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  • Devesh Mishra Pathology

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    Nishesh Tiwari
    5K views423 pages
    - Keratomalacia due to vitamin A deficiency and myositis ossificans are mentioned. - Intestinal columnar cells, cardiac muscle, skeletal muscle and nerves cannot undergo cell division and will only show hypertrophy, not hyperplasia. - Cellular markers like ALT and AST may leak from tissues during conditions like liver cirrhosis.

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    Pathology by Devesh Mishra

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    - Keratomalacia due to vitamin A deficiency and myositis ossificans are mentioned. - Intestinal columnar cells, cardiac muscle, skeletal muscle and nerves cannot undergo cell division and will only show hypertrophy, not hyperplasia. - Cellular markers like ALT and AST may leak from tissues during conditions like liver cirrhosis.

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Flag for inappropriate content
    5K views423 pages

    Devesh Mishra Pathology

    Uploaded by

    Nishesh Tiwari
    - Keratomalacia due to vitamin A deficiency and myositis ossificans are mentioned. - Intestinal columnar cells, cardiac muscle, skeletal muscle and nerves cannot undergo cell division and will only show hypertrophy, not hyperplasia. - Cellular markers like ALT and AST may leak from tissues during conditions like liver cirrhosis.

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 423

    - keratomalacia d/t vit A deficiency

    - myositis osificans

    5
    intestinal columnar
    cells

    - keratomalacia d/t vit A deficiency

    not a part of cell adaptation.


    exception- bening prostatic hyperplasia
    exception- apocrine metaplasia

    - cardiac muscle , skeletal muscle, nerve cant undergo cell division.


    SO WILL ONLY SHOW HYPERTROPHY NO HYPERPLASIA
    1.
    2

    thus decreased protein synthesis


    - cellular markers leaked
    e.g.- ALT,AST in liver cirrhosis

    methemoglobinemia CO poisoning
    abscess
    pancreatitis-enzymes from pancrease digest parenchyma
    tombstone appearance of coagulative necrosis normal renal parenchyma

    wedge shaped pale infarction


    wet gangrene

    varients of coagulative necrosis

    dry gangrene

    caseous necrosis

    also seen in PIH-


    placenta shows
    fibrinoid necrosis
    apoptotic cell

    eosinophilic cytoplasm
    shrinkage of nucleus
    pro

    bcl2 protects cytochrome c from


    leaking out of cell.
    now bcl2 is lost, cell membrane is
    damaged and cyt c leaks out.
    associated death domain)

    1. 2.
    ?
    MI

    - CAUSES FATTY LIVER

    - SEEN IN MI

    ?
    ?
    and?
    ?

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