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Evaluation and Applicability of Counterconditioning in Fear Extinction
Evaluation and Applicability of Counterconditioning in Fear Extinction
Extinction
Boris Kitanov
28.04.2023
Word count:
In the ‘’Fear Extinction and Relapse: State of the Art’’ by (Vervliet et el., 2013) is provided a
thorough insight of the Pavlovian classical conditioning which is fundamental for treating
anxiety disorder and phobias. Of which the core idea is to avoid a relapse of an already
extinct fear or replace it with something that would erase the undesired feeling from the
modified as a trigger for it is in the core of the fear, which makes counterconditioning
suitable and effective when it comes to erasing the feeling associated with a certain context
by enhancing it with a pleasant stimulus. Hence, it plays a vital role in fear extinction as well
as avoiding unpredicted shifts and relapse in behaviour. However, fear extinction could take
time to remember, merely because the new memory is more recent while the memory of fear
has been there for longer period which makes it stronger and outweighs the novel modified
more pleasant one. Thus, there is a high chance of reoccurrence of the extinct response
(Pavlov 1927, as cited in Vervliet et el., 2013, p. 218). It is important because nowadays
thousands of people experience phobias, anxiety, and many other mental problems related to
conditioning in fear extinction. In this essay, I argue that fear extinction can be most
counterconditioning. Where the context which triggers the fear is paired with a positive or
neutral stimulus to avoid relapse of the unconditioned fear stimulus and increase the chance
of long-term extinction.
The classical conditioning initially coined by Pavlov after conducting an example of a pre-
clinical trial on his dog. Where he establishes how different stimulus work such as
conditioned stimulus, unconditioned stimulus, and neutral stimulus where such modifications
in behaviour first were created in process called the synaptic plasticity. Which have a vital
role in fear extinction and the change of the behaviour of the stimulus. The control over the
stimulus is executed in series of clinical trials with the patient and exposure to US in the same
and different context until the fear response decreases or in very little number of cases it is
completely erased. To test the level of extinction, hence renewal test is presented in ABB and
ABA variations in which a few steps are followed (Vervliet et el., 2013, pp. 221-222). Such
test are initially done in pre clinical trials first trials with animals are executed mostly rodents,
which are done to establish the safety of the methods before applied on humans. For example,
Vansteenwegen et al., (2005, pp. 331-332) displays the charts and results from ABA and
AAA groups with four block tests M1-acquisition, M2-second acquisition, M3-extinction,
M4-test where the AAA group had a much higher CS+ score than the ABA which could lead
to increase in the chances of a relapse. Suggesting that relapse of fear is almost inevitable.
Furthermore, part of the pre-clinical trials in distinguishing some cognitive disorders such as
attention deficit hyperactivity disorder (ADHD), schizophrenia, and traumatic brain injury
mentioned above making it more effective due its enhancements in the methodology of fear
extinction. The concept of counterconditioning was first developed in the experiment called
the ‘’Little Peter” by Mary Cover Jones (1924) which was noted as the earliest behavioural
modification (Keller et al., 2020, p. 2). The term ‘’counterconditioning’’ was coined by
Joseph Wolpe in 1950 during the process of replacing anxiety and phobias response with
desirable feelings which was extracted from the model of classical conditioning (Mcleod,
2022, November 3). The core idea of counterconditioning (CC) is first to pair both stimulus
until a later phase of complete replacement of the unconditioned stimulus (US) negative
stimulus with US positive stimulus (e.g., food, music, favourite cloth, etc.) (Keller et al.,
2020, p. 2). This method aims to avoid what exposure therapy proposes ‘’ systematic
desensitization, individuals progress through increasingly more anxiety’’ (Craske et al., 2012,
p.2) while in the counterconditioning the anxiety is decreased. Thus, the counterconditioning
in clinical conditions would be conducted in the following order: person with phobia or fear
not only exposed to it as in the exposure therapy, but appetitive is introduced simultaneously
with it. Which is the main reason why it is suggested to be more effective. During the
appetitive phase where the prediction error system leads to dopamine release in the ventral
tegmental area (VTA) located at the back part of the brain through the Ventral Straitum to the
of the amygdala (Keller et al., 2020, pp. 5-6). In this lengthy looking process, the fear
extinction is reduced, in short ‘’the higher the dopamine, lower is the possibility of a
relapse’’. For instance, a person with fear of dogs is put in a room with a calm and friendly
dog while being given their favourite food which dominates the feeling of fear by releasing
dopamine through the reward system (explained above). Furthermore, the
efficiency in manner of combing two different stimulus and solidifying the prediction error
According to Button (2004, p. 2) the ‘’saved’’ original learning would not be remembered as
well as during a counterconditioning process because the memory would be modified by the
appetitive presented in the specific context. Although, studies also suggest that
extinction should be short term treatment to ease the fears or phobias of a person, with an in-
depth focus on kids (Wolpe, 1956, pp. 236-239). Nowadays, it is used more often with kids
On the other hand, counterconditioning is based on the release of a vital for our existence
chemical, the dopamine. Without the dopamine we would not be motivated to do anything
but with enough of it a person can not only overcome a fear or phobia but do much better in
life as suggested in the book The Molecule of More by Dr. Lieberman. As mentioned above
the key difference in the counterconditioning is that it uses this dopamine release as fear
phobias (Garcia, 2017), until extinction is reached. Finally, this method suggests one less
harmful, lengthier, and more natural manner of fear extinction, by the use of dopamine which
is part of our daily life and turned into medication by being manipulated in various ways.
Reference list:
Bouton, M. E. (2004). Context and Behavioral Processes in Extinction. Learning & Memory, 11(5),
485–494. https://doi.org/10.1101/lm.78804
Keller, N. E., Hennings, A. C., & Dunsmoor, J. E. (2020). Behavioral and neural processes in
counterconditioning: Past and future directions. Behaviour Research and Therapy, 125,
103532. https://doi.org/10.1016/j.brat.2019.103532
Vervliet, B., Craske, M. G., & Hermans, D. (2013). Fear Extinction and Relapse: State of the Art.
clinpsy-050212-185542
M. Craske, Liao, B., Brown, L., & B. Vervliet. (2017). Role of Inhibition in Exposure Therapy.
Inhibition-in-Exposure-Therapy-Craske-Liao/
fc05d73a23a5dc4d45a4911ea63a2666bba73515
https://metallicman.com/wp-content/uploads/asgarosforum/5912/The-Molecule-of-More-by-
Daniel-Z.-Lieberman-Michael-E.-Long-pdf-version.pdf
Garcia, R. (2017b). Neurobiology of fear and specific phobias. Learning & Memory, 24(9), 462–471.
https://doi.org/10.1101/lm.044115.116