Intra Pump: mi, chf

Pre Post leaky: nephrosis, gastrosis

Prerenal cirrhosis
hole: d/d/d/h
clog: fmd, ras
nephrotic
glomerulonephritis >3.5g/d
Glomerulonephritis rbc casts chol
Intrarenal edema
AIN
wbc casts, wbc Infxn
Abx: tmx- smp
atn eosinophils pcn, cephalo
Prodrome cr
AtN Ischemia
contrast IV Fluids
cr
IV
oliguric u Muddy brown Toxins: myoglobin
casts

ain polyuric u Ureters: Ca, Stones

Pre bun:cr >20 Postrenal Bladder: Ca, Stones
una <10 prerenal Neurogenic
fena Fena <1%
ivf or diuresis
Acute HD Urethra: Ca, Stones
FEUrea FEUrea <35% bph, foley
a cidosis
k
e lytes Ca
Post u/s hydroureter
or postrenal I ntoxication
ct hydronephrosis
Foley Surgery Overload
Nephrostomy
u remia
Intrarenal HX
u/a
phys DX
Dz-specific
BX
 I HATE THIS BOOK.

Just kidding. Well, not really.

Let’s talk about what this book is and isn’t. It is a companion to our video curriculum. It’s a great
place to take notes, and to reference my final whiteboards. It ISN’T a shortcut. Nor is it an excuse
to skip writing out your own notes as you follow along.

OME’s power comes from two places: 1) Medical knowledge belongs to no one, and so it is acces-
sible to everyone. This is why the videos are free. 2) The PACE paradigm—reading the notes (and
taking your own), watching the video (and taking more notes), doing the challenge questions (and
taking more notes), and enforcing them through repetition—works. Purposeful engagement of the
content in multiple modalities is the path to success.

This ISN’T a replacement for PACE. I advocated that we not create this book, as it might facilitate
students in skipping steps: not writing out their notes, not following along with me at the board,
etc., thus compromising their learning. But, I was overruled by you, the user. We made this because
you asked for it, and we realized you were spending hundreds of dollars printing out the whiteboard
graphics anyway.

So I acquiesced. I recommend you follow PACE and follow along with me at the board. I designed
the lessons to flow a certain way. The positioning on the board, the order in which the material is
presented, the colors used, and even the cadences of speech are not accidental.

BUT, I’d rather you have an awesome, well produced, ring bound, color book for cheaper than
doing it yourself. I’d also rather you spend time studying, not wasting time organizing and tracking
down the logistics.

This book is a companion. It’s not a crutch.

It also has a cute cat animation in the lower left corner. Check it out.
Copyright © 2019 by OnlineMedEd
First Edition

For information about permission to use or reproduce selections

from this book, email help@onlinemeded.org.

By Dustyn Williams and Rachel Evers-Meltzer

ISBN 978-0-9969501-3-8
Dustyn Williams, MD
Published by OnlineMedEd, www.onlinemeded.org

Printed in the United States of America

Coronary Artery Disease.Pdf NOTES

Cardiology
ASx cp cath
cad sa ua nstemi stemi
1,2 3+
pain + exertion rest rest rest ekg
st
cath
stent #vessels cabg
e-merge
relieves with rest m orphine bb
o2 a ce-i
bio troponins urgently n itrates s tatin
markers a sa h eparin?
st s ? c lopidogrel?
tpa? des....1 year
occlusion <50% 70% 90% 90% 100% stress bms....1mo
.......
Supply test electively
demand ischemia ischemia rest

hpi risk factors assoc sxs physical stress evaluation

Dm sdb nonpleuritic exercise ecg
1 substernal presyncope normal at risk dead
smoking nonpositional echo
2 worse exertion htn n/v non-tender
d NTG hld pharm nuclear
3 Relieve
obesity stress
3/3 = typical
Fam HX
2/3 = atypical
Age >45
0-1/3 = nonanginal >55

Heart Failure.Pdf

doe βb
orthopnea i asa
pnd ace-i/arb statin
loop
jvd ii diuretics
r l pmi crackles aicd
isdn-Hydralazine ef<35%
abd pain
iii Spironolactone
hsm
body
iv inotropes

l weight gain
everyone: h2o <2L/day
peripheral
edema nacl <2g/day chf
exac
body
dx: 1 cxr cxr ecg stemi mi
2 ecg bnp trop
mona
r 3 bnp ef= 55% bash
4 echo pap
diastolic sys cath
chf
body
5 lhc ischemia ef
l atix
ischemic or not m orphine
dia n itrates
htn o2
ef p osition
infiltrative
normal

© 2019 OnlineMedEd 5
Valvular Disease.Pdf NOTES

Cardiology
mitral mitral
insufficiency
path: infxn
infarction
hcm path: sarcomere mutations
stenosis path: rhuematic
grade PT: acute chronic pt: young athlete
PT: younger
cardiogenic CHF sudden death
chf sxs
I s1 s2 > murmur afib
shock
pulm edema
afib sob, syncope c exertion
dx: diastolic dx: systolic dx: systolic
II s1 s2 = murmur apex apex as....more blood=
*opening snap* holosystolic makes it better
rumbling Tx: replacement tx: “avoid dehydration”
III s1 s2 < murmur tx: balloon valvuloplasty
replacement β-blockade
IV palpable thrill mvp path: congenital
aortic aortic Dissection Aortic
V stethoscope 1/2 off insufficiency path: infxn stenosis path: as ca pt: young women
Chest infarction pt: old ♂ ..bicuspid dx: “mr”...
pt: acute chronic
better c more blood
VI without stethoscope
c as..cp. chf, syncope
cardiogenic chf
shock dx: systolic tx: β-blockade
flash pulm cp base
Systolic 3+ crescendo decrescendo avoid dehydration
edema
dia echo cp
tx: replacement
dx: diastole
base
rumbling cabg
tx: acute: emergent
chronic: urgent replacement

Cardiomyopathy.Pdf

restrictive CM
dilated cm hocm concentric hypertrophy
ca
path: amyloid
path: contractility path: genetics path: htn sarcoid fibrosis
virus, etoh, sarcomeres pt: dia chf hemochromatosis
ischemia pt: murmur=as
pt: sys chf: young athletes pt: dia chf
amyloid neuropathy
orthopne a/pnd doe sarcoid pulm disease
doe syncope cirrhosis, dm
crackles, edema scd hemo
dx: echo=asymmetric dx: echo=concentric dx: echo=restrictive
dx: echo=dilated fat pad bx
tx: dia chf amyloid
tx: avoid dehydration endo
tx: chf: βb, ace-i avoid dehydration sarcoid cmr myocardial
diuretics βb=ccb βb=ccb hemo ferritin genetic
etoh etoh ablation, myectomy tx: dia chf
chemotheraphy
aicd= risk of death βb=ccb
transplant
transplant transplant gentle diuresis
htn transplant
f/u: 1st degree relatives
screened underlying DZ

© 2019 OnlineMedEd 7
 $100.00
ISBN 978-0-9969501-3-8
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