Professional Documents
Culture Documents
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Advanced Life Support Training Manual
MOH/P/PAK/347.17(GU)
ISBN 978-967-0769-85-1
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content
Foreword
Director General of Health Malaysia i
Deputy Director General of Health (Medical) ii
Director Medical Development Division iii
Chapter 1-11
Chapter 1
Course Overview 1
Chapter 2
The Systematic Approach 3
The BLS Primary Survey 3
The ALS Secondary Survey 4
Chapter 3
Team Dynamics 6
Chapter 4
Airway Management & Ventilation 8
Overview of Airway Management 8
Oxygen Delivering Devices 9
(nasal cannula/simple face mask/venturi mask
/mask with O2 reservoir)
Non-invasive Airway Devices 12
(oropharyngeal airway/nasopharyngeal airway)
Manual Assist Ventilation 13
(mouth to mask ventilation/bag-mask ventilation)
Advanced Airway 15
(supra-glottic airways eg: LMA/ETT)
Waveform Capnography 25
Tracheobronchial Suctioning 27
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Chapter 5
Defibrillation and Electrical Therapy 28
What is Defibrillation? 28
The Importance of Early Defibrillation 28
Defibrillators 28
Preparing the Patient 29
Safety Issues 30
Synchronized Cardioversion 32
Ventricular Tachycardia 32
Refibrillation versus Refractory VF 33
Pacing 33
Summary 33
Chapter 6
ALS Core ECG Rhythms and Recognition 34
Chapter 7
Drugs in Resuscitation 43
Chapter 8
ALS Algorithms 50
Chapter 9
Resuscitation in Special Situation 53
Chapter 10
Post Resuscitation Care 57
Chapter 11
Ethics of resuscitation and end of life issues 60
Appendix
Skill Station Competency Checklist 64
National Committee on Resuscitation Training (NCORT) 70
Foreword
Advanced Life Support Training Manual
Advanced Life Support Training Manual
Training to ensure,
the continuing
updated teaching of
Advanced Life
Support in line with
the international
community
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understanding and
care
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quality
cardiopulmonary Thank you.
resuscitation and
post-resuscitation
care
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Chapter
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Advanced Life Support Training Manual
Course Overview
The Advanced Life Support Course aims to train doctors and healthcare providers working in
critical care areas in the resuscitation of patients beyond the ABC of resuscitation.
The course emphasizes on enhancing your skills in the treatment of arrest patients through
active participation in a series of simulated cardiopulmonary cases. These simulations are
designed to reinforce important concepts, including
Course Objectives
Upon completion of this course, you should be able to
Course Description
The course concentrates on skills both individually and as part of a team. Lectures are short and
few. Therefore you are expected to have read the ALS provider training manual before the course.
In addition, strong BLS skills are the foundation of ALS. You must have passed the 1-rescuer
BLS/Automated External Defibrillator (AED) course before enrolment into the ALS course. The
course programme is as follows:
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Course Overview
The Advanced Life Support Course aims to train doctors and healthcare providers working in
critical care areas in the resuscitation of patients beyond the ABC of resuscitation.
The course emphasizes on enhancing your skills in the treatment of arrest patients through
active participation in a series of simulated cardiopulmonary cases. These simulations are
designed to reinforce important concepts, including
Course Objectives
Upon completion of this course, you should be able to
Course Description
The course concentrates on skills both individually and as part of a team. Lectures are short and
few. Therefore you are expected to have read the ALS provider training manual before the course.
In addition, strong BLS skills are the foundation of ALS. You must have passed the 1-rescuer
BLS/Automated External Defibrillator (AED) course before enrolment into the ALS course. The
course programme is as follows:
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Day 1
Day 2
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• Check for responsiveness before shouting for help and open the airway
• Check breathing before starting chest compressions
• Attach an AED, then analyze for a shockable rhythm before delivering a shock
Remember:
Assess...then perform appropriate action.
Table 1 below shows an overview of BLS Primary Survey. DRS is included before ABCD for
completeness.
Assess Action
Danger
Wear PPE (gloves, apron, mask) if available
- Are there blood spills, sharps, electric Make sure you, the victim and
wires? bystanders are safe
- Is the scene dangerous?
‘Emergency! Emergency!
Shout for help Call ambulance 999 or bring emergency
trolley & defibrillator if available
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Advanced Support
Life Training
Support Manual
Manual
Training
Assess Action
NB. Make every effort to minimize interruptions in chest compressions. Limit interruptions in chest
compressions to no longer than 10s
Avoid:
• Prolonged rhythm analysis • Frequent or inappropriate pulse checks
• Taking too long to give breaths • Unnecessarily moving the patient
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Differential Diagnosis ● Search for, find and treat reversible causes (i.e.
- Why did this patient develop
definitive care). Look for 5H and 5T causes.
cardiac arrest?
● 5H: Hypoxia, Hydrogen ion, Hypothermia,
- Why is the patient still in arrest?
Hypovolemia, Hypo/hyperkalemia.
- Can we identify a reversible cause
● 5T: Tamponade (cardiac), Tension pneumothorax,
of this arrest?
Thrombosis (pulmonary or coronary), Toxins
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Roles
Team Leader
Organizes the group, monitors individual performance of team members, backs up team members,
models excellent team behavior, trains and coaches, facilitates understanding and focuses on
comprehensive patient care.
Team Member
Must be proficient to perform skills within their scope of practice. They are clear about their role
assignment, prepared to fulfill the role responsibilities, well-practiced in resuscitation skills,
knowledgeable about the algorithms and committed to success.
When communicating with team members, the leader should use closed loop communication.
The leader gives an order or assignment and then confirms that the message is heard. The team
member verbally repeats the order to confirm that the order or assignment is heard and informs the
leader when the task is complete.
Clear Messages
All messages and orders should be delivered in a calm and direct manner without yelling or shouting. The team
leader should speak clearly while the team members should question an order if they are unsure what is said.
Every member of the team should know his/her role and responsibilities. To avoid inefficiencies, the team
leader should clearly delegate tasks. A team member should not accept assignments above his/her level of
expertise.
Every member of the team should know his/her imitations and capabilities and the team leader should be
aware of them. A new skill should not be attempted during the arrest, instead call for expert help at an early
stage.
