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Subject XXVI. Quarantine infections. Sepsis.

To learn macroscopic specimens:

65. Hemorrhagic pneumonia.


In the dissection of tissue of lungs the dense air-free dark red lesions places merging with each other are
visible.

224. Sero-hemorrhagic inflammation of an intestine at malignant anthrax (plate).


The centres of sero-hemorrhagic inflammation are visible on the mucosa of intestine.

7. Moulage. A malignant anthrax: an anthracic carbuncle (hemorrhagic inflammation).


On the skin of face the ulcer of the spherical form up to 2,0 cm in diameter, dark red colour, at centre by
which one 3 whitish lesions, on peripherals zone of oedema is visible.

10. Moulage. A smallpox.


The polymorphic papulo-vesicular viral rash with haemorrhages and black crusts are visible on skin of the
person.

301. Bacterial thrombotic endocarditis of the aortal valves.


The vegetations are located on the ventricular surface of the semilunar valves. They begin from the
contact areas of the valve and extend along the surface of the valves and on to the adjacent endocardium.

331. Septic (bacterial) endocarditis of the aortal valves.


Vegetations reminding polyps are visible on two semilunums of the aortal valve on a place of ulceration.

309. Purulent meningitis.


The gyri of brain impressed, in sulci cream-like pus of yellow-white colour places. Blood vessels of pia
mater are congested.

321. Abscess of cerebellum.


In the dissection of cerebellum the cavity up to 1,5 cm is visible, the walls are covered by friable dim
grayish tissue.

166. Purulent hemorrhagic pneumonia.


The multiple centres of purulent inflammation are visible on cut section of an airless reddish lungs tissue.

332. Purulent inflammation and purulent thrombophlebitis of the stump of femur.


The extensive ulcer with purulent dull edges and grey dirty bottom is visible on the skin of femur stump.
There is occlusive thrombus with purulent fusion in lumen of the vessel.

124. Septic hyperplasia of spleen.


The spleen enlarged in the sizes, on a cut section the pulp is quaggy flabby (gives a plentiful scrape).

518. Subacute hyperplasia and ischemic infarct of spleen at septic endocarditis.


The spleen is enlarged in the sizes, dense and with an ischemic infarction.

525. Chronic pneumonia with abscesses.


The lobe of lung is firm with cords of connective tissue. The cavities (abscesses) with thick walls are
visible in depth of lung with bands of sclerosis around it. The pleura is thickening.

15. Chronic nephritis.


Surface of a kidney granular, motley view.
Study of microscopic specimens:

45. Hemorrhagic pneumonia.


The erythrocytes are predominated in alveolar exudates. Non affected sites of lungs are inflated
(emphysema).
To indicate in the figure:
Hemorrhagic exudates in alveoli.

173. Serous inflammation of oesophagus at a smallpox.


There are bubbles in the submucous layer of oesophagus vesicles containing light serous exudates and
simple leucocytes. The multiplayer flat epithelium by places is desquamated.
To indicate in the figure:
1 bubbles,
2 desquamation of an epithelium.

100. Bacterial endocarditis at a sepsis.


The valve are thickened by the proliferation of young connective tissue cells. A clump of leucocytes is
visible, in centre of the valve. The thrombotic masses from fibrin and colonies of microorganisms are
visible on a surface of the valve.
To indicate in the figure:
1 thrombus,
2 valve,
3 leucocytes,
4 colonies of microorganisms.

61. Cellulitis (Phlegmon).


Diffuse infiltration of a fatty tissue predominantly by neutrophilic leucocytes.

58. Purulent meningitis.


The piamater of spinal cord of the child are soaked with purulent exudates, containing a large quantity of
leucocytes.

36. A pneumonia with abscesses.


The multiple centres of fusion abscesses are visible in the pulmonary tissue on a background of purulent
inflammation.

The literature

1. .., ... Pathological anatomy. 1993, page 492 494, 497 498, 520 - 525.
2. Harsh Mohan .Textbook of Pathlogy. 4th Edition, page 92, 131, 163-165, 308-311, 759-760, .
3. Robbins. Pathologic basis of disease. 1999, page 134 135, 357-358, 387-388, 572 - 576.

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