ir Pollution S-moking A-sthma, chronic
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Based on Types:
CENTRIBULAR
Blue Bloater Stage 1st stage Most bronchioles and alveoli plugged with mucus Central airway dilated Danger: Cor Pulmonale
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Blue
Bloater Type Cyanotic Edematous W/ prod. Cough D.O.E. Weakness Nail Clubbing ABG: Resp. Acidosis S/S of hypoxia S/S of R-sided CHF Barrel-shaped chest
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PAN-LOBULAR
2nd stage Most alveoli and bronchioles dilated Mucus expelled Hyperventilating (compensation to high pCO2)
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Pink
Puffers Pinkish skin color Emaciated Non-productive cough Severe weakness Anorexia Dyspnea ABG: Resp. Alkalosis
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fatigue Pursed lip breathing Barrel Chest Dyspnea, orthopnea Retractions Prolonged I:E ratio Wheezing on expiration Clubbing
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Nursing Interventions
NDx1: Gas Exchange, Imp. R/t ventilation: perfusion mismatching (Physiologic shunting) Goal 1: Normal ABG values 2: No Hypoxia
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Monitor: ABGs, s/sx of resp. acidosis. s/sx of hypoxia, pulse oximeter (O2 sat) Give bronchodilators as ordered (p.o., IV, rectal, nebulizer). Check side effects:
Dysrhythmias HR, BP Excitation (L.O.C.) N&V Tremors
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Low flow O2 therapy with venturi mask at 24-30% concentration or nasal cannula at 1-3 L/min Good humidification Liquify secretions Suction PRN Avoid narcotics- depress RR
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NDx2: Airway clearance, Ineffective r/t chronic asthma, bronchitis, smoking, pollution Goal 1: Open airway Goal 2: Adequate ventilation
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Assess: VS, cough, record consistency of sputum secretion, s/sx of hypoxia Increase fluids p.o.-6-10 glasses (3L)/day unless C.I. Nebulization as ordered Mucolytics as ordered Avoid milk, creams
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Respiratory therapy Antibiotics or antihistaminics as ordered Position: High fowlers lean forward. Use overbed table Administer steroids as ordered to decrease swelling of airway
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NDx3: Breathing pattern, impaired r/t airway obstruction Goal 1: Improve pattern of breathing or ventilation Goal 2: Relief of Dyspnea
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Position Pursed-lip breathing Blow bottle exercises IPPB with nebulization Alternate activities with rest
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Teach use of inspiratory muscle traininer (use 10 min/day to strengthen respiratory muscles) Teach to coordinate diaphragmatic breathing with activity Use controlled breathing while bending, walking, bathing Teach postural drainage
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NDx4: High Risk : Complications Goal 1: Prevent complications: CO2 Narcosis Resp. acidosis Cor Pulmonale Respiratory Failure
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Ensure low flow O2 Monitor ABG Fluid intake= 1.0-1.5 l/day if w/ Rsided CHF or pulmonary edema Diuretics as ordered IVF tkvo-use D5W Tracheostomy tube if necessary
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Encourage catharsis Involve in self-care and improve self-esteem Allow to make decisions about his care (shaving, bathing , eating, etc) Adopt a hopeful and encouraging attitude towards pt Encourage activity to level of tolerance to improve self-esteem
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Monitor compliance to regimen Allow use of O2 during activities Teach relaxation tech, energy conservation Gradually increasing exercise program using an insp. Resistive device (blow bottle) Pulmonary Rehab. Tech
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LUNG CANCER
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Description
Refers
to malignant tumor growth within the bronchial tissue or lung parenchyma. Types include:
Squamous cell 35 50% of all lung cancers. Adenocarcinoma 15 35% of all lung cancers. Small cell (oat cell) 20-25% of all lung cancers Large cell 10-15% of all lung cancers 46
Family Tend
to have poor prognosis, unless it is very well defined and removed by surgery.
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Pathophysiology
As the lung tissue experiences irritation, it undergoes a series of changes and eventually gives rise to a tumor. Metastases can occur, especially when the mother tumor is near areas of lymph drainage. Some tumors secrete hormones: ADH reabsorption of water ACTH stimulates adrenal glands to produce steroids
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Symptoms
may include:
Cough Wheezing Shortness of breath Chest pains Hoarseness Dysphagia (compression of esophagus) Weight loss
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Nursing Interventions
Prepare the patient for surgery if tumor is small enough to be removed Prepare patient for planned treatments chemotherapy / radiation therapy Analgesics as ordered to control pain Adequate oxygenation through oxygen therapy or planned activityrest Maintain nutritional status Provide emotional support to the patient and family 50