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The document outlines a comprehensive guide for clinical examination and case taking in medicine, detailing various systems such as respiratory, cardiovascular, and abdominal examinations. It includes references to standard medical texts and emphasizes the importance of thorough patient history, including personal, treatment, and socioeconomic factors. Additionally, it discusses specific conditions and examination techniques, particularly in assessing pulse characteristics and their clinical significance.
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Edited by:
Dr. Dipendra Khadl
MBBS (KU), MD (KU) Medicins
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RATURE V4
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bration 198
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Ly \(PH NODE EXAMINATION 196
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RAL EXAMINATION OF A
NT ASS.
RESPIRATOTY SYSTEM 44%
CARDIOVASCULAR SYSTEM 8826
ABDOMINAL SYSTEM 02 98
GENITOURINARY SYSTEM 77°.
NERVOUSSYSTEM 274
CHEST X-RAY 339.
roc Boe
REFERENCES:
Hutehison's clinical methods
Macleod's clinical examination
Manual of Practical Medicine, R, Alagappan
Davidson's Principles and Practice of Medicine
Harrison's Principles of Internal Medicine
Bedside Clinics in Medicine Part I, Arup Kumar Kundi
Bedside Clinics in Medicine Part Il, Arup Kumar Kundu
Medicine: Prep Manual for Undergraduates, K. George
Mathews; Praveen Aggarwal
ECG made easy, Atul Luthra
The ECG made easy, John R. Hampton
(Chest X-ray made easy
BATES Guide to Physical Examination & History Taking,
‘Textbook of Human Neuroanatomy (Fundamental and
Clinical), Inderbir Singh
Clinical neuroanatomy, Richard S. Snell
BD Chaurasia's Human Anatomy Volume 1, 2 and 3Particulars
Chief complaints
History of present illness
“History of past illness
Personal history
Treatment history
Socioeconomic history
Drug and allergy history
General examination’
Local examination
Systemic examination
Respiratory)
Cardiovascular
‘Abdominal!
Name; Identity; clue about country, state and
religion. Needed for obtaining information from.
CASE TAKING
2
Ischemic heart disease,
Bronchogenic carcinoma
Haemophilia
nale: Autoimmune diseases like
SLE
Thyroid disorder
ws/ Muslims (citcumeision); Less prone to
CaPenis
Muslims (co
to decompensated liver disease
Less prone to Ca lung
Less
Certain Hindus (cons
prone to Ca colon
Occupation:
« Silicosis: Mine workers
(@romatic amine)
© Mesothelioma: Asbestos
hours of standing)
‘Marital status
h. Date of examination
Chief Complaints:
= Current complaint which brought patient to
hospital & their duration in chronological order
History of present illness:
Elaborate patient complaints in their own words
from its onset to its present state
© No leading questionsSiar ilness nthe Past i
afletier medic! o° UMMA
saad disenses tt he
Wont had suffered
i: Hypertension
Re Rheumatic fee"
EE
‘a: Asthma,
Aniety
Arthritis
patient
Disease
had to take medi
g duration? pee
Depression
forlong
How long? ;
staking 5: Surgical history
What type (Cigarette, Cigar or Pipe)
Number of cigarette per day
Duration of smoking in years
Pack years =
‘Number of cigarettes smoked per day *
‘Duration of smoking in
‘Rskfor lang cancer=30 pack years American
‘Associaton for Thoracic Surgery)
‘Smoking index (S1)=
Number of cigarettes smoked per day *
‘Duration of smoking in years
51 < 100 = mild smoker,
101 to 300 = moderate smoker,
> 300 heavy smoker
Lung cancers common if SI > 300,
Alcohol
8 What type (Local, Wine, Whiskey, Beet)
Daily/weekly pattern (specially binge
Arinking and morning drinking)
Usual place of drinking
Alone or accompanied
= Purpose
Amount of money spent on alcohol
* Attitude to alcohol
7
{alcohol deP'
inquiry: .
sor felt the need to Cut dwg,
Enel consumption?
aspiave you tlt Angry st otnetaag
inking?
