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  • Facial Paralysis

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    RISHAV BHAGAT
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    Facial paralysis can be caused by central or peripheral lesions of the facial nerve. Peripheral lesions are more common, accounting for about 2/3 of cases. Bell's palsy is the most common cause of peripheral facial paralysis, characterized by sudden onset of weakness or paralysis of the muscles on one side of the face. It is usually idiopathic but can be caused by infections like herpes zoster oticus. Trauma from temporal bone fractures can also cause facial paralysis by damaging the facial nerve as it exits the skull. Diagnosis involves identifying the problem with the facial nerve through clinical examination. Most cases of Bell's palsy resolve on their own within 6 months, while some people develop permanent weakness

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    2. Facial Paralysis

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    Facial paralysis can be caused by central or peripheral lesions of the facial nerve. Peripheral lesions are more common, accounting for about 2/3 of cases. Bell's palsy is the most common cause of peripheral facial paralysis, characterized by sudden onset of weakness or paralysis of the muscles on one side of the face. It is usually idiopathic but can be caused by infections like herpes zoster oticus. Trauma from temporal bone fractures can also cause facial paralysis by damaging the facial nerve as it exits the skull. Diagnosis involves identifying the problem with the facial nerve through clinical examination. Most cases of Bell's palsy resolve on their own within 6 months, while some people develop permanent weakness

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    3 views7 pages

    Facial Paralysis

    Uploaded by

    RISHAV BHAGAT
    Facial paralysis can be caused by central or peripheral lesions of the facial nerve. Peripheral lesions are more common, accounting for about 2/3 of cases. Bell's palsy is the most common cause of peripheral facial paralysis, characterized by sudden onset of weakness or paralysis of the muscles on one side of the face. It is usually idiopathic but can be caused by infections like herpes zoster oticus. Trauma from temporal bone fractures can also cause facial paralysis by damaging the facial nerve as it exits the skull. Diagnosis involves identifying the problem with the facial nerve through clinical examination. Most cases of Bell's palsy resolve on their own within 6 months, while some people develop permanent weakness

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    of 7

    Facial Paralysis

    Causes:
    ·
    Can be central or
    peripheral
    its
    · The peripheral lesion
    may
    involve the nerve in

    intracranial, intratemporal or extratemporal parts.

    Peripheral lesions common about 2/3rd


    of
    ·
    are more

    hem
    ↓
    are
    of Idiopathic variety.

    Examples:
    (A) Central -

    i Brain abscess

    iii) Poutine gliomas


    viii) Poliomyelin's
    sclerosis
    live Multiple

    (B) Intracranial part


    -
    (i Aconetic Neuroma

    (ii) Meningioma
    cholesteatoma
    iii) Congenital
    ive Metastatic carvinoma

    (x)
    Meningh's
    (c) Intratemporal Part -

    Idiopathic. nfections:
    -I

    vi Bell's palsy (i) AOM

    ii) COM
    iii) Melkersson syndrome zostesoticus
    (iii) Herpes
    in Ollis externa
    Trauma o Malignant
    is surgical -

    Mastoidectomy & Stapedeclomy


    ii) Accidental - Fractures of Temporal bone
    Neoplasms:
    Ext. Middle
    i
    Malignancies of ear

    ii) Glomu jugular tumor


    viii) Facial Nerve neuroma

    to bone
    (iv) Metastasis Temporal

    (P) Extracranial Part -

    (i)
    Malignancy of Parolid
    ii) Surgery of Parohid
    (iii) Accidental injury
    Neonatal
    in facial injury

    (E)
    Systemic Diseases -

    is Diabetes mellitus

    ii) Hypothyroidism
    viiis Uraemia

    nodosa
    is Polyarteria's
    1x
    Kegener's
    xi) Sarcoidosis

    (vii) Leprosy
    (viii) Leukaemia
    disease
    lix Demyelinating
    Bell’s Palsy
    Weakness Paralysis of nucles
    of the
    face.
    ·

    or

    to 7th Cranial Nerve Facial Nerve.


    Damage
    ·
    -

    Idiopathic (unknown camel

    IFialNere
    -

    Lacrimal Gland
    -

    Submingual Glowd

    -
    Submandibular Gland
    -
    In Ear ->Stapeding nucle

    -
    Taste (Anterior 2/3rds Tongue)

    Innervahia ⑰

    Gets Damaged by ②

    "FatEwtechinreplen ⑤
    -

    Epstein-Barr
    -
    Varicella Zoster ④

    12 materia ⑤
    -borrelia
    burgdorferi -

    Weakness / Paralysis - Facial Nerve.


    Symphome;
    1
    Drooping Eyelid
    (2) Abseney Nasolabial Fold

    13)
    Drooping mouth
    Dayness in affected mouth
    14)
    &
    Eye.
    (5)
    Hypersensitivity to loud Noices
    16 Taste Sensation
    Lossy on

    Anterior 23 is
    Tongue
    Diagnosis.
    ·

    Identifying problem with


    facial
    Nerve (Not stroke or Brain tumor
    Most within Months
    people 6
    ·

    recover

    Some
    people develop permanent weakness / Paralysis
    ·

    Treatments:
    ·
    Not needed in all cares

    · Severcomes - Corticosteroids can

    Reduce Nerve Inflammation.

    Tclinical diagnosis
    (MRI for
    Belle
    Paley
    Melkersson Syndrome

    HERPES ZOSTER OTICUS (RAMSAY–HUNT SYNDROME

    I
    Trauma
    ·
    Fractures of Temporal Bone.
    A

    Can be mixed
    longitudinal, transverse or

    Facial
    ·

    paby is seen more


    often in Transverse
    fractures (50%)

    longitudinal fracture:
    · Passesthrough the roof of external

    auditory canal, middle ear & anterior

    to inner ear

    · Conduction hearing loss

    ·
    Rophene of TM

    Middle cranial
    · CSF Leak through
    Fossa ->

    Tegman tympani - CSF

    & Howhea

    transverse Frachere:

    · TM intact

    will collect in middle Eustachian Tobe


    · CSF
    ear-Through
    I,
    CSF Oftorhinorrhea
    Frachene of Temporal bone:

    is Hemotympanum
    behind Battle
    (ii) Bruising &
    Exchymosis ear -

    sign.

    (Hemotympanum)

    & Battle sign)

    C
    MedNotes [ www.mednotes.in ]
    Full MedNotes : mednotesebooks.company.site
    App Available on PlayStore

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