Antagonism The use of antagonistic microorganisms to control fungal pathogens is very limited because very few of the species

which showed potential under experimental conditions were successful in the guava field.

Possible mechanisms of antagonism, including antibiosis, hyperparasitism, competition for volatile and nonvolatile nutrients, interference with pathogenesis, physical modification of wounds, and induced resistance.
Antibiosis is the process of secretion of anti-microbial compounds by antagonist fungi to suppress and/or kill pathogenic fungi in the vicinity of its growth area hyphal penetration was absent, suggesting that mycoparasitism was not the sole cause for the observed inhibitory effects so the skeleton of filamentous fungi cell walls

contains chitin, glucan and proteins, enzymes that hydrolyze these components have to be present in a successful antagonist in order to play a significant role in cell wall lysis of the pathogen by of -1,3
glucanases, chitinases, lipases, proteases and xylanases volatiles, extracellular enzymes and/or antibiotics endochitinases, exochitinases and 1,4--N-acetylglucosaminidases,

There are several mechanisms involved in fungal antagonism namely antibiosis whereby the antagonist fungus shows production of antibiotics; competition for nutrients; and mycoparasitism whereby aspergillus sp directly attacks the plant pathogen by excreting lytic enzymes such as chitinases, -1,3 glucanases and proteases

These data indicated that the antifungal potential of the exponential culture filtrate was mainly due to the presence of extracellular chitinase enzyme, whereas the antifungal activity of the stationary culture filtrate involved the action of unknown thermostable antifungal compound(s).

classified the mycoparasitic interactions as: (1) replacement (unilateral antagonism), (2) deadlock (mutual antagonism), and (3) intermingling (no antagonism), with lack of explanation at microscopic level. There are

several mechanisms involved in fungal antagonism namely antibiosis whereby the antagonist fungus shows production of antibiotics; competition for nutrients; and mycoparasitism whereby aspergillus sp directly attacks the plant pathogen by excreting lytic enzymes such as chitinases, -1,3 glucanases and proteases