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Knowledge Sharing
A critical component of effective team performance is information sharing. The team leader can ask
for suggestions when the resuscitation efforts seem to be ineffective.
Constructive Intervention
During a code, a team leader or member may need to intervene if an action is about to occur at an
inappropriate time. For example, the person recording the event may suggest that adrenaline be
given as the next drug because it has been 5 minutes since the last dose. In actual fact the adrenaline
should be repeated every 3 to 5 minutes. All suggestions for a different intervention or action should
be done tactfully by the team leader or member.
An essential role of the team leader is monitoring and reevaluation of the status of the
patient, interventions that have been done and assessment findings.
Mutual Respect
The best teams are composed of members who share a mutual respect for each other and work
together in a collegial, supportive manner. All team members should leave their egos at the door.
Team debriefing
A team debriefing could be done, led by the Team leader after the resuscitation has been
completed and the patient has been admitted to a critical care ward to discuss pertinent issues that
occurred during the resuscitation and reinforce teaching points among members.
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The support of ventilation and oxygenation during CPR and peri-arrest period is important. The
purpose of ventilation during CPR is to maintain adequate oxygenation and sufficient elimination of
carbon dioxide. Airway management during resuscitation is dependent on patient factors, the phase of
resuscitation (during CPR or after ROSC) and the skill of the rescuers.
A variety of airway management modalities e.g. bag valve mask (BVM), supraglottic airway devices
(e.g. LMA,SUPREME,Igel) and endotracheal tube (ETT) are often used during resuscitation as a part of
stepwise approach to airway management. After ROSC, ultimately an endotracheal intubation is
needed for the post resuscitation care.
1. When supplementary oxygen is available, use the maximal feasible inspired oxygen
concentration during CPR. The detrimental effects of hypoxia during low cardiac output state
(CPR) supersede hyperoxia that may exist in the immediate post cardiac arrest period.
2. After ROSC, titrate the inspired oxygen concentration to achieve the SpO 2 in the range of 94 –
98%.
1. There are various modalities for managing the airway during resuscitation. It depends mainly
on the rescuer skill and the familiarities of the technique to provide effective oxygenation and
ventilation. Options includes bag mask ventilation (BMV), supraglottic airway devices (SGAs)
and endotracheal intubation.
2. There is inadequate evidence to show the difference in survival or favorable neurological
outcome with the use of bag mask devices (BMV), supraglottic airway devices (SGAs) and
endotracheal tube (ETT). Either BMV or an advanced airway (SGAs,ETT) may be used for
oxygenation and ventilation during CPR. Advanced airways should be inserted if the rescuer is
familiar with the devices and technique of insertion.
3. Supraglottic airway devices e.g. LMA, Supreme, I-gel are relatively easier to insert in
comparison to endotracheal intubation. Interruption to chest compression should be
minimized during insertion of an advanced airway.
4. Once an advanced airway has successfully been inserted, cyclical CPR (30:2) should be stopped.
The ventilation rate should be 10 breaths per minutes (1 breath every 6 seconds) while chest
compression being performed at the rate of 100 – 120 per minutes.
5. Delivery of oxygen through a cannula or surgical cricothyrodotomy may be lifesaving in CICO
conditions (Cannot Intubate Cannot Oxygenate).
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Oxygen supplement is always appropriate for patient in respiratory distress. There are various devices
that can deliver supplemental concentration from 21% - 100%.
Table . Delivery of Supplemental Oxygen: Flow Rates and Percentages of Oxygen Delivered.
Venturi mask
4-8L/minute 24 - 40
(Device specific)
10-12L/minute 40 - 50
Mask with O2
Reservoir
• Rebreathing 10-15L/minute 70 - 80
• Non-rebreathing 10-15L/minute 95 - 100
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Nasal Cannula
• Consists of 2 prongs
• Every 1L/minute increase in O2 flow rate increase in FiO2 by 4%
• Usually 1-6L/minute O2 given
• Do not use more than 6L/minute O2 as this does not increase oxygenation much, yet dries up nasal
passages and is uncomfortable to patient
• O2 concentration depends on: - O2 supply flow rate
- Pattern of ventilation
- Patient inspiratory flow rate
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Venturi Mask
• Based on Bernoulli principle
- O2 is passed through a narrowed orifice and this creates a high-velocity stream of gas. This
high-velocity jet stream generates a shearing force known as viscous drag that pulls room
air into the mask through the entrainment ports on the mask.
• Gives desired concentration of oxygen to patient (24% to 60%)
• Ideally used for patient with Chronic Obstructive Pulmonary Disease (COPD)
• The addition of a reservoir bag to a standard face mask increases the capacity of the O2
reservoir by 600 to 1000 ml. If the reservoir bag is kept inflated, the patient will inhale
only the gas contained in the bag.
• There are two types of mask-reservoir bag devices:
• No valve and so gas exhaled in the initial phase • Presence of a one-way valve that prevents any
of expiration returns to the reservoir bag exhaled gas from returning to the reservoir bag
• Provides up to 70% to 80% O2 with flow rates of • Provides up to 95% to 100% O2 with flow rates
10 - 15L/minute. of 10 - 15L/minute.
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Oropharyngeal Airway
• A semicircular tube to hold the tongue away from the posterior wall of the pharynx
• Used in comatose patient or patient with loss of airway reflex
• May cause laryngospasm in semi-comatose patient
• Various sizes (3,4,5)
- The appropriate size is measured from angle of mouth to angle of jaw
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Nasopharyngeal Airway
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• Two ways of carrying out mouth to mask ventilation depending on whether there are 1 or 2 rescuers
Bag-mask Ventilation
Ventilation Oxygen Air/Oxygen
Bag Supply Inlet Intake Valve
Connection
Oxygen
Reservoir
Non breathing Valve
Exhalation
Port
Oxygen Supply
Face Mask
Tubing
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Advanced Airways
Bag-mask ventilation is not suitable for prolonged periods of ventilation as it also inflates the
stomach. Therefore, ALS providers should be trained to use advanced airways (supraglottic airway
devices (SGAs) and ETT).
Supraglottic airways are devices designed to maintain an open airway and facilitate ventilation.
Insertion of a supraglottic airway device does not require visualization of vocal cords, therefore can
be done with minimal chest compression interruptions.
