Patient in tha hospital were: Ph(756), PCO2(38), The amount of calcium is filtgred by the glomeruli is 240 mmol/24 h In the Calcitriol increases the absorption of calcium and phosphate from GI system in the case of PTH deficiency, sybsequently, an increase in the excretion of any excess in the level of Zaicium and phosphate in ECF by kidney. * Or @F Increase of Ethylene glycol metabolism leads to decrease of anion gap * — Or De The main reason for carbon dioxide pass into red blood cells is haemoglobin acts as buffer solution *Increase of Ethylene glycol metabolism leads to decrease of anion gap * Or @F ‘The main reason for carbon dioxide pass into red blood cells is. haemoglobin acts as buffer solution * Or @Fr The results of unknown blood sample in clinical biochemistry lab. For 1 point Certain patient in the hospital were: Ph(7.25), PCO2(37), HCO3(20), subsequently, The abnormal case of patient is called respiratory acidosis * Increase of lactic acid leads to decrease of the anion gap * C7.CIE er od A eet @ docs.google.com ri] Dehydrogenase (J) Isomerase (1) Decarboxylase Regarding Type III 1 point hyperlipoproteinaemia , all of the followings are true except * eee (FJ Palmar xanthoma a broad pre-beta band Page 3 of 4 Back Next ee submit passwords through Google Forms € + Oean OU @ docs.google.com All of the following plasma 1 point lipoproteins are normally founded in the blood of healthy subjects except * (J) Hou fa IDL 0 chylomicrons (1D veo. Regarding LDL deficiency , all of 1 point the followings are false except * =— 5) Defects of apoB metabolism wi impaired VLDL synthesis wm acanthocytosis (J) Lat deficiencySee oe Sea @ docs.google.com The most common cause of 1 point hypoglycemia is * PF emenanean hypoglycaemia O Hypoinsulinaemic hypoglycaemia O Reactive hypoglycemia (FJ Miscellaneous causes The precursor of testosterone is * 1 point oO Aldosterone O Methyl testosterone oO Estrone Pregnenolone All of the following plasma 1 point lipoproteins are normally |: | founded in the blood of healthy eit sire dinesPSE Pra @ docs.google.com In Cushing’s syndrome-a tumour 1 point associated disease of adrenal cortex, there is * ([) Decreased epinephrine production Bb Excessive cortisol production (1) Excessive epinephrine production Decreased cortsoil production Diabetic ketoacidosis is caused 1 point by * Low insulin level fo low intracellular glucose level V beta cell dysfunction we level of plasma free fatty acids The most common cause of 1 point fi hypoglycemia is *Ser @ docs.google.com Plasma lipids consist of * 1 point (F) triacylglycerols (18%), oO phospholipids (35%), x cholesterol (14%) oO cholesteryl esters (38%) A hormone secreted from 1 point anterior pituitary is * Growth hormone (1) vasopressin (J) oxytocin O Epinephrine Ketotic hypoglycemia is caused 1 point by * Vy oO Severe hypothyroidismPerera) @ docs.google.com Precursor of ACTH is * 1 point Cholesterol Pregnenolone Corticotropin SBO00 Pro-opiomelanocortin A hormone produced by corpus 1 point luteum and placenta, concerned with relaxationof pelvis tissue is * HCG Chorionic somatommotropin Relaxin OsBsond Progestins In Cushing's syndrome-a tumour 1 point associated disease of adrenal fa cortex, there is *SSPE @ docs.google.com Hyperlipidemia is more 1 point dangerous than hypolipidemia * y2) True @ False The leyding cell activity is 1 point controlled by * Intestitial cell stimulating hormone oO Adernocortex stimulating hormone oO Thyroid stimulating hormone oO Melanocyte stimulating harmone Precursor of ACTH is * 1 point oO Cholesterol 0 PregnenoloneREE @ docs.google.com Ketotic hypoglycemia is caused 1 point by* Z Severe hypothyroidism (LJ End-stage renal disease wm Adrenal insufficiency ws Von Gierke's disease The following organs are highly 1 point dependent on glucose as a fuel source except * yf Skeletal muscle SF adipose tissue fa The defect in adrenal cortex 1 pointSEs @ docs.google.com responsible for lack of glucocorticoids and mineralcor- ticoids is * i) Androstenedione deficiency oO 17 a-OH