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suffer respiratory distress syndrome, meconium aspiration and sepsis compared to inborn
neonates. Similar adverse outcomes, including longer LOS, greater duration of total parenteral
nutrition (TPN) andslower introduction of full enteral feeds, were also identifiedin outborns by
Kitchanan et al. [83] in a review of 21patients with AWD admitted to their unit in Queensland,
Australia.
The intestine is frequently foreshortened, covered with gelatinous exudates, matted together,
and/or is edematous due to its exposure to amniotic fluid and compression of the mesenteric
blood supply at the defect. Usus seringkali lebih pendek, ditutupi dengan eksudat agar-
agar, kusut bersama-sama, dan / atau edematosa karena paparan cairan ketuban dan
kompresi suplai darah mesenterika pada defek.
The principles of management however remain the same. First, to reduce the viscera safely,
second, to close the abdominal wall defect with an acceptable cosmetic appearance and, third,
proper nutrition support, in addition to detection and proper managemen. Namun prinsip-
prinsip manajemen tetap sama. Pertama, untuk mengurangi visera dengan aman, kedua,
untuk menutup cacat dinding perut dengan penampilan kosmetik yang dapat diterima
dan, ketiga, dukungan nutrisi yang tepat, selain deteksi dan manajemen yang tepat
Initial management aimed at maintaining circulation to the bowel and preventing infection by
covering the defect with sterile dressing soaked in warm saline to prevent fluid loss, stabilizing
infant (temperature/fluids), gastric decompression, intravenous . Manajemen awal bertujuan
menjaga sirkulasi ke usus dan mencegah infeksi dengan menutup cacat dengan
pembalut steril yang direndam dalam larutan garam hangat untuk mencegah kehilangan
cairan, menstabilkan bayi (suhu / cairan), dekompresi lambung, intravena
Preterm delivery of infants with gastroschisis was recommended by some researchers to avoid
the intestinal damage that may occur due to prolonged exposure to the amniotic fluid, which
contain inflammatory factors that lead to intestinal ischemia or damage [17,18]. In contrary,
Maramreddy et al. [19] reported that there is no benefit of preterm delivery in reducing the
morbidities in patients with gastroschisis. In addition, preterm delivery increased complication
rate in those infants with regard to sepsis, longer hospital stay, and prolonged period to
establish feeding and to tolerate full feeding.
DAFPUS
Holland, A. J., Walker, K., & Badawi, N. (2010). Gastroschisis: an update. Pediatric surgery
international, 26(9), 871-878.
Cassandra Kelleher, Jacob C. Langer. Congenital Abdominal Wall Defects. J. Patrick Murphy
George W. Holcomb. Ashcraft's Pedatric Surgery 5th edition. Philadelphia : Saunders Elselvier,
2010: 625-36.
Mortellaro VE, Peter SD, Fike FB, Islam S. Review of the Evidence on the Closure of abdominal
Wall Defects. Pediatr Surg Int. 2010.
Valente, L., Pissarra, S., Henriques-Coelho, T., Flor-de-Lima, F., & Guimarães, H. (2015).
Gastroschisis: factors influencing 3-year survival and digestive outcome. Journal of Pediatric
and Neonatal Individualized Medicine (JPNIM), 5(1), e050114.
Molik KA, Gingalewski CA, West KW, Rescorla FJ, Scherer LR, Engum SA, Grosfeld JL.
Gastroschisis: a plea for risk categorization. J Pediatr Surg. 2001;36(1):51-5.
Harris EL, Minutillo C, Hart S, Warner TM, Ravikumara M, Nathan EA, Dickinson JE. The long
term physical consequences of gastroschisis. J Pediatr Surg. 2014;49(10):1466-70.
Gamba, P., & Midrio, P. (2014, October). Abdominal wall defects: prenatal diagnosis, newborn
management, and long-term outcomes. In Seminars in pediatric surgery (Vol. 23, No. 5, pp.
283-290). WB Saunders.
Minutillo, C., Rao, S. C., Pirie, S., McMichael, J., & Dickinson, J. E. (2013). Growth and
developmental outcomes of infants with gastroschisis at one year of age: a retrospective study.
Journal of pediatric surgery, 48(8), 1688-1696.