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Size 1 < 5 kg 4 ml
Size 1.5 5 to 10 kg 7 ml
Size 2 10 to 20 kg 10 ml
Size 2.5 20 to 30 kg 14 ml
Size 4 50 to 70 kg (adult) 30 ml
Insertion of LMA
Before any attempt to insert an LMA, the following equipment has to be prepared:
The following are the steps necessary for successful insertion of LMA:
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• Inspect interior of LMA airway tube to ensure that it is free from blockage or loose
particles
- Any particles present in the airway tube should be removed as patient
may inhale them after insertion
• LMA can be inserted even if the head is in the neutral position as long as the mouth
opening is adequate
• Avoid LMA fold over:
- Assistant pulls the lower jaw downwards
- Visualize the posterior oral cavity
- Ensure that LMA is not folding over in the cavity as it is inserted
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Tape
Bite Block
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LIMITATION OF SGAs
1. In the presence of high airway resistance or poor lung compliance (pulmonary oedema,
bronchospasm) there is a risk of significant leak around the cuff causing hypoventilation.
The leaks gas normally escapes through the patient’s mouth but some gastric inflation may
occur.
2. No data demonstrating whether or not it is possible to provide adequate ventilation via
SGAs without interruption of the chest compression. Uninterrupted chest compressions are
likely to cause some leaks around the SGAs cuff when ventilation is attempted. Attempt
continuous chest compression initially but abandon this if persistent leaks occur.
3. There is theoretical risk of aspiration of stomach contents; however this complication has
not been documented widely in clinical practice.
4. If the patient is not deeply unconscious, insertion of the SGAs may cause coughing, straining
or laryngospasm. This will not occur in cardiopulmonary arrest patients.
5. If adequate ventilation is not achieved, withdraw the SGAs and re-attempt insertion after
ensuring good alignment of the head and neck.
The ETT was once considered the optimal method of managing airway during cardiac arrest. It keeps
the airway patent, permits suctioning of airway secretions, enables delivery of a high concentration of
oxygen, provides an alternative route for the administration of some drugs, facilitates delivery of a
selected tidal volume, and with the use of a cuff, may protect the airway from aspiration. However,
there is insufficient evidence to support or refute the use of any specific technique to maintain an
airway and provide positive pressure ventilation in resuscitation. Endotracheal intubation should only
be performed by trained personnel with high level of skill and competence.
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Laryngoscope
Endotracheal Tube
Choosing The Correct Size ETT
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The following steps are necessary for successful endotracheal intubation during cardiac arrest:
A B
B
A
Step 2: Preoxygenation
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3A: Laryngoscopy
Use left hand to hold laryngoscope
Enter at right side of mouth and push tongue towards left aside
Move the laryngoscope blade towards midline and advance to the base of the tongue.
Advance the blade to the vallecula if the curved blade is used or to just beyond tip of
epiglottis if the straight blade is used
Lift upward and forward to bring up the larynx and vocal cords into view. The direction of
force necessary to lift the mandible and tongue is 45 degrees. Do not use teeth as a fulcrum
or a lever
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Step 6: Ventilate with a tidal volume of 6-8 ml/kg (visible chest rise) at a rate of 8-10 breath
per minute
Waveform Capnography
Continuous waveform capnography is recommended as the most reliable method of confirming and
monitoring correct placement of the endotracheal tube (ETT). Studies of waveform capnography to
verify ETT position in patients in cardiac arrest have shown high sensitivity and specificity in identifying
correct ETT placement. It can also detect a patient’s deterioration associated with declining clinical
status or ETT displacement.
Confirms ETT placement; note that EtCO2 detection will not differentiate between tracheal
and endobronchial tube placement. Careful auscultation is essential.
Correlates with cardiac index
Assesses adequacy of ventilation
Indicates quality of CPR
Signifies ROSC
Carries prognostic value for survival during resuscitation
Waveform Capnography.
Normal range (approximately 35 to 45 mmhg)
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During intubation Hypoxia from the procedure itself, esophageal intubation and/or
laryngospasm and bronchospasm
Pulmonary aspiration
Hoarseness
Laryngeal granuloma
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Tracheobronchial Suctioning
Suction Catheter
• Size (FG) = ETT internal diameter (mm) x 3/2 or outer diameter should not exceed 1/2 to 2/3
ETT internal diameter
• Minimal trauma to mucosa with molded ends and side holes
• Long enough to pass through tip of ETT
• Minimal friction resistance during insertion through ETT
• Sterile and disposable
Suction Pressure
• 100 to-120mmHg (adults) • 80 to-100mmHg (children) • 60 to-80 mmHg (infants)
Point to note:
In patient with elevated intracranial pressure (e.g. head injury), temporary
hyperventilation before and after suctioning may be indicated
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What is Defibrillation?
• The passage of an electrical current across the myocardium to depolarise a critical
mass of myocardium and enable restoration of coordinated electrical activity
• An electrophysiological event that occurs 30-50 ms after shock delivery-the heart is stunned
and hopefully the sino-atrial (SA) node will take over
• Aims to restore sinus rhythm
• Shock success typically defined as the termination of ventricular fibrillation (VF) within 5
seconds after the shock. Shock success using this definition does not equal to resuscitation
outcome
• Indicated only for VF or pulseless ventricular tachycardia (pVT) where a single shock is given
followed immediately by chest compression without any pulse check or rhythm reanalysis
after a shock.
For every minute that passes between collapse and defibrillation, survival rates from witnessed
VF SCA decrease 7% to 10% if no CPR is provided. When bystander CPR is provided, the decrease
in survival rate is more gradual and averages 3% to 4% per minute from collapse to defibrillation.
CPR prolongs VF, delays the onset of asystole and extends the window of time during which
defibrillation can occur. Basic CPR alone, however, is unlikely to terminate VF and restore a
perfusing rhythm.
Defibrillators
Modern defibrillators are classified according to 2 types of waveforms: monophasic and
biphasic. Monophasic waveform defibrillators were introduced first, but biphasic waveforms are
used in almost all Automated External Defibrillators (AEDs) and most manual defibrillators sold today.
Energy levels vary by type of device and manufacturer.
Although it is recognized that some areas continue to use the older monophasic waveforms,
defibrillation using biphasic waveform are preferred (2015 guidelines). Studies show greater success
in arrhythmia termination.