Seen ever feel Guilty about extess
ver drink in morning a8 Bye
.nswer for problemof
G:Doyou
E:Doyou
up 1 Know alcohol by volume
SP cer 5%, Cider 4.5%, Winemy
Champagne 12%, Spirit 40%) Ned
Romy Gin 375%, Whisky 40%, Home
13% (NHRC 2015)isu
roe cot]
« traditional volumes:
step 2: Know t I
Tea glass 150 ml, Water glass 250 mi
bottle 650 ml
step 3: Number of units
Volume * % of alcohol by volume:
: 700
(since, 1 unit= 10 gram]
regular consumption of more thang
ceetbrmen and more than 2 uniter
Tobacco betel nut chewing: More PRO
ral cavity
Food habit: Vep/ nomveg
Bladder and bowel habits
local
f, Sleep and appetite
g. lf femal
‘Ask: Menstrual history, Obstetric his
Contraceptive history (As given |
gynae section)
Family history:
"= Similar illness in any of the family met
1 Diseases (THREAD) like Diabetes,
‘which may be hereditary;
= Recent exposure to i 8 dis
Recent expa infectious
Socioeconomic history:
Occupation, Family income, Educa
"Housing condition (Type of hous
rooms, separate toilet, kitchen)
Ventilation facility, Surrounding
© Source of water
Drug
ind allergic history.
's the patient taking any drug at
Any history of allerg
Any history of allergies to any’ mi
y ther things like some food) ust
ial pulse 18 21
emitted along arte
Miuring cardiac ¢)
} t
F
volume | 1
Character) J
Condition of
Radio-radial d
Radio-femoral
Rhythm
Palpation of p
fal artery: P
Bx0r Carpi Rary atothets crite
silty about excess d
‘by volume
onal volumes:
1, Water glass 250 mi
nits
alcohol by volume:
7000
10 gram}
ion of more than 3 Uy
nore than 2 unit per d
hewing: More prone
veg
abits
ry, Obstetric history,
nistory (As given inv
of the family mem
ncome, Education
Type of house,
kitchen)
srrounding sani
ny drug at Pre
jes to any med
‘ome food, dus
pulse is a palpable pressure wave form
Tong arterial Wall and generated at root of
z cardiac cycle
| ra
ravthm J palpating radial artery
volume | bestassessed by
character! palpating carotid artery
Condition of vessel wall
adio-raial delay
fadio-femoral delay
palpation of peripheral arteries
Posterior
isla
Racial artery: Palpated at the wrist, lateral to the
Bésos Carpi Radialis tendon against lower border
lth font of radius (radial styloid process)
® old the patient's wrist as if you are going to
shake your hands (hold the patient's right hand
With your right hand or viee versa)
B Now slightly flex the wrist and assess Radial
Pulse with3 fingers
ipheralartr
ee
Distal finger: Fixes
Middle finger: Feels
‘Assesses state of w
Patpate both radial pulses simultaneously for
Radio-radial delay
Palpate the Radial and Femoral pulse
simultaneously for Radio-femoral delay
Check for the condition of arterial wall—ediion otvesew |
[Comment Rate, uae
| Radio-adial del |
ecubital F852,
atpated inthe atest
"I rosie lowe!
aa to bleps tendon: oP
apne with eum wih 7
rack he elbow
around the|
vee eeainer mi ruPPoR REDS
tbo wight
le with ee thu \
eames ery; Palpated medial fo
seid mscte atthe level Of MEPS
eae
ge gat cro
ee c process of 6th
ede of ere) of anes
(Coase aera incaota ian |
Soo ea unb i eenecedemast |
Pa lotta tte! |
Sly toward te a
rend of
sur finger cuPPed |
nisrelaxed |
ey emmpressing te bFachal |
[Votume and charact
palpating it
|
ses simultaneous! |
Tempora avery: alpen ont |
Sflne agus ofthe ear agaist zygomatic Bone.
