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MiKaHe Gbi cami HaryrMnn, cerania 8 2:25 tOnio | Oreename tOnnit asna0a | Powat, Tak HME BBHAY “TO B HecKONBIGIX GONBLIMK M CONAAHEX SANAMHBIX Hay nnyGnnnaunsx -oGs0pax MeToROS neHEHHNA AN M ap 322014 w 2015 ro He GeiTO HM anHOrO ynownnarns o abxpecmsrocrn nannkrae C al. pep ulna ToBKO © MOHOTEpATIAH HE commurreneHocm.8 mory alin sr nyGnnkauin.no MoeMy QaMol coasTop Dade Cancor.sar0 Ha sanage “2cTo uTApytOr oHOFO AOKTOpa ,KOTOpEAi NHCAN CTaTeH O AN ewe 8 1975 rony : combination of an SSRI and Modafinil “the hidden pearl that can really help depersonalization disorder”.Dr. Evan Torch. flo Mom HaGnioneHsM Ha cbopyMaX SaMAnHBIK- MHOTME OTSEIBGIOTCA C BOCTOPOM,O TOM {TO HM MrHOBEHHO NoMor Suboxone. 9 Poany 1 tOnnii Dapigos | Potal, KCTaTH .HaCHET NPOKOTHITTHBHBIX CBOVICTS LCT Takan CTpaeHian Hay'tian nySnMNauNna soT ee saxnoenie :Finally, when the team gave low doses of clonazepam (Klonopin), which modulates the GABA neurotransmitter system employed by FS interneurons, to mice lacking DS ‘and Db, the mice agan performed the rule-shifting task normally. The cognitive improvements following direct interneurcn stimulation persisted for a week after the ‘experiments, suggesting that targeting FS interneuron dysfunction may result in durable improvements in cognitive function in schizophrenia. ‘Clonazepam and other GABA-modulating drugs such as lorazepam (Ativan) and diazepam (Valium) are now used to treat anxiety assodated with schizophrenia, but usually at higher doses that also increase sedation, which may mask any cognition-enhancing effect. Lower doses, or better-designed compounds that specifically target FS interneurons in the prefrontal cortex, might be better options, Sohal said, adding that measuring gamma oscillations may be 2 useful quide in developing these approaches. corona 5 6:15 Pomanty 1 Pomal Bexkep JOnni, 370 one over rny6owas Teopus. A pai TaKcEA, M70 KnoWaSenaM XOPOUIO TAK NYTAT To KoFHIATHBKe B KTUHUECKEA TpUMeHBEMEY N32 tOnito | reer HK a Powart Bexxep (Onnil, @ He BvOky HinkaKinx CBexvNx "O630N08 METOROB NeHEHMA JIM ZIP" 3a 2014 W 2015. CTaTEM (CEEKIE MO TEME BVPKY, CEEK/N HMEHHO CO3008 HE BIDKY. FOKAIKH? To 4TO He YNOMMaIOT KOMBWHaUWO ~ He SHAMITT 4TO He PabOTaeT. MOF 3A6bITS Cel GhaKT B ‘0630p. KPHMTUUECKOrO-To YNOMMHGHH® TOXE HET. ATO TO MOHOTEDANHIA COMHIFTETBHS ~ TAK 370 HIN OBUIEe MECTO Alacra Crmeou ynommiaer naMoTpHANHMH cpenit MeTOROS neueHHI [I s coe kHUre 2013 rons. seca Hasag 1Onno | OrsenaTe tonnit aenaos Powatt, a coseTarine HeOBpaTHMbIX VIMAO ¢ aMAKTANOM -HMEET NOTEHUAT? Tpit \aca Hasag Poway a Onnii Daengos |) Powalt, nowyxan.tanpnvep There is no known pharmacotherapy for the treatment of Doo. 13 The Iterature dudes tnals of domipramine, selective serotonin reuptake inhibitors, and lamotrigine. 12,13 Research using selective serotonin reuptake inhibitors in patients with DDD has shown that serotonin agonists such as metachlorphenylpiperazine ‘can induce symptoms of depersonalization and/or derealizationt . Sierral2 suggested possible agents may indude opioid ‘agonists, N-methyl-D-aspartate (NMDA) agonists, and serotonin 2 agonists. No efficacy for any of these medications or dasses was proven. More research is needed as the evidence is incondusive. Tpit vaca Hasag Powaty ie Onn Dapigos H) Powan, http://www.ncbi.nim.nih. gov/pmc/artices/PMC4204471/p.. sa vaca Hasan PomaHy Pomax Bexxep fOnnit, weer. Aba vaca Hasag tOnm | OreraTb Powal Bexxep Oni. 06309 HenowHeii. TyT HET HanpHMep HU cnoa npo KnodasenaM W HM CcBa Mo aTUNUKH. Minn cnosa npo manacusun. 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