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Electric/Paddle force
• 8kg in adult
• 5kg in 1-8years when using adult paddles
Transthoracic Impedance
• Use gel pads or electrode paddles or self-adhesive pads to reduce transthoracic impedance.
The average adult human impedance is 70 to 80 Ω. When transthoracic impedance is too
high, a low-energy shock will not generate sufficient current to achieve defibrillation
Electrode/Paddle Placement
• Can be at antero-lateral, antero-posterior, anterior-left infrascapular and anterior-right
infrascapular locations on the chest/back. All these 4 positions are equally effective. For ease
of placement and education, anterolateral is a reasonable default electrode placement.
• Ensure that the paddle and gel or pads are in full contact with the skin
• Special considerations:
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Breasts
- Place lateral pads/paddles under breast tissue
- Move pendulous breasts gently out of the way
Wet Chest
- Briskly wipe the chest dry before attaching electrode pads and attempting defibrillation
Hirsutism
- Shave hirsute males prior to application of pads
- Remove excess chest hair by briskly removing an electrode pad (which will remove some hair)
or by rapidly shaving the chest in that area
Safety Issues
Fire
• Ignited by sparks from poorly applied defibrillator paddles in the presence of an oxygen-enriched
atmosphere
• Avoid defibrillation in an oxygen-enriched atmosphere
• Use self-adhesive defibrillation pads
• Ensure good pad–chest-wall contact
• If manual paddles are used, gel pads are preferable to electrode pastes and gels
because the pastes and gels can spread between the 2 paddles, creating the potential
for a spark
Accidental Electrocution
• Charge paddles after being placed on patient’s chest rather than prior to being taken out
from the defibrillator
• Ensure that none of the rescuer team members is in contact with
patient/victim/resuscitation trolley prior to defibrillator discharge
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• Make sure that no oxygen is flowing across the patient’s chest or openly across the
electrode pads
• Carry out the above steps quickly to minimize the time from the last compression to
shock delivery
When pressing the shock button, the defibrillator operator should face the patient, not the machine.
This helps to ensure coordination with the chest compressor and to verify that no one resumed the
contact with the patient.
No need to use exact words, but a clear and firm warning of about a delivering a shock and everyone
must stand clear of patient. The entire sequence should take less than 5 seconds.
An Example:
• To avoid pre-shock pause of more than 5 sec. Even a 5-10 seconds delay will reduce the chance of
survival. Use of adhesive pad is encouraged to reduce delay.
• To continue giving high quality chest compression for 2 minutes after delivery of shock to improve
coronary perfusion pressure and cerebral perfusion. Only to check the pulse and rhythm after a
complete 2 minutes of cycle of CPR.
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Synchronized Cardioversion
• A shock delivery that is timed (synchronized) with the QRS complex
• Avoids shock delivery during the relative refractory portion of the cardiac cycle when a shock could produce VF
• Indicated in a hemodynamically unstable patient (low blood pressure) with a perfusing rhythm (pulse present)
• Recommended in supraventricular tachycardia due to re-entry, atrial fibrillation, atrial flutter,
and atrial tachycardia
• Recommended in monomorphic VT with pulses
• Not effective for treatment of junctional tachycardia or multifocal atrial tachycardia
Witnessed and monitored patient with VF/pVT,(in catheter lab, coronary care unit, critical
care area where manual defibrillator is rapidly available) 3 quick and successive (stacked )
shocks is recommended. Reason being, the chest compression is unlikely to improve the
already very high chance of ROSC when defibrillation occurs early in the electrical phase,
immediately after onset of VF. If this initial three-shock strategy is unsuccessful, the ALS
algorithm should be followed and these three-shocks are treated as first single shock given.
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Usage of escalating energy level is beneficial for the termination of the refibrillation and patient with
failed shock.
Refractory VF is when fibrillation persist after three defibrillation shocks. Amiodarone 300mg bolus
followed with infusion 900mg to be given. (Amiodarone 150mg after 300 mg bolus, maximum daily
dose 2.2g).
Points to know:
Synchronized cardioversion is preferred for treatment of an organized ventricular rhythm.
However, for some arrhythmias, the many QRS configurations and irregular rates that
comprise polymorphic ventricular tachycardia make it difficult or impossible to reliably
synchronize to a QRS complex. If there is any doubt whether monomorphic or polymorphic VT
is present in the unstable patient, do not delay shock delivery to perform detailed
rhythm analysis-provide high energy unsynchronized shocks (i.e. defibrillation doses,
360J monophasic or 120-200J biphasic).
Pacing
• Not recommended for patients in asystolic cardiac arrest as it is not effective and may delay
or interrupt the delivery of chest compressions
It is reasonable for healthcare providers to be prepared to initiate pacing in patients who do not
respond to atropine (or second-line drugs if these do not delay definitive management).
Immediate pacing might be considered if the patient is severely symptomatic. If the patient does
not respond to drugs or transcutaneous pacing, transvenous pacing is probably indicated.
Summary
The recommendations for electrical therapies described in this section are designed to improve
survival from SCA and life threatening arrhythmias. Whenever defibrillation is attempted,
rescuers must coordinate high-quality CPR with defibrillation to minimize interruptions in chest
compressions and to ensure immediate resumption of chest compressions after shock delivery.
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Regular rhythm.
Normal P wave morphology and axis (upright in I and II, inverted in aVR).
Narrow QRS complexes (< 100 ms wide).
Each P wave is followed by a QRS complex.
The PR interval is constant. ( P-R interval < 0.2sec )
2. Sinus Arrhythmia
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3. Sinus Tachycardia
4. Sinus Bradycardia
• Sinus rhythm.
• A resting heart rate of < 60 bpm.
• Normal QRS complex
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7. Atrial Fibrillation
8. Atrial Flutter
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9. Supraventricular Tachycardia
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14. 2 Degree AV Block Mobitz I (Wenkebach phenomenon)
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nd
15. 2 Degree AV block, Mobitz II
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17. Asystole
No ventricular activity.
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Adrenaline
Atropine
Introduction Dose and Administration
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• First line drug for symptomatic Bradycardia • Use atropine cautiously in the presence
• Organophosphate poisoning of acute coronary ischemia or MI;
increased heart rate may worsen ischemia
or increase infarction size.