Gubelavin atery: Palpated just posterior tothe | ?
idle third of clavicle by shrugging the shoulder
(cgans the 1st). |
Femoral artery Pal
‘ginal lig
in the groin just below the
Imidway between the anterior
|
| 10,
Je and the pubic symphysis (mid |
®agrnst the head ofthe femur and the |]
major.
patient tolie supine with hip flexed about | puuse RATE:
|2 Sep ia
swith finger extended places
palpate
a fingers over the femora
index and middle
rea ife patente suey ‘cree Reed
itmakes 135°
co thumbs onthe tial Suber a
aay fossa with 4 Fingers ofeach
ett ane Pai a
ec of upper endo HO
Place
Foptructive
Fpaiced Intra cranial Press
Byasovagal attack
Dyicart blocks”
gs: Beta blockers Vet
palpate in prone position with knee flex
seth relax the popliteal fossa (est mela
ee, poplite! artery is palpated agaist ae ihe
femur. |
Posterior tibial artery: Palpated 2 em Bel
behind the medial malleolus midway betwee
tendon Achilles, against the calcanewm:
a artery: Palpated anterioniy
tnidwray between the two maleol agains
tnd of tibia just above the ankle joint la
‘Extensor Hallucis Longus tendon.
Dorsalis pedis; Palpated just lateral to thes
allucis Longus tendon athe prox
the groove between the 15" and’
against the navicular and middle
the difference bed
‘hen counted si
Normal: 60-100 / min.
| Physfotogical:
Infants
| 2. Children:ned
ove een
with knee exed gp
ibial tuberosity & p
{ngers of each hand,
yi palpated agama poate
pleat blocks
—Fimus bradycardia (60/ min)
so
Be vere hypoxia ’
Fypotnermia .
fk sins syndrome
iypotnyroidiso ©
Hopructive jaundice
Poised intra cranial pressure 7
Wasovagal attack
Drugs: Beta-blockers, Verapamil, Diltiazem, Digoxin
ieficit (Apex-Pulse Defic
Ttis the difference between the heart rate & pulse
fate, when counted simultaneously for-one full
‘Sinus Trachycardia (°100/ min)
my
ee
Pathological:
‘Tachyarrhythmia ©
High output states:
= Thyrotoxicosis
= Anaemia
~ Pyrexia ~
= Pheochromocytoma(@TAPP)
= Beriberi ~
Cardic failure —
CCardiogenic shock“
Hypovolemia
Hypotension
Drugs: Atropine, Nifedipine, Thyroxine,
Catecholamines, Salbutamol
Arrhythmias:
= Atrial Fibrillation —
= Atrial Flutter
PSVT <
‘Ventricular tachycardia ~
Causes:
fe Atrial fibrillation
‘= Ventricular premature beats ~~
<10/min
Present
Decreases or disappears
‘Ventricular premature beats
hort pause followed by along pause-|
‘Atrial fibrillation
>On,
Absent
ba
Persists or increases “~
Pauses ae variable chaotic
ed by palpating the radial artery:
iy iregular shythm:
‘Arterial tachyamhytmias (PAT & arterial ution)
th fixed AV block
Ay iregular rhythm:
Asal or ventricular ectopics\—
Pulse volume:
“Assessed by palpating the carotid artery.
Pulse pressure gives accurate measure of pulse
volume
fe Normal volume pulse: 30-60 mmHg pulse
pressure
‘e- Small volume pulse: Less than 30-60 mmig,
pulse pressure
fa Large volume pulse: Greater than 60 mmHgSe features {if any)
I pro a because of objection of bolus of ‘plood
Meera ota daring 31
Triste oly ptpable wae
sls and generates
atic after distention itrevols and
porate
E re which is T wave.
some pressur
ta during diastole.
Because of esting pressure in aorta GUNNS
jeri ote
Hypokinetc pulse
Sina weak pase (6mall volume and narrow pulse |
|
Fig: Hypokinerc puis
i: Hyponete ise
fictive pericardial disease
Pals parvus Slow volume pulse d/t diminish
lt verter vue congas hart
flue or.a¥ in systemic arterial pressure
Pu
stole
Slow ring pss at eas nein |
Eg. Aortic stenosis
se pulse ane wide pulse p
tan tee eh 2d
2 fgh output states
“Thyrotoxicosis
“Anaemia
AV fistula
pyrexia
= Pregnancy
MR
vsb ‘
; pulse (Collapsing pulsefOaH
~» volume pulse with rapid upstroke
var gh) sustained pe a
Pres toke (dastolic pressures IOW)e
Rapid upstroke: d/t markedly
stroke volume;
Rapid down stroke: d/t
Diastolic leak back into left
ventricle
Rapid run off to the periphery
systemic vascular resistance
Procedure:
a Palpate the wrist in such a way that
fall on the radial artery and rest of
over the ulnar artery.
