HMB273

ADENOSINE TRIPHOSPHATE

McArdle, Katch & Katch

Pages 138-141, 379-381

100m – Ben Johnson Seoul 1988

Split Time
(m) (sec) 12

10 1.83 10
20 2.87
Velocity (m/s)

30 3.80 8

40 4.66 6
50 5.50
60 6.33 4

70 7.17 2
80 8.02
90 8.89 0
10 20 30 40 50 60 70 80 90 100
100 9.79
Distance (m)

1
Why does the velocity decrease?
13

12
Velocity (m/s)

11

a. The neural signal to the muscle slows

10
b. Muscle cross-bridge cycling slows
c. The drugs Ben was taking have run out
9

8
20 30 40 50 60 70 80 90 100

Distance (m)

EVENTS LEADING TO MUSCLE ACTION

Fig 18.14

2
ATP – IMMEDIATE SUPPLY

Fig 6.5

ATP AND PCr DURING SPRINTING

3
Why does the velocity decrease?
13

12
Velocity (m/s)

11

a. The neural signal to the muscle slows

10
b. Muscle cross-bridge cycling slows
c. The drugs Ben was taking have run out
9

8
20 30 40 50 60 70 80 90 100

Distance (m)

Why does the velocity drop?

13

12
Velocity (m/s)

11

10

8
20 30 40 50 60 70 80 90 100

Distance (m)

The rate (speed) with which ATP can be resynthesised (made

available) determines the rate at which ATP can be used for
muscle shortening

4
Learning Objectives
1. Describe skeletal muscle contraction from the release of ACh
to excitation-contraction coupling (revision)
2. Identify the high-energy phosphates and discuss their
contributions to powering biologic work
3. Quantify the body’s reserves of adenosine triphosphate
(ATP) and phosphocreatine (PCr) and give examples of
physical activities in which each of these energy sources
predominates