• Will not be effective in infranodal (type II)
AV block and new third-degree block with
wide QRS complexes
Adenosine
Introduction Dose and Administration
Amiodarone
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• A membrane-stabilising anti-arrhythmic
drug that increases the duration of Adverse Effects and Precautions
the action potential and refractory
period in atrial and ventricular • Can cause hypotension, bradycardia
myocardium and heart block
• A mild negative inotropic action • The adverse hemodynamic effects of the IV
• Causes peripheral vasodilation formulation of amiodarone are
through non-competitive alpha attributed to vasoactive solvents
blocking effects. Atrioventricular (polysorbate 80 and benzyl alcohol)
conduction is slowed, and a similar • Beware of accumulations with multiple
effect is seen with accessory pathways. dosing (cumulative doses >2.2g are
associated with hypotension)
Indications
• Refractory pulseless VT/VF ( persistent
after at least 3 shock and adrenaline )
• Unstable tachyarrhythmias ( failed
3x cardioversion )
• Stable tachyarrhythmias
Calcium
Introduction Dose and Administration
• Essential for nerve and muscle activity • The initial dose of 10 ml 10% calcium
• Plays a vital role in the cellular mechanism chloride (6.8 mmol Ca2+) may be repeated
underlying myocardial contraction if necessary
• No data supporting any beneficial
• Administer calcium chloride via a central
action for calcium after cardiac arrest
• Some studies have suggested a line only
possible adverse effect when given
routinely during cardiac arrest (all Side Effects and Precautions
rhythms)
• Calcium can slow heart rate and
Indications precipitate arrhythmias
• In cardiac arrest, calcium may be given by
Only in Pulseless Electrical Activity caused by rapid intravenous injection
• In the presence of a spontaneous
• hyperkalaemia circulation give it slowly
• hypocalcaemia • Do not give calcium solutions and sodium
• overdose of calcium channel blocker bicarbonate simultaneously via the
same route
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Lignocaine
Introduction Dose and Administration
• Act as a sodium channel blocker • Cardiac arrest from VT/VF Initial dose:
1-1.5mg/kg IV or IO
• For refractory VF: may give additional
dose 0.5-0.75mg/kg and repeat 5-
Indications 10 minutes up to 3 times or maximal
dose of 3mg/kg
• Alternative to amiodarone in
cardiac arrest from VT/VF Side Effects and Precautions
• Stable monomorphic VT with
preserved ventricular function • In overdose it can cause slurred speech,
altered consciousness, muscle twitching
and seizure
• It also can cause hypotension,
bradycardia, heart block and asytole
Dopamine
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Magnesium
Noradrenaline
Introduction Dose and Administration
Introduction Dose and Administration
• An electrolye important for maintaining • Cardiac arrest due to Torsades
Strongmembrane
beta-1 andstability
alpha-adrenergic effects de pointes as
0.05– 1mcg/kg/min or continuous
hypomagnesemia:
infusion
1-2g diluted in 10 mL D5% to
and• moderate
Hypomagnesemia can cause myocardial
beta-2 effects be given over 5-20 minute
hyperexcitability especially in the presence
Is a potent vasoconstrictor
of hypokalemia with positive
or digoxin • Torsades de pointes with
inotropic effect evidence to recommend for or
• Insufficient Side-effectpulse
and Precautions
or AMI with
against its routine use in cardiac arrest hypomagnesemia:
Cause tissueLoading
necrosisdose of 1-2g mixed
if extravasation occurs
with 50 mL D5% over 5-60
Indication Do not administer Sodium Bicarbonate
minute, followed with 0.5 to
Indications through the1g/hour
same IV line containing
(titrate to control
Torsades)
Noradrenaline
• •Torsades
Used for hypotension in post
de pointes Increase afterload and beta-effects may
resuscitation period Sidemyocardial
increase Effects and Precautions
work and oxygen
• •Hypomagnesemia
Cardiogenic shock consumption
• Occasional fall in blood pressure
• Life threatening ventricular arrhythmias due to Very high dose
with can
rapidlead into peripheral limb
administration
digitalis toxicity • Use with caution if renal failure is
ischaemia
present
Verapamil
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Sodium Bicarbonate
Lignocaine
Introduction Dose and Administration
• A strong alkaline agent with
Introduction Dose and• Administration
1 mEq/kg IV bolus
high sodium and bicarbonate load
• Not
• Act recommended
as a sodium for routine use
channel blocker • Cardiac arrest from VT/VF Initial dose:
in cardiac arrest 1-1.5mg/kg IV or IO
Adverse Effects and Precautions
• For refractory VF: may give additional
Indications dose 0.5-0.75mg/kg and repeat 5-
Indications • May cause tissue necrosis if
10 minutes up to occurs
extravasation 3 times or maximal
• Known prexisting hyperkalemia dose
• Alternative to amiodarone
• Known preexisting in
bicarbonate • ofDo
3mg/kg
not administer together with
cardiac arrest from
responsive VT/VF
acidosis e.g. : aspirin IV line used for vasopressors or
Side EffectsCalcium
and Precautions
• Stable monomorphic
overdose, diabeticVT with
ketoacidosis,
preserved
tricyclicventricular function
antidepressant or cocaine • In overdose it can cause slurred speech,
• Prolonged r e s u s c i t a t i o n with altered consciousness, muscle twitching
effective ventilation. Upon return of and seizure
spontaneous circulation after long • It also can cause hypotension,
arrest interval bradycardia, heart block and asytole
• Not useful nor effective in
hypercarbic acidosis (e.g. cardiac
arrest or CPR)
without tracheal intubation
Dopamine
Dobutamine
Introduction Dose and Administration
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Noradrenaline
Introduction Dose and Administration
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Can consider:
-3 stacks of shocks in witnessed,
monitored VF/pVT
-Escalation of energy in
refibrillation/failed shock
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Adapted from European Resuscitation Council (ERC) 2015 Guidelines for resuscitation.
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Survival after an asphyxia-induced cardiac arrest is rare and survivors often have severe
neurological impairment. Thus, during CPR early effective ventilation of supplementary oxygen is
essential. Those who are unconscious but have not progressed to a cardiac arrest are much more
likely to make a good neurological recovery.