‘= Examine the volume of the pulse
both radial and ulnar artery) fora
'= Now elevate the whole upper lil
above the level of the heart & ny
any changes in the volume of the}
‘= Abrupt downstroke of the pul
collapsing feel.
Right sided pulse shoul be
hil standing on the right sides
"sv Raise the arm to feel fOPt
Jevate the arm:
sto!
cada to fall of Blood «
Br scodilatation) and ths
Yjostolic pressure more
widens.
oR
1¢ may be so that the art
More in the Tine of aorta
Bem, and thus allows dite
Gisstolic backward flow
of collapsing pulse:
“nortic regurgitation
Hyperkinetic circulatory
‘Anaemia
Fever
Thyrotoxcosis
Pregnancy
Exercise
Beribert
Patent cuuctusartriosu
“Arteriovenous fistala
fulsus Bisferiens:
pulse wave with tw
i/t Percussion wave (PSe al of Hod coll ase
or |
ions des yeelicgetonara)
swardfow. |
|
Gj sortic regurgitation
GF Hyperkinetic circulatory sates i
upstroke (ysi
peak and a ra Anaemia
Fever
Thyrotoxicosis
Pregnancy
Exercise
= Beribert
ip) Patent ductus arteriosus (PDA)
IF Arteriovenous fistula
BF Ruptured sinus of valsalva
Palsus Bis
le pulse wave with two peaks in systole
Beat percussion wave (P) and 24 dt tidal wave
Haaren: jection of pit of loo Hough
Beta |
fat in big arteries like brachial and femoral |
vere aortic regurgitation
Hypertrophic obstructive cardiomyopathy
Fig: Pulsus Bisferiens
ollapsiti
ingle putse wave with one peak in systole and one
peak in diastole.
‘Mechanism: D/t a very low stroke volume with
decreased peripheral resistance.
Left ventricular failure
Typhoid fever
Extreme dehydration
Dilated cardiomyopathy
Cardiac tamponade
nalsus alternans:
‘Alternating small and large volume pulse in regular
rhythm.
Best appreciated by palpating radial, femoral pulses
rather than carotid.
Cause; Left ventricular failure
Mechanism:
‘D/tatternating left ventricular contractile force ie
altemate strong and weak beats.
‘Note; One should search for $3 gallop rhythmand
‘basal crepitation when pulse alternans is felt
Fig: Pulsus alternons
Pulsus bigeminus:
‘Normal beat followed by a premature beat and a
ompensatory pause, occurring in rapid succession,
sarang in alteration of the strength of the pulse,
producing irregular rhythm.supe
Digs toxic y Palos par esph
Sig of ign atory pase! saree the BP cult 0 SUPFasystolic feel gl es
sent whee asin puts RSA anil =
compensatory pause SP Peaksystoic presume during oP OR a Po
: Then cuffs dfted even mot slowly angi °°!
rescporated inspiratory fallin systolic Pressure sien noted when KorotKoff sound berg
Amy during quit thi 1 a sughout the respiration cya i
sare Heart aounds are sll avaible at atime ere
chen radial pus il ein
ee Inflate the BP cuff to suprasytoliclevel and da
© Consttive pericarditis i
DE il Present if there is an alteration in the intensiyf
Superior venacava obs conte "
Inspiation
ssessng the condtion of vessel
Normaly arterial wal impalpable and may be papal
inold aged/ arteriosclerosis ;,
Inartriosclersis, the artery becomes tortuous tickeMufemo
tues Ie cord k/a"Monckenberg’smedialsdem Gi.
Ps doxus | Demonstration: hand ar
Compress the brachial artery above tiga
Mechanism: by ball of the left thumb and now roll the rad) ©auses:
Noxmally, fallin systolic BP i < 1mm during artery over radius by index and middle ings | m_Coa
Inspiration. Because of the right hand,
Duringinpzaton therein intrathoracic pressure oR
> poolingof ocd inpulmonary vasculature and | & First place the index and middle fingers of
Fight ventricle > in venous return in left atrium & left and tight hand over radial artery side by:
‘ventricle in cardiac output, and exsanguinate the artery by moving the mi
In puss paradoxus, more blood comes in right fingers in opposite direction,
ventricle Which pushes the interventricular septum | © The radial artery is now rolled over the
to theleft side 4 inthe volume of left ventricular by two index fingers.
cavity Further in cardiac output.
