1. CTG monitoring involves monitoring the fetal heart rate and uterine contractions during labor to assess fetal well-being and risk of distress.
2. Various patterns in the fetal heart rate tracing including baseline heart rate, variability, accelerations, and decelerations are evaluated using an assessment tool to determine if the tracing is predictive of normal or abnormal acid-base status.
3. Potential interventions for abnormal tracings showing late or variable decelerations include oxygen administration, changing maternal positioning, stopping oxytocin infusion, tocolysis with terbutaline, amnioinfusion, or expedited delivery by cesarean section or assisted vaginal delivery if the fetus is at station +1 or below.
1. CTG monitoring involves monitoring the fetal heart rate and uterine contractions during labor to assess fetal well-being and risk of distress.
2. Various patterns in the fetal heart rate tracing including baseline heart rate, variability, accelerations, and decelerations are evaluated using an assessment tool to determine if the tracing is predictive of normal or abnormal acid-base status.
3. Potential interventions for abnormal tracings showing late or variable decelerations include oxygen administration, changing maternal positioning, stopping oxytocin infusion, tocolysis with terbutaline, amnioinfusion, or expedited delivery by cesarean section or assisted vaginal delivery if the fetus is at station +1 or below.
1. CTG monitoring involves monitoring the fetal heart rate and uterine contractions during labor to assess fetal well-being and risk of distress.
2. Various patterns in the fetal heart rate tracing including baseline heart rate, variability, accelerations, and decelerations are evaluated using an assessment tool to determine if the tracing is predictive of normal or abnormal acid-base status.
3. Potential interventions for abnormal tracings showing late or variable decelerations include oxygen administration, changing maternal positioning, stopping oxytocin infusion, tocolysis with terbutaline, amnioinfusion, or expedited delivery by cesarean section or assisted vaginal delivery if the fetus is at station +1 or below.
-Def: monitoring FH while he is under stress of contraction *labor*
-how to read any CTG? Dr C BrVADO (determine risk *Hx* , uterine contraction: No. , baseline assessment, variability , acceleration , deceleration , order) 1-Baseline rate: Normal: 110 to 160 b/min , >160 tachycardia , <110 bradycardia 2-Variability: take long segment and takes highest - lowest point = the Normal fluctuates between 5 and 26 beats/minute ↓ beat-to-beat variability <5 : *ASD* : asphyxia, sleep(physiological and last for 25 minutes) or drugs(pethidine or Mg sulfate) -FHR Changes: 1.Acceleration: FHR ↑ by 15bpm for at least 15s (This is a normal response “Ynz3g mn alcontraction and move” -> sympathetic activated -> ↑ FHR. 2.No change: 3.Deceleration: FHR ↓ by 15BPM for at least 15s -Decelerations may be: early, late, variable, or mixed. All except early decelerations are abnormal - Early deceleration (head compression): - This pattern: onset, peak, recovery of deceleration are co-incident with onset, peak, and recovery of uterine contraction (mirroring) - uterine contraction compressing fetal head ->↑ I.C.P-> vagal response-> deceleration (normal physiological response) - Mang: observe only. - - - Late deceleration (U.P insufficiency ) : This pattern : onset, peak, and recovery of deceleration are shifted to right *lag* in relation to uterine contraction MD1TALK Severe repetitive late decelerations : indicates Chronic asphyxia-> shunt of blood to vitals and shift to anaerobic metabolism-> fetal Acidosis -> hypercalcemia->slow conduction of nerve->delayed response of FH Causes: pre-uterine -> hypotension / Uterine -> tachysystole *PG or oxytocin* Intra uterine : insufficiency in PTP /PIH / APH -Variable deceleration (cord compression): This pattern: variable in onset , shape*W* , intensity caused: cord compression ->causes a sudden increase in BP in circulation of fetus -> carotid body stretch -> vagal response -late decelerations with variability of <5 beats/ minute -> indicates severe fetal distress
Small box = 15s in duration
Strategies for Intervention :
Variable and Late DECELERATIONS during labor: -intra uterine Resuscitation : 1-ABC: 100% o2 mask -> inc blood perfusion 2-maternal position to right or left lateral (relieves fetal pressure on the cord and IVC) 3-Empty u.bladder 4-Stop oxytocin infusion 5-TOCO: Terbutaline 6-Amnioinfusion : NS is infused to amniotic sac particularly for variable. If its in 2nd stage + vertex >=+1 station -> do instumental delivery* *آسرع لك, If not ? EM C/S if there is no accelerations + lack of beat-to-beat variability *<5 —> Acidosis or sleep: do acoustic stimulation or DSS(Waking baby)-> acceleration? -> absence of acidosis
FETAL TACHYCARDIA: FHR >160 for > 10 min
MD1TALK tachycardia : fetal anemia ,*choriamonits* , post-term pregnancy , epidural Tachycardia for 15 to 30 minutes are associated with excessive oxytocin augmentation of labor (moves a lot) FETAL BRADYCARDIA : FHR <110 for >10min —> deliver immediately either by c-section or assisted vaginal delivery Causes: narcotics , heart block , severe acidosis FHR <110 for more than 2 minute-> prolonged deceleration. Order : Category (I): tracing predictive of normal acid-base status Features: -Baseline rate 110 to 160 -variability moderate -absent Late or variable decelerations -present or absent Early decelerations Category (III): tracing predictive of abnormal acid-base status Features: Absent variability and any of the following: - Recurrent late decelerations - Recurrent variable decelerations - Bradycardia - Sinusoidal patten - Category (II): ينحط هنا٢ وال١ اي شي مو pt on syntocinon to induce labour : -If there is >5 contraction per 10m -> uterine hyper-stimulation(tachysystole) -Uterine vesseles every time with contraction close -> deceleration 1st thing you do : stop syntocinon notes : Supine hypotensive syndrome (IVC compression syndrome) is caused by gravid uterus compresses IVC in a supine position ->leading to decreased venous return. ... In severe cases, women can have loss of consciousness , and fetal bradycardia MD1TALK
Raising Mentally Strong Kids: How to Combine the Power of Neuroscience with Love and Logic to Grow Confident, Kind, Responsible, and Resilient Children and Young Adults
Dark Psychology & Manipulation: Discover How To Analyze People and Master Human Behaviour Using Emotional Influence Techniques, Body Language Secrets, Covert NLP, Speed Reading, and Hypnosis.
Empath: The Survival Guide For Highly Sensitive People: Protect Yourself From Narcissists & Toxic Relationships. Discover How to Stop Absorbing Other People's Pain