A high degree of clinical suspicion and aggressive treatment can prevent cardiac arrest from
electrolytes abnormalities especially from life-threatening hyperkalaemia. Early recognition and
prompt treatment must be done immediately and a new algorithm has been introduced for
management of hyperkalaemia. Other electrolytes disorders are also important to be managed
correctly to minimize complications leading to cardiac arrest namely hypercalcaemia,
hypocalcaemia, hypermagnesaemia and hypomagnesaemia.
Hypothermic patient without signs of cardiac instability (systolic blood pressure > 90 mm Hg,
absence of ventricular arrhythmias or core temperature > 28o C ) can be rewarmed externally
using minimally invasive techniques ( e.g. with warm forced air and warm intravenous fluid ).
Patients with signs of cardiac instability should be transferred directly to a centre capable of
extracorporeal life support (ECLS). The mainstay of therapy for hyperthermia including heat
stroke is still supportive and rapidly cooling the victim.
Hypovolaemia is a potentially treatable cause of cardiac arrest that usually results from a reduced
intravascular volume (i.e. haemorrhage), but relative hypovolaemia may also occur in patients
with severe vasodilation (e.g. anaphylaxis, sepsis). Early recognition and immediate treatment
with intramuscular adrenaline remains the mainstay of emergency treatment for anaphylaxis.
Intravenous adrenaline should only be used by those experienced in the use and titratioin of
vasopressors in their normal clinical practice ( e.g. anaesthetists, emergency physicians, intensive
care doctors).
The mortality from traumatic cardiac arrest (TCA) is very high. The most common cause of death
is haemorrhage. It is recognized that most survivors do not have hypovolaemia, but instead have
other reversible causes (hypoxia, tension pneumothorax, cardiac tamponade) that must be
immediately treated. The new treatment algorithm for TCA was developed to prioritize the
sequence of life-saving measures. Chest compressions should not delay the treatment of
reversible causes. Principles of damage control resuscitation in trauma including hypotensive
resuscitation, haemostatic resuscitation and damage-control surgery. Cardiac-arrest of non-
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traumatic origin leading to a secondary traumatic event should be recognized and treated with
standard algorithms.
There is limited evidence for recommending the routine transport of patients with continuing CPR
after out-of-hospital cardiac arrest (OHCA) of suspected cardiac origin. Transport may be
beneficial in selected patients where there is immediate hospital access to the catheterisation
laboratory and an infrastructure providing pre-hospital and in-hospital teams experienced in
mechanical or haemodynamic support and percutaneous coronary intervention (PCI) with ongoing
CPR.
Asthma
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Full training in the procedures for equipment failure and the cardiac arrest situation associated with
ventricular assist devices (VADs) is recommended. External chest compression may be particularly
useful to decompress a non-functional right ventricle in cardiac arrests that might be the cause of the
loss of cardiac output. Few implantable left ventricular assist devices (LVAD) in the market currently
such as a HeartMate (Thoratec, Pleasanton, CA, USA) or HeartWare (HeartWare, Framingham, MA,
USA).
Management of cardiac arrest in patient with VADs are ; Start ALS algorithm ;Check the rhythm;
perform defibrillation for shockable rhythms (VF/pVT), start pacing for asystole and turn the pacing off
in pulseless electrical activity (PEA).
ERC recommended, because it is possible for a patient to have asystole or VF, but still have adequate
cerebral blood flow due to adequate and continued pump flow. If the patient is conscious and
responding then you will have more time in which to resolve this arrhythmia and external chest
compressions will not be needed.
Obesity
The World Health Organization (WHO) uses body mass index (BMI; weight in kg divided by height in
m2) to define obesity in adults as;
• Overweight (25.0–29.9kgm−2);
• Obese (30.0–34.9kgm−2);
• Very obese (≥35.0 kg m−2 ).
No changes to sequence of actions are recommended in resuscitation of obese patients. CPR may be
challenging because of physical and physiological factors related to obesity: patient access and
transportation, patient assessment, difficult IV access, airway management, quality of chest
compressions, the efficacy of vasoactive drugs, and the efficacy of defibrillation because none of these
measures are standardised to a patient’s BMI or weight. (710). Higher inspiration pressure is needed
for positive pressure ventilation due to increased intra-abdominal pressure.
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pregnancy. During cardiac arrest, Gravid uterus compromised venous return and eventually and cardiac
output limits the effectiveness of chest compressions. During cardiac arrest, Gravid uterus (from 20th
weeks onwards) compromised venous return and eventually and cardiac output limits the effectiveness
of chest compressions.
Place the patient in the left lateral position or manually and gently displace the uterus to the
left.
Give oxygen, guided by pulse oximetry to correct any hypoxaemia.
Give a fluid bolus if there is hypotension or evidence of hypovolaemia.
Immediately re-evaluate the need for any drugs being given.
Activate Code Red. Obstetric and neonatal specialists should
be involved early in the
resuscitation.
Identify and treat the underlying cause, e.g. rapid recognition and
treatment of sepsis,
including early intravenous antibiotics.
Discussion with O&G team regarding the the need for an emergency hysterotomy or Caesarean section
as soon as a pregnant woman goes into cardiac arrest (about 4 min after cardiac arrest). Based on ERC
guidelines 2015 recommendations are :
At gestational age less than 20 weeks, urgent Caesarean delivery need not be considered,
because a gravid uterus of this size is unlikely to significantly compromise maternal cardiac
output.
At gestational age approximately 20–23 weeks, initiate emergency hysterotomy to enable
successful resuscitation of the mother, not considering survival of the delivered infant, which
is unlikely at this gestational age.
At gestational age approximately ≥24–25 weeks, initiate emergency hysterotomy to save the
life of both the mother and the infant.
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There is increase recognition on the existence of post cardiac arrest syndrome in cardiac arrest victim
whom has been successfully resuscitated and achieved ROSC. This involves a complete
pathophysiological process.
The presentations of post cardiac arrest syndrome include myocardial dysfunction ( first 2-3 days )
and anoxic brain injury. In addition, the ischaemic-reperfusion problem activates immunological and
systemic inflammatory response and contributing to multi-organ failure and increase risk of infection.
