Nodule iit: St y/o Asymptomatic Male
‘The riame and date have been verified
- Previous films ive been obtained for
t comparison. This is a PA view. The patient has
tken a nearsadequate inspiration. There are 8
visible posterior ribs inthe right hemithorax.
‘The film is slightly underpenetrated but there
is adequate lung detail. The trachea is midline
and the clavieles are equidistant from the
trachea. The patient is well-positioned,
b
‘What can be seen of the soft tissues and bony
structures appears to be symmetric. There is no
calcification or mass. There is no bony
deformity, fracture, lytic lesion or dislocation,
‘The mediastinum is narrow with no obvious
deferities
‘There is a nodular tesion adjacent to the
heart in the left hemithorax. The nodule has
white, punctated deasities that represent
calcifications. The densities are distributed
in peculiar “popcorn” pattern. It
measures 6 em in size. The nodule does not
obliterate the heart border,
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‘The width of the heart is less than 30% of the width of the ribcage. ‘The hemidiaphragm margins are sharp, clear and at &
reasonable height. There is o evidence of pleural fluid
‘There are no visible sifhouette signs,
air-beonchograms or fluid menisci. There is no.
‘evidence of atelectasis or lobar collapse.
‘On the lateral view, this large module is seen
beneath the oblique fissure. is
‘well-circumscribed, with sharp margins and
1 spiculations. It is 6 cm in diameter.
Based on our CXR findings we can make the
diagnosis of @ large wodule ln the superior
Segment of the left lower lobe. The
‘calcification, smooth borders and
wellcircumscribed features suggest that this
és a benign lesion. The “popeorn” patiern of
‘calcification is consistent with a hamartoma
This hamartoma is quite targe as most are
Jess than 4-cmt in diameter, but this lesion bs 6
‘em. The character of the calcification is what
suggests the diagnosis of hamartoma.
‘Remember ta correlate these findings with the
differential diagnosis from the Nistory and
physical (ériengutation)!!Aelectasis #1: 25 y/o female with non-productive cough and wheezing
‘The name and date have been verified. Previous films: have been
‘obtained for comparison. The first film is a PA view. The patient has
‘taken an adequate inspiration. There are 9 visible posterior ribs in
‘the right hemithorax. The film is well penetrated with adequate lung
detail, The trachea is midline and the clavieles are equidistant from
‘the trachea. The patient is well-positioned,
‘The soft tissues and bony structures are symmetric. There is no
‘calcification or mass, There is no bony deformity, fracture, lytic
lesion or dislocation.
‘The mediastinum is narrow with no deformities. The width of the
heart is less than 50% of the width of the ribcage,
‘There is an area of increased density om the left side of the heart
shadow which blurs the edge of the descending thoracic aorta
(silhouetie sign). Also, if you look carefully, you will note that
the feft mainstem broschus and the left hilar vessels are
displaced downwards. This may be easier te see if you compare with the right side,
‘The medial portion of the left hemidiphragm Is obscured (silhouette sign). The right hemidiaphragm margins are sharp are
clear, The diaphragms are at a reasonable height. There is no evidence of pleural fluid,
‘The left upper lung appears “blacker” than usual with increased spreading of the vessels. There ane no visible,
air-bronchograms, nodules oF fluid menisci.
On the Lateral view, the left posterior diaphragm is obseured which is
consistent with our findings on the PA (silhouette sign).
Based on our CXR findings we cam make the diagnosis of atelectasis of
the left lower lobe. The collapsed lobe is contiguous with the mediat
(posterior) diaphragm and the descending thoracic aorta, The collapse
leads to shift of the left mainstem bronchus and left hilar vessels as well as
hyperacration of the left upper lobe.‘A'59-year-old woman with hypertension and diabetic nephropathy presented with a sudden onset of dyspnoes after
discontinuing her medications, Physical examination revealed hypenension (BP 225/122 mmlg), tachycardia (HR. 112 bpm).
tachypnoes (24 breaths per minute), and hypoxemia (Sa02 = 94% despite treatment wit supplementary oxygen). The patient
also had elevated IVP, bilateral rales, and oedema of the legs. The level of BNP was elevated (780.8 pg/ml. Normal level <
18.4). A chest radiograph showed an enlarged cardiac sihouctter a dilated azygous vein adn peribronchial euffing, in addition
to Kerley's A, B, and C lines:
Kerley A lines (arrows) are linear opacities extending form the periphery to hila, they are caused by distention of anastomotic