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  • Pathway ALO

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    Shila Wisnasari
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    This document discusses the pathophysiology of cardiogenic and non-cardiogenic pulmonary edema. Cardiogenic causes include left heart failure, pneumonia, and gastric aspiration. Non-cardiogenic causes include lymphangitic carcinomatosis, silicosis, pulmonary embolism, and preeclampsia. Pulmonary edema occurs due to an imbalance in Starling forces that results in a buildup of fluid in the lungs, including increased capillary pressure and decreased plasma oncotic pressure. This fluid accumulation in the alveoli leads to impaired gas exchange, reduced tissue oxygenation, and respiratory issues. Treatment involves bed rest, endotracheal intubation, mechanical ventilation, and management of infection risk and

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    acute lung oedem

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    This document discusses the pathophysiology of cardiogenic and non-cardiogenic pulmonary edema. Cardiogenic causes include left heart failure, pneumonia, and gastric aspiration. Non-cardiogenic causes include lymphangitic carcinomatosis, silicosis, pulmonary embolism, and preeclampsia. Pulmonary edema occurs due to an imbalance in Starling forces that results in a buildup of fluid in the lungs, including increased capillary pressure and decreased plasma oncotic pressure. This fluid accumulation in the alveoli leads to impaired gas exchange, reduced tissue oxygenation, and respiratory issues. Treatment involves bed rest, endotracheal intubation, mechanical ventilation, and management of infection risk and

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
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    6K views1 page

    Pathway ALO

    Uploaded by

    Shila Wisnasari
    This document discusses the pathophysiology of cardiogenic and non-cardiogenic pulmonary edema. Cardiogenic causes include left heart failure, pneumonia, and gastric aspiration. Non-cardiogenic causes include lymphangitic carcinomatosis, silicosis, pulmonary embolism, and preeclampsia. Pulmonary edema occurs due to an imbalance in Starling forces that results in a buildup of fluid in the lungs, including increased capillary pressure and decreased plasma oncotic pressure. This fluid accumulation in the alveoli leads to impaired gas exchange, reduced tissue oxygenation, and respiratory issues. Treatment involves bed rest, endotracheal intubation, mechanical ventilation, and management of infection risk and

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    PATHWAY ALO Faktor kardiogenik Gagal jantung kiri ARSD Pnemonia, Aspirasi As.

    . Lambung Bahan Toksik inhalan Faktor non-kardiogenik Isufisiensi limfatik Post. Lung transplant Lymphangitic arsinomiclosis Silicosis Unkwnown Pulmonary Embolism Eclamasia High altitude Pulmonary edema

    Ketidakseimbangan Staling Force

    Tekanan Kapiler Paru

    Tekanan Onkotik Plasma

    Tekanan Negative Interstitial

    Tekanan Onkotik Interstitial

    Cairan berpindah ke interstitial

    Akumulasi cairan berlebih (transudat / eksudat)

    Alveoli terisi cairan

    Cardiac ouput

    Pemasangan alat bantu nafas (ventilator)

    Gangguan pertukaran gas

    O2 jaringan

    Bed rest fisik

    Pemasangan selang endotrakheal

    Area invasi M.O

    Gangguan perfusi jaringan

    Pengambilan O2 Gangguan pola nafas

    Kelelahan

    Defisit perawatan diri

    Resiko tinggi infeksi

    Intoleransi aktivitas

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