Therefore, a comprehensive, structured, multidisciplinary system of care should be implemented in a
consistent manner for the treatment of post cardiac arrest patient. The post resuscitation phase
starts at the location where ROSC is achieved. But, once stabilized, the patient should be transferred
to the most appropriate area of high level care ( eg : intensive care unit, coronary care unit or cardiac
catheterization laboratory ).
The clinical intensity of post cardiac arrest syndrome varies, but is roughly proportional to the
duration of the cardiac arrest and CPR. The overall outcome depends on the underlying cause of
collapse, the availability of early high quality CPR and post cardiac arrest care.
To ensure the success of post cardiac arrest care, healthcare provider must:
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2. Hemodynamic optimization
• Significant myocardial dysfunction is common during post cardiac arrest period. This
accounts for the major cause of death post ROSC but typically recovers by 2-3 days.
• Post cardiac arrest myocardial dysfunction causes hemodynamic instability, which
manifests as hypotension, low cardiac index and arrhythmias. Echocardiography is
indicated during post cardiac arrest care period
• Most of the patient will require inotropic support and intravascular volume expansion.
Some may need invasive device such as IABP (intra-aortic balloon pump) to maintain
hemodynamic stability.
• In post cardiac arrest victims who are maintained a MAP ≥ 70 mmHg as part of post
resuscitation care management, have been reported to have better neurological outcome
and survival.
Hypothermia :
The comatose adult patient with ROSC after cardiac arrest, must have TTM
Select and maintain a constant core temperature between 32-36○C for at least 24 hours
Prehospital cooling with large volume of cooled intravenous drip immediately after ROSC is
not recommended
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5. Seizure Control
• Seizure is common after cardiac arrest. It occurs in approximately one third of patients who
remain comatose after ROSC. Myoclonus is the most common presentation
• Seizure may increase CmRO2 and has potential to worsen the brain insult caused by cardiac
arrest. It should be treated with anticonvulsant
• However, prophylactic use of anticonvulsant in post cardiac arrest patient is not
recommended
6. Glucose Control
• Maintain blood glucose level at ±10mmol/l
• Do not implement strict glucose control in patient with ROSC post cardiac arrest because it
increases the risk of hypoglycaemia
• Hyperglycaemia in patient post cardiac arrest is bad but hypoglycaemia is more disastrous.
Prognostication
Death is common after ROSC post cardiac arrest. Therefore, optimal timing for prognostication in
post resuscitation care patient is essential
The earliest time for prognostication in patient treated with TTM may be 72 hours after return to
normothermia.
The earliest time to prognosticate a poor neurological outcome in patient not treated with TTM is 72
hours after cardiac arrest. However, time must be given to allow the effect of sedation/ muscle
relaxant to wear off.
However, feedback for the resuscitation team should be constructive and not based on fault/blame
culture. Whether the resuscitation attempt was successful or not, the patient’s relatives will require
considerable support. Consider the needs of all those associated with the arrest.
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Beneficience
Life sustaining therapy including CPR should be done in the best benefit of the victim.
However, a balance between risk and benefit when attempting CPR should be considered.
Withholding CPR should be considered if the risk for proceeding with CPR outweighs its benefit.
Non-maleficience
CPR should not be attempted in those whom it will not succeed, where no benefit is likely but
there is obvious risk of harm.
Autonomy
A person with decision making capacity should be allowed to make informed decisions pertaining
to their health and resuscitation options. An advanced medical plan or directives, once
established should be respected in uplifting his rights to medical autonomy.
Justice
This implies to our duty in distributing care equally within the society. If CPR is provided,
it should be available to all who shall benefit from it.
As modern medicine continues to evolve alongside technologies that can sustain and prolong
life, it is imperative that the ethical basis of resuscitation should evolve as well. New resuscitation
techniques and strategies like extracorporal CPR requires new approaches in dealing with decision
to discontinue support. Health care providers will inevitably face difficulties to decide on the
direction of resuscitation without a good understanding of such therapy, its appropriate use,
limitations, implications and likely benefits.
A shift from doctor-centered (beneficence) to patient-centered (autonomy) care in
resuscitation and end of life decisions have been emphasized in the 2015 guidelines. Greater
importance has been given to respecting patient’s wishes for CPR as part of medical therapy. As
such healthcare providers are responsible to equip themselves with sound knowledge in
resuscitation, communication and ethical issues pertaining to it, in making end of life decisions.
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Therefore, as we move towards a family and patient-centered care, family members should be
given the OPTION to be present during resuscitation attempt after considering the cultural,
religious and social background. Observing the resuscitation attempt may allow time for family
members and loved ones to accept the reality of death, reduce guilt or disappointment and may
help the grieving process. Whenever possible, an experienced staff should facilitate and support
the relative during this process.
A proper written document eg. policy, procedure or standard of care on FPDR in patient-care
areas should be made available to assist and ensure health care providers are aware of such
arrangement, thus providing family members the opportunity to be with their loved ones
during this trying period. In Malaysia, such practice should be allowed when proper written policy
or guidelines are in place.
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Appendix
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Advanced Life Support Training Manual
SkillSkill Station
Station Competency
Competency Checklist
Checklist
Management
Management of of Respiratory
Respiratory Arrest
Arrest ififdone
done correctly
correctly
Checks and opens the Airway (head tilt-chin lift or if trauma is suspected,
jaw thrust without head extension)
Administers oxygen
Critical Action
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CPR/AED
CPR/AEDTesting
TestingChecklist
Checklist
VF TreatedWith
VF Treated WithCPR
CPRand
andAutomated
AutomatedExternal
ExternalDefibrillation
Defibrillation(1(1 Rescuer)
Rescuer)
11 Assesses
AssessesDanger
Danger
2
2 Checks unresponsiveness
Checks unResponsiveness
33 Shoutsfor
Shouts forhelp:
help:someone
someonetotoactivate
activateEMS
EMSand
andget
getAED
an an AED
Opens Airway using head tilt-chin lift
44 Opens Airway using head tilt-chin lift
Checks for absent or abnormal Breathing
5 Checks for absent or abnormal Breathing
5
66 Locates
LocatesCPR hand position
CPR hand position
Delivers
Delivers 30
30 compressions
compressions at
at100/min
100/minAcceptable <
20 seconds for 30 compressions
Acceptable < 20 seconds for 30 compressions
7 Gives
7 Gives 22 breaths
breaths(1
(1second
secondeach)
each)
AED Arrives
AED 1 Turns AED on
Instructor signature affirms that skills test were done Instructor Signature:
according to NCORT ALS guidelines
Print Inst.Name:
Save this sheet with course record.