progesterone deficiency C-21 hydroxylase deficiency (J) Testosterone deficiency Cortisol is synthesized in * 1 point Zona fasiculata (J) Zona glomerulosa (J) Zona reticularis O Chromaffin cells The following criteria are 1 point characteristic of Type 2 diabetes {EB mellitus except *Aer 2) @ docs.google.com Ine tollowing criteria are 1 point characteristic of Type 2 diabetes mellitus except * oO 2hr postprandial serum glucose 11.1mmol/L (J) Fasting serum glucose 7 mmol/L Aa HbA1c 6.4% AY 2hr OGTT 10 mmol/L All of the followings are 1 point characteristic features of familial hypercholesterolemia except * oO Tendon xanthoma 4 eruptive xanthoma oO corneal arcus y lipaemia retinalis fa Hyperlipidemia is more 1 pointA Peer) @ docs.google.com ri] Excessive secretion of ACTH 1 point causes * Cushing's syndrome oO Addison's disease oO Myxoedema (J) Thyrotoxicosis Secretion of prolactin is 1 point regulated by * (J) Feedback inhibition (J) Prolactin releasing hormone (F) Prolactin release inhibiting hormone All the above Plasma lipids consist of * 1 point € + OoSPE @ docs.google.com The production of progesterone 1 point by corpus luteum cell is stimulated by * LH (D TsH (CD ActH (0 MsH The following organs are 1 point gluconeogenic except * (CD liver (J) kidneys sa Skeletal muscles 5 adipose tissue Excessive secretion of ACTH 1 point causes *1:09 Gad <@ SALE) -aSUrement of the enzyme ionlis more Oya RS RSS Ee eee [Perea Bees oer eC Ee mercer eM (oR 11-101] [Ted roolaR | * € 100f15 MAcute.cell.damage, for example in viral; . (hepatitis, y cause very high plasma aminotransferase activities that reduce.as the Elle ae we ele kh aces el este IL)aR ee | i € 90f15 es,)such as amylase;! can be la aaasiiaiheiaae eys.) Thus, plasma OE RESIN Cee ist mI Ber SRO E TNC Ie REY oreo Super ne cn eee eT ernC ne | Nd“7 '"ENer or lower than nor Nzyme activi h Thee De hig Et m ty in plasma may be: Che » due to the|proliferation ¢ Yate of. emg D Screen Saleem ee err AER resl Res PARES UND *. Pirie 321:09 Gad <@ bd € 110f15 OCardial infarction; fo Plasma levels of Cand aspartate aminotransferase (AST) -fall:to:normal before those ofd.DHpwhich has a longer half-life Errweea ir |ErRRCe- Wel Shy LNB Ey el | MO ESRC UNIAN) ane RCTS rene Sees Bear 10D]: MUES ESO EF) reer ape erie)SRO ew a € = 120f15 = — o aralysis)may be improved by tt Ge’ Serial enzyme estimations es Wich anil estimations, (e\lsoenzyme determination (ie Estimation of more than one enzyme Pa ieee nt mn RS aaL a aa ee Roa) > Before attributing a changeiin plasma e activity to a specific disease process, it is important to exclude 1Moderate exercise, or a large intram injection, may lead to a rise in/plasma'CK 2-Some drugs, such as the anticonvulsants | phenytoin and phenobarbital, may induce the synthesis of the microsomal enzyme g-glutamy! | transferase (GGT) cular lage LDH) ALPiand CAL JoSl Sao Joh La Se See e Stk RM CLs ce nee eer ee eben!lip “=? Y" VAISEQ plas a AST in Vitro release from erythro i Orel om er cytes if thi S€paration of plasma from cells is delve : during the neonatal period, ti : pper adult eee timith period |(about 1.5 times the @ Marked increase (may be gre: Se aes he upper reference limit or URL entre failure with : aaa ~ hypoxia} — myocardial infarction) — acute viral or toxic hepatitis (Moderate Zo(Slight increasé (usually less than five times URL): ~ Hepatic steatosis{fatty liver or non-alcoholic fatty liver disease (NAFLD)], — cirrhosis (may be normal sometimes), — infectious Aono i iovolvelnen te ai jaundi i i f the liver, (may be normal), — , — malignant infiltration o nay be norr $ eat uscle disease, — after trauma or surgery (especially after cardiac surgery), — severe haemolytic episodes (of erythrocyte origin), ” Tien FAST 6 Weslo! Lich a (a EI 4 3 A cy SS . z *aR ee a ce ee | Ce a El ane oxaloacetate aminotransferase, GOT)is _ Present in high concentrations in cells of cardiac)and skeletalimuscle liver, kidney. and erythrocytes.) Damage to any of these tissues may increase plasma AST levels. CU CN ee ela