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  • Pa Tho Physiology of Gastritis

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    Excessive alcohol intake Prolonged use of NSAID's Traumatic injury Infections Severe physiological stress Irritation of gastric mucosa Non-modifiable Age (55 years) reduction in prostaglandin synthesis which is responsible for the protection of the mucosal from the effects of the gastric acids. Gastric mucous membrane undergoes superficial obstruction Disruption of capillary walls with loss of plasma Ulceration Gastric glands responsible for the production of acid

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    Excessive alcohol intake Prolonged use of NSAID's Traumatic injury Infections Severe physiological stress Irritation of gastric mucosa Non-modifiable Age (55 years) reduction in prostaglandin synthesis which is responsible for the protection of the mucosal from the effects of the gastric acids. Gastric mucous membrane undergoes superficial obstruction Disruption of capillary walls with loss of plasma Ulceration Gastric glands responsible for the production of acid

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    26 views1 page

    Pa Tho Physiology of Gastritis

    Uploaded by

    gerald_ichigo402
    Excessive alcohol intake Prolonged use of NSAID's Traumatic injury Infections Severe physiological stress Irritation of gastric mucosa Non-modifiable Age (55 years) reduction in prostaglandin synthesis which is responsible for the protection of the mucosal from the effects of the gastric acids. Gastric mucous membrane undergoes superficial obstruction Disruption of capillary walls with loss of plasma Ulceration Gastric glands responsible for the production of acid

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    of 1

    PATHOPHYSIOLOGY OF GASTRITIS

    Predisposing Factors

    Modifiable Non-modifiable
     Excessive alcohol intake  Age (<55 years
     Prolonged use of NSAID’s
     Traumatic injury
     Infections
     Severe physiological stress
     Bile reflux following gastric
    surgery

    Irritation of gastric
    mucosa
    Reduction in prostaglandin synthesis which is
    responsible for the protection of the mucosa from the
    effects of the gastric acids

    Stomach tissue unprotected from auto digestion of hydrochloric acid


    and pepsin
    Gastric mucous membrane becomes
    edematous
    Gastric mucous membrane undergoes superficial
    obstruction

    Disruption of capillary Gastric glands responsible for the


    walls with loss of plasma production of acid is damage due to

    Ulceration Scanty amount of gastric


    juices is secreted with very

    If If not
    treated: treated:

    Patient recovers within GI bleeding


    24 hrs although the
    appetite maybe Shock
    diminished for an
    additional of 2-3 days.
    Death

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