In cardiac tamponade: above effect + raised
intrapericardal pressure (compresses the heart from,
Raa Radioradial delay:
Inacute severe asthma:
and Radiof
Simultaneously palpate both the arteries by bol
Both ings ate expanded compres th heart Your and using your left and or ighada
JSPs timely memes a ‘and vice versa for the other hand,
| = Pulsus paradoxus
—* Pulsus alterans
Normal anatomical variation:
"Thoracic inlet syndrome e.g. cervical rib
Aneurysm of the arch of aortaslowly and
ound best
on eycle.
) pressures
gupravalvular aortic stenosis,
Feripheral embolism or atheromatous plaque
iauneroxclerosis of aorta |
pressure over axillary artery by ymphnode |
iatrogenic: Blalock Taussing shunt operation in |
Tetralogy of Fallot
Readiofemoral del
Gimultancously palpate the radial artery with left
Fand and femoral artery of same side with right hand
Tollook or the radio-femoral delay.
Gis:
B)Coarctation of aorta
Atherosclerosis of aorta
Thrombosis or embolism of aorta
Acrtoartertis
[ Carotid
Brachial
ee
| Radiat __Rate
Rhythm
Tea ccathing pattern may be
al bres
—
respiration Events
Moving thea into and out of he 4
Met ethed entation ona.
achanging ges between the int
Cingn ad the blood
ansport of oxygen to the body ee
ae pnd the return of carbon dione,
ee omy ;
: _achanging gases between the blooad | @ Re
Be Cohn) a : s
Rate (RR
Normal:
Nervousness
* Pain i
o Fever
© Hypoxia i
Respiratory conditions: |
= aie pulsonary oor
Perce
ines enntin
nos
Metblacidss
= Asthma
Rhyth
It includes entire breathing (inspiration and
expiration) cycle
Duration of inspiratory phase is longer
than expiratory phase with no pause in betw
and this cycle occu ae
= Women: Thoraco-abominal
= Men: Abdomino- thoracic |
| CGitSen > erect cbr lose
‘sig the oxygen in cel processes ae a Cardiae(left ventric
Renal failure
= Narcotic poisoning,
fs Raised intracranial
Ee ii. Kussmaul's breathing
Al depth of breathing,
pea idi = Metabolic acidosis-
EI = Pontine lesions
Narevi drug poisoning = Pontinel
Raised intracranial tension.
Cardiac arrest
Reason:
In males, diaphragms are stronger compared
epnteon whe the oposite true for
Depth:
fers to the amount of ar thats inhaled andes
Hyperventilation: Increase in depth of
ntlation: Decrease in depth of
rpnoea: Increase in rate and deplll
respiraton
~ Acidosis
~ Brainstem lesion
~ Hysterian events
no :ne nt and out of the nga.
Re verttation!
rgng ones between thesia th
ratte bod
portfongento the bea el
Percent atoon doe
rng gues between the Eso
rey co
ne onrgen in calprocesses at
Sacto ot crbon deat
Onna
a mae |
eet |
followed by apnoea,
's Cardiac(left ventricular) failure
fs Renal failure
‘s Narcotic poisoning,
fs Raised intracranial pressure
fi, Kussmaul's breathing: Increase in rate and
depth of breathing,
2 Metabolic acidosis-DKA.
= Pontine lesions
b. Irregular abnormal pat
= Uraemia
= Hepatic failure
Shock
i. Biot's breathing: Apnoea between several
shallow or few deep inspirations
= Meningitis
‘Deep and shallow breaths
‘occurs randomly
18 Brain tem lesions
fi, Apneustic breathing: Pause at full inspiration
iterating witha pause in expiration, lasting,
for 2t0 3 secs
f= Pontine lesions
iv. Cogwheel breathing: Interrupted type of
breathing
Nervous individuals
+. Pursed lip breathing: Patients breathes out
against pursed lip
= COPD esp. Emphysema
“Helps in increasing the intrabronchial pressure
above the surrounding alveoli and prevents
itscollapse