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    Francisco Javier Bernal Avila
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    Case report NSH

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    8 views6 pages

    Case Report NSH

    Uploaded by

    Francisco Javier Bernal Avila

    Copyright:

    © All Rights Reserved
    Download as PDF or read online from Scribd
    Flag for inappropriate content
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    RRQUINE VIERMRINARY EDUCATION Ruins vst Bd, (2000) 12 (6) 297-902 Case Report 2 Unusual presentation of nutritional secondary hyperparathyroidism in a Paint colt D. Lrrn, W. R. Reppine, K. A. Spautpre*, S. H. Duprent ann S, L. Jones Department of Clinical Sciences, “Anatomy, Physiology and Radiology and tVeterinary Teaching Hospital, College of Yeterinary Medicine, North Carolina State University, 4700 Hillsborough Street, Raleigh, North Carolina 2%606, USA. Introdudion Parathyrofa hormone (PTH), calcitonin and vitemin D3 control calcium and phosphorus motnbotism and hence, to a Inrgo ttent, bone metabolism, Clinical signs atiributable to ‘dysfunctional bone metabolism. are manifest whm abnormal levels of these hormones exist ‘There axe 2 main types of hyperparathyroidiem. Psimery Ihyperperathmroidism, which results from excessive and uncontrolledproduetion of PTH, is rarely reported in the hhorse Rousel and Thatcher 1987; Roussel ef ai, 1987; Penger et 190d; Frank ef al. 1998). Secondary Iiyperparatlgroidism is an ostaodyatrophie disease resulting fom decompensation of physiological mechanisms of ealciam homeostasis, Renal recondary hyperparatlgroidism rarely occurs in the horse, but nutritional tecondary hyperparatinyroidism (NSH) is voll recognited disorder, The incidenco of NEE appears to have decline! throw this century as knowledge of the rele of nutciton in this disorder ins beeoro known, and it is now eorsdard to be en uncomman diseaso of the horse ‘Breestone and Seshorn 1999; Clarke ef al. 1996). ‘As with most, metabolic bone discasas, thoro ie « spectrum of clinical prosontations depenting on the soverity of adeodyatrophy. However, the classical clinical presentationis fibrous osteodystrophia ofthe facial bonas, from which te colloquial name for the disease, ‘higheed?, jg derived, Othar clinienl signs inetodo « stiff gait or shifting-limblameness, abnormal masticstion, weight loss and uppor eirway siridor ‘These clinical siens are Aecceibed Gpically in conjunction with clinical and radlographieevidenco of thickoning ofthe facial hones. Case details History A.16-month

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