Date:
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Megacode
Megacode Testing
Testing Checklist
Checklist 1/21/2
Bradycardia • VF/Pulseless VT • Asystole
Bradycardia • VF/Pulseless VT • Asystole
Critical Performance
Critical Steps
Performance Steps ifif done
donecorrectly
correctly
Team Leader
Ensures high-quality CPR at all times
Assigns team members roles
Bradycardia Management
Starts oxygen, places monitor, starts IV
Places monitor leads in proper position
Recognizes symptomatic bradycardia
Administers appropriate drug(s) and doses
Verbalizes the need for transcutaneous
VF/Pulseless
pacing VT Management
Recognizes VF
Clears before ANALYZE and SHOCK
Immediately resumes CPR after shocks
Appropriate airway management
Asystole Management
Recognizes asystole
Verbalizes potential reversible causes of Asystole/PEA (H’s and T’s)
Administers appropriate drug(s) and doses
Immediately resumes CPR after rhythm checks
Instructor signature affirms that skills test were done Instructor Signature:
according to NCORT ALS guidelines
Print Inst.Name:
Save this sheet with course record.
Date:
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Tachycardia Management
Starts oxygen, places monitor, starts IV
Places monitor leads in proper position
Recognizes unstable tachycardia
Recognizes symptoms due to
tachycardia
VF/Pulseless VT Management
Performs immediate synchronized cardioversion
Recognizes VF
Clears before ANALYZE and SHOCK
Immediately resumes CPR after shocks
Appropriate airway management
Appropriate cycles Drug-Rhythm Check/Shock - CPR
Administers appropriate drug(s) and doses
Asystole Management
Recognizes asystole
Verbalizes potential reversible causes of Asystole/PEA (H’s and T’s)
Administers appropriate drug(s) and doses
Immediately resumes CPR after rhythm checks
STOP THE
STOP THETEST
TEST
Instructor signature affirms that skills test were done Instructor Signature:
according to NCORT ALS guidelines
Print Inst.Name:
Save this sheet with course record.
Date:
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Advanced LifeSupport
SupportTraining Manual
TrainingManual
Team Leader
Ensures high-quality CPR at all times
Assigns team members roles
Bradycardia Management
Starts oxygen, places monitor, starts IV
Places monitor leads in proper position
Recognizes tachycardia (specific diagnosis)
Recognizes no symptoms due to tachycardia
Attempts vagal maneuvers
Gives appropriate initial drug thetapy
VF/Pulseless VT Management
Recognizes VF
Clear before ANALYZE and SHOCK
Immediately resumes CPR after shocks
Appropriate airway management
Appropriate cycles Drug-Rhythm Check/Shock-CPR
Administers appropriate drug(s) and doses
Asystole Management
Recognizes asystole
Verbalizes potential reversible causes of Asystole/PEA (H’s and T’s)
Administers appropriate drug(s) and doses
Immediately resumes CPR after rhythm checks
STOP THE
STOP THETEST
TEST
Instructor signature affirms that skills test were done Instructor Signature:
according to NCORT ALS guidelines
Print Inst.Name:
Save this sheet with course record.
Date:
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National
Committee
Resuscitation
Training
(NCORT)
Advanced Life
Advanced Support
Life Training
Support Manual
Training
Manual
Advanced Life
Dato’ Dr Support
Luah Sub Committee
Lean Wah Dr Ridzuan bin Dato’ Mohd Isa
Consultant Anaesthesiologist Consultant Emergency Physician
Penang General Hospital Penang Ampang Hospital
Selangor
Dato’ Dr Luah Lean Wah Dr Ridzuan bin Dato’ Mohd Isa
Dr Ismail
Consultant Tan bin Mohd Ali Tan
Anaesthesiologist Consultant
Dr WanEmergency Physician
Nasrudin bin Wan Ismail
Penang General Hospital
Consultant Penang and
Anaesthesiologist Ampang HospitalAnaesthesiologist and
Consultant
Intensivist Kuala Lumpur Hospital Selangor
Intensivist Raja Perempuan Zainab II
Kuala Lumpur Hospital Kota Bahru
Dr Ismail Tan bin Mohd Ali Tan Dr Khairuddin bin Ismail
Consultant Anaesthesiologist and Consultant Anaesthesiologist
Intensivist Kuala Lumpur Hospital Sultanah Nur Zahirah Hospital
Kuala Lumpur Terengganu
Dr Anuwar Ariff bin Mohamed Omar Datin Dr Najah binti Tan Sri Harussani
Consultant Anaesthesiologist Consultant Anaesthesiologist
Melaka Hospital Raja Permaisuri Bainun Hospital Ipoh
Melaka Perak
Secretariat
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National Committee
National OnOn
Committee Resuscitation Training
Resuscitation (NCORT)
Training (NCORT)
Sub Committee
Sub Committee
Advanced Life
Advanced Support
Life Sub
Support Committee
Sub Committee
Dato’ Dr Dr
Luah Lean Wah Dr Ridzuan bin Dato’ Mohd Isa
Dato’ Luah Lean Wah Dr Ridzuan bin Dato’ Mohd Isa
Consultant Anaesthesiologist Consultant Emergency Physician
Consultant Anaesthesiologist Consultant Emergency Physician
Penang General Hospital Ampang Hospital
Penang General Hospital Ampang Hospital
Penang Selangor
Penang Selangor
DrDr Ismail
Ismail TanTan
binbin Mohd
Mohd AliAli
TanTan Dr Dr
AdiAdi
binbin
Osman
Osman
Consultant
Consultant Anaesthesiologist
Anaesthesiologist andand Intensivist
Intensivist Consultant Emergency
Consultant Physician
Emergency Physician Raja
Kuala
Kuala Lumpur
Lumpur Hospital
Hospital Raja Permaisuri
Permaisuri Bainun
Bainun HospitalIpoh
Hospital
Kuala
Kuala Lumpur
Lumpur Ipoh
Perak
71 Page
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