LI 2 : relation of stoke and sensory impairment

Common disorders that occur after a stroke are loss of sensation or sensory disturbance.
Information received through sensory receptors is called a somatic sensation, which includes
Exteroception and proprioception (Sherwood, 1997). Exteroception is sensory information about
the external environment received from receptors on the skin and subcutaneous tissue
(O'Sullivan and Schmitz, 1988). Exteroception is responsible for pain perception, temperature,
light touch and pressure. Proprioception is the awareness of body position in space and time,
through receptors in muscles, tendons, ligaments and fascia (Sherwood, 1997).

Although sensory disorders occur a lot, sensory disturbances can be restored with lots of
exercise, for example physiotherapy, so that it can be said that sensory function can be cured.
(Carey, 1993)

Anamnesis in patients with "numbness" is very important. First of all, as is the case with most
neurological complaints. We must determine what the patient means by "numbness." Some
patients describe loss of sensitivity (anesthesia or hypesthesia) or aberrant sensation
(paresthesia), which is often described as tingling. The loss of actual sensitivity is more likely to
represent true damage to the sensory pathways in the nervous system, while paresthesia has a far
wider difference. Dysesthesia, which is the perception of unpleasant sensations (often burning)
or allodynia (harmless perception of pain as pain) can occur due to damage to the nervous system
or, more generally, a manifestation of the underlying pain condition.

Sherwood, L. 1997. Human Physiology: Foam Cells to System 3rd. Belmont. CA: Wadsworth.

O’ Sullivan, SB, Schmitsz, TJ. 1988. Physical rehabilitation: Assesment and treatment.
Philadelphia. FA Davis.

Carey, JR. 1993. Applied Demography for Biologists: with Special Emphasis on Insects. New
York. Oxford University Press.

Swenson, Rand. 2008. Disorders of the Nervous System. -. Dartmouth Medical School.