Study Student Disorder & Methodology Results Evaluation

name Aim

Amick et al. (2015) Dmitrii Depression & Review 11 No statistical +: The made
“CBT vs. anti- Leonidovi Compare randomized differences in the research is very
depressant drugs” ch CBT control trials effectiveness of specific
Oshchepk (Cognitive (including CBT or the other +: Research based
ov Behavioral qualitative antidepressant on statistics tends
Therapy) to assessment and to be strong
the other meta-analysis) -: CBT differs with
antidepressan comparing CBT each client’s type
ts to the different of behaviour
antidepressants

Khan et al (2012) Momo Investigate Analyse previous Combination of -Meta-analysis is

“Comparative Yang the studies (100+) depressants and strong, but the
Efficacy” effectiveness psychotherapy is reliability of each
of different more effective study included is
treatments for than either alone. not clear.
depression
-individuals
difference

March et al (2004) Dung Evaluate the 439 adolescent 71% effective for - High external
“Antidepressants & effectiveness of patients with major combination validity (439 valid pts
CBT” 4 depression depression. and 95% confidence
treatments for 60% for fluoxetine interval).
adolescents. 12 weeks of alone
treatment: - Strong
fluoxetine alone, 43.2% for CBT methodology
CBT alone, CBT alone (blinded for placebo
with fluoxetine, and fluoxetine)
placebo. 34.8% placebo
- Unethical: patients
do not receive the
most desired
treatments

Dunlop et al (2017) Natalia Aim to check 122 participants Out of 122 It is better to
“Biomarkers, CBT, & María the randomly assigned Only 58 (47.5%) understand the
antidepressants” Ramos effectiveness of to receive CBD or improved to the treatment of depression
Nufio CBT and other another treatment. through neural
drugs for antidepressive: When the treatment networks
different SSRIs or SNRIs. matched with biotype Ethical consideration
biomarkers of treatment 74% were into
depression improved. consideration.
When treatment did
not with biotype
treatment 85.5% of
the participants failed.
Drysdale et al (2017) Shachar Investigate Method: They Observed: Strength:
“Biomarkers for Witzrabin the role Correlational ‘common - Indication that
depression” neural study neurological core’ diagnostic criteria
Sample: 1188 for depression may
networks - showing all
USA and be flawed
might have in Canada subtypes of
depression Used: fMRI to depression - Data driven
examine Then they were approach
connectivity framed into 4
across biotypes, Limitations:
neural networks on the basis of - Correlational
(responsible for study, thus no
biomarkers
mode regulation) cause and effect
Finally: indicating
relationship can be
Correlated connectivity in the shown
patterns of neural networks,
connectivity with connecting Method:
symptoms of various brain - Results cross-
depression structures. validated with
different samples

- Findings were
replicated with
other samples

Kirsch (2014) Souhung Investigate the Meta analysis of 82% of the The drug-placebo
“Antidepressants & role of the datas from US effectiveness of effect size was 0.32
Placebo Effect” placebo effect food and drug antidepressant drug which measure the
in administration is due to the effectiveness of drug
antidepressant (FDA) and placebo effect. based on clinical
drugs published and Resulting in only improvement. (it
unpublished data two points needs to be at least
difference on the 0.5 to be approved).
Variables: 53-point HAM-D
antidepressants scale between the S: included the
and placebo two variables. vulnerability of
relapse.
So the most parts of L: not triangulated,
the effectiveness of other studies
SSRIs comes from showed the
the placebo effect. antidepressant
works for those with
mild depression.

Ethics: no inclusion
of negative studies
can be misleading
Mukherjee (2012)
“Antidepressants and
Depression Severity”

Kuyken et al. (2016) Noa Fišer Investigate Statistical Normal MBCT appeared
“MCBT & Depressive the analysis of nine treatment , ( long to lessen the risk
Relapse” effectiveness randomised term treatment , of relapse
of MBCT with controlled trials short term considerably for
other forms of comparing results )possible those participants
treating MBCT with other relapse into with the most
depression treatments for depression after severe depression
depression 60 weeks, MCBT
treatment of 8
weeks no relapse

Baldwin et al (2011) Nimaty To investigate Trials on drug There is no 'ideal - Supporting

“SSRI’s to Treat GAD” Abu Assab GAD’s drug treatments of GAD treatment' for GAD, treatments
treatments . based on a meta- and that the existing for GAD
analysis of a treatments are wasn’t as
random sampling relatively substantial
methd. ineffective in both as the
the short and long research
term. supporting
treatments
for
depression

Cuijpers et al (2014) Sofía Generalized -meta-analysis CBT (Cognitive S: Meta-analysis

“CBT & GAD” Peñaherr Anxiety that compared Behavioral for broad
era Disorder. psychotherapies Theraphy) is conclusions about
To test the and drug effective in treatment (CBT)
effectiveness treatments for treatment of GAD. L: Self Report data
of GAD against That treatment collection causes
psychological each other. method is reliable demand
treatments of (conducted in characteristics.
GAD other studies with Participant Bias.
good results.) M.L: not enough
sample size of
compared studies
in the meta-
analysis to
conclude
effectiveness of
treatment.

Olatunji et al (2010) Momo Investigate Meta- analysis -Efficacy of CBT -no ethical
“CBT & GAD” Yang the efficacy of on anxiety is problems.
CBT on GAD supported -correlation not
-exposures in cause-effect
 CBT might be relationship
necessary and -strong
sufficient for OCD methodology but
and phobia individual study
might be invalid to
certain extent.

Evans et al (2008) Hagar To investigate 11 GAD patients The results

“MBCT for GAD” Diller the were indicated that
effectiveness participants. MBCT led to
of MBCT in -The first part reductions in
treating GAD. was MBCT anxiety, worry and
classes for 8 depression.
weeks in focus
on MBSR.
-The second
part, cognitive
exercises in
group sessions.
-Also completed
meditations
homework.
-disorders were
measured using
a variety of
scales, using
self-reporting.

Hoge et al (2013) Tova To test the 93 participants, - Both

“Mindfulness-based Lebens effectiveness diagnosed with lowered
stress reduction” of GAD, randomly the anxiety
Mindfulness assigned to an symptoms
as a 8-week long - The
treatment of group statistics
GAD intervention showed
treatment either that the
using MSBR treatments
(Mindfulness- were both
based stress effective,
reduction) or they didn't
SME (Stress significantl
Management y
Education) statistically
differ.
-

DeAngelis (2008)
“Therapy treatment for
disorders”
Mitte (2005) Mohamad To investigate Meta-analysis of In reality, Strengths:
“Medication vs. CBT” the studies significant Credible because
effectiveness differences its based on
of CBT and between both multiple studies
medicinal therapies Ethical.
treatments on because the
GAD studies were Weakness:
influenced by Complicated
different factors. methodology.
But CBT showed
more Difficulty finding
effectiveness suitable studies.
under the
conditions of this researcher's
study. Bias.

Roy-Byrne et al Phạ m To investigate Select patients A significantly + Participants were

(2010) “CALM Tuấ n whether a with anxiety greater carefully selected →
Model” Long flexible disorders, improvement for exterminate
treatment- excluding CALM vs UC in confounding
delivery patients who global anxiety variables → better
model fo won’t benefit symptoms was external validity
from CALM. found - triangulation of
r multiple Interview. Then method → reliability
primary care Stratified - Stratified
anxiety randomization to randomization →
disorders 2 groups: UC external validity
would be and CALM.
better than Blinded follow-up
usual care assessments
(